Готові списки джерел за темами / RecF

Добірка наукової літератури з теми "RecF"

Автор: Grafiati
Опубліковано: 6 вересня 2023

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Статті в журналах з теми "RecF"

1

Luisi-DeLuca, C., S. T. Lovett, and R. D. Kolodner. "Genetic and physical analysis of plasmid recombination in recB recC sbcB and recB recC sbcA Escherichia coli K-12 mutants." Genetics 122, no. 2 (June 1, 1989): 269–78. http://dx.doi.org/10.1093/genetics/122.2.269.

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Abstract The effect of mutations in known recombination genes (recA, recB, recC, recE, recF, recJ, recN, recO, recQ and ruv) on intramolecular recombination of plasmids was studied in recB recC sbcB and recB recC sbcA Escherichia coli mutants. The rate of recombination of circular dimer plasmids was at least 1000-fold higher in recB recC sbcB or recB recC sbcA mutants as compared to wild-type cells. The rate was decreased by mutations in recA, recF, recJ, recO, ruv or mutS in recB recC sbcB mutants, and by mutations in recE, recN, recO, recQ, ruv or mutS in recB recC sbcA mutants. In addition to measuring the recombination rate of circular dimer plasmids, the recombination-mediated transformation of linear dimer plasmids was also studied. Linear dimer plasmids transformed recB recC sbcB and recB recC sbcA mutants 20- to 40-fold more efficiently than wild-type cells. The transformation efficiency of linear dimer plasmids in recB recC sbcB mutants was decreased by mutations in recA, recF, recJ, recO, recQ or lexA (lexA3). In recB recC sbcA mutants the transformation efficiency of linear dimers was decreased only by a recE mutation. Physical analysis of linear dimer- or circular dimer-transformed recB recC sbcB mutants revealed that all transformants contained recombinant monomer genotypes. This suggests that recombination in recB recC sbcB cells is very efficient.
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2

Lovett, S. T., C. Luisi-DeLuca, and R. D. Kolodner. "The genetic dependence of recombination in recD mutants of Escherichia coli." Genetics 120, no. 1 (September 1, 1988): 37–45. http://dx.doi.org/10.1093/genetics/120.1.37.

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Abstract RecBCD enzyme has multiple activities including helicase, exonuclease and endonuclease activities. Mutations in the genes recB or recC, encoding two subunits of the enzyme, reduce the frequency of many types of recombinational events. Mutations in recD, encoding the third subunit, do not reduce recombination even though most of the activities of the RecBCD enzyme are severely reduced. In this study, the genetic dependence of different types of recombination in recD mutants has been investigated. The effects of mutations in genes in the RecBCD pathway (recA and recC) as well as the genes specific for the RecF pathway (recF, recJ, recN, recO, recQ, ruv and lexA) were tested on conjugational, transductional and plasmid recombination, and on UV survival. recD mutants were hyper-recombinogenic for all the monitored recombination events, especially those involving plasmids, and all recombination events in recD strains required recA and recC. In addition, unlike recD+ strains, chromosomal recombination events and the repair of UV damage to DNA in recD strains were dependent on one RecF pathway gene, recJ. Only a subset of the tested recombination events were affected by ruv, recN, recQ, recO and lexA mutations.
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3

Sawitzke, J. A., and F. W. Stahl. "Phage lambda has an analog of Escherichia coli recO, recR and recF genes." Genetics 130, no. 1 (January 1, 1992): 7–16. http://dx.doi.org/10.1093/genetics/130.1.7.

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Abstract The RecF pathway catalyzes generalized recombination in Escherichia coli that is mutant for recBC, sbcB and sbcC. This pathway operating on conjugational recombination requires the recA, recF, recJ, recN, recO, recQ, recR, ruvA, ruvB and ruvC genes. In contrast, lambda mutant for its own recombination genes, int, red alpha and red beta, requires only the recA and recJ genes to recombine efficiently in recBC sbcB sbcC cells. Deletion of an open reading frame in the ninR region of lambda results in an additional requirement for recO, recR and recF in order to recombine in recBC sbcB sbcC mutant cells. This function, designated orf for recO-, recR- and recF-like function, is largely RecF pathway specific.
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4

Stohl, Elizabeth A., and H. Steven Seifert. "Neisseria gonorrhoeae DNA Recombination and Repair Enzymes Protect against Oxidative Damage Caused by Hydrogen Peroxide." Journal of Bacteriology 188, no. 21 (August 25, 2006): 7645–51. http://dx.doi.org/10.1128/jb.00801-06.

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ABSTRACT The strict human pathogen Neisseria gonorrhoeae is exposed to oxidative damage during infection. N. gonorrhoeae has many defenses that have been demonstrated to counteract oxidative damage. However, recN is the only DNA repair and recombination gene upregulated in response to hydrogen peroxide (H2O2) by microarray analysis and subsequently shown to be important for oxidative damage protection. We therefore tested the importance of RecA and DNA recombination and repair enzymes in conferring resistance to H2O2 damage. recA mutants, as well as RecBCD (recB, recC, and recD) and RecF-like pathway mutants (recJ, recO, and recQ), all showed decreased resistance to H2O2. Holliday junction processing mutants (ruvA, ruvC, and recG) showed decreased resistance to H2O2 resistance as well. Finally, we show that RecA protein levels did not increase as a result of H2O2 treatment. We propose that RecA, recombinational DNA repair, and branch migration are all important for H2O2 resistance in N. gonorrhoeae but that constitutive levels of these enzymes are sufficient for providing protection against oxidative damage by H2O2.
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5

Ivančić-Baće, Ivana, Erika Salaj-Šmic, and Krunoslav Brčić-Kostić. "Effects of recJ, recQ, and recFOR Mutations on Recombination in Nuclease-Deficient recB recD Double Mutants of Escherichia coli." Journal of Bacteriology 187, no. 4 (February 15, 2005): 1350–56. http://dx.doi.org/10.1128/jb.187.4.1350-1356.2005.

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ABSTRACT The two main recombination pathways in Escherichia coli (RecBCD and RecF) have different recombination machineries that act independently in the initiation of recombination. Three essential enzymatic activities are required for early recombinational processing of double-stranded DNA ends and breaks: a helicase, a 5′→3′ exonuclease, and loading of RecA protein onto single-stranded DNA tails. The RecBCD enzyme performs all of these activities, whereas the recombination machinery of the RecF pathway consists of RecQ (helicase), RecJ (5′→3′ exonuclease), and RecFOR (RecA-single-stranded DNA filament formation). The recombination pathway operating in recB (nuclease-deficient) mutants is a hybrid because it includes elements of both the RecBCD and RecF recombination machineries. In this study, genetic analysis of recombination in a recB (nuclease-deficient) recD double mutant was performed. We show that conjugational recombination and DNA repair after UV and gamma irradiation in this mutant are highly dependent on recJ, partially dependent on recFOR, and independent of recQ. These results suggest that the recombination pathway operating in a nuclease-deficient recB recD double mutant is also a hybrid. We propose that the helicase and RecA loading activities belong to the RecBCD recombination machinery, while the RecJ-mediated 5′→3′ exonuclease is an element of the RecF recombination machinery.
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6

Ivančić-Baće, Ivana, Petra Peharec, Sunčana Moslavac, Nikolina Škrobot, Erika Salaj-Šmic†, and Krunoslav Brčić-Kostić. "RecFOR Function Is Required for DNA Repair and Recombination in a RecA Loading-Deficient recB Mutant of Escherichia coli." Genetics 163, no. 2 (February 1, 2003): 485–94. http://dx.doi.org/10.1093/genetics/163.2.485.

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Abstract The RecA loading activity of the RecBCD enzyme, together with its helicase and 5′ → 3′ exonuclease activities, is essential for recombination in Escherichia coli. One particular mutant in the nuclease catalytic center of RecB, i.e., recB1080, produces an enzyme that does not have nuclease activity and is unable to load RecA protein onto single-stranded DNA. There are, however, previously published contradictory data on the recombination proficiency of this mutant. In a recF– background the recB1080 mutant is recombination deficient, whereas in a recF+ genetic background it is recombination proficient. A possible explanation for these contrasting phenotypes may be that the RecFOR system promotes RecA-single-strand DNA filament formation and replaces the RecA loading defect of the RecB1080CD enzyme. We tested this hypothesis by using three in vivo assays. We compared the recombination proficiencies of recB1080, recO, recR, and recF single mutants and recB1080 recO, recB1080 recR, and recB1080 recF double mutants. We show that RecFOR functions rescue the repair and recombination deficiency of the recB1080 mutant and that RecA loading is independent of RecFOR in the recB1080 recD double mutant where this activity is provided by the RecB1080C(D–) enzyme. According to our results as well as previous data, three essential activities for the initiation of recombination in the recB1080 mutant are provided by different proteins, i.e., helicase activity by RecB1080CD, 5′ → 3′ exonuclease by RecJ- and RecA-single-stranded DNA filament formation by RecFOR.
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7

Jain, Kanika, Elizabeth A. Wood, and Michael M. Cox. "The rarA gene as part of an expanded RecFOR recombination pathway: Negative epistasis and synthetic lethality with ruvB, recG, and recQ." PLOS Genetics 17, no. 12 (December 22, 2021): e1009972. http://dx.doi.org/10.1371/journal.pgen.1009972.

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The RarA protein, homologous to human WRNIP1 and yeast MgsA, is a AAA+ ATPase and one of the most highly conserved DNA repair proteins. With an apparent role in the repair of stalled or collapsed replication forks, the molecular function of this protein family remains obscure. Here, we demonstrate that RarA acts in late stages of recombinational DNA repair of post-replication gaps. A deletion of most of the rarA gene, when paired with a deletion of ruvB or ruvC, produces a growth defect, a strong synergistic increase in sensitivity to DNA damaging agents, cell elongation, and an increase in SOS induction. Except for SOS induction, these effects are all suppressed by inactivating recF, recO, or recJ, indicating that RarA, along with RuvB, acts downstream of RecA. SOS induction increases dramatically in a rarA ruvB recF/O triple mutant, suggesting the generation of large amounts of unrepaired ssDNA. The rarA ruvB defects are not suppressed (and in fact slightly increased) by recB inactivation, suggesting RarA acts primarily downstream of RecA in post-replication gaps rather than in double strand break repair. Inactivating rarA, ruvB and recG together is synthetically lethal, an outcome again suppressed by inactivation of recF, recO, or recJ. A rarA ruvB recQ triple deletion mutant is also inviable. Together, the results suggest the existence of multiple pathways, perhaps overlapping, for the resolution or reversal of recombination intermediates created by RecA protein in post-replication gaps within the broader RecF pathway. One of these paths involves RarA.
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8

Poteete, Anthony R. "Modulation of DNA Repair and Recombination by the Bacteriophage λ Orf Function in Escherichia coli K-12". Journal of Bacteriology 186, № 9 (1 травня 2004): 2699–707. http://dx.doi.org/10.1128/jb.186.9.2699-2707.2004.

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ABSTRACT The orf gene of bacteriophage λ, fused to a promoter, was placed in the galK locus of Escherichia coli K-12. Orf was found to suppress the recombination deficiency and sensitivity to UV radiation of mutants, in a Δ(recC ptr recB recD)::P tac gam bet exo pae cI ΔrecG background, lacking recF, recO, recR, ruvAB, and ruvC functions. It also suppressed defects of these mutants in establishing replication of a pSC101-related plasmid. Compared to orf, the recA803 allele had only small effects on recF, recO, and recR mutant phenotypes and no effect on a ruvAB mutant. In a fully wild-type background with respect to known recombination and repair functions, orf partially suppressed the UV sensitivity of ruvAB and ruvC mutants.
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9

Nakayama, Koji, Susumu Shiota, and Hiroaki Nakayama. "Thymineless death in Escherichia coli mutants deficient in the RecF recombination pathway." Canadian Journal of Microbiology 34, no. 7 (July 1, 1988): 905–7. http://dx.doi.org/10.1139/m88-157.

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Like recF and recQ mutants studied earlier, two other classes of Escherichia coli mutants defective in the RecF conjugal recombination pathway, recJ and recO, were found to be partially resistant to thymineless death. In contrast, a recN mutant, also belonging to the pathway, was indistinguishable from the wild type with respect to thymineless death.
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10

Shiraishi, Kouya, Katsuhiro Hanada, Yoichiro Iwakura, and Hideo Ikeda. "Roles of RecJ, RecO, and RecR in RecET-Mediated Illegitimate Recombination in Escherichia coli." Journal of Bacteriology 184, no. 17 (September 1, 2002): 4715–21. http://dx.doi.org/10.1128/jb.184.17.4715-4721.2002.

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ABSTRACT We analyzed effects of overexpression of RecE and RecT on illegitimate recombination during prophage induction in Escherichia coli and found that frequencies of spontaneous and UV-induced illegitimate recombination are enhanced by coexpression of RecE and RecT in the wild type, but the enhanced recombination was reduced by recJ, recO, or recR mutation. The results indicated that RecET-mediated illegitimate recombination depends on the functions of RecJ, RecO, and RecR, suggesting that the RecE and RecJ exonucleases play different roles in this recombination pathway and that the RecO and RecR proteins also play important roles in the recombination. On the other hand, the frequency of the RecET-mediated illegitimate recombination was enhanced by a recQ mutation, implying that the RecQ protein plays a role in suppression of RecET-mediated illegitimate recombination. It was also found that RecET-mediated illegitimate recombination is independent of the RecA function with UV irradiation, but it is enhanced by the recA mutation without UV irradiation. Based on these results, we propose a model for the roles of RecJOR on RecET-mediated illegitimate recombination.
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Більше джерел

Дисертації з теми "RecF"

1

Raharjo, Joko Purnomo. "The performance analysis of real estate construction firms (RECF) in Indonesia." Thesis, Queensland University of Technology, 2022. https://eprints.qut.edu.au/228029/1/Joko%20Purnomo_Raharjo_Thesis.pdf.

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This thesis examines the efficiency, productivity, and survival of real estate construction firms in Indonesia. By applying a meta-frontier approach, the results show that efficiency and productivity of the firms are relatively low. The gap between the industry and the small, medium, and large is high. This suggest that the overall industry could obtain remarkable efficiency and productivity improvement if firms could access technologies used by more efficient firms. Designing policy and managerial interventions for each group would have greater impacts. The survival analysis demonstrates that profit margin, income diversification, experience and structure are strongly related to firm survival.
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2

Taylor, L. "The recB and recC gene products of Escherichia coli." Thesis, University of Newcastle Upon Tyne, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.355080.

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3

Wilson, R. E. "The recB-recC region of the Escherichia coli chromosome." Thesis, University of Newcastle Upon Tyne, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.375598.

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4

Vickridge, Elise. "Management of E. coli sister chromatid cohesion in response to genotoxic stress." Thesis, Université Paris-Saclay (ComUE), 2018. http://www.theses.fr/2018SACLS172/document.

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La réplication fidèle de l’ADN au cours du cycle cellulaire est essentielle au maintien de l’intégrité du génome à travers les générations. Toutefois, de nombreux éléments peuvent perturber et compromettre la réplication et donc cette intégrité. La mitomycine C (MMC) est une molécule génotoxique utilisée en chimiothérapie. Elle forme des liaisons covalentes entre les deux brins d’ADN, ce qui est un obstacle à la bonne réplication de l’ADN. La rencontre de la fourche de réplication avec une liaison covalente entre les deux brins d’ADN va aboutir à une cassure double brin. Escherichia coli (E.coli) est un modèle d’étude très étendu car facile d’utilisation, permettant d’aborder des notions complexes. E coli possède divers mécanismes pour réparer ces lésions dont le régulon SOS. Le régulon SOS est un ensemble de gènes sous contrôle d’un promoteur réprimé par la protéine LexA. En réponse à des dommages à l’ADN, LexA est dégradé et les gènes du régulon sont activés.En utilisant une technique de biologie moléculaire qui permet de quantifier l’interaction entre deux chromatides sœurs restées cohésives derrière la fourche de réplication (étape appelée cohésion des chromatides sœurs), nous avons montré qu’en réponse à des cassures double brin générées par la MMC, la cohésion entre les chromatides sœurs nouvellement répliquées est maintenue. Ce phénomène est dépendant de RecN, une protéine induite de façon précoce dans le régulon SOS. RecN est une protéine de type SMC (structural maintenance of chromosomes), un groupe de protéines impliqué dans la dynamique et la structure du chromosome. En parallèle, des techniques de microscopie confocale et de marquage du chromosome par des protéines fluorescentes ont permis de montrer que la protéine RecN est impliquée dans une condensation globale du nucléoide suite à un traitement par la MMC. Cette condensation du nucléoide s’accompagne d’un rapprochement des chromatides sœurs ségrégées. Ces deux phénomènes, médiés par RecN pourraient permettre une stabilisation globale des nucléoides et favoriser l’appariement des chromatides sœurs pour permettre la recombinaison homologue.De façon intéressante, l’inhibition de Topoisomérases de type II (Topoisomerase IV et Gyrase) permettent de restaurer le phénotype d’un mutant recN en viabilité et en cohésion des chromatides sœurs. Les Topoisomérases sont des protéines qui prennent en charge les liens topologiques générés par la réplication et la transcription). Les liens topologiques non éliminés par les Topoisomerases permettraient de garder les chromatides sœurs cohésives et favoriser la réparation, même en l’absence de RecN.De plus, une expérience de RNA seq (séquençage de tout le transcriptome de la bactérie) a révélé que dans un mutant recN, le régulon SOS est moins induit que dans les cellules sauvages. Ceci va de pair avec une déstructuration des foci de réparation RecA. Il est possible que le rapprochement des chromatides sœurs médié par RecN permettrait de stabiliser le filament RecA et donc l’induction du SOS.L’ensemble de ces résultats suggère que RecN, une protéine de type SMC, permet de maintenir la cohésion entre les chromatides sœurs nouvellement répliquées, favorisant la réparation de cassures double brins par recombinaison homologue
Maintaining genome integrity through replication is an essential process for the cell cycle. However, many factors can compromise this replication and thus the genome integrity. Mitomycin C is a genotoxic agent that creates a covalent link between the two DNA strands. When the replication fork encounters the DNA crosslink, it breaks and creates a DNA double strand break (DSB). Escherichia coli (E.coli) is a widely used model for studying complex DNA mechanisms. When facing a DNA DSB, E. coli activates the SOS response pathway. The SOS response comprises over 50 genes that are under the control of a LexA-repressed promoter. Upon a DSB induction, RecA, a central protein of the SOS response will trigger the degradation of LexA and all the SOS genes will be expressed.We have developed a novel molecular biology tool that reveals contacts between sister chromatids that are cohesive. It has been shown in the lab (Lesterlin et al. 2012) that during a regular cell cycle, the two newly replicated sister chromatids stay in close contact for 10 to 20 min before segregating to separate cell halves thanks to the action of Topoisomerase IV. This step is called sister chromatid cohesion. We have used this molecular biology tool to study sister chromatid cohesion upon a genotoxic stress induced by mitomycin C (MMC). We have shown that sister chromatid cohesion is maintained and prolonged when the cell is facing a DSB. Moreover, this sister chromatid cohesion is dependent on RecN, an SOS induced structural maintenance of chromosome-like (SMC-like) protein. In the absence of RecN, the proximity between both sister chromatids is lost and this has a deleterious effect on cell viability. By tagging the chromosome with fluorescent proteins, we have revealed that RecN can also mediated a progressive regression of two previously segregated sister chromatids and this is coordinated with a whole nucleoid compaction. Further studies showed that this genome compaction is orderly and is not the result of a random compaction in response to DNA damage.Interestingly, inhibiting TopoIV in a recN mutant fully restores viability and sister chromatid cohesion suggesting that RecN’s action is mainly structural. Preserving cohesion through precatenanes is sufficient to favor repair and cell viability even in the absence of RecN.An RNA-seq experiment in a WT strain and a recN mutant revealed that the whole SOS response is downregulated in a recN mutant. This suggests that RecN may have an effect on the induction of the SOS response and thus RecA filament formation. This is in good agreement with the change in RecA-mcherry foci formation we observed. In the WT strain, the RecA-mcherry foci are defined as described in previous work. However, in the recN, the RecA-mcherry foci seemed to form bundle like structures. These RecA bundles were previsously described by Lesterlin et al. in the particular case of a DSB occurring on a chromatid that has already been segregated from its homolog. This could mean that in the absence of recN, the sister chromatids segregate and RecA forms bundle like structures in order to perform a search for the intact homologous sister chromatid.Altogether, these results reveal that RecN is an essential protein for sister chromatid cohesion upon a genotoxic stress. RecN favors sister chromatid cohesion by preventing their segregation. Through a whole nucleoid rearrangement, RecN mediates sister chromatid regression, favoring DNA repair and cell viability
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5

Dulermo, Rémi. "Etude des mécanismes de l'extrême tolérance aux radiations de la bactérie Deinococcus deserti par une approche de génomique fonctionnelle." Aix-Marseille 2, 2009. http://theses.univ-amu.fr.lama.univ-amu.fr/2009AIX22100.pdf.

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Le génome de Deinococcus deserti, une bactérie très radiotolérante, a été analysé et comparé à ceux de D. Radiodurans et D. Geothermalis. Environ 230 protéines sont spécifiquement conservées chez ces 3 espèces, dont IrrE, un régulateur essentiel pour la radiotolérance. D. Deserti possède plusieurs gènes supplémentaires liés à la réparation de l’ADN, dont imuY et dnaE2 (ADN polymérases translesionnelles). En plus, D. Deserti a 3 recA qui codent pour 2 protéines RecA différentes (RecAC et RecAP). Pour étudier ces gènes, des outils génétiques ont été mis au point. Différents résultats suggèrent qu’IrrE, nécessaire pour l’induction de plusieurs gènes après irradiation, a une activité peptidase. Les 2 RecA sont fonctionnelles pour la réparation de l’ADN. D. Deserti est mutable par UV, ce qui nécessite ImuY, DnaE2 et RecAC, mais pas RecAP
The genome of Deinococcus deserti, a highly radiation-tolerant bacterium, was analyzed and compared to those of D. Radiodurans and D. Geothermalis. About 230 proteins are specifically conserved in these 3 species, including IrrE, a regulator protein essential for radiotolerance. D. Deserti has several supplementary DNA repair genes, like imuY and dnaE2 (translesion DNA polymerases). Moreover, D. Deserti has 3 recA that code for 2 different RecA proteins (RecAC et RecAP). To study these genes, genetic tools were developed for D. Deserti. Different results suggest that IrrE, required for the induction of several genes after irradiation, has peptidase activity. The 2 RecA proteins are functional for DNA repair. D. Deserti is mutable by UV, which requires ImuY, DnaE2 and RecAC, but not RecAP
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6

Ames, Cory. "Reef Fish Assemblage Biogeography Along the Florida Reef Tract." NSUWorks, 2017. http://nsuworks.nova.edu/occ_stuetd/459.

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Understanding the biogeography of reef fish assemblages is paramount to reef conservation, management, and conducting appropriate population survey designs. Reef fish assemblages are a multispecies complex of reef-associated fish and are shaped by multiple environmental and biological factors (e.g. temperature, depth, benthic habitat, and topographic relief), which determine the species constituents residing in an area. Assemblages typically change with latitude where the number of families, genera, and/or densities of species specific to warmer climates decrease poleward into colder climate regimes. The Florida Reef Tract (FRT) extends for 595 km from the Dry Tortugas in the south-west to Martin County in the north, crossing a sub-tropical to temperate climate transition. This study investigates the biogeography of reef fish assemblages throughout the FRT to determine if they correspond to previous regional delineations that were primarily based on coastal geomorphology. Multivariate density analyses show that depth, habitat, relief, and region are major factors in determining the assemblages. Four main ecoregions were evident based on depth, benthic habitat, relief and latitudinal region: Dry Tortugas (DT), Florida Keys (FK), Southeast mainland (SE), and Bahamas Fracture Zone (BF). DT split into four biogeographic assemblage regions primarily based on depth, and relief. FK split into five biogeographic assemblage regions with a sixth extending through Broward County primarily based on depth, habitat type, and relief. SE split into four biogeographic assemblage regions primarily based on depth, and region. BF split into three biogeographic assemblage regions primarily based on depth, and region. These sixteen assemblages represent the current composition of reef fish based on four factors. Numerous other factors also affect reef fish assemblages (e.g. past and present fishing pressure, mangrove nursery habitat, and coral death) that were not part of the analysis but are discussed. The final reef fish assemblage regions were associated with previous benthic habitat maps in order to view their spatial extent. Having a map of current biogeographic reef fish assemblages serves as a baseline and allows more accurate management and monitoring of future reef fish populations.
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7

Rech, Alexander [Verfasser]. "Werkwohnungen / Alexander Rech." Frankfurt : Peter Lang GmbH, Internationaler Verlag der Wissenschaften, 2016. http://d-nb.info/1102805289/34.

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8

Geange, Shane Wallace. "An evaluation of prior residency and habitat effects on the persistence of settling reef fishes : a thesis submitted to the Victoria University of Wellington in fulfilment of the requirements for the degree of Doctor of Philosophy in Marine Biology /." ResearchArchive@Victoria e-Thesis, 2010. http://hdl.handle.net/10063/1169.

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9

Scales, Helen Joanna. "Exploitation of coral reef fishes for the Live Reef Fish Trade." Thesis, University of Cambridge, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.615075.

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Nothdurft, Luke David. "Microstructure and early diagenesis of recent reef building scleractinian corals, Heron reef, Great Barrier Reef : implications for paleoclimate analysis." Thesis, Queensland University of Technology, 2008. https://eprints.qut.edu.au/16690/1/Luke_D._Nothdurft_Thesis.pdf.

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Scleractinian corals increasingly are studied as geochemical archives of modern- and palaeoclimate, but microsampling for geochemical data is complicated by: 1) the microstructural complexity and spatial variability in skeletal growth in different coral genera; and 2) the rapidity and scale of diagenetic alteration that occurs in living coralla. Geochemical sampling techniques now have spatial resolution into the sub-micrometer to tens of micrometers range, and it is hoped that the spatial resolution can be translated to temporal resolution. This study investigated the effects on geochemical analyses imposed by microstructure and diagenesis in different live-collected coral genera representing somewhat different depositional environments. Suites of samples of four reef-building genera (Acropora, Pocillopora, Goniastrea and Porites) were collected from three adjacent environments in intertidal and subtidal positions near the reef edge at Heron Reef, Great Barrier Reef and studied by means of optical and scanning electron microscopy, combined with vibrational and energy dispersive spectroscopy. The first section of this study compares and documents the microstructure of the four coral genera. Each genus was found to have very different three-dimensional arrangements of microstructural elements, and a new general growth model was proposed for Acropora, to take into account differences in the timing of precipitation of trabeculae and thickening deposits. The results highlight the complexity and spatial variability of skeletal growth in different coral genera. Because microstructural patterns vary in different genera, direct observation of microstructural elements and growth lines are necessary to allow geochemical microsamples to be placed into series that represent temporal sequences with known degrees of time averaging. Coral growth rates (i.e., rates of extension) are discussed to determine the range of temporal relationships that exist between closely spaced skeletal microstructural elements. Such data are necessary in order for coral skeletogenesis to be understood and are critical for constraining microsampling strategies aimed at developing true time series geochemical data at very fine spatial and temporal scales. The second part of the study focused on early diagenetic alteration of the corals, which is an equally important concern for geochemical analysis. Early marine diagenesis was documented in the same live-collected samples of the four common reef-building coral genera. Samples show extensive early marine diagenesis where parts of the coralla less than three years old contain abundant macro- and microborings (sponges, algae, cyanobacteria and fungi) and significant amounts of aragonite, high-Mg calcite, low-Mg calcite and brucite [Mg(OH)2] cements. Many of the cements are associated with microendoliths and endobionts that inhabit recently abandoned parts of the skeleton. The cements are problematic for palaeoclimate reconstruction because geochemical proxies used for paleoclimate studies are meant to reflect ambient seawater chemistry and conditions, but the occurrence of brucite and low-Mg calcite demonstrates how far fluid chemistry in microenvironments within the corals has evolved from ambient seawater. Some Porites lobata specimens have had as much as 60% of the most recently deposited skeletal aragonite (i.e., the part of the skeleton that projects into the layer of living polyps) bored and replaced by low-Mg calcite cement. The low-Mg calcite cement has significantly different trace element ratios (Sr/Ca(mmol/mol) = 6.3 ± 1.4; Mg/Ca(mmol/mol) = 12.0 ± 5.1) than the host coral skeletal aragonite (Sr/Ca(mmol/mol) = 9.9 ± 1.3; Mg/Ca(mmol/mol) = 4.5 ± 2.3), thus providing a serious challenge for Sr/Ca or Mg/Ca based sea surface temperature calculations. This study illustrates that many diagenetic changes that can radically alter important geochemical characteristics of coral skeleton occur very early on the sea floor (i.e., while corals are still alive). Documented cements altered trace element inventories (e.g., Sr and Mg), thus, interfering with the use of those elements in palaeotemperature calculations. Hence, significant diagenetic changes that jeopardise palaeoclimate data do not require long-term diagenesis or meteoric exposure. Some of the diagenetic changes (e.g., calcite filled borings) occur at scales that are very difficult to detect short of visual inspection using SEM. Hence, vetting of coral samples with SEM is required before any sample is subjected to geochemical analysis.
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Книги з теми "RecF"

1

Gunesekera, Romesh. Reef. New York: New Press, 1995.

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2

Reef. London: Granta, 1995.

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(Firm), Scubazoo. Reef. New York: DK Pub., 2009.

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(Firm), Scubazoo, ed. Reef. [ New York: DK Publishing, 2007.

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5

Ivanovic, Radomir. Rec o reci: Poetika Ace Sopova. Beograd, 1986.

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6

Rec Ref Bks Small & Med. Not Avail, 1997.

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7

Dr. Bart A De Boer. Nos Ref di Koral / Ons Koraalrif / Our Coral Reef. Dieren Bescherming: Curacao, 2007.

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8

DeLoach, Ned, and Paul Humann. The Reef Set: Reef Fish, Reef Creature and Reef Coral (3 Volumes) (Reef Set). 2nd ed. New World Publications, 2002.

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9

Rech'. Rech'. Rech'. Detskaya ritorika dlya mladshih shkol'nikov. Astrel' - Rodnichok, 2000.

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10

Sheppard, Charles R. C., Simon K. Davy, Graham M. Pilling, and Nicholas A. J. Graham. Reef fisheries and reef aquaculture. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198787341.003.0007.

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Fisheries are of key importance in provision of protein, livelihood opportunities and income to islanders and coastal populations with few alternative food sources, including some of the world’s poorest people. The variety of reef fisheries for reef-associated invertebrates and vertebrates around the world is examined. Fishing methods used and particular issues with these fisheries are discussed. Exploitation of reef resources also occurs to supply luxury food markets and hobbies related to aquaria, and the international live reef fish trade is highlighted. The development of reef-based aquaculture is examined, and issues that need to be addressed to deliver sustainable expansion of this approach are discussed. In the face of increasing pressures on reef resources from a number of sources, resultant impacts on reef renewable resources and the reef ecosystem are detailed, and potential ways in which fisheries management may control these pressures are described.
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Частини книг з теми "RecF"

1

Spies, Maria, and Stephen C. Kowalczykowski. "Homologous Recombination by the RecBCD and RecF Pathways." In The Bacterial Chromosome, 389–403. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555817640.ch21.

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2

Andréfouët, Serge, and Guy Cabioch. "Barrier Reef (Ribbon Reef)." In Encyclopedia of Modern Coral Reefs, 102–7. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-90-481-2639-2_43.

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3

Yap, Helen T. "Coral Reef coral reef Ecosystems coral reef ecosystem." In Encyclopedia of Sustainability Science and Technology, 2489–509. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0851-3_728.

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4

Munro, John L. "The scope of tropical reef fisheries and their management." In Reef Fisheries, 1–14. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-015-8779-2_1.

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5

McManus, John W. "Social and economic aspects of reef fisheries and their management." In Reef Fisheries, 249–81. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-015-8779-2_10.

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6

Bohnsack, James A. "Maintenance and recovery of reef fishery productivity." In Reef Fisheries, 283–313. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-015-8779-2_11.

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7

Ruddle, Kenneth. "Traditional management of reef fishing." In Reef Fisheries, 315–35. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-015-8779-2_12.

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8

Adams, Timothy J. H. "Modern institutional framework for reef fisheries management." In Reef Fisheries, 337–60. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-015-8779-2_13.

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9

Polunin, Nicholas V. C., Callum M. Roberts, and Daniel Pauly. "Developments in tropical reef fisheries science and management." In Reef Fisheries, 361–77. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-015-8779-2_14.

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10

Sadovy, Yvonne J. "Reproduction of reef fishery species." In Reef Fisheries, 15–59. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-015-8779-2_2.

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Тези доповідей конференцій з теми "RecF"

1

Guzma´n, Amador M., Tania A. Aracena, Felipe A. Urzua, and Rodrigo A. Escobar. "Flow Bifurcations and Transition Scenarios in Confined Flows: Channel Geometry and Operational Parameter Dependency." In ASME 2006 International Mechanical Engineering Congress and Exposition. ASMEDC, 2006. http://dx.doi.org/10.1115/imece2006-14292.

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We have performed numerical simulations of confined flows in both symmetric and asymmetric wavy channels to investigate the flow bifurcations and transition scenarios dependency on geometrical and operational parameters. Laminar and time-dependent transitional flow regimes are obtained by direct numerical simulations of the mass and momentum equations using a computational program based on the spectral element method. Computational meshes for periodic computational domains are used to determine the transitional flow behavior for increasing Reynolds numbers and changing geometrical parameters. For the asymmetric wavy channel, the transition scenarios are highly dependent on the aspect ratio of the channel geometrical parameters. Depending on a specific geometric aspect ratio—one of the control parameters, the following scenarios develop: a) a first transition scenario with one flow bifurcation to a relatively low Reynolds numbers, Rec; b) a second scenario, with also one flow bifurcation, to a relatively high Reynolds numbers, Rec*; and, c) a third flow transition scenario with two Hopf bifurcations B1 and B2, occurring in critical Reynolds numbers Rec1 y Rec2, respectively, similar to the Ruelle-Takens-Newhouse scenario. In this third scenario, fundamental frequencies ω1 and ω2, and super harmonic combinations of both develop as the Reynolds number increases from laminar to transitional flow regimes. For the symmetric wavy channel and a high aspect ratio of r=0.375, a transition scenario with one flow bifurcation develops to a critical Reynolds numbers Rec** leading to a periodic flow. Further increases in the Reynolds number leads to successive periodic flows where the fundamental frequency ω1, increases continuously, in a scenario of frequency-doubling.
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2

Guzman, Amador M., Fernando A. Donoso, and Alfonso Ortega. "Transition Scenarios Due to Flow Bifurcations in Asymmetric Wavy Channel Flows With Different Spatial Periodicity on the Sinusoidal Walls." In ASME 2008 International Mechanical Engineering Congress and Exposition. ASMEDC, 2008. http://dx.doi.org/10.1115/imece2008-66216.

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Numerical investigations of transition scenarios due to flow bifurcations from laminar to time-dependent transitional flows in asymmetric wavy channels with different spatial periodicity on the sinusoidal wall are performed by direct numerical simulations of the mass and momentum conservation equations using a spectral element method computational program. Three aspect ratios r = a/(2L) are considered in this study: 0.125, 0.25, y 0.375. Computational meshes for both extended and periodic computational domains are used to determine first, the existence of spatial periodicity and second, the transitional flow behavior for increasing Reynolds numbers. Numerical results shows that the transition scenarios are highly dependent on the aspect ratio, r. The following scenarios develop: a) a first transition scenario with one flow bifurcation to a relatively low Reynolds numbers, Rec; b) a second scenario, with also one flow bifurcation, to a relatively high Reynolds numbers, Rec*; and, c) a third flow transition scenario with two Hopf bifurcations B1 and B2, occurring at critical Reynolds numbers Rec1 y Rec2, respectively. In this third scenario, fundamental frequencies ω1 and ω2, and sub and super harmonic combinations of the fundamental frequencies develop as the Reynolds number increases from a laminar to higher transitional flow regime. This transition scenario from a laminar flow to high transitional flow regimes is very similar to the Ruelle-Takens-Newhouse (RTN) found in another confined flow channels such as symmetric wavy, grooved and communicating channels.
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3

"Gesamtband." In Ref-NEKP. Wien: Verlag der Österreichischen Akademie der Wissenschaften, 2020. http://dx.doi.org/10.1553/0x003b72a1.

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4

"Referenzplan als Grundlage für einen wissenschaftlich fundierten und mit den Pariser Klimazielen in Einklang stehenden Nationalen Energie- und Klimaplan für Österreich (Ref-NEKP) - Executive Summary." In Ref-NEKP. Vienna: Austrian Academy of Sciences Press, 2020. http://dx.doi.org/10.1553/ref-nekp-summarys1.

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5

"Referenzplan als Grundlage für einen wissenschaftlich fundierten und mit den Pariser Klimazielen in Einklang stehenden Nationalen Energie- und Klimaplan für Österreich (Ref-NEKP) Vision 2050 und Umsetzungspfade: Österreich im Einklang mit den Pariser Klimazielen und der Weg dorthin." In Ref-NEKP. Wien: Verlag der Österreichischen Akademie der Wissenschaften, 2020. http://dx.doi.org/10.1553/ref-nekp-vision2050s1.

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6

Huang, Chuan-Che (Jeff), Sheng-Yuan Liang, Bing-Hsun Wu, and Mark W. Newman. "Reef." In DIS '17: Designing Interactive Systems Conference 2017. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3064663.3064685.

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7

Weimer, Markus, Yingda Chen, Byung-Gon Chun, Tyson Condie, Carlo Curino, Chris Douglas, Yunseong Lee, et al. "REEF." In SIGMOD/PODS'15: International Conference on Management of Data. New York, NY, USA: ACM, 2015. http://dx.doi.org/10.1145/2723372.2742793.

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8

Datta, Anwitaman, Jackson Tan Teck Yong, and Anthony Ventresque. "T-RecS." In the 20th international conference companion. New York, New York, USA: ACM Press, 2011. http://dx.doi.org/10.1145/1963192.1963289.

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9

Achara, Jagdish Prasad, Maaz Mohiuddin, Wajeb Saab, Roman Rudnik, Jean-Yves Le Boudec, and Lorenzo Reyes-Chamorro. "T-RECS." In e-Energy '18: The Ninth International Conference on Future Energy Systems. New York, NY, USA: ACM, 2018. http://dx.doi.org/10.1145/3208903.3208928.

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10

Wu, Chia-Han, Wei-Jen Chen, and Jyh-Cheng Chen. "Poster: RECO." In MobiCom '17: The 23rd Annual International Conference on Mobile Computing and Networking. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3117811.3131257.

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Звіти організацій з теми "RecF"

1

Katherine Comer Santos, Katherine Comer Santos. Coral reef health monitoring. Experiment, February 2023. http://dx.doi.org/10.18258/48280.

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2

Altman, Safra, R. Harris, S. McKay, Michael Kjelland, and Todd Swannack. Oyster reef connectivity : ecological benefits and associated vulnerabilities. Engineer Research and Development Center (U.S.), August 2022. http://dx.doi.org/10.21079/11681/45020.

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Global oyster abundance has declined ~85 % over the past 200 years, primarily because of overharvesting (Beck, Brumbaugh, and Airoldi 2011; Kirby 2004). Healthy oyster reef systems benefit the environment in many ways, including water-quality improvement, shoreline protection, increased biological and habitat diversity, and carbon sequestration. To maintain these environmental benefits, reef-restoration efforts that produce healthy, sustainable oyster reefs are essential. To this end, the US Army Corps of Engineers (USACE) has been involved in reef-restoration projects in many locations, including extensive efforts in the Chesapeake Bay (Virginia, Maryland), coastal regions of New York and New Jersey, and the Gulf of Mexico. There are many benefits to creating and maintaining oyster reef systems that are well connected, for both oysters and other organisms within the reef and surrounding habitats. This technical note presents the current knowledge of benefits and costs to restore oyster-reef connectivity along the East and Gulf Coasts of North America. Connectivity of oyster reefs can refer to the physical location of reefs with respect to one another as well as to the dynamics of the genetic links within a metapopulation or to the extent to which larval transport and recruitment unite reef communities. For the purposes of this technical note, connectivity is defined as the spatial aggregation of reefs, though we address impacts of genetic and larval flow as well. Reef connectivity positively affects many ecosystem services and dynamics but can also have unintended consequences (that is, negative externalities). This technical note reviews the benefits and costs of increasing connectivity and presents a brief example of how trade-offs may occur between these potentially opposing ecological objectives. Here, we focus on the eastern oyster, Crassostrea virginica, which inhabits the East and Gulf Coasts of North America, though many of the concepts and principles discussed may apply to other oyster species as well.
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3

Lobel, Lisa K., and Phillip Lobel. Coral Reef Protection Implementation Plan. Fort Belvoir, VA: Defense Technical Information Center, October 2000. http://dx.doi.org/10.21236/ada410910.

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4

Luckie, M. Really Explicit Congestion Notification (RECN). RFC Editor, April 2015. http://dx.doi.org/10.17487/rfc7514.

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5

Chapman and Keshavarz-Valian. L51988 Development of Turbocharger-Reciprocating Engine Simulation (T-RECS). Chantilly, Virginia: Pipeline Research Council International, Inc. (PRCI), December 2002. http://dx.doi.org/10.55274/r0010947.

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The objective of this project was to develop a numerical simulation system that can be used to optimize the performance of a large-bore reciprocating engine. The simulation system will include sub-models of all major components between the inlet filter and the exhaust pipe of the engine. The deliverable product of this project is the software program, Turbocharger-Reciprocating Engine Computer Simulation (T-RECS), which was developed at the National Gas Machinery Laboratory at Kansas State University. The simulation program calls upon a database of system components to allow the user specify a specific system. The database in this edition of T-RECS contains nominal components, but will be expanded under the Populate T-RECS project that has been funded by the Gas Research Institute. This report contains 1) a discussion of the methodology utilized to develop T-RECS; 2) a user�s guide; and 3) an example problem.
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6

Mücher, Sander, Juha Suomalainen, John Stuiver, and Erik Meesters. Hyperspectral Coral Reef Classification of Bonaire. Den Helder: Wageningen Marine Research, 2017. http://dx.doi.org/10.18174/422722.

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7

Haver, Samara. Analysis of underwater soundscape conditions at Buck Island Reef National Monument during the COVID-19 pandemic: Focused condition assessment report. National Park Service, October 2022. http://dx.doi.org/10.36967/2294883.

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In partnership with the National Oceanic and Atmospheric Administration and Oregon State University, the National Park Service has been collecting continuous acoustic recordings at a stationary autonomous recorder in Buck Island Reef National Monument since 2016. The audio data were previously analyzed to establish baseline soundscape conditions as well as monitor the acoustic presence of vessels and humpback whales. This report specifically investigates potential changes to the soundscape environment during the onset of the COVID-19 pandemic and the consequent “anthro-pause” when human activities such as tourism and commercial shipping were interrupted by public health guidance. Although major declines of anthropogenic activities were observed in other regions of the world, soundscape conditions in Buck Island Reef National Monument were only minimally impacted during early 2020. Furthermore, in latter months of 2020 and into 2021, vessel movement and related noise levels slightly increased from historic levels. Humpback whale vocalizations were also analyzed for seasonal presence in Buck Island Reef National Monument, revealing a consistent pattern with previously analyzed seasons. Ongoing passive acoustic soundscape monitoring will provide data that can be used to evaluate continued impacts of anthropogenic activity in and near Buck Island Reef National Monument.
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8

Stone-Jovicich, Samantha, Tracy Shultz, Kirsten Maclean, Elizabeth Hobman, and Angela Dean. Stewardship for the Great Barrier Reef: Exploring perspectives and suggestions for monitoring stewardship for the Great Barrier Reef. Brisbane, QLD, Australia: The University of Queensland, May 2023. http://dx.doi.org/10.14264/f905c86.

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9

Meredith Wingate. REC Tracking Systems Design Guide. Office of Scientific and Technical Information (OSTI), February 2004. http://dx.doi.org/10.2172/822444.

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10

Johnston, Robert K., Robert D. George, Kenneth E. Richter, P. F. Wang, and William J. Wild. Ex-ORISKANY Artificial Reef Project: Ecological Risk Assessment. Fort Belvoir, VA: Defense Technical Information Center, January 2006. http://dx.doi.org/10.21236/ada484457.

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