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Готові списки джерел за темами / Chloride channels

Добірка наукової літератури з теми "Chloride channels"

Автор: Grafiati

Опубліковано: 4 червня 2021

Оновлено: 31 січня 2023

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Статті в журналах з теми "Chloride channels":

1

Jentsch, Thomas J. "Chloride channels." Current Opinion in Neurobiology 3, no. 3 (June 1993): 316–21. http://dx.doi.org/10.1016/0959-4388(93)90123-g.

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2

Lísal, Jiří, and Merritt Maduke. "Proton-coupled gating in chloride channels." Philosophical Transactions of the Royal Society B: Biological Sciences 364, no. 1514 (October 28, 2008): 181–87. http://dx.doi.org/10.1098/rstb.2008.0123.

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The physiologically indispensable chloride channel (CLC) family is split into two classes of membrane proteins: chloride channels and chloride/proton antiporters. In this article we focus on the relationship between these two groups and specifically review the role of protons in chloride-channel gating. Moreover, we discuss the evidence for proton transport through the chloride channels and explore the possible pathways that the protons could take through the chloride channels. We present results of a mutagenesis study, suggesting the feasibility of one of the pathways, which is closely related to the proton pathway proposed previously for the chloride/proton antiporters. We conclude that the two groups of CLC proteins, although in principle very different, employ similar mechanisms and pathways for ion transport.

3

Kicińska, A., G. D bska, W. Kunz, and A. Szewczyk. "Mitochondrial potassium and chloride channels." Acta Biochimica Polonica 47, no. 3 (September 30, 2000): 541–51. http://dx.doi.org/10.18388/abp.2000_3977.

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Channels selective for potassium or chloride ions are present in inner mitochondrial membranes. They probably play an important role in mitochondrial events such as the formation of delta pH and regulation of mitochondrial volume changes. Mitochondrial potassium and chloride channels could also be the targets for pharmacologically active compounds such as potassium channel openers and antidiabetic sulfonylureas. This review describes the properties, pharmacology, and current observations concerning the functional role of mitochondrial potassium and chloride channels.

4

Kim, Hyeong Jae, Peter Chang-Whan Lee, and Jeong Hee Hong. "Chloride Channels and Transporters: Roles beyond Classical Cellular Homeostatic pH or Ion Balance in Cancers." Cancers 14, no. 4 (February 9, 2022): 856. http://dx.doi.org/10.3390/cancers14040856.

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The canonical roles of chloride channels and chloride-associated transporters have been physiologically determined; these roles include the maintenance of membrane potential, pH balance, and volume regulation and subsequent cellular functions such as autophagy and cellular proliferative processes. However, chloride channels/transporters also play other roles, beyond these classical function, in cancerous tissues and under specific conditions. Here, we focused on the chloride channel-associated cancers and present recent advances in understanding the environments of various types of cancer caused by the participation of many chloride channel or transporters families and discuss the challenges and potential targets for cancer treatment. The modulation of chloride channels/transporters might promote new aspect of cancer treatment strategies.

5

Higgins, Chris. "Chloride channels revisited." Nature 358, no. 6387 (August 1992): 536. http://dx.doi.org/10.1038/358536a0.

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6

Vinson, V. "Controlling Chloride Channels." Science Signaling 3, no. 146 (November 2, 2010): ec338-ec338. http://dx.doi.org/10.1126/scisignal.3146ec338.

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7

Bretag, A. H. "Muscle chloride channels." Physiological Reviews 67, no. 2 (April 1987): 618–724. http://dx.doi.org/10.1152/physrev.1987.67.2.618.

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8

Ackerman, Michael J., and David E. Clapham. "Cardiac chloride channels." Trends in Cardiovascular Medicine 3, no. 1 (January 1993): 23–28. http://dx.doi.org/10.1016/1050-1738(93)90024-z.

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9

Fahlke, Christoph, Timothy Knittle, Christina A. Gurnett, Kevin P. Campbell, and Alfred L. George. "Subunit Stoichiometry of Human Muscle Chloride Channels." Journal of General Physiology 109, no. 1 (January 1, 1997): 93–104. http://dx.doi.org/10.1085/jgp.109.1.93.

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Voltage-gated Cl− channels belonging to the ClC family appear to function as homomultimers, but the number of subunits needed to form a functional channel is controversial. To determine subunit stoichiometry, we constructed dimeric human skeletal muscle Cl− channels in which one subunit was tagged by a mutation (D136G) that causes profound changes in voltage-dependent gating. Sucrose-density gradient centrifugation experiments indicate that both monomeric and dimeric hClC-1 channels in their native configurations exhibit similar sedimentation properties consistent with a multimeric complex having a molecular mass of a dimer. Expression of the heterodimeric channel in a mammalian cell line results in a homogenous population of Cl− channels exhibiting novel gating properties that are best explained by the formation of heteromultimeric channels with an even number of subunits. Heteromultimeric channels were not evident in cells cotransfected with homodimeric WT-WT and D136G-D136G constructs excluding the possibility that functional hClC-1 channels are assembled from more than two subunits. These results demonstrate that the functional hClC-1 unit consists of two subunits.

10

Duszyk, Marek, Andrew S. French, and S. F. Paul Man. "Cystic fibrosis affects chloride and sodium channels in human airway epithelia." Canadian Journal of Physiology and Pharmacology 67, no. 10 (October 1, 1989): 1362–65. http://dx.doi.org/10.1139/y89-217.

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Abnormalities of epithelial function in cystic fibrosis (CF) have been linked to defects in cell membrane permeability to chloride or sodium ions. Recently, a class of chloride channels in airway epithelial cells have been reported to lack their usual sensitivity to phosphorylation via cAMP-dependent protein kinase, suggesting that CF could be due to a single genetic defect in these channels. We have examined single chloride and sodium channels in control and CF human nasal epithelia using the patch-clamp technique. The most common chloride channel was not the one previously associated with CF, but it was also abnormal in CF cells. In addition, the number of sodium channels was unusually high in CF. These findings suggest a wider disturbance of ion channel properties in CF than would be produced by a defect in a single type of channel.Key words: ion channels, cystic fibrosis, airway, epithelium.

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Дисертації з теми "Chloride channels":

1

Low, Wendy. "Chloride channels in epithelia." Thesis, McGill University, 1993. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=68206.

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The outwardly rectifying chloride channel is found in most vertebrate cells however its physiological role is uncertain. Patch clamp, short-circuit current, and electronic cell sizing techniques were used to investigate the role of the outward rectifier in transepithelial chloride secretion and cell volume regulation, the two main functions that have been proposed for this channel in epithelia. Patch clamp studies of the human cell lines PANC-1 and T$ sb{84}$ showed that the chloride channel blockers IAA-94 and NPPB decrease the open probability of the outward rectifier, with half-maximal inhibition at 15 $ mu$M and 23 $ mu$M, respectively. At these concentrations the blockers did not affect cAMP-induced short-circuit current. They did inhibit the regulatory volume decrease (RVD) which occurs after hypotonic cell swelling, but only at much higher concentrations. Moreover, the commonly-used inhibitor DIDS, which blocks the outward rectifier in the 10-20 $ mu$M range, had no effect on the RVD when tested at 100 $ mu$M. The results indicate that the outwardly rectifying Cl channel does not mediate a significant fraction of transepithelial Cl secretion across T$ sb{84}$ cells. Although the data do not exclude a role for the outward rectifier in cell volume regulation, the selectivity and pharmacological properties of the swelling-induced anion conductance in T$ sb{84}$ cells is more similar to the ClC-2 channel than to the outward rectifier.

2

Thompson, Christopher Hal. "Identification and characterization of a peptide toxin inhibitor of ClC-2 chloride channels." Diss., Atlanta, Ga. : Georgia Institute of Technology, 2008. http://hdl.handle.net/1853/26604.

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Thesis (Ph.D)--Biology, Georgia Institute of Technology, 2009.
Committee Chair: McCarty, Nael; Committee Co-Chair: Harvey, Stephen; Committee Member: Hartzell, Criss; Committee Member: Kubanek, Julia; Committee Member: Lee, Robert. Part of the SMARTech Electronic Thesis and Dissertation Collection.

3

Sabanov, Victor. "Chloride Channels and Brown Fat Cells." Doctoral thesis, Stockholm : Department of Physiology, Wenner-Gren Institute, Arrhenius Laboratories, Stockholm University, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-474.

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4

Bhandal, Narotam Singh. "Arthropod chloride channels as targets for pesticides." Thesis, University of Nottingham, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335651.

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5

Joo, Nam Soo. "Regulation of duodenal ion transport by uroguanylin and cloning of murine intestinal CIC-2 chloride channel." free to MU campus, to others for purchase, 1998. http://wwwlib.umi.com/cr/mo/fullcit?p9924893.

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6

Sin, Sai-lung Steven, and 冼世隆. "Chloride channel in glioma cell invasion." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41508555.

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7

Sin, Sai-lung Steven. "Chloride channel in glioma cell invasion." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B41508555.

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8

Zeltwanger, Shawn. "Gating of cystic fibrosis transmembrane conductance regulator (CFTR) chloride channels by nucleoside triphosphates." free to MU campus, to others for purchase, 1998. http://wwwlib.umi.com/cr/mo/fullcit?p9924950.

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9

Starc, Tanja. "Structure function analysis of glutamate gated chloride channels." Thesis, McGill University, 2003. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=79135.

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Glutamate-gated chloride channels (GluCl) belong to then icotinic ligand-gated ion channel family and are thus assumed to be heteropentamers. Each subunit contains a large extracellular N-terminal domain, four transmembrane domains (TM1--TM4), and an extracellular C terminal. Caenorhabditis elegans expresses various GluCl channels formed by alpha1, alpha2, alpha3, alpha4 and beta subunits. The best understood GluCl channel is expressed in pharyngeal muscle cells where it mediates response to the M3 motor neuron. alpha2 forms this channel, probably in association with beta. The alpha2 mutant lacks M3 neurotransmission which can be rescued by pharynx-specific alpha2 expression. My results show that alpha1 and alpha3 subunits cannot substitute for alpha2. Formation of chimeric constructs of alpha1, alpha2 and alpha3 pinpoints the M1--M3 transmembrane region of alpha2 as the minimal rescuing domain. This region may therefore be important for localization or, in association with another subunit, in the formation of the active channel.

10

Halstead, Meredith. "Putative glutamate-gated chloride channels from Onchocerca volvulus." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=29439.

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Onchocerca volvulus, a filarial nematode, is the causative agent of onchocerciasis.
O. volvulus is a human parasite with no animal model host and is endemic in the tropics. O. volvulus material is scarce and must be conserved as part of the Onchocerciasis Control Program. A genomic library was constructed to provide a substantial source of renewable genetic material, in place of original parasite DNA.
Currently there is only one glutamate-gated chloride channel that has been sequenced from O. volvulus, but this has not yet been characterized. This GluClx partial cDNA sequence isolated by Cully et al., 1997, may be found in GenBank, accession number U59745. Specific primers were designed to amplify this gene from the genomic library. A fragment of this gene was isolated but the primers were non-specific, amplifying genes in addition to GluClx.
A motif is a short recognition sequence within a protein that may allow the modification of the protein. The cysteine loop in the N-terminal of all the ligand-gated ion channels is interesting because it contains the neurotransmitter-gated ion channel signature sequence. (Abstract shortened by UMI.)

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Книги з теми "Chloride channels":

1

J, Alvarez-Leefmans F., Russell John M. 1942-, and International Brain Research Organization. Congress, eds. Chloride channels and carriers in nerve, muscle, and glial cells. New York: Plenum Press, 1990.

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2

W, Olsen Richard, and Venter J. Craig, eds. Benzodiazepine/GABA receptors and chloride channels: Structural and functional properties. New York: A.R. Liss, 1986.

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3

Alvarez-Leefmans, Francisco J., and John M. Russell, eds. Chloride Channels and Carriers in Nerve, Muscle, and Glial Cells. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-9685-8.

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4

Kirk, Kevin L. The cystic fibrosis transmembrane conductance regulator. Georgetown, TX: Landes Bioscience : Eurekah.com, 2004.

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5

Kirk, Kevin L. The cystic fibrosis transmembrane conductance regulator. Georgetown, Tex: Landes Bioscience / Eurekah.com, 2003.

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6

Ahmed, Najma Nusarat. Proteins which interact with and regulate the chloride channel, CIC-2. Ottawa: National Library of Canada, 2001.

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7

Wong, Simeon. Regulation of Clc-2 chloride channel by protein kinase C phosphorylation. Ottawa: National Library of Canada, 2000.

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8

Kleinzeller, Arnost, Douglas M. Fambrough, and William B. Guggino. Chloride Channels. Elsevier Science & Technology Books, 1994.

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9

Roland, Kozlowski, ed. Chloride channels. Oxford: Isis Medical Media, 1999.

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10

Kozlowski, Ronald. Chloride Channels. Informa Healthcare, 2000.

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Частини книг з теми "Chloride channels":

1

Prescott, Steven A. "Chloride Channels." In Encyclopedia of Computational Neuroscience, 601–5. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4614-6675-8_226.

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2

Prescott, Steven A. "Chloride Channels." In Encyclopedia of Computational Neuroscience, 1–4. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-7320-6_226-1.

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3

Greger, R., and K. Kunzelmann. "Epithelial Chloride Channels." In Epithelial Secretion of Water and Electrolytes, 3–13. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75033-5_1.

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4

Hume, Joseph R., Paul C. Levesque, Pádraig Hart, Mei Lin Collier, John D. Warth, Yvonne Geary, Todd Chapman, and Burton Horowitz. "Chloride channels in heart." In Developments in Cardiovascular Medicine, 187–96. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-011-3990-8_16.

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5

Cuppoletti, John, Danuta H. Malinowska, and Ryuji Ueno. "ClC-2 Chloride Channels." In Ion Channels and Transporters of Epithelia in Health and Disease, 491–518. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-3366-2_15.

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6

Gallos, George, and Charles W. Emala. "Calcium-Activated Chloride Channels." In Calcium Signaling In Airway Smooth Muscle Cells, 85–106. Cham: Springer International Publishing, 2013. http://dx.doi.org/10.1007/978-3-319-01312-1_5.

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7

Gerencser, G. A. "Chloride Channels in Molluscs." In Advances in Comparative and Environmental Physiology, 133–53. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78261-9_8.

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8

Cuppoletti, John, Danuta H. Malinowska, and Ryuji Ueno. "ClC-2 Chloride Channels." In Studies of Epithelial Transporters and Ion Channels, 495–522. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-55454-5_13.

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9

Liedtke, Carole M. "Chloride Channels in Cystic Fibrosis." In Ion Channels and Ion Pumps, 500–525. New York, NY: Springer New York, 1994. http://dx.doi.org/10.1007/978-1-4612-2596-6_23.

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10

Blatz, Andrew L. "Chloride Channels in Skeletal Muscle." In Chloride Channels and Carriers in Nerve, Muscle, and Glial Cells, 407–20. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-9685-8_16.

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Тези доповідей конференцій з теми "Chloride channels":

1

Shenoy, Ambika, Sascha Kopic, Michael Murek, Christina Caputo, John Geibel, and Marie Egan. "Cystic Fibrosis Transmembrane Conductance Regulator Protein And Calcium Activated Chloride Channels Mediate Chloride Efflux In Murine Macrophages." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6575.

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2

Jenson, Lacey J. "Voltage- and calcium-activated chloride channels in insect physiological systems." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93221.

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3

Chang, Chen-Ling, John Guofeng Bai, Kyong-Hoon Lee, Jae-Hyun Chung, Yaling Liu, and Wing Kam Liu. "Ion Diffusion Upon Concentrations in Open Nanofluidic Channels." In ASME 2007 International Mechanical Engineering Congress and Exposition. ASMEDC, 2007. http://dx.doi.org/10.1115/imece2007-42362.

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The ion flow in nanochannels is investigated by using nanochannels in an open configuration that allows the direct observation of fluid diffusion through an optical microscope. An “open nanochannel” is a channel with the top open to air such that fluidics can be introduced from both the entrance and the top of the channels. The experimental results showed that the diffusion length of the potassium chloride and phosphate buffer decreased with their concentration. The observed behaviors were analyzed by the contact angle variation due to the electrowetting phenomena involving the interaction between electrical double layer and counter-ions in the solution.

4

Gallos, George, Peter Yim, Yi Zhang, James M. Cook, Sundari Rallapalli, and Charles W. Emala. "Gabaa Channels Containing The Alpha5 Subunit Directly Relax Airway Smooth Muscle And Increase Chloride Channel Flux." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6034.

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5

Yim, Peter, George Gallos, Yi Zhang, and Charles W. Emala. "Concomitant Blockade Of Calcium-Activated Chloride Channels (CACC) And Sodium Potassium Chloride Cotransporter (NKCC) Attenuates Acetylcholine Contractions In Human Airway Smooth Muscle." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6033.

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6

Ozoe, Yoshihisa. "Molecular and functional characterization of histamine-gated chloride channels from the house fly, Musca domestica." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93015.

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7

Vemulakonda, Srilakshmi, Demosthenes G. Papamatheakis, A. Forrest, N. Leblanc, J. Angermann, L. D. Longo, and Sean M. Wilson. "INFLUENCE OF POSTNATAL MATURITY AND CHRONIC HYPOXIA ON CALCIUM ACTIVATED CHLORIDE CHANNELS IN PULMONARY ARTERIAL VASOCONSTRICTION." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6278.

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8

Brett, T., M. Sala-Rabanal, K. Berry, D. F. Steinberg, and C. G. Nichols. "Modulation of TMEM16B Channel Activity by the Calcium-Activated Chloride Channel Regulator 4 Suggests a Common Function for CLCA Proteins in Modifying TMEM16 Channels." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a2127.

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9

Vemulakonda, S., DG Papamatheakis, J. Angermann, D. Nguyen, WJ Pearce, LD Longo, and SM Wilson. "The Role of Calcium Activated Chloride Channels in Pulmonary Arterial Vasoconstriction Is Influenced by Long-Term Hypoxic Stress." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a6245.

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10

Remy, Kenneth E., Ding Bang Xu, Herng-Yu S. Chang, Peter Yim, and Charles W. Emala. "Novel Expression Of The TMEM16 Family Of Calcium Activated Chloride Channels In Human Airway Epithelium And Smooth Muscle Cells." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6035.

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