Готові списки джерел за темами / Marmoset monkeys

Добірка наукової літератури з теми "Marmoset monkeys"

Автор: Grafiati
Опубліковано: 10 грудня 2022
Оновлено: 28 січня 2023

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Статті в журналах з теми "Marmoset monkeys"

1

LaBonte, Jason A., Gregory J. Babcock, Trushar Patel, and Joseph Sodroski. "Blockade of HIV-1 Infection of New World Monkey Cells Occurs Primarily at the Stage of Virus Entry." Journal of Experimental Medicine 196, no. 4 (August 12, 2002): 431–45. http://dx.doi.org/10.1084/jem.20020468.

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HIV-1 naturally infects chimpanzees and humans, but does not infect Old World monkeys because of replication blocks that occur after virus entry into the cell. To understand the species-specific restrictions operating on HIV-1 infection, the ability of HIV-1 to infect the cells of New World monkeys was examined. Primary cells derived from common marmosets and squirrel monkeys support every phase of HIV-1 replication with the exception of virus entry. Efficient HIV-1 entry typically requires binding of the viral envelope glycoproteins and host cell receptors, CD4 and either CCR5 or CXCR4 chemokine receptors. HIV-1 did not detectably bind or utilize squirrel monkey CD4 for entry, and marmoset CD4 was also very inefficient compared with human CD4. A marmoset CD4 variant, in which residues 48 and 59 were altered to the amino acids found in human CD4, supported HIV-1 entry efficiently. The CXCR4 molecules of both marmosets and squirrel monkeys supported HIV-1 infection, but the CCR5 proteins of both species were only marginally functional. These results demonstrate that the CD4 and CCR5 proteins of New World monkeys represent the major restriction against HIV-1 replication in these primates. Directed adaptation of the HIV-1 envelope glycoproteins to common marmoset receptors might allow the development of New World monkey models of HIV-1 infection.
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2

Woller, Michael J., Pam L. Tannenbaum, Nancy J. Schultz-Darken, Bruce D. Eshelman, and David H. Abbott. "Pulsatile gonadotropin-releasing hormone release from hypothalamic explants of male marmoset monkeys compared with male rats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 298, no. 1 (January 2010): R70—R78. http://dx.doi.org/10.1152/ajpregu.00193.2009.

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The present study was conducted to quantify in vitro gonadotropin-releasing hormone (GnRH) release parameters in the male marmoset. We established primary cultures of marmoset hypothalamic tissues for ∼2 days (marmosets) to assess GnRH release profiles in vitro in hypothalamic explants from testis-intact and gonadectomized males. Pulsatile GnRH release profiles were readily demonstrated from in vitro hypothalamic explants isolated from adult male marmoset monkeys. Gonadectomy of male marmosets resulted in elevated mean GnRH and pulse amplitude from hypothalamic explants on the 1st day of culture ( day 0). GnRH pulse amplitude increased by day 2 in ∼67% of hypothalamic explants from testis-intact marmosets, suggesting release from an endogenous regulator of GnRH. We also measured GnRH release profiles in vitro in hypothalamic explants from testis-intact and gonadectomized rats. Male rats showed no changes in any concentration or frequency release parameters for GnRH following gonadectomy or during successive days in culture. The present study represents a unique examination of GnRH release from male marmoset monkey hypothalamic tissue and compares release dynamics directly with those obtained from male rat, suggesting a species difference in feedback regulation of GnRH release.
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3

Takahashi, N., S. Suda, T. Shinki, N. Horiuchi, Y. Shiina, Y. Tanioka, H. Koizumi та T. Suda. "The mechanism of end-organ resistance to 1α,25-dihydroxycholecalciferol in the common marmoset". Biochemical Journal 227, № 2 (15 квітня 1985): 555–63. http://dx.doi.org/10.1042/bj2270555.

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The common marmoset, a New World monkey, requires a large amount of cholecalciferol (110 i.u./day per 100g body wt.) to maintain its normal growth. In a previous report, we demonstrated that the circulating levels of 1 alpha, 25-dihydroxycholecalciferol [1 alpha,25(OH)2D3] in the marmosets are much higher than those in rhesus monkeys and humans, but the marmosets are not hypercalcaemic [Shinki, Shiina, Takahashi, Tanioka, Koizumi & Suda (1983) Biochem. Biophys. Res. Commun. 14, 452-457]. To compare the effect of the daily intake of cholecalciferol, two rhesus monkeys were given a large amount of cholecalciferol (900 i.u./day per 100g body wt). Their serum levels of calcium, 25-hydroxycholecalciferol and 24R,25-dihydroxycholecalciferol were markedly elevated, but the serum 1 alpha,25(OH)2D3 levels remained within a range similar to those in the rhesus monkeys fed the normal diet (intake of cholecalciferol 5 i.u./day per 100g body wt). Intestinal cytosols prepared from both monkeys contained similar 3.5 S macromolecules to which 1 alpha,25(OH)2D3 was bound specifically. However, the cytosols from the marmosets contained only one-sixth as many 1 alpha,25(OH)2D3 receptors as those from the rhesus monkeys. Furthermore, the activity of the 1 alpha,25(OH)2D3-receptor complex in binding to DNA-cellulose was very low in the marmosets. These results suggest that the marmoset possesses an end-organ resistance to 1 alpha,25(OH)2D3 and is a useful animal model for studying the mechanism of vitamin D-dependent rickets, type II.
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4

Hori, Yuki, Justine C. Cléry, Janahan Selvanayagam, David J. Schaeffer, Kevin D. Johnston, Ravi S. Menon, and Stefan Everling. "Interspecies activation correlations reveal functional correspondences between marmoset and human brain areas." Proceedings of the National Academy of Sciences 118, no. 37 (September 7, 2021): e2110980118. http://dx.doi.org/10.1073/pnas.2110980118.

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The common marmoset has enormous promise as a nonhuman primate model of human brain functions. While resting-state functional MRI (fMRI) has provided evidence for a similar organization of marmoset and human cortices, the technique cannot be used to map the functional correspondences of brain regions between species. This limitation can be overcome by movie-driven fMRI (md-fMRI), which has become a popular tool for noninvasively mapping the neural patterns generated by rich and naturalistic stimulation. Here, we used md-fMRI in marmosets and humans to identify whole-brain functional correspondences between the two primate species. In particular, we describe functional correlates for the well-known human face, body, and scene patches in marmosets. We find that these networks have a similar organization in both species, suggesting a largely conserved organization of higher-order visual areas between New World marmoset monkeys and humans. However, while face patches in humans and marmosets were activated by marmoset faces, only human face patches responded to the faces of other animals. Together, the results demonstrate that higher-order visual processing might be a conserved feature between humans and New World marmoset monkeys but that small, potentially important functional differences exist.
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5

Tardif, Suzette, and Corinna Ross. "MARMOSET MONKEYS AS A MODEL OF AGING." Innovation in Aging 3, Supplement_1 (November 2019): S8—S9. http://dx.doi.org/10.1093/geroni/igz038.028.

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Abstract Interest in the New World Monkey, the common marmoset, as a nonhuman primate aging model is growing. Because marmosets have a fast maturation and short life span compared with more commonly used Old World monkey models, the aging research community began to explore the potential of this model species. In addition, the relative ease with which marmosets can be bred in a barrier environment enhances their value as a life-span model. Since that time, efforts to better define what aging actually looks like in marmosets has intensified. Important findings of the past decade include: (1) a refined definition of lifespan in this species and what affects age-specific survival; (2) assessments of age-related pathological changes; (3) development of functional phenotyping relevant to aging, such as activiyy, strength, body composition, cytokine profiling; (4) support of studies using the marmoset as a preclinical model to test intervention that may modulate the aging process.
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6

Žagar, Žiga, Klemen Šmalc, Pia Kristina Primožič, Pavel Kvapil, and Ana Nemec. "Oral and Dental Examinations Findings in 15 Zoo Bolivian Squirrel Monkeys (Saimiri boliviensis) and Black-Tufted Marmosets (Callithrix penicillata)." Journal of Veterinary Dentistry 38, no. 2 (June 2021): 67–74. http://dx.doi.org/10.1177/08987564211041781.

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As part of an annual wellness evaluation, we performed oral and dental examination under general anesthesia in 7 zoo Bolivian squirrel monkeys aged 10 and 15 years, and 8 zoo black-tufted marmosets aged between 1 and 7 years. No oral discomfort was observed in any animal prior to the procedure. Apart from dilacerated roots of second mandibular incisor teeth in Bolivian squirrel monkeys and one case of presumably odontodysplasia in a black-tufted marmoset, no major variations in number and shape of the present teeth and roots were revealed. All 15 animals had gingivitis, but periodontitis was only diagnosed in 3 black-tufted marmosets. Most commonly diagnosed dental pathology in Bolivian squirrel monkeys was attrition/abrasion, affecting 11.9% of all teeth, followed by caries, which was only diagnosed in older animals. Altogether 8 fractured teeth were diagnosed in Bolivian squirrel monkeys only, with root fracture being the most common type, followed by complicated crown fracture and complicated crown-root fracture. Radiographic signs of endodontic disease were found in 10 teeth in Bolivian squirrel monkeys and in one nonvital tooth with intact crown in a black-tufted marmoset. We associated high occurrence of caries in the older Bolivian squirrel monkeys with their diet and saliva characteristics of these animals. Lack of any periodontitis in Bolivian squirrel monkeys may partially be attributed to limitations of radiography technique, although squirrel monkeys appear to be far less susceptible to naturally occurring periodontitis than marmosets.
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7

Pattison, J. Christina, David H. Abbott, Wendy Saltzman, Ann D. Nguyen, Gary Henderson, Hongwu Jing, Christopher R. Pryce, Amy J. Allen, Alan J. Conley, and Ian M. Bird. "Male Marmoset Monkeys Express an Adrenal Fetal Zone at Birth, But Not a Zona Reticularis in Adulthood." Endocrinology 146, no. 1 (January 1, 2005): 365–74. http://dx.doi.org/10.1210/en.2004-0689.

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Neonatal human males produce high levels of dehydroepiandrosterone (DHEA) and its sulfo-conjugated form (DS) that decline within a few months of birth, due to regression of the adrenal fetal zone (FZ). Adult male humans and rhesus monkeys produce C19 steroids in abundance from the adrenal zona reticularis (ZR). Male marmoset monkeys produce DS at birth, but unlike humans and rhesus monkeys, do not produce comparable amounts of DHEA and DS in adulthood. To determine whether male marmosets express a functional ZR in adulthood, we examined adult and neonatal male marmosets for the presence of a ZR and FZ, respectively. Exogenous ACTH failed to stimulate DHEA or DS in adults, and dexamethasone treatment failed to suppress DHEA and DS, although cortisol levels changed as expected. In steroidogenic tissues, the key proteins necessary to synthesize C19 steroids from pregnenolone are P450c17, 3β-hydroxysteroid dehydrogenase (3β-HSD), nicotinamide adenine dinucleotide phosphate (reduced) oxido-reductase cytochrome P450 (reductase), and cytochromeb5 (cytb5). Adult adrenal cross sections showed P450c17 and reductase protein expression throughout the cortex but showed no expected decrease in 3β-HSD and increase in cytb5 in the innermost region. Western analysis confirmed these data, demonstrating comparable P450c17 expression to rhesus monkeys, but not cytb5. HPLC analysis revealed similar 17α-hydroxylase action on pregnenolone for adult marmoset and rhesus adrenal microsomes but greatly diminished 17,20-lyase activity in marmosets. Neonatal marmoset adrenals exhibited staining indicative of a putative FZ (with P450c17, reduced 3β-HSD and increased cytb5). We conclude that neonatal marmosets exhibit a C19 steroid-secreting FZ similar to humans, but adult males fail to acquire a functional ZR.
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8

Ma, Liya, Janahan Selvanayagam, Maryam Ghahremani, Lauren K. Hayrynen, Kevin D. Johnston, and Stefan Everling. "Single-unit activity in marmoset posterior parietal cortex in a gap saccade task." Journal of Neurophysiology 123, no. 3 (March 1, 2020): 896–911. http://dx.doi.org/10.1152/jn.00614.2019.

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Abnormal saccadic eye movements can serve as biomarkers for patients with several neuropsychiatric disorders. The common marmoset ( Callithrix jacchus) is becoming increasingly popular as a nonhuman primate model to investigate the cortical mechanisms of saccadic control. Recently, our group demonstrated that microstimulation in the posterior parietal cortex (PPC) of marmosets elicits contralateral saccades. Here we recorded single-unit activity in the PPC of the same two marmosets using chronic microelectrode arrays while the monkeys performed a saccadic task with gap trials (target onset lagged fixation point offset by 200 ms) interleaved with step trials (fixation point disappeared when the peripheral target appeared). Both marmosets showed a gap effect, shorter saccadic reaction times (SRTs) in gap vs. step trials. On average, stronger gap-period responses across the entire neuronal population preceded shorter SRTs on trials with contralateral targets although this correlation was stronger among the 15% “gap neurons,” which responded significantly during the gap. We also found 39% “target neurons” with significant saccadic target-related responses, which were stronger in gap trials and correlated with the SRTs better than the remaining neurons. Compared with saccades with relatively long SRTs, short-SRT saccades were preceded by both stronger gap-related and target-related responses in all PPC neurons, regardless of whether such response reached significance. Our findings suggest that the PPC in the marmoset contains an area that is involved in the modulation of saccadic preparation. NEW & NOTEWORTHY As a primate model in systems neuroscience, the marmoset is a great complement to the macaque monkey because of its unique advantages. To identify oculomotor networks in the marmoset, we recorded from the marmoset posterior parietal cortex during a saccadic task and found single-unit activities consistent with a role in saccadic modulation. This finding supports the marmoset as a valuable model for studying oculomotor control.
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9

Fereydouni, B., C. Drummer, N. Aeckerle, S. Schlatt, and R. Behr. "The neonatal marmoset monkey ovary is very primitive exhibiting many oogonia." REPRODUCTION 148, no. 2 (August 2014): 237–47. http://dx.doi.org/10.1530/rep-14-0068.

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Oogonia are characterized by diploidy and mitotic proliferation. Human and mouse oogonia express several factors such as OCT4, which are characteristic of pluripotent cells. In human, almost all oogonia enter meiosis between weeks 9 and 22 of prenatal development or undergo mitotic arrest and subsequent elimination from the ovary. As a consequence, neonatal human ovaries generally lack oogonia. The same was found in neonatal ovaries of the rhesus monkey, a representative of the old world monkeys (Catarrhini). By contrast, proliferating oogonia were found in adult prosimians (now called Strepsirrhini), which is a group of ‘lower’ primates. The common marmoset monkey (Callithrix jacchus) belongs to the new world monkeys (Platyrrhini) and is increasingly used in reproductive biology and stem cell research. However, ovarian development in the marmoset monkey has not been widely investigated. Herein, we show that the neonatal marmoset ovary has an extremely immature histological appearance compared with the human ovary. It contains numerous oogonia expressing the pluripotency factors OCT4A, SALL4, and LIN28A (LIN28). The pluripotency factor-positive germ cells also express the proliferation marker MKI67 (Ki-67), which has previously been shown in the human ovary to be restricted to premeiotic germ cells. Together, the data demonstrate the primitiveness of the neonatal marmoset ovary compared with human. This study may introduce the marmoset monkey as a non-human primate model to experimentally study the aspects of primate primitive gonad development, follicle assembly, and germ cell biology in vivo.
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10

Mills, Penniston, and Tanumihardjo. "Extra-Hepatic Vitamin A Concentrations in Captive Rhesus (Macaca Mulatta) and Marmoset (Callithrix Jacchus) Monkeys Fed Excess Vitamin A." International Journal for Vitamin and Nutrition Research 75, no. 2 (March 1, 2005): 126–32. http://dx.doi.org/10.1024/0300-9831.75.2.126.

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Recent work examining vitamin A (VA) status of rhesus monkeys (Macaca mulatta) used as models for human biomedical research has revealed subtoxic hepatic VA concentrations. Livers of marmoset monkeys (Callithrix jacchus), another experimental animal, were also high in VA as was serum retinyl ester concentration. Both species consumed common research diets that provided up to four times the amount of VA (retinyl acetate) as currently recommended by the National Research Council. To further define the effects of chronically high dietary VA as found in many human subpopulations, we analyzed lung and kidney tissues from subtoxic rhesus and marmoset monkeys (n = 10 each) for retinol and retinyl esters. Marmoset kidneys contained 0.88 ± 0.66 mumol VA/g and was nearly the same as hepatic VA at 1.40 ± 0.44 mumol/g (p = 0.143). In contrast, rhesus kidney VA concentrations were 0.0100 ± 0.0032 mumol/g, even though liver reserves were 18.8 ± 6.4 mumol VA/g (p < 0.0001). Lung tissue VA concentrations, 0.0022 ± 0.0012 and 0.0061 ± 0.0025 mumol/g for marmosets and rhesus, respectively, were lower as compared with kidney (p < 0.011). Kidney and lung VA in monkeys with adequate, but not excessive, VA stores have not been determined; hence, interpretation of these findings is limited to tissue retinol and retinyl ester profiles and extrapolation from other species rather than direct comparison to "normal" values.
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Більше джерел

Дисертації з теми "Marmoset monkeys"

1

Seehase, Sophie [Verfasser]. "Marmoset monkeys as a preclinical model in respiratory research / Sophie Seehase." Hannover : Bibliothek der Tierärztlichen Hochschule Hannover, 2013. http://d-nb.info/1030452954/34.

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2

Hauser, Jonas. "Long-term neuro-behavioural effects of prenatal dexamethasone treatment in Wistar rats and marmoset monkeys." kostenfrei, 2007. http://e-collection.ethbib.ethz.ch/view/eth:30028.

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3

Le, Friec Alice. "Evolution of corticospinal tract integrity in stroked marmoset monkeys : Towards a bioimplant and stem cell therapeutic strategy." Thesis, Toulouse 3, 2020. http://www.theses.fr/2020TOU30031.

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L'Accident Vasculaire Cérébral (AVC) ischémique endommage fréquemment des régions cérébrales impliquées dans le contrôle du mouvement volontaire. De fait, cette pathologie est l'une des premières causes de handicap acquis à l'âge adulte. Bien que des centaines de stratégies thérapeutiques aient montré de potentiels effets bénéfiques dans des modèles animaux d'AVC, seule la rééducation motrice est validée comme traitement des déficits moteurs après la phase aiguë chez l'Homme. Ce constat souligne l'importance de développer et de caractériser des modèles pré cliniques reproductibles chez une espèce proche de l'homme, qui permettront de mieux évaluer l'efficacité de thérapies innovantes. Le premier objectif de ma thèse était donc de caractériser les conséquences anatomiques et fonctionnelles d'une lésion cérébrale induite par une toxine mitochondriale, le malonate, chez le rongeur et le primate non humain. Sur le plan anatomique, l'Imagerie par Résonance Magnétique multimodale a permis un suivi longitudinal non-invasif des altérations tissulaires. Celles-ci ont été explorées plus spécifiquement par des analyses histologiques. Les déficits moteurs ont été évalués par une batterie de tests sensorimoteurs. Nous montrons premièrement que l'injection stéréotaxique de malonate dans la capsule interne du rat permet une lésion ciblée des fibres du faisceau corticospinal (FCS). Cette lésion est associée à des déficits moteurs de longue durée, similaires à ceux observés suite à un AVC lacunaire chez l'Homme. Dans un deuxième temps, j'ai caractérisé les conséquences de l'injection stéréotaxique de malonate au niveau du cortex moteur primaire chez le marmouset. Ce modèle a été conçu afin de reproduire les effets d'une lésion corticale du FCS qui est fréquente dans l'AVC ischémique. Cette approche produit une lésion focale de volume et de localisation reproductible. Des lésions secondaires hypointenses en IRM pondérée T2 et hyperintenses en IRM pondérée T1 et associées à une infiltration d'astrocytes et de microglie sont observées dans la substance blanche à distance du site de la lésion, vraisemblablement suite à la perte de neurones qui font partie des boucles motrices cortico-sous-corticales. Fait important à noter, ces dommages sont associés à une perte durable de force et de dextérité du membre supérieur des animaux. L'injection stéréotaxique de malonate reproduit donc les conséquences de l'AVC ischémique, conduit à des déficits chroniques et permettra donc l'évaluation de nouvelles stratégies thérapeutiques. Parmi celles-ci, la thérapie cellulaire semble un moyen prometteur de favoriser la réparation tissulaire.[...]
Ischemic stroke frequently damages brain regions involved in the control of voluntary movement and remains a leading cause of adult-acquired disability. Although hundreds of therapeutic strategies have shown potential benefits in animal models of stroke, motor rehabilitation and physiotherapy remain the only validated treatments in Humans after the acute phase. This observation highlights the need to develop and characterize reproducible pre-clinical models, which will allow the assessment of experimental therapies. The first objective of this work was therefore to characterize the anatomical and functional consequences of a brain lesion induced by stereotaxic injection of malonate, a mitochondrial toxin, in rodents and primates. Multimodal Magnetic Resonance Imaging allowed longitudinal non-invasive assessment of tissue alterations. We then performed histological analyses to further describe tissue damage. Motor deficits and their recovery were evaluated using a battery of sensorimotor tests. We first show that stereotaxic injection of malonate into the internal capsule of rats creates targeted destruction of corticospinal tract fibers. This lesion is associated with long term motor impairments similar to those observed after lacunar stroke in humans. Secondly, I characterized the consequences of stereotaxic injection of malonate into the primary motor cortex of marmoset monkeys. This model was developed in order to reproduce the effects of middle cerebral artery stroke in Humans. Indeed, the blood supply of motor territories strongly depends on this vessel, which is often occluded in ischemic stroke. We show that this approach causes a focal lesion of predictable size and location. Secondary lesions together with astrocyte and microglial infiltration were observed in white matter tracts distant to the lesion site, and likely occur after degeneration of cortico-sub-cortical motor loop axons. Importantly, the lesion was associated with long-lasting loss of dexterity and grip strength of the contralateral forelimb. Stereotaxic injection of malonate therefore reproduces the consequences of ischemic stroke and should allow the investigation of innovative therapies. Stem cell therapy may hold promise for tissue regeneration in the central nervous system (CNS). Co-transplantation of stem cells with biomaterials is currently investigated to enhance the survival and maturation of transplanted cells within the lesion site. Biomaterials can help to create a microenvironment permissive to cell integration within host tissue. An approach combining intracerebral engraftment of semi-rigid micro patterned biomaterials with human neural stem cells (to form a "neuro-implant") improved the recovery of grip strength in stroked rats. [...]
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4

Rutherford, Julienne Noelle. "Litter size effects on placental structure and function in common marmoset monkeys (Callithrix jacchus) implications for intrauterine resource allocation strategies /." [Bloomington, Ind.] : Indiana University, 2007. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3278218.

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Анотація:
Thesis (Ph.D.)--Indiana University, Dept. of Anthropology, 2007.
Source: Dissertation Abstracts International, Volume: 68-09, Section: A, page: 3930. Adviser: Kevin D. Hunt. Title from dissertation home page (viewed May 8, 2008).
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5

Pomberger, Thomas [Verfasser], and Steffen [Akademischer Betreuer] Hage. "Audio-Vocal Integration Mechanisms and Volitional Control of Vocal Behavior in Marmoset Monkeys (Callithrix jacchus) / Thomas Pomberger ; Betreuer: Steffen Hage." Tübingen : Universitätsbibliothek Tübingen, 2019. http://d-nb.info/1196633789/34.

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6

Young, Fiona Margaret. "Luteal regression in the marmoset monkey." Thesis, University of Edinburgh, 2000. http://hdl.handle.net/1842/23277.

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Ovaries were studied on luteal days 10, 18 and 22 (corresponding to the mid luteal phase, functional luteal regression and structural luteal regression respectively), and also 12 and 24 hours after administration of either PGF2a or GnRH antagonist. Decreased progesterone concentrations indicative of functional luteal regression were apparent 12 hours later. Analysis of haematoxylin and eosin stained sections of corpora lutea indicated that the administration of PGF2a or GnRH antagonist resulted in apoptosis, and also in the formation of cytoplasmic vacuoles in steroidogenic cells. Apoptosis in corpora lutea was further investigated by 3' end labelling DNA extracted from corpora lutea, and by in situ 3' end labelling of sections of ovarian tissue. Apoptosis was found to occur after induced luteolysis and in naturally regressing corpora lutea but only after progesterone had decreased to follicular phase values. Therefore the decline in progesterone characteristic of functional luteal regression was not caused by the apoptotic cell death of steroidogenic cells. However, apoptosis played a role in structural luteal regression. Ubiquitin is expressed only by live cells undergoing a process of non-apoptotic cell death. Ubiquitin expression was only found in PGF2a, but not in GnRH antagonist treated luteal tissue, suggesting three possible explanations: that the cells in GnRH antagonist treated animals were dead prior to the collection point of 12 hours, or that the cells were not in a cell death pathway, or that cell death was occurring via different mechanisms in PGF2a and GnRH antagonist treated animals. The importance of the vasculature in luteal regression was investigated by labelling endothelial cells with an antibody against von Willebrand Factor VIII Antigen. Endothelial cell numbers remained constant after administration of luteolytic agents, indicating that induced luteal regression was not effected by vascular changes. Similarly, the vascualture did not change during functional regression in untreated animals. Vascular remodelling, however, occurred during structural luteal regression, when the vasculature changed from an extensive network of small capillaries to a system comprised of a lower number of larger blood vessels.
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7

Dalrymple, Annette. "Prolactin receptor expression and signalling in the marmoset monkey uterus." Thesis, University of Edinburgh, 2001. http://hdl.handle.net/1842/23319.

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Immunohistochemical studies localised PRL expression to the stromal compartment of the marmoset endometrium. Expression was minimal during the proliferative phase and was up-regulated during the mid-late secretory phase of the ovulatory cycle. Similarly to PRL, PRL-R expression was minimal during the proliferative phase and was dramatically up-regulated during the mid-late secretory phase. However, expression of the PRL-r was localised to the glandular epithelium of the endometrium. The temporal pattern of PRL-R gene expression in the marmoset uterus across the ovulatory cycle was further confirmed by ribonuclease protection assay. The role of Jak2, Stat1 and Stat5 in the intracellular signalling pathway of PRL were also assessed in the mid-late secretory phase. Jak2/Stat1/Stat5 proteins were co-localised with the PRL-R to the glandular epithelial compartment. Moreover within the marmoset uterus, Jak2, Stat1 and Stat5 were temporarily phosphorylated in response to PRL. The pattern of expression of the IRF-1 gene, a PRL target gene, and the effect of PRL on transcription of IRF-1 were also investigated. IRF-1 expression in the marmoset uterus was encoded by a protein of 48 kDa and was localised to the glandular epithelial compartment, as was observed for the PRL-R and Jak2/Stat1/Stat5 proteins. Incubation of mid-late secretory uterine tissue with PRL for 3 hours resulted in 2.4 ± 0.5 (P<0.05) fold induction of IRF-1 gene expression. These studies confirm (a) high sequence and functional similarity between the marmoset and human PRL-R and (b) the expression of both PRL and its receptor in the uterus of the marmoset monkey. Expression of both genes is up-regulated during the mid-late secretory phase of the ovulatory cycle. PRL function in the marmoset uterus is linked to the Jak/Stat signalling pathway leading to the regulation of expression of PRL-responsive genes such as IRF-1. The site of expression of PRL, PRL-R and IRF-1 in the marmoset uterus suggest that PRL may influence glandular epithelial function and direct gene transcription in these cells in a paracrine fashion. In conclusion, the data strongly suggest that the marmoset monkey may provide a useful tool to investigate the role of PRL in human reproduction.
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8

Ronacher, Katharina. "Internalisation of the type II gonadotropin-releasing hormone receptor of marmoset monkey." Doctoral thesis, University of Cape Town, 2003. http://hdl.handle.net/11427/8599.

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Bibliography: leaves 102-124.
The mammalian type II GnRH receptor has a C-terminal tail unlike the mammalian type I GnRH receptor, which uniquely lacks the cytoptasmic C- terminal domain. lnternalisation of a mammalian type ll GnRH receptor has never been investigated, therefore this thesis studies the internalisation pathway of the type ll GnRH receptor. As the C-terminal tail mediates rapid internalisation of many G protein-coupled receptors this research investigates the functional role of the C-terminal tail and intracellular loop in receptor internalisation. The internalisation pathway of the type ll GnRH receptor in COS-1 cells was investigated by co expressing dominant negative mutants and wild- type constructs of G protein-coupled receptor kinases (GRKs), dynamin-1 and β-arrestin 1 and 2 with the type II GnRH receptor. The results show that internatisation of the receptor requires GRK 2 and dynamin but does not require β-arrestin 1 and 2. Furthermore, inhibitors to both the caveolae pathway as well as the clathrin coated vesicle endocytosis abolished receptor internalisation indicating that both structures are involved in internalisation of the receptor. Even though in COS-1 cells the type ll GnRH receptor internatises in a β-arrestin independent manner, internalisation of this receptor can be enhanced by over-expression of wild type β-arrestin. This indicates that the type ll GnRH receptor is able to utilise a β-arrestin mediated internaltsation pathway if high levels of β-arrestin are present in the cell. The mammalian type ll GnRH receptor internalises with enhanced rate and extent compared to the tail-less human type I GHRH receptor. The role of the C-terminal tail of the type ll GnRH receptor in internalisation was investigated by measuring internalisation of C-terminally truncated mutants. It was found that the region between Gly 343 and Ser 335 within the C-terminal domain is important for receptor internalisation. Substitution of putative phosphorylation sites within this region revealed that Ser 338 and Ser 339 are critical for rapid receptor internalisation. Furthermore a serine residue in intracellular loop three (Ser 251) was shown to play a role in signalling as well as in internalisation. Since dominant negative GRK 2 could not inhibit internalisation of a mutant lacking all three serine residues, but could reduce internalisation of the wild-type receptor, we suggest that Ser 251, 338 and 339 are target of phosphorylation by GRK. However these phosphorylation sites as well as the C-terminal tail are not necessary for β-arrestin dependent internalisation. Taken together this thesis elucidates the internalisation pathway of a mammalian type lI GnRH receptor and identified residues within the C-terminal tail and intracellular loop three that are critical for rapid internalisation.
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9

Gameau, Louise J. "The effect of cytokines on chorionic gonadotrophin expression in the marmoset monkey embryo /." Title page, contents and summary only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phg192.pdf.

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10

Eildermann, Katja [Verfasser]. "Potentially pluripotent cells in the common marmost monkey (Callithrix Jacchus) testis / Katja Eildermann." Bremen : IRC-Library, Information Resource Center der Jacobs University Bremen, 2012. http://d-nb.info/1035266067/34.

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Книги з теми "Marmoset monkeys"

1

Pygmy marmosets. North Mankato, Minn: Capstone Press, 2013.

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2

Lucie, Papineau, and Sarrazin Marisol 1965 ill, eds. Gilda the giraffe and Marvin the marmoset. Minneapolis, Minn: Picture Window Books, 2006.

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3

S, Ashwell Ken W., ed. Stereotaxic and chemoarchitectural atlas of the brain of the common marmoset. Boca Raton: Taylor & Francis, 2012.

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4

Nunez, Sigrid. Mitz: The marmoset of Bloomsbury. New York: HarperFlamingo, 1998.

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5

ill, Judge Lita, ed. Quick, little monkey! Honesdale, Pennsylvania: Boyds Mills Press, 2016.

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6

Petrides, Michael, Charles Watson, George Paxinos, Marcello Rosa, and Hironobu Tokuno. Marmoset Brain in Stereotaxic Coordinates. Elsevier Science & Technology Books, 2011.

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7

Fox, James G., Keith Mansfield, Suzette D. Tardif, Robert P. Marini, and Lynn M. Wachtman. Common Marmoset in Captivity and Biomedical Research. Elsevier Science & Technology Books, 2018.

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8

Fox, James G., Keith Mansfield, Suzette D. Tardif, Robert P. Marini, and Lynn M. Wachtman. Common Marmoset in Captivity and Biomedical Research. Elsevier Science & Technology, 2018.

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9

The Marmoset Brain In Stereotaxic Coordinates. Springer, 2008.

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10

Palazzi, Xavier, and Nicole Bordier. The Marmoset Brain in Stereotaxic Coordinates. Springer, 2009.

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Частини книг з теми "Marmoset monkeys"

1

Abbott, David H. "Reproduction in Female Marmoset Monkeys, Callithrix jacchus." In Reproductive Biology of South American Vertebrates, 245–61. New York, NY: Springer New York, 1992. http://dx.doi.org/10.1007/978-1-4612-2866-0_17.

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2

Schulz, Thomas G., Diether Neubert, Donald S. Davies, and Robert J. Edwards. "Induction of Cytochromes P450 by Dioxins in Liver and Lung of Marmoset Monkeys (Callithrix jacchus)." In Advances in Experimental Medicine and Biology, 443–46. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9480-9_53.

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3

Abbott, David H., and John P. Hearn. "The Effects of Neonatal Exposure to Testosterone on the Development of Behaviour in Female Marmoset Monkeys." In Novartis Foundation Symposia, 299–327. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720448.ch14.

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4

Einspanier, Almuth. "Relaxin is an important factor for uterine differentiation and implantation in the marmoset monkey." In Relaxin 2000, 73–82. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-017-2877-5_9.

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5

Hillier, Stephen G., Christopher R. Harlow, Helen J. Shaw, E. Jean Wickings, Alan F. Dixson, and J. Keith Hodges. "Granulosa Cell Differentiation in Primate Ovaries: The Marmoset Monkey (Callithrix Jacchus) as a Laboratory Model." In The Primate Ovary, 61–73. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4615-9513-7_5.

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6

Charnock, J. S., T. M. Bridle, J. Turner, P. L. McLennan, and M. Y. Abeywardena. "Vitamin E and Health in the Marmoset Monkey: A Non-Human Primate Model for Nutritional Research." In Lipid-Soluble Antioxidants: Biochemistry and Clinical Applications, 134–51. Basel: Birkhäuser Basel, 1992. http://dx.doi.org/10.1007/978-3-0348-7432-8_12.

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7

Tani, Kenzaburo, Hitoshi Hibino, Hajime Sugiyama, Ming-Shiuan Wu, Kiyoko Izawa, Tsuyoshi Tanabe, Hidenori Hase, et al. "Hematological Aspects of Common Marmoset Monkey Transplanted with Autologous MDR1 Gene Transduced Peripheral Blood Stem Cells." In Molecular Biology of Hematopoiesis 6, 307–19. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4797-6_38.

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8

Ghazanfar, A. A., and D. Y. Takahashi. "The Evo-Devo of Vocal Communication: Insights From Marmoset Monkeys." In Evolution of Nervous Systems, 317–24. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-12-804042-3.00137-8.

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9

"25(OH)D3, BUT NOT 1,25(OH)2D3 CURES OSTEOMALACIA IN MARMOSET MONKEYS." In Vitamin D, 450–51. De Gruyter, 1988. http://dx.doi.org/10.1515/9783110846713.450.

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10

Craig F., Ferris, and Snowdon Charles T. "Functional Magnetic Resonance Imaging in Conscious Marmoset Monkeys: Methods and Applications in Neuroscience Research." In The Laboratory Primate, 353–69. Elsevier, 2005. http://dx.doi.org/10.1016/b978-012080261-6/50021-0.

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Тези доповідей конференцій з теми "Marmoset monkeys"

1

Veale, Richard, Chih-yang Chen, and Tadashi Isa. "Marmoset Monkeys Model Human Infant Gaze?" In 2021 IEEE International Conference on Development and Learning (ICDL). IEEE, 2021. http://dx.doi.org/10.1109/icdl49984.2021.9515602.

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2

Takahashi, Daniel Y., Darshana Narayanan, and Asif A. Ghazanfar. "Development of self-monitoring essential for vocal interactions in marmoset monkeys." In 2013 IEEE International Conference on Development and Learning and Epigenetic Robotics (ICDL). IEEE, 2013. http://dx.doi.org/10.1109/devlrn.2013.6652553.

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3

Takahashi, Daniel Y., Darshana Narayanan, and Asif A. Ghazanfar. "A computational model for vocal exchange dynamics and their development in marmoset monkeys." In 2012 IEEE International Conference on Development and Learning and Epigenetic Robotics (ICDL). IEEE, 2012. http://dx.doi.org/10.1109/devlrn.2012.6400844.

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4

Seehase, Sophie, Vanessa Neuhaus, Franz-Josef Kaup, O. Pfennig, C. Förster, Jens M. Hohlfeld, Sascha Knauf, Armin Braun, and Katherina Sewald. "Ex Vivo Lung Culture Models Of Marmoset Monkeys And Humans For Anti-Inflammatory Drug Testing." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4169.

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5

Lauenstein, Hans-Dieter, Sophie Seehase, Simone Switalla, Frauke Prenzler, Katherina Sewald, Franz-Joseg Kaup, Eberhard Fuchs, Christina Schlumbohm, and Armin Braun. "Establishment Of A Tiered Model Of Airway Inflammation In Marmoset Monkey For Preclinical Pharmaceutical Testing." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2886.

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6

Marrouch, Natasza, Heather L. Read, Joanna Slawinska, and Dimitrios Giannakis. "Data-driven spectral decomposition of ECoG signal from an auditory oddball experiment in a marmoset monkey: Implications for EEG data in humans." In 2018 International Joint Conference on Neural Networks (IJCNN). IEEE, 2018. http://dx.doi.org/10.1109/ijcnn.2018.8489475.

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