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Статті в журналах з теми "Pancreatiti"
Zakharova, M. A., D. S. Gorin, L. A. Marinova, and A. G. Kriger. "X-ray-negative pancreatic dust stones in patients with chronic pancreatiti." Khirurgiya. Zhurnal im. N.I. Pirogova, no. 12 (2019): 137. http://dx.doi.org/10.17116/hirurgia2019121137.
Повний текст джерелаRisse, O., C. Arvieux, J. Abba, and C. Létoublon. "Chirurgia delle complicanze delle pancreatiti acute." EMC - Tecniche Chirurgiche Addominale 19, no. 1 (March 2013): 1–14. http://dx.doi.org/10.1016/s1283-0798(13)63954-6.
Повний текст джерелаHadjiangelis, Nicos P., and Doreen J. Addrizzo-Harris. "Cryptic Miliary Tuberculosis With a Prodrome Resembling Pancreatiti." Chest 124, no. 4 (January 2003): 330S. http://dx.doi.org/10.1378/chest.124.4_meetingabstracts.330s.
Повний текст джерелаShaikh, Danial H., Ahmed Alemam, Jennifer von Ende, Haider Ghazanfar, Anil Dev, and Bhavna Balar. "Ansa Pancreatica, an Uncommon Cause of Acute, Recurrent Pancreatitis." Case Reports in Gastroenterology 15, no. 2 (July 1, 2021): 587–93. http://dx.doi.org/10.1159/000516686.
Повний текст джерелаBhattacharyya, B. K., S. Chowdhury, S. Das, S. Mukherjee, and D. Bhattacharjee. "Treatment of Pancreatic Exocrine Insufficiency with Enteric Coated Pancreatin Formulations: An Overview." International Journal of Pharmaceutical Sciences and Nanotechnology 6, no. 3 (November 30, 2013): 2125–30. http://dx.doi.org/10.37285/ijpsn.2013.6.3.3.
Повний текст джерелаAdibelli, Zehra Hilal, Mustafa Adatepe, Cetin Imamoglu, Ozgur Sipahi Esen, Nazif Erkan, and Mehmet Yildirim. "Anatomic variations of the pancreatic duct and their relevance with the Cambridge classification system: MRCP findings of 1158 consecutive patients." Radiology and Oncology 50, no. 4 (December 1, 2016): 370–77. http://dx.doi.org/10.1515/raon-2016-0041.
Повний текст джерелаBragado, M. J., J. I. San Roman, A. González, L. J. García, M. A. López, and J. J. Calvo. "Impairment of Intracellular Calcium Homoeostasis in the Exocrine Pancreas after Caerulein-Induced Acute Pancreatitis in the Rat." Clinical Science 91, no. 3 (September 1, 1996): 365–69. http://dx.doi.org/10.1042/cs0910365.
Повний текст джерелаSommer, Camille Anne, and C. Mel Wilcox. "Pancreatico-pericardial fistula as a complication of chronic pancreatitis." F1000Research 3 (January 29, 2014): 31. http://dx.doi.org/10.12688/f1000research.3-31.v1.
Повний текст джерелаMihai, Catalina, Mariana Floria, Radu Vulpoi, Loredana Nichita, Cristina Cijevschi Prelipcean, Vasile Drug, and Viorel Scripcariu. "Pancreatico-Pleural Fistula – from Diagnosis to Management. A Case Report." Journal of Gastrointestinal and Liver Diseases 27, no. 4 (December 31, 2018): 465–69. http://dx.doi.org/10.15403/jgld.2014.1121.274.ple.
Повний текст джерелаSajika Dighe, Raju Shinde, Sangita Shinde, and Mohit Gupte. "A rare case of pancreaticopleural fistula patient presented in surgery OPD." International Journal of Research in Pharmaceutical Sciences 11, SPL4 (December 21, 2020): 1329–32. http://dx.doi.org/10.26452/ijrps.v11ispl4.4301.
Повний текст джерелаДисертації з теми "Pancreatiti"
Fernandes, Cátia Conceição da Encarnação. "Clínica médica e cirúrgica em animais de companhia: pancreatite em animais de companhia." Master's thesis, Universidade de Évora, 2015. http://hdl.handle.net/10174/18224.
Повний текст джерелаFitzsimmons, Deborah. "Quality of life in pancreatic cancer and chronic pancreatitis." Thesis, University of Southampton, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326844.
Повний текст джерелаMalluta, Éverson Fernando. "Avaliação do pâncreas através da ecoendoscopia em pacientes portadores de Doença de Crohn." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/5/5147/tde-28012009-151207/.
Повний текст джерелаBACKGROUND: Pancreas injury usually is not mentioned as an extra-intestinal manifestation of Crohns disease, but the few available series suggest pancreatic injury in a significant proportion of these patients, ranging from 1.2% to 58% in this series. Endoscopic ultrasound (EUS) presents the advantage of having a much higher sensitivity than the abdominal ultrasound or computadorized tomography in evaluating pancreas abnormalities. Compared to the endoscopic retrograde cholangiopancreatography, EUS has at least an equal sensitivity, with a lower complication rate. AIMS: To evaluate the incidence of pancreatic alterations by means of the EUS in Crohns disease, correlating them with clinical, endoscopic and biochemical data. METHODS: Fifty one patients with Crohns disease, age between 18 and 60 years-old (mean = 38), without previous history of pancreatic disease, diabetes mellitus or alcoholism, were submitted to EUS. The control-group was formed by 20 patients submitted to EUS with no previous history of pancreatic or Crohns disease. Clinical, endoscopic and biochemical data were collected in order to determine possible predictive factors. Eleven variables were analyzed, both in pancreatic parenchyma and ducts. The patients with 3 or more alterations were submitted to magnetic resonance. Pancreatic function was determined using fecal elastase assay in 39 patients. RESULTS: Of the 51 analyzed patients, 56% were female, with mean diagnosis time of seven years (1-25) and Crohns disease Activity Index (CDAI) of 102 (20-419). Two patients (3.9%) presented 4 alterations in the EUS exam, 3 (5.9%) presented 3 alterations, 11 (21.5%) had 2 alterations and 13 (25.5%) had 1 alteration in the EUS, which were statistically significant when compared to the control-group, in whom only 16% presented 1 exam alteration (p<0.001). The parenchymal abnormalities were more common, totalizing 39 findings when compared to 11 ductal abnormalities. The patients with 3 or more alterations in the exam were submitted to magnetic resonance; however, pancreatic lesions were not detected. Four patients (10%) had low fecal elastase measurement, suggestive of exocrine pancreatic insufficiency. None of these patients had significant pancreatic alterations in EUS. The only predictive factor that correlated with the number of alterations in EUS was isolated ileal disease. CONCLUSIONS: Patients with Crohns disease had a higher incidence of pancreatic abnormalities (10% with 3 or more alterations in endoscopic ultrasound) in comparison to the control-group. These alterations were found most frequently in the pancreatic parenchyma, which might explain the lack of correlation with the magnetic resonance, which is more prone to detect duct abnormalities. The only predictive factor to these alterations on EUS was ileal disease
Sampaio, Cristina Cardoso. "Pancreatite alcoólica." Master's thesis, [s.n.], 2014. http://hdl.handle.net/10284/4842.
Повний текст джерелаA pancreatite alcoólica é uma das principais complicações do consumo excessivo de álcool. O risco de desenvolver a doença aumenta com a ingestão de doses crescentes de álcool, sugerindo que os efeitos tóxicos estão relacionados com a quantidade de álcool ingerida. No entanto, apenas uma minoria dos alcoólicos desenvolve pancreatite, o que indica a necessidade de um fator desencadeador adicional para iniciar a lesão pancreática clinicamente evidente. A pancreatite alcoólica começa como um processo agudo necroinflamatório/ autodigestivo, progredindo com repetidos episódios de necroinflamação. O seu espectro clínico inclui pancreatite aguda (dor abdominal aguda e elevados níveis séricos das enzimas pancreáticas) e pancreatite crónica (dor abdominal, má digestão e diabetes). Têm sido feitos avanços significativos que fornecem uma visão sobre os mecanismos moleculares da lesão pancreática provocada pelo álcool, principalmente no que diz respeito aos seus efeitos tóxicos sobre as células acinares pancreáticas e recentemente, nas células estreladas pancreáticas (PSCs), que desempenham um papel fundamental na fibrose, caraterística da pancreatite crónica alcoólica. Alcoholic pancreatitis is a major complication of alcohol abuse. The risk of developing pancreatitis increases with increasing doses of alcohol, suggesting that alcohol exerts dose-related toxic effects on the pancreas. However, it is also clear that only a minority of alcoholics develop the disease, indicating that an additional trigger may be required to initiate clinically evident pancreatic injury. Alcoholic pancreatitis is thought to begin as an acute necroinflammatory/autodigestive process in a susceptible individual and to progress with repeated episodes. The clinical spectrum of the disease includes acute pancreatitis (acute abdominal pain and raised sérum levels of pancreatic enzymes) and chronic pancreatitis (abdominal pain, maldigestion, diabetes). Significant advances have been made in recent years that provide an insight into the molecular mechanisms of alcohol-related pancreatic injury, particularly with respect to the direct toxic effects of alcohol on pancreatic acinar cells and on the recently characterized PSCs, with may play a key role in the fibrosis of alcoholic chronic pancreatitis.
Шевченко, Володимир Порфирович, Владимир Порфирьевич Шевченко, Volodymyr Porfyrovych Shevchenko, V. O. Bratushka, Y. I. Sobolev, I. A. Myslovsky, O. V. Kravez, et al. "The results of surgical treatment of pancreatic pseudocysts following acute pancreatitis." Thesis, Видавництво СумДУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/27527.
Повний текст джерелаIkuta, Kozo. "Nardilysin inhibits pancreatitis and suppresses pancreatic ductal adenocarcinoma initiation in mice." Kyoto University, 2019. http://hdl.handle.net/2433/242408.
Повний текст джерелаWilson, Benjamin Gregg. "Feline pancreatic lipase: purification and validation of a clinically significant radioimmunoassay for the diagnosis of feline pancreatitis." Texas A&M University, 2003. http://hdl.handle.net/1969.1/1581.
Повний текст джерелаAbou, Khalil Jad. "Pancreatic fistulas after pancreatico-duodenectomies: are pancreatico-gastrostomies safer than pancreatico-jejunostomies? a quasi-experiment and propensity-score adjusted analysis." Thesis, McGill University, 2014. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=122998.
Повний текст джерелаCONTEXTE les fistules pacreatiques (PF) constituent une cause significative de la morbiditée et mortalité subie par les patients qui recoivent une pancreaticoduodenectomie (PD). La technique ideale pour retablir la continuité pancreatico-enterique est inconnue. Il n'est pas donné que les Pancreatico-Gastrostomies (PG) donne moi de PF et de complications post-operatives que les Pancreatico-Jejunostomies(PJ). BUT: Nous avons comparé le profile de complications post-operatoire chez les patients ayant subi une PG or PJ apres PD au Centre Universitaire de Sante McGill. METHODOLOGIE: Nous avons collecté des données pre-operatoires ainsi que les complications post- operatoires pour les patients ayant subi une PD dans notre base de données entre 1999 et 2011 et ayant subi une reconstruction par PG ou PJ. Nous avons performé une regression logistique ajustée pour un" propensity-score" pour identifier l'effet de la technique chirurgicale sur les PF, les delais de la motilitée gastrique (DGE), et les complications totales. nous avons utilisé les classifications ISGPF et Strasberg et Linehan pour PF et la definition ISGPS pour DGE. La morbidité totale a été evaluée par la classification Clavien-Dindo et l'Index Comprehensif de Morbidité (CCI). RESULTATS 23/103 et 20/103 (p=0.49) des patients ont developpé une PF et 74/103 et 55/103 patients ont eu DGE en periode post operatoire dans les groupes PG et PJ respectivement (p=0.02). Le grade Clavien-Dindo des complications n'etait pas different entre les groupes (p=0.29) mais le CCI l'etait (38.4 vs. 31.4 for PG vs. PG respectivement, p=0.02.) l'analyse multivariable ajustée pour le "Propensity-score" n'a pas montré d'effet de la technique chirurgical sur PF (p=0.89), DGE grades B/C (p=0.9) ou CCI (p=0.41) mais il restait un effet sur le DGE de toutes les grades de severité (p=0.012.) CONCLUSION Les patients ayant recu une PG n'avaient pas moins de PF que ceux ayant recu une PJ aprés PD a notre institution; Les deux groupes ont souffert du meme profile de complications, mais le groupe PG avait plus de DGE de toutes grades.
Camargo, Enilton Aparecido. "Caracterização da pancreatite aguda induzida por fosfolipases 'A IND. 2' secretorias." [s.n.], 2007. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308940.
Повний текст джерелаTese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas
Made available in DSpace on 2018-08-10T04:13:39Z (GMT). No. of bitstreams: 1 Camargo_EniltonAparecido_D.pdf: 1739135 bytes, checksum: faa68531f796834af8491c48ada0ccc0 (MD5) Previous issue date: 2007
Resumo: A pancreatite aguda e uma doença inflamatória do pâncreas caracterizada por intensa necrose pancreática e efeitos sistêmicos secundários como lesão pulmonar, os quais são a principal causa da mortalidade observada nessa doença. Ha evidencia de que as fosfolipases A2 (PLA2) tem um importante papel na fisiopatologia da pancreatite aguda. O objetivo deste trabalho foi investigar a capacidade de PLA2s de induzir pancreatite em ratos e os mecanismos envolvidos nesse fenômeno. As seguintes PLA2s foram utilizadas: piratoxina-I (homologo Lys-49 de PLA2 desprovido de atividade catalitica), bothropstoxina-II (homologo Asp-49 de PLA2 com baixa atividade catalítica) e a PLA2 proveniente do veneno de Naja moçambique moçambique (que possui alta atividade catalítica). Ratos Wistar machos (200-250 g) provenientes do CEMIB/UNICAMP foram utilizados. As PLA2s foram injetadas no ducto biliopancreatico de animais anestesiados e apos diferentes tempos experimentais foram avaliados o extravasamento de proteínas plasmáticas no pâncreas, infiltrado de neutrofilos no pâncreas e pulmão e amilase serica. A analise histológica do pâncreas e pulmão também foi realizada em alguns grupos experimentais. Piratoxina-I foi capaz de causar extravasamento de proteínas plasmáticas no pâncreas, infiltrado de neutrofilos no pulmão e os níveis sericos de amilase. Alem disso, a piratoxina-I causou alterações histológicas nos tecidos pancreático (infiltrado de neutrofilos, necrose de células acinares e edema intersticial) e pulmonar (edema intersticial e diminuição do espaço alveolar), que foram mais evidentes nos tempos iniciais da pancreatite (4-12h). Bothropstoxina-II e a PLA2 do veneno de Naja moçambique moçambique, a semelhança da Piratoxina-I, também foram capazes de aumentar o extravasamento de proteínas plasmáticas e o influxo de neutrofilos no tecido pancreático por mecanismos nao relacionados a sua atividade catalítica. Entretanto, o influxo de neutrofilos para o pulmão e o aumento dos níveis sericos de amilase causados por essas PLA2s foi dependente de sua atividade catalítica. As PLA2s também causaram secreção deamilase de acinos pancreáticos isolados, que foi dependente da atividade catalítica dessas enzimas. Adicionalmente, com o objetivo de entender o mecanismo envolvido na pancreatite induzida pela PLA2 de Naja mocambique mocambique animais foram tratados com os seguintes agentes farmacológicos: pentoxifilina (inibidor da sintese de TNF-á), SR140333 (antagonista de receptor NK1), icatibant (antagonista de receptor B2), L-NAME (inibidor não seletivo das NOS), aminoguanidina (inibidor preferencial da NOS induzivel), indometacina (inibidor não seletivo de COX), celecoxib (inibidor seletivo de COX-2), PCA4248 (antagonista dos receptores de PAF) e AA861 (inibidor da 5-lipoxigenase). Em conjunto, nossos dados mostraram que os efeitos locais e secundários são multimediados, envolvendo a participação de bradicinina, substancia P, NO, TNF-á, PAF e metabolitos das COXs. Em conclusão, demonstramos que as PLA2s secretorias são capazes de induzir pancreatite aguda em ratos quando injetadas no ducto biliopancreatico, um quadro caracterizado por efeitos inflamatórios locais e secundários cuja mediação farmacológica envolve vários fatores. Alem disso, a pancreatite aguda induzida pelas PLA2s reproduz algumas alterações observadas na pancreatite aguda em humanos, representando uma nova estratégia de estudo da fisiopatologia da pancreatite aguda
Abstract: Acute pancreatitis is an inflammatory disease of the pancreas that is characterized by intense pancreatic necrosis and remote systemic effects such as the lung injury that is the main cause of death during acute pancreatitis. There are evidences that phospholipases A2 (PLA2) have an important role in the acute pancreatitis pathophysiology. The aim of this work was to investigate the ability of PLA2 to induce acute pancreatitis in rats, and the mechanisms underlying this phenomenon. The following PLA2s were used: piratoxin-I (a Lys-49 PLA2 homologue devoid of catalytic activity), bothropstoxin-II (an Asp-49 PLA2 homologue with low catalytic activity) and PLA2 from Naja mocambique mocambique venom (high catalytic activity). Male Wistar rats (200-250g) provided by CEMIBUNICAMP have been used. The PLA2s were injected into the common bile duct of anesthetized rats, after which pancreatic plasma protein extravasation, pancreatic and lung neutrophil infiltration and serum levels of amylase were measured. Histology of the pancreatic and lung tissue has also been carried out in some experimental group. Piratoxin-I was able to increase the pancreatic plasma protein extravasation, lung neutrophil infiltration and serum amylase levels. In addition, Piratoxin-I caused histological changes in the pancreatic (neutrophil infiltration, areas of acinar cell necrosis and interstitial edema) and lung (interstitial edema and diminuition of alveolar space) tissues, which were more evident in the early periods (4-12h) after the injection. Bothropstoxin-II and PLA2 from Naja mocambique mocambique venom were also able to increase the plasma protein extravasation and neutrophil influx in the pancreatic tissue by mechanisms unrelated to their catalytic activity. However, the remote lung neutrophil influx caused by these PLA2s was shown to depend on their catalytic activity. The enhancement of serum amylase levels was also dependent on the catalytic activity of these enzymes. The PLA2s also caused amylase secretion from isolated pancreatic acini, which was dependent on their catalytic activity. Next, in order to further understand the mechanisms involved in pancreatitis induced by PLA2 Naja mocambique mocambique, animals were treated with the following pharmacological agents: pentoxifylline (TNF-á synthesis inhibitior), SR140333 (NK1receptor antagonist), icatibant (B2 receptor antagonist), L-NAME (non-selective NOS inhibitor), aminoguanidine (preferential inducible NOS inhibitor), indomethacin (nonselective COX inhibitor), celecoxib (selective COX-2 inhibitor), PCA4248 (PAF receptor antagonist) and AA861 (5-lipoxygenase inhibitor). Taken together our data showed that local and remote effects are multimediated, involving the participation of bradykinin, substance P, NO, TNF-á, PAF and COXs metabolites. In conclusion, we have shown that secretory PLA2s are able to induce acute pancreatitis in rats when injected into the common bile duct. Therefore, PLA2-induce acute pancreatitis reproduces some aspects of the human disorder representing a new strategy to study the pathophysiology of acute pancreatitis
Doutorado
Farmacologia
Doutor em Farmacologia
Karjula, H. (Heikki). "Diagnosis, treatment and prophylaxis of pancreatic fistulas in severe necrotizing pancreatitis and the long-term outcome of acute pancreatitis." Doctoral thesis, Oulun yliopisto, 2019. http://urn.fi/urn:isbn:9789526224312.
Повний текст джерелаTiivistelmä Akuutti nekrotisoiva haimatulehdus ja erityisesti siihen liittyvä bakteeri-infektio on sairaus, johon liittyy korkea komplikaatio- ja kuolleisuusriski. Tautia usein komplisoi infektion lisäksi nekroosiin liittyvä haimafisteli, joka tekee hoidosta entistä haasteellisemman. Viime aikaisissa tutkimuksissa on käsitelty runsaasti mini-invasiivista nekrosektomiaa, mutta suhteellisen vähän on tutkimuksia nekrotisoivaan haimatulehdukseen liittyvästä fisteliongelmasta. Haimatulehdus-potilaiden pitkäaikaisennuste on myös epäselvä. Tämän väitöskirjatutkimuksen tavoitteena oli selvittää nekrotisoivaan haimatulehdukseen liittyvän haimafistelin yleisyyttä, diagnostiikkaa, ehkäisyä ja hoitoa. Lisäksi tarkasteltiin akuuttiin haimatulehdukseen sairastuneiden potilaiden pitkäaikaisennustetta. Ensimmäisessä osatyössä ilmeni, että kaikille potilaille, joille suoritettiin haiman nekrosektomia kehittyi fisteli ja endoskooppinen transpapillaarinen haimateiden stenttaus (ETPS) osoittautui hyväksi ja turvalliseksi hoidoksi fistelin hoidossa. Toisessa prospektiivisessa randomoidussa kontrolloidussa osatyössä tutkittiin profylaktista haimateiden stenttausta nekrotisoivassa haimatulehduksessa. Tutkimus osoitti, etteivät potilaat hyötyneet stenttauksesta: toimenpiteestä oli enemmän haittaa kuin hyötyä. Tämän tutkimuksen mukaan protetisointia ei suositella tehtäväksi taudin alkuvaiheessa. Kolmannessa osatyössä selvitettiin haiman nekrosektomian jälkeisen haimafistelin diagnosointia. Tutkimustuloksen mukaan haimafistelin osoittamiseksi dreenieritteen amylaasitasoa mittaamalla tarvitaan useita mittauskertoja, koska yksittäisen mittauksen sensitiivisyys on matala. Neljännessä osatyössä analysoitiin Oulun yliopistollisessa sairaalassa 1995–2012 akuutin haimatulehduksen sairastaneiden työikäisten potilaiden pitkäaikaisennustetta ja kuolinsyitä. Noin kymmenen vuoden seurannassa tutkimusryhmän (n = 1 644) kuolleisuus oli yli nelinkertainen verrattuna ikä- ja sukupuolivakioituihin verrokeihin (n = 8 220). Merkittävin kuolleisuutta lisäävä tekijä oli alkoholi. Tutkimuksemme osoitti, että infektoituneen haimanekroosiin liittyvä haimafisteli on huomioitava hoidossa. Varhaisesta profylaktisesta haimateiden protetisoinnista ei tutkimuksessa osoitettu olevan hyötyä. Alkoholin aiheuttaman haimatulehduksen pitkäaikaisennusteen mortaliteetti on korkea johtuen alkoholin käytöstä ja siihen liittyvistä sairauksista
Книги з теми "Pancreatiti"
V, Balakrishnan, Kumar Harish, S. Sudhindran, and A. G. Unnikrishnan. Chronic pancreatitis and pancreatic diabetes in India. Edited by Indian Pancreatitis Study Group. Cochin, India]: Indian Pancreatitis Study Group, 2006.
Знайти повний текст джерелаG, Beger H., Büchler Markus 1955-, and Malfertheiner P. 1950-, eds. Standards in pancreatic surgery. Berlin: Springer-Verlag, 1993.
Знайти повний текст джерелаKanojia, Ravi P. Laparoscopic lateral pancreatico-jejunostomy for chronic pancreatitis in children. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-7849-4.
Повний текст джерелаC, Langley William, ed. Pancreatitis research advances. New York: Nova Biomedical Books, 2007.
Знайти повний текст джерела1952-, Johnson C. D., and Imrie C. W, eds. Pancreatic disease: Towards the year 2000. 2nd ed. London: Springer, 1999.
Знайти повний текст джерела1932-, Burns Gerard P., and Bank S, eds. Disorders of the pancreas: Current issues in diagnosis and management. New York: McGraw-Hill, Health Professions Division, 1992.
Знайти повний текст джерелаLankisch, P. G., and Peter A. Banks. Pancreatitis. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-80320-8.
Повний текст джерелаAdams, David B., Peter B. Cotton, Nicholas J. Zyromski, and John Windsor, eds. Pancreatitis. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781118924907.
Повний текст джерелаA, Banks Peter, ed. Pancreatitis. Berlin: Springer, 1998.
Знайти повний текст джерелаC, Carter David, and Warshaw Andrew L. 1939-, eds. Pancreatitis. Edinburgh: Churchill Livingstone, 1989.
Знайти повний текст джерелаЧастини книг з теми "Pancreatiti"
Tuan, Le Quan Anh, and Pham Minh Hai. "Laparoscopic Internal Drainage of Pancreatic Pseudocysts." In Mastering Endo-Laparoscopic and Thoracoscopic Surgery, 345–48. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-3755-2_50.
Повний текст джерелаForsmark, Chris E. "Pancreatitis-Related Pancreatic Masses: Chronic Pancreatitis." In Pancreatic Masses, 75–84. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-19677-0_6.
Повний текст джерелаAl-Salem, Ahmed H. "Pancreatitis and Pancreatic Pseudocyst." In Atlas of Pediatric Surgery, 221–27. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-29211-9_25.
Повний текст джерелаArshad, Ali, and Ashley Dennison. "Acute Pancreatitis, Chronic Pancreatitis and Pancreatic Neoplasms." In Gastroenterology For General Surgeons, 103–17. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-92768-8_10.
Повний текст джерелаCarter, D. C. "Pancreatico-duodenectomy for Chronic Pancreatitis." In Aktuelle Pankreaschirurgie, 149–57. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75265-0_21.
Повний текст джерелаBadalov, Nison, and Scott Tenner. "Chronic Pancreatitis and Pancreatic Pseudocysts." In Practical Gastroenterology and Hepatology: Small and Large Intestine and Pancreas, 420–26. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444328417.ch57.
Повний текст джерелаGuarise, Alessandro, Niccolò Faccioli, Giovanni Morana, and Alec J. Megibow. "Chronic Pancreatitis vs Pancreatic Tumors." In Imaging of the Pancreas, 329–69. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-68251-6_18.
Повний текст джерелаSunderland, G. T., and C. W. Imrie. "Pancreatic Fistulas in Acute Pancreatitis." In Pancreatic Fistulas, 61–69. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77418-8_4.
Повний текст джерелаVesentini, S. "Pancreatic Fistulas in Chronic Pancreatitis." In Pancreatic Fistulas, 70–74. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77418-8_5.
Повний текст джерелаHindy, Pierre, and Scott Tenner. "Chronic Pancreatitis and Pancreatic Pseudocysts." In Practical Gastroenterology and Hepatology Board Review Toolkit, 378–82. Oxford, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781119127437.ch63.
Повний текст джерелаТези доповідей конференцій з теми "Pancreatiti"
Hasanova, S. Y. k. "To the question of the treatment of pancreatic necrosis." In General question of world science. Наука России, 2021. http://dx.doi.org/10.18411/gq-31-07-2021-06.
Повний текст джерелаThomas, David, Emma Bermingham, Mark Roberts, and Wayne Young. "An investigation into the effect of high fat and carbohydrate diets on a range of biomarkers associated with pancreatitis in dogs." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/uvdt4784.
Повний текст джерелаClark, K., and K. E. Gross. "Pancreatico-Pleural Fistula and Pancreatico-Pericardial Fistula: Unusual Complications of Pancreatitis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6439.
Повний текст джерелаMARINGHINI, A., R. PATTI, A. TERMINI, M. CIAMBRA, and P. BIFFARELLA. "PANCREATIC FUNCTION TESTS IN DIAGNOSIS OF CHRONIC PANCREATITIS." In Proceedings of the 92nd Course of the International School of Medical Sciences. WORLD SCIENTIFIC, 1999. http://dx.doi.org/10.1142/9789814447249_0012.
Повний текст джерелаBudzinskiy, S., S. Shapovalianz, E. Fedorov, M. Zakharova, and E. Platonova. "ENDOSCOPIC PANCREATIC DUCT STENTING IN MANAGEMENT OF CHRONIC PANCREATITIS." In ESGE Days. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1704663.
Повний текст джерелаRuud, T. E., W. Müller-Esterl, H. Fritz, J. O. Stadaas, and A. O. Aasen. "RELATIONS BETWEEN APROTININ CONCENTRATIONS AND HEMODYNAMICS IN EXPERIMENTAL ACUTE PANCREATITIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644333.
Повний текст джерелаMalinka, T., B. Globke, L. Timmermann, F. Klein, J. Pratschke, and M. Bahra. "Necrotising pancreatitis of the pancreatic remnant (NECROREMNANTITIS): A new definition of severe pancreatitis after pancreatectomy." In Viszeralmedizin 2019. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1695503.
Повний текст джерелаBOCKMAN, DALE E. "PATHOLOGY OF CHRONIC PANCREATITIS: RELATIONSHIP TO PANCREATIC ANATOMY AND NERVES." In Proceedings of the 92nd Course of the International School of Medical Sciences. WORLD SCIENTIFIC, 1999. http://dx.doi.org/10.1142/9789814447249_0001.
Повний текст джерелаLAYER, PETER, and JUTTA KELLER. "HUMAN EXOCRINE PANCREATIC SECRETION IN NORMAL PANCREAS AND CHRONIC PANCREATITIS." In Proceedings of the 92nd Course of the International School of Medical Sciences. WORLD SCIENTIFIC, 1999. http://dx.doi.org/10.1142/9789814447249_0010.
Повний текст джерелаLi, Lisha, Heping Wang, and Yulin Li. "Network Analysis Reveals the Connection Between Chronic Pancreatitis and Pancreatic Cancer." In International Conference on Biomedical and Biological Engineering. Paris, France: Atlantis Press, 2016. http://dx.doi.org/10.2991/bbe-16.2016.59.
Повний текст джерелаЗвіти організацій з теми "Pancreatiti"
Research, Gratis. Gallstone Pancreatitis. Gratis Research, January 2020. http://dx.doi.org/10.47496/gr.blog.08.
Повний текст джерелаAlseidi, Adnan. Pancreatic Anatomy. Touch Surgery Simulations, 2018. http://dx.doi.org/10.18556/touchsurgery/2018.s0111.
Повний текст джерелаJayasinghe, Ravindri, Sonali Ranasinghe, Chandrani Kuruppu, Umesh Jayarajah, and Sanjeewa Seneviratne. Clinical characteristics and outcomes of acute pancreatitis following spinal surgery: a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2022. http://dx.doi.org/10.37766/inplasy2022.7.0017.
Повний текст джерелаSahin-Toth, Miklos. Mouse Model of Human Hereditary Pancreatitis. Fort Belvoir, VA: Defense Technical Information Center, September 2015. http://dx.doi.org/10.21236/ada624309.
Повний текст джерелаMiller, George. Divergent Effects of Dendritic Cells on Pancreatitis. Fort Belvoir, VA: Defense Technical Information Center, September 2015. http://dx.doi.org/10.21236/ada624310.
Повний текст джерелаBeatty, Gregory, Patrick Guirnalda, and Santiago L. Luque. Listeria Vaccines for Pancreatic Cancer. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada600605.
Повний текст джерелаLu, Lei, Hangbin Jin, Jing Yang, Jianfeng Yang, and Xiaofeng Zhang. Early versus delayed cholecystectomy for mild gallstone pancreatitis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0033.
Повний текст джерелаWang, Lei. Molecular Probes for Pancreatic Cancer Imaging. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.3105.
Повний текст джерелаYu, David. The Replication Stress Response in Pancreatic Cancer. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada599228.
Повний текст джерелаYu, Davis S. The Replication Stress Response in Pancreatic Cancer. Fort Belvoir, VA: Defense Technical Information Center, December 2014. http://dx.doi.org/10.21236/ada621841.
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