Добірка наукової літератури з теми "Cell carcinomas"
Оформте джерело за APA, MLA, Chicago, Harvard та іншими стилями
Ознайомтеся зі списками актуальних статей, книг, дисертацій, тез та інших наукових джерел на тему "Cell carcinomas".
Біля кожної праці в переліку літератури доступна кнопка «Додати до бібліографії». Скористайтеся нею – і ми автоматично оформимо бібліографічне посилання на обрану працю в потрібному вам стилі цитування: APA, MLA, «Гарвард», «Чикаго», «Ванкувер» тощо.
Також ви можете завантажити повний текст наукової публікації у форматі «.pdf» та прочитати онлайн анотацію до роботи, якщо відповідні параметри наявні в метаданих.
Статті в журналах з теми "Cell carcinomas"
Sica, Gabriel, Patrick L. Wagner, Nassar Altorki, Jeffrey Port, Paul C. Lee, Madeline F. Vazquez, and Anjali Saqi. "Immunohistochemical Expression of Estrogen and Progesterone Receptors in Primary Pulmonary Neuroendocrine Tumors." Archives of Pathology & Laboratory Medicine 132, no. 12 (December 1, 2008): 1889–95. http://dx.doi.org/10.5858/132.12.1889.
Повний текст джерелаCapelli, Marco, Giulia Bertino, Patrizia Morbini, Chiara Villa, Stefano Zorzi, and Marco Benazzo. "Neuroendocrine Carcinomas of the Upper Airways: A Small Case Series with Histopathological Considerations." Tumori Journal 93, no. 5 (September 2007): 499–503. http://dx.doi.org/10.1177/030089160709300517.
Повний текст джерелаShilo, Konstantin, Tatiana Dracheva, Haresh Mani, Junya Fukuoka, Isabell A. Sesterhenn, Wei-Sing Chu, Joanna H. Shih, Jin Jen, William D. Travis та Teri J. Franks. "α-Methylacyl CoA Racemase in Pulmonary Adenocarcinoma, Squamous Cell Carcinoma, and Neuroendocrine Tumors: Expression and Survival Analysis". Archives of Pathology & Laboratory Medicine 131, № 10 (1 жовтня 2007): 1555–60. http://dx.doi.org/10.5858/2007-131-1555-mcripa.
Повний текст джерелаFrixen, U. H., J. Behrens, M. Sachs, G. Eberle, B. Voss, A. Warda, D. Löchner, and W. Birchmeier. "E-cadherin-mediated cell-cell adhesion prevents invasiveness of human carcinoma cells." Journal of Cell Biology 113, no. 1 (April 1, 1991): 173–85. http://dx.doi.org/10.1083/jcb.113.1.173.
Повний текст джерелаCheuk, W., M. Y. Kwan, Saul Suster, and John K. C. Chan. "Immunostaining for Thyroid Transcription Factor 1 and Cytokeratin 20 Aids the Distinction of Small Cell Carcinoma From Merkel Cell Carcinoma, But Not Pulmonary From Extrapulmonary Small Cell Carcinomas." Archives of Pathology & Laboratory Medicine 125, no. 2 (February 1, 2001): 228–31. http://dx.doi.org/10.5858/2001-125-0228-ifttfa.
Повний текст джерелаLiddell, Heath, Anton Mare, Sean Heywood, Genevieve Bennett, and Hin Fan Chan. "Clear Cell Papillary Renal Cell Carcinoma: A Potential Mimic of Conventional Clear Cell Renal Carcinoma on Core Biopsy." Case Reports in Urology 2015 (2015): 1–4. http://dx.doi.org/10.1155/2015/423908.
Повний текст джерелаMilroy, C. M., P. J. Robinson, and H. R. Grant. "Primary composite squamous cell carcinoma and large cell neuroendocrine carcinoma of the hypopharynx." Journal of Laryngology & Otology 103, no. 11 (November 1989): 1093–96. http://dx.doi.org/10.1017/s0022215100111107.
Повний текст джерелаNikiforova, Marina N., Edna T. Kimura, Manoj Gandhi, Paul W. Biddinger, Jeffrey A. Knauf, Fulvio Basolo, Zhaowen Zhu, et al. "BRAF Mutations in Thyroid Tumors Are Restricted to Papillary Carcinomas and Anaplastic or Poorly Differentiated Carcinomas Arising from Papillary Carcinomas." Journal of Clinical Endocrinology & Metabolism 88, no. 11 (November 1, 2003): 5399–404. http://dx.doi.org/10.1210/jc.2003-030838.
Повний текст джерелаWysocki, John, Rishi Agarwal, Laura Bratton, Jeremy Nguyen, Mandy Crause Weidenhaft, Nathan Shores, and Hillary Z. Kimbrell. "Mixed Large Cell Neuroendocrine Carcinoma and Adenocarcinoma with Spindle Cell and Clear Cell Features in the Extrahepatic Bile Duct." Case Reports in Pathology 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/347949.
Повний текст джерелаRudland, P. S., S. J. Leinster, J. Winstanley, B. Green, M. Atkinson, and H. D. Zakhour. "Immunocytochemical identification of cell types in benign and malignant breast diseases: variations in cell markers accompany the malignant state." Journal of Histochemistry & Cytochemistry 41, no. 4 (April 1993): 543–53. http://dx.doi.org/10.1177/41.4.8450194.
Повний текст джерелаДисертації з теми "Cell carcinomas"
Spandidos, Athanasia. "Proteomic methods applied to renal cell carcinomas." Thesis, University of Cambridge, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.620561.
Повний текст джерелаLlewelyn, Jenefer Kirstin. "Analysis of gene expression in squamous cell carcinomas." Thesis, King's College London (University of London), 2006. https://kclpure.kcl.ac.uk/portal/en/theses/analysis-of-gene-expression-in-squamous-cell-carcinomas(4ce3ae48-c239-4bc0-918e-e4aa691bd0d2).html.
Повний текст джерелаXinarianos, George. "Genetic alterations in non-small cell lung carcinomas." Thesis, University of Liverpool, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343688.
Повний текст джерелаDowlatshahi, Mitra. "Suppressed Activity of Tumor-Specific T Cells in Human Merkel Cell Carcinomas." Thesis, Harvard University, 2017. http://nrs.harvard.edu/urn-3:HUL.InstRepos:32676135.
Повний текст джерелаHudon, Valerie. "Investigating tumor suppressor genes involved in renal cell carcinomas." Thesis, McGill University, 2010. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=86607.
Повний текст джерелаLe cancer du rein est une maladie complexe qui comprend différents types de cancers classifiés selon leurs caractéristiques histologiques. Certains cas de cancers rénaux sont attribuables à une prédisposition héréditaire et l'étude de ces formes héréditaires de cancer a largement contribué au développement des connaissances concernant la pathogenèse des cancers rénaux. Le travail décrit dans cette thèse porte sur deux maladies héréditaires prédisposant au développement de tumeurs rénales soit la maladie de Von Hippel-Lindau (VHL) et le syndrome de Birt-Hogg-Dubé (BHD) ainsi que les gènes associés à ces dernières, VHL et FLCN respectivement. Premièrement, nous avons étudié le rôle de la régulation de l'assemblage de la matrice extracellulaire (MEC) par VHL durant la tumorigénèse et nous avons démontré que l'assemblage inadéquat de la MEC corrèle avec une croissance tumorale accrue et induit la formation de tumeurs fort vascularisées. Nous avons conclu que la perte de l'intégrité de la MEC favorise l'angiogénèse tumorale en fournissant une voie permettant aux vaisseaux sanguins d'infiltrer la tumeur. Deuxièmement, nous avons développé un modèle murin pour étudier la coopération potentielle entre VHL et p53 durant le développement tumoral. Nous avons observé que l'inactivation simultanée de VHL et p53 accélère la progression d'hémangiomes du foie et d'hémangiosarcomes de la rate. De plus, la perte concomitante de VHL et p53 inhibe le développement de lymphomes normalement associés à l'inactivation de p53. Nos résultats indiquent que les phénotypes apparaissant suite à l'inactivation de VHL et p53 varient selon l'organe étudié. Finalement, nous avons développé un modèle murin pour étudier de la pathologie associée au syndrome BHD et à avons observé la formation d'un continuum de lésions rénales allant de l'hyperprolifération des tubules rénaux à de rares adénomes. Finalement, nous avons conf
Sandström, Karl. "Radioimmunodiagnosis of Head and Neck Squamous Cell Carcinomas : Preclinical Studies." Doctoral thesis, Uppsala universitet, Öron-, näs- och halssjukdomar, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-156523.
Повний текст джерелаEkberg, Tomas. "Diagnosis and Radioimmunotherapy of Head and Neck Squamous Cell Carcinomas." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Universitetsbiblioteket [distributör], 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-8395.
Повний текст джерелаSantos, Harim Tavares dos 1989. "Gotas lipídicas intracitoplasmáticas em carcinomas de glândulas salivares = estudo imunohistoquímico." [s.n.], 2014. http://repositorio.unicamp.br/jspui/handle/REPOSIP/289546.
Повний текст джерелаDissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba
Made available in DSpace on 2018-08-24T17:05:31Z (GMT). No. of bitstreams: 1 Santos_HarimTavaresdos_M.pdf: 2437979 bytes, checksum: 15b0ad672ced32a375d9670dcdeb69ad (MD5) Previous issue date: 2014
Resumo: Introdução: Gotas lipídicas (GL) são organelas altamente reguladas envolvidas na ativação e metabolismo celular assim como em processos inflamatórios e neoplásicos. O aumento da lipogênese levando a um aumento no número de GL é um fenótipo comum a numerosos carcinomas humanos e tem sido associado com: diferenciação, proliferação e agressividade tumoral. Objetivo: Avaliarem carcinomas salivares: a) a frequência e quantidade das GLs citoplasmáticas, correlacionando-as com a morfologia tumoral, grau de diferenciação e proliferação celular e b) a expressão de proteínas relacionadas ao processo secretório celular (STAT5a e mamoglobina). Material e métodos:em 79 casos de carcinomas de glândulas salivares foram utilizados os anticorpos Ki-67, adipophilin, STAT 5a e mamoglobina. A quantificação da imunoreatividade ao adipophilin, STAT 5a e mamoglobina foi através de uma escala semi-quantitativa. A intensidade de marcação do STAT 5a e mamoglobina foi subjetivamente avaliada como fraca/moderada ou intensa. A positividade ao Ki-67 foi calculada através da relação entre o número de células positivas e o número total de células tumorais em três áreas selecionadas da amostra. Resultados:os subtipos histopatológicos que apresentaram positividade ao adipophilin em 50% ou mais das células, foram os casos: MASC (100%), CCA (85,64%), CM (83%), CDS (75%) e CSeb (50%). Índice de proliferação maior que 10% das células tumorais foi observado no CSeb (Ki-67 = 29%), seguido do CDS (Ki-67 = 23,11%) e do CM (Ki-67 = 12,15%). Nos casos de CAC com transformação para alto grau a área transformadaapresentou maior proliferação e lipogênese quando comparada à área convencional. Somente os casos de MASC apresentaram imunorreatividade acentuada para STAT5a e mamoglobina. Conclusões: Embora exista correlação entre o acúmulo de gotas lipídicas e o índice proliferativo do tumor, em alguns tipos de carcinomas salivares tal depósito está possivelmente relacionado à diferenciação celular (CSeb e MASC) ou alteração metabólica (CCA). O acúmulo de Gl refletindo diferenciação celular se apresenta como gotas maiores em contraste com as microgotas frequentemente detectadas nos carcinomas com alto índice proliferativo. No MASC a forte expressão de STAT5a e mamoglobina sugere que a que o acúmulo de GL possivelmente reflete diferenciação do tipo lactacional.Mais estudos são necessários para entender o papel das gotas lipídicas citoplasmáticas em carcinomas de glândulas salivares. (Apoio FAPESP: 2012/18104-4 e 2012/18086-6)
Abstract: Introduction: Lipid droplets (LD) are highly regulated organelles involved in cell activation and metabolism as well as in inflammatory and neoplastic processes Upregulated lipogenesis leading to an increased number of cytoplasmic LD is a common phenotype to numerous human carcinomas and has been associated with: differentiation, proliferation and aggressiveness of the tumor. Objective: to evaluate in salivary carcinomas: a) the frequency and quantity of cytoplasmics LDs, correlating it with tumoral morphology, differentiation grade e cellular proliferation and b) the expression of proteins associated with cellular secretor process (STAT5a and mammaglobin). Material and Methods:in 79 cases of salivary gland carcinomas, an immunohistochemical study was performed with the antibodies Ki-67, adipophilin, STAT 5a and mammaglobin. The positive neoplastic cells were assessed regarding quantity using a semi-quantitative scale. The intensity of expression for each antibody was subjectively evaluated as weak/ moderate or intense.The positivity for Ki-67 was calculated based on the relation between the number of positive cells and the total number of the tumoral cells in three selected areas. Results: the histopatologic subtypes that presented positivity for adipophilin in 50% or more of cells were: AciCC (85,64%), MASC (100%), MC (83%), SDC (75%), SebC (50%). Proliferation index higher than 10% of tumoral cells was observed in SebC (Ki-67 = 29%), SDC (Ki-67 = 23,11%) and MC (Ki-67 = 12,15%). In ACC with high grade transformation the, the transformed area presented both higher proliferation and lipogenesis when compared to the convencional area. Only the cases of MASC presented accentuated immunoreactivity for STAT5a and mammaglobin. Conclusions: Although in salivary carcinomas there is correlation between the accumulation of lipid droplets and the proliferative index of the tumor, in some types of carcinomas such deposit is possibly related to cellular differentiation (SebC and MASC) or metabolic alteration (AciCC). The accumulation of LD that reflects cellular differentiationpresents features of macro droplets in contrast to micro-droplets frequently detected in carcinomas with high proliferative index. In MASC, the strong expression of STAT5a and mammaglobin suggests that LD accumulation is probably due to lactational-like differentiation. More studies are necessary to understand the role of cytoplasmic lipid droplets in salivary gland carcinomas (Supported by FAPESP: 2012/18104-4and2012/18086-6)
Mestrado
Patologia
Mestre em Estomatopatologia
Kass, Youssef Khalil. "Identification of cellular origin and molecular mechanism in basal and squamous cell carcinomas." Doctoral thesis, Universite Libre de Bruxelles, 2012. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209771.
Повний текст джерелаBCCs result from aberrant activation of HH signaling and several mouse models carrying mutations in HH signaling genes are capable to form tumors resembling to human BCCs.
To identify the cell lineage at the origin of BCC and to investigate the role of stem cells in tumor initiation, we followed a genetic approach where we conditionally expressed SmoM2 oncogene (a constitutively active Smoothened mutant) in distinct skin epidermal compartments including SCs. Targeting basal epidermis cells, showed that only SmoM2-clones in the inter follicular epidermis (IFE) and the infundibulum can progress into BCC, whereas SmoM2 expression in Bulge SCs or in matrix transit amplifying progenitor cells never leads to BCC formation. Progressively after SmoM2 expression, tumor-initiating cells lose their normal differentiation to adopt a hair placode-like shape and markers, demonstrating that biochemical and morphological tumour features can be misleading in extrapolating their cellular origin.
The molecular changes occurring in tumor initiating cells and the mechanisms regulating the early steps of cancer development are poorly characterized for the majority of tumors. To address these questions in BCC, we took advantage of our ability to isolate SmoM2 expressing cells at different stages of tumor initiation and progression. Transcriptional profiling of SmoM2-basal IFE cells isolated one week (normal histology) and 4 weeks (dysplastic lesion), suggests that adult IFE cells undergo a reprogramming into embryonic hair follicle (EHFP) like fate. In addition, we showed that Wnt/β-catenin signaling is essential for BCC initiating cell reprogramming into EHFP like fate and for tumor initiation in a cell autonomous manner. Finally, we show that EHFP reprogramming occurs also in human BCCs in addition to the presence of a similar canonical Wnt activation signature to the one revealed in the SmoM2-BCC mouse model.
SCC is the second most frequent skin cancers after BCC and mutations in p53 and Ras genes has been suggested to be potentially the primary events in this tumour. SCCs present signs of squamous differentiation, suggesting that SCCs may originate from the inter follicular epidermis (IFE). To identify the cell lineage at the origin of SCC and the role of the hair follicle SCs in tumor initiation, we use a genetic tools driving oncogenic KRas (KRasG12D) expression at physiological levels in different epidermal compartments.
Targeting KRasG12D expression in bulge SCs and their progeny or in IFE results in benign tumor development with no sign of malignant transformation. In contrast, KRasG12D expression in HF Transit amplifying (TA) matrix cells do not promotes any macroscopic tumors or microscopic defects in the epidermis. Interestingly, papillomas arising from the IFE express follicular markers such as CD34 and K17, indicating that the expression of HF markers by tumor cells does not necessarily reflect their cellular origin. Using a combination of deletion of both p53 alleles together with KRasG12D expression, we showed that bulge SCs and/or their progeny but not HF matrix TA cells, promote SCC formation, suggesting that additional genetic hits such as p53 are required to promote full-blown invasive skin SCC.
In summary, our work demonstrated the non-follicular origin of BCC resulting from Smo mutation, as well as the implication of the IFE progenitors in tumor initiation. We also revealed the progressive reprogramming of BCC initiating cells towards an EHFP-like fate and the key role of Wnt/β-catenin pathway in this process. In contrast, we showed the competence of several epidermal lineages to initiate benign tumors upon expression of KRasG12D oncogene at physiological levels. We also demonstrated that lineage -specific markers expression within tumor cells does not necessarily reflect their cellular origin. Finally, we demonstrated the requirement of additional hits, such as P53 loss, to promote malignant progression in the context of oncogenic Ras.
Doctorat en Sciences biomédicales et pharmaceutiques
info:eu-repo/semantics/nonPublished
Blat, Irene Catherine. "Functional miRNA regulation of metastatic genes promotes tumor cell dissemination in non-small cell and small cell lung carcinomas." Thesis, Massachusetts Institute of Technology, 2013. http://hdl.handle.net/1721.1/80982.
Повний текст джерелаCataloged from PDF version of thesis. Vita.
Includes bibliographical references.
Tumor progression, from initiation to advanced metastatic disease, requires the orchestration of a diverse group of cell-intrinsic and extrinsic factors. This multifactorial disease is promoted by an accumulation of genetic and epigenetic changes that confer selective advantage to cells and enable unrestrained proliferation, survival, motility, and self-renewal. While much emphasis over the last 35 years has been dedicated to understanding the regulators of tumor initiation, the number of cancer-related deaths worldwide continues to rise, of which the majority are attributed to metastasis. The lengthy progression to metastasis requires invasion out of the primary tumor site and into the bloodstream, survival in and exit from circulation, and colonization and expansion in a foreign environment. Developmental pathways such as the Transforming Growth Factor [beta] (TGF[beta]) signaling network are frequently dysregulated during metastatic progression due to the similarities between early embryogenesis and tumor progression. Furthermore, the TGF[beta] pathway highlights how cell-intrinsic and extrinsic signals help coordinate the complex interactions required between tumor cells, as well as those of the tumor microenvironment to achieve metastasis. Facilitating alterations to pathways such as TGF[beta] and many others are modulators of gene expression that can target multiple nodes of the signaling cascade instead of requiring genetic alterations to single genes. Moreover, in the last decade, emphasis on the role of noncoding RNAs in post-transcriptional modifications has revealed their important contribution in the regulation of developmental programs across metazoan species. More recently, the role of alterations in expression of small noncoding RNAs, microRNAs (miRNAs) has emerged as a significant contributor to disease states, including each stage of tumor progression from initiation to metastatic colonization. miRNAs hold great promise not only as biomarkers but also as potential therapeutics. For these reasons, we have characterized the role of two important examples of miRNA families - the miRNA-200 family and the miRNA-1 7~92 cluster - that regulate early stages of tumor initiation in addition to later steps of cell migration, invasion, survival, and colonization. Examination of their contribution to tumor progression in relevant in vitro and in vivo cellular contexts using genetic tools reveals they are functional contributors to tumor cell dissemination. Furthermore, modulation of their expression in the appropriate tumor microenvironments elucidates a network of targets underlying the molecular mechanisms of metastasis.
by Irene Catherine Blat.
Ph.D.
Книги з теми "Cell carcinomas"
Small cell carcinomas: Causes, diagnosis and treatment. New York: Nova Biomedical Books, 2009.
Знайти повний текст джерелаMaldonado, Jonathon G. Small cell carcinomas: Causes, diagnosis and treatment. New York: Nova Biomedical Books, 2009.
Знайти повний текст джерелаMaldonado, Jonathon G., and Mikayla K. Cervantes. Small cell carcinomas: Causes, diagnosis and treatment. New York: Nova Biomedical Books, 2009.
Знайти повний текст джерелаHastie, G. Michael. Ribonucleic acid isolation from small cell lung carcinomas. Sudbury, Ont: Laurentian University, 1992.
Знайти повний текст джерелаEl-Salam, Mahmoud Abd. The prevalence of different human papillomavirus types and p53 mutations in laryngeal squamous cell carcinomas. [s.l.]: typescript, 1994.
Знайти повний текст джерелаKuo, Michael Jeo-Ming. Aberrations of chromosome arms 5q and 8p in squamous cell carcinomas of the head and neck. Birmingham: University of Birmingham, 1998.
Знайти повний текст джерелаBurton, Jean. A study of cellular proliferation rates in squamous cell carcinomas of the lung, with relation to p53 status. [S.l: The Author], 1994.
Знайти повний текст джерелаRenal cell carcinoma. Shelton, Conn: People's Medical Pub. House, 2009.
Знайти повний текст джерелаMortensen, Daniel V. Squamous cell carcinoma. Hauppauge, N.Y: Nova Science Publishers, 2011.
Знайти повний текст джерелаBukowski, Ronald M., and Andrew Novick. Renal Cell Carcinoma. New Jersey: Humana Press, 2000. http://dx.doi.org/10.1385/1592592295.
Повний текст джерелаЧастини книг з теми "Cell carcinomas"
Knowlton, Christin A., Michelle Kolton Mackay, Tod W. Speer, Robyn B. Vera, Douglas W. Arthur, David E. Wazer, Rachelle Lanciano, et al. "Carcinomas: Basal Cell Carcinoma." In Encyclopedia of Radiation Oncology, 96. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-540-85516-3_1059.
Повний текст джерелаMeyer-Breiting, Erhard, and Arne Burkhardt. "Squamous Cell Carcinomas." In Tumours of the Larynx, 79–113. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-71100-8_5.
Повний текст джерелаCorrin, B. "Small Cell Carcinomas Versus (Atypical) Carcinoids." In Clinical and Experimental Pathology of Lung Cancer, 47–52. Dordrecht: Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-5036-8_5.
Повний текст джерелаChahoud, Jad, Gabriel G. Malouf, and Nizar M. Tannir. "Translocation Renal Cell Carcinomas." In Rare Genitourinary Tumors, 41–52. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-30046-7_3.
Повний текст джерелаWeissferdt, Annikka, and Cesar A. Moran. "Non-Small Cell Carcinomas." In Diagnostic Pathology of Pleuropulmonary Neoplasia, 53–120. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0787-5_3.
Повний текст джерелаJonasch, Eric, and Patrick G. Pilie. "Hereditary Renal Cell Carcinomas." In Rare Kidney Tumors, 1–10. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-96989-3_1.
Повний текст джерелаLiu, Dongyou. "Basal Cell and Squamous Cell Carcinomas." In Tumors and Cancers, 9–14. Boca Raton : Taylor & Francis, 2018. | Series: Pocket guides to biomedical sciences | “A CRC title, part of the Taylor & Francis imprint, a member of the Taylor & Francis Group, the academic division of T&F Informa plc.”: CRC Press, 2017. http://dx.doi.org/10.1201/9781315120553-3.
Повний текст джерелаStöppler, H., M. Conrad Stöppler, M. Kisiela, A. Holzbach, I. Moll, P. Houdek, and R. Moll. "Merkel Cell Carcinomas Possess Telomerase Activity." In The Merkel Cell, 237–40. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-662-10358-6_35.
Повний текст джерелаSchwartz, Robert A. "Appendageal Carcinomas, Merkel Cell Carcinoma, and Cutaneous Sarcomas." In Skin Cancer, 71–79. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3790-7_8.
Повний текст джерелаCompton, Carolyn C., David R. Byrd, Julio Garcia-Aguilar, Scott H. Kurtzman, Alexander Olawaiye, and Mary Kay Washington. "Cutaneous Squamous Cell Carcinoma and Other Cutaneous Carcinomas." In AJCC Cancer Staging Atlas, 357–70. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-2080-4_29.
Повний текст джерелаТези доповідей конференцій з теми "Cell carcinomas"
Mishra, Amita, Archit Pandit, Namit Kalra, and Bhawna Narula. "Primary signet ring cell mucinous carcinoma ovary: A very rare neoplasm." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685405.
Повний текст джерелаStravinskaite, Kristina, and Raimundas Sakalauskas. "Multiple Primary Carcinomas: Kidney Clear Cell Carcinoma, Rectum Adenocarcinoma, Recessus Piriformis Squamose Cell Carcinoma And Non Small Cell Lung Carcinoma." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5876.
Повний текст джерелаLinka, RM, Q. Zhu, C. Wiek, J. Schipper, W. Birchmeier, and K. Scheckenbach. "Stem Cell Therapies of Head and Neck Squamous Cell Carcinomas." In Abstract- und Posterband – 90. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Digitalisierung in der HNO-Heilkunde. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1686023.
Повний текст джерелаFeil, L., J. Senz, M. Ta, J. Huvila, K. Greif, B. Krämer, S. Brucker, et al. "Molecular stratification of clear cell ovarian carcinomas." In Kongressabstracts zur Tagung 2020 der Deutschen Gesellschaft für Gynäkologie und Geburtshilfe (DGGG). © 2020. Thieme. All rights reserved., 2020. http://dx.doi.org/10.1055/s-0040-1718135.
Повний текст джерелаRollison, Dana E., Anna R. Giuliano, Jane L. Messina, Neil A. Fenske, Basil S. Cherpelis, Vernon K. Sondak, Richard G. Roetzheim, et al. "Abstract 950: Merkel cell polyomavirus infection in cutaneous squamous cell carcinomas." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-950.
Повний текст джерелаHe, Qingli, Harvey Lui, David Zloty, Bryce Cowan, Larry Warshawski, David I. McLean, and Haishan Zeng. "Microscopic fluorescence spectral analysis of basal cell carcinomas." In SPIE Proceedings, edited by Qingming Luo, Lihong V. Wang, Valery V. Tuchin, and Min Gu. SPIE, 2007. http://dx.doi.org/10.1117/12.741593.
Повний текст джерелаSoni, Abhishek, Nupur Bansal, A. K. Dhull, Vivek Kaushal, Rajeev Atri, and Monica Verma. "Diagnostic dilemma of mesonephric adenocarcinoma cervix." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685283.
Повний текст джерелаSalmanzadeh, Alireza, Harsha Kittur, Michael B. Sano, Mark A. Stremler, P. Christopher Roberts, Eva M. Schmelz, and Rafael V. Davalos. "Investigating Dielectrophoretic Signature of Mouse Ovarian Surface Epithelial Cells, Macrophages and Fibroblasts." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80872.
Повний текст джерелаLal, Anita, Rebecca Panos, Mira Marjanovic, Michael Walker, Eloisa Fuentes, W. David Henner, Ljubomir Buturovic, and Meredith Halks-Miller. "Abstract 1724: A gene expression profile test to resolve squamous cell carcinomas of head & neck from squamous cell carcinomas of the lung." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1724.
Повний текст джерелаSalas-García, I., F. Fanjul-Vélez, N. Ortega-Quijano, and J. L. Arce-Diego. "Photodynamic diagnosis model for depth evaluation of basal cell carcinomas." In Biomedical Optics. Washington, D.C.: OSA, 2012. http://dx.doi.org/10.1364/biomed.2012.bsu3a.6.
Повний текст джерелаЗвіти організацій з теми "Cell carcinomas"
Mahoney, My G., Ulrich Rodeck, and Jouni Uitto. Molecular Characterization of Squamous Cell Carcinomas From Recessive Dystrophic Epidermolysis Bullosa. Fort Belvoir, VA: Defense Technical Information Center, September 2006. http://dx.doi.org/10.21236/ada463709.
Повний текст джерелаMahoney, My G., Ulrich Rodeck, and Jouni Uitto. Molecular Characterization of Squamous Cell Carcinomas Derived from Recessive Dystrophic Epidermolysis Bullosa. Fort Belvoir, VA: Defense Technical Information Center, June 2005. http://dx.doi.org/10.21236/ada446877.
Повний текст джерелаMahoney, My G., Ulrich Rodeck, and Jouni Uitto. Molecular Characterization of Squamous Cell Carcinomas Derived from Recessive Dystropic Epidermolysis Bullosa. Fort Belvoir, VA: Defense Technical Information Center, June 2004. http://dx.doi.org/10.21236/ada427184.
Повний текст джерелаMahoney, My G., Ulrich Rodeck, and Jouni Uitto. Molecular Characterization of Squamous Cell Carcinomas Derived From Recessive Dystrophic Epidermolysis Bullosa. Fort Belvoir, VA: Defense Technical Information Center, June 2003. http://dx.doi.org/10.21236/ada419358.
Повний текст джерелаAlessa, Mohammed, Tayba Wahedi, Jumanah Alsairafi, Nouf Almatrafi, Wisal Shuaib, Johara Alnafie, Fatimah Alzubaidi, and Soha Elmorsy. Prevalence of Thyroid cancer in Saudi Arabis: Systematic review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0088.
Повний текст джерелаJones, Richard. Ovarian Carcinoma Stem Cells. Fort Belvoir, VA: Defense Technical Information Center, May 2009. http://dx.doi.org/10.21236/ada508216.
Повний текст джерелаFreire, Mariana, Diana Martins, Maria Filomena Botelho, and Fernando Mendes. Biomarkers of resistance mechanisms in innovative lung cancer treatments - A systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0011.
Повний текст джерелаGiancotti, Filippo G. Cell-Matrix Interactions in Breast Carcinoma Invasion. Fort Belvoir, VA: Defense Technical Information Center, January 1996. http://dx.doi.org/10.21236/ada305438.
Повний текст джерелаCuratola, Anna M. Cell-Matrix Interactions in Breast Carcinoma Invasion. Fort Belvoir, VA: Defense Technical Information Center, January 2000. http://dx.doi.org/10.21236/ada391446.
Повний текст джерелаDrucker, Aaron, Gaelen P. Adam, Valerie Langberg, Abhilash Gazula, Bryant Smith, Farah Moustafa, Martin A. Weinstock, and Thomas A. Trikalinos. Treatments for Basal Cell and Squamous Cell Carcinoma of the Skin. Agency for Healthcare Research and Quality, 2017. http://dx.doi.org/10.23970/ahrqepccer199.
Повний текст джерела