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Готові списки джерел за темами / Energy metabolism

Добірка наукової літератури з теми "Energy metabolism"

Автор: Grafiati

Опубліковано: 4 червня 2021

Оновлено: 4 травня 2024

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Статті в журналах з теми "Energy metabolism":

1

Perera, PAJ, and Faiz MMT Marikar. "Energy Metabolism." Bangladesh Journal of Medical Biochemistry 6, no. 2 (January 13, 2014): 68–76. http://dx.doi.org/10.3329/bjmb.v6i2.17646.

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This review considers how our understanding of energy utilized by energy metabolism has progressed since the pioneering work on this topic in the late 1960s and early 1970s. Research has been stimulated by a desire to understand how metabolic events contribute to the development of the body into the different phases, the need of considering health with which to improve the success of implication on public health. Nevertheless, considerable progress has been made in defining the roles of the traditional nutrients: pyruvate, glucose, lactate and amino acids; originally considered as energy sources and biosynthetic precursors, but now recognised as having multiple, overlapping functions. Other nutrients; notably, lipids, are beginning to attract the attention they deserve. The review concludes by up-dating the state of knowledge of energy metabolism in the early 1970s and listing some future research questions. DOI: http://dx.doi.org/10.3329/bjmb.v6i2.17646Bangladesh J Med Biochem 2013; 6(2): 68-76

2

Almeida Castro, Luis Henrique, Leandro Rachel Arguello, Nelson Thiago Andrade Ferreira, Geanlucas Mendes Monteiro, Jessica Alves Ribeiro, Juliana Vicente de Souza, Sarita Baltuilhe dos Santos, et al. "Energy metabolism." International Journal for Innovation Education and Research 8, no. 9 (September 1, 2020): 359–68. http://dx.doi.org/10.31686/ijier.vol8.iss9.2643.

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Most animal cells are able to meet their energy needs from the oxidation of various types of compounds: sugars, fatty acids, amino acids, but some tissues and cells of our body depend exclusively on glucose and the brain is the largest consumer of all. That is why the body has mechanisms in order to keep glucose levels stable. As it decreases, the degradation of hepatic glycogen occurs, which maintains the appropriate levels of blood glucose allowing its capture continues by those tissues, even in times of absence of food intake. But this reserve is limited, so another metabolic pathway is triggered for glucose production, which occurs in the kidneys and liver and is called gluconeogenesis, which means the synthesis of glucose from non-glucose compounds such as amino acids, lactate, and glycerol. Most stages of glycolysis use the same enzymes as glycolysis, but it makes the opposite sense and differs in three stages or also called deviations: the first is the conversion of pyruvate to oxaloacetate and oxaloacetate to phosphoenolpyruvate. The second deviation is the conversion of fructose 1,6 biphosphate to fructose 6 phosphate and the third and last deviation is the conversion of glucose 6 phosphate to glucose.

3

Flight, Monica Hoyos. "Shifting energy metabolism." Nature Reviews Drug Discovery 9, no. 4 (April 2010): 272. http://dx.doi.org/10.1038/nrd3146.

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4

Gutierrez, Guillermo, Fernando Palizas, and Carlo E. Marini. "Cellular Energy Metabolism." Chest 97, no. 4 (April 1990): 975–82. http://dx.doi.org/10.1378/chest.97.4.975.

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5

Lochner, A. "Myocardial energy metabolism." Cardiovascular Drugs and Therapy 4, no. 3 (May 1990): 756. http://dx.doi.org/10.1007/bf01856567.

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6

Crunkhorn, Sarah. "Disrupting energy metabolism." Nature Reviews Drug Discovery 17, no. 10 (October 2018): 708. http://dx.doi.org/10.1038/nrd.2018.172.

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7

Blum, J. Joseph. "Energy metabolism inLeishmania." Journal of Bioenergetics and Biomembranes 26, no. 2 (April 1994): 147–55. http://dx.doi.org/10.1007/bf00763063.

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8

Alcaraz, Miquel. "Pavlova E.V. Movement and energy metabolism of marine planktonic organisms." Scientia Marina 70, no. 4 (December 30, 2006): 767–68. http://dx.doi.org/10.3989/scimar.2006.70n4767.

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9

Lee, Sujin, and Yumie Rhee. "Bone and Energy Metabolism." Journal of Korean Diabetes 14, no. 4 (2013): 174. http://dx.doi.org/10.4093/jkd.2013.14.4.174.

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10

Nieuwenhuizen, Arie G., and Evert M. van Schothorst. "Energy Metabolism and Diet." Nutrients 13, no. 6 (June 1, 2021): 1907. http://dx.doi.org/10.3390/nu13061907.

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Дисертації з теми "Energy metabolism":

1

Darcy, Justin. "Energy metabolism and aging." OpenSIUC, 2017. https://opensiuc.lib.siu.edu/dissertations/1430.

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Ames dwarf mice have a spontaneous homozygous Prophet of Pituitary Factor 1 (Prop1) loss-of-function mutation. The Prop1 mutation results in a lack of differentiation of lactotrophs, thyrotrophs, and somatotrophs in the anterior pituitary. Without these endocrine cell types, Ames dwarf mice have essentially no circulating levels of growth hormone (GH), thyroid-stimulating hormone (TSH), and prolactin, and exhibit downstream hormonal deficiencies including insulin-like growth factor 1 (IGF-1), 3’,3,5-triiodothyronine (T3), and thyroxine (T4). Ames dwarf mice are exceptionally long-lived (40% to over 60% depending on sex and diet). They are also extremely insulin sensitive, have a delayed incidence of cancer, and have improved energy metabolism. While the extended lifespan and the many characteristics of an extended healthspan have been known for some time in Ames dwarf mice, the revelation that dwarf mice have improved energy metabolism was less than a decade ago. This finding came about at the molecular level (improved efficiency of the electron transport chain) and at the whole-animal level (increased oxygen consumption and decreased respiratory quotient). To date, however, few studies have been directed at furthering our understanding of the possible mechanism(s) by which Ames dwarf mice have altered energy metabolism. The goal of the studies presented in this dissertation is to delineate these mechanisms and to lay the groundwork for future studies that broaden our understanding of the role(s) of energy metabolism in the aging process. Project 1 examines the effects of early-life T4 replacement therapy in Ames dwarf mice. Previous work established that life-long T4 replacement therapy shortens lifespan in Snell dwarf mice (these mice have endocrine deficits that are essentially identical to those of Ames dwarf mice), while short-term replacement therapy during the early postnatal period of Ames dwarf mice does not. We hypothesized that T4 replacement therapy causes transient impairment of energy metabolism, which is why long-term T4 replacement therapy shortens longevity, and short-term replacement therapy does not. Supporting our hypothesis, we showed that short-term T4 replacement therapy during the early postnatal period transiently impaired energy metabolism as measured by indirect calorimetry. Following early-life T4 replacement therapy, we also observed an accelerated rate of sexual development, as well as lasting effects on bone physiology. Project 2 continued our investigation of energy metabolism by examining a highly metabolic tissue: brown adipose tissue (BAT), which is responsible for non-shivering thermogenesis. Our laboratory has already demonstrated functional alterations in visceral adipose tissue of Ames dwarf mice, and given the altered energy metabolism of Ames dwarf mice, we hypothesized that BAT may also be functionally unique compared to their normal littermates. Supporting our hypothesis, we observed alterations in gene expression, relative weight, and histological structure of BAT in Ames dwarf mice. Moreover, surgical removal of the interscapular BAT depot resulted in a unique physiological response, where Ames dwarf mice lost adiposity in their subcutaneous, perirenal, and epididymal white adipose tissue depots, thus contrasting with normal mice that gained adiposity. Project 3 built upon the findings of our second study, where we continued to examine the role of non-shivering thermogenesis and core body temperature in Ames dwarf mice. To further understand the role of non-shivering thermogenesis in glucose homeostasis and energy metabolism, we housed a cohort of Ames dwarf mice and their normal littermates at room temperature (23˚C), and another cohort at thermoneutrality (for mice this is 30˚C). We found that Ames dwarf mice placed at thermoneutrality had impaired glucose homeostasis and energy metabolism. This is an important finding because we and others believe both of these metabolic processes are important factors for longevity. Taken together, these studies indicate that the improved energy metabolism in Ames dwarf mice is dependent upon several factors, including a loss of thyroid hormone signaling and improved non-shivering thermogenesis.

2

Bojanowska, Magdalena. "Wpływ opóźniania terminu pierwszego unasieniania krów z zaburzeniami metabolizmu energetycznego na ich płodność." Rozprawa doktorska, Uniwersytet Technologiczno-Przyrodniczy w Bydgoszczy, 2018. http://dlibra.utp.edu.pl/Content/1229.

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Zasadnicznym celem przeprowadzonych badań była ocena skuteczności wykorzystania danych z okresowej kontroli użytkowości mlecznej w typowaniu krów z zaburzeniami metabolizmu energetycznego i wpływie opóżnienia u nich terminu pierwszej inseminacji na wskaźniki rozrodcze stada
The aim of the research was to assess the effectiveness of the use of data from periodic control of dairy utility in the selection of cows with energy metabolism disturbances and the impact of their delay in the first insemination on the reproductive indicators of the herd

3

Fredrix, Elisabeth Wilhelmina Hubertina Maria. "Energy metabolism in cancer patients." Maastricht : Maastricht : Datawyse ; University Library, Maastricht University [Host], 1990. http://arno.unimaas.nl/show.cgi?fid=5567.

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4

Vasquez-Velasquez, Jose Lionel. "The energy metabolism of children." Thesis, University of Cambridge, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.315979.

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5

Kotwica, Aleksandra Olga. "Dietary nitrate and the modulation of energy metabolism in metabolic syndrome." Thesis, University of Cambridge, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708924.

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6

Schrauwen, Patrick. "Determinants of energy and substrate metabolism." Maastricht : Maastricht : Shaker ; University Library, Maastricht University [Host], 1998. http://arno.unimaas.nl/show.cgi?fid=8500.

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7

Etten, Ludovicus Maria Leonardus Anna van. "Weight training: implications for energy metabolism." Maastricht : Maastricht : Universiteit Maastricht ; University Library, Maastricht University [Host], 1997. http://arno.unimaas.nl/show.cgi?fid=6819.

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8

Lambert, D. "Perioperative energy metabolism in hepatobiliary disease." Thesis, University of Newcastle Upon Tyne, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.234422.

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9

Smith, Ruth Deborah. "Potassium intake, growth and energy metabolism." Thesis, University of Southampton, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295704.

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10

Shelton, Laura Marie. "Targeting Energy Metabolism in Brain Cancer." Thesis, Boston College, 2010. http://hdl.handle.net/2345/1183.

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Thesis advisor: Thomas N. Seyfried
It has long been posited that all cancer cells are dependent on glucose for energy, termed the "Warburg Effect". As a result of an irreversible injury to the mitochondria, cancer cells are less efficient in aerobic respiration. Therefore, calorie restriction was thought to be a natural way to attenuate tumor growth. Calorie restriction lowers blood glucose, while increasing the circulation of ketone bodies. Ketone bodies are metabolized via oxidative phosphorylation in the mitochondria. Only cells that are metabolically capable of aerobic respiration will thus be able to acquire energy from ketone bodies. To date, calorie restriction has been shown to greatly reduce tumor growth and angiogenesis in the murine CT2A, EPEN, and human U87 brain tumor models. Using the novel VM-M3 model for invasive brain cancer and systemic metastatic cancer, I found that though calorie restriction had some efficacy in reducing brain tumor invasion and primary tumor size, metastatic spread was unaffected. Using a bioluminescent-based ATP assay, I determined the viability of metastatic mouse VM-M3 tumor cells grown in vitro in serum free medium in the presence of glucose alone (25 mM), glutamine alone (4 mM), or in glucose + glutamine. The VM-M3 cells could not survive on glucose alone, but could survive in glutamine alone indicating an absolute requirement for glutamine in these metastatic tumor cells. Glutamine could also maintain viability in the absence of glucose and in the presence of the F1 ATPase inhibitor oligomycin. Glutamine could not maintain viability in the presence of the Krebs (TCA) cycle enzyme inhibitor, 3-nitropropionic acid. The data indicate that glutamine can provide ATP for viability in the metastatic VM-M3 cells through Krebs cycle substrate level phosphorylation in the absence of energy from either glycolysis or oxidative phosphorylation. I therefore developed a metabolic therapy that targeted both glucose and glutamine metabolism using calorie restriction and 6-diazo-5-oxo-L-norleucine (DON), a glutamine analog. Primary tumor growth was about 20-fold less in DON treated mice than in untreated control mice. I also found that DON treatment administered alone or in combination with CR inhibited metastasis to liver, lung, and kidney as detected by bioluminescence imaging and histology. Although DON treatment alone did not reduce the incidence of tumor metastasis to spleen compared to the controls, DON administered together with CR significantly reduced the incidence of metastasis to the spleen, indicating a diet/drug synergy. In addition, the phagocytic capabilities of the VM-M3 tumor cells were enhanced during times of energy stress. This allowed for the digestion of engulfed material to be used in energy production. My data provide proof of concept that metabolic therapies targeting both glucose and glutamine metabolism can manage systemic metastatic cancer. Additionally, due to the phagocytic properties of the VM-M3 cell line also seen in a number of human metastatic cancers, I suggest that a unique therapy targeting metabolism and phagocytosis will be required for effective management of metastatic cancer
Thesis (PhD) — Boston College, 2010
Submitted to: Boston College. Graduate School of Arts and Sciences
Discipline: Biology

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Книги з теми "Energy metabolism":

1

McCandless, David W., ed. Cerebral Energy Metabolism and Metabolic Encephalopathy. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-1209-3.

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2

1941-, McCandless David W., ed. Cerebral energy metabolism and metabolic encephalopathy. New York: Plenum Press, 1985.

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3

Jong, Jan Willem de, 1942-, ed. Myocardial energy metabolism. Dordrecht: Nijhoff, 1988.

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4

De Jong, Jan Willem, ed. Myocardial Energy Metabolism. Dordrecht: Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-1319-6.

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5

Hirrlinger, Johannes, and Helle S. Waagepetersen, eds. Brain Energy Metabolism. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1059-5.

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6

Hirrlinger, Johannes, and Helle S. Waagepetersen. Brain energy metabolism. New York: Heidelberg, 2014.

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7

Boulton, Alan A., Glen B. Baker, and Roger Butterworth. Carbohydrates and Energy Metabolism. New Jersey: Humana Press, 1989. http://dx.doi.org/10.1385/0896031438.

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8

Donohoue, Patricia A., ed. Energy Metabolism and Obesity. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-139-4.

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9

A, Little R., and Wernerman J, eds. Energy metabolism in trauma. London: Baillière Tindall, 1997.

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10

(Project), BIOTOL, Open Universiteit (Heerlen Netherlands), and Thames Polytechnic, eds. Energy sources for cells. Oxford [England]: Butterworth-Heinemann, 1992.

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Частини книг з теми "Energy metabolism":

1

Hawkins, Richard. "Cerebral Energy Metabolism." In Cerebral Energy Metabolism and Metabolic Encephalopathy, 3–23. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-1209-3_1.

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2

Green, J. Hilary. "Energy metabolism." In The Autonomic Nervous System and Exercise, 72–103. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4899-2919-8_4.

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3

Whittow, G. C. "Energy Metabolism." In Avian Physiology, 253–68. New York, NY: Springer New York, 1986. http://dx.doi.org/10.1007/978-1-4612-4862-0_10.

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4

Giudice, Giovanni. "Energy Metabolism." In The Sea Urchin Embryo, 73–78. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70431-4_3.

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5

Ellenbroek, Bart, Alfonso Abizaid, Shimon Amir, Martina de Zwaan, Sarah Parylak, Pietro Cottone, Eric P. Zorrilla, et al. "Energy Metabolism." In Encyclopedia of Psychopharmacology, 481. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_1388.

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6

Köhler, Peter, and Louis Tielens. "Energy Metabolism." In Encyclopedia of Parasitology, 902–17. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_1057.

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7

Proske, Uwe, David L. Morgan, Tamara Hew-Butler, Kevin G. Keenan, Roger M. Enoka, Sebastian Sixt, Josef Niebauer, et al. "Energy Metabolism." In Encyclopedia of Exercise Medicine in Health and Disease, 293–97. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_66.

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8

Wu, Guoyao. "Energy Metabolism." In Principles of Animal Nutrition, 449–78. Boca Raton : Taylor & Francis, 2018.: CRC Press, 2017. http://dx.doi.org/10.1201/9781315120065-8.

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9

McCandless, David W., and Marc S. Abel. "Hypoglycemia and Cerebral Energy Metabolism." In Cerebral Energy Metabolism and Metabolic Encephalopathy, 27–41. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-1209-3_2.

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10

Amaral, Ana I., Paula M. Alves, and Ana P. Teixeira. "Metabolic Flux Analysis Tools to Investigate Brain Metabolism In Vitro." In Brain Energy Metabolism, 107–44. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1059-5_5.

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Тези доповідей конференцій з теми "Energy metabolism":

1

Tsuda, V., S. DeCamp, N. C. Ogassavara, J. Mitchel, S. Koehler, J. P. Butler, and J. J. Fredberg. "Energy Metabolism and Unjamming." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5655.

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2

Yuan, Tai-Yi, Hanan N. Fernando, Jessica Czamanski, Chong Wang, Wei Yong Gu, and Chun-Yuh Huang. "Effects of Static Compression on Energy Metabolism of Porcine Intervertebral Disc." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19600.

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Degeneration of the intervertebral disc (IVD) has been associated with low back pain, which is one of the major socio-economic problems in the United States. Since IVD is the largest avascular cartilaginous structure in the human body, poor nutrient supply has been suggested as a potential mechanism for IVD degeneration. Biosynthesis of extracellular matrix is an energy demanding process which is required to maintain tissue integrity [1]. Cells consume glucose and oxygen to produce adenosine triphosphate (ATP), the main energy form in cells. Glycolysis, the primary metabolic pathway for production of ATP in IVD cells, is strongly regulated by local oxygen concentration and pH (which is governed by lactate concentration) [2]. Therefore, energy metabolism may play an important role in the malnutrition pathway leading to IVD degeneration. The objective of this study was to investigate the effect of mechanical loading on cellular energy metabolism in whole disc and in agarose gels.

3

Карпин, Владимир Александрович, and Наргиз Мираддин кызы Джафарова. "SELF-REGULATION OF THE BIOLOGICAL PROCESSES: COUPLING OF METABOLISM AND ENERGY." In Психология. Спорт. Здравоохранение: сборник избранных статей по материалам Международной научной конференции (Санкт-Петербург, Апрель 2022). Crossref, 2022. http://dx.doi.org/10.37539/psm302.2022.49.17.004.

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Фундаментальным движением материи, первоосновой ее организации является колебательный принцип. Организация живых организмов, появившихся в таком неорганическом мире, также стала формироваться на этой основе. Важнейшим общим проявлением жизни является обмен веществ и энергии. Колебательное движение является сопрягающим фактором метаболических и энергетических процессов. The fundamental movement of matter, the fundamental basis of its organization, is the oscillatory principle. The organization of living organisms that appeared in such an inorganic world also began to form on this basis. The most important general manifestation of life is the metabolism of substances and energy. Oscillatory motion is a coupling factor of metabolic and energy processes.

4

Noack, Raymond, Chetan Manjesh, Miklos Ruszinko, Hava Siegelmann, and Robert Kozma. "Resting state neural networks and energy metabolism." In 2017 International Joint Conference on Neural Networks (IJCNN). IEEE, 2017. http://dx.doi.org/10.1109/ijcnn.2017.7965859.

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5

Cai, Jian-Guang, and Xin-Kang Zhang. "Energy Metabolism and Nutrition Supplement of Aerobics." In 2015 International Conference on Medicine and Biopharmaceutical. WORLD SCIENTIFIC, 2016. http://dx.doi.org/10.1142/9789814719810_0159.

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6

Morozov, G. A., and P. P. Krynitskiy. "Microwave field energy as baker's yeast metabolism regulator." In 2015 International Conference on Antenna Theory and Techniques (ICATT). IEEE, 2015. http://dx.doi.org/10.1109/icatt.2015.7136888.

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7

Kounelakis, M. G., M. E. Zervakis, G. C. Giakos, C. Narayan, S. Marotta, D. Natarajamani, G. J. Postma, L. M. C. Buydens, and X. Kotsiakis. "Targeting brain gliomas energy metabolism for classification purposes." In 2010 IEEE International Conference on Imaging Systems and Techniques (IST). IEEE, 2010. http://dx.doi.org/10.1109/ist.2010.5548526.

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8

Al-GHamdi, Sami G., and Abdulrahman AL-Tamimi. "Energy Metabolism Analysis in Qatar From Socioeconomic Dimensions." In Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2018. http://dx.doi.org/10.5339/qfarc.2018.eepd1117.

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9

DeCamp, S., N. C. Ogassavara, and J. J. Fredberg. "Unjamming and Energy Metabolism in the Epithelial Layer." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a7322.

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Chong, Cher-Rin, Mark Cole, Carolyn Carr, Henry Lee, Brianna Stubbs, Azrul bin Abdul Kadir, Rhys Evans, Pete Cox, and Kieran Clarke. "P22 Cardiac energy metabolism increases with ketone oxidation." In British Society for Cardiovascular Research, Autumn Meeting 2017 ‘Cardiac Metabolic Disorders and Mitochondrial Dysfunction’, 11–12 September 2017, University of Oxford. BMJ Publishing Group Ltd and British Cardiovascular Society, 2018. http://dx.doi.org/10.1136/heartjnl-2018-bscr.27.

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Звіти організацій з теми "Energy metabolism":

1

Corscadden, Louise, and Anjali Singh. Metabolism And Measurable Metabolic Parameters. ConductScience, December 2022. http://dx.doi.org/10.55157/me20221213.

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Анотація:

Metabolism is the sum of chemical reactions involved in sustaining the life of organisms.[1] It constantly provides your body with the energy to perform essential functions. The process is categorized into two groups:[2] Catabolism: It’s the process of breaking down molecules to obtain energy. For example, converting glucose to pyruvate by cellular respiration. Anabolism: It’s the process of synthesis of compounds required to run the metabolic process of the organisms. For example, carbohydrates, proteins, lipids, and nucleic acids.[2] Metabolism is affected by a range of factors, such as age, sex, muscle mass, body size, and physical activity affect metabolism or BMR (the basal metabolic rate). By definition, BMR is the minimum amount of calories your body requires to function at rest.[2] Now, you have a rough idea about the concept. But, you might wonder why you need to study it. What and how metabolic parameters are measured to determine the metabolism of the organism? Find the answer to all these questions in this article.

2

Glaser, M. Cellular energy metabolism. Office of Scientific and Technical Information (OSTI), June 1991. http://dx.doi.org/10.2172/5714213.

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3

Popov, V. S., N. V. Vorobeva, and G. A. Svazlian. The relationship of energy metabolism and metabolism in pigs. Вестник Курской государственной сельскохозяйственной академии, 2019. http://dx.doi.org/10.18411/issn1997-0749.2019-03-74-79.

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4

Aulick, Louis H. Effects of Wound Bacteria on Postburn Energy Metabolism. Fort Belvoir, VA: Defense Technical Information Center, March 1990. http://dx.doi.org/10.21236/ada242721.

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5

Murugesan, G. Raj, and Michael E. Persia. New Model for Examining the Energy Metabolism of Laying Hens. Ames (Iowa): Iowa State University, January 2013. http://dx.doi.org/10.31274/ans_air-180814-188.

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6

Jacobs, Ira. Energy Metabolism in Cold-Stressed Females: Implications for Predictive Modeling. Fort Belvoir, VA: Defense Technical Information Center, October 1997. http://dx.doi.org/10.21236/ada338905.

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7

Hompodoeva, U. Features of energy metabolism in the young Yakut horses in winter. ООО «Информационно-консалтинговый центр», 2019. http://dx.doi.org/10.18411/konevodstvo.2019.6.70rus.

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8

Morfin, C., and G. G. Loots. Characterizing the role of Mef2c in regulating osteoclast differentiation and energy metabolism. Office of Scientific and Technical Information (OSTI), April 2018. http://dx.doi.org/10.2172/1459127.

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9

Overbeek, Ross. An Integrative Approach to Energy Carbon and Redox Metabolism In Cyanobacterium Synechocystis. Office of Scientific and Technical Information (OSTI), June 2003. http://dx.doi.org/10.2172/824924.

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10

Glaser, M. Cellular energy metabolism. Final technical report, May 1, 1987--April 30, 1991. Office of Scientific and Technical Information (OSTI), June 1991. http://dx.doi.org/10.2172/10127387.

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