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Статті в журналах з теми "Endocannabinois"
Fonseca, B. M., G. Correia-da-Silva, M. Almada, M. A. Costa, and N. A. Teixeira. "The Endocannabinoid System in the Postimplantation Period: A Role during Decidualization and Placentation." International Journal of Endocrinology 2013 (2013): 1–11. http://dx.doi.org/10.1155/2013/510540.
Повний текст джерелаLee, Yunna, Jeongbin Jo, Hae Young Chung, Charalabos Pothoulakis, and Eunok Im. "Endocannabinoids in the gastrointestinal tract." American Journal of Physiology-Gastrointestinal and Liver Physiology 311, no. 4 (October 1, 2016): G655—G666. http://dx.doi.org/10.1152/ajpgi.00294.2015.
Повний текст джерелаSuchopár, Josef, Zdeněk Laštůvka, Simona Mašková, Miroslava Alblová, and Antonín Pařízek. "Endocannabinoids." Česká gynekologie 86, no. 6 (December 21, 2021): 414–20. http://dx.doi.org/10.48095/cccg2021414.
Повний текст джерелаAyakannu, Thangesweran, Anthony H. Taylor, Timothy H. Marczylo, Jonathon M. Willets, and Justin C. Konje. "The Endocannabinoid System and Sex Steroid Hormone-Dependent Cancers." International Journal of Endocrinology 2013 (2013): 1–14. http://dx.doi.org/10.1155/2013/259676.
Повний текст джерелаKeereetaweep, Jantana, and Kent D. Chapman. "Lipidomic Analysis of Endocannabinoid Signaling: Targeted Metabolite Identification and Quantification." Neural Plasticity 2016 (2016): 1–13. http://dx.doi.org/10.1155/2016/2426398.
Повний текст джерелаChan, Hsiu-Wen, Natalie C. McKirdy, Hassendrini N. Peiris, Gregory E. Rice, and Murray D. Mitchell. "The role of endocannabinoids in pregnancy." REPRODUCTION 146, no. 3 (September 2013): R101—R109. http://dx.doi.org/10.1530/rep-12-0508.
Повний текст джерелаFriend, Lindsey, Ryan Williamson, Collin Merrill, Scott Newton, Michael Christensen, Jake Petersen, Bridget Wu, Isaac Ostlund, and Jeffrey Edwards. "Hippocampal Stratum Oriens Somatostatin-Positive Cells Undergo CB1-Dependent Long-Term Potentiation and Express Endocannabinoid Biosynthetic Enzymes." Molecules 24, no. 7 (April 3, 2019): 1306. http://dx.doi.org/10.3390/molecules24071306.
Повний текст джерелаKano, Masanobu, Takako Ohno-Shosaku, Yuki Hashimotodani, Motokazu Uchigashima, and Masahiko Watanabe. "Endocannabinoid-Mediated Control of Synaptic Transmission." Physiological Reviews 89, no. 1 (January 2009): 309–80. http://dx.doi.org/10.1152/physrev.00019.2008.
Повний текст джерелаEhrenkranz, Joel, and Michael A. Levine. "Bones and Joints: The Effects of Cannabinoids on the Skeleton." Journal of Clinical Endocrinology & Metabolism 104, no. 10 (August 8, 2019): 4683–94. http://dx.doi.org/10.1210/jc.2019-00665.
Повний текст джерелаWang, Yanqing, and Brian D. Burrell. "Differences in chloride gradients allow for three distinct types of synaptic modulation by endocannabinoids." Journal of Neurophysiology 116, no. 2 (August 1, 2016): 619–28. http://dx.doi.org/10.1152/jn.00235.2016.
Повний текст джерелаДисертації з теми "Endocannabinois"
Serzysko, Malgorzata. "Endocannabinoids and excitotoxicity: lessons from hypoglossal motoneurons." Doctoral thesis, SISSA, 2015. http://hdl.handle.net/20.500.11767/3908.
Повний текст джерелаMateu, Codina Gerard Àngel. "Factores genéticos en patología dual." Doctoral thesis, Universitat Autònoma de Barcelona, 2019. http://hdl.handle.net/10803/667189.
Повний текст джерелаThe term dual pathology describes the coexistence of one or more mental illnesses and substance use disorders which is very common and is often associated with higher psychiatric severity, and worse medical conditions and social status. Likewise, there is now an important body of research on the important role that the endocannabinoid system (eCB) could play on the neurobiological bases of various neuropsychiatric conditions, such as anxiety, mood disorders or schizophrenia, in addition to several substance use disorders. The purpose of this study is to investigate the association between various polymorphisms of the genes encoding for some endocannabinoid system proteins (cannabinoid receptor 1 [CNR1] and fatty acid amide hydrolase [FAAH]) according to (1) the presence or absence of substance use disorders in the whole sample and (2) according to the presence or absence of dual pathology within the group of subjects diagnosed with a substance use disorder. This is a case-control study with a total sample size of 675 subjects, of whom 362 are controls (C; individuals without any psychiatric diagnosis) and 313 cases (Cs; individuals with a substance use disorder without any co-morbid neuropsychiatric condition [TCS] and those individuals with a substance use disorder with co-morbid neuropsychiatric condition [PD]). A comprehensive comparison including demographics, parental and personal background of psychiatric and substance use disorders (according to DSM-IV-R and performed by using the Spanish validated version of Psychiatric Research Interview for Substance and Mental Disorders (PRISM-IV)) and personality characteristics (evaluated using the Spanish version of the Cloninger’s Temperament and Character Inventory (TCI-R))was performed between both Cs and C groups and between TCS and PD groups. 768 single-nucleotide polymorphisms (SNPs) were genotyped wich were used as genetic markers in an approximation of candidate gene study. Simple statistical analysis of polymorphisms, genes and haplotypes and subsequent multivariate analysis were performed. We will relate the results of genetic analysis with sociodemographic and clinical characteristics. The phenotypical results of the comparison between both Cs and C groups showed that Cs subjects had greater prevalence of family background of mental illness and substance use disorders and a lower score on reward dependence, self-directness, cooperativeness and self-transcendence as regarding on personality traits. Likewise, TCS and PD groups contrast show that PD subjects had higher prevalence of alcohol and cocaine use disorders and lower prevalence of cannabis use disorder, as well as a higher prevalence of family background of mental illness and substance use disorders. In addition, TCS subjects had higher score on harm avoidance and self-transcendence but, interestingly, PD subjects score significantly lower on self-directness. We have found a significant prevalence of the AA/AC variants of SNP rs324420 and TT of SNP rs11576941 (both SNPs belonging to the same haplotype of the FAAH gene) in the Cs group in the association study of CS vs. C. The prevalence was significant in the case of the TT variant of the SNP rs11576941 belonging to the FAAH gene in the TCS group in the association study of TCS vs. C. Furthermore, a subgroup of subjects of the PD group with significantly lower scores in the personality dimension harm avoidance that are characterized by being homozygous GG for the SNP rs806380 of the CNR1 gene have been detected. This allelic variation has been associated with a greater sensitivity for positive stimuli and could represent an endophenotypic marker for those subjects with higher emotional instability and, therefore, with greater clinical potential severity.
Strohm, Daniela. "Modulation der Insulinsignalgebung durch Prostaglandin E2 und Endocannabinoide." Phd thesis, Universität Potsdam, 2010. http://opus.kobv.de/ubp/volltexte/2011/4967/.
Повний текст джерелаThe obesity related insulin resistance is accompanied by a low grade inflammation. In response to inflammatory stimuli, PGE2 is released from Kupffer cells and signals through four G-Protein coupled PGE2-receptors (EP1-EP4). Previous work showed that PGE2 attenuated insulin signaling in rat hepatocytes through an EP3ß- and ERK1/2-dependent mechanism. Since EP-receptor expression on hepatocytes varies between species and physiological conditions, the effect of the individual EP receptor subtypes on insulin signaling was studied in hepatoma cell lines expressing individual EP receptor subtypes. HepG2 cells lacking functional EP-receptors, and derivatives stably expressing either EP1 receptor (HepG2-EP1), EP3ß receptor (HepG2-EP3ß) or EP4 receptor (HepG2-EP4) and Fh-hTert cells expressing EP2- and EP4-receptor were pre-incubated with PGE2 for 330 min to mimic the sub-acute inflammation. The cells were subsequently stimulated with insulin for 15 min. Akt and ERK1/2 activation was determined by Western Blotting with phospho-specific antibodies. PGE2 inhibited insulin stimulated Akt phosphorylation in all cell lines expressing EP receptors, except in HepG2 cells which are lacking functional EP receptors. PGE2 increased insulin stimulated phosphorylation of the serine/threonine kinase ERK1/2 in all EP R expressing HepG2 cell lines except in Fh-hTert cells. In HepG2-EP1 and HepG2 EP3ß cells PGE2 increased the serine phosphorylation of the insulin receptor substrate, presumably through an ERK1/2 activation. This IRS-serine phosphorylation leads to attenuation of insulin signal transduction. Inhibiting ERK1/2 activation with a specific inhibitor attenuated the PGE2-dependent inhibition of insulin signal transmission in HepG2 EP3ß cells to some extent. ERK1/2 activation in these cells seems to be of major importance for the observed attenuation of insulin stimulated Akt phosphorylation. Application of inhibitors in the other cell lines stably expressing EP receptors provided evidence that other mechanisms contributed to the attenuation of insulin signaling. Insulin signal transduction in Fh-hTert cells by PGE2 was apparently blocked by an ERK1/2-independent mechanism. Increased PGE2 production during obesity is not limited to the periphery. Signs of inflammation have been detected in the hypothalamus, which might be associated with an increased PGE2 production. Therefore, the EP receptor profile of primary neurons as well as neuronal cell models was characterised in order to investigate, whether PGE2 attenuates insulin signal transduction in neuronal cells similar to what was observed in hepatocytes. Pre-incubation with PGE2 did not attenuate insulin stimulated Akt phosphorylation in all neuronal cells. The EP receptor profile in SH SY5Y cells and in primary neurons varied depending on the differentiation status of the cells. Although Akt-kinase was phosphorylated in response to insulin stimulation in all neuronal cells studied, gene expression of insulin target genes (POMC, AgRP) was not modulated by insulin. This might be due to the low level of differentiation of the investigated cells. In the course of obesity, an over-activation of the endocannabinoid system is detected. Since endocannabinoid receptors are related to EP receptors, it was investigated whether endocannabinoids can interfere with insulin signaling in a similar way as PGE2. Pre-incubation of the neuronal cell line N 41 for 330 min with an endocannabinoid receptor agonist, increased insulin stimulated Akt phosphorylation. This implies an insulin sensitising effect of endocannabinoids. This is contradictory to the endocannabinoid-dependent insulin resistance described in the literature and might be caused by indirect endocannabinoid-triggered mechanisms.
LISAI, SARA. "Nutritional factors influencing tissue omega-3 metabolism and endocannabinoids levels in experimental models and humans." Doctoral thesis, Università degli Studi di Cagliari, 2016. http://hdl.handle.net/11584/266644.
Повний текст джерелаPatel, Annie. "Endocannabinoid turnover and function." Thesis, University of Nottingham, 2010. http://eprints.nottingham.ac.uk/12625/.
Повний текст джерелаKilaru, Aruna. "Endocannabinoid System in Plants?" Digital Commons @ East Tennessee State University, 2012. https://dc.etsu.edu/etsu-works/4781.
Повний текст джерелаNorris, Leonie. "Endocannabinoid modulation of nociceptive processing." Thesis, University of Nottingham, 2010. http://eprints.nottingham.ac.uk/28701/.
Повний текст джерелаChilufya, Jedaidah Y., Shivakumar P. Devaiah, Richard R. Sante, and Aruna Kilaru. "Endocannabinoid-Like Lipids in Plants." Digital Commons @ East Tennessee State University, 2015. https://dc.etsu.edu/etsu-works/4747.
Повний текст джерелаLenz, Frederike. "The endocannabinoid system and autistic behavior in the Fmr1- KO mouse." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2018. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-231397.
Повний текст джерелаBonneville, Marika. "Endocannabinoid Modulation of Post-Ischemia Depression." Thesis, Université d'Ottawa / University of Ottawa, 2016. http://hdl.handle.net/10393/35056.
Повний текст джерелаКниги з теми "Endocannabinois"
Pertwee, Roger G., ed. Endocannabinoids. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-20825-1.
Повний текст джерелаAbood, Mary E., Roger G. Sorensen, and Nephi Stella, eds. endoCANNABINOIDS. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-4669-9.
Повний текст джерелаMaccarrone, Mauro, ed. Endocannabinoid Signaling. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-2728-0.
Повний текст джерелаMaccarrone, Mauro, ed. Endocannabinoid Signaling. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3539-0.
Повний текст джерелаMaccarrone, Mauro, ed. New Tools to Interrogate Endocannabinoid Signalling. Cambridge: Royal Society of Chemistry, 2020. http://dx.doi.org/10.1039/9781839160752.
Повний текст джерелаKendall, Dave, and Stephen Alexander, eds. Behavioral Neurobiology of the Endocannabinoid System. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-88955-7.
Повний текст джерелаBehavioral neurobiology of the endocannabinoid system. Berlin: Springer-Verlag, 2009.
Знайти повний текст джерелаMelis, Miriam, ed. Endocannabinoids and Lipid Mediators in Brain Functions. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57371-7.
Повний текст джерелаJ, Plutzky, Woods Stephen C, and sanofi aventis (Firm), eds. The endocannabinoid system and regulation of energy metabolism. [S.l: sanofi aventis, 2006.
Знайти повний текст джерелаS, Onaivi Emmanuel, Sugiura Takayuki, and Di Marzo Vincenzo, eds. Endocannabinoids: The brain and body's marijuana and beyond. Boca Raton: Taylor & Francis, 2005.
Знайти повний текст джерелаЧастини книг з теми "Endocannabinois"
Godlewski, Grzegorz, and George Kunos. "Overview of Nonclassical Cannabinoid Receptors." In endoCANNABINOIDS, 3–27. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4669-9_1.
Повний текст джерелаBurston, James, and David Kendall. "Peroxisome Proliferator-Activated Receptors and Inflammation." In endoCANNABINOIDS, 221–33. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4669-9_10.
Повний текст джерелаMascia, Paola, Gianluigi Tanda, Sevil Yasar, Stephen J. Heishman, and Steven R. Goldberg. "Peroxisome Proliferator-Activated Nuclear Receptors and Drug Addiction." In endoCANNABINOIDS, 235–60. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4669-9_11.
Повний текст джерелаAbood, Mary E., Roger G. Sorensen, and Nephi Stella. "Conclusions: Therapeutic Potential of Novel Cannabinoid Receptors." In endoCANNABINOIDS, 263–80. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4669-9_12.
Повний текст джерелаKotsikorou, Evangelia, and Patricia Reggio. "Overview of Non-CB1/CB2 Cannabinoid Receptors: Chemistry and Modeling." In endoCANNABINOIDS, 29–51. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4669-9_2.
Повний текст джерелаLu, Hui-Chen, Jane E. Lauckner, John W. Huffman, and Ken Mackie. "GPR55 in the CNS." In endoCANNABINOIDS, 55–69. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4669-9_3.
Повний текст джерелаWhyte, Lauren S., and Ruth A. Ross. "The Role of GPR55 in Bone Biology." In endoCANNABINOIDS, 71–113. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4669-9_4.
Повний текст джерелаAndradas, Clara, María M. Caffarel, Eduardo Pérez-Gómez, Manuel Guzmán, and Cristina Sánchez. "The Role of GPR55 in Cancer." In endoCANNABINOIDS, 115–33. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4669-9_5.
Повний текст джерелаMcHugh, Douglas, and Heather B. Bradshaw. "GPR18 and NAGly Signaling: New Members of the Endocannabinoid Family or Distant Cousins?" In endoCANNABINOIDS, 135–42. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4669-9_6.
Повний текст джерелаRimmerman, Neta, Ewa Kozela, Rivka Levy, Zvi Vogel, and Ana Juknat. "Cannabinoid Signaling Through Non-CB1R/Non-CB2R Targets in Microglia." In endoCANNABINOIDS, 143–71. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4669-9_7.
Повний текст джерелаТези доповідей конференцій з теми "Endocannabinois"
"PV-003 - SISTEMA ENDOCANNABINOIDE EN DEPRESIÓN DUAL." In 24 CONGRESO DE LA SOCIEDAD ESPAÑOLA DE PATOLOGÍA DUAL. SEPD, 2022. http://dx.doi.org/10.17579/abstractbooksepd2022.pv003.
Повний текст джерелаBilgic, Elif. "Endocannabinoid induced apoptotic cell death on endometriotic cells." In 15th International Congress of Histochemistry and Cytochemistry. Istanbul: LookUs Scientific, 2017. http://dx.doi.org/10.5505/2017ichc.op-12.
Повний текст джерелаBouter, Y., M. Brzózka, C. Rohleder, R. Rygula, M. Leweke, J. Wiltfang, and U. Havemann-Reinecke. "Effects of social defeat on the endocannabinoid system." In Abstracts of the 30th Symposium of the AGNP. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1606396.
Повний текст джерелаGertsch, J. "Endocannabinoid signaling across species – evolution and perspectives for drug discovery." In 67th International Congress and Annual Meeting of the Society for Medicinal Plant and Natural Product Research (GA) in cooperation with the French Society of Pharmacognosy AFERP. © Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-3399680.
Повний текст джерелаAbohalaka, Reshed, Turgut Emrah Bozkurt, Emirhan Nemutlu, Sevgen Celik Onder, and Inci Sahin-Erdemli. "The effect of endocannabinoid metabolism inhibition on airway inflammation in mice." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa3878.
Повний текст джерелаCruz, Leonardo Cardoso, Luis Gustavo Fraga Belotto, Sofia Dias Campos Machado, and Fabrício de Araújo Moreira. "Possible mechanisms of action of cannabidiol in the epilepsies: a review." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.045.
Повний текст джерелаOnnis, Valentina, Alessandro Deplano, and Monica Demurtas. "Discovery of novel endocannabinoid level modulators by modification of old analgesic drugs." In 4th International Electronic Conference on Medicinal Chemistry. Basel, Switzerland: MDPI, 2018. http://dx.doi.org/10.3390/ecmc-4-05590.
Повний текст джерелаOscoz Irurozqui, Maitane, Maria Guardiola-Ripoll, Carmen Almodóvar-Payà, Salavador Sarró, Amalia Guerrero-Pedraza, Edith Pomarol-Clotet, and Mar Fatjó-Vilas. "Cannabis use and genes of endocannabinoid system: their role in psychotic symptoms and cognition in first-episode psychosis." In 22° Congreso de la Sociedad Española de Patología Dual (SEPD) 2020. SEPD, 2020. http://dx.doi.org/10.17579/sepd2020o031.
Повний текст джерелаClendenen, S., R. Mcclain, and N. Clendenen. "B307 The effect of acetaminophen and total knee arthroplasty on endogenous plasma endocannabinoid levels." In ESRA Abstracts, 39th Annual ESRA Congress, 22–25 June 2022. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/rapm-2022-esra.381.
Повний текст джерелаUer, O., C. Weisheit, L. Bindila, M. Velten, H. Treede, and G. D. Duerr. "Role of the Endocannabinoid System in Modulation of the Inflammatory Reaction in Aortic Valve Degeneration." In 50th Annual Meeting of the German Society for Thoracic and Cardiovascular Surgery (DGTHG). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1725674.
Повний текст джерелаЗвіти організацій з теми "Endocannabinois"
Sallaberry, Chad, and Laurie Astern. The Endocannabinoid System, Our Universal Regulator. Journal of Young Investigators, June 2018. http://dx.doi.org/10.22186/jyi.34.5.48-55.
Повний текст джерелаGuzmán, Manuel. Endocannabinoides: un nuevo sistema de comunicación en el cerebro. Sociedad Española de Bioquímica y Biología Molecular (SEBBM), November 2010. http://dx.doi.org/10.18567/sebbmdiv_anc.2010.11.1.
Повний текст джерелаGewirtz, David A. The Endocannabinoid System as a Target for Treatment of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, August 2010. http://dx.doi.org/10.21236/ada535994.
Повний текст джерелаMolina, Patricia E. Endocannabinoids as a Target for the Treatment of Traumatic Brain Injury. Fort Belvoir, VA: Defense Technical Information Center, November 2012. http://dx.doi.org/10.21236/ada576663.
Повний текст джерелаMolina, Patricia E. Endocannabinoids as a Target for the Treatment of Traumatic Brain Injury. Fort Belvoir, VA: Defense Technical Information Center, November 2013. http://dx.doi.org/10.21236/ada599310.
Повний текст джерелаLichtman, Aron. The Endocannabinoid System as a Target for Treatment of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, August 2009. http://dx.doi.org/10.21236/ada550908.
Повний текст джерелаLichtman, Aron. The Endocannabinoid System as a Target for Treatment of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, August 2011. http://dx.doi.org/10.21236/ada558526.
Повний текст джерелаKamaruzzaman, Mohd Amir, Muhammad Hibatullah Romli, Razif Abas, Sharmili Vidyadaran, Mohamad Taufik Hidayat Baharuldin, Muhammad Luqman Nasaruddin, Vishnnumukkala Thirupathirao, et al. Impact of Endocannabinoid Mediated Glial Cells on Cognitive Function in Alzheimer’s Disease: A Systematic Review and Meta-Analysis of Animal Studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2022. http://dx.doi.org/10.37766/inplasy2022.8.0094.
Повний текст джерелаEmery, Sean M., Aron H. Lichtman, and David A. Gewirtz. Involvement of the Endocannabinoid System in the Development and Treatment of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, February 2013. http://dx.doi.org/10.21236/ada575843.
Повний текст джерелаEmery, Sean. Involvement of the Endocannabinoid System in the Development and Treatment of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, February 2012. http://dx.doi.org/10.21236/ada560646.
Повний текст джерела