Готові списки джерел за темами / Oral cancers

Добірка наукової літератури з теми "Oral cancers"

Автор: Grafiati
Опубліковано: 10 грудня 2022
Оновлено: 29 січня 2023

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Статті в журналах з теми "Oral cancers"

1

G. Robayo, D. Adriana, Raquel F. Hernandez, Alveiro T. Erira, Ljubov Kandaurova, Celia L. Juarez, Victoria Juarez, and Angel Cid-Arregui. "Oral Microbiota Associated with Oral and Gastroenteric Cancer." Open Microbiology Journal 14, no. 1 (February 10, 2020): 1–17. http://dx.doi.org/10.2174/1874285802014010001.

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Анотація:
When the normal microbiota-host interactions are altered, the commensal microbial community evolves to a dysbiotic status resulting in some species becoming pathogenic and acting synergistically in the development of local and systemic diseases, including cancer. Advances in genetics, immunology and microbiology during the last years have made it possible to gather information on the oral and gastrointestinal microbiome and its interaction with the host, which has led to a better understanding of the interrelationship between microbiota and cancer. There is growing evidence in support for the role of some species in the development, progression and responses to treatment of various types of cancer. Accordingly, the number of studies investigating the association between oral microbiota and oral and gastrointestinal cancers has increased significantly during the last years. Here, we review the literature documenting associations of oral microbiota with oral and gastroenteric cancers.
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2

Savita Sharma. "Oral Cancer and HPV Connection: A Review." International Healthcare Research Journal 1, no. 8 (November 10, 2017): 240–42. http://dx.doi.org/10.26440/ihrj/01_08/122.

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Анотація:
HPV is the leading cause of oropharyngeal cancers and a few oral cavity cancers with increasing mortality across the globe. The risk factors for oral cancer are well known. Factors like tobacco chewing, smoking, alcohol consumption and actinic radiation have been extensively studied and clinically validated. However , Recently HPV has been shown to be a significant risk factor for oral and oropharyngeal cancers. The HPV family contains around 200 strains but it is important to note that only nine out of them are high risk and associated with cancers. Amongst them HPV16 is most strongly related with oral cancer. HPV associated cancers are different from cancers originating from other etiologies and thus , require a novel multidisciplinary treatment approach. The article is a review of Molecular Biology , Risk Factors ,Clinical aspects ,Diagnosis ,and Treatment of HPV associated Oral cancer .
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3

Soufi, Hissam E., Mohan Kameswaran, and Tarek Malatani. "Khat and oral cancer." Journal of Laryngology & Otology 105, no. 8 (August 1991): 643–45. http://dx.doi.org/10.1017/s0022215100116913.

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AbstractOral cancers in the Asir region of Saudi Arabia have been observed to occurmostly among patients who have been long-term khat users. In a survey that reviewed cancers for the past two years there were 28 head and neck cancer patients, 10 of whom presented with a history of having chewed khat. One of these was a case of metastatic cervical lymph node and unknown primary, one was a parotid tumour, and the remaining eight presented with oral cancers. All were non-smoking khat chewers and all of them had used itover a period of 25 years or longer. We conclude that this strong correlation between khat chewing and oral cancer warrants attention.
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4

Takano, Atsushi, Yoshihiro Yoshitake, Masanori Shinohara, Yohei Miyagi, and Yataro Daigo. "Characterization of URST1 as a prognostic biomarker and therapeutic target for lung and oral cancers." Journal of Clinical Oncology 35, no. 15_suppl (May 20, 2017): e14057-e14057. http://dx.doi.org/10.1200/jco.2017.35.15_suppl.e14057.

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e14057 Background:Lung cancer and oral cavity cancer belong to aerodigestive cancers whose clinical outcome after multimodal therapy remains poor. Methods:To identify new biomarkers and therapeutic targets for lung and oral cancers, we screened genes using our original gene expression profile database for various solid cancers, and characterized clinical and oncogenic values of up-regulated in solid tumor 1 (URST1) protein as a candidate. Results: URST1 was highly expressed in the most of lung or oral cancer cell lines and tumor tissues, but was hardly detected in normal lung or oral epithelial cells and tissues as detected by real-time PCR and western blot analyses. Immunocytochemical analysis revealed that URST1 was mainly localized in the nucleus and cytoplasm of lung or oral cancer cell lines. Immunohistochemical analysis using tissue microarray showed that positive staining of URST1 was observed in 231 of 358 (64.5%) lung cancers, but not in normal lung, and it was significantly associated with poor prognosis after curative surgery. In addition, URST1 was expressed in 64 of 96 (66.7%) oral cancers, but not in oral mucosa, and it was significantly correlated with poor clinical outocome after surgery. Multivariate analysis confirmed that URST1 expression was an independent prognostic factor for oral cancer. Suppression of URST1 expression by siRNA or treatment with synthesized inhibitor specific for URST1 activity inhibited mitosis and growth of lung or oral cancer cell lines. Conclusions:URST1 is likely to be a prognostic biomarker and therapeutic target for aerodigestive cancers such as lung and oral cancers.
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5

Lim, Asher AT, Tze Haur Wee, and Raymond CW Wong. "Epidemiology of Oral Cancer Diagnosed at a Singapore Tertiary Healthcare Institution." Annals of the Academy of Medicine, Singapore 43, no. 2 (February 15, 2014): 96–101. http://dx.doi.org/10.47102/annals-acadmedsg.v43n2p96.

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Анотація:
Introduction: Oral and pharyngeal cancers grouped together are the sixth most common cancer seen worldwide. Oral cancers are, however, relatively not common in Singapore. There are few published epidemiological studies of oral cancers seen in Singapore. This article is a retrospective study of oral cancer incidence in a major tertiary institution in Singapore from 1991 to 2001. Materials and Methods: All oral cancers diagnosed from 1991 to 2001 were extracted from the register of histopathology results and case notes were reviewed. Results: In our study, it was found that oral malignancies preferentially affect older males. Chinese, being the predominant ethnicity, reports the highest incidence of oral malignancy. Squamous cell carcinoma was the most common oral malignancy with the tongue being the most commonly affected site. The majority of patients who sought treatment had complaints of swelling and were referred from restructured government hospitals and clinics. Conclusion: Dental professional should be aware of the common clinical presentation of oral cancers. As oral cancer is a disease of high morbidity and mortality, a concerted effort from the government and healthcare profession will be required to improve the outcome of the disease. Key words: Oral malignancies, Oral squamous cell carcinoma
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6

Bora, Panchali, Sanjeet Singh, Kanika Sharma, Nishant Singh, and Paramjit Singh. "ADVANCED DIAGNOSTIC AIDS IN ORAL PATHOLOGY." International Journal of Advanced Research 10, no. 08 (August 31, 2022): 511–17. http://dx.doi.org/10.21474/ijar01/15204.

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Анотація:
Oral cancers are one of the most common cancers worldwide today. They are usually neglected by the common population when compared to systemic cancers such as the lung cancer, colon cancer etc. However, they also may be extremely fatal if left untreated even at a very initial stage of the lesion. Early detection and treatment gives the best chance for its cure. The five-year survival rate of oral cancer still remains low and delayed diagnosis is suggested to be one of the major reasons. The detection and diagnosis are currently based on clinical examination, histopathological evaluation of the biopsy material and molecular methods. Several diagnostic aids have been developed over the years for early detection of oral cancer. The purpose of this article is to review the advanced available diagnostic adjuncts for the detection of oral cancer.
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7

Komma, Siva Teja, Anil Kumar Sakalecha, Mohiyuddin S. M. Azeem, A. V. Darshan, Rajeswari, and G. R. Varshitha. "Multi detector computed tomographic evaluation of neck spaces in locally advanced oral cancers." JOURNAL OF CLINICAL AND BIOMEDICAL SCIENCES 12, no. 1 (March 15, 2022): 22–29. http://dx.doi.org/10.58739/jcbs/v12i1.7.

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Анотація:
Background: Malignancy of the oral cavity account for about 7.6% of total cancer in India. Local anatomical spread of oral cancers into the face and maxilla and further extension into neck spaces is critical for staging of cancers. Cancer staging helps in the treatment of oral cancers. CT is considered the primary modality of investigation as it helps in delineating the size and extent of primary tumor. Objective: To perform multi-detector computed tomography (MDCT) and to evaluate its usefulness in as-sessing the involvement of deep neck spaces in locally advanced oral cancers. To assess CT morphology specifi-cally to know resectability or non-resectability of oral cancers. Methods and Material: Study included a total of 39 cases of locally advanced oral cancer referred for CECT neck. MDCT findings were analyzed with regard to location, size and extent of the disease and findings were correlated with either clinical follow up or surgical findings. Results: In our study highest number of cases were of carcinoma buccal mucosa (71.8 %) followed by carcinoma tongue and lower alveolus. Most commonly involved neck space was buccal space (94.9 %). CT was 100% accurate in detecting the bone erosion. Conclusion: MDCT evaluation of neck spaces in locally advanced oral cancers is a superior diagnostic tool in tumour staging and appropriate treatment planning. Surgical management of T4b oral cancers in our study subjects showed good results with > 50% of patients showing loco-regionally controlled disease after surgery. Keywords: Computed tomography, Neck spaces, Oral, Cancers
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8

Shinohara, Shogo, Masahiro Kikuchi, Hiroyuki Harada, Kiyomi Hamaguchi, Ryo Asato, Hisanobu Tamaki, Masanobu Mizuta, et al. "Clinicopathological Characteristics and Survival Outcomes of Patients with Buccal Squamous Cell Carcinoma: Results of a Multi-Institutional Study." Medicina 57, no. 12 (December 13, 2021): 1361. http://dx.doi.org/10.3390/medicina57121361.

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Background and Objectives: To investigate clinicopathological characteristics and survival outcomes of patients with buccal cancer in Japan. Materials and Methods: This study was conducted using a database of 1055 patients with oral cancers treated between 2010 and 2017 at 12 institutions in Japan. Ninety-two patients (8.7%) with primary buccal cancer were extracted and clinicopathological characteristics and survival outcomes were compared between patients with buccal cancers and patients with other oral cancers. Results: Ages were significantly higher in the patients with buccal cancer (73 years old vs. 69 years old). Buccal cancer had less advanced cT stage and cN stage than other oral cancers. Overall 5-year survival (OS) was 80.6%, and recurrence-free 5-year survival (RFS) of buccal cancers was 67.8%, and there were no significant differences in survival compared with other oral cancers in terms OS or RFS (5y-OS: 82.5%, 5y-RFS: 74.4%). However, patients with stage IV buccal cancer showed poorer prognosis in terms of OS and RFS compared with the same stage patients with other oral cancer. Advanced T stage was the only factor independently associated with both OS and RFS of patients with buccal cancer in this study. Conclusions: Postoperative radiotherapy or chemoradiotherapy should be considered to improve survival outcome of buccal cancer patients, especially for the patients with advanced primary site disease or a higher cancer stage.
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Reitano, Elisa, Nicola de’Angelis, Paschalis Gavriilidis, Federica Gaiani, Riccardo Memeo, Riccardo Inchingolo, Giorgio Bianchi, Gian Luigi de’Angelis, and Maria Clotilde Carra. "Oral Bacterial Microbiota in Digestive Cancer Patients: A Systematic Review." Microorganisms 9, no. 12 (December 14, 2021): 2585. http://dx.doi.org/10.3390/microorganisms9122585.

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Анотація:
The relation between the gut microbiota and human health is increasingly recognized. Recently, some evidence suggested that dysbiosis of the oral microbiota may be involved in the development of digestive cancers. A systematic review was conducted according to the PRISMA guidelines to investigate the association between the oral microbiota and digestive cancers. Several databases including Medline, Scopus, and Embase were searched by three independent reviewers, without date restriction. Over a total of 1654 records initially identified, 28 studies (2 prospective cohort studies and 26 case-controls) were selected. They investigated oral microbiota composition in patients with esophageal squamous cell carcinoma (n = 5), gastric cancer (n = 5), colorectal cancer (n = 9), liver carcinoma (n = 2), and pancreatic cancer (n = 7). In most of the studies, oral microbiota composition was found to be different between digestive cancer patients and controls. Particularly, oral microbiota dysbiosis and specific bacteria, such as Fusobacterium nucleatum and Porphyromonas gingivalis, appeared to be associated with colorectal cancers. Current evidence suggests that differences exist in oral microbiota composition between patients with and without digestive cancers. Further studies are required to investigate and validate oral–gut microbial transmission patterns and their role in digestive cancer carcinogenesis.
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10

Chaturvedi, Anil K., Neal D. Freedman, and Christian C. Abnet. "The Evolving Epidemiology of Oral Cavity and Oropharyngeal Cancers." Cancer Research 82, no. 16 (August 16, 2022): 2821–23. http://dx.doi.org/10.1158/0008-5472.can-22-2124.

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Анотація:
In 1988, Blot and colleagues reported results from a U.S. case–control study of oral cavity or pharyngeal (oropharyngeal and hypopharyngeal) cancers, with results showing independent associations of smoking and alcohol with increased risk, multiplicative interaction effects between smoking and alcohol, and that nearly three-quarters of these cancers are attributable to smoking and alcohol. The report by Blot and colleagues represents a landmark in oropharyngeal cancer epidemiology. This study, the largest at the time, introduced several novel concepts in oropharyngeal cancer epidemiology that remain relevant today—etiologic heterogeneity, statistical interaction effects, adjusted attributable fractions, and disparities by sex and race/ethnicity. Perhaps the most significant recognition in the field since 1988 is the etiologic association of human papillomavirus (HPV, primarily HPV16) with cancers arising in the oropharynx. Today, more than 80% of oropharyngeal cancers in the United States are caused by HPV while only approximately 3% of oral cavity cancers are caused by HPV. This etiologic heterogeneity across head and cancer subsites revealed by HPV is manifest at the genetic/genomic, epidemiologic, and clinical levels. Tobacco and alcohol remain the major etiologic factors for oral cavity cancers while HPV is the major cause of oropharyngeal cancers. Thus, tobacco and alcohol control and prophylactic HPV vaccination remain the most promising prevention tools for oral cavity and oropharyngeal cancers at this time. Importantly, the ever-emerging alternative tobacco products, such as smokeless tobacco/snus, hookah and water pipes, e-cigarettes, flavored cigars and cigarillos, and oral dissolvable products, represent a key public health concern and the carcinogenic effects of these products remains an active area of investigation. See related article by Blot and colleagues, Cancer Res 1988;48:3282–7
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Більше джерел

Дисертації з теми "Oral cancers"

1

Sastry, Preeti. "Effect of Metro Living on Oral Cancers in Virginia: 2001-2005." VCU Scholars Compass, 2008. http://scholarscompass.vcu.edu/etd/1649.

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Анотація:
Background: Forty percent of all head and neck cancers occur in the oral cavity. According to ICD-O (International classification of diseases for oncology) C00-C14 includes cancers of the lip, oral cavity and pharynx. Studies have indicated that increased population density or Metro living have increased oral cancer incidence. The objectives of this study are to look at the distribution of Oral and Oro pharyngeal Cancers in Virginia from 2001-2005 The study aims to determine if there is an association between metro living (beale code 3) and advanced Oral Cancers. This study is also being done to determine if Metro living is a predictor of Oral Cancer after adjusting for gender. Methods: The data for this study was obtained from the Virginia Cancer Registry. Cancer counts were obtained based on gender, beale code distribution and stage at diagnosis. The counts were collected for the years 2001-2005 based on the ICD-O codes C00-C14. Analysis of this secondary data was done using SAS 9.1. Descriptive statistics presents the distribution of oral cancer according to the stage, gender and urbanity level of the patient. A log-linear model was done to look for association between metro living and Oral Cancers in Virginia after adjusting for gender and stage. This model was fit using a Poisson’s regression to observe if the cancer counts are influenced by the urban beale code 3. Results: During the five year period of 2001- 2005 the Virginia Cancer registry received a total of 3,390 reported cases of oral and pharyngeal cancers. Out of the 3,390 cases 67.35% (2283) of the cases were diagnosed in males and the rest 32.65% were females (1107). Based on the stage at diagnosis, 34.45% (1168) of Oro-pharyngeal cancers were diagnosed to have localized staging as compared to 50.18% (1701) regional and 11.03% (374) distant. 4.34% of the cancers were unstaged (N=147). 82% of all Oro-pharyngeal cancers were seen amongst whites. Majority of oral cancers were seen amongst age groups 35-74 years (78.41%). While looking at the distribution of oral cancers reported from the urban populations; 82.3% (2790) were reported from beale code1. Only 9.73% (330) cases were reported from beale code 2 and 7.96% (270) cases were reported from beale code 3 (population fewer than 250,000 people). More than 50% of cancers were diagnosed at an advanced stage in the urban populations; we did not see a significant relation between advanced oral cancers and metro living. (p =0.2878). After performing a scaled deviance Poisson regression model indicated that there was a stable trend in counts of advanced oral cancer after adjusting for race, age and gender.(θ=-0.04 p-value =0.617). Conclusions: With a linear trend between increased population density and advanced oral cancers, our study observed a stable trend within the metro populations. Due to lack of a clear understanding of all the possible contributing factors further research is recommended to observe the various etiological differences within the urban populations and advanced oral cancers.
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2

Chiriseri, Edina. "Human papilloma virus and oral cancers : sexual behaviour as a risk factor." Thesis, De Montfort University, 2017. http://hdl.handle.net/2086/16084.

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Анотація:
AIM & OBJECTIVES: Human papilloma virus (HPV) has been related to cervical infection, however, its part in Head and Neck Squamous Cell Carcinoma (HNSCC) is still debatable and is easy to refute. Suspicion of HPV causation is heightened when carcinomas arise in patients that are young and have never smoked. The present UK based study undertaken at Northampton NHS Trust endeavoured to determine the extent to which HPV is an entity in HNSCC in the UK. Furthermore, the study investigated whether sexual behaviour (as measured by sexual health clinic (SHC) attendance) is linked the acquisition of HPV associated HNSCC in young age groups. HNSCC incidences and sexual trends in the UK were collected from publicly available databases to identify if there were any changes at a national level in sexual behaviours and their influence on HNSCC in young age groups. MATERIALS & METHODS: PCR was used to evaluate the presence of HPV in biopsy samples from of 99 patients diagnosed with HNSCC at Northampton Hospital from 2006 to 2014. Patient demographics on age, sex, smoking, alcohol use and SHC attendance were also collected. All HPV PCR positive biopsies were further genotyped using an ABI 3130xl genetic analyser. Databases in the UK; including GLOBOCAN, NATSAL and PHE were searched for data on HNSCC prevalence, sexual behaviour trends and vaccine uptake. Multinomial regression explored the relationship between HPV positivity and sex, age, smoking, drinking, race and SHC attendance. RESULTS: PCR showed that 25.2% (25/99) of biopsies tested were positive for HPV and were all obtained from white participants. Most specimens (23, 92%) were high-risk (HR) HPV 16 positive with a mean age of 56 for HPV positivity and 72% of the cases 50-60 years old. Smokers were 11% in total (11/99) with most 88.9% participants (88/99) being non-smokers. HPV positivity was strongly linked with non-smoking history (p < 0.001); no alcohol abuse (p < 0.001); male gender (p < 0.001); young age less than 60 years (p < 0.001) and SHC attendance (p < 0.001). A Kruskal-Wallis post hoc test affirmed the impact of age on HPV positivity (p= < 0.05). GLOBOCAN and Cancer Research demonstrated a rising UK HNSCC pattern of over 200% for both sexes from 1975 to 2011. The three NATSAL surveys undertaken in 1990-1991, 1999-2001 and 2010-2012 demonstrated an overall increase in opposite and same sex partners. The UK average of individuals engaging in oral sex was in the younger age groups of between 16 and 54 with at least 70% of males and 63% females of that age engaging in oral sex. Finally, NASTAL 1, 2 and 3 surveys reported 20 vs 15; 25 vs 55; 55 vs 65 of males and females respectively with more than 10 sexual partners to have attended the SHC. The UK immunization take-up was over 90% countrywide. CONCLUSION: Few research studies have been conducted to date on HPV as a cause of HNSCC in the UK. The present research showed 25.2% of HNSCC to be caused by HPV, with the high risk (HR) genotype 16 (the leading cause of cervical cancer) accounting for 92% (23/25) of the cases. These outcomes affirmed the high prevalence of HR-HPV in HNSCC, with a rate of 25.2% similar to those reported previously. Routine HPV testing in those aged below 60 is therefore warranted. Smoking and drinking showed negative correlation; the young age of below 60 and attendance of the SHC for both sexes showed a positive correlation with HPV positive HNSCC. NATSAL data showed increased sexually risky behaviour coupled with attending the SHC in younger ages for both sexes. Increased sexually risky behaviour as shown in NASTAL surveys may be the reason why young age and SHC attendance is positively correlated with HPV HNSCC. The study highlights a conceivable relationship between HPV positive HNSCC in those under 60 years with no smoking history who attended the SHC. Smoking and drinking are known risks for HNSCC in those past 65 years of age; the negative association with HPV HNSCC in the young in the present research revealed smoking and drinking to have reduced association with HPV HNSCC. The reported HR-HPV positive HNSCC in young age groups inform future vaccination strategies and consequently decrease the quantity of HPV HNSCC's.
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3

De, Camargo Cancela Marianna. "Les cancers de la cavité buccale et de l’oropharynx dans le monde : incidence internationale et classification TNM dans les registres du cancer." Thesis, Lyon 1, 2010. http://www.theses.fr/2010LYO10311/document.

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Анотація:
L’objectif de ces travaux est de connaître et évaluer les caractéristiques épidémiologiques des cancers de la cavité orale et de l’oropharynx. Ces deux localisations partagent des facteurs de risque en commun, et sont de fait souvent regroupées dans les études épidémiologiques. Cependant, la découverte de facteurs de risque spécifiques, telle l’infection par le virus du papillome humain pour les cancers de l’oropharynx, nous conduit à fournir des taux d’incidence spécifiques avec la classification anatomique de ces cancers. En réorganisant les données disponibles dans la base des données du Centre International de Recherche sur le Cancer, nous avons recherché les cas incidents au niveau mondial et recalculé les taux d’incidence dans les registres de 60 pays, pendant la période 1998-2002. La classification TNM n’est pas disponible dans les bases de données du CIRC. Nous avons identifié et contacté les registres du cancer qui ont déclaré son recueil. Cela nous a permis de créer et structurer une base des données innovante et inédite, dont les informations ont été analysées par rapport à la qualité. Finalement nous avons comparé la distribution de stades précoces et avancés dans 8 pays. Les résultats montrent que l’incidence des cancers de la cavité buccale et de l’oropharynx est très hétérogène au niveau mondial par rapport à la sous localisation des tumeurs, à l’âge d’incidence, au ratio homme/femme et au stade clinique
Oral cavity and oropharynx cancers : International incidence and TNM classification in population-based cancer registries The aim of this work was to know and to evaluate the epidemiological patterns of oral cavity and ororpharynx cancers. These topographies share some common risk factors and they are often grouped in epidemiological studies. However, the implication of the human papilloma virus in oropharyngeal tumors lead us to provide incidence rates according to the anatomical classification of these tumors. We reorganized the incidence data available at the International Agency for Research on Cancer, for the period 1998-2002. Incidence rates were calculated for oral cavity and oropharynx cancers separately for 60 countries. As the TNM classification is not available on the IARC database we contacted the cancer registries that declared to abstract and collect it. Based on their data we created and structure a new, innovative and quality controlled. Finally, we compared the TNM stage distribution among 8 countries. The results show that the oral cavity and oropharynx cancers have a very heterogeneous distribution in the studied registries concerning tumor sub-sites, age of incidence, male to female ratio and clinical stage
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4

Auluck, Ajit. "Epidemiological shifts and risk behaviours for oral and oropharyngeal cancers in multicultural population of British Columbia, Canada." Thesis, University of British Columbia, 2012. http://hdl.handle.net/2429/41390.

Повний текст джерела
Анотація:
Although smoking prevalence in British Columbia (BC) is decreasing, numbers of oral cancers are increasing. This change may reflect new emerging risk factors, including an increase in human papillomavirus (HPV) infections and greater immigration from high-risk countries. Currently, in BC there is no data on the trends in oral cancer incidence and survival by ethnicity or by etiologically clustered oral cancer subsites (oral cavity cancers, OCC, which are predominantly tobacco related; and oropharyngeal cancers, OPC, which are predominantly HPV-related). Oral cancers were retrieved from BC Cancer Registry (BCCR) from 1980 to 2006 and the following information collected: names, demographic, tumor, treatment and outcome information. When specific information was not complete, chart review was done. South Asian (SA) or Chinese ethnicities were determined by using previously generated ethnic surname list. Age-adjusted incidence rates (AAIR), age-specific incidence rates (ASIR) and 5-year survival rates for these three populations were calculated by sex, grouping the cancers into etiologically clustered subsites. Calculations were done for each year from 1980 to 2006. An ethnographic study was then conducted to describe the patterns of access, use and perceptions of SA men towards chewing tobacco-containing betel quid (BQ). Extensive field work included participant observations and semi-structured interviews. We have for the first time shown that the incidence of HPV-related OPC has surpassed that of tobacco-related OCC in men. For female, the incidence rates of OPC increased and OCC unchanged. AAIR for OCC was highest in SA males and females while rates of OPC were highest in general population males and Chinese males. Survival rates for OCC were unchanged and for OPC improved in males. SA had poorest survival rates for OCC. Ethnographic findings revealed that among SA males chewing tobacco-containing BQ was viewed as a culturally accepted practice. Availability of BQ, perceived benefits of chewing, ability to conceal the habit, and a lack of awareness of health risks also supported chewing practices. These findings provide a strong foundation for continued work in this field aimed at identifying effective prevention and treatment strategies for oral cancer.
Dentistry, Faculty of
Graduate
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5

Auluck, Ajit. "Epidemiological shifts and risk behaviors for oral and oropharyngeal cancers in multicultural population of British Columbia, Canada." Thesis, University of British Columbia, 2012. http://hdl.handle.net/2429/41390.

Повний текст джерела
Анотація:
Although smoking prevalence in British Columbia (BC) is decreasing, numbers of oral cancers are increasing. This change may reflect new emerging risk factors, including an increase in human papillomavirus (HPV) infections and greater immigration from high-risk countries. Currently, in BC there is no data on the trends in oral cancer incidence and survival by ethnicity or by etiologically clustered oral cancer subsites (oral cavity cancers, OCC, which are predominantly tobacco related; and oropharyngeal cancers, OPC, which are predominantly HPV-related). Oral cancers were retrieved from BC Cancer Registry (BCCR) from 1980 to 2006 and the following information collected: names, demographic, tumor, treatment and outcome information. When specific information was not complete, chart review was done. South Asian (SA) or Chinese ethnicities were determined by using previously generated ethnic surname list. Age-adjusted incidence rates (AAIR), age-specific incidence rates (ASIR) and 5-year survival rates for these three populations were calculated by sex, grouping the cancers into etiologically clustered subsites. Calculations were done for each year from 1980 to 2006. An ethnographic study was then conducted to describe the patterns of access, use and perceptions of SA men towards chewing tobacco-containing betel quid (BQ). Extensive field work included participant observations and semi-structured interviews. We have for the first time shown that the incidence of HPV-related OPC has surpassed that of tobacco-related OCC in men. For female, the incidence rates of OPC increased and OCC unchanged. AAIR for OCC was highest in SA males and females while rates of OPC were highest in general population males and Chinese males. Survival rates for OCC were unchanged and for OPC improved in males. SA had poorest survival rates for OCC. Ethnographic findings revealed that among SA males chewing tobacco-containing BQ was viewed as a culturally accepted practice. Availability of BQ, perceived benefits of chewing, ability to conceal the habit, and a lack of awareness of health risks also supported chewing practices. These findings provide a strong foundation for continued work in this field aimed at identifying effective prevention and treatment strategies for oral cancer.
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6

Guillet, Julie. "Les papillomavirus Humains dans les cancers des Voies Aéro-Digestives Supérieures : optimisation de méthodes de détection et étude de populations à risque." Thesis, Université de Lorraine, 2016. http://www.theses.fr/2016LORR0050/document.

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Les Papillomavirus Humains (HPV) sont responsables de près de 100% des cancers du col utérin. Récemment, ces HPV sont apparus comme étant aussi la cause de certaines tumeurs des voies aérodigestives supérieures, et particulièrement des carcinomes épidermoïdes de l’oropharynx. En France, la proportion des tumeurs oropharyngées HPV-induites est mal connue, notamment parce que le dépistage viral n’est pas recommandé. De plus, il est difficile d’évaluer la proportion de tumeurs HPV positives dans les tumorothèques car les échantillons tumoraux sont fixés dans du formol puis inclus en paraffine (FFIP), ce qui complexifie les techniques de détection. Nous avons, au cours de nos travaux, testé une méthode de détection des HPV à haut risque oncogène indiquée pour le traitement des frottis en phase liquide. Nous l’avons mise à l’épreuve sur des prélèvements FFIP et comparée à la technique de référence qu’est la PCR (Polymerase Chain Reaction) suivie d’une électrophorèse sur gel. Nos résultats indiquent que cette technique est applicable aux prélèvements tissulaires et apparaît même comme étant plus sensible. En France, deux tiers des patients atteints de tumeurs des VADS sont pris en charge à des stades tardifs. Ceci s’explique en partie par l’absence de dépistage organisé de ces cancers. Nous avons donc mené une étude prospective sur des patients atteints d’une tumeur des VADS afin de tester le frottis oral comme technique de dépistage des cancers mais également des infections par les HPV. Nos résultats indiquent que le frottis a une spécificité proche de celle de la biopsie (94,4%) pour le dépistage des cancers des VADS, mais une moindre sensibilité (66,7%). Cette étude nous a permis de mettre en évidence une tumeur HPV-induite dans 12,2% des cas. Parmi eux, nous avons détecté grâce à un frottis buccal (en zone saine) une infection par un HPV à haut risque oncogène dans 53,3% des cas. L’OMS a classé les HPV comme agents carcinogènes depuis 1995, et a établi que les patientes ayant développé un cancer du col utérin avaient un risque 6 fois plus élevé de développer une autre tumeur HPV-induite. Dans ce contexte, nous avons prévu une étude prospective multi-centrique visant à dépister une infection orale par un HPV oncogène chez des patientes porteuses d’une lésion pré-néoplasique ou néoplasique du col utérin. Le taux de co-infection des deux sites anatomiques est inconnu chez les femmes infectées au niveau génital. Dans la mesure où l’infection orale pourrait être à l’origine d’une seconde localisation tumorale, il semble important d’en connaître la proportion afin de proposer par la suite un suivi particulier aux populations « à risque ». Au-delà des traitements des cancers avérés se pose la question de la vaccination préventive, qui existe contre les HPV 16 et 18 dans la prévention des cancers du col utérin. Le type 16 étant retrouvé dans 90% des tumeurs épidermoïdes de l’oropharynx HPV-induites, l’extension des recommandations vaccinales apparaît comme une nouvelle question de santé publique
The Human Papillomavirus (HPV) are involved in almost 100% of cervical cancers. Recently, HPVs have been recognized as the cause of tumors of the upper aerodigestive tract, especially of squamous cell carcinoma of the oropharynx. In France, the proportion of oropharyngeal HPV-related tumors is unknown, partly because viral testing is not in guidelines. Moreover, assess the proportion of HPV-positive tumors in tumor banks is difficult because the tumor samples were fixed in formalin and embedded in paraffin (FFPE), which complicates detection techniques. We tested a high risk HPV detection method, indicated for liquid based pap smear, on FFPE samples. We compared this technique to the gold-standard : PCR (Polymerase Chain Reaction) followed by electrophoresis. Our results indicate that this technique is applicable to FFPE samples and even appears to be more sensitive. The majority of French patients (2/3) with head and neck consult with an advanced stage of disease. This is explained in part by the lack of organized screening of these cancers, contrary to breast, prostate, cervical, or colorectal cancers. But an early treatment is essential to increase the survival rate. We therefore conducted a prospective study on patients with head and neck tumors to test the oral brushing as screening cancer and HPV detection. We found tumor and/or dystrophic cells in 97.8% of patients with biopsy, and in 88.9% of patients by brushing. Compared with biopsy, our results suggested that smear has similar specificity for HPV detection in tumors (94.4%), but lower sensitivity (66.7%). This study has shown an HPV-related tumor in 12.2% of cases. Among them, we detected by brushing (in healthy area) an oral infection by high-risk HPV in 53.3% of cases. WHO has classified HPV as carcinogenic agents since 1995, and determined that patients who developed cervical cancer are six-times more likely to develop another HPV-related tumor. In this context, we have planned a multicenter prospective study to detect oral HPV infection in patients with a pre-neoplastic or neoplastic lesion of the cervix. Co-infection rate of the two anatomical sites is unknown in women infected with genital level. Insofar oral infection could be the cause of a second tumor location, it seems important to know how much women are co-infected to propose thereafter a special monitoring. The preventive vaccination, which exists against HPV 16 and 18 in the prevention of cervical cancer, is a future perspective. Because HPV 16 is found in 90% of HPV-related squamous cell carcinoma of the oropharynx, extending vaccine recommendations emerge as a new public health issue
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7

Kauppila, J. (Joonas). "Toll-like receptor 9 in alimentary tract cancers." Doctoral thesis, Oulun yliopisto, 2014. http://urn.fi/urn:isbn:9789526204482.

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Abstract Cancers of the alimentary tract include many common cancer types, some of which have well-established treatment protocols and relatively good prognosis, such as colorectal cancer, and others, which have generally very poor prognosis. The gastrointestinal canal is colonized by a multitude of bacteria, the effects of which are currently poorly understood. Toll-like receptor 9 (TLR9) in cells of the alimentary tract recognizes the bacterial DNA-fragments and regulates immune functions in the host and the cancer. This thesis examines the function and prognostic significance of Toll-like receptor 9 in oral and esophageal squamous cell carcinoma as well as in esophageal, gastric and colorectal adenocarcinoma. The studies were made using tissue samples from patient cohorts and various cell culture techniques. Our data indicate that high expression of Toll-like receptor 9 in cancer cells associates with metastatic properties in oral and esophageal cancers and poor prognosis in esophageal adenocarcinoma and oral squamous cell carcinoma. Cell culture studies further suggest that TLR9 is functional in alimentary tract cancers and mediates cellular invasion when activated. Based on the results, TLR9 is active in alimentary tract cancers and its expression is related to poor cancer prognosis. Thus, TLR9 may represent a novel therapeutic target in alimentary tract cancers and might provide a link between bacteria and oral and gastrointestinal cancer
Tiivistelmä Ruuansulatuskanavan syöpiin lukeutuu useita yleisiä syöpätyyppejä, kuten kohtalaisen hyväennusteinen paksusuolen syöpä, jonka hoitokäytäntö on vakiintunut. Toisissa ruuansulatuskanavan syövissä puolestaan ennuste on hyvin huono. Mahasuolikanavaa asuttavat moninaiset bakteerikannat, joiden vaikutuksia ymmärretään vielä kehnosti. Tollinkaltainen reseptori 9 (TLR9) tunnistaa näiden bakteerien DNA-rakenteita ja vaikuttaa yksilön ja syövän immuunivasteeseen. Tämä väitöstutkimus selvittää TLR9:n toimintaa ja ennustevaikutusta suun ja ruokatorven levyepiteelisyövissä, sekä ruokatorven, mahalaukun ja paksusuolen adenokarsinoomassa. Tutkimus toteutettiin käyttäen syöpäpotilaiden kudosnäytteitä sekä soluviljelytekniikoita. Tuloksemme osoittavat, että TLR9:n lisääntynyt ilmentyminen syöpäsoluissa yhdistyy metastasointiin suu- ja ruokatorvisyövissä, sekä korkeaan kuolleisuuteen suun levyepiteelisyövässä ja ruokatorven adenokarsinoomassa. Soluviljelykokeidemme tuloksiin nojaten TLR9 toimii ruuansulatuskanavan syövissä ja sen aktivaatio saa aikaan solujen invasoitumisen. Tutkimustuloksiimme vedoten TLR9 on aktiivinen ja toimiva ruuansulatuskanavan syövissä ja sen ilmentyminen liittyy huonoon ennusteeseen. TLR9 saattaa osoittautua uudeksi syöpähoitojen kohteeksi tai yhdistäväksi tekijäksi syövän ja bakteerien välillä ruuansulatuskanavan syövissä tulevaisuudessa
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8

Le, Béguec Céline. "Analyse intégrative des ARN longs non-codants chez le chien et leurs implications dans le mélanome oral canin, modèle des mélanomes humains." Thesis, Rennes 1, 2018. http://www.theses.fr/2018REN1B030/document.

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Les ARN longs non-codants (lncRNAs) constituent une famille d'ARN hétérogènes qui jouent un rôle majeur dans de nombreux cancers et notamment dans les mélanomes. Le chien est un modèle naturel et spontané pour l’analyse génétique comparée des cancers et, l'annotation du génome canin a récemment été enrichie avec l'identification de plus 10 000 lncRNAs. Afin de réaliser des prédictions fonctionnelles bioinformatiques des lncRNAs, nous avons caractérisé les profils d'expression des lncRNAs canins à partir de 26 tissus distincts. Nous avons défini la spécificité tissulaire de l’expression des lncRNAs et inféré leur fonctionnalité potentielle par des analyses de génomique et de transcriptomique comparatives avec des données humaines issues du projet ENCODE (ENCyclopedia Of DNA Elements). Comme chez l'homme et la souris, une grande proportion de lncRNAs canins (44 %) est exprimée de manière spécifique au sein d’un tissu. Par une approche de génomique comparative, nous avons identifié plus de 900 lncRNAs orthologues entre l’homme et le chien et pour 26 % d’entre eux, des patrons d'expression entre tissus significativement conservés (p < 0,05). Dans le cadre de l'étude des mélanomes canins, nous avons analysé les données de RNA-seq de 52 échantillons tumeurs/contrôles de mélanomes oraux. Nous avons identifié plus de 750 lncRNAs différentiellement exprimés entre la tumeur et le contrôle (FDR < 0,01), dont plus de 100 conservés avec l’homme. Ces lncRNAs constituent de bons candidats pour étudier la régulation de la progression tumorale des mélanomes chez le chien et pourront être évalués pour leurs potentiels diagnostic et thérapeutique en médecine humaine et vétérinaire
Long non-coding RNAs (lncRNAs) are a family of heterogeneous RNAs that play a major role in many cancers, particularly in melanomas. The dog is a natural and spontaneous model for the comparative genetic analysis of cancers and, the annotation of the canine genome has recently been enriched with the identification of over 10,000 lncRNAs. In order to perform functional bioinformatic predictions of lncRNAs, we have characterized the expression patterns of canine lncRNAs from 26 distinct tissues representative of the major functions of the organism. We defined the tissue specificity of lncRNAs expression and inferred their potential functionality by comparative genomic and transcriptomic analyses with human data from the ENCODE project (ENCyclopedia Of DNA Elements). As in humans and mice, we show that a large proportion of canine lncRNAs (44%) are expressed specifically within a tissue. Using a comparative genomic approach, we have identified more than 900 orthologue lncRNAs between humans and dogs, and we show that for 26% of them, tissue expression patterns are also significantly conserved (p < 0.05). In the study of canine melanomas, we investigated the lncRNAs from RNA-seq data from 52 tumour/control samples of oral melanoma. We identified more than 750lncRNAs differentially expressed between tumour and control (FDR < 0.01), of which more than 100 were conserved with humans. These lncRNAs are good candidates to study the regulation of tumour progression of melanomas in dogs and can be evaluated for their diagnostic and therapeutic potential in human and veterinary medicine
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9

Carton, Matthieu. "Facteurs de risque professionnels des cancers des voies aérodigestives supérieures chez les femmes : analyse des données de l’étude Icare." Thesis, Université Paris-Saclay (ComUE), 2017. http://www.theses.fr/2017SACLV002/document.

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Contexte : Peu d’études ont recherché le rôle des facteurs de risque professionnels dans la survenue des cancers des voies aérodigestives supérieures (VADS). Ces études ont été conduites principalement chez des hommes.Objectif : L’objectif de cette thèse était d’étudier les associations entre les cancers des VADS et les expositions professionnelles chez les femmes.Méthodes : Icare est une étude cas-témoins en population générale incluant 296 cas féminins de cancers épidermoïdes des VADS et 775 femmes témoins. Les historiques de carrières recueillis ont été codés et croisés avec les matrices emplois-expositions du programme Matgéné. Outre les intitulés d’emplois, les expositions à 5 solvants chlorés (chloroforme, chlorure de méthylène, perchloréthylène, trichloréthylène, tétrachlorure de carbone), 5 solvants oxygénés (éthylène glycol, tétrahydrofurane, éther éthylique, cétones, alcools), 5 solvants pétroliers (essences carburants, essences spéciales, gazole, benzène, white-spirit) et à 7 poussières et fibres (amiante, farine, cuir, fibres céramiques réfractaires, ciment, laines minérales, silice) ont été étudiées. Les odds-ratios et leurs intervalles de confiance à 95% ajustés sur l’âge, le département, les consommations de tabac et d’alcool ont été estimés par régressions logistiques non conditionnelles.Résultats :Plusieurs professions et secteurs d’activité associés à un risque élevé de cancer des VADS ont été identifiés. Certaines professions (ouvrières de l’alimentation et des boissons, monteuses en appareillage électrique ou électronique, soudeuses) peuvent être à l’origine d’expositions professionnelles aux solvants, aux métaux, aux fumées de soudage et à diverses poussières. Les analyses par nuisance ont mis en évidence des associations significatives entre le risque de cancer des VADS et l’exposition au perchloréthylène et au au trichloréthylène. Aucune association claire n’est observée avec les solvants pétroliers et oxygénés, certains largement utilisés par les femmes L’exposition aux poussières de farine augmente significativement le risque de cancer des VADS. Une exposition probable à l’amiante est associée à une augmentation modérée et non significative du risque. Les analyses par localisation de cancer (cavité orale, pharynx, larynx), limitées par des effectifs faibles, ne mettent pas en évidence d’association spécifique.Conclusion : Nos résultats suggèrent un rôle des expositions professionnelles au trichloréthylène, au perchloréthylène et aux poussières de farine dans la survenue des cancers des VADS chez les femmes
Background : Few occupational studies have addressed head and neck cancer, and these studies have been predominantly conducted in men. Objective : Our objective was to investigate the associations between head and neck cancer and occupational exposures in women Population and methods : ICARE, a French population-based case–control study, included 296 squamous cell carcinomas of the head and neck (HNSCC) in women and 775 female controls. Lifelong occupational history was collected. Job-exposure matrices were used to assess exposure to five chlorinated solvents (carbon tetrachloride; chloroform; methylene chloride; perchloroethylene; trichloroethylene), 5 petroleum solvents (benzene; special petroleum product; gasoline; white-spirits and other light aromatic mixtures; diesel, fuels and kerosene), 5 oxygenated solvents (alcohols; ketones and esters; ethylene glycol; diethyl ether; tetrahydrofuran) and 7 fibers and dusts (asbestos, flour dust, leather dust, refractory ceramic fibers, cement dust, mineral wools and silica) . An analysis by job title was conducted, and then associations with specific occupational exposures were investigated.Odds ratio (ORs) and 95% confidence intervals (CI), adjusted for smoking, alcohol drinking, age and residence area, were estimated with logistic models. Results : Significantly increased HNSCC risks were found for several jobs and industries. Some of these occupations (food and beverage processors, electrical and electronic equipment assemblers, welders and flame cutters) may entail exposure to agents such as solvents, metals, welding fumes and various dusts. Analyses for specific occupational exposures showed a significantly elevated risk of HNSCC associated with exposure to trichloroethylene and perchloroethylene. There is no clear evidence that petroleum or oxygenated solvents, some of them commonly used by women, are risk factors for HNSCC. Exposure to flour dust increased significantly HNSCC risk. Probable exposure to asbestos was associated with a moderate, non-significant elevation in risk. Analyses by cancer site (oral cavity, pharynx, larynx) were hampered by small numbers and did to reveal any specific association.Conclusion : These findings suggest that occupational exposure to perchloroethylene, trichloroethylene and flour dust may increase the risk of HNSCC in women
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10

supsavhad, wachiraphan. "Novel Molecular Targets for Feline Oral Squamous Cell Carcinoma." The Ohio State University, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=osu1471628009.

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Книги з теми "Oral cancers"

1

Bahadur, Sudhir, and Subramania Iyer, eds. Management of Oral Cancers. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-6499-4.

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2

Ogbureke, Kalu U. E. Oral cancer. Rijeka, Croatia: InTech Europe, 2012.

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3

W, Johnson Newell, and Batsakis John G, eds. Oral cancer. London: Martin Dunitz, 2003.

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4

Kirita, Tadaaki, and Ken Omura, eds. Oral Cancer. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-54938-3.

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5

1926-, Silverman Sol, and American Cancer Society, eds. Oral cancer. 3rd ed. Atlantic: American Cancer Society, 1990.

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6

Oral cancer metastasis. New York: Springer, 2010.

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7

Myers, Jeffrey, ed. Oral Cancer Metastasis. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-0775-2.

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8

M. Fribley, Andrew, ed. Targeting Oral Cancer. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-27647-2.

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9

Panta, Prashanth, ed. Oral Cancer Detection. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-61255-3.

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10

Appleton, Julia. Working with oral cancer. Bicester: Winslow, 1995.

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Частини книг з теми "Oral cancers"

1

Levine, Ronnie, and Catherine Stillman-Lowe. "Oral Cancers." In BDJ Clinician’s Guides, 57–62. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-98207-6_7.

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2

Unger, Keith, Nadeem Riaz, Allen Chen, and Nancy Y. Lee. "Oral Cavity Cancers." In Target Volume Delineation and Field Setup, 35–44. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-28860-9_5.

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3

Unger, Keith, Matthew Forsthoefel, Nadeem Riaz, Allen Chen, and Nancy Y. Lee. "Oral Cavity Cancers." In Practical Guides in Radiation Oncology, 75–86. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-99590-4_6.

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4

Liu, Dongyou. "Oral Cavity Cancer." In Tumors and Cancers, 39–44. Boca Raton : Taylor & Francis, 2018. | Series: Pocket guides to biomedical sciences | “A CRC title, part of the Taylor & Francis imprint, a member of the Taylor & Francis Group, the academic division of T&F Informa plc.”: CRC Press, 2017. http://dx.doi.org/10.1201/b22275-8.

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Sari, Sezin Yuce, Gokhan Ozyigit, Melis Gultekin, Gozde Yazici, Pervin Hurmuz, Mustafa Cengiz, and Murat Beyzadeoglu. "Oral Cavity." In Radiation Therapy for Head and Neck Cancers, 67–91. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-10413-3_5.

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6

Kumar, Piyush, and C. Murali Krishna. "Optical Techniques: Investigations in Oral Cancers." In Oral Cancer Detection, 167–87. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-61255-3_8.

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7

Feng, Zipei, Carlo B. Bifulco, Rom Leidner, R. Bryan Bell, and Bernard A. Fox. "Immunotherapy in Head and Neck Cancers." In Targeting Oral Cancer, 211–24. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-27647-2_9.

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8

Filho, Victor Wünsch. "Cancer of the Oral Cavity, Pharynx, and Nasopharynx." In Occupational Cancers, 49–106. London: Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-2825-0_4.

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Dokhe, Yogesh, Sivakumar Vidhydharan, Krishnakumar Thankappan, and Subramania Iyer. "Epidemiology." In Management of Oral Cancers, 1–22. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-6499-4_1.

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10

Bahadur, Sudhir. "Guidelines in Management of Carcinoma of the Tongue and Floor of Mouth." In Management of Oral Cancers, 133–47. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-6499-4_10.

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Тези доповідей конференцій з теми "Oral cancers"

1

William, William N. "Abstract IA05: Immunoprevention of oral cancers." In Abstracts: AACR-AHNS Head and Neck Cancer Conference: Optimizing Survival and Quality of Life through Basic, Clinical, and Translational Research; April 23-25, 2017; San Diego, CA. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1557-3265.aacrahns17-ia05.

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2

Musulin, Jelena, Daniel Štifanić, Ana Zulijani, and Zlatan Car. "SEMANTIC SEGMENTATION OF ORAL SQUAMOUS CELL CARCINOMA ON EPITHELLIAL AND STROMAL TISSUE." In 1st INTERNATIONAL Conference on Chemo and BioInformatics. Institute for Information Technologies, University of Kragujevac, 2021. http://dx.doi.org/10.46793/iccbi21.194m.

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Oral cancer (OC) is among the top ten cancers worlwide, with more than 90% being squamous cell carcinoma. Despite diagnostic and therapeutic development in OC patients’ mortality and morbidity rates remain high with no advancement in the last 50 years. Development of diagnostic tools in identifying pre-cancer lesions and detecting early-stage OC might contribute to minimal invasive treatment/surgery therapy, improving prognosis and survival rates, and maintaining a high quality of life of patients. For this reason, Artificial Intelligence (AI) algorithms are widely used as a computational aid in tumor classification and segmentation to help clinicians in the earlier discovery of cancer and better monitoring of oral lesions. In this paper, we propose an AI-based system for automatic segmentation of the epithelial and stromal tissue from oral histopathological images in order to assist clinicians in discovering new informative features. In terms of semantic segmentation, the proposed AI system based on preprocessing methods and deep convolutional neural networks produced satisfactory results, with 0.878 ± 0.027 mIOU and 0.955 ± 0.014 F1 score. The obtained results show that the proposed AI-based system has a great potential in diagnosing oral squamous cell carcinoma, therefore, this paper is the first step towards analysing the tumor microenvironment, specifically segmentation of the microenvironment cells.
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3

Singh, S. P., Atul Deshmukh, Pankaj Chaturvedi, and C. Murali Krishna. "In vivo Raman spectroscopy for oral cancers diagnosis." In SPIE BiOS, edited by Anita Mahadevan-Jansen and Wolfgang Petrich. SPIE, 2012. http://dx.doi.org/10.1117/12.905453.

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4

Sahu, Aditi, Sneha Talathi, Sharada Sawant, and C. Murali Krishna. "Classification of oral cancers using Raman spectroscopy of serum." In SPIE BiOS, edited by Anita Mahadevan-Jansen and Wolfgang Petrich. SPIE, 2014. http://dx.doi.org/10.1117/12.2034941.

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5

Lim, Yenkai, Naoki Fukuma, Makrina Totsika, Liz Kenny, Mark Morrison, and Chamindie Punyadeera. "Abstract 577: Oral microbiome biomarker panel to detect oral and oropharyngeal cancers in a clinical setting." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-577.

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6

Lane, Pierre, Catherine F. Poh, J. Scott Durham, Lewei Zhang, Sylvia F. Lam, Miriam Rosin, Michele Follen, and Calum MacAulay. "Endogenous Fluorescence Imaging for the Management of Oral and Cervical Cancers." In Bio-Optics: Design and Application. Washington, D.C.: OSA, 2011. http://dx.doi.org/10.1364/boda.2011.btud3.

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Symer, David E., Keiko Akagi, Kevin R. Coombes, Weihong Xiao, Robert K. L. Pickard, Amit Agrawal, and Maura L. Gillison. "Abstract 132: Comprehensive genomic analysis of human papillomavirus-associated oral cancers." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-132.

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Fouzat, Arij. "Elaeagnus Angustifolia extract inhabits cell invasion of human colorectal cancer cells and increases the survival rate of the Drosophila colon cancer model." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0103.

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Colorectal cancer (CRC), the third most common type of cancer in the world, is an aggressive type of cancer with high tendency to metastasize and invade to other tissues and distant organs. Traditional CRC treatment regimen includes 5-fluorouracil (5-FU); however, tumors develop a resistance against these drugs, apart from the severe side effects that develop upon these therapies. Nowadays, traditional medicinal plants are the focus of increased interest as a source for new potential drugs, particularly those that serve as anti-cancerous agents. Elaeagnus angustifolia (EA) is a medicinal plant that can be used traditionally to manage several human ailments including cancers especially oral and HER2-positive breast cancer as recently reported by our group. However, the effect of EA flower extract on human CRC has not been investigated yet. Therefore, EA effect was explored in vitro using KRAS CRC cell lines (HCT-116 and LoVo) and in vivo using transgenic Drosophila melanogaster model for KRAS gene, which is known to develop CRC. Our results from the in vitro investigations revealed that EA flower extract significantly inhibits cell motility and invasion in addition to colony formation. Moreover, we found that EA extract modulates the epithelial–mesenchymal transition (EMT) event and its related genes; EMT is a known hallmark of cancer invasion and metastasis. More significantly, our in vivo data pointed out that EA extract increases the survival rate of KRAS mutation D. melanogaster model. Our findings implicate that EA extract may possess chemo-preventive effects against human CRC.
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Doyle, Scott, Margaret Brandwein-Gensler, and John Tomaszewski. "Quantification of tumor morphology via 3D histology: application to oral cavity cancers." In SPIE Medical Imaging, edited by Metin N. Gurcan and Anant Madabhushi. SPIE, 2016. http://dx.doi.org/10.1117/12.2217015.

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Jung, Hyun Min, Rushi S. Patel, Donald M. Cohen, Andrew Jakymiw, William W. Kong, Jin Q. Cheng, and Edward KL Chan. "Abstract 191: Subclassification of oral cancers using a miRNA-based combinatorial approach." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-191.

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Звіти організацій з теми "Oral cancers"

1

Archilla, Alberto Rodriguez. Oral Cancers Adjacent to Dental Implants: A Descriptive Study. Science Repository, July 2019. http://dx.doi.org/10.31487/j.dobcr.2019.03.04.

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zhixia, Zhang, Song Jiating, Pan lanlan, xiaoting Lin, and jing li. The Effect of different exercise methods in the treatment of cancer-related fatigue: a network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0004.

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Review question / Objective: To compare the clinical effects of different exercise methods for cancer fatigue by using mesh Meta-analysis, and to choose the best exercise method for cancer fatigue. Condition being studied: Cancer-related fatigue. Eligibility criteria: Inclusion criteria: (1) Study subjects: the patients is caused by fatigue.(2) Intervention: A group of patients used exercise intervention. (3) Study type: RCT. (4) Outcome index: Cancer-related fatigue score.(5) Grey literature is available.(6) Language in Chinese or English.Exclusion criteria:(1) Using oral drugs. (2) It can not provide complete data. (3) Repeatedly published literature. (4) Conference papers. (5) Literature with inconsistent data types:(1) Using oral drugs. (2) It can not provide complete data. (3) Repeatedly published literature. (4) Conference papers. (5) Literature with inconsistent data types.
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3

Dioguardi, Mario, and Diego Sovereto. Application of the Extracts of Pomegranate in Oral Cancer. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0027.

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Review question / Objective: In this scoping review we will focus on identifying those studies that investigated the effects of Punica granatum extracts on oral cancer and more specifically on OSCCs, summarizing the main results and the state of the research at the present time. Eligibility criteria: All studies investigating Punica granatum L. in association with oral and precancerous cancer were considered potentially admissible, no restrictions were applied in relation to the year of publication and based on the language provided that an abstract in English is available. literature reviews were excluded and were used only as sources for bibliographic research.
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Mukhtar, Hasan. Sustained Release Oral Nanoformulated Green Tea for Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, May 2011. http://dx.doi.org/10.21236/ada545577.

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Mukhtar, Hasan. Sustained Release Oral Nanoformulated Green Tea for Prostate Cancer Prevention. Fort Belvoir, VA: Defense Technical Information Center, May 2013. http://dx.doi.org/10.21236/ada585226.

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6

Mukhtar, Hasan, Nihal Ahmad, Vaqar M. Adhami, and Naghma Khan. Sustained Release Oral Nanoformulated Green Tea for Prostate Cancer Prevention. Fort Belvoir, VA: Defense Technical Information Center, May 2012. http://dx.doi.org/10.21236/ada589659.

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Greer, Joseph, Jaime Jacobs, and Molly Ream. Does a Smartphone App Help Patients with Cancer Take Oral Chemotherapy as Planned? Patient‐Centered Outcomes Research Institute (PCORI), April 2019. http://dx.doi.org/10.25302/4.2019.ihs.130603616.

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Gammon, Marilie. Oral Contraceptive Use and HER-2/neu-Positive Breast Cancer Among White and Black Women. Fort Belvoir, VA: Defense Technical Information Center, February 1996. http://dx.doi.org/10.21236/ada306141.

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Ling, Yang, Hou Lili, Zhao Yan, Chen Weihong, Zhang Jinfeng, and Mao Yan. A meta-analysis of the detection rate of mouth opening difficulty in patients with oral cancer. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2022. http://dx.doi.org/10.37766/inplasy2022.6.0064.

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Zhang, Xinyu, Kelu Yang, Xiaonan Liu, Yang Li, Yang Zhao, Kewei Jiang, and Quan Wang. Can oral dietary nutrition prevent cancer chemotherapy-induced cardiotoxicity? An evidence mapping of published in vivo records. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0015.

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