Готові списки джерел за темами / Cerebral circulation

Добірка наукової літератури з теми "Cerebral circulation"

Автор: Grafiati
Опубліковано: 4 червня 2021
Оновлено: 29 липня 2024

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Статті в журналах з теми "Cerebral circulation"

1

Hickey, Joanne V. "Cerebral Circulation Demystified." AACN Advanced Critical Care 2, no. 4 (November 1, 1991): 657–64. http://dx.doi.org/10.4037/15597768-1991-4005.

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Basic anatomic and physiologic concepts related to cerebral circulation are summarized. The arterial blood supply is traced from its origins to the major divisions of anterior and posterior circulation. The circle of Willis, the major arterial vessels and territories, and the peculiarities of the cerebral venous circulation are discussed. Finally, concepts of cerebral circulations are applied to clinical practice to assist the nurse in accurately assessing, monitoring, and predicting human responses to alterations in cerebral blood supply
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2

Hamel, Edith. "Cerebral Circulation." Journal of Cardiovascular Pharmacology 65, no. 4 (April 2015): 317–24. http://dx.doi.org/10.1097/fjc.0000000000000177.

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3

Cipolla, Marilyn J. "The Cerebral Circulation." Colloquium Series on Integrated Systems Physiology: From Molecule to Function 1, no. 1 (January 2009): 1–59. http://dx.doi.org/10.4199/c00005ed1v01y200912isp002.

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4

Townsend, P., and M. G. Knowles. "The cerebral circulation." Current Anaesthesia & Critical Care 10, no. 2 (April 1999): 77–82. http://dx.doi.org/10.1016/s0953-7112(99)90005-4.

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5

Moss, Edward. "The cerebral circulation." BJA CEPD Reviews 1, no. 3 (June 2001): 67–71. http://dx.doi.org/10.1093/bjacepd/1.3.67.

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6

Cipolla, Marilyn J. "The Adaptation of the Cerebral Circulation to Pregnancy: Mechanisms and Consequences." Journal of Cerebral Blood Flow & Metabolism 33, no. 4 (January 16, 2013): 465–78. http://dx.doi.org/10.1038/jcbfm.2012.210.

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The adaptation of the cerebral circulation to pregnancy is unique from other vascular beds. Most notably, the growth and vasodilatory response to high levels of circulating growth factors and cytokines that promote substantial hemodynamic changes in other vascular beds is limited in the cerebral circulation. This is accomplished through several mechanisms, including downregulation of key receptors and transcription factors, and production of circulating factors that counteract the vasodilatory effects of vascular endothelial growth factor (VEGF) and placental growth factor. Pregnancy both prevents and reverses hypertensive inward remodeling of cerebral arteries, possibly through downregulation of the angiotensin type 1 receptor. The blood–brain barrier (BBB) importantly adapts to pregnancy by preventing the passage of seizure provoking serum into the brain and limiting the permeability effects of VEGF that is more highly expressed in cerebral vasculature during pregnancy. While the adaptation of the cerebral circulation to pregnancy provides for relatively normal cerebral blood flow and BBB properties in the face of substantial cardiovascular changes and high levels of circulating factors, under pathologic conditions, these adaptations appear to promote greater brain injury, including edema formation during acute hypertension, and greater sensitivity to bacterial endotoxin.
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7

Nikitin, Vladislav Nikolaevich, and Ekaterina Valerevna Kozhemyakina. "MODELING REDISTRIBUTION CEREBRAL CIRCULATION." SOFT MEASUREMENTS AND COMPUTING 1, no. 4 (2021): 13–18. http://dx.doi.org/10.36871/2618-9976.2021.04.002.

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The brain is one of the most important organs responsible for the health and functioning of the entire body. The blood supply to the brain is carried out through 2 internal carotid and 2 vertebral arteries in norm. The brain, like other body systems, has protective (compensatory) mechanisms aimed at maintaining the necessary blood flow, one of which is the circle of Willis. The article proposes a mechanism for how blood flow is redistributed through the arteries feeding the brain, which is based on the assumption that the central nervous system controls in such a way that it minimizes flows through the connective arteries of the circle of Willis, the flows along which are normal (with symmetry of the left and right sides) practically equal to zero. Сase of the structure of the circle of Willis is considered in norm. The indicated redistribution mechanism is still only the first step towards an attempt to predict cases of changes in blood flow through the cerebral arteries, especially in stroke. In further works, it is planned to consider the inverse problem, i.e. determine the flows through the internal carotid and vertebral arteries, provided that the flows through the cerebral arteries extending from the circle of Willis have normal flow values.
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8

Tan, A., and D. Roberts. "Cerebral circulation 1: anatomy." BJA Education 21, no. 10 (October 2021): 390–95. http://dx.doi.org/10.1016/j.bjae.2021.05.004.

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9

Chang, Steven D., Stephen I. Ryu, and Gary K. Steinberg. "Posterior Cerebral Circulation Revascularization." Neurosurgery Clinics of North America 12, no. 3 (July 2001): 519–40. http://dx.doi.org/10.1016/s1042-3680(18)30041-x.

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10

Hermann, Dirk M., and Claudio L. Bassetti. "Cerebral circulation and sleep." Sleep Medicine Reviews 6, no. 6 (December 2002): 425–27. http://dx.doi.org/10.1053/smrv.2002.0259.

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Дисертації з теми "Cerebral circulation"

1

Cirovic, Srdjan. "Cerebral circulation during acceleration stress." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/NQ58910.pdf.

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2

Grolimund, Peter. "Doppler-sonographic evaluation of the cerebral circulation /." [S.l.] : [s.n.], 1987. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=8433.

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3

Critchley, Giles Roderic. "Cerebral ischaemia following subarachnoid haemorrhage : a laboratory and clinical investigation of the cerebral microcirculation." Thesis, St George's, University of London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268382.

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4

Jonsson, Ove. "Cerebral Perfusion and Metabolism during Experimental Extracorporeal Circulation." Doctoral thesis, Uppsala universitet, Anestesiologi och intensivvård, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-147486.

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Neurologic injuries are major causes of mortality and morbidity after cardiac surgery. This thesis aimed to investigate cerebral metabolism and perfusion abnormalities in pigs during hypothermic circulatory arrest, selective antegrade cerebral perfusion (SACP) and extracorporeal circulation following progressive venous stasis. Hypothermic circulatory arrest induced a metabolic pattern consistent with overt ischaemia, which was absent following SACP. In contrast, metabolism during SACP was influenced by the perfusate temperature, where a colder perfusate (20 °C) preserved cellular metabolism and membrane integrity better than a warmer perfusate (28 °C). The minimum SACP flow required to maintain metabolism during hypothermia at 20 °C was investigated with magnetic resonance imaging, protein S100β, near infrared spectroscopy and microdialysis. The findings suggested an ischaemic threshold close to 6 ml/kg/min in the present models. Furthermore, regional differences in perfusion with a hemispheric distribution were apparent at all flow levels and differed from earlier studies where the differences were uniform and followed a neuranatomical pattern. Venus stasis following superior vena cava congestion produced measurable signs of impaired cerebral perfusion and patterns of cerebral ischaemia were evident in individual animals. As venous pressure increased, the mean arterial pressure stayed more or less unchanged, generating reduced cerebral perfusion pressure and consequently an increased risk of ischaemia, which may impair cerebral perfusion, especially in cases of compromised arterial flow during extracorporeal circulation. In conclusion, cerebral metabolism and perfusion are influenced by temperature, SACP flow levels and venous congestion. In clinical practice, the regional differences in perfusion during SACP may be of pathogenic importance in focal cerebral ischaemia. Furthermore, the reduced superior vena cava cannula flow may pass undetected during bicaval cardiopulmonary bypass if the superior vena cava flow is not specifically monitored.
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5

Raynor, Sheila Marie. "Analysis of Doppler waveforms of the cerebral circulation." Thesis, Imperial College London, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.444367.

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Kenton, Anthony Robert. "The assessment of the cerebral circulation following stroke." Thesis, University of Leicester, 1998. http://hdl.handle.net/2381/29544.

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Budohoski, Karol Paweł. "Cerebral autoregulation and subarachnoid haemorrhage." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648435.

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Patel, Toshal R. "The pathophysiologic significance of endothelins in the cerebral circulation." Thesis, University of Glasgow, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.297004.

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9

Belfort, Michael A. "The cerebral circulation in preeclampsia : abnormalities in autoregulation and perfusion /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4622-1/.

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Modaresi, Kamran Bahari. "Computerised techniques for improved imaging and monitoring of the arterial circulation." Thesis, King's College London (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.281685.

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Книги з теми "Cerebral circulation"

1

Cipolla, Marilyn J. The cerebral circulation. [San Rafael, Calif.?]: Morgan & Claypool Life Sciences, 2010.

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2

Lars, Edvinsson, and McCulloch J. 1951-, eds. Peptidergic mechanisms in the cerebral circulation. Weinheim, Federal Republic of Germany: VCH, 1987.

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3

1983), Tbilisi Symposium on Cerebral Circulation (5th. Otek golovnogo mozga: Rassmotrenie patofiziologicheskikh mekhanizmov na osnove sistemnogo podkhoda na 5-m Tbilisskom simpoziume po mozgovomu krovoobrashchenii͡u︡, 20-23 apreli͡a︡ 1983 g. = Brain edema : consideration of pathophysiological mechanisms on the basis of systems approach at 5th Tbilisi Symposium on Cerebral Circulation, April 20-23, 1983. Tbilisi: Izd-vo "Met͡s︡niereba", 1986.

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4

Mitagvariia, N. P. Cerebral blood flow regulation. New York: Nova Science Publishers, 2009.

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5

1947-, Mraovitch Sima, and Sercombe Richard, eds. Neurophysiological basis of cerebral blood flow control: An introduction. London: J. Libbey, 1996.

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6

International Symposium of Applied Physiology of the Peripheral Circulation (5th 2000 Pittsburgh, Pa.). Cerebral blood flow: Mechanisms of ischemia, diagnosis and therapy. New York: Springer, 2002.

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7

1930-, Bevan John A., ed. Arterial behavior and blood circulation in the brain. New York: Consultants Bureau, 1986.

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8

1921-, Mchedlishvili G. I., та Momcʻeliże Nana, ред. Mikrocʻirculacʻia, hemoreologia, tʻavis tvinis sisxlis mimokʻcʻeva: Giorgi Mčedlišvilis šromebi. Tʻbilisi: Megobari, 2002.

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9

Madsen, Jörn Bo, and Georg Emil Cold. The Effects of Anaesthetics upon Cerebral Circulation and Metabolism. Vienna: Springer Vienna, 1990. http://dx.doi.org/10.1007/978-3-7091-3680-5.

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10

K, Perktold, ed. Computer Simulation lokaler arterieller Strömungsformen unter besonderer Beachtung der cerebralen Gefässe. Graz: Forschungsgesellschaft Joanneum, 1987.

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Частини книг з теми "Cerebral circulation"

1

Kam, Peter, Ian Power, Michael J. Cousins, and Philip J. Siddal. "Cerebral Circulation." In Principles of Physiology for the Anaesthetist, 39–41. Fourth edition. | Boca Raton : CRC Press, Taylor & Francis Group, 2020.: CRC Press, 2020. http://dx.doi.org/10.1201/9780429288210-6.

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Muresian, Horia. "The Cerebral Circulation." In Arterial Revascularization of the Head and Neck, 1–43. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-34193-4_1.

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3

Ainslie, Philip N., Mark H. Wilson, and Christopher H. E. Imray. "Cerebral Circulation and Brain." In High Altitude, 141–70. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-8772-2_7.

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4

Tong, Lu-Sha, Yan-nan Yu, Jiping Tang, Min Lou, and John H. Zhang. "Cerebral Venous Collateral Circulation." In Cerebral Venous System in Acute and Chronic Brain Injuries, 103–17. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-96053-1_5.

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5

Iadecola, Costantino, and Kiyoshi Niwa. "Neural Regulation of the Cerebral Circulation." In Cerebral Blood Flow, 7–16. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-56036-1_2.

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6

Peerless, Sydney J., and Charles G. Drake. "Posterior Circulation Aneurysms." In Advances in Surgery for Cerebral Stroke, 53–56. Tokyo: Springer Japan, 1988. http://dx.doi.org/10.1007/978-4-431-68314-8_6.

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Symon, Lindsay. "Posterior Circulation Aneurysms." In Advances in Surgery for Cerebral Stroke, 63–66. Tokyo: Springer Japan, 1988. http://dx.doi.org/10.1007/978-4-431-68314-8_8.

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8

Marsh, W. Richard, and Thoralf M. Sundt. "Posterior Circulation Aneurysms." In Advances in Surgery for Cerebral Stroke, 67–69. Tokyo: Springer Japan, 1988. http://dx.doi.org/10.1007/978-4-431-68314-8_9.

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9

Edvinsson, L. "Neurogenic Control of Cerebral Circulation." In Cerebral Ischemia and Hemorheology, 13–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71787-1_2.

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Quayle, John M., and Mark T. Nelson. "Ion Channels in Cerebral Arteries." In The Human Brain Circulation, 145–56. Totowa, NJ: Humana Press, 1994. http://dx.doi.org/10.1007/978-1-4612-0303-2_11.

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Тези доповідей конференцій з теми "Cerebral circulation"

1

Utsuki, Tomohiko. "Construction of a Dynamic Cerebral Blood Circulation Model." In International Conference on Research in Science, Engineering and Technology. Acavent, 2019. http://dx.doi.org/10.33422/icrset.2019.03.69.

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2

Bozkurt, Surhan, and Umut Engin Ayten. "Regional Resistance Value Optimization of Cerebral Blood Circulation." In 2021 8th International Conference on Electrical and Electronics Engineering (ICEEE). IEEE, 2021. http://dx.doi.org/10.1109/iceee52452.2021.9415923.

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3

Lieber, Baruch B., Chander Sadasivan, David J. Fiorella, and Henry H. Woo. "Phototherapy Enhanced Exclusion of Aneurysms From the Cerebral Circulation." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14135.

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Accumulated experience using flow diverters in humans suggests that complete cure of the aneurysm is usually a protracted process that can last up to twelve months [1]. While it is well established that a properly designed flow diverter serves as a scaffold for neointimal proliferation, the process of its formation over the aneurysm neck is delayed until the aneurysm cavity itself is occluded by a thrombus, negating flow of fresh blood through the neck, and thus allowing the neointimal formation to bridge the aneurysm neck. The notion that induction of some injury to the luminal surface of the aneurysmal tissue, particularly to the endothelium, may result in a healing response that is faster than just placing a flow diverter and waiting for thrombus formation within the aneurysm has been tried in the past using various experimental models. Some of the injuries to the aneurysm tissue that have been tried in the past include mechanical scraping, thermal heating and UV irradiation. All these attempts, while showing that hastening the thrombus formation is feasible, have not resulted in any success due to the fact that the processes that were tried suffered from lack of proper control to be implemented in actual aneurysmal tissue that is weakened and diseased a priori.
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Terskov, Andrey V., Olga N. Pavlova, Arkady S. Abdurashitov, Alexey N. Pavlov, and Oxana Semyachkina-Glushkovskaya. "Synchronization of cerebral and peripheral blood circulation: stress-induced changes." In Saratov Fall Meeting 2019: Computations and Data Analysis: from Nanoscale Tools to Brain Functions, edited by Dmitry E. Postnov. SPIE, 2020. http://dx.doi.org/10.1117/12.2559692.

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5

Reymond, Philippe, Fabrice Merenda, Fabienne Perren, Daniel Rüfenacht, and Nikos Stergiopulos. "One Dimensional Model of the Systemic Arterial Tree Including Cerebral Circulation." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176452.

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The aim of this study is to develop a distributed model of the entire systemic arterial tree, coupled to a heart model and including a detailed description of the cerebral arteries. Distributed models of the arterial tree have been studied extensively in the past (Avolio [1]; Cassot et al [2]; Meister [3]; Schaaf and Abbrecht [4]; Stergiopulos et al [5]; Westerhof et al [6]; Zagzoule and Marc-Vergnes [7]), however, no model has been developed so far that offers a physiologically relevant coupling to the heart and includes the entire cerebral artery network.
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Albuquerque, Tamara Melissa Zavadzki, Annelise Akemi Higa Lee, João Paulo Santiago de Oliveira, Rosa Maria Nascimento Marcusso, Felipe Torres Pacheco, Vivian Baptista Dias Gagliardi, and Rubens José Gagliardi. "Predictive factors for brain collateral circulation analysis in ischemic stroke." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.539.

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Introduction: Cerebral collateral circulation has become increasingly important in recent years. It is a physiological vascular protection circuit, designed to preserve cerebral irrigation in a failure context of the primary pathways. Methods: This retrospective, descriptive and analytical study aims to analyze the predictive factors for the collateral circulation presence in patients with acute ischemic stroke, seen in the emergency room of the Santa Casa de Misericórdia Hospital in São Paulo, with large vessels occlusion on angiotomography. Results: 21 patients were analysed between january 2018 and april 2020, 10 men and 11 women, with a median age of 71 years. The median NIHSS score was 16 and intravenous thrombolysis was performed in 71.4% of the patients. We identified the presence of hyperglycemia, cervical obstruction and female gender as factors independently associated with an unsatisfactory leptomeningeal collateral state. Systemic arterial hypertension, smoking, alcoholism and a history of stroke had no association with poor collateral status, as well as the previous use of sinvastatin does not improve the combined score. Conclusion: Knowing the risk factors and comorbidities associated with the rarefaction of cerebral collateral circulation is of great importance in assessing the ischemic stroke acute phase, contributing to improvement in treatment and primary and secondary prevention.
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Sadasivan, Chander, Liliana Cesar, and Baruch B. Lieber. "Mixing of Angiographic Contrast With Blood During Injections in the Cerebral Circulation." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192336.

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In the past, various techniques such as indicator dilution, transit time, parametric imaging, or first-pass distribution have been used to estimate blood flow rates during angiographic contrast injections. We have previously employed the method of modeling contrast concentration curves to assess changes in flow exchange between parent cerebral vessels and cerebral aneurysms due to endovascular treatment by flow divertors [1]. There has been concern, however, that contrast injected under such situations may remain as a separate slug or stream flowing with blood or that contrast may settle from blood in the direction of gravity due to its higher density [2,3]. According to this argument, therefore, the analysis of the transport of angiographic contrast visualized under X-ray cannot be used to represent the transport of blood.
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Reymond, Philippe, Fabrice Merenda, Fabienne Perren, Daniel Rüfenacht, and Nikos Stergiopulos. "Validation of 1D Model of the Systemic Arterial Tree Including the Cerebral Circulation." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192529.

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The aim of this study is to develop a distributed model of the entire systemic arterial tree, coupled to a heart model and including a detailed description of the cerebral arteries. Distributed models of the arterial tree have been studied extensively in the past (Avolio [1], Stergiopulos et al [2], Westerhof et al [3]), however, no model has been developed so far that offers a physiologically relevant coupling to the heart and includes the entire cerebral arterial tree.
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9

Dholakia, R. J., C. Sadasivan, D. J. Fiorella, H. H. Woo, and B. B. Lieber. "Flow Diverted Aneurysmal Hemodynamic Simulations and Validation With Experiments." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14686.

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Brain aneurysms occur due to abnormal ballooning of cerebral arteries. Rupture of the cerebral aneurysms can result in subarachnoid hemorrhage and may prove fatal for patients [1]. Surgical clipping is a highly invasive option for treatment of aneurysms. Endovascular flow diverting stents have recently emerged as a less invasive treatment for cerebral aneurysms. Flow diverters for intracranial aneurysms, are porous metallic mesh tubes deployed across the neck of the aneurysm to exclude the aneurysm from the circulation [2–4]. By producing a substantial reduction of flow inside the aneurysm and by promoting activation of platelets that cross the device into the aneurysm, intra-aneurysmal thrombus is generated and the aneurysm is eventually excluded from the circulation.
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10

Salehi, Seyedeh Sarah, Bahar Firoozabadi, and Mohamad Said Saidi. "Numerical and 1-D modeling of systemic circulation along with cerebral vasculature." In 2012 19th Iranian Conference of Biomedical Engineering (ICBME). IEEE, 2012. http://dx.doi.org/10.1109/icbme.2012.6519652.

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Звіти організацій з теми "Cerebral circulation"

1

Gao, Shanshan, Dongdong Yang, Hengni Yan, Yuxuan Chao, and Yu Fang. Efficacy and safety analysis of Chinese herb injections promoting blood circulation combined with intravenous thrombolysis with alteplase in hyperacute cerebral infarction patients：a systematic review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2020. http://dx.doi.org/10.37766/inplasy2020.6.0093.

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2

Wu, Xiaoqi, Maoxia Fan, Yaobo Pan, and Dona Guo. Quality of Evidence Supporting the Effects of Ginkgo Terpene Lactone Preparations in Ischemic Stroke: An Overview of Systematic Reviews and Meta-Analyses. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0124.

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Анотація:
Review question / Objective: 2.2.1 Type of studies SRs/MAs of Randomized Controlled Trials (RCTs) of GTLP for IS in any language. 2.2.2 Type of Participants Included patients were diagnosed with IS according to international or national standards, regardless of race, age, gender, time of onset, and source of cases. 2.2.3 Type of Intervention The intervention method in the control group was routine treatment, and the intervention method in the intervention group was GTLP treatment or GTLP combined with the treatment of the control group. 2.2.4 Types of outcomes Conclusions at least need to include clinical efficacy analysis and National Institute of Health Stroke Scale (NIHSS). Condition being studied: Stroke is the second leading cause of death and third leading cause of disability globally.Among them, ischemic stroke (IS) accounts for 70% of all stroke types. It is a central nervous system disease caused by cerebral blood circulation disorder, ischemia and hypoxia .The incidence rate is high and increasing year by year, the age of onset is younger, the disability rate is high, and most patients have different degrees of limb motor dysfunction.In order to reduce the burden of stroke on the society and the patient's family, many articles proposed to strengthen the primary stroke prevention - behavior change and drug intervention.
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