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Статті в журналах з теми "Rice Diet"
Wiwanitkit, Viroj. "Rice and Spicy Diet." Journal of Neurogastroenterology and Motility 16, no. 3 (July 31, 2010): 340. http://dx.doi.org/10.5056/jnm.2010.16.3.340.
Повний текст джерелаMarsono, Yustinus, Richard J. Illman, Julie M. Clarke, Rodney P. Trimble, and David L. Topping. "Plasma lipids and large bowel volatile fatty acids in pigs fed on white rice, brown rice and rice bran." British Journal of Nutrition 70, no. 2 (September 1993): 503–13. http://dx.doi.org/10.1079/bjn19930144.
Повний текст джерелаLEE, Jae-Sung, Nese SREENIVASULU, Ruaraidh Sackville HAMILTON, and Ajay KOHLI. "Brown Rice, a Diet Rich in Health Promoting Properties." Journal of Nutritional Science and Vitaminology 65, Supplement (October 11, 2019): S26—S28. http://dx.doi.org/10.3177/jnsv.65.s26.
Повний текст джерелаWeissbort, Daniel. "Venice and the Rice Diet." Iowa Review 31, no. 3 (December 2001): 69–70. http://dx.doi.org/10.17077/0021-065x.5444.
Повний текст джерелаKusnandar, Feri, Trina Kharisma, Nancy Dewi Yuliana, Safrida Safrida, and Slamet Budijanto. "Hypocholesterolemic Effect of Analogue Rice with the Addition of Rice Bran." Current Research in Nutrition and Food Science Journal 10, no. 1 (April 29, 2022): 183–94. http://dx.doi.org/10.12944/crnfsj.10.1.13.
Повний текст джерелаFelipoff, Ana Lia, Angela Zuleta, Maria Elena Sambucetti, and Maria Esther Rio. "Not any type of rice performs equally to improve lactose-induced diarrhea characteristics in rats: is amylose an antidiarrheal factor?" Food Science and Technology 32, no. 2 (May 29, 2012): 323–28. http://dx.doi.org/10.1590/s0101-20612012005000057.
Повний текст джерелаHamzat, R. A., A. J. Omole, B. B. Babatunde, M. O. Adejumo, and O. G. Longe. "Comparative value of kola testa, corn-bran and rice- bran with maize for mature african giant land snail (Archachatina marginata)." Nigerian Journal of Animal Production 34, no. 2 (January 9, 2021): 265–76. http://dx.doi.org/10.51791/njap.v34i2.1203.
Повний текст джерелаHuang, Pei-Xin, Chiu-Li Yeh, Suh-Ching Yang, Hitoshi Shirakawa, Chao-Lin Chang, Li-Hsin Chen, Yen-Shuo Chiu, and Wan-Chun Chiu. "Rice Bran Supplementation Ameliorates Gut Dysbiosis and Muscle Atrophy in Ovariectomized Mice Fed with a High-Fat Diet." Nutrients 15, no. 16 (August 9, 2023): 3514. http://dx.doi.org/10.3390/nu15163514.
Повний текст джерелаGonlachanvit, Sutep. "Rice and Spicy Diet: Author's Reply." Journal of Neurogastroenterology and Motility 16, no. 3 (July 31, 2010): 341. http://dx.doi.org/10.5056/jnm.2010.16.3.341.
Повний текст джерелаZhang, Hong, Pei Cao, Luis B. Agellon, and Cheng-Kai Zhai. "Wild rice (Zizania latifolia(Griseb) Turcz) improves the serum lipid profile and antioxidant status of rats fed with a high fat/cholesterol diet." British Journal of Nutrition 102, no. 12 (July 27, 2009): 1723–27. http://dx.doi.org/10.1017/s0007114509991036.
Повний текст джерелаДисертації з теми "Rice Diet"
O, Jung Mi, University of Western Sydney, of Science Technology and Environment College, and of Science Food and Horticulture School. "Food habits and eating patterns of Korean adult immigrants in Australia." THESIS_CSTE_SFH_O_J.xml, 2003. http://handle.uws.edu.au:8081/1959.7/462.
Повний текст джерелаMaster of Science (Hons)
Oliveira, Liliane Soares Corrêa de. "Dieta rica em sacarose: perfil inflamatório e danos hepáticos em camundongos." Universidade do Estado do Rio de Janeiro, 2013. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=6922.
Повний текст джерелаAinda não está bem definido na literatura se uma dieta rica em sacarose, mesmo sendo isoenergética, provoca danos à saúde. Camundongos C57BL/6 foram alimentados com uma dieta controle (10% da energia proveniente de gordura, 8% da energia proveniente da sacarose - SC), uma dieta rica em sacarose (10% de energia proveniente da gordura, 32% da energia proveniente da sacarose - HSu), uma dieta hiperlipídica (42% da energia proveniente de gordura, 8% da energia proveniente da sacarose - HF) ou uma dieta combinada HF/HSu (42% da energia proveniente de gordura, 32% da energia proveniente da sacarose), durante oito semanas. Apesar da massa corporal e do índice de adiposidade não terem sofrido alteração, o grupo HSu apresentou hipertrofia dos adipócitos, o que também foi observado nos grupos HF e HF/HSu. Os grupos HF, HSu e HF/HSu foram intolerantes à glicose e apresentaram níveis séricos de insulina elevados. Os níveis séricos de leptina, resistina e proteína quimiotática de monócitos-1 (MCP-1) aumentaram, enquanto adiponectina sérica reduziu nos grupos HF, HSu e HF/HSu. No tecido adiposo, os animais HF, HSu e HF/HSu apresentaram maiores níveis de expressão protéica de leptina e níveis mais baixos de expressão protéica de adiponectina, em comparação ao grupo SC. Colesterol hepático foi maior nos grupos HF e HF/HSu, enquanto TG hepático foi maior nos grupos HSu e HF/HSu. Os animais dos grupos HF, HSu e HF/HSu apresentaram esteatose hepática, aumento da expressão protéica hepática de elemento regulador de esterol ligante da proteína 1 (SREBP-1c) e diminuição da expressão protéica do receptor ativador de proliferação peroxissomal alfa (PPAR-α). Em conclusão, a dieta rica em sacarose não provoca obesidade nos animais, mas provoca alterações nos adipócitos (hipertrofia), intolerância à glicose, hiperinsulinemia, hiperlipidemia, esteatose hepática e aumento de citocinas inflamatórias. Os efeitos prejudiciais da dieta rica em sacarose, mesmo quando a sacarose substitui isocaloricamente o amido na alimentação, pode ter consequências para a saúde.
It is still unclear if an isoenergetic, sucrose-rich diet leads to health conse-quences. Mice were fed a control diet (10% energy from fat, 8% energy from sucrose - SC), a high-sucrose diet (10% energy from fat, 32% energy from sucrose - HSu), a high-fat diet (42% energy from fat, 8% energy from sucrose - HF) or a HF/HSu diet (42% energy from fat, 32% energy from sucrose) for eight weeks. Despite the un-changed body mass and adiposity indices, HSu presented adipocyte hypertrophy, which was also observed in the HF and HF/HSu. The HF, HSu and HF/HSu were glucose intolerant and had elevated serum insulin levels. The levels of leptin, resistin and Monocyte Chemotactic Protein-1 (MCP-1) increased, while the serum adiponectin decreased in the HF, HSu and HF/HSu. In the adipose tissue, the HF, HSu and HF/HSu showed higher levels of leptin protein expression and lower levels of adiponectin protein expression in comparison with the SC. Hepatic cholesterol was higher in the HF and HF/HSu, while hepatic TG was higher in the HSu and HF/HSu. Liver steatosis was higher, sterol regulatory element-binding protein-1c (SREBP-1c) hepatic expression was increased, and peroxisome proliferator-activated receptor-alpha (PPAR-α) hepatic protein expression was decreased in the HF, HSu and HF/HSu in comparison with the SC. In conclusion, a sucrose-rich diet does not lead to a state of obesity but has the potential to cause changes in the adipocytes (hypertrophy) as well as glucose intolerance, hyperinsulinemia, hyperlipidemia, hepatic steatosis, and increases in the number of inflammatory cytokines. The deleterious effects of a sucrose-rich diet in an animal model, even when the sucrose replaces starch isocalorically in the feed, can have far-reaching consequences for health.
Gomes, Arthur Rocha. "Efedrina altera a ansiedade e locomo??o de ratos Wistar tratados desde a lacta??o com dietas de cafeteria ou restri??o cal?rica." UFVJM, 2016. http://acervo.ufvjm.edu.br/jspui/handle/1/1369.
Повний текст джерелаOrientador do trabalho n?o mencionado na lista da Folha de Aprova??o.
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O objetivo desse trabalho foi avaliar os efeitos da efedrina no comportamento de ratos tratados com ra??o padr?o (comercial), dieta de cafeteria ou dieta de restri??o cal?rica desde a lacta??o. Foramutilizadas 12 ninhadas de ratos da linhagem Wistar (Rattus novergicus). Os animais foram alojados em gaiolas individuais, sob condi??es padr?es (umidade natural; temperatura de 23 ?C ? 2; e ciclo claro/escuro de 12 horas). Os ratos machos de cada ninhada formaram, da lacta??o at? a fase adulta, osgrupos: Controle (C) ?receberamra??o padr?o e?guaadlibitum(n = 24);Cafeteria (CAF)?receberam dieta de cafeteria e ?gua ad libitum (n = 24); Restri??o (R) ?receberam 50%dara??oconsumidapelo grupoControle e ?guaadlibitum (n = 24). Entre o 113? e o 117? dia de vida, os animais foram subdividos (n = 12) para receberemo tratamento com salina (C, CAF e R) ou efedrina (C-E, CAF-E e R-E) e realizarem os testes comportamentais. No 118? foram anestesiados e eutanasiados por exsanguina??o. Foram avaliados: o peso corporal, ganho de peso, consumo de ra??o, ingest?o cal?rica, coeficiente de efici?ncia alimentar, comprimento naso-anal e ?ndice de massa corporal; peso dos ?rg?os e tecido adiposo abdominal; comprimento do f?mur e t?bia, e teor de minerais totais; teores de colesterol total e fra??es, triacilglicerol e glicemia do soro; teor de lip?dios, colesterol total e triacilglicerol do f?gado; e os efeitos no comportamento pelos testes Labirinto em Cruz Elevado (LCE) eCampo Aberto. O grupo CAF demonstrou maior ingest?o cal?rica, configurando em maior CEA e ac?mulo de tecido adiposo abdominal, al?m de uma tend?ncia em aumento do peso e do IMC. Ademais, CAF obteve eleva??o dos n?veis de triacilglicerol plasm?tico e hep?tico, que possivelmente foram fatores respons?veis pelo aumento do tecido adiposo. Ao mesmo tempo, houve uma rela??o ruim entre as fra??es do colesterol plasm?tico (HDL-c, LDL-c e VLDL-c), com desenvolvimento de dislipidemia. Portanto, a dieta de cafeteria foi capaz de reproduzir um modelo de obesidade humana e de s?ndrome metab?lica em CAF. Os animais de dieta de restri??o obtiveram menor peso corporal e dos ?rg?os, apresentando ainda retardo no crescimento (menor CNA, da t?bia e do f?mur), e menores valores de IMC. O quadro nutricional desse grupo de animais indica que foi poss?vel desenvolver um modelo de desnutri??o. O grupo CAF obteve maior n?mero de entradas nos bra?os fechados do LCE e uma tend?ncia em atravessar maior n?mero de quadrantes no in?cio do teste de Campo Aberto, sugerindoaumento da locomo??o. A efedrina proporcionou: maior n?mero de entradas nos bra?os fechados; de entradas e tempo de perman?ncia nos bra?os abertos do LCE; maior n?mero de entradas, tempo de perman?ncia no centro do e n?mero de quadrantes atravessados no campo aberto; para ambos os tratamentos (CAF-E e R-E), sugerindo um efeito ansiol?tico e de aumento da locomo??o. As altera??es no comportamento frente ? efedrina indicam que os animais CAF e R possuem uma altera??o no sistema dopamin?rgico, que culmina com aumento do efeito ansiol?tico e de locomo??o da droga.
Disserta??o (Mestrado) ? Programa de P?s-gradua??o em Ci?ncias Farmac?uticas, Universidade Federal dos Vales do Jequitinhonha e Mucuri, [2016].
The objective of this work was to evaluate the effects of ephedrine on the behavior of rats treated with standard (commercial) chow, cafeteria diet or caloric restriction diet since lactation. Twelve litters of Wistar rats (Rattus novergicus) were used. The animals were housed in individual cages under standard conditions (natural humidity, temperature of 23 ? C ? 2 and light / dark cycle of 12 hours). The male rats of each litter formed, from lactation to adulthood, the following groups: Control (C) - received standard chow and water ad libitum (n = 24); Cafeteria (CAF) - received cafeteria diet and water ad libitum (n = 24); Restriction (R) - received 50% of the chow consumed by the Control group and water ad libitum (n = 24). Between the 113th and 117th days of life, the animals were subdivided (n = 12) to receive treatment with saline (C, CAF and R) or ephedrine (C-E, CAF-E and R-E) and perform behavioral tests. At 118th, they were anesthetized and euthanized by exsanguination. Body weight, weight gain, feed intake, caloric intake, food efficiency coefficient, naso-anal length and body mass index; weight of organs and abdominal adipose tissue; Length of femur and tibia, and total mineral content; levels of total cholesterol and fractions, triacylglycerol and serum glycemia; lipid content, total cholesterol and triacylglycerol of the liver; and effects on behavior through the Elevated Plus Maze (EPM) and Open Field tests were evaluated. The CAF group demonstrated a higher caloric intake, resulting in an increased CEA and abdominal adipose tissue accumulation, in addition to an increasing tendency of weight and BMI. In addition, CAF obtained an elevation in plasma and hepatic triacylglycerol levels, which were possibly responsible for the increase in adipose tissue. At the same time, there was a great relationship between fractions of plasma cholesterol (HDL-c, LDL-c and VLDL-c), with development of dyslipidemia. Therefore, the cafeteria diet was able to reproduce a model of human obesity and metabolic syndrome in CAF. Restriction diet animals had lower body and organ weight, with slower growth (lower CNA, tibia and femur), and lower BMI values. The nutritional picture of this group of animals indicates that it was possible to develop a model of malnutrition. The CAF group had a higher number of entries in the closed arms of the EPM and a tendency to cross more quadrants at the beginning of the Open Field test, suggesting an increase in locomotion. Ephedrine provided: high number of entries in the closed arms; entries and length of stay in the open arms of the EPM; high number of entries, length of stay in the center and number of quadrants crossed in the open field; for both treatments (CAF-E and R-E), suggesting an anxiolytic effect and increased locomotion. Changes in the behavior against ephedrine indicate that the CAF and R animals have a change in the dopaminergic system, which culminates with aniological increase and locomotion of the drug.
Johnson, Raegan C. "The rise and fall of fad diets how the news media frame and represent the Atkins diet, 1972-2005 /." Diss., Columbia, Mo. : University of Missouri-Columbia, 2006. http://hdl.handle.net/10355/4600.
Повний текст джерелаThe entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file viewed on (February 8, 2007) Includes bibliographical references.
Costa, Mariane Róvero. "Efeitos da suplementação do licopeno sobre processos fisiopatológicos da doença gordurosa não alcoólica do fígado induzida por dieta hipercalórica rica em carboidratos simples." Botucatu, 2019. http://hdl.handle.net/11449/183191.
Повний текст джерелаResumo: Introdução: Nos últimos anos a população mundial tem vivenciado mudanças nos hábitos alimentares, tais alterações caracterizam a transição nutricional. Ressalta-se o maior consumo de alimentos de alta densidade energética, produtos industrializados, acrescidos de açúcar e ricos em gordura e uma redução no consumo de vegetais e frutas. Este padrão alimentar é conhecido como dieta ocidental, que associada a inatividade física, reflete em alterações na composição corporal e, consequentemente, obesidade. O sobrepeso e a obesidade atualmente são um preocupante problema de saúde pública devido à elevada prevalência bem como associação com outras patologias como a doença gordurosa não alcoólica do fígado (NAFLD, do inglês nonalcoholic fatty liver disease). A fisiopatologia da NAFLD está amplamente relacionada aos processos oxidativo e inflamatório. Por isso, o licopeno, um carotenoide reconhecido pela sua ação antioxidante e anti-inflamatória, tem sido investigado como um potencial protetor hepático na NAFLD. Objetivo: Investigar o efeito da suplementação com licopeno sobre os processos fisiopatológicos da NAFLD induzida por dieta hipercalórica rica em carboidrato simples. Materiais e Métodos: Esse estudo foi aprovado pela Comissão Ética no Uso de Animais da Faculdade de Medicina de Botucatu (1266/2018). Foram utilizados 24 ratos Wistar aleatoriamente distribuídos em 2 grupos com diferentes dietas para indução de obesidade: dieta controle (n=12) e hipercalórica (n=12). Após 20 seman... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: Introduction: In recent years the world population has experienced changes in eating habits, which characterize the nutritional transition. Higher consumption of high energy density foods, industrialized products, high sugar and high fat products, and a reduction in the consumption of vegetables and fruits are also noteworthy. This dietary pattern is known as western diet, which associated with physical inactivity, reflects on changes in body composition and consequently obesity. Overweight and obesity are currently a worrying public health problem due to the high prevalence as well as association with other pathologies such as non-alcoholic fatty liver disease (NAFLD). The pathophysiology of NAFLD is largely related to oxidative and inflammatory processes. Therefore, lycopene, a carotenoid recognized for its antioxidant and anti-inflammatory action, has been investigated as a potential liver protector in NAFLD. Aim: To investigate the effect of lycopene supplementation on the pathophysiological processes of NAFLD induced by a simple carbohydrate-rich hypercaloric diet. Materials and Methods: This study was approved by the Animal Ethical Committee of Botucatu Medical School (1266/2018). 24 Wistar rats were randomly distributed in 2 groups with different diets for obesity induction: control diet (n=12) and hypercaloric (n=12). After 20 weeks, the animals were redistributed in 4 groups for the study of lycopene supplementation: control group (Co, n=6), control group supplemente... (Complete abstract click electronic access below)
Mestre
Aboosadi, Masoud Arab. "Enzyme supplementation of rice bran based broiler diets." Thesis, University of Aberdeen, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265041.
Повний текст джерелаLima, Flávia Fernandes de. "Dieta hiperlipídica e/ou rica em sacarose em camundongos suíços: metabolismo de carboidratos, fígado e tecido adiposo." Universidade do Estado do Rio de Janeiro, 2013. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=7379.
Повний текст джерелаA doença hepática gordurosa não alcoólica é uma desordem multifatorial causada principalmente por excesso nutricional e resistência à insulina, com prevalência estimada de 20-40% nos países ocidentais. A dieta hiperlipídica e/ou rica em sacarose pode influenciar no desenvolvimento da esteatose hepática associada à obesidade e a resistência à insulina. O fígado, por assumir papel central no controle metabólico, é um órgão alvo nos casos de excesso alimentar, ocasionando, principalmente, acúmulo de gotículas de gordura nos hepatócitos. Este trabalho teve como objetivo avaliar o início das alterações morfológicas e metabólicas no fígado e no tecido adiposo de camundongos suíços machos alimentados com dieta hiperlipídica e/ou rica em sacarose. Camundongos suíços machos aos três meses de idade foram divididos em quatro grupos nutricionais: dieta padrão (SC), dieta hiperlipídica (HF), dieta rica em sacarose (HSu) e dieta hiperlipídica rica em sacarose (HFHSu). Os animais receberam as respectivas dietas durante quatro semanas. A massa corporal, a ingestão alimentar e a tolerância oral à glicose foram avaliados. Ao sacrifício, o fígado e os depósitos de gordura corporal foram removidos e processados para análises histomorfométricas e moleculares. As amostras de sangue foram obtidas para análises bioquímicas plasmáticas. Os dados foram expressos como média e erro padrão da média e as diferenças foram testadas por one-way ANOVA com pós-teste de Holm-Sidak, e foi considerado o nível de significância de p<0,05. Os grupos HF e HFHSu apresentaram-se mais pesados quando comparados aos grupos SC e HSu. Os animais dos grupos HF, HSu e HFHSu apresentaram intolerância à glicose, esteatose hepática e aumento de triglicerídeos hepáticos quando comparados ao grupo SC (p<0,0005). Adicionalmente, houve elevação na expressão hepática das proteínas transportador de glicose 2 (GLUT-2), proteína de ligação ao elemento regulador do esterol 1-c (SREBP1-c), fosfoenolpiruvato carboxiquinase (PEPCK), glicose -6- fosfatase (G6PASE), substrato do receptor da insulinaI-1 (IRS-1) e proteína quinase B (AKt/ou PKB) e redução da expressão no fígado do receptor ativador de proliferação peroxissomal (PPAR-α) nos grupos experimentais em comparação com o grupo SC (p<0,0005). A administração de dieta hiperlipídica e/ou rica em sacarose promoveu intolerância à glicose e danos hepáticos (hepatomegalia, esteatose, redução da beta-oxidação, aumento na lipogênese e na produção de glicose) em camundongos machos adultos.
The non-alcoholic fatty liver disease is a multifactorial disorder caused mainly by excess nutritional and insulin resistance, with an estimated prevalence of 20-40% in Western countries. The diet can to influence in the development of fatty liver associated with obesity and insulin resistance. The liver plays a central role in metabolic control, is a target organ, in case of excess of food, mainly causing accumulation of fat droplets in hepatocytes. This study aimed to evaluate the early morphological and metabolic changes in the liver and adipose tissue of male Swiss mice fed high-fat and/or high-sucrose diets. Twenty three-month-old male Swiss Webster mice were divided into 4 groups: standard chow (SC), high-fat diet (HF), high-sucrose diet (HSu) and high-fat-high-sucrose diet (HFHSu). Animals received the respective diets for 4 weeks; throughout the experiment, body mass, food intake and oral glucose tolerance were evaluated. After the mice were euthanized, the liver was removed and processed for histomorphometrical and molecular analysis. Blood samples were obtained for serum analysis. The data were tested by one-way ANOVA with a Holm-Sidak post-hoc test and were expressed as the mean standard error of the mean; the significance level was set at p < 0.05. The HF and HFHSu groups were heavier than the SC and HSu groups. Animals from the HF, HSu and HFHSu groups presented glucose intolerance, hepatomegaly, liver steatosis and augmented hepatic triglycerides when compared to the SC group (p<0.0005). Additionally, there was an elevation in glucose transporter 2 (GLUT-2), sterol regulatory element binding protein-1c (SREBP-1c), phosphoenolpyruvate carboxykinase (PEPCK), glucose 6 phosphatase (G6PASE), insulin receptor substrate 1 (IRS-1), protein kinase B (AKT/ or PKB) protein expression and a reduction in peroxisome proliferator-activated receptor alpha (PPAR-alpha) expression in liver from the experimental groups compared to those of the SC group (p<0.0005). Administration of high-fat and/or high-sucrose diets promoted glucose intolerance and liver damage (hepatomegaly, steatosis, reduced beta-oxidation, increased lipogenesis and glucose production) in adult male mice.
Silva, Sandra Barbosa da. "Efeitos benéficos do tratamento com óleo de peixe em camundongos c57bl/6 alimentados com dieta hiperlipídica e rica em sacarose." Universidade do Estado do Rio de Janeiro, 2008. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=3258.
Повний текст джерелаAvaliar os efeitos benéficos do tratamento com óleo de peixe sobre mudanças metabólicas e morfológicas no pâncreas e tecido adiposo de camundongos C57BL/6 alimentados com dieta rica em lipídeos e sacarose (HLS).Camundongos machos da linhagem C57BL/6, foram alimentados com dieta padrão (P) ou dieta HLS. Aos 3 meses de idade, os camundongos do grupo HLS foram separados em grupo não-tratado (HLS) ou grupo tratado com óleo de peixe (HLS-Px, 1,5g/kg/dia). Aos 4 meses de idade os animais foram sacrificados. O grupo HLS apresentou aumento da massa corporal (MC) e no acúmulo do tecido adiposo total, porém o grupo HLS-Px apresentou menor MC e massa de tecido adiposo comparado ao grupo HLS. As concentrações de glicose plasmática e insulina não foram afetadas entre os grupos, no entanto os grupos HLS e HLS-Px apresentaram maior HOMA-IR. Os grupos HLS e HLS-Px apresentaram maiores concentrações plasmáticas do colesterol total e LDL-C, porém o grupo HLS-Px apresentou maior concentração plasmática do HDL-C e redução da concentração de triglicerídeos. Os adipócitos do grupo HLS apresentaram maior diâmetro quando comparado aos grupos controle e HLS-Px. A massa do pâncreas foi menor no grupo HLS-Px e as ilhotas pancreáticas apresentaram maior diâmetro no grupo HLS, quando comparado ao grupo controle. A expressão de insulina, glucagon e GLUT-2 mostrou-se forte em todas as ilhotas pancreáticas do grupo controle, mas o grupo HLS apresentou fraca expressão para o GLUT-2. Entretanto, HLS-Px apresentou maior expressão do GLUT-2. O tratamento com óleo de peixe foi capaz de reduzir o ganho de massa corporal e a concentração de triglicerídeos, assim como reduzir o acúmulo de tecido adiposo,hipertrofia dos adipócitos, das ilhotas pancreáticas, assim como prevenir a redução do GLUT-2 em camundongos C57BL/6.
To evaluate the fish oil treatment upon morphological and metabolic changes in the pancreas and adipose tissue of C57BL/6 mice fed high-fat-high-sucrose (HFHS) diet. Male C57BL/6 mice were fed HFHS chow or standard chow (SC). At 3 mo-old, HFHS mice were separated into untreated group (HFHS) or treated with fish oil (HFHS-Fo, 1.5 g/kg/day). At 4-mo-old animals were sacrificed. HFHS had increase in body mass (BM) and in total body fat, but HFHS-Fo had smaller BM and total body fat in relation to HFHS. Plasma glucose and insulin levels were not affected among the groups, but HFHS and HFHS-Fo had higher HOMA-IR ratio. HFHS and HFHS-FO had increased plasma total cholesterol and LDL-C, but HFHS-Fo increased plasma HDL-C and decreased triglycerides levels. The adipocytes size were greater in HFHS, when compared to SC and HFHS-Fo groups. HFHS-Fo had smaller pancreas mass and HFHS presented higher islet pancreatic diameter, when compared to SC group. SC group showed strong expression for insulin, glucagon and GLUT-2 in all pancreatic islets, and HFHS presented lesser expression for GLUT-2. However, HFHS-Fo presented increase of GLUT-2 expression. Fish oil treatment was able to reduce body mass gain and plasma TG, reduce fat pad adiposity as well as adipocyte and pancreatic islet hypertrophy, prevent decrease GLUT-2 in C57BL/6.
Benedet, Lynda Louise. "Use of a rice bran in tropical grower-finisher swine diets." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ27481.pdf.
Повний текст джерелаSantos, Aline de Sousa dos. "Disfunção mitocondrial e cardíaca em camundongos induzida por dieta rica em ácidos graxos poliinsaturados." Universidade do Estado do Rio de Janeiro, 2012. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=5951.
Повний текст джерелаIndivíduos obesos apresentam maior risco de morbidade e mortalidade atribuída às doenças cardiovasculares. A composição da dieta é um fator que prediz o fenótipo cardíaco em resposta a obesidade e, o tipo de ácido graxo pode afetar de forma diferencial a estrutura e a função do miocárdio. Estudos têm mostrado que a disfunção mitocondrial exerce um papel chave na patogênese da insuficiência e hipertrofia cardíaca, e as alterações mitocondriais observadas em falhas cardíacas apontam para defeitos em sítios específicos da cadeia transportadora de elétrons. Desta forma, o objetivo deste estudo foi avaliar a função contrátil ventricular em camundongos, alimentados com dieta hiperlipídica, rica em ácidos graxos poliinsaturados, buscando elucidações através da bioenergética mitocondrial. Após desmame, camundongos machos C57Bl/6 passaram a receber dieta manipulada contendo 7% (C) ou 19% (HF) de óleo de soja, até os 135 dias de idade. A ingestão alimentar e a massa corporal foram monitoradas e foi realizado teste de tolerância à glicose. No final do período experimental, os animais foram anestesiados e submetidos à avaliação da composição corporal por Absortimetria de Raios X de Dupla Energia (DXA), e em seguida, sacrificados por exsanguinação. No plasma foram determinados o perfil lipídico e a insulina. O coração, o tecido adiposo intra-abdominal e o subcutâneo foram coletados, pesados, processados para análise histomorfológica. Fibras cardíacas do ventrículo esquerdo foram utilizadas para análise da respiração mitocondrial através de oxígrafo. O coração também foi utilizado para a técnica de perfusão de coração isolado de Langendorff, e para análise da expressão de proteínas relacionadas à bioenergética de cardiomiócitos, através de Western Blotting. O índice de HOMA e de adiposidade foram calculados. O grupo HF apresentou maior adiposidade, sem alteração na ingestão alimentar. Foi observada intolerância a glicose, hiperinsulinemia e resistência à insulina, além de alterações desfavoráveis no perfil lipídico. Foi observado alteração na morfologia cardíaca e quadro de cardiomiopatia hipertrófica, refletindo em alteração hemodinâmica, determinando maior contratilidade, maior pressão ventricular e função diastólica prejudicada. Em relação à atividade mitocondrial dos cardiomiócitos foi observada menor oxidação de carboidratos (-47%) e de ácidos graxos (-60%). Porém, sem alteração na expressão de proteínas relacionadas à bioenergética de cardiomiócitos, CPT1, UCP2, GLUT1, GLUT4, AMPK e pAMPK. A partir desses resultados, concluímos que o tipo e a quantidade de ácidos graxos predizem o fenótipo cardíaco na obesidade, promovendo alteração na capacidade oxidativa mitocondrial, na morfologia e na hemodinâmica cardíaca
Obese individuals have a higher risk of morbidity and mortality attributed to cardiovascular disease. The diet composition is one factor that predicts the cardiac phenotype in response to obesity and the type of fatty acid differentially influences the myocardial structure and function. Studies have showed that mitochondrial dysfunction is considered to play a key role in the pathogenesis of cardiac hypertrophy and failure, also the mitochondrial alterations present in heart failure indicate to defects at specific sites in electron transport chain. Thus, the aim of the study was evaluated the ventricular contractile function in mice fed high fat diet, rich in polyunsaturated fatty acids, looking through mitochondrial bioenergetics. After weaning, mouse C57Bl/6 received manipulated diet containing 7% (C) or 19% (HF) of soybean oil, until 135 days of age. The food intake and the body mass were monitored, and the glucose tolerance test was realized. At the end of the experimental period, the animals had their body composition evaluated by Dual-energy X-ray Absorptiometry (DXA), after, were sacrificed by exsanguination. In plasma was determined the lipid profile and insulin. The heart, intraabdominal and subcutaneous adipose tissue were collected, weighted and processed to morphological analysis. The left ventricular myocardial fibers were used to analyze mitochondrial respiration by technique of high resolution respirometry. The heart was used to the Langendorff technique of isolated heart perfusion, and to analyze the expression of proteins related to the cardiomyocytes bioenergetics, through of Western Blotting. The HOMA-IR and the adiposity index were calculated. The group HF showed higher adiposity, but did not differ about food intake. Was observed glucose intolerance, hiperinsulinemia, insulin resistance and also unfavorable alterations in lipid profile. Was observed alterations in cardiac morphology and hypertrophic cardiomyopathy, reflecting in hemodinamic alterations with increase of contractility, higher ventricular pressure and impaired diastolic function. About the mitochondrial activity of cardiomyocytes was observed lower oxidation of carbohydrates (-47%) and fatty acids (-60%). However, the expression of proteins related to the cardiomyocytes bioenergetics, CPT1, UCP2, GLUT1, GLUT4, AMPK e pAMPK, did not differ between the groups. From these results, we conclude that the type and amount of fatty acids predict the cardiac phenotype in obesity, promoting the impairment of mitochondrial oxidative capacity, alterations in cardiac morphology and hemodynamics
Книги з теми "Rice Diet"
Robert, Rosati, ed. The rice diet solution: The world famous low-sodium, good-carb, detox diet for quick and lasting weight loss. New York: Simon & Schuster, 2006.
Знайти повний текст джерелаRobert, Rosati, ed. The rice diet solution: The world-famous low-sodium, good-carb, detox diet for quick and lasting weight loss. New York: Simon & Schuster, 2007.
Знайти повний текст джерелаRosati, Kitty Gurkin. The rice diet cookbook: 150 easy, everyday recipes and inspirational success stories from the rice diet community. New York: Berkley Books, 2007.
Знайти повний текст джерелаFlinn, J. C. Contributions of modern rice varieties to nutrition in Asia. Manila, Philippines: International Rice Research Institute, 1985.
Знайти повний текст джерелаMoscovitz, Judy. The rice diet report: How I lost up to 12 pounds a week on the world-famous weight-loss plan. New York: G.P. Putnam's Sons, 1986.
Знайти повний текст джерелаMoscovitz, Judy. The rice diet report: How I lost up to 12 poundsa week on the world-famous weight-loss plan. New York: G.P. Putnam's Sons, 1986.
Знайти повний текст джерелаNgô, Thanh Nhàn. Ăn gạo lứt muối mè: Tăng cường sức khỏe và trị bệnh theo phương pháp thực dưỡng Ohsawa = Macrobiotic eating for health and happiness. 5-те вид. [Đà Nẵng]: NXB Đà Nẵng, 2008.
Знайти повний текст джерелаDrago, Lorena. Beyond rice and beans. Alexandria: American Diabetes Association, 2005.
Знайти повний текст джерелаNewborg, Barbara. Walter Kempner and the rice diet: Challenging conventional wisdom. Durham, N.C: Carolina Academic Press, 2011.
Знайти повний текст джерелаSams, Craig. The macrobiotic brown rice cookbook: Delicious and wholesome grain-based dishes. Rochester, Vt: Healing Arts Press, 1993.
Знайти повний текст джерелаЧастини книг з теми "Rice Diet"
Bresciani, Andrea, Maria Ambrogina Pagani, and Alessandra Marti. "Rice: A Versatile Food at the Heart of the Mediterranean Diet." In Cereal-Based Foodstuffs: The Backbone of Mediterranean Cuisine, 193–229. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-69228-5_8.
Повний текст джерелаGanguly, Kavery, and Ashok Gulati. "Pulses Value Chain- Pigeon Pea and Gram." In India Studies in Business and Economics, 253–86. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-33-4268-2_8.
Повний текст джерелаSeedorf, Thomas. "Buxtehude, Diet(e)rich." In Metzler Komponisten Lexikon, 114–16. Stuttgart: J.B. Metzler, 1992. http://dx.doi.org/10.1007/978-3-476-03421-2_41.
Повний текст джерелаSeedorf, Thomas. "Buxtehude, Diet(e)rich." In Komponisten Lexikon, 90–91. Stuttgart: J.B. Metzler, 2003. http://dx.doi.org/10.1007/978-3-476-05274-2_40.
Повний текст джерелаChen, Chang-Hwei. "Diets Rich in Enzyme Modulators." In Activation and Detoxification Enzymes, 103–11. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4614-1049-2_11.
Повний текст джерелаMatres, Jerlie Mhay, Erwin Arcillas, Maria Florida Cueto-Reaño, Ruby Sallan-Gonzales, Kurniawan R. Trijatmiko, and Inez Slamet-Loedin. "Biofortification of Rice Grains for Increased Iron Content." In Rice Improvement, 471–86. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-66530-2_14.
Повний текст джерелаXu, J., L. Hulett, A. Barnes, R. Albridge, and N. Tolk. "Rotational Distributions Following DIET: CN* from Alkali-Rich Surfaces." In Springer Series in Surface Sciences, 313–16. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-78080-6_51.
Повний текст джерелаIslam, S., M. M. Rahman, and R. Naidu. "Health risk of inorganic arsenic from rice-based diets." In Arsenic in the Environment: Bridging Science to Practice for Sustainable Development As2021, 280–81. London: CRC Press, 2024. http://dx.doi.org/10.1201/9781003317395-114.
Повний текст джерелаAhmed, A. U., and J. Ghostlaw. "Diversifying rice-centric agriculture and diets: the Bangladesh experience." In Agriculture for improved nutrition: seizing the momentum, 154–64. Wallingford: CABI, 2019. http://dx.doi.org/10.1079/9781786399311.0154.
Повний текст джерелаDreher, Mark L. "Fiber-Rich Diets in Chronic Kidney Disease." In Dietary Fiber in Health and Disease, 305–18. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-50557-2_15.
Повний текст джерелаТези доповідей конференцій з теми "Rice Diet"
Parklak, W., N. Munkong, S. Somnuk, N. Sompara, J. Naowaboot, B. Yoysungnoen, and N. Lerdvuthisopon. "Effect of rice bran extract on pancreas of rats fed a high-fat diet." In GA 2017 – Book of Abstracts. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1608340.
Повний текст джерелаFernández, C., T. Romero, and M. Lachica. "Orange leaves and rice straw in the diet of lactating goats: effect on energy balance." In 6th EAAP International Symposium on Energy and Protein Metabolism and Nutrition. The Netherlands: Wageningen Academic Publishers, 2019. http://dx.doi.org/10.3920/978-90-8686-891-9_74.
Повний текст джерелаBadrul Zaman, Maslia Manja, Amelia Najwa Ahmad Hairi, Norliza Saparin, and Ahmadilfitri Md Noor. "Candelilla and Rice Bran Wax as Oleogelators in Soybean Oil for Deep Frying Application." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/jltk8162.
Повний текст джерелаYulistiani, Dwi, Wisri Puastuti, and Yeni Widiawati. "In Vitro Digestibility and Rumen Fermentation of Grass or Rice Straw Basal Diet With or Without Complete Rumen Modifier Supplementation." In Proceedings of International Seminar on Livestock Production and Veterinary Technology. Indonesian Center for Animal Research and Development (ICARD), 2016. http://dx.doi.org/10.14334/proc.intsem.lpvt-2016-p.310-317.
Повний текст джерелаShin, Woo-Kyoung, Hwi-Won Lee, Aesun Shin, Jong-koo Lee, and Daehee Kang. "Abstract LB-279: Multi-grain rice diet decreases risk of breast cancer in Korean women: Results from the Health Examinees study." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-lb-279.
Повний текст джерелаGunawan, Winarti Erna, Sutardi, and Kusnadi Harwi. "Effect of rice bran and mineral supplementation on forage-based diet on growth performance and cost efficiency of ongole crossbred bulls." In INTERNATIONAL CONFERENCE ON ORGANIC AND APPLIED CHEMISTRY (ICOAC) 2022. AIP Publishing, 2024. http://dx.doi.org/10.1063/5.0184975.
Повний текст джерелаDaulay, Muhammad Rizqi Hariz, Dimas Hand Vidya Paradhipta, Cuk Tri Noviandi, Ali Agus, Andriyani Astuti, and Karen Harper. "Supplementation Effects of Ground Cassava and Cassava Leaves with Different Ratios on In Vitro Digestibility of Rice Straw Based-Diet." In 9th International Seminar on Tropical Animal Production (ISTAP 2021). Paris, France: Atlantis Press, 2022. http://dx.doi.org/10.2991/absr.k.220207.004.
Повний текст джерелаStoin, Daniela, Mariana-Atena Poiana, Calin Jianu, Ileana Cocan, and Monica Negrea. "SENSORY AND PHYSICO-CHEMICAL EVALUATION OF GLUTEN-FREE CRUFFINS OBTAINED FROM ALMOND FLOUR AND RICE FLOUR." In 22nd SGEM International Multidisciplinary Scientific GeoConference 2022. STEF92 Technology, 2022. http://dx.doi.org/10.5593/sgem2022v/6.2/s25.37.
Повний текст джерелаFrljak, Jasna, Senita Isaković, Enver Karahmet, Almir Toroman, Ervina Bečić, Fahir Bečić, and Zoran Marković. "Status and quality of life of people with celiac and people on a gluten-free diet." In 2nd International Conference on Chemo and Bioinformatics. Institute for Information Technologies, University of Kragujevac, 2023. http://dx.doi.org/10.46793/iccbi23.269f.
Повний текст джерелаVasyukova, A. T., D. A. Tikhonov, R. A. Edwards, M. V. Vasyukov, and Talbi Mounir. "Macro- and microelements in new products for the population of ecological territories at risk." In III All-Russian Scientific Conference with International Participation "Science, technology, society: Environmental engineering for sustainable development of territories". Krasnoyarsk Science and Technology City Hall, 2022. http://dx.doi.org/10.47813/nto.3.2022.6.617-621.
Повний текст джерелаЗвіти організацій з теми "Rice Diet"
Kanner, Joseph, Mark Richards, Ron Kohen, and Reed Jess. Improvement of quality and nutritional value of muscle foods. United States Department of Agriculture, December 2008. http://dx.doi.org/10.32747/2008.7591735.bard.
Повний текст джерелаGERGELY SZOCSI, ANDRADA ELISABETA, and Juan Gambini. El potencial uso del hidroxitirosol en humanos. Fundación Avanza, May 2024. http://dx.doi.org/10.60096/fundacionavanza/5032024.
Повний текст джерелаProvenza, Frederick, Avi Perevolotsky, and Nissim Silanikove. Consumption of Tannin-Rich Forage by Ruminants: From Mechanism to Improved Performance. United States Department of Agriculture, April 2000. http://dx.doi.org/10.32747/2000.7695840.bard.
Повний текст джерелаCzowalla, Lucas, Dahlia Busch, Andrea Fromberg, Peter Gwiasda, Mathias Wilde, and Martin Lanzendorf. Neuere Entwicklungen zur Integration von Fahrrad und Öffentlichem Verkehr in Deutschland: Überblick zum Stand des Wissens und der Praxis. Goethe-Universität, Institut für Humangeographie, October 2017. http://dx.doi.org/10.21248/gups.40314.
Повний текст джерелаAparicio Llanos, Amada. Serie sobre hospitalizaciones evitables y fortalecimiento de la atención primaria en salud: El caso de Costa Rica. Inter-American Development Bank, December 2012. http://dx.doi.org/10.18235/0007671.
Повний текст джерелаJHA, Anil. Revitalising millets in Northeast India: A healthy choice - Policy Brief. International Centre for Integrated Mountain Development (ICIMOD), December 2023. http://dx.doi.org/10.53055/icimod.1041.
Повний текст джерелаHouzer, Ella, and Ian Scoones. Are Livestock Always Bad for the Planet? Rethinking the Protein Transition and Climate Change Debate. Institute of Development Studies (IDS), September 2021. http://dx.doi.org/10.19088/steps.2021.003.
Повний текст джерелаBernal, Pedro, Giuliana Daga, and Florencia Lopez Boo. Do Behavioral Drivers Matter for Healthcare Decision-making in Times of Crisis?: A study of Low-Income Women in El Salvador During the COVID-19 Pandemic. Inter-American Development Bank, August 2023. http://dx.doi.org/10.18235/0005094.
Повний текст джерелаLandau, Sergei Yan, John W. Walker, Avi Perevolotsky, Eugene D. Ungar, Butch Taylor, and Daniel Waldron. Goats for maximal efficacy of brush control. United States Department of Agriculture, March 2008. http://dx.doi.org/10.32747/2008.7587731.bard.
Повний текст джерелаEinarsson, Rasmus. Nitrogen in the food system. TABLE, February 2024. http://dx.doi.org/10.56661/2fa45626.
Повний текст джерела