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Статті в журналах з теми "Metabolic derangements"
Allan, Russell, and Cristopher Foster. "Problem-Based Review: A patient with metabolic acidosis." Acute Medicine Journal 11, no. 4 (October 1, 2012): 251–56. http://dx.doi.org/10.52964/amja.0587.
Повний текст джерелаGaboury, Cynthia L., Norman K. Hollenberg, Paul N. Hopkins, Roger Williams, and Gordon H. Williams. "Metabolic derangements in nonmodulating hypertension*." American Journal of Hypertension 8, no. 9 (September 1995): 870–75. http://dx.doi.org/10.1016/0895-7061(95)00160-q.
Повний текст джерелаPinder, Elizabeth M., Gurprit S. S. Atwal, Abraham A. Ayantunde, Sarah Khan, Mike Sokal, Tom McCulloch, and Simon L. Parsons. "Tumour Lysis Syndrome Occurring in a Patient with Metastatic Gastrointestinal Stromal Tumour Treated with Glivec (Imatinib Mesylate, Gleevec, STI571)." Sarcoma 2007 (2007): 1–5. http://dx.doi.org/10.1155/2007/82012.
Повний текст джерелаMuoio, Deborah M., and Christopher B. Newgard. "Obesity-Related Derangements in Metabolic Regulation." Annual Review of Biochemistry 75, no. 1 (June 2006): 367–401. http://dx.doi.org/10.1146/annurev.biochem.75.103004.142512.
Повний текст джерелаPuente-Maestu, Luis, Alberto Lázaro, and Blanca Humanes. "Metabolic derangements in COPD muscle dysfunction." Journal of Applied Physiology 114, no. 9 (May 1, 2013): 1282–90. http://dx.doi.org/10.1152/japplphysiol.00815.2012.
Повний текст джерелаGebreegziabher, Yohannes, Amgad N. Makaryus, John N. Makaryus, and Samy I. McFarlane. "Heart failure: metabolic derangements and therapeutic rationale." Expert Review of Cardiovascular Therapy 5, no. 2 (March 2007): 331–43. http://dx.doi.org/10.1586/14779072.5.2.331.
Повний текст джерелаYadav, Hariom. "Whole Grains in Amelioration of Metabolic Derangements." Journal of Nutritional Health & Food Science 4, no. 4 (October 24, 2016): 1–11. http://dx.doi.org/10.15226/jnhfs.2016.00173.
Повний текст джерелаMazzoccoli, Gianluigi, Calogero Longhitano, and Manlio Vinciguerra. "Cardio-Hepatic Metabolic Derangements and Valproic Acid." Current Clinical Pharmacology 9, no. 2 (April 2014): 165–70. http://dx.doi.org/10.2174/1574884708999140101144839.
Повний текст джерелаBaker, Susan S., Ellen Dwyer, and Patt Queen. "Metabolic Derangements in Children Requiring Parenteral Nutrition." Journal of Parenteral and Enteral Nutrition 10, no. 3 (May 1986): 279–81. http://dx.doi.org/10.1177/0148607186010003279.
Повний текст джерелаGovindarajan, Gurushankar, Melvin R. Hayden, Shawna A. Cooper, Said Daibes Figueroa, Lixin Ma, Timothy J. Hoffman, Craig S. Stump, and James R. Sowers. "Metabolic Derangements in the Insulin‐Resistant Heart." American Journal of Geriatric Cardiology 15, no. 6 (November 2006): 102–6. http://dx.doi.org/10.1111/j.1559-4564.2006.05683.x.
Повний текст джерелаДисертації з теми "Metabolic derangements"
Taveroff, Arlene. "Metabolic derangements following bone marrow transplantation : an integrated analysis." Thesis, McGill University, 1989. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=74259.
Повний текст джерелаFitzgerald, Danielle. "Pasture-associated metabolic derangements of horses: Pathogenesis and identification." Thesis, Queensland University of Technology, 2019. https://eprints.qut.edu.au/134255/1/Danielle%20Maree_Fitzgerald_Thesis.pdf.
Повний текст джерелаLi, Shiying, and 李诗盈. "Maternal green tea epigallocatechin gallate supplementation counteracts high-fat diet-induced metabolic derangements in dams andtheir male offspring: a programming effect." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B47156132.
Повний текст джерелаpublished_or_final_version
Biological Sciences
Doctoral
Doctor of Philosophy
Romrig, Franziska [Verfasser], Melek Canan [Akademischer Betreuer] Arkan-Greten, Bernhard [Akademischer Betreuer] Küster, and Martin [Akademischer Betreuer] Klingenspor. "Metabolic Derangements in Pancreatic Cancer / Franziska Romrig. Gutachter: Bernhard Küster ; Melek Canan Arkan-Greten ; Martin Klingenspor. Betreuer: Melek Canan Arkan-Greten." München : Universitätsbibliothek der TU München, 2013. http://d-nb.info/1034952099/34.
Повний текст джерелаReinhart, Jennifer M. "Derangements of tonicity and implications for veterinary patients." Thesis, Kansas State University, 2014. http://hdl.handle.net/2097/19761.
Повний текст джерелаDepartment of Clinical Sciences
Thomas Schermerhorn
Tonicity is property of a solution that is defined as the total effective (impermeable) osmole concentration that drives fluid movement across a semipermeable membrane via osmosis. Tonicity is related to but distinct from solution osmolality, which is a summation of all solute concentrations, regardless of the solute membrane permeability. In the mammalian body, tonicity is tightly regulated at both a cellular and systemic level; tonic derangements cause rapid change in cell and tissue volume leading to significant dysfunction. Input from the central nervous, circulatory, endocrine, gastrointestinal, and urinary systems are integral to osmoregulation, so many diseases in veterinary medicine are associated with tonicity disorders. However, because the homeostatic mechanisms that control tonicity overlap with those regulating electrolyte and acid-base balance as well as hydration and vascular volume, tonic consequences of disease can be difficult to isolate. Understanding of disease-associated changes in tonicity is further complicated by the fact that the tonic contributions of many solutes that accumulate in disease are unknown. Additionally, direct assessment of tonicity is difficult because tonicity is not just a physiochemical property, but it implies a physiologic effect. Thus, simple summation of osmole concentrations is an inadequate measurement of tonicity. The following report includes three studies investigating various aspects of tonicity as it applies to veterinary patients. Chapter 2 reports a study that examines the tonic effects of ketoacids and lactate using two different in vitro red blood cell assays. Results demonstrated that the ketoacids, beta-hydroxybutyrate and acetoacetate, behave as ineffective osmoles while the tonic behavior of lactate is variable, implying a more complex cellular handling of this anion. Two additional studies examine whether the mean corpuscular volume difference (dMCV) is a novel clinical marker for hypertonicity in dogs. Results of separate retrospective (Chapter 3) and prospective (Chapter 4) studies provide evidence that dMCV is a useful clinical marker for hypertonicity in dogs.
Sturney, Sharon C. "Breath biomarkers of inflammation, infection and metabolic derangement in the intensive care unit." Thesis, University of Nottingham, 2015. http://eprints.nottingham.ac.uk/28648/.
Повний текст джерелаAbolghasemi, Armita. "Lipid mediators as regulators of lipid and energy metabolism during energy balance derangement in animal models." Doctoral thesis, Université Laval, 2020. http://hdl.handle.net/20.500.11794/67011.
Повний текст джерелаLes facteurs environnementaux jouent un rôle clé dans le développement du syndrome métabolique et de l'obésité, qui sont maintenant de véritables épidémies soulevant des problèmes de santé publique. Les excès de graisse corporelle dans l'obésité et la perte de masse grasse et maigre dans les conditions de dénutrition ou d'anorexie mentale sont le résultat d'un déséquilibre énergétique. Par conséquent, le maintien de l'équilibre énergétique est crucial à la fois pour la prévention de l'obésité et pour le traitement de l'anorexie mentale et d'autres formes de dénutrition. Les signaux lipidiques tels que ceux médiés par les médiateurs des endocannabinoidomes sont profondément impliqués dans le contrôle du métabolisme énergétique. Dans cette thèse, au sein de deux projets différents, nous avons étudié comment différents facteurs environnementaux, y compris la restriction calorique, l'activité physique, la supplémentation en vitamine D et les médicaments antipsychotiques, peuvent conduire à une modification du métabolisme énergétique par la modulation de la signalisation des endocannabinoïdomes. Les résultats des travaux expérimentaux montrent comment les différentes conditions étudiées provoquent des changements dans les niveaux tissulaires du médiateur lipidique endocannabinoïdome ainsi que dans l'expression de leurs récepteurs et enzymes métaboliques, ce qui peut contribuer aux changements observés de la masse grasse corporelle et du métabolisme énergétique au sein des modèles. Nous concluons que les conditions étudiées peuvent provoquer des changements dans le bilan énergétique par altération de l'endocannabinoidome.
Environmental factors play a key role in the development of obesity-induced metabolic syndrome and obesity, which are now true epidemics raising public health concerns. Both excess body fat in obesity, and fat and lean mass loss in undernutrition conditions or anorexia nervosa, are the result of energy imbalance. Therefore, maintaining energy balance is crucial for both the prevention of obesity and the treatment of anorexia nervosa and other forms of undernutrition. Lipid signals such as those mediated by endocannabinoidome mediators are deeply involved in the control of energy metabolism. In this thesis, within two different projects, we studied how different environmental factors including calorie restriction, physical activity, vitamin D supplementation and antipsychotic drugs, may lead to energy metabolism modification through modulation of the endocannabinoidome signaling. The results of the experimental work show how the different studied conditions cause changes in the tissue levels of endocannabinoidome lipid mediator as well as in the expression of their receptors and metabolic enzymes, which may contribute to the observed changes in body fat mass and energy metabolism within the models. We conclude that the investigated conditions may cause changes in energy balance through alteration of the endocannabinoidome.
"Lactate, a novel marker of disordered energy homeostasis in the metabolic derangements of obesity." THE JOHNS HOPKINS UNIVERSITY, 2009. http://pqdtopen.proquest.com/#viewpdf?dispub=3340035.
Повний текст джерелаYun, Chun-Ho, and 惲純和. "The Association among Metabolic Derangements, Atherosclerosis and Regional-Specific Visceral Fat Tissue Quantified by Multiple Detector Computed Tomography." Thesis, 2016. http://ndltd.ncl.edu.tw/handle/07816281421667517219.
Повний текст джерела國立陽明大學
生物醫學影像暨放射科學系
104
Excessive visceral adipose tissue accumulation is strongly associated with metabolic derangements including type 2 diabetes, hyperlipidemia, hypertension, cardiovascular disease risk and its factors. Visceral adipose tissue has been demonstrated to contribute to systemic inflammation and to be more relevant to unfavorable clinical outcomes. However, the most popular method, anthropometric measures of waist circumference for central obesity is relatively clinical convenient but may not precisely reflect the extent of visceral adipose tissue due to subcutaneous adipose tissue. Recently, multi-detector computed tomography (MDCT) provided a reliable and accurate method in assessing visceral adipose tissue. It is noninvasive and could quantify fat volume by semi-automatic segmentation technique to select all pixels in fat density of the region of interest. Current research studies of visceral adipose tissue quantified by MDCT mainly focused on pericardial fat (PCF) and thoracic peri-aortic fat (TAT). In 2008, the first article of PCF of MDCT published by Harvard medical school was from Framingham study. The definition of PCF is adipose tissue directly adjacently to myocardiaum and coronary arteries as well as confined by pericardium, isolated from other tissue and organs of thorax. TAT is adipose tissue surrounding a segment of thoracic descending aorta, not locally confined in thorax. Up-to-date, most published research studies discussed the relationship between these two regional visceral adipose tissue, coronary atherosclerosis and metabolic disease. However, is there strong association among different regional specific adipose tissue, pre-diabetes and carotid intimamedia thickness? Is the volume changes of PCF, TAT with aging? There is no definite conclusion now. In our study, in order to further assess the relationship between carotid atherosclerosis and regional specific adipose tissue, we created a novel way to define "peri-aortic root adipose fat" (PARF). We have measured the volume of adipose tissue surrounding a short segment of ascending aortic root of MDCT images and evaluated the influence of this regional specific adipose tissue on carotid morphology. The purposes in the present thesis study are: 1) To investigate the clinical significance, biological effects, and related cardiometabolic derangements of body-site specific visceral adipose tissue accumulation in a relatively healthy population. 2) To evaluate the degree of pericardial and thoracic peri-aortic adipose tissue, when quantified by multi-detector computed tomography, differs significantly in a normal, pre-diabetic, and overtly diabetic population. 3) To describe the relationship between a novel measurement of peri-arotic root fat and ultrasound measures of carotid artery remodeling. In our study, we have shown that age-related body-site specific adipose tissue accumulation may have distinct biological effects. Compared to other measures of adiposity, thoracic peri-aortic tissue is more closely associated with cardiometabolic risk and subclinical atherosclerosis in a relatively healthy population. Besides, pre-diabetic and diabetic subjects, compared to normoglycemia, were associated with significantly higher pericardial and peri-aortic adipose tissue burden. Visceral fat accumulation adjacent to the thoracic descending aorta seemed to exert a significant impact on insulin resistance and systemic inflammation. Peri-aortic root fat is associated with carotid intima-media thickness, even after adjustment for cardiometabolic risks, age and coronary atherosclerosis.
HSUAN, CHIA, and 楊佳璇. "To investigate the role of vit D3 on metabolic derangement of patient with chronic kidney disease." Thesis, 2009. http://ndltd.ncl.edu.tw/handle/73s9t9.
Повний текст джерела靜宜大學
食品營養研究所
98
The important relationship among serum Vitamin D3(25(OH)D3)、metabolic syndrome and insulin resistance in patient with chronic kidney disease(CKD)was investigated.It’s might be adjust the physiological metabolism and abnormalities of lipid metabolism are risk factor for the development of CKD.The aim of our study is to determine chronic kidney disease patients(CKD p’t) of serum Vitamin D3,whether abnormalities of lipid metabolism to cause the insulin resistence and the fat storeds. The experimental subject in 41 non-diabetes patient with CKD, tests for the time 12 weeks, by the questionnaire way collection clinical correlation data, illness medical history, 24hrs record the diet history、medicine use and anthropometric measurements, after the experiment ended, collects the urine and the hematologic analysis. Results showed that there was no significant relationship between sex, waistline, buttocks, body weight, BMI, smoke, activity, vitamins premix, sugar fasting, siastolic blood pressure and vitamin D3 concentration.Results also showed that Body weight、height、Hemoglobin、RBC、Vitamin D3 concentration、urine creatinine and the calories、carbohydrates、protein intake in the stage 3 of CKD p’t is higher than the stage 4、5 of of CKD p’t but the BUN、Creatine are lower than the stage 4、5 CKD p’t;Next, the Vitamin D3 concentration is greater than 25nmol/L,its Homa-IR、BUN、Creatine are to be lower than the concentration is smaller than 25 nmol/L,but the Vitamin D3 concentration is greater than 50 nmol/L,it’s blood sugar and the cholesterol is lower; In addition, we have also demonstrated that serum Vitamin D3 concentration and CKD are associated with insulin resistence and other components of the metabolic syndrom,such as triglyceride,and the CKD and insulin resistance is also related.In conclusion,we extrapolate that maintains the serum Vitamin D3 concentration In the non-diabetes CKD p’t to be possible to avoid the kidney function degree dropping、insulin resistance and abnormalities of lipid metabolism ,then further prevention metabolic syndrome.
Книги з теми "Metabolic derangements"
Hormonal and Metabolic Derangements in Renal Failure. S. Karger AG (Switzerland), 1986.
Знайти повний текст джерелаDierdorf, Stephen F. Introduction to Metabolic and Endocrine Diseases. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0024.
Повний текст джерелаCheng, Ning, Susan A. Masino, and Jong M. Rho. Metabolic Therapy for Autism Spectrum Disorder and Comorbidities. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0014.
Повний текст джерелаJordan, Nerissa. Non-metastatic neurological manifestations of malignancy. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0238.
Повний текст джерелаFranzese, Adriana, Enza Mozzillo, and Francesco Maria Rosanio. Glucose Metabolism Derangements in Pediatric Age. Cambridge Scholars Publishing, 2022.
Знайти повний текст джерелаMeier, Petra M., and Thomas O. Erb. Craniosynostosis and Apert Syndrome. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0021.
Повний текст джерелаNelson, Jonathon, and Franklyn P. Cladis. Pediatric Liver Transplantation. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0038.
Повний текст джерелаRuskin, David N. Metabolic Therapy and Pain. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0022.
Повний текст джерелаTaljanovic, Mihra S., Imran M. Omar, Kevin B. Hoover, and Tyson S. Chadaz, eds. Musculoskeletal Imaging Volume 1. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190938161.001.0001.
Повний текст джерелаTaljanovic, Mihra S., Imran M. Omar, Kevin B. Hoover, and Tyson S. Chadaz, eds. Musculoskeletal Imaging Volume 2. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190938178.001.0001.
Повний текст джерелаЧастини книг з теми "Metabolic derangements"
Jaworski, Nikki, and Ansgar Brambrink. "Electrolyte and Metabolic Derangements." In Handbook of Neurocritical Care, 13–35. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6842-5_2.
Повний текст джерелаCiardella, F. "Effects of Nutritional Treatment on Hormonal and Metabolic Derangements of the Uremic Syndrome." In Nutritional Treatment of Chronic Renal Failure, 241–54. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-1583-4_27.
Повний текст джерелаGupta, Diptesh, and Madhukar Misra. "Metabolic Derangements Seen in Chronic Kidney Disease and End-Stage Renal Disease Patients." In Hemodialysis, 113–19. Basel: KARGER, 2011. http://dx.doi.org/10.1159/000327232.
Повний текст джерелаde la Monte, Suzanne M. "The Full Spectrum of Alzheimer’s Disease Is Rooted in Metabolic Derangements That Drive Type 3 Diabetes." In Advances in Experimental Medicine and Biology, 45–83. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-3540-2_4.
Повний текст джерелаKawamata, Tatsuro, Y. Katayama, N. Tsuji, and H. Nishimoto. "Metabolic derangements in interstitial brain edema with preserved blood flow: selective vulnerability of the hippocampal CA3 region in rat hydrocephalus." In Brain Edema XII, 545–47. Vienna: Springer Vienna, 2003. http://dx.doi.org/10.1007/978-3-7091-0651-8_111.
Повний текст джерелаLeverve, X. M. "Derangements in Cellular Oxygen Metabolism." In Mechanisms of Organ Dysfunction in Critical Illness, 52–68. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56107-8_4.
Повний текст джерелаMorii, H., K. Matsumoto, G. Endo, S. Uematsu, T. Konishi, and K. Maekawa. "Clinical Presentation of Derangements of Mineral Metabolism." In Calcium in Internal Medicine, 3–12. London: Springer London, 2002. http://dx.doi.org/10.1007/978-1-4471-0667-8_1.
Повний текст джерелаSchiraldi, F., A. R. Derosa, and F. Paladino. "Metabolic Disorders and Electrolyte Derangement during Hypoperfusion Syndrome." In Anaesthesia, Pain, Intensive Care and Emergency Medicine - A.P.I.C.E., 691–96. Milano: Springer Milan, 1998. http://dx.doi.org/10.1007/978-88-470-2278-2_75.
Повний текст джерелаRiley, P. A. "Derangements of Cellular Metabolism in The Pre-Malignant Syndrome." In Free Radicals: from Basic Science to Medicine, 199–205. Basel: Birkhäuser Basel, 1993. http://dx.doi.org/10.1007/978-3-0348-9116-5_17.
Повний текст джерелаYanagihara, Takehiko. "Metabolic Derangement and Cell Damage in Cerebral Ischemia with Emphasis on Protein and Nucleic Acid Metabolism." In Advances in Brain Resuscitation, 77–98. Tokyo: Springer Japan, 1991. http://dx.doi.org/10.1007/978-4-431-68538-8_5.
Повний текст джерелаТези доповідей конференцій з теми "Metabolic derangements"
Fessel, Joshua P., Rizwan Hamid, Eric D. Austin, Yuji Tada, Bryan Wittmann, Anna Hemnes, and James West. "Metabolomic Analysis Reveals Widespread Metabolic Derangements In Pulmonary Arterial Hypertension." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6512.
Повний текст джерелаLeduc-Gaudet, J. P., K. Miguez, J. Faitg, F. Eduardo Broering, O. Reynaud, D. Mayaki, L. Huck, M. Sandri, G. Gouspillou, and S. N. A. Hussain. "The Influence of Autophagy Inhibition on Sepsis-Induced Skeletal Muscle Wasting, Metabolic Derangements and Mortality." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2377.
Повний текст джерела