Готові списки джерел за темами / Sudden infant death syndrome – Etiology; Sudden infant death syndrome – Pathogenesis

Добірка наукової літератури з теми "Sudden infant death syndrome – Etiology; Sudden infant death syndrome – Pathogenesis"

Автор: Grafiati

Опубліковано: 10 грудня 2022

Оновлено: 28 січня 2023

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Статті в журналах з теми "Sudden infant death syndrome – Etiology; Sudden infant death syndrome – Pathogenesis"

Pisaniak, Paulina, Dominika Piękoś, Katarzyna Bąk, Patryk Stokłosa, and Dorota Ozga. "The battle with uneven opponent – Sudden Infant Death Syndrome." Pielegniarstwo XXI wieku / Nursing in the 21st Century 18, no. 2 (June 1, 2019): 132–35. http://dx.doi.org/10.2478/pielxxiw-2019-0013.

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AbstractSIDS is one of the biggest problems of modern medicine. In the diagnosis of SIDS, we take into account all possible diseases that may be the cause of death, as well as factors indicating an accident or murder. The etiology of SIDS is not yet known. There are several pathogenetic concepts, most of which refer to pathophysiological changes associated with nervous system hypoplasia. The most important risk factors include the effects of tobacco smoke, obstetric history, and incorrect sleep position. The role of risk factors in the pathogenesis of SIDS and their interdependence is still the subject of many studies. There are many theories developed on this subject, but none have been supported by scientific research and which is extremely difficult to carry out in this group of newborns. In most cases, medical help finds a newborn already dead, so it is difficult to say what is the main cause or marker of cot death. A considerable success in preventing the onset of sudden infant death syndrome turned out to be educational campaigns for parents - in order to follow up, an information leaflet was prepared with the basic recommendations in the prevention of SIDS. Among the parents of newborn children there are still many controversial opinions about risk factors in the onset of sudden infant death syndrome, the article contains and explains the meaning of individual activities that are considered to predispose to SIDS.

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ben-Aaron, M. "Sudden infant death syndrome: a cybernetic etiology." Medical Hypotheses 61, no. 5-6 (November 2003): 601–4. http://dx.doi.org/10.1016/s0306-9877(03)00242-1.

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Byard, Roger W., and Henry F. Krous. "Sudden Infant Death Syndrome: Overview and Update." Pediatric and Developmental Pathology 6, no. 2 (March 2003): 112–27. http://dx.doi.org/10.1007/s10024-002-0205-8.

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The past decade and a half has seen marked changes in the epidemiology of sudden infant death syndrome (SIDS). The avoidance of certain risk factors such as sleeping prone and cigarette smoke exposure has resulted in the death rate falling dramatically. Careful evaluation of environmental factors and endogenous characteristics has led to a greater understanding of the complexities of the syndrome. The development and implementation of death scene and autopsy protocols has led to standardization in approaches to unexpected infant deaths with increasing diagnoses of accidental asphyxia. Despite these advances, there is still confusion surrounding the diagnosis, with deaths being attributed to SIDS in many communities and countries where death scene investigations and autopsies have not been conducted. The following review provides a brief overview of the historical background, epidemiology, pathology, and pathogenesis of SIDS. Contentious issues concerning the diagnosis and current problems are discussed. Despite calls to abandon the designation, SIDS remains a viable term for infants who die in their sleep with no evidence of accident, inflicted injury, or organic disease after a full investigation has been conducted according to standard guidelines.

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Perrone, Serafina, Chiara Lembo, Sabrina Moretti, Giovanni Prezioso, Giuseppe Buonocore, Giorgia Toscani, Francesca Marinelli, Francesco Nonnis-Marzano, and Susanna Esposito. "Sudden Infant Death Syndrome: Beyond Risk Factors." Life 11, no. 3 (February 26, 2021): 184. http://dx.doi.org/10.3390/life11030184.

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Sudden infant death syndrome (SIDS) is defined as “the sudden death of an infant under 1 year of age which remains unexplained after thorough investigation including a complete autopsy, death scene investigation, and detailed clinical and pathological review”. A significant decrease of SIDS deaths occurred in the last decades in most countries after the beginning of national campaigns, mainly as a consequence of the implementation of risk reduction action mostly concentrating on the improvement of sleep conditions. Nevertheless, infant mortality from SIDS still remains unacceptably high. There is an urgent need to get insight into previously unexplored aspects of the brain system with a special focus on high-risk groups. SIDS pathogenesis is associated with a multifactorial condition that comprehends genetic, environmental and sociocultural factors. Effective prevention of SIDS requires multiple interventions from different fields. Developing brain susceptibility, intrinsic vulnerability and early identification of infants with high risk of SIDS represents a challenge. Progress in SIDS research appears to be fundamental to the ultimate aim of eradicating SIDS deaths. A complex model that combines different risk factor data from biomarkers and omic analysis may represent a tool to identify a SIDS risk profile in newborn settings. If high risk is detected, the infant may be referred for further investigations and follow ups. This review aims to illustrate the most recent discoveries from different fields, analyzing the neuroanatomical, genetic, metabolic, proteomic, environmental and sociocultural aspects related to SIDS.

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Leiter, J. C., and Ines Böhm. "Mechanisms of pathogenesis in the Sudden Infant Death Syndrome." Respiratory Physiology & Neurobiology 159, no. 2 (November 2007): 127–38. http://dx.doi.org/10.1016/j.resp.2007.05.014.

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Glinge, Charlotte, Sára Rossetti, Louise Bruun Oestergaard, Niels Kjær Stampe, Thomas Hadberg Lynge, Regitze Skals, Bo Gregers Winkel, et al. "Risk of Sudden Infant Death Syndrome Among Siblings of Children Who Died of Sudden Infant Death Syndrome in Denmark." JAMA Network Open 6, no. 1 (January 25, 2023): e2252724. http://dx.doi.org/10.1001/jamanetworkopen.2022.52724.

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ImportanceSudden infant death syndrome (SIDS) remains a leading cause of death during the first year of life. The etiology of SIDS is complex and remains largely unknown.ObjectiveTo evaluate whether siblings of children who died of SIDS have a higher risk of SIDS compared with the general pediatric population.Design, Setting, and ParticipantsThis register-based cohort study used Danish nationwide registers. Participants were all infants (&lt;1 year) in Denmark between January 1, 1978, and December 31, 2016, including siblings of children who died of SIDS. Siblings were followed up from the index cases’ date of SIDS, date of birth, or immigration, whichever came first, and until age 1 year, emigration, developing SIDS, death, or study end. The median (IQR) follow-up was 1 (1-1) year. Data analysis was conducted from January 2017 to October 2022.Main Outcomes and MeasuresStandardized incidence ratios (SIRs) of SIDS were calculated with Poisson regression models relative to the general population.ResultsIn a population of 2 666 834 consecutive births (1 395 199 [52%] male), 1540 infants died of SIDS (median [IQR] age at SIDS, 3 [2-4] months) during a 39-year study period. A total of 2384 younger siblings (cases) to index cases (first sibling with SIDS) were identified. A higher rate of SIDS was observed among siblings compared with the general population, with SIRs of 4.27 (95% CI, 2.13-8.53) after adjustment for sex, age, and calendar year and of 3.50 (95% CI, 1.75-7.01) after further adjustment for mother’s age (&lt;29 years vs ≥29 years) and education (high school vs after high school).Conclusions and RelevanceIn this nationwide study, having a sibling who died of SIDS was associated with a 4-fold higher risk of SIDS compared with the general population. Shared genetic and/or environmental factors may contribute to the observed clustering of SIDS. The family history of SIDS should be considered when assessing SIDS risk in clinical settings. A multidisciplinary genetic evaluation of families with SIDS could provide additional evidence.

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Kaler, Jasndeep, Azhar Hussain, and Stanley Lee. "Manifestation and Pathogenesis of Sudden Infant Death Syndrome: A Review." Critical Reviews in Eukaryotic Gene Expression 30, no. 2 (2020): 111–20. http://dx.doi.org/10.1615/critreveukaryotgeneexpr.2020033009.

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Wilders, Ronald. "Cardiac Ion Channelopathies and the Sudden Infant Death Syndrome." ISRN Cardiology 2012 (December 5, 2012): 1–28. http://dx.doi.org/10.5402/2012/846171.

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The sudden infant death syndrome (SIDS) causes the sudden death of an apparently healthy infant, which remains unexplained despite a thorough investigation, including the performance of a complete autopsy. The triple risk model for the pathogenesis of SIDS points to the coincidence of a vulnerable infant, a critical developmental period, and an exogenous stressor. Primary electrical diseases of the heart, which may cause lethal arrhythmias as a result of dysfunctioning cardiac ion channels (“cardiac ion channelopathies”) and are not detectable during a standard postmortem examination, may create the vulnerable infant and thus contribute to SIDS. Evidence comes from clinical correlations between the long QT syndrome and SIDS as well as genetic analyses in cohorts of SIDS victims (“molecular autopsy”), which have revealed a large number of mutations in ion channel-related genes linked to inheritable arrhythmogenic syndromes, in particular the long QT syndrome, the short QT syndrome, the Brugada syndrome, and catecholaminergic polymorphic ventricular tachycardia. Combining data from population-based cohort studies, it can be concluded that at least one out of five SIDS victims carries a mutation in a cardiac ion channel-related gene and that the majority of these mutations are of a known malignant phenotype.

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Meny, Robert G., John L. Carroll, Mary Terese Carbone, and Dorothy H. Kelly. "Cardiorespiratory Recordings From Infants Dying Suddenly And Unexpectedly at Home." Pediatrics 93, no. 1 (January 1, 1994): 44–49. http://dx.doi.org/10.1542/peds.93.1.44.

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Objective. Little is known about the mechanism of death during sudden infant death. To study the mechanism, we obtained data on six infants who died while on a memory-equipped cardiorespiratory monitor. Methods. Waveforms of respiration and heart rate trend were available for five infants; an alarm log only was available for the sixth. These printouts were reviewed with attention to mechanism and time to death. Results. All infants were born prematurely; autopsies reported the cause of death as sudden infant death syndrome in three cases and bronchopulmonary dysplasia in the others. Bradycardia, which played a more prominent role than central apnea, was preceded by tachycardia in two deaths. Resuscitation occurred within 1 minute in four cases; no response to alarms occurred in the other two cases, apparently because the parents were desensitized by prior meaningless alarms. Five patients died within 20 minutes, whereas one death due to sudden infant death syndrome was prolonged. Conclusion. Bradycardia is an important feature in all six of these infant deaths. Although its etiology is unknown, hypoxemia or obstructive apnea may precede bradycardia. Home monitors equipped to detect these possible antecedents will yield further insight into sudden infant death.

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Ponsonby, A. L., T. Dwyer, and M. E. Jones. "Sudden infant death syndrome: seasonality and a biphasic model of pathogenesis." Journal of Epidemiology & Community Health 46, no. 1 (February 1, 1992): 33–37. http://dx.doi.org/10.1136/jech.46.1.33.

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Дисертації з теми "Sudden infant death syndrome – Etiology; Sudden infant death syndrome – Pathogenesis"

Byard, Roger William. "Sudden natural death in infancy and early childhood : an analysis of aetiological mechanisms and pathological features / Roger W. Byard." 1993. http://hdl.handle.net/2440/38619.

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Includes bibliographic references.
2 v. :
Title page, contents and abstract only. The complete thesis in print form is available from the University Library.
A detailed description of possible aetiological mechanisms in sudden infant death syndrome and reports original work investigating a variety of causal mechanisms and pathological markers.
Thesis (M.D.)--University of Adelaide, Dept. of Paediatrics, 1994?

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Byard, Roger William. "Sudden natural death in infancy and early childhood : an analysis of aetiological mechanisms and pathological features / Roger W. Byard." Thesis, 1993. http://hdl.handle.net/2440/38619.

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Includes bibliographic references.
2 v. :
A detailed description of possible aetiological mechanisms in sudden infant death syndrome and reports original work investigating a variety of causal mechanisms and pathological markers.
Thesis (M.D.) -- University of Adelaide, Dept. of Paediatrics, [1994?]

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Paulino, Inês Pereira. "Current concepts on the pathogenesis of sudden infant death syndrome." Master's thesis, 2017. http://hdl.handle.net/10362/44175.

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Registo Grau Estrangeiro Medicina (Medical Faculty, Pavol Jozef Šafárik University in Košice, Slovakia.). A dissertação em questão cumpre o disposto no nº2 do artº 11º da Portaria nº 29/2008 de 10 de janeiro (alterada pela Portaria nº 227/2017 de 20 de julho)

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Книги з теми "Sudden infant death syndrome – Etiology; Sudden infant death syndrome – Pathogenesis"

Langwith, Jacqueline. Sudden infant death syndrome. Detroit: Greenhaven Press, 2011.

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Crib death: The sudden infant death syndrome. 2nd ed. Mount Kisco, N.Y., USA: Futura Pub. Co., 1989.

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Crib death: The sudden infant death syndrome. 3rd ed. Armonk, NY: Futura Pub. Co., 1995.

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W, Tyler James, ed. Sudden infant death (S.I.D.S.): Probable causes & simple prevention. New York: Sterling Impact Book, 1986.

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Sudden infant death syndrome: The possible role of "the fear paralysis reflex". Oslo: Norwegian University Press, 1986.

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The cot death: Cover-up? Auckland, N.Z: Penguin Books, 1996.

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Hogan, Glenn P. The unified theory of AIDS: Reconciling Duesberg, Root-Bernstein, and the HIV : with postscript unexplained stillbirths and sudden infant death. Ann Arbor, MI: G.P. Hogan, 1998.

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Lavezzi, Anna M., and Conrad E. Johanson, eds. New Approaches to the Pathogenesis of Sudden Intrauterine Unexplained Death and Sudden Infant Death Syndrome. Frontiers Media SA, 2017. http://dx.doi.org/10.3389/978-2-88945-301-6.

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Medicine, Institute of. Immunization Safety Review: Vaccinations and Sudden Unexpected Death in Infancy. National Academy Press, 2003.

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(Editor), Kathleen Stratton, Donna A. Almario (Editor), Theresa M. Wizemann (Editor), and Marie C. McCormick (Editor), eds. Immunization Safety Review: Vaccinations and Sudden Unexpected Death in Infancy. National Academies Press, 2003.

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Частини книг з теми "Sudden infant death syndrome – Etiology; Sudden infant death syndrome – Pathogenesis"

Wiemann, Martin, Stilla Frede, Frank Tschentscher, Heidrun Kiwull-Schöne, Peter Kiwull, Dieter Bingmann, Bernd Brinkmann, and Thomas Bajanowski. "NHE3 in the Human Brainstem: Implication for the Pathogenesis of the Sudden Infant Death Syndrome (SIDS)?" In Integration in Respiratory Control, 508–13. New York, NY: Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-73693-8_89.

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Yanger, Sheryl. "Sudden Infant Death Syndrome." In Pediatric Emergencies, 35–36. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190073879.003.0004.

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Sudden infant death syndrome (SIDS) is the third leading cause of infant mortality in the United States. SIDS is defined as the sudden death of an infant younger than age 1 year that cannot be explained after a thorough investigation, including autopsy, scene investigation, and clinical history. Despite research and advances in understanding of SIDS, it remains a diagnosis of exclusion after ruling out accidental causes such as suffocation/strangulation and cardiac, infectious, metabolic, or traumatic etiologies. Although the exact pathogenesis is unknown, a working model of SIDS includes a combination of exogenous factors, such as overbundling, prone sleep position, and airway obstruction, and intrinsic factors such as immature cardiorespiratory or arousal systems.

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Rognum, T. O., and R. W. Byard. "SUDDEN INFANT DEATH SYNDROME, ETIOLOGY AND EPIDEMIOLOGY." In Encyclopedia of Forensic and Legal Medicine, 117–29. Elsevier, 2005. http://dx.doi.org/10.1016/b0-12-369399-3/00332-3.

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Byard, R. W., and T. O. Rognum. "Autopsy Findings: Sudden Infant Death Syndrome – Epidemiology and Etiology." In Encyclopedia of Forensic and Legal Medicine, 354–67. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-12-800034-2.00049-5.

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Li, Aihua, Robert A. Darnall, Susan Dymecki, and James C. Leiter. "Animal Models: Illuminating the Pathogenesis of Sudden Infant Death Syndrome." In SIDS Sudden infant and early childhood death: The past, the present and the future, 759–828. University of Adelaide Press, 2018. http://dx.doi.org/10.20851/sids-33.

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