Добірка наукової літератури з теми "Tumeurs du cerveau – Enfant"
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Статті в журналах з теми "Tumeurs du cerveau – Enfant":
Pawluski, Jodi, and Pascal Vrticka. "Le cerveau parental : structure, fonction et synchronisation intercérébrale parent-enfant." Spirale N° 107, no. 3 (January 16, 2024): 127–37. http://dx.doi.org/10.3917/spi.107.0127.
Nguyen, D. T., M. Felix-Ravelo, and R. Jankowski. "Kystes à l’interface tumeur-cerveau et tumeurs nasosinusiennes." Annales françaises d'Oto-rhino-laryngologie et de Pathologie Cervico-faciale 131, no. 5 (November 2014): 307–8. http://dx.doi.org/10.1016/j.aforl.2014.07.008.
de Ribaupierre, S., O. Vernet, M. Vinchon, and B. Rilliet. "Phacomatoses et tumeurs génétiquement déterminées : la transition enfant–adulte." Neurochirurgie 54, no. 5 (October 2008): 642–53. http://dx.doi.org/10.1016/j.neuchi.2008.07.004.
Rapin, I. "modularité du cerveau et du développement du petit enfant: perspective neuropsychologique." Journal de Pédiatrie et de Puériculture 10, no. 8 (December 1997): 494–95. http://dx.doi.org/10.1016/s0987-7983(97)80480-x.
Marcelli, D. "Regard, langage et subjectivité: Comment le cerveau d’un enfant peut dire « je »”?" Neuropsychiatrie de l'Enfance et de l'Adolescence 60, no. 5 (July 2012): S5. http://dx.doi.org/10.1016/j.neurenf.2012.04.028.
Baud, Olivier, Marit Knoop, Alice Jacquens, and Marie-Laure Possovre. "Ocytocine : une nouvelle cible de neuroprotection ?" Biologie Aujourd’hui 216, no. 3-4 (2022): 145–53. http://dx.doi.org/10.1051/jbio/2022012.
Vallélian, Joëlle. "Projet Insula : Création de marionnettes et d'outils de médiation symboliques pour aider l'enfant qui a un Trouble du Spectre de l'Autisme à exercer les habiletés sociales." Cortica 1, no. 1 (March 21, 2022): 161–75. http://dx.doi.org/10.26034/cortica.2022.1943.
Castagna, J., J. Clerc, A. S. Dupond, and C. Laresche. "Tumeurs à cellules granuleuses multiples chez un enfant atteint d’un syndrome de Noonan compliqué de leucémie myélomonocytaire juvénile." Annales de Dermatologie et de Vénéréologie 144, no. 11 (November 2017): 705–11. http://dx.doi.org/10.1016/j.annder.2017.06.008.
Kane, Bourama, and Et Al. "Des abcès cérébraux révélateurs d’un ventricule droit à double issue chez un enfant de 5 ans dans le service de pédiatrie de l’Hôpital du Mali." Revue Malienne d'Infectiologie et de Microbiologie 17, no. 1 (April 30, 2022): 54–60. http://dx.doi.org/10.53597/remim.v17i1.2227.
Muscatelli, Françoise. "L’ocytocine, dès la naissance, conditionne le comportement alimentaire et social d’un individu." Biologie Aujourd’hui 216, no. 3-4 (2022): 131–43. http://dx.doi.org/10.1051/jbio/2022017.
Дисертації з теми "Tumeurs du cerveau – Enfant":
Lefevre, Philippe. "Tumeurs cérébrales primitives de l'enfant : expérience du service de cancérologie de Bordeaux." Bordeaux 2, 1994. http://www.theses.fr/1994BOR23088.
SUC, ETIENNE. "Tumeurs cerebrales chez les enfants de moins de trois ans : etude retrospective de 98 cas traites a l'institut gustave roussy." Toulouse 3, 1990. http://www.theses.fr/1990TOU31082.
DE, SURVILLE BRESSAC SANDRINE. "Tumeurs cerebrales chez des enfants irradies pour l. A. L." Nantes, 1993. http://www.theses.fr/1993NANT256M.
OLMOS, GRAZIELLA. "Tumeur et epilepsie : signes revelateurs exclusifs d'une maladie de bourneville : a propos de 2 cas." Aix-Marseille 2, 1990. http://www.theses.fr/1990AIX20236.
Ogunleke, Abiodun. "Imagerie chimique 3D de tumeurs du cerveau." Thesis, Bordeaux, 2019. http://www.theses.fr/2019BORD0040/document.
Three-dimensional (3D) histology is a new advanced tool for cancerology. The whole chemical profile and physiological characteristics of a tissue is essential to understand the rationale of pathology development. However, there is no analytical technique, in vivo or histological, that is able to discover such abnormal features and provide a 3D distribution at microscopic resolution.Here, we introduce a unique high- throughput infrared (IR) microscopy method that combines automated image correction and subsequent spectral data analysis for 3D-IR image reconstruction. I performed spectral analysis of a complete organ for a small animal model, a mouse brain with animplanted glioma tumor. The 3D-IR image is reconstructed from 370 consecutive tissue sectionsand corrected using the X-ray tomogram of the organ for an accurate quantitative analysis of thechemical content. A 3D matrix of 89 x 106 IR spectra is generated, allowing us to separate the tumor mass from healthy brain tissues based on various anatomical, chemical, and metabolic parameters. I demonstrate for the first time that quantitative metabolic parameters (glucose, glycogen and lactate) can be extracted and reconstructed in 3D from the IR spectra for the characterization of the brain vs. tumor metabolism (assessing the Warburg effect in tumors). Our method can be further exploited by searching for the whole spectral profile, discriminating different anatomical landmarks in the brain. I demonstrate this by the reconstruction of the corpus callosum and basal ganglia region of the brain
DEBAVELAERE, VINCENT. "Polyamines erythrocytaires et tumeurs cerebrales." Lille 2, 1991. http://www.theses.fr/1991LIL2M048.
Rakotomalala-Andrianasolo, Andria. "Développement et caractérisation de modèles cellulaires pour l'étude du rôle de l'oncohistone H3.3 K27M dans le phénotype agressif et la réponse aux thérapies des gliomes pédiatriques diffus de la ligne médiane." Electronic Thesis or Diss., Université de Lille (2022-....), 2024. http://www.theses.fr/2024ULILS015.
H3K27-altered DMG treatment is one of the most significant challenges in pediatric neuro-oncology,with no improvement in patient survival over the past 50 years. In 2012, it was shown that DMGs harbor a specific histone 3 mutation (oncohistone) called H3.3 K27M with a very high prevalence (70-80% of cases). Although theH3.3 K27M impact on the epigenetic landscape has been well described, studies are needed to understand betterits role in DMG cells’ aggressiveness and response to therapies.To study the H3.3 K27M mutation impact on DMG cell phenotypes precisely, we developed andcharacterized pediatric high-grade glioma isogenic cellular models induced and knock-out for the oncohistone.Using these models, we aimed to decipher the oncohistone impact on DMG cell biology and response to anticancertherapies, including radiation therapy, the only current standard of care for DMGs. Characterization of our H3.3 K27M-induced pediatric supratentorial glioma cell lines reveals that the oncohistone affects the response to ionizing radiations and specific targeted therapies in a cellular context-dependentway. Based on these results, we settled to characterize oncohistone biological impacts in a more relevant cellular context of DMG. In that sense, we established H3.3 K27M knock-out cellular models and characterized them regarding their parental DMG H3.3 K27M mutated cell lines. Through omic (transcriptomic and proteomic)and cell metabolism characterizations of these models, we notably showed the H3.3 K27M mutation impact on DMG cells’ lipid metabolism. In 3D spheroid models, this H3.3 K27M-induced lipid metabolism rewiring appeared conditioned by microenvironment factors still under investigation.On the other hand, a functional pharmacological screen identified H3.3 K27M-driven dependencies tospecific DNA repair pathways. In addition, ongoing radiobiological characterization of our models indicates anH3.3 K27M-associated radiosensitivity correlating with a decrease in DNA repair efficiency following ionizingradiations. Beyond this DNA repair impact, our pharmacological screen also revealed an H3.3 K27M-relatedsensitivity to cardiac glycoside drugs. This result makes sense with our transcriptomic data showing enrichmentin genes involved in cardiomyopathies and ion homeostasis among differentially expressed genes with theoncohistone. In this context, we began unraveling the molecular and biological processes underlying thisH3.3 K27M-driven effect.Finally, we used our isogenic cellular models to show that the H3.3 K27M oncohistone drives lipidmetabolism modifications. These metabolic changes could prime H3K27-altered DMG cells to specific regulatedcell death pathways (e.g., ferroptosis) and affect the response to certain therapies. Moreover, the H3.3 K27Mseems to drive specific sensitivities, notably to radiation therapy and cardiac glycoside drugs. Understanding the underlying molecular mechanisms governing these H3.3 K27M-associated Achille heels could highlight newinsights into the oncohistone role in DMG cells and provide rationales for developing new therapeutic strategies
DUFONT, PHILIPPE, and BIQUILLON CATHERINE CARIN. "La neurocysticercose chez l'enfant." Lille 2, 1989. http://www.theses.fr/1989LIL2M117.
Favier, Olivier. "Le medullobastome de l'enfant : etude retrospective sur une serie de 79 cas." Aix-Marseille 2, 1992. http://www.theses.fr/1992AIX20154.
VEINANTE, ROCHE MARTINE. "Les tumeurs cardiaques de l'enfant." Besançon, 1994. http://www.theses.fr/1994BESA3050.
Книги з теми "Tumeurs du cerveau – Enfant":
E, Cohen Michael. Brain tumors in children: Principles of diagnosis and treatment. 2nd ed. New York: Raven Press, 1994.
Violet, Lydie. La vie sauve. [Paris]: Points, 2006.
Claudine, Amiel-Tison, and Stewart Ann Dr, eds. L' enfant nouveau-né: Un cerveau pour la vie. Paris: Editions INSERM, 1995.
Connolly, Barbara. A sense of tumour: A collection of works by members and friends of the Brain Tumour Support Group. Dublin: E print Limited, 2000.
Shlanger, Dafi. Brain surgeons don't do facelifts: A woman's journey through brain surgery. Central Milton Keynes, Buckinghamshire: AuthorHouse, 2008.
Lacoursière, Louise. Lunes bleues: Roman. Montréal: Libre expression, 2008.
Brun, Danièle. L' enfant donné pour mort. Paris: Eshel, 2001.
Siegel, Daniel J. Le cerveau de votre enfant: Manuel d'éducation positive pour parents d'aujourd'hui. Laval (Québec) Canada: Guy Saint-Jean éditeur, 2015.
Servan-Schreiber, David. On peut se dire au revoir plusieurs fois. Paris: Pocket, 2012.
Hart, Elizabeth. Maman, pourquoi a dix ans je dois mourir? Paris: Garanciere, 1987.