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Статті в журналах з теми "Molecular Physiology and Pharmacology"
Kiehn, Johann, Antonio E. Lacerda, Barbara Wible, and Arthur M. Brown. "Molecular Physiology and Pharmacology of HERG." Circulation 94, no. 10 (November 15, 1996): 2572–79. http://dx.doi.org/10.1161/01.cir.94.10.2572.
Повний текст джерелаUEDA, Hiroshi, and Makoto INOUE. "Molecular pharmacology and physiology of nociceptin." Folia Pharmacologica Japonica 114, no. 6 (1999): 347–56. http://dx.doi.org/10.1254/fpj.114.347.
Повний текст джерелаBleakman, D. "Kainate receptor pharmacology and physiology." Cellular and Molecular Life Sciences (CMLS) 56, no. 7-8 (November 1, 1999): 558–66. http://dx.doi.org/10.1007/s000180050453.
Повний текст джерелаMasaki, Tomoh, Masashi Yanagisawa, and Katsutoshi Goto. "Physiology and pharmacology of endothelins." Medicinal Research Reviews 12, no. 4 (July 1992): 391–421. http://dx.doi.org/10.1002/med.2610120405.
Повний текст джерелаFisher, James W. "Erythropoietin: Physiology and Pharmacology Update." Experimental Biology and Medicine 228, no. 1 (January 2003): 1–14. http://dx.doi.org/10.1177/153537020322800101.
Повний текст джерелаNakanishi, Shigetada. "Molecular physiology of glutamate receptors." Japanese Journal of Pharmacology 67 (1995): 7. http://dx.doi.org/10.1016/s0021-5198(19)35554-4.
Повний текст джерелаBrooks, G. T. "Comprehensive insect physiology, biochemistry and pharmacology." Insect Biochemistry 15, no. 5 (January 1985): i—xiv. http://dx.doi.org/10.1016/0020-1790(85)90131-3.
Повний текст джерелаCiarimboli, Giuliano. "Physiology, Biochemistry, and Pharmacology of Transporters for Organic Cations." International Journal of Molecular Sciences 22, no. 2 (January 13, 2021): 732. http://dx.doi.org/10.3390/ijms22020732.
Повний текст джерелаAnderson, Warwick P. "Molecular biology in the service of physiology, pharmacology and endocrinology." Clinical and Experimental Pharmacology and Physiology 22, no. 12 (December 1995): 934. http://dx.doi.org/10.1111/j.1440-1681.1995.tb02329.x.
Повний текст джерелаde Almeida, Luiz G. N., Hayley Thode, Yekta Eslambolchi, Sameeksha Chopra, Daniel Young, Sean Gill, Laurent Devel, and Antoine Dufour. "Matrix Metalloproteinases: From Molecular Mechanisms to Physiology, Pathophysiology, and Pharmacology." Pharmacological Reviews 74, no. 3 (June 23, 2022): 712–68. http://dx.doi.org/10.1124/pharmrev.121.000349.
Повний текст джерелаДисертації з теми "Molecular Physiology and Pharmacology"
Bahnasi, Yahya Mohamed. "Molecular physiology and pharmacolgy of TRPC5 ion channels." Thesis, University of Leeds, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.496554.
Повний текст джерелаElnakish, Mohammad T. "Mechanisms and Functional Consequences of Cardiac Remodeling: Role of Myocardial Rac1 and Vascular Profilin1 Genes." The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1363694358.
Повний текст джерелаBonilla, Ingrid Marie. "Acquired Electrophysiological Remodeling and Cardiac Arrhythmias." The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1396024058.
Повний текст джерелаSzabo, Elod Zala. "Molecular and cellular properties of the human brain Na+H+ exchanger isoform 5." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=38420.
Повний текст джерелаPharmacological analyses demonstrated that H+ i-activated 22Na+ influx mediated by NHE5 was inhibited by several classes of drugs at half-maximal concentrations that were intermediate to those determined for the high-affinity NHE1 and the low-affinity NHE3 isoforms. Kinetic analyses showed that the extracellular Na+-dependence of NHE5 activity followed a simple hyperbolic relationship and, unlike other NHE isoforms, the intracellular H+-dependence also exhibited first-order kinetics. Extracellular monovalent cations, such as H+ and Li+, but not K+, acted as effective competitive inhibitors of 22Na+ influx by NHE5.
To find novel interacting proteins that are involved in NHE5 regulation, a yeast two-hybrid screen of human brain cDNA library was conducted using NHE5 as bait. A clone encoding the AMP-activated protein kinase (AMPK) alpha2 subunit was further analyzed. AMPK is a serine/threonine kinase that is activated by elevated ratios of [AMP]/[ATP], regulating various biological processes in response to hypoxia or exercise. AMPK alpha2 binds NHE5 in vitro and in vivo, and directly phosphorylates it in vitro. Activation of endogenous AMPK by AICAR, a membrane permeable AMP analogue, as well as heterologous expression of the full-length and constitutive active forms of alpha2 subunit increased the transporter activity measured by 22Na+ influx.
The regulatory protein arrestin3 was also found to interact with NHE5 in the yeast two-hybrid screen. Arrestins were previously shown to associate with and regulate transmembrane proteins of the G protein-coupled receptor family. We demonstrate that NHE5 binds arrestin3 both in vitro and in vivo; and the binding is phosphorylation-dependent. When co-expressed in CHO cells, arrestin3 and NHE5 co-localize, and arrestin3 expression seems to attenuate the basal activity of the transporter. The data presented in this thesis reveals new aspects of both NHE regulation, and AMPK and arrestin function.
Zicha, Stephen. "Molecular basis for ion current heterogeneity in normal and diseased hearts." Thesis, McGill University, 2004. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=85660.
Повний текст джерелаHere, we describe the variable dependence on repolarizing K+ currents in different species as being the result of the lack of Ito subunits in guinea pig heart with a greater expression of IK subunits, while rabbits express all hypothesized Ito subunits, but express IK subunits at low levels. Humans are found to lie in between these two species in terms of the expression of these voltage-gated K+ channel subunits. The specialized function of certain regions of the heart, such as the ventricles and the SAN, have been attributed to the heterologous expression of Ito and the pacemaker current (I f) respectively. Here were demonstrate that both Kv4.3 and KchIP2 gradients underlie an observed Ito transmural gradient and contribute to the dispersion of repolarization, while a greater expression of HCN2 and HCN4 subunits in the SAN compared to the right atrium account for the larger I f current in this region. Cardiovascular diseases such as congestive heart failure (CHF) have been associated with ion channel remodelling. Here, we report the finding of changes in Nav1.5, Kv4.3, HCN2 and HCN4 expression which may underlie some of the electrophysiological changes associated with this disease. Furthermore, we characterise a genetic polymorphism which is associated with another disease, atrial fibrillation.
The heterologous expression of voltage-gated ion channel subunits may account for many of the species-, region- and disease-specific differences which have been observed in the heart. Such heterogeneity contributes to the proper functioning of the heart under normal conditions, but may also contribute to the pathogenesis of cardiovascular disease.
Richman, Jeremy Golding 1970. "Characterization of α₂-adrenergic receptor localization and functional responses". Diss., The University of Arizona, 1998. http://hdl.handle.net/10150/282583.
Повний текст джерелаSinha, Sayantani. "Role of TRPA1 and TRPV1 in Propofol Induced Vasodilation." Thesis, Kent State University, 2014. http://pqdtopen.proquest.com/#viewpdf?dispub=3618926.
Повний текст джерелаAims: Propofol, clinically named as Diprivan is an intravenous anesthetic known to cause hypotension in patients presenting for surgery. We have investigated the vasodilatory signaling cascade by which propofol causes hypotension using both in vivo and in vitro experimental approaches.
Methods and Results: Using high-fidelity microtip transducer catheter, mean arterial blood pressure (MAP) was measured in control, transient receptor potential ankyrin subtype 1 knock-out (TRPA1-/-), transient receptor potential vanilloid 1 knock-out (TRPV1-/-) and TRPA1-TRPV1 double-knockout mice (TRPAV-/-) in the presence and absence of L-NAME (an endothelial nitric oxide synthase inhibitor) and penitrem A [a big-conductance calcium gated (BKCa) channel inhibitor]. To further support our in-vivo data, murine coronary microvessels were isolated and cannulated for vasoreactivity studies. Furthermore, NO production from endothelial cells isolated from mouse aorta was also measured and immunocytochemical (ICC) studies were performed to show the intracellular localization of TRPA1 and TRPV1. Our in-vivo data shows that the characteristic propofol-induced depressor response is dependent on TRPA1-NO-BKCa pathway. Interestingly, vasoreactivity studies in isolated murine left anterior ascending (LAD) arteries demonstrate that TRPA1 and TRPV1 communicate with each other and propofol-induced vasodilation is dependent on both TRPA1 and TRPV1. Moreover our data also suggest that NO production and BK channel activation are the downstream mediators in this pathway. Finally, we demonstrate that NO production is attenuated in primary endothelial cells isolated from TRPAV-/- mice. ICC data also shows the co-localization of these channels in mouse aortic endothelial cells.
Conclusions: This is the first study which has shown that propofol-induced vasodilation involves TRPA1 in-vivo and also there is an implication of cross-talk between TRPA1 and TRPV1 in the coronary bed. Furthermore by understanding the mechanisms by which this anesthetic causes hypotension and coronary dilation will help to mitigate the potential harmful side-effects of anesthesia in patients with little cardiovascular reserve. This will in turn ensure a better and faster post-operative recovery in patients, especially benefiting those suffering from diabetes and other cardiovascular disorders.
Harris, Tanoya L. "Ouabain Regulates Caveolin-1 Vesicle Trafficking by a Src-Dependent Mechanism." University of Toledo Health Science Campus / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=mco1333732028.
Повний текст джерелаBarr, Larry A. "The Role of Calcium in the Regulation of Pathological Hypertrophy." Diss., Temple University Libraries, 2014. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/254617.
Повний текст джерелаPh.D.
Pathological hypertrophy leads to cardiac dysfunction and heart failure. It is not clearly defined how this process occurs in the cardiomyocyte, or how the pathology can be effectively treated. There are numerous processes that lead to pathological hypertrophy. We developed two models to study pathological hypertrophy and the role that Ca2+ plays. In one model, we administered clinical doses of the leukemia therapeutic drug imatinib to neonatal ventricular cardiomyocytes. This drug has recently been found to be cardiotoxic, and we set out to understand if Ca2+ is involved. In the second model, we developed mice with overexpression of the Ca2+ entrance channel, the L-type calcium channel (LTCC), which leads to pathological hypertrophy over time. We instituted a chronic exercise regimen on these mice to learn if physiological hypertrophy can ameliorate detrimental aspects of pathological hypertrophy. After cardiomyocytes were treated with imatinib, they expressed enhanced Ca2+ activity. Levels of atrial natriuretic peptide (ANP) were up, signifying pathological hypertrophy. We determined that Ca2+ was activating Calcineurin, leading to translocation of nuclear factor of activated T-cells (NFAT) into the nucleus, resulting in hypertrophy. This activity was blocked by Ca2+ and Calcineurin inhibitors. We concluded that imatinib causes Ca2+ induced pathological hypertrophy. When mice with LTCC overexpression were exercised, they exhibited enhanced cardiac function. They also had thicker septal walls and increased chamber diameter, hallmarks of physiological hypertrophy. Heart weight to body weight ratio was significantly higher after exercise. When isolated hearts were administered ischemia/reperfusion injury, the exercised hearts showed a significant improvement in recovery compared to sedentary LTCC overexpressed hearts. Calcium activity was enhanced at the cardiomyocyte level in both mouse lines of exercised mice. In conclusion, hearts with a pathological hypertrophic phenotype can enhance function and achieve cardioprotection through chronic exercise.
Temple University--Theses
Blatherwick, Eleanor Q. "Imaging mass spectrometry approaches for the detection and localisation of drug compounds and small molecules in tissue." Thesis, University of Warwick, 2013. http://wrap.warwick.ac.uk/57257/.
Повний текст джерелаКниги з теми "Molecular Physiology and Pharmacology"
1953-, Webb David J., ed. Molecular biology and pharmacology of the endothelins. New York: Springer, 1995.
Знайти повний текст джерелаKenakin, Terrence P. Molecular pharmacology: A short course. Cambridge, Mass: Blackwell Science, 1997.
Знайти повний текст джерелаMorad, Martin, Setsuro Ebashi, Wolfgang Trautwein, and Yoshihisa Kurachi, eds. Molecular Physiology and Pharmacology of Cardiac Ion Channels and Transporters. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-011-3990-8.
Повний текст джерелаBurch, Ronald M. Molecular biology and pharmacology of bradykinin receptors. Austin: R.G. Landes, 1993.
Знайти повний текст джерелаGlossmann, H. Methods in Pharmacology: Molecular and Cellular Biology of Pharmacological Targets. Boston, MA: Springer US, 1993.
Знайти повний текст джерелаVitamin D: Physiology, molecular biology, and clinical applications. 2nd ed. [New York]: Humana Press, 2010.
Знайти повний текст джерела1929-, Inoki Reizo, Kudō Teruo 1929-, and Olgart Leif 1938-, eds. Dynamic aspects of dental pulp: Molecular biology, pharmacology and pathophysiology. London: Chapman and Hall, 1990.
Знайти повний текст джерелаSternberg, Esther M., and Lewis L. Judd. Glucocorticoids and mood: Clinical manifestations, risk factors and molecular mechanisms. Boston, Mass: Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2009.
Знайти повний текст джерелаP, Czech Michael, ed. Molecular basis of insulin action. New York: Plenum Press, 1985.
Знайти повний текст джерелаNestler, Eric J. Molecular Neuropharmacology. New York: McGraw-Hill, 2008.
Знайти повний текст джерелаЧастини книг з теми "Molecular Physiology and Pharmacology"
Benn, Caroline. "Physiology and Treatment of Hyperuricemia and Gout." In Encyclopedia of Molecular Pharmacology, 1234–43. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-57401-7_10042.
Повний текст джерелаBenn, Caroline. "Physiology and Treatment of Hyperuricemia and Gout." In Encyclopedia of Molecular Pharmacology, 1–10. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-21573-6_10042-1.
Повний текст джерелаCui, Meng, Lucas Cantwell, Andrew Zorn, and Diomedes E. Logothetis. "Kir Channel Molecular Physiology, Pharmacology, and Therapeutic Implications." In Pharmacology of Potassium Channels, 277–356. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/164_2021_501.
Повний текст джерелаBorn, W., and J. A. Fischer. "Calcitonin Gene Products: Molecular Biology, Chemistry, and Actions." In Physiology and Pharmacology of Bone, 569–616. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77991-6_16.
Повний текст джерелаMartin, T. J. "Parathyroid Hormone-Related Protein: Molecular Biology, Chemistry, and Actions." In Physiology and Pharmacology of Bone, 617–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77991-6_17.
Повний текст джерелаFrench, Deborah. "The Molecular Pathology of Glanzmann’s Thrombasthenia." In Handbook of Platelet Physiology and Pharmacology, 394–423. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-5049-5_18.
Повний текст джерелаJohnston, Graham A. R. "Molecular Biology, Pharmacology, and Physiology of GABAC Receptors." In The GABA Receptors, 297–323. Totowa, NJ: Humana Press, 1997. http://dx.doi.org/10.1007/978-1-4757-2597-1_11.
Повний текст джерелаLehmann-Horn, F., and R. Rüdel. "Molecular pathophysiology of voltage-gated ion channels." In Reviews of Physiology, Biochemistry and Pharmacology, 195–268. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/3-540-61343-9_9.
Повний текст джерелаBenos, Dale J., Sonia Cunningham, R. Randall Baker, K. Beth Beason, Youngsuk Oh, and Peter R. Smith. "Molecular characteristics of amiloride-sensitive sodium channels." In Reviews of Physiology, Biochemistry and Pharmacology, 31–113. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/bfb0036122.
Повний текст джерелаPette, Dirk, and Robert S. Staron. "Cellular and molecular diversities of mammalian skeletal muscle fibers." In Reviews of Physiology, Biochemistry and Pharmacology, 1–76. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/3540528806_3.
Повний текст джерелаТези доповідей конференцій з теми "Molecular Physiology and Pharmacology"
Lu, Yi-Ru, and Yu-Hsin Chiu. "Physiology and pharmacology of ATP-releasing pannexin 1 channels." In THE 4TH INTERNATIONAL CONFERENCE ON LIFE SCIENCE AND TECHNOLOGY (ICoLiST). AIP Publishing, 2023. http://dx.doi.org/10.1063/5.0112732.
Повний текст джерелаJerde, Travis J., and Stephen Y. Nakada. "The Role of Pharmacology in Ureteral Physiology and Expulsive Therapy." In RENAL STONE DISEASE: 1st Annual International Urolithiasis Research Symposium. AIP, 2007. http://dx.doi.org/10.1063/1.2723585.
Повний текст джерелаOrdu, Y., J. Augustin, E. V. Hodenberg, V. Bode, and J. Harenberg. "COMPARATIVE CLINICAL PHARMACOLOGY OF LOW MOLECULAR WEIGHT HEPARINS IN MAN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643228.
Повний текст джерелаBielby-Clarke, Keren. "0161 Enhancing Anatomy, Physiology And Pharmacology Teaching In A New Undergraduate Pharmacy Curriculum Using Simulation Technology." In Association for Simulated Practice in Healthcare Annual Conference 11–13 November 2014 Abstracts. The Association for Simulated Practice in Healthcare, 2014. http://dx.doi.org/10.1136/bmjstel-2014-000002.184.
Повний текст джерела"Anti-tumor Molecular Mechanism of Steroidal Drugs Based on Network Pharmacology." In 2018 3rd International Conference on Life Sciences, Medicine, and Health. Francis Academic Press, 2018. http://dx.doi.org/10.25236/iclsmh.18.020.
Повний текст джерелаOuyang, Xinyao. "Molecular mechanism of catalpol in the treatment of diabetes mellitus based on network pharmacology and molecular docking." In 3RD INTERNATIONAL CONFERENCE ON FRONTIERS OF BIOLOGICAL SCIENCES AND ENGINEERING (FBSE 2020). AIP Publishing, 2021. http://dx.doi.org/10.1063/5.0048403.
Повний текст джерелаBarth, Connor W., Lei G. Wang, Antonio Montaño, Alexander L. Antaris, Jonathan M. Sorger, and Summer L. Gibbs. "Pharmacology of near infrared nerve-specific fluorophores for fluorescence-guided nerve sparing surgical procedures." In Molecular-Guided Surgery: Molecules, Devices, and Applications VII, edited by Summer L. Gibbs, Brian W. Pogue, and Sylvain Gioux. SPIE, 2021. http://dx.doi.org/10.1117/12.2583265.
Повний текст джерелаPiermarini, Peter M. "The molecular physiology of inward rectifier potassium (Kir) channels in mosquitoes." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93039.
Повний текст джерелаTekutskaya, E., I. Raybova, and Lyubov Ramazanovna Gusaruk. "THE DEGREE OF OXIDATIVE DAMAGE TO DNA IN VITRO AS A MOLECULAR PREDICTOR OF DISORDERS CAUSED BY EPIGENETIC AND EXOGENOUS FACTORS." In NEW TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2021. http://dx.doi.org/10.47501/978-5-6044060-1-4.49.
Повний текст джерелаKawachi, N., N. Suzui, S. Ishii, S. Ito, N. S. Ishioka, K. Kikuchi, T. Tsukamoto, T. Kusakawa, and F. Fujimaki. "Molecular imaging for plant physiology: Imaging of carbon translocation to sink organs." In 2009 IEEE Nuclear Science Symposium and Medical Imaging Conference (NSS/MIC 2009). IEEE, 2009. http://dx.doi.org/10.1109/nssmic.2009.5402366.
Повний текст джерелаЗвіти організацій з теми "Molecular Physiology and Pharmacology"
Tabita, F. R. Summer Workshop: Molecular Basis, Physiology and Diversity of Microbial Adaptation. Office of Scientific and Technical Information (OSTI), May 2002. http://dx.doi.org/10.2172/836588.
Повний текст джерелаHeven Sze. Regulating Intracellular Calcium in Plants: From Molecular Genetics to Physiology. Office of Scientific and Technical Information (OSTI), June 2008. http://dx.doi.org/10.2172/932554.
Повний текст джерелаUnsworth, Nancy. The Physiology and Molecular Biology of Iron Nutrition for Cyanobacteria. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.6413.
Повний текст джерелаZeikus, J. G. Molecular Physiology of Succinic Acid Based Fermentation In Anaerobes: Control of Chemical Yield by CO2 Fixation and Electron Donors. Office of Scientific and Technical Information (OSTI), May 2003. http://dx.doi.org/10.2172/1183577.
Повний текст джерелаHulata, Gideon, Thomas D. Kocher, Micha Ron, and Eyal Seroussi. Molecular Mechanisms of Sex Determination in Cultured Tilapias. United States Department of Agriculture, October 2010. http://dx.doi.org/10.32747/2010.7697106.bard.
Повний текст джерелаLaBonte, Don, Etan Pressman, Nurit Firon, and Arthur Villordon. Molecular and Anatomical Characterization of Sweetpotato Storage Root Formation. United States Department of Agriculture, December 2011. http://dx.doi.org/10.32747/2011.7592648.bard.
Повний текст джерелаBloch, Guy, Gene E. Robinson, and Mark Band. Functional genomics of reproduction and division of labor in a key non-Apis pollinator. United States Department of Agriculture, January 2011. http://dx.doi.org/10.32747/2011.7699867.bard.
Повний текст джерелаFait, Aaron, Grant Cramer, and Avichai Perl. Towards improved grape nutrition and defense: The regulation of stilbene metabolism under drought. United States Department of Agriculture, May 2014. http://dx.doi.org/10.32747/2014.7594398.bard.
Повний текст джерелаSavaldi-Goldstein, Sigal, and Siobhan M. Brady. Mechanisms underlying root system architecture adaptation to low phosphate environment. United States Department of Agriculture, January 2015. http://dx.doi.org/10.32747/2015.7600024.bard.
Повний текст джерелаLers, Amnon, Majid R. Foolad, and Haya Friedman. genetic basis for postharvest chilling tolerance in tomato fruit. United States Department of Agriculture, January 2014. http://dx.doi.org/10.32747/2014.7600014.bard.
Повний текст джерела