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Статті в журналах з теми "Humane Animal Treatment"
Lansdell, Herbert. "Laboratory Animals Need Only Humane Treatment: Animal “Rights” May Debase Human Rights." International Journal of Neuroscience 42, no. 3-4 (January 1988): 169–78. http://dx.doi.org/10.3109/00207458808991594.
Повний текст джерелаWang, Yuanfang, and Peng Chan. "Animal Mistreatment in Business: Ethical Challenges and Solutions." International Business Research 10, no. 5 (April 24, 2017): 159. http://dx.doi.org/10.5539/ibr.v10n5p159.
Повний текст джерелаThompson, Kelly, and Eleonora Gullone. "An Investigation into the Association between the Witnessing of Animal Abuse and Adolescents' Behavior toward Animals." Society & Animals 14, no. 3 (2006): 221–43. http://dx.doi.org/10.1163/156853006778149163.
Повний текст джерелаHarde, Roxanne. "“Better Friends”: Marshall Saunders Writing Humane Education and Envisioning Animal Rights." Jeunesse: Young People, Texts, Cultures 1, no. 2 (December 2009): 85–108. http://dx.doi.org/10.3138/jeunesse.1.2.85.
Повний текст джерелаNelson, Michael L. "Book Review: Animal Rights and Welfare: A Documentary and Reference Guide." Reference & User Services Quarterly 55, no. 2 (December 16, 2015): 175. http://dx.doi.org/10.5860/rusq.55n2.175a.
Повний текст джерелаGleba, Ol'ga Vladimirovna. "Legal Problems of Improving Legislation on the Protection of Farm Animals." Сельское хозяйство, no. 4 (April 2022): 1–10. http://dx.doi.org/10.7256/2453-8809.2022.4.39344.
Повний текст джерелаGleba, Ol'ga Vladimirovna. "Legal Problems of Improving Legislation on the Protection of Farm Animals." Сельское хозяйство, no. 2 (February 2022): 24–33. http://dx.doi.org/10.7256/2453-8809.2022.2.39344.
Повний текст джерелаРебенко, Галина, Олександр Нечипоренко, Валерій Пономаренко, Юрій Мусієнко, Анатолій Фотін, Юлія Сторчак, and Олександрa Баканова. "Decreasing of the risk of zoonoses by solving the problem with stray dogs." Bulletin of Sumy National Agrarian University. The series: Veterinary Medicine, no. 1-2(44-45) (July 1, 2019): 42–49. http://dx.doi.org/10.32845/bsnau.vet.2019.1-2.6.
Повний текст джерелаOberg, Andrew. "ALL TOO HUMAN? SPECIESISM, RACISM, AND SEXISM." Think 15, no. 43 (2016): 39–50. http://dx.doi.org/10.1017/s1477175616000051.
Повний текст джерелаWrenn, Corey Lee. "Nonhuman Animal Rights, Alternative Food Systems, and the Non-Profit Industrial Complex." PhaenEx 8, no. 2 (December 26, 2013): 209. http://dx.doi.org/10.22329/p.v8i2.4093.
Повний текст джерелаДисертації з теми "Humane Animal Treatment"
Beck, Daniel Phillip. "Animals on Lifeboats: a Defense of a Sliding Scale Model of Moral Status." Miami University Honors Theses / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=muhonors1240373673.
Повний текст джерелаChacko, Anu. "Comparison of human and animal Chlamydia pneumoniae responses to interferon gamma and penicillin treatment." Thesis, Queensland University of Technology, 2015. https://eprints.qut.edu.au/85438/1/Anu_Chacko_Thesis.pdf.
Повний текст джерелаHuff, Michael W. "The biblical responsibility of dominion and man's use and treatment of animals." Theological Research Exchange Network (TREN), 1987. http://www.tren.com.
Повний текст джерелаHoffmann, Willem Abraham. "The determination of plasma adrenocorticotropic hormone during the treatment of women experiencing dog phobia." Pretoria : [s.n.], 2009. http://upetd.up.ac.za/thesis/available/etd-11162006-093620/.
Повний текст джерелаVan, den Heever Martine. "A comparison of the effect of curcumin treatment on apoptosis, necrosis and autophagy in a MCF-7 mammary adenocarcinoma and a MCF-12A healthy mammary epithelial cell line." Thesis, Stellenbosch : University of Stellenbosch, 2009. http://hdl.handle.net/10019.1/2970.
Повний текст джерелаBreast cancer is currently the primary cause of cancer-related death in women worldwide. Conventional treatments such as radiation and chemotherapy have many deleterious and long lasting side-effects, some of which are permanent, such as infertility. As certain tumour cells can also acquire resistance to chemotherapy, the need for the development of a less severe, yet more effective, targeted anti-cancer treatment exists. Curcumin, a plant polyphenol from Curcuma longa, has long been thought to possess antitumour, antioxidant, anti-arthritic, anti-amyloid, anti-ischemic and anti-inflammatory properties. Numerous studies conducted over the past sixty years confirm this. We aimed at examining the effect of curcumin on cell viability and the different modes of cell death, namely apoptosis, necrosis and autophagy, in the MCF-12A (non-tumorigenic mammary epithelial) and MCF-7 (mammary adenocarcinoma) cell lines. Cells were incubated with different doses of curcumin to evaluate the dose response through a MTT assay. Thereafter, cells were incubated with 200 μM curcumin for 48 hrs and stained with markers and DNA stains for apoptosis (Hoechst, Caspase-3, PARP), necrosis (Propidium Iodide) and autophagy (LC3B and Beclin-1). Cells were examined via fluorescence microscopy, Western Blot- and FACS analyses. MTT results showed no significant decrease in viability in the MCF-12A cell line after curcumin treatment. However, a significant decrease in viability was observed in MCF-7 cells after treatment with 200 μM curcumin (p < 0.05). Treated MCF-7 cells also show clear LC3B expression. FACS results show a significant difference in Hoechst mean fluorescence intensity in MCF-7 cells after curcumin treatment (p < 0.05). This study provides evidence that MCF-7 cells respond to a 200 μM dose of curcumin treatment through metabolic change and induction of the autophagic pathway. The model system used in this study provides groundwork for further cell culture based studies regarding breast cancer and curcumin.
Hoffmann, Willem Abraham. "The determination of plasma adrenocorticotropic hormone during the treatment of women experiencing dog phobia." Diss., University of Pretoria, 2000. http://hdl.handle.net/2263/29490.
Повний текст джерелаDissertation (MSc (Veterinary Ethology))--University of Pretoria, 1999.
Production Animal Studies
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Costantini, Veronica P. "Animal enteric viruses: gene expression, epidemiology and their role in shellfish and environmental contamination." The Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=osu1186687895.
Повний текст джерелаSinisalo, Johanna. "Interactions between humans and dogs : Neurobiological factors relevant for the treatment of exhaustion-related disorders." Thesis, Högskolan i Skövde, Institutionen för biovetenskap, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-11436.
Повний текст джерелаLeisching, Gina Renata. "Manipulation of the autophagic pathway sensitises cervical cancer cells to cisplatin treatment." Thesis, Stellenbosch : Stellenbosch University, 2013. http://hdl.handle.net/10019.1/80107.
Повний текст джерелаENGLISH ABSTRACT: Introduction Cisplatin has been widely used to treat solid tumours and much success has come from the use of this drug in the treatment of head and neck, ovarian, testicular, cervical and small-cell lung cancers. However, the success of cisplatin treatment is limited due to its dose-limiting toxicity and its resulting side-effects, such as nephro- and ototoxicity. The devastating side-effects induced by cisplatin treatment provided the platform for this study whereby the aim was to lower the concentration of cisplatin while maintaining its cancer-specific cytotoxic action. Equally concerning is, cisplatin resistance which is becoming increasingly common, and this radically limits the clinical efficacy and utility of the drug. Adjuvant therapy has thus become necessary in an attempt to possibly curb or lessen the extent of cisplatin resistance. Due to the large body of evidence implicating the importance of autophagy in cancer, the prospect of targeting this mechanism has generally been accepted. Various chemotherapy agents induce autophagy in cancer cells; however the effect of cisplatin on autophagic induction has not been very well explored. We thus hypothesise that the manipulation of the autophagic pathway will sensitise cancer cells to a low concentration of cisplatin treatment. Furthermore, due to the functional interaction between Bcl-2 and Beclin-1 and its role in the regulation of autophagy, ratio analysis of Beclin-1 to Bcl-2 as means of detecting the role of autophagy within the cell under homeostatic and treatment/stress conditions has been conducted. Additionally, Bcl-2 has a prominent role in the malignant cell and it’s over-expression has been found to confer resistance in a variety of cancerous cell lines. We therefore hypothesise that the silencing of Bcl-2 prior to cisplatin treatment will sensitise cervical cancer cells to apoptosis and increase the Beclin-1/Bcl-2 ratio in favour of apoptosis. Materials and Methods Three human cervical cell lines were used: a non-cancerous ectocervical epithelial cell line (Ect1/E6E7) and two cancerous cervical cell lines (HeLa and CaSki). In order to determine a concentration of cisplatin that was non-toxic to the non-cancerous Ect1/E6E7 cell line, a dose-response was performed. With the use of an autophagy inhibitor (bafilomycin A1) and an autophagy inducer (rapamycin), autophagic flux capacities were assessed in each cell line through the Western blotting technique. In order to assess whether the chosen concentration of cisplatin induced autophagy, flow cytometry with the use of a Lysotracker™ dye was utilised, as well as analysis of autophagy protein levels (LC-3 II, Beclin-1 and p62). Autophagy modulation was achieved through two methods: pharmacological modulation with use of two recognised agents, namely bafilomycin A1 and rapamycin, and biological manipulation with the use of ATG5 and mTOR mRNA silencing. The effects of different treatment regimes on cell death was assessed with the use of PARP and caspase-3 cleavage through Western blotting, caspase-3/-7 activity (Caspase-Glo®), PI inclusion, LDH release and MTT reductive capacity. Additionally the effects of these treatment regimes on cell-cycle progression were also analysed. Beclin-1 and Bcl-2 expression was determined through Western blotting and immunocytochemistry before and after treatment with cisplatin in HeLa and CaSki cells. To assess the reliance of the cervical cancer cells on Bcl-2 after cisplatin treatment, Bcl-2 knock-down was achieved through RNA interference, where after the Beclin-1/Bcl-2 ratio was assessed as well as apoptosis with the use of cleaved PARP analysis (Western blotting) and Caspase-Glo©. For the ex vivo analysis, biopsies were collected from patients undergoing routine colposcopy screenings and hysterectomies at Tygerberg Hospital, Tygerberg, Western Cape. A total of 10 normal, 29 low-grade squamous intraepithelial lesions (LSIL), 33 high-grade squamous intraepithelial lesions (HSIL) and 13 carcinoma biopsies were collected for analysis, where after the expression profiles of two autophagy markers (mTOR and LC-3 II), as well as one anti-apoptotic marker (Bcl-2) were assessed. Protein levels were analysed through Western blot and confirmed through immunohistochemistry. Results Dose-response curves revealed that 15 μM of cisplatin did not induce cell death in the normal cervical epithelial cell line (Ect1/E6E7) and was therefore utilised through-out the remainder of the study. It was additionally determined that the CaSki cells were more resistant to cisplatin treatment when compared to the HeLa and Ect1/E6E7 cells. Autophagic flux analysis revealed that, although all three cell lines were cervix derived, their autophagic flux capacities differed. It was observed that the chosen concentration of cisplatin was able to induce autophagy in all three cell lines, with the HeLa cells demonstrating a particularly pronounced response. Autophagy modulation in conjunction with cisplatin treatment revealed the following: Autophagy inhibition with bafilomycin A1 lead to significant increases in caspase-3 and PARP cleavage and LDH release in both cervical cancer cell lines. The inhibition of autophagy through silencing of ATG5 induced caspase-3 cleavage and agrees with results obtained from pharmacological inhibition of autophagy with bafilomycin A1. In addition to autophagic induction, a low concentration of cisplatin induced the up-regulation of Bcl-2, which when silenced significantly improved cisplatin-induced apoptosis in both cervical cancer cell lines. Analysis of the expression profiles of mTOR and LC-3 in normal, pre-malignant (LSIL and HSIL) and cancerous cervical tissue revealed that autophagy is significantly up-regulated in HSILs and carcinoma of the cervix. Additionally, Bcl-2 expression is significantly increased in cervical carcinoma tissue, which agrees with results from other studies. Conclusion Autophagic flux capacities between the three cell lines investigated, derived from the same organ, differ significantly. This should be taken into consideration when autophagic modulation is being used as an adjuvant treatment. With regard to chemotherapy treatment in cervical cells, a low-concentration of cisplatin significantly induces autophagy in malignant and non-malignant cervix-derived cell lines where it serves a pro-survival mechanism. Inhibition of autophagy with bafilomycin A1 and ATG5 siRNA confirmed this survival effect in both cancerous cell lines where apoptosis was significantly increased. Interestingly, rapamycin pre-treatment together with cisplatin did not induce significant levels of apoptosis in HeLa cells where autophagy induction may have provided additional protection from the cytotoxic effects of cisplatin. Therefore the inhibition of autophagy through pharmacological and biological inhibition improves the cytotoxicity of a low concentration of cisplatin and provides a promising new avenue for the future treatment of cervical cancer. Bcl-2 up-regulation in response to cisplatin treatment also serves as a protective mechanism by which cervical cancer cells survive. The extent of apoptotic cell death observed after biological inhibition of Bcl-2 reiterates the fact that this response may be exploited in order to favour the use of lower concentrations of cisplatin. Analysis of clinical specimens emphasised the value of the in vitro work: Cervical cancer biopsies had increased expression of both LC-3 II and Bcl-2, indicating autophagy induction and apoptosis inhibition, respectively. Thus two novel methods of improving cisplatin cytotoxicity have been demonstrated in the following study. Treatment regimens may administer more frequently and prolonged due to the minimal side-effects that accompanies low-dose cisplatin treatment.
AFRIKAANSE OPSOMMING: Inleiding Sisplatien word algemeen gebruik vir die behandeling van soliede gewasse. Baie sukses is reeds deur die gebruik van díe middel behaal in die behandeling van kop en nek, ovariale, terstikulêre, servikale en klein-sel kankers. Die sukses van Sisplatien-behandeling word wel ingeperk deur die dosis-beperkende toksisiteit en die gevolglike newe-effekte soos nefrotoksisiteit. Hierdie verwoestende newe-effekte wat deur sisplatien behandelings geïnduseer word, het as die platform vir hierdie studie gedien. Die doel was om die sisplatien konsentrasies te verlaag, maar terselfdertyd die kankerspesifieke sitotoksisiteit te behou. Nog ʼn punt van kommer is dat sisplatien-weerstandigheid aan die toeneem is, wat die kliniese effektiwiteit en gebruik van hierdie middel geweldig beperk. Byvoegmiddels het dus noodsaaklik geraak in die poging om die sisplatien-weerstandigheid te verhoed. As gevolg van verskeie bewyse wat die belangrikheid van outofagie in kanker impliseer, is die vooruitsig om hierdie meganisme te teiken, algemeen aanvaar. Verskeie chemoterapeutiese middels induseer outofagie in kanker selle, hoewel die effek van Sisplatien op outofagiese induksie nog nie goed ondersoek is nie. Ons hipotese is dus dat die manipulasie van die outofagiese pad die kankerselle sensitiseer tot ʼn lae konsentrasie van sisplatien. Verder, as gevolg van die funksionele interaksie tussen Bcl-2 en Beclin-1, en hul rol in die regulering van outofagie, is verhouding-analises van Beclin-1 tot Bcl-2 uitgevoer met die doel om die rol van outofagie in die sel onder homeostatiese en behandeling/stres kondisies te bepaal. Verder is Bcl-2 bekend daarvoor om ʼn prominente rol te speel in kwaadaardige selle, en die ooruitdrukking daarvan is gevind om weerstandigheid aan te help in ʼn verskeidenheid van kankeragtige sellyne. Ons hipotetiseer dus dat geenonderdrukking van Bcl-2 voor die behandeling met sisplatien die servikale kanker selle sal sensitiseer tot apoptose en ʼn verhoging in die verhouding van Beclin-1/Bcl-2 veroorsaak, wat in die guns van apoptose is. Materiale en Metodes Drie menslike servikale sellyne was gebruik: ʼn nie-kankeragtige servikale epiteel sellyn (Ect/E6E7) en twee kankeragtige servikale sellyne (HeLa en CaSki). Om ʼn konsentrasie van sisplatien te bepaal wat nie-toksies tot die nie-kankeragtige Ect1/E6E7 sellyn is, was ʼn dosisrespons uitgevoer. Met die gebruik van ʼn outofagiese inhibeerder (bafilomycin A1) en ʼn outofagiese induseerder (rapamycin), is die outofagiese-fluks kapasiteite van elke sellyn deur die Western Blotting tegniek geassesseer. Om te bepaal of die gekose konsentrasie van sisplatien outofagie induseer, is vloeisitometrie met ʼn Lysotracker™ kleurstof gebruik, sowel as analises op outofagie proteïenvlakke (LC-3 II, Beclin-1 en p62). Outofagie modulering is behaal deur twee metodes: farmakologiese modulering met twee erkende middels, naamlik bafilomycin A1 en rapamycin, en biologiese manipulasie met die gebruik van ATG5 en mTOR geenonderdrukking. Die effekte van die verskillende behandeling skedules op seldood was geassesseer deur gebruik te maak van PARP en kaspase-3 splitsing deur Western Blotting, kaspase-3/-7 aktiwiteit deur Caspase-Glo ®, PI-insluiting, LDH vrystelling en MTT reduserende kapasiteit. Verder is die effekte van hierdie behandeling skedules op selsiklus progressie ook geanaliseer. Beclin-1 en Bcl-2 uitdrukking was ook bepaal deur Western Blotting en immunohistochemie voor en na behandeling met sisplatien in HeLa en CaSki selle. Om die afhanklikheid van die servikale kankerselle op Bcl-2 na sisplatien behandelings te toets, is Bcl-2 onderdruk deur RNA-inmenging, waarna Beclin-1/Bcl-2 verhouding geassesseer is, sowel as opoptose deur die gebruik van gesplitste PARP analises (Western Blotting) en Caspase-Glo©. Vir die ex vivo analises is biopsies vanaf pasiënte wat roetine kolposkopie en histerektomies ondergaan, verkry (Tygerberg Hospitaal, Tygerberg, Westelike Provinsie). ʼn Totaal van 10 normale, 29 lae-graad plaveisel intraepiteel letsels (LSIL), 33 hoe-graad plaveisel intraepiteel letsels (HSIL) en 13 karsinoom biopsies is verkry vir analises. Die uitdrukkingsprofiel van twee outofagiese merkers (mTOR en LC-3 II), asook een merker vir apoptose (Bcl-2), was geassesseer. Proteïen vlakke was ook deur Western Blotting geanaliseer en deur immunohistochemie bevestig. Resultate Dosisrespons kurwes het getoon dat 15 μM sisplatien nie seldood in die normale sellyn (Ect1/E6E7) geïnduseer het nie, en was daarom gebruik deur die res van hierdie studie. Verder is daar ook gevind dat CaSki selle meer weerstandig tot sisplatien behandelings is wanneer vergelyk word met die HeLa en Ect1/E6E7 selle. Outofagiese-fluks analises het getoon dat, alhoewel al drie sellyne vanaf die serviks afkomstig is, daar verskille is in hul outofagiese-fluks kapasiteit. Daar is ook waargeneem dat die gekose konsentrasie van sisplatien in staat was om outofagie te induseer in al drie sellyne, met HeLa selle wat die mees merkbare respons getoon het. Modulering van outofagie in samewerking met sisplatien behandelings het die volgende onthul: inhibisie van outofagie deur bafilomycin A1 het gelei tot ʼn beduidende verhoging in kaspase-3, PARP splitsing en LDH vrylating in beide servikale kankersellyne. Geenonderdrukking van ATG5 induseer kaspase-3 splitsing en stem ooreen met resultate wat verkry is deur farmakologiese inhibisie van outofagie met bafilomycin A1. Bykomend tot outofagiese indusering, het ʼn lae konsentrasie sisplatien die opregulering van Bcl-2 geïnduseer. Wanneer Bcl-2 geenonderdrukking in hierdie scenario toegepas was, het dit ʼn beduidende verbetering in sisplatien-geïnduseerde apoptose in beide servikale kankersellyne getoon. Analises van die uitdrukkingsprofiel van mTOR en LC-3 in normale, pre-maligne (LSIL en HSIL) en kankeragtige servikale weefsel, het getoon dat outofagie beduidend opgereguleer is in HSILs en servikale karsinome. Verder is Bcl-2 uitdrukking ook gevind om beduidend verhoog te wees in servikale karsinoomweefsel, wat ooreenstem met resultate verkry in ander studies. Gevolgtrekking Outofagiese-fluks kapasiteite tussen die drie sellyne, afkomstig van dieselfde orgaan, toon beduidende verskille. Hierdie bevinding moet in ag geneem word wanneer outofagiese-modulering as ʼn bevorderingsbehandeling gebruik word. Met betrekking tot chemoterapie behandeling in servikale selle; ʼn lae konsentrasie van sisplatien veroorsaak ʼn beduidende indusering van outofagie in kwaadaardige en nie-kwaadaardige serviks-afkomstige sellyne, en dien as ʼn oorlewingsmeganisme. Inhibisie van outofagie met bafilomycin A1 en ATG5 siRNA het hierdie beskermings effek bevestig, aangesien apoptose beduidend verhoog was in beide kankersellyne. Interessant genoeg het rapamycin pre-behandeling tesame met sisplatien nie beduidende vlakke van apoptose in HeLa selle geïnduseer nie. Outofagie induksie mag dalk addisionele beskerming teen die sitotoksiese effekte van sisplatien gebied het. Daarom het die inhibisie van outofagie deur farmakologiese en biologiese inhibering die sitotoksisiteit van ʼn lae konsentrasie sisplatien bevorder, wat ʼn belowende bevinding is vir die toekomstige behandeling van servikale kanker. Bcl-2 opregulering as gevolg van sisplatien behandelings dien ook as beskermings meganisme waarby servikale kankerselle oorleef. Die mate van apoptotiese seldood wat waargeneem word na biologiese inhibering van Bcl-2, wys weer op die feit dat hierdie respons uitgebuit kan word vir die gebruik van laer konsentrasies van sisplatien. Analises van die kliniese monsters het ook die waarde van die in vitro werk versterk: Servikale kanker biopsies het verhoogde uitdrukking van beide LC-3 II en Bcl-2 getoon, wat aandui dat outofagie geïnduseer en apoptose geïnhibeer word. Daar is dus twee nuwe metodes vir die verbetering van sisplatien-toksisiteit in hierdie studie gedemonstreer. Behandeling regimes kan meer gereeld en vir langer tydperke toegepas word, aangesien die newe-effekte van lae-dosis sisplatien behandelings minimaal is.
MRC for funding
Hoyt, Kaleigh. "Raptors and Humans: Exploring Alternative Therapies in Non-Clinical Environments using Birds of Prey." Scholar Commons, 2018. https://scholarcommons.usf.edu/etd/7307.
Повний текст джерелаКниги з теми "Humane Animal Treatment"
The extended circle: A dictionary of humane thought. Fontwell, Sussex: Centaur Press, 1985.
Знайти повний текст джерелаTrumbauer, Lisa. Exploring animal rights and animal welfare. Westport, Conn: Greenwood Press, 2002.
Знайти повний текст джерелаCare, United States Animal and Plant Health Inspection Service Regulatory Enforcement Animal. Humane treatment for horses: How the Horse Protection Act works. Hyattsville, Md.?]: U.S. Dept. of Agriculture, Animal and Plant Health Inspection Service, Regulatory Enforcement Animal Care, 1989.
Знайти повний текст джерелаHyland, J. R. God's covenant with animals: A biblical basis for the humane treatment of all creatures. New York, NY: Lantern Books, 2000.
Знайти повний текст джерелаJory, Brian. The AniCare model of treatment for animal abuse. Washington Grove, Md: Doris Day Animal Foundation and Psychologists for the Ethical Treatment of Animals, 2000.
Знайти повний текст джерелаSateren, Shelley Swanson. The humane societies: A voice for the animals. Parsippany, N.J: Dillon Press, 1997.
Знайти повний текст джерелаHyland, J. R. The slaughter of terrified beasts: A Biblical basis for the humane treatment of animals. Sarasota, Fla: Viatoris Ministries, 1988.
Знайти повний текст джерелаIngrid, Newkirk, ed. 50 awesome ways kids can help animals: Fun and easy ways to be a kind kid. New York: Warner Books, 2006.
Знайти повний текст джерелаNewkirk, Ingrid. Kids can save the animals!: 101 easy things to do. New York, NY: Warner Books, 1991.
Знайти повний текст джерелаPresnall, Judith Janda. Animal actors. San Diego, CA: Kidhaven Press, 2002.
Знайти повний текст джерелаЧастини книг з теми "Humane Animal Treatment"
Centner, Terence J. "The humane treatment of food animals." In Consumers, Meat and Animal Products, 70–80. Milton Park, Abingdon, Oxon ; New York, NY : Routledge, 2019. | Series: Earthscan food and agriculture: Routledge, 2019. http://dx.doi.org/10.4324/9780429430572-7.
Повний текст джерелаCregier, Sharon E. "The Psychology and Ethics of Humane Equine Treatment." In Advances in Animal Welfare Science 1986/87, 77–87. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3331-6_7.
Повний текст джерелаDe Paula Vieira, Andreia, and Raymond Anthony. "Reimagining Human Responsibility Towards Animals for Disaster Management in the Anthropocene." In The International Library of Environmental, Agricultural and Food Ethics, 223–54. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-63523-7_13.
Повний текст джерелаEskens, Erno. "Comment: Animals in ‘Non-Ideal Ethics’ and ‘No-Deal Ethics’." In The International Library of Environmental, Agricultural and Food Ethics, 275–84. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-63523-7_15.
Повний текст джерелаCorley, F. G. "Bites—Human and Animal." In Current Orthopedic diagnosis & treatment, 22–23. London: Current Medicine Group, 2000. http://dx.doi.org/10.1007/978-1-4613-1107-2_12.
Повний текст джерелаCohen, Erik. "Rethinking the animal in tourism studies." In Tourism in development: reflective essays, 245–55. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789242812.0021.
Повний текст джерелаBennett, Richard. "People's preferences in relation to animal welfare." In The economics of farm animal welfare: theory, evidence and policy, 75–97. Wallingford: CABI, 2020. http://dx.doi.org/10.1079/9781786392312.0075.
Повний текст джерелаBlandford, David. "Humane Treatment of Farm Animals." In US Programs Affecting Food and Agricultural Marketing, 471–504. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4930-0_18.
Повний текст джерелаEadie, Edward N. "Immorality of the Treatment of Animals by Humans." In Animal Welfare, 33–43. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-30577-1_4.
Повний текст джерелаMcVety, Dani. "Maintaining the Human-Animal Bond." In Treatment and Care of the Geriatric Veterinary Patient, 7–9. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781119187240.ch2.
Повний текст джерелаТези доповідей конференцій з теми "Humane Animal Treatment"
Mess, Griffin, Rasika Thombre, Max Kerensky, Eli Curry, Fariba Abhabaglou, Safwan Alomari, Henry Brem, Nicholas Theodore, Betty Tyler, and Amir Manbachi. "Designing a Murine Model of Human Glioblastoma Brain Tumor: Development of a Platform for Validation Using Ultrasound Elastography." In 2022 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2022. http://dx.doi.org/10.1115/dmd2022-1025.
Повний текст джерелаWester, J., F. W. J. Van Mensvoort, D. G. Meuleman, H. ten Cate, C. P. Henny, and J. W. ten Cate. "EFFECTS OF ORG 10172, A NOVEL LMW HEPARINOID ON PRIMARY HAEMOSTASIS IN RATS AND HUMANS. A MORPHOLOGICAL STUDY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643238.
Повний текст джерелаVoigt, Elizabeth, Cara F. Buchanan, M. Nichole Rylander, and Pavlos Vlachos. "Blood Flow Characterization in a Perfused Collagen Vessel Bioreactor Using X-Ray Micro-PIV." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80700.
Повний текст джерелаNatarajan, Raghu N., Alejandro Espinoza, and Gunnar B. J. Andersson. "Effect of Needle Puncture Injury on Human Intervertebral Disc Mechanics." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19116.
Повний текст джерелаHerfat, Safa T., Daniel V. Boguszewski, and Jason T. Shearn. "Intact Knee and ACL Forces for the Human and Ovine Model During Simulated In Vivo Human and In Vivo Ovine Motions." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53532.
Повний текст джерелаPitta, Marina Galdino da Rocha, Jordy Silva de Carvalho, Luzilene Pereira de Lima, and Ivan da Rocha Pitta. "iPSC therapies applied to rehabilitation in parkinson’s disease." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.022.
Повний текст джерелаChen, Jing, and Sihong Wang. "Thermal Effects on Osteogenesis of Human Mesenchymal Stem Cells." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80885.
Повний текст джерела"Targeting peroxisomal transport in trypanosoma." In 4th International Conference on Biological & Health Sciences (CIC-BIOHS’2022). Cihan University, 2022. http://dx.doi.org/10.24086/biohs2022/paper.566.
Повний текст джерелаD.V., Rudoy, Olshevskaya A.V., Pavlov P.D., Odabashyan M.Yu., Ugrekhelidze N.T., and Egyan M.A. "LAVENDER AND ITS INTERACTION WITH THE NERVOUS SYSTEM." In INTERNATIONAL FORUM "YOUTH IN THE AGRIBUSINESS". DSTU-Print, 2022. http://dx.doi.org/10.23947/young.2022.41-44.
Повний текст джерелаMarshall, Lauren, Andra Frost, Tim Fee, and Joel Berry. "Assembly and Characterization of 3D, Vascularized Breast Cancer Tissue Mimics." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14199.
Повний текст джерелаЗвіти організацій з теми "Humane Animal Treatment"
Huijser, Marcel, E. R. Fairbank, and K. S. Paul. Best Practices Manual to Reduce Animal-Vehicle Collisions and Provide Habitat Connectivity for Wildlife. Nevada Department of Transportation, September 2022. http://dx.doi.org/10.15788/ndot2022.2.
Повний текст джерелаJames, Christian, Ronald Dixon, Luke Talbot, Stephen James, Nicola Williams, and Bukola Onarinde. Assessing the impact of heat treatment on antimicrobial resistant (AMR) genes and their potential uptake by other ‘live’ bacteria. Food Standards Agency, August 2021. http://dx.doi.org/10.46756/sci.fsa.oxk434.
Повний текст джерелаCahaner, Avigdor, Susan J. Lamont, E. Dan Heller, and Jossi Hillel. Molecular Genetic Dissection of Complex Immunocompetence Traits in Broilers. United States Department of Agriculture, August 2003. http://dx.doi.org/10.32747/2003.7586461.bard.
Повний текст джерелаHansen, Peter J., Zvi Roth, and Jeremy J. Block. Improving oocyte competence in dairy cows exposed to heat stress. United States Department of Agriculture, January 2014. http://dx.doi.org/10.32747/2014.7598163.bard.
Повний текст джерелаNoga, Edward J., Angelo Colorni, Michael G. Levy, and Ramy Avtalion. Importance of Endobiotics in Defense against Protozoan Ectoparasites of Fish. United States Department of Agriculture, September 2003. http://dx.doi.org/10.32747/2003.7586463.bard.
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