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Добірка наукової літератури з теми "Sleep breathing disorder"

Автор: Grafiati
Опубліковано: 17 листопада 2023

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Статті в журналах з теми "Sleep breathing disorder"

1

Choi, Soo Jeon. "Sleep Breathing Disorder." Tuberculosis and Respiratory Diseases 63, no. 1 (2007): 5. http://dx.doi.org/10.4046/trd.2007.63.1.5.

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2

Koirala, Puru, Narendra Bhatta, Ram Hari Ghimire, Deebya Raj Mishra, Bidesh Bista, and Bhupendra Shah. "Overlap of Sleep Disorders and Chronic Respiratory Diseases: An Emerging Health Dilemma." Nepalese Medical Journal 2, no. 2 (December 22, 2019): 243–49. http://dx.doi.org/10.3126/nmj.v2i2.26007.

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Introduction: The burden of Overlap Syndrome (coexistence of sleep-related breathing disorders in patients with respiratory diseases) is high in developing countries, and such a phenomenon implies higher morbidity. The study was conducted to measure the prevalence of sleep-related breathing disorders in patients with Respiratory Symptom Complex and to identify factors associated with the severity of sleep-related breathing disorders.Materials and Methods: A hospital-based cross-sectional study of 50 patients with respiratory symptom complex was conducted at BP Koirala Institute of Health Sciences. Structured proforma and Polysomnography were used for analysis.Results: There were 24 patients (48%) with COPD, 18 (36%) with Bronchial Asthma. 6 patients (12%) with Bronchiectasis and 2 patients with Interstitial Lung Disease. 60% (n=30) patients had sleep-related breathing disorder or Overlap syndrome, 14 patients (46.67%) had mild sleep-related breathing disorder while 16 (53.33%) patients had moderate to severe type. 62.5% COPD patients, 55.55% Bronchial Asthma patients, 50% of patients with Interstitial Lung Disease and 50% Post-TB Bronchiectasis patients had a sleep-related breathing disorder. There was a significant positive correlation between the presence of sleep-related breathing disorder in patients with respiratory symptom complex and high neck circumference (0.499, p-value <0.001), waist circumference (0.293, p-value = 0.039) and hip circumference (0.371, p-value = 0.008).Conclusions: Overlap Disorders comprising sleep disorders in patients with chronic respiratory diseases are high in developing countries. Routine sleep history and polysomnography in all patients with Respiratory Symptom Complex can detect sleep-related breathing disorders.
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3

Renjen, Pushpendra N., Dinesh Chaudhari, Sudha Kansal, and Mahir Meman. "Stroke-sleep disorder breathing." Apollo Medicine 12, no. 1 (March 2015): 18–21. http://dx.doi.org/10.1016/j.apme.2015.02.008.

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4

Cheng, Philip, Melynda D. Casement, Chiau-Fang Chen, Robert F. Hoffmann, Roseanne Armitage, and Patricia J. Deldin. "Sleep-disordered breathing in major depressive disorder." Journal of Sleep Research 22, no. 4 (January 25, 2013): 459–62. http://dx.doi.org/10.1111/jsr.12029.

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5

Krakow, Barry, Ali Artar, Teddy D. Warner, Dominic Melendrez, Lisa Johnston, Michael Hollifield, Anne Germain, and Mary Koss. "Sleep Disorder, Depression, and Suicidality in Female Sexual Assault Survivors." Crisis 21, no. 4 (July 2000): 163–70. http://dx.doi.org/10.1027//0227-5910.21.4.163.

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The role of sleep in psychiatric illness in general, and depression and suicidality in particular, is poorly understood and has not been well researched despite the pervasiveness of sleep complaints in these conditions. As an exploratory, hypothesis-generating study, female sexual assault survivors with posttraumatic stress disorder (n = 153) who had enrolled in a nightmare-treatment program were assessed for subjectively determined sleep breathing and sleep movement disorders. Diagnoses of potential disorders were based on clinical practice parameters and research algorithms from the field of sleep disorders medicine. Potential sleep breathing and sleep movement disorders were present in 80% of the participants (n = 123) and included three subgroups: sleep-disordered breathing only (n = 23); sleep movement disorder only (n = 45); and both sleep disorders (n = 55). Based on the HamiltonDepression Rating Scale and Suicide subscale, participants with potential sleep disorders suffered greater depression (Cohen's d = .73-.96; p < .01) and greater suicidality (Cohen's d = .57-.78; p < .05) in comparison to participants without potential sleep disorders. The group with both sleep disorders suffered from the most severe depression and suicidality. A provisional hypothesis is formulated that describes how sleep disorders may exacerbate depression and suicidality through the effects of chronic sleep fragmentation.
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6

Han, JinKyu. "0558 Sleep Bruxism and Sleep Breathing Disorder." SLEEP 46, Supplement_1 (May 1, 2023): A246. http://dx.doi.org/10.1093/sleep/zsad077.0558.

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Abstract Introduction Sleep bruxism (SB) is reported by 8-9% of the adult population and is mainly associated with rhythmic masticatory muscle activity (RMMA) characterized by repetitive jaw muscle contractions (3 bursts or more at a frequency of 1 Hz). The purpose of this study was to evaluate the relationship between the nature of sleep bruxism and the sleep breathing disoredr. Methods We prospectively studied 32 patients who were referred to the clinical sleep apnea laboratory for study. They underwent standard nocturnal polysomnographic examination; in addition, masticatory activity was measured with a masseter electromyogram. Patients slept in the supine and lateral decubitus positions. Results Nocturnal clenching was higher in patients with higher respiratory disturbance index. 28 among 32 patients were included in the criteria of obstructive sleep apnea; average respiratory disturbance index (RDI) was 12.7 280 clenches demonstrated in all patients. Conclusion We conclude that there is an association between sleep related breathing disorder and bruxisms that sleep position affects the incidence of both sleep disordered breathing and bruxisms, and that analysis of apneas and hypopneas and clenching events in both supine and lateral decubitus sleeping positions may be helpful. Support (if any)
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7

Jung, Chaewon, Youngkeun Yoo, Hyun-Woo Kim, and Hyun-Chool Shin. "Detecting Sleep-Related Breathing Disorders Using FMCW Radar." Journal of Electromagnetic Engineering and Science 23, no. 5 (September 30, 2023): 437–45. http://dx.doi.org/10.26866/jees.2023.5.r.189.

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Polysomnography (PSG) is currently the sole method for detecting breathing disorders that occur during sleep, such as apnea and hypopnea. However, PSG monitoring is quite inconvenient for test subjects. This study proposes a non-contact method for detecting sleep-related breathing disorders that uses frequency-modulated continuous wave (FMCW) radar. The proposed method effectively extracts respiratory signals from radar signals and uses the degree of reduction in the amplitude of the respiratory signals and the duration of the reduction interval as data to detect breathing disorder intervals. The experimental results show that the respiratory signals extracted using the proposed method are similar to the actual respiratory signals. Furthermore, the breathing disorder intervals detected during sleep using FMCW radar are consistent with the actual apnea and hypopnea intervals as confirmed by the PSG results. These results demonstrate that the proposed method is suitable for non-contact detection of breathing disorders during sleep using FMCW radar.
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8

Pigeon, Wilfred R., and Michael J. Sateia. "Is Insomnia a Breathing Disorder?" Sleep 35, no. 12 (December 2012): 1589–90. http://dx.doi.org/10.5665/sleep.2222.

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9

Janssen, Hennie C. J. P., Lisette N. Venekamp, Geert A. M. Peeters, Angelique Pijpers, and Dirk A. A. Pevernagie. "Management of insomnia in sleep disordered breathing." European Respiratory Review 28, no. 153 (September 30, 2019): 190080. http://dx.doi.org/10.1183/16000617.0080-2019.

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Both obstructive sleep apnoea (OSA) and chronic insomnia disorder are highly prevalent in the general population. Whilst both disorders may occur together by mere coincidence, it appears that they share clinical features and that they may aggravate each other as a result of reciprocally adverse pathogenetic mechanisms. Comorbidity between chronic insomnia disorder and OSA is a clinically relevant condition that may confront practitioners with serious diagnostic and therapeutic challenges. Current data, while still scarce, advocate an integrated and multidisciplinary approach that seems superior over the isolated treatment of each sleep disorder alone.
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Anderson, M., J. Lavela, and T. Mellman. "0457 Sleep-Disordered Breathing Events and Posttraumatic Stress Disorder." Sleep 41, suppl_1 (April 2018): A173. http://dx.doi.org/10.1093/sleep/zsy061.456.

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Дисертації з теми "Sleep breathing disorder"

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Sutton, Amy M. "Executive Function in the Presence of Sleep Disordered Breathing." Digital Archive @ GSU, 2008. http://digitalarchive.gsu.edu/cps_diss/16.

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The purpose of the study was to investigate whether sleep-disordered breathing (SDB) impairs executive functioning in children. Additionally, the study sought to identify the executive functions at risk in SDB and the contribution of daytime sleepiness. SDB represents a spectrum of upper airway conditions that can be mild, such as snoring, or severe, such as obstructive sleep apnea (OSA). Children with these problems may present with excessive sleepiness, failure to thrive, and a variety of cognitive and behavioral dysfunctions including impaired executive functioning. Beebe and Gozal (2002) developed a theoretical model to explain the impact of sleepiness and hypoxia on executive functioning. This model provided a framework to examine links between the medical disorder and the neuropsychological consequences. Twenty-seven children with suspected SDB were tested with polysomnography (PSG) and a neuropsychological battery. Parents completed subjective measures of cognitive function and sleep symptoms. The children were ages 8 to 18 and had no congenital or acquired brain damage. They were matched for age and gender with 21 healthy controls. The executive function protocol included subtests from the Delis-Kaplan Executive Function System (D-KEFS), the digit span subtest from the Wechsler Intelligence Scale for Children (WISC-IV), the Tower of London-II-Drexel University (TOL-II), the Behavioral Rating Inventory of Executive Functioning (BRIEF), and the Conners’ Continuous Performance Test (CPT-II). Statistical analysis was performed using 2 statistical software packages, SAS and NCSS. Regression analysis was used to evaluate all variables. Due to significant group differences in socio-economic status (SES), SES was included as a covariate, along with IQ. No group differences in IQ were found. Significantly less robust executive function in children with SDB was identified in the domains of cognitive flexibility and impulsivity. Additionally, poorer executive planning and overall inattentiveness was also associated with SDB. Level of significance was set at 0.05 and trends (0.05 < p < 0.10) were acknowledged. Other areas of executive function, including working memory, behavioral and emotional inhibition, and processing speed were not associated with SDB. Moreover, academic functioning was significantly lower in children with SDB, although the differences can be shared equally with SDB, SES and IQ.
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2

BALIAN, ARAXI. "EFFECTIVENESS OF ORTHODONTIC TREATMENT WITH PREFABRICATED MYOFUNCTIONAL APPLIANCES IN CHILDREN WITH SLEEP-RELATED BREATHING DISORDERS AND OBSTRUCTIVE SLEEP APNEA: AN 18-MONTH FOLLOW-UP." Doctoral thesis, Università degli Studi di Milano, 2022. http://hdl.handle.net/2434/915635.

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Sleep-Related Breathing Disorders (SRBD) include various clinical entities, from primary snoring to Obstructive Sleep Apnea Syndrome (OSAS). They are characterized by intermittent partial or complete (hypopnea/apnea) upper airway obstruction during sleep which lead to sleep disturbance, cardio-respiratory, and neuro-behavioral impairments. Malocclusion and oral-facial dysfunctions such as having a retrusive bite, narrow maxilla, mandibular hypoplasia, oral breathing, or visceral swallowing are considered risk factors for SRBD and OSAS in pre-schoolers. The present study aims to assess the effectiveness of orthodontic treatment in childhood SRBD and the changes which occur in oral functions and cranio-facial structures. Participants were recruited if they were less than 6 years of age, presented signs and symptoms of SRBD together with malocclusion and/or oral dysfunctions, and had a baseline apnea index < 5 event/h. Children were assigned to a 12-month treatment with a preformed myofunctional appliance (EFline®, Orthoplus), then to an additional 6-month retention period. At baseline, all children underwent physical examination, orthodontic assessment, nocturnal polygraphy, lateral cephalograms and dental casts. A 6-month nocturnal polygraphy was performed only in OSAS patients. Validated tools for assessing risk of SRBD and OSAS (Sleep Clinical Score, SCS), frequency of sleep disturbances (Sleep Disturbances Scale for Children, SDSC) and neuro-behavioral impairment (Child Behavior Checklist, CBCL) were performed at baseline (T0) and after 12 months (T1). Custom cephalometric analyses and dental arch width measurements were digitally performed at baseline and after 18 months (T2). Of the 12 patients initially recruited (4 females, 8 males; mean age 5.0±0.47 years), one child dropped-out. Overall, children tolerated the treatment well and recovered from oral dysfunctions (p<0.001). The OSAS subgroup completely recovered from apnea (AHI<1). Significant improvements were seen in respiratory, sleep and dento-skeletal variables: mean SCS (p<0.001), SDSC and CBCL (p<0.01) scores decreased after 12 months; a mean increase of 2-3 mm occurred in pharyngeal airway widths (p<0.01) and 6 mm in mandibular length (p<0.001), a less hyperdivergent facial growth pattern and favourable advancement of the hyoid bone occurred; mean inter-canine widths increased by 2.3-3 mm in the mandible (p<0.001) and in the maxilla (p<0.05). No significant differences were detected between OSAS and SRBD non-OSAS subgroups with the exception of the SCS and CBCL scores at baseline which were resolved after treatment. Orthodontic treatment with PMAs may produce significant improvements in respiratory and sleep patterns together with significant cranio-facial and dental changes. PMAs might be an effective tool in the multidisciplinary treatment of SRBD and mild-to-moderate OSAS in pre-schooled children.
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3

Li, Chutu. "The effects of CPAP tube reverse flow." Click here to access this resource online, 2008. http://hdl.handle.net/10292/659.

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CPAP is the most common treatment for moderate to severe sleep apnea in adults. Despite its efficacy, patients’ safety, comfort and compliance are issues to be considered and improved in CPAP design. The issues include condensation, carbon dioxide in inhaled air, humidity and temperature of inhaled air. When a CPAP user breaths deeply, there will be some air not fully expelled and may be driven back into the heated air delivery tube (HADT). An interest has existed in what impacts this so called reverse flow may bring about to the CPAP use. The main objectives of this research are to quantify the reverse flow and its influence on carbon dioxide re-breathing, delivered humidity to the patient and condensation in the HADT. Within this thesis, two computer models of the CPAP system have been constructed on Simulink™ in the Matlab™ environment. One is about the CPAP fluid dynamic performance and carbon dioxide re-breathing and the other is on thermodynamic performance. The models can predict the dynamic behaviour of the CPAP machine. They are able to mimic the breath induced airflow fluctuation, and flow direction changes over wide real working ranges of ambient conditions, settings and coefficients. These models can be used for future analysis, development, improvement and design of the machine. The fluid dynamic and thermodynamic models were experimentally validated and they have proved to be valuable tool in the work. The main conclusions drawn from this study are: • Reverse flow increases when breaths load increases and pressure setting decreases. • Reverse flow does not definitely add exhaled air to the next inhalation unless the reverse flow is relatively too much. • Mask capacity does not influence the reverse flow. • The exhaled air re-breathed is mainly due to that stays in the mask, therefore larger mask capacity increases the exhaled air re-breath and the percentage of exhaled air in next inhalation drops when the breath load increases. • Deep breathing does not significantly change the total evaporation in chamber. • When deep breathing induced reverse flow occurs, condensation occurs or worsens in the HADT near the mask. This happens only when the humidity of the airflow from the CPAP is much lower than that of the exhaled air and the tube wall temperature is low enough for condensation to occur. • The deep breathing and reverse flow do not significantly influence the average inhaled air temperature. • The overall specific humidity in inhaled air is lower under deep breathing. • Mask capacity does not influence the thermal conditions in the HADT and the inhaled air specific humidity. Also the mask capacity does not significantly influences the inhaled air temperature.
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Zhang, Xinyan. "Uncovering the sleep pathway in the social profile of Rett syndrome." Electronic Thesis or Diss., Lyon 1, 2022. http://www.theses.fr/2022LYO10128.

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Le sommeil est essentiel pour maintenir une santé optimale. Le sommeil problématique se retrouve avec une plus grande fréquence et sévérité chez les enfants atteints de troubles neurodéveloppementaux et psychiatriques. De plus, le sommeil problématique est associé à un fonctionnement psychosocial plus faible pendant la journée. Le syndrome de Rett (RTT), en tant que l'un des handicaps multiples génétiques les plus courants et les plus graves chez la femme, est fortement lié au gène mutant de la protéine de liaison méthyl-CpG 2 (MECP2) sur le chromosome X. Les formes phénotypiques variantes de l'RTT présentent un spectre de symptomatologie similaire à celui de l'RTT classique, mais présentent des différences subtiles dans certaines caractéristiques cliniques, variante d’épilepsie précoce (variante de Hanefeld, liée au gène mutant X-linked cyclin-dependent kinase-like 5, CDKL5), variante congénitale (variante de Rolando, liée au gène forkhead box G1, FOXG1) et variante de la parole préservée (variante de Zappella, également liée à MECP2). Le syndrome de Rett concerne 1 naissance sur 10 000 à 15 000, ce qui représente 40 à 50 nouveaux enfants malades chaque année en France. Le RTT se caractérise par un arrêt du développement environ 6 à 18 mois après la naissance, la présence de mouvements stéréotypés de la main et des anomalies de la démarche coïncidant avec la perte des compétences acquises de la main intentionnelle et du langage parlé. L’enfant se retire socialement. D'autres signes également décrits dans les profils cliniques du RTT comprennent les crises d'épilepsie, les difficultés respiratoires, le tonus musculaire anormal, la scoliose, ainsi que les troubles du sommeil. En général, les résultats physiopathologiques du RTT suggèrent des activités corticales anormales et une dysmaturité de la fonction du tronc cérébral, ce qui est essentiel pour maintenir un état adéquat pendant le sommeil ou l'éveil. Cependant, il n'existe pas d'étude scientifique sur la relation entre les anomalies du sommeil et les troubles sociaux dans le RTT. Ainsi, ce travail de doctorat s'est orienté vers ce sujet pour lier le jour et la nuit en RTT. Premièrement, nous avons entrepris au total cinq revues systématique de toutes les études précédentes sur les performances sociales non verbales et le sommeil réalisé sur des personnes atteintes de RTT. Deuxièmement, nous avons analysé les enregistrements polysomnographiques dans un échantillon clinique d'individus atteints de RTT présentant les mutations MECP2. Nous avons étudié leur macrostructure du sommeil et leur respiration pendant le sommeil. En outre, nous avons examiné les traits phénotypiques possibles via une approche analytique stratifiée par caractéristiques cliniques et génétiques. Pour examiner les profils sociaux chez les personnes atteintes de RTT, nous avons extrait 25 items liés au comportement social du questionnaire ‘Rett Syndrome Behavior Questionnaire’, qui étaient corrélés à leur sommeil.De manière générale, nous pouvons conclure que le sommeil dans le phénotype social des individus atteints de RTT est lié à des déficiences sensorimotrices progressives. Par conséquent, à l'avenir, la physiopathologie du système sensorimoteur devrait faire l'objet d'une plus grande attention dans l'étude du sommeil et de la vie sociale des personnes atteintes de RTT. En outre, nous attendons avec impatience de nouvelles recherches sur la démonstration des effets des thérapies sensorimotrices sur les troubles du sommeil et les déficiences sociales
Sleep is essential for maintaining optimal health. In children with neurodevelopmental and psychiatric disorders, problematic sleep is found with greater frequency and severity. Furthermore, problematic sleep is associated with poorer psychosocial functioning during the daytime. Rett Syndrome (RTT), one of the most common and severe genetic multi-disabilities in females, is strongly linked to the mutant methyl-CpG binding protein 2 gene (MECP2) on the X chromosome. Variant phenotypic forms of RTT present a spectrum of symptomatology similar to that of classical RTT but show subtle differences in some clinical features, including the Early Seizure Variant (ESV, Hanefeld variant, linked to mutant gene X-linked cyclin-dependent kinase-like 5, CDKL5), congenital variant (CV, Rolando variant, linked to the forkhead box G1 gene, FOXG1) and preserved speech variant (PSV, Zappella variant, also linked to MECP2). RTT affects 1 in 10,000 to 15,000 births, which represents 40 to 50 new cases each year in France. RTT is characterized by developmental arrest around 6-18 months after birth, the presence of stereotypical hand movements, and gait abnormalities coinciding with the loss of acquired purposeful hand skills and spoken language. The child withdraws socially. Other signs also described in RTT clinical profiles include epileptic seizure, breathing difficulties, abnormal muscle tone, scoliosis/kyphosis, as well as disturbed sleep. Accumulating pathophysiological findings in RTT suggest abnormal cortical activities and dysmaturity of the brainstem function, which is key in maintaining proper status during sleep or wakefulness. However, there is no scientific study investigating the relationship between sleep abnormalities and social impairments in RTT. Therefore, this doctoral work is subjected to this topic to link the day and night together in RTT. First, we undertook five systematic reviews of all previous studies on non-verbal social performance and sleep in RTT. Then, we analyzed polysomnographic recordings in a clinical sample of RTT individuals with MECP2 mutations. We studied their sleep macrostructure and respiration during sleep. In addition, we examined possible phenotypic traits via a stratified analytical approach to clinical and genetic characteristics. Lastly, to examine social profiles in RTT individuals, we extracted 25 social behavior items from the Rett Syndrome Behavior Questionnaire, and correlated them to their sleep. Overall, we can conclude that sleep in the social phenotype of individuals with RTT is related to progressive sensorimotor impairments. Therefore, in the future, the pathophysiology of the sensorimotor system should receive more attention in the study of sleep and the social life of individuals with RTT. In addition, we look forward to furthering research demonstrating the effects of sensorimotor therapies on sleep and social impairments
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5

Ng, Kwok-keung Daniel. "Sleep related breathing disorders in children /." View the Table of Contents & Abstract, 2006. http://sunzi.lib.hku.hk/hkuto/record/B36223724.

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Ng, Kwok-keung Daniel, and 吳國強. "Sleep related breathing disorders in children." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B45007688.

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Davies, Robert J. O. "Sleep disordered breathing and the cardiovascular system." Thesis, University of Southampton, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.404009.

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8

Smith, Lindsay Anne. "Sleep-disordered breathing and chronic heart failure." Thesis, University of Edinburgh, 2009. http://hdl.handle.net/1842/29371.

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Methods: Patients with stable symptomatic chronic heart failure were screened for sleep-disordered breathing by home sleep study. Daytime sleepiness was assessed by Epworth Sleepiness Scale and heart failure severity by symptom class, left ventricular ejection fraction and serum N-terminal pro-brain natriuretic peptide concentrations. In a subset of patients, synchronous in-laboratory limited sleep studies and polysomonography, and home limited sleep studies, were performed prospectively. Patients with obstructive sleep apnoea and stable symptomatic chronic heart failure were randomised to nocturnal auto-titrating continuous positive airway pressure or sham for six weeks each in crossover design. Results: In the era of modern therapy, sleep-disordered breathing is common in patients with stable symptomatic chronic heart failure, predominantly obstructive in aetiology, without clear relationship to heart failure severity and is difficult to diagnose because of major overlap in symptomatology. Limited sleep studies compare well diagnostically to polysomnography when tested under identical patient and environmental conditions but less so when tested in the home setting. Auto-titrating continuous positive airway pressure improves daytime sleepiness is patients with obstructive sleep apnoea and chronic heart failure but not other subjective or objective measures of heart failure severity. Conclusions: Sleep-disordered breathing is difficult to detect clinically in patients with chronic heart failure, and as such, the diagnosis is reliant on accurate sleep studies. However, the clinical utility of limited sleep studies in detection and diagnosis of sleep-disordered breathing is restricted by a number of technical and situational factors which are exacerbated in patients with chronic heart failure. The potential therapeutic benefits of continuous positive airway pressure in patients with obstructive sleep apnoea and chronic heart failure are achieved by alleviation of obstructive sleep apnoea rather than by improvement in cardiac function.
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Pirilä-Parkkinen, K. (Kirsi). "Childhood sleep-disordered breathing – dentofacial and pharyngeal characteristics." Doctoral thesis, Oulun yliopisto, 2011. http://urn.fi/urn:isbn:9789514296024.

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Abstract The aim of this work was to examine distinct dentofacial and pharyngeal features in children with sleep-disordered breathing (SDB). A further aim was to test the validity of the conventional methods used in orthodontics for recognition of obstructed airways. Seventy children (36 girls, 34 boys, age range 4.2–11.9 years), who were diagnosed as having SDB during the years 2000–2002, constituted the source of subjects for four separate studies. The study protocol included otorhinolaryngological and orthodontic examinations. Dental impressions and lateral cephalograms were taken. In addition, upper airway was imaged in 36 children in different head postures by magnetic resonance imaging (MRI). Age- and gender-matched healthy children were used as controls. The results showed that children with SDB had increased overjet, smaller overbite, narrower upper and shorter lower dental arches and higher incidence of anterior open bite and distal molar relationship when compared with control children. The severity of the obstruction correlated with larger deviations from normal in the dental characteristics. There were more children with crowding and with anterior open bite with increased severity of the disorder. Children with SDB displayed an increased sagittal jaw relationship, a more vertical growth pattern of the mandible, a longer and thicker soft palate, a lower hyoid bone position, larger craniocervical angles and narrower pharyngeal airway measured at multiple levels, when compared with the controls. Deviation in pharyngeal variables showed highest correlation with the severity of SDB. MRI indicated a smaller than normal oropharyngeal airway in children with SDB. The effect of head posture on pharyngeal airway dimensions differed in children with SDB when compared with the controls. These findings verify that the developmental course of occlusal characteristics and craniofacial structures may be modulated by breathing pattern, and, on the other hand, that certain features may predispose to the development of the disorder. It is important to recognize these signs in order to guide further development in a more favorable direction. The results confirmed that cephalometry is a valid screening tool when assessing nasopharyngeal and retropalatal airway dimensions. Clinical examination of tonsillar size was found reliable when retroglossal airway size was evaluated
Tiivistelmä Väitöskirjatyön tarkoituksena oli selvittää lasten unenaikaisten hengityshäiriöiden yhteyttä kasvojen ja leukojen kasvuun, purennan kehitykseen sekä ylempien hengitysteiden rakenteeseen. Lisäksi tutkittiin, kuinka luotettavasti hammaslääkäreiden käytössä olevin menetelmin pystytään arvioimaan lasten ahtautuneita ilmateitä. Tutkimusaineistossa oli mukana 70 lasta (36 tyttöä, 30 poikaa, ikä 4.2–11.9 vuotta), joilla diagnosoitiin unenaikainen obstruktiivinen hengityshäiriö vuosina 2000–2002. Lapsille tehtiin kliiniset tutkimukset oikojahammaslääkärin ja korva-, nenä- ja kurkkutautien erikoislääkärin toimesta sekä otettiin lateraalikallokuva ja hampaistosta kipsimallijäljennökset. Lisäksi ylempien hengitysteiden magneettitutkimus suoritettiin 36 lapselle eri pään asennoissa. Verrokkiryhmässä oli sama ikä- ja sukupuolijakauma kuin tutkimusryhmässä. Tulokset osoittivat, että unenaikaisista hengityshäiriöistä kärsivillä lapsilla horisontaalinen ylipurenta oli suurentunut, vertikaalinen ylipurenta oli pienentynyt, ylähammaskaari oli kapeampi ja alahammaskaari lyhyempi kuin terveillä verrokeilla. Alaetualueen ahtaus ja etualueen avopurenta olivat yleisempiä niillä lapsilla, joilla oli vaikeampiasteinen hengityshäiriö. Lasten unenaikaiset hengityshäiriöt liittyivät suurentuneeseen leukojen väliseen kokoepäsuhtaan, alaleuan avautuvaan kasvumalliin, pidempään ja paksumpaan pehmeään suulakeen, kieliluun alempaan asentoon, kohonneeseen pään asentoon sekä ahtaampiin nielun ilmatilan mittoihin verrattaessa terveisiin lapsiin. Häiriön vaikeusaste korreloi suurempiin poikkeamiin nielun mitoissa. Lapsilla, joilla todettiin unenaikainen hengityshäiriö, magneettitutkimukset osoittivat nielun olevan ahdas. Pään asennon vaikutukset ilmatien rakenteeseen poikkesivat unenaikaisista hengityshäiriöistä kärsivillä lapsilla. Saadut tulokset osoittivat, että hengitystapa voi vaikuttaa hampaiston, kasvojen ja leukojen rakenteiden kehitykseen. Tietyt kasvojen ja leukojen piirteet puolestaan saattavat altistaa häiriön kehittymiselle. On tärkeää tunnistaa nämä merkit, jotta kehitystä voidaan ohjata suotuisampaan suuntaan. Tutkimukset osoittivat, että kefalometrinen analyysi on luotettava arvioitaessa nenänielun sekä pehmeän suulaen takana olevan ilmatilan kokoa. Nielurisojen kliinisen koon arviointi on luotettava arvioitaessa alemman suunielun ilmatilan kokoa
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10

Aran, Reza. "Craniofacial morphology and sleep disordered breathing in children." Thesis, University of British Columbia, 2013. http://hdl.handle.net/2429/44379.

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Objective: The aim of this study is to understand how craniofacial morphology and the severity of a malocclusion can contribute to sleep disordered breathing (SDB) symptoms in children when controlled for age, gender and body mass index (BMI). Methods: A total of 301 subjects with complete records were included in this study. Two hundred and thirty-seven were preadolescents, of which 97 were male and 140 were female (mean age 9.9±1.6); 64 were adolescents, of which 24 were male and 40 were female (mean age 13.8±0.9). All the subjects’ parents were asked to complete a SDB questionnaire. Lateral cephalometric images were analyzed to assess the position of the hyoid bone, length of the soft palate, and the maxillary and mandibular dental and skeletal relationship. A clinical examination was performed to determine the Angle classification, Mallampati score, tonsil size (Brodesky), and BMI. Results: Data from 301 children that completed the questionnaires and underwent a cephalometric analysis were evaluated. Subjects were divided into two groups based on their age and each group was further divided based on gender. By comparing preadolescents with adolescents we found that preadolescents presented a significantly higher incidence of hyperactivity, morning headaches, more frequent snoring, and bedwetting. Adolescents exhibited significantly higher daytime sleepiness, difficulty getting up, and impaired daytime function. When comparing female and male subjects, we found that frequent snoring, and morning headaches were more prevalent among females, while daytime sleepiness, and hyperactivity were more common among males. Craniofacial features that have a significant relationship with SDB symptoms are, a lower position of the hyoid bone, retruded mandible, steeper mandibular plane angle, and retroclined lower incisors. There was no statistically significant relationship between Angle classifications, tonsil size, Mallampati score, and BMI with SDB symptoms in this sample. Conclusion: This study suggests that craniofacial morphology, but not severity of malocclusions, could be a potential contributing factor to SDB symptom severity.
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Книги з теми "Sleep breathing disorder"

1

H, Edelman Norman, and Santiago Teodoro V, eds. Breathing disorders of sleep. New York: Churchill Livingstone, 1986.

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2

editor, Nangia Vivek, and Shivani Swami. Sleep related breathing disorders. New Delh: Jaypee, the health sciences publisher, 2015.

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3

1942-, Zwick Hartmut, ed. Sleep related breathing disorders. Wien ; New York: Springer-Verlag, 1992.

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Won, Christine, ed. Complex Sleep Breathing Disorders. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-57942-5.

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5

Zwick, Hartmut, ed. Sleep Related Breathing Disorders. Vienna: Springer Vienna, 1992. http://dx.doi.org/10.1007/978-3-7091-6675-8.

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6

Breathing disorders in sleep. Philadelphia: W.B. Saunders, 1992.

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7

Thomas, Verse, ed. Surgery for sleep disordered breathing. 2nd ed. Berlin: Springer, 2010.

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8

Kheirandish-Gozal, Leila, and David Gozal, eds. Sleep Disordered Breathing in Children. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60761-725-9.

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9

Hörmann, Karl, and Thomas Verse. Surgery for Sleep Disordered Breathing. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-77786-1.

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Hörmann, Karl. Surgery for sleep-disordered breathing. New York: Springer, 2005.

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Частини книг з теми "Sleep breathing disorder"

1

Krakow, Barry, Bret Moore, and Victor A. Ulibarri. "Sleep-Disordered Breathing and Posttraumatic Stress Disorder." In Sleep and Combat-Related Post Traumatic Stress Disorder, 243–52. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7148-0_21.

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2

Mandolini, Marco, Manila Caragiuli, Daniele Landi, Antonio Gracco, Giovanni Bruno, Alberto De Stefani, and Alida Mazzoli. "Evaluation of the Effects Caused by Mandibular Advancement Devices Using a Numerical Simulation Model." In Lecture Notes in Mechanical Engineering, 101–7. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-70566-4_17.

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AbstractObstructive sleep apnea syndrome (OSAS) is a sleep disorder that causes pauses in breathing or periods of shallow breathing during sleep. Mandibular advancement devices (MADs) represent a non-invasive treatment for OSAS that has had the highest development in recent years. Nevertheless, literature has not primarily investigated the effects of mandibular advancement. This paper presents a finite element method numerical simulation model for evaluating the stress/strain distribution on the temporomandibular joint (TMJ) and periodontal ligaments caused by advancement devices used for the treatment of OSAS. Results highlight that the mandible lift phase generates significant stress values on TMJ, which cannot be neglected for extended usage of MADs. Furthermore, mandible molar teeth are more loaded than incisor ones.
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McCullough, Lindsay, and Alejandra C. Lastra. "Breathing Pauses." In Sleep Disorders, 95–122. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-65302-6_5.

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4

Franco-Avecilla, Dennys, Mouaz Saymeh, and Tetyana Kendzerska. "Changes in Positive Airway Pressure Application and Practices in Individuals with Sleep-Related Breathing Disorder During the COVID-19 Pandemic." In COVID-19 and Sleep: A Global Outlook, 369–83. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-0240-8_23.

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Davies, Eryl. "Sleep Breathing Disorders." In The Final FFICM Structured Oral Examination Study Guide, 414–15. Boca Raton: CRC Press, 2022. http://dx.doi.org/10.1201/9781003243694-141.

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6

Mendelson, Wallace B. "Sleep-Related Breathing Disorders." In Human Sleep, 183–220. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1921-4_6.

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7

Kreitinger, Kimberly, Matthew Light, Sagar Patel, and Atul Malhotra. "Sleep-Disordered Breathing." In Sleep Medicine and Mental Health, 131–50. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-44447-1_7.

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8

Suri, Jagdish Chander, and Tejas M. Suri. "Sleep-Disordered Breathing." In ICU Protocols, 139–45. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-0898-1_15.

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9

Rohde, Kerstin, and Thomas Verse. "Sleep Disordered Breathing." In Surgery for Sleep Disordered Breathing, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-77786-1_1.

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Suri, Jagdish Chander. "Sleep-Disordered Breathing." In ICU Protocols, 101–6. India: Springer India, 2012. http://dx.doi.org/10.1007/978-81-322-0535-7_13.

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Тези доповідей конференцій з теми "Sleep breathing disorder"

1

T. V., Rajagopal, Vimi Varghese, and Prince James. "Acute heart failure - Central breathing disorder - Hypercapnia: The uncommon trio." In ERS Sleep and Breathing 2023 abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2023.121.

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2

Ahn, So-Hyun. "Preliminary analysis of the impact of REM sleep behavior disorder on the severity of OSA." In ERS Sleep and Breathing 2023 abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2023.95.

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3

Georgiev, Todor, Krasimir Avramov, Aneliya Draganova, Valentin Dichev, Nikolay Mehterov, and Kiril Terziyski. "Downregulation of miRNA-125b and let-7 provides a novel aspect to chronic insomnia disorder – a pilot study." In ERS Sleep and Breathing 2023 abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2023.125.

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4

Ghosh, Rahul, and Susmita Kundu. "Sleep related breathing disorder patterns in DPLD patients." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa934.

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5

Shivaram, Suganti, Anjani Muthyala, Zahara Z. Meghji, Susan Karki, and Shivaram Poigai Arunachalam. "Multiscale Entropy Technique Discriminates Single Lead ECG’s With Normal Sinus Rhythm and Sleep Apnea." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6948.

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Sleep apnea is characterized by abnormal interruptions in breathing during sleep due to partial or complete airway obstructions affecting middle-aged men and women on an estimated ∼4% of the population [1]. While the disorder is clinically manageable to relieve patients, the challenge occurs with diagnosis, with many patients going undiagnosed leading to further complications such as ischemic heart diseases, stroke etc. Sleep apnea also significantly affects the quality of day to day life causing sleepiness and fatigue. Polysomnography (PSG) technique is currently a used for detecting sleep apnea which is a comprehensive sleep test to diagnose sleep disorders by recording brain waves, the oxygen level in the blood, heart rate, breathing, eye and leg movements during the study. However, PSG test is very expensive, requires patients to stay overnight and is known to cause inconvenience to the patients.
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6

Hamnvik, Sondre, Pierre Bernabé, and Sagar Sen. "Yolo4Apnea: Real-time Detection of Obstructive Sleep Apnea." In Twenty-Ninth International Joint Conference on Artificial Intelligence and Seventeenth Pacific Rim International Conference on Artificial Intelligence {IJCAI-PRICAI-20}. California: International Joint Conferences on Artificial Intelligence Organization, 2020. http://dx.doi.org/10.24963/ijcai.2020/754.

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Obstructive sleep apnea is a serious sleep disorder that affects an estimated one billion adults worldwide. It causes breathing to repeatedly stop and start during sleep which over years increases the risk of hypertension, heart disease, stroke, Alzheimer's, and cancer. In this demo, we present Yolo4Apnea a deep learning system extending You Only Look Once (Yolo) system to detect sleep apnea events from abdominal breathing patterns in real-time enabling immediate awareness and action. Abdominal breathing is measured using a respiratory inductance plethysmography sensor worn around the stomach. The source code is available at https://github.com/simula-vias/Yolo4Apnea
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7

Dongol, Eptehal, Panagis Drakatos, Rexford Tapiwa Muza, Mohamed Shahat Badawy, Brian Kent, Ahmed Younis, and Adrian Williams. "Periodic limb movement disorder: a Trojan horse for residual sleepiness in patients with OSAS while on CPAP?" In ERS/ESRS Sleep and Breathing Conference 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/23120541.sleepandbreathing-2017.p29.

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8

Zavrel, Erik A., and Matthew R. Ebben. "A Novel Genioglossal Strengthening Device for Obstructive Sleep Apnea Treatment." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6809.

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Obstructive sleep apnea (OSA) is a condition characterized by temporary diminutions or cessations of breathing caused by repetitive collapse of the upper airway (UA) during sleep [1]. OSA is a common disorder — the American Academy of Sleep Medicine estimates the condition affects 29.4 million Americans — in which the muscles that hold the airway open weaken, resulting in partial or complete UA collapse during inspiration [2]. These pauses in breathing lead to blood oxygen desaturation and induce neurological arousal resulting in sleep disruption and fragmentation. The cycle of airway collapse and arousal can repeat hundreds of times per night.
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9

Tork, Sepideh, Mohammad Reza Yazdchi, and Arezoo Karimizadeh. "How health related to quality of sleep and breathing: How to associate sleep disorder breathing and extracted respiratory signal." In 2013 21st Iranian Conference on Electrical Engineering (ICEE). IEEE, 2013. http://dx.doi.org/10.1109/iraniancee.2013.6599645.

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10

Krichen, H., R. Kammoun, S. Abid, A. Hentati, A. Kotti, I. El Wadhane, S. Msaad, A. Bahloul, W. Feki, and S. Kammoun. "Predictors of chronic hypercapnia in obese patients with suspected breathing sleep disorder." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.1333.

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Звіти організацій з теми "Sleep breathing disorder"

1

Sitrin, Robert G. Home-Based Diagnosis and Management of Sleep-Related Breathing Disorders in Spinal Cord Injury. Fort Belvoir, VA: Defense Technical Information Center, October 2012. http://dx.doi.org/10.21236/ada569677.

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