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Добірка наукової літератури з теми "Catecholamines"

Автор: Grafiati

Опубліковано: 4 червня 2021

Оновлено: 30 липня 2024

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Статті в журналах з теми "Catecholamines"

Wang, Wenping, Ximing Wu, Chung S. Yang, and Jinsong Zhang. "An Unrecognized Fundamental Relationship between Neurotransmitters: Glutamate Protects against Catecholamine Oxidation." Antioxidants 10, no. 10 (September 30, 2021): 1564. http://dx.doi.org/10.3390/antiox10101564.

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Neurotransmitter catecholamines (dopamine, epinephrine, and norepinephrine) are liable to undergo oxidation, which copper is deeply involved in. Catecholamine oxidation-derived neurotoxicity is recognized as a pivotal pathological mechanism in neurodegenerative diseases. Glutamate, as an excitatory neurotransmitter, is enriched in the brain at extremely high concentrations. However, the chemical biology relationship of these two classes of neurotransmitters remains largely unknown. In the present study, we assessed the influences of glutamate on the autoxidation of catecholamines, the copper- and copper-containing ceruloplasmin-mediated oxidation of catecholamines, the catecholamine-induced formation of quinoprotein, catecholamine/copper-induced hydroxyl radicals, and DNA damage in vitro. The results demonstrate that glutamate, at a physiologically achievable molar ratio of glutamate/catecholamines, has a pronounced inhibitory effect on catecholamine oxidation, catecholamine oxidation-evoked hydroxyl radicals, quinoprotein, and DNA damage. The protective mechanism of glutamate against catecholamine oxidation could be attributed to its restriction of the redox activity of copper via chelation. This previously unrecognized link between glutamate, catecholamines, and copper suggests that neurodegenerative disorders may occur and develop once the built-in equilibrium is disrupted and brings new insight into developing more effective prevention and treatment strategies for neurodegenerative diseases.

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Padbury, J. F., A. M. Martinez, S. L. Thio, E. E. Burnell, and J. A. Humme. "Free and sulfoconjugated catecholamine responses to hypoxia in fetal sheep." American Journal of Physiology-Endocrinology and Metabolism 257, no. 2 (August 1, 1989): E198—E202. http://dx.doi.org/10.1152/ajpendo.1989.257.2.e198.

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Plasma catecholamines circulate either in conjugated or unconjugated forms. In adult humans, sulfoconjugated catecholamines predominate; however, there is considerable variation between species. In a variety of pathophysiological states catecholamine conjugation is believed to represent an important mechanism of inactivation of high circulating catecholamine levels. To date, there have been few data in developing animals or humans on catecholamine sulfoconjugation. We studied the differences in free and sulfoconjugated catecholamines in full term (141 +/- 1 days) and preterm (123 +/- 1 days) chronically catheterized fetal sheep and determined the changes in free and sulfoconjugated catecholamines in response to hypoxia. The results demonstrate that term and preterm animals have a comparable percentage of basal circulating sulfoconjugated catecholamines (free-to-total ratio 50-60%). In response to hypoxia, both free and sulfoconjugated catecholamines were promptly elevated with significant increases in each by 5 min of hypoxia. This was true for both term and pretern animals. The proportion of free and total catecholamines remained relatively constant during hypoxia despite a 5- to 10-fold increase in circulating levels of each. These data demonstrate that fetal sheep, as early as 80% gestation, have a well developed mechanism for sulfoconjugation and subsequent inactivation of the high circulating levels of catecholamines seen during fetal and newborn life.

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Koller, M. "Results for 74 substances tested for interference with determination of plasma catecholamines by "high-performance" liquid chromatography with electrochemical detection." Clinical Chemistry 34, no. 5 (May 1, 1988): 947–49. http://dx.doi.org/10.1093/clinchem/34.5.947.

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Abstract Various catecholamine metabolites, catecholamine-related compounds, catechols, drugs, amines, and other nitrogen compounds were injected onto an HPLC system ("ClinRep Catecholamine-Plasma" assay kit with a reversed-phase C18 column) used for measuring catecholamines. None of the 74 substances tested co-eluted with any of the catecholamines--norepinephrine, epinephrine, or dopamine--or with the internal standard, 3,4-dihydroxybenzylamine.

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Stein, H. M., A. Martinez, K. Oyama, L. Blount, and J. F. Padbury. "Effect of corticosteroids on free and sulfoconjugated catecholamines at birth in premature newborn sheep." American Journal of Physiology-Endocrinology and Metabolism 268, no. 1 (January 1, 1995): E28—E32. http://dx.doi.org/10.1152/ajpendo.1995.268.1.e28.

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We previously demonstrated that prenatal corticosteroids attenuated the expected exponential increase in circulating catecholamines at birth. The present studies were undertaken to determine if alteration in sulfoconjugation could account for this attenuation. Catheterized fetal lambs received saline (n = 6) or corticosteroids (n = 8) intravenously for 60 h. The lambs were delivered by cesarean section at 130 +/- 1 days gestation. Ventilatory and cardiovascular responses and plasma catecholamine concentrations were measured for 2 h after birth. Although plasma free catecholamines levels were higher in controls than in corticosteroid-treated fetuses, the sulfoconjugated levels were similar in the two groups. Thus the corticosteroid-treated fetuses had a higher proportion of plasma sulfoconjugated catecholamines consistent with the possibility that sulfoconjugation was augmented during intrauterine life. After birth, the corticosteroid-treated animals showed an attenuated increase in plasma free catecholamine levels compared with controls but a similar increase in sulfoconjugated catecholamine levels to the control animals. The proportion of plasma sulfoconjugated catecholamines was higher in the corticosteroid-treated animals; however, the increase in sulfoconjugated catecholamines was insufficient to account for the attenuated overall increase in total catecholamines in the corticosteroid-treated animals.

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Davidson, D. Fraser. "Phaeochromocytoma with Normal Urinary Catecholamines: The Potential Value of Urinary Free Metadrenalines." Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 39, no. 6 (November 2002): 557–66. http://dx.doi.org/10.1177/000456320203900603.

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Background Normal urine catecholamine values in patients with phaeochromocytoma is an occasional finding and may lead to a missed diagnosis. Additional urinary free metadrenaline analysis may be of value in this situation. Methods In addition to vanillylmandelic acid, homovanillic acid and the catecholamines, urinary free normetadrenaline (fNMA) and free metadrenaline (fMA) were measured. This report describes six confirmed cases of phaeochromocytoma showing normal urinary catecholamine output and compares fMA results and tumour size with other confirmed cases where the urine catecholamines were increased. Results Urine catecholamines in these patients with, on average, smaller tumours, were all normal. Urinary fNMA and fMA were available on five patients, and were increased in three. The data suggest that, unlike the catecholamines, urinary fNMA and fMA could be a useful predictor of tumour size. Conclusion The inclusion of fNMA and fMA in the test profile is likely to be of additional benefit in tumour detection, particulariy when catecholamines or other metabolites are normal.

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Rosano, T. G., T. A. Swift, and L. W. Hayes. "Advances in catecholamine and metabolite measurements for diagnosis of pheochromocytoma." Clinical Chemistry 37, no. 10 (October 1, 1991): 1854–67. http://dx.doi.org/10.1093/clinchem/37.10.1854.

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Abstract Assessment of catecholamine production and excretion is important in the laboratory detection of pheochromocytoma, a rare but curable cause of hypertension. Advances in catecholamine and metabolite methodologies have enhanced the diagnostic acumen by increasing analytical sensitivity and eliminating many of the interferences observed with earlier methods. Estimation of urinary catecholamines metanephrine and vanillylmandelic acid is routinely used in the biochemical detection of pheochromocytoma and in monitoring the completeness of tumor excision as well as the possibility of recurrence. Traditional spectrophotometric and fluorometric methods for urinary catecholamines and their metabolites are being replaced by highly sensitive and selective chromatographic methods. The ability to quantify individual catecholamines and metanephrines by high-performance liquid chromatography is of particular value for detecting familial forms of the tumor that may secrete epinephrine. Plasma norepinephrine and epinephrine measurements are of additional diagnostic value in determining recent catecholamine release and response to clonidine suppression. For either urine or plasma measurements, appropriate patient preparation, sample collection, and method validation along with an understanding of the variable pattern of catecholamine secretion and metabolism in pheochromocytoma are essential. Advances in laboratory methodology and reference intervals for catecholamines for clinical interpretation are reviewed.

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Zhao, Lin, Xiaoran Zhang, Xu Meng, Ting Zhang, Hua Fan, Qiongyu Zhang, Yecheng Liu, Xianliang Zhou, and Huadong Zhu. "The Clinical Characteristics of Pheochromocytomas and Paragangliomas with Negative Catecholamines." Journal of Clinical Medicine 11, no. 19 (September 23, 2022): 5583. http://dx.doi.org/10.3390/jcm11195583.

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Pheochromocytomas and paragangliomas (PPGLs) associated with negative catecholamines are not uncommon. However, few studies have examined clinical features of patients with these tumors. In the absence of available data, it is difficult to identify characteristics of patients with potential PPGLs and normal serum and urine screens. Therefore, an analysis of patients with PPGLs was conducted retrospectively to compare the clinical features of patients with positive and negative catecholamines. This study included 214 patients, including 69 patients with negative catecholamines. Prevalence rates of diabetes (p < 0.001) and hypertension (p < 0.001) were lower and tumor diameter (p < 0.001) was smaller in the negative-catecholamine group compared with the positive-catecholamine group. Multivariable logistic regression analysis showed that extra-adrenal PPGLs were independently positively associated with negative catecholamines (p = 0.004); hypertension (p = 0.001) and tumor diameter (p = 0.016) were independently negatively associated with negative catecholamines. There was no significant difference in tumor recurrence between the two groups (mean follow-up, 20.54 ± 11.83 months) (p = 0.44). The results demonstrated that PPGL patients with negative catecholamines were more likely to have extra-adrenal tumors and less likely to have comorbidities, and these patients should also be closely monitored for tumor recurrence.

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Oyama, K., J. Padbury, A. Martinez, B. Chappell, H. Stein, L. Blount, and E. Buhl. "Free and sulfoconjugated catecholamine responses at birth in newborn sheep." American Journal of Physiology-Endocrinology and Metabolism 263, no. 1 (July 1, 1992): E23—E27. http://dx.doi.org/10.1152/ajpendo.1992.263.1.e23.

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There have been little data on catecholamine sulfoconjugation in developing animals or humans. We studied the differences in free and sulfoconjugated catecholamines at birth in newborn sheep. Baseline concentrations of sulfoconjugated norepinephrine and epinephrine were the predominant form of circulating catecholamine, representing 77 +/- 4 and 65 +/- 12% of total circulating catecholamines, respectively. At birth the free epinephrine concentration increased 10-fold (49 +/- 27 to 653 +/- 21 pg/ml, respectively), and plasma free norepinephrine concentration rose 4-fold (307 +/- 92 to 1,178 +/- 389 pg/ml). In contrast, there was only a transient twofold increase in the sulfoconjugated epinephrine. There was no increase in the sulfoconjugated form of norepinephrine. These data demonstrate that, while the near-term newborn sheep has a well-developed mechanism for sulfoconjugation of circulating catecholamines, this does not occur rapidly. During the logarithmic increases of circulating catecholamines at birth, there are not commensurate increases in the concentration of sulfoconjugated norepinephrine or epinephrine. Thus sulfoconjugation does not appear to represent a significant mechanism for inactivation of the high circulating levels of catecholamines seen at birth.

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Waele, Jean-Pascal De, Michel Anctil, and Mats Carlberg. "Biogenic catecholamines in the cnidarian Renilla köllikeri: radioenzymatic and chromatographic detection." Canadian Journal of Zoology 65, no. 10 (October 1, 1987): 2458–65. http://dx.doi.org/10.1139/z87-371.

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The presence of biogenic catecholamines in the colonial anthozoan Renilla köllikeri was assessed with a radioenzymatic assay and thin-layer chromatographic separation of extracts from different parts of the colony. Confirmation of catecholamine detection was also obtained using an HPLC technique with electrochemical detection. All three catecholamines, i.e., dopamine, noradrenaline, and adrenaline, were detected to varying degrees in the colonial compartments. Unidentified inhibitory factor(s) endogenous to Renilla tissues prevented the detection of internal catecholamine standards to an extent that was dependent upon time of sampling and part of the colony assayed. Peaks in catecholamine levels fluctuated sharply over the 10-month sampling period. Dopamine was the most frequently detected catecholamine, with levels generally higher than those of the other two amines, especially in the autozooids. The presence of adrenaline is reported for the first time in a coelenterate species. These results suggest that catecholamines are present in the most primitive metazoan phylum known to possess a nervous system. The physiological significance of these findings is discussed.

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Kjeldsen, S. E., K. Gjesdal, P. Leren, and I. K. Eide. "Decreased Platelet-Free Dopamine and Unchanged Noradrenaline and Adrenaline in Essential Hypertension." Thrombosis and Haemostasis 60, no. 02 (1988): 251–54. http://dx.doi.org/10.1055/s-0038-1647040.

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SummaryThe content of free-catecholamines in blood platelets is much higher than in plasma and platelet catecholamines must be taken up from plasma, since platelets lack the enzymes for catecholamine synthesis. There is some evidence that platelet catecholamine content under certain circumstances may be an integrated measure of plasma catecholamine concentrations over time. Platelet-free catecholamines were therefore assayed in 18 untreated patients with essential hypertension and in 16 normotensive control subjects. Mean platelet-free dopamine in the hypertensive group was 3.7 ± 0.4 pg/mg platelet weight, i.e. significantly less than the 6.5 ± 0.9 pg/mg found in the normotensive (p <0.005). Platelet contents of noradrenaline and adrenaline did not differ. Decreased platelet-free dopamine and unchanged platelet noradrenaline and adrenaline persisted after adjustment for increased body weight in the hypertensive group. Although the reasons for decreased platelet-free dopamine in the hypertensive group remain unknown, this finding may add to previous result showing facilitated release of granular contents from blood platelets in patients with essential hypertension. Our data do not support platelet levels of free-catecholamines to be a marker of increased sympathetic tone in essential hypertension.

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Дисертації з теми "Catecholamines"

Jonsson, Anna. "Reference interval for urinary catecholamines and methylated catecholamines analysed using HPLC." Thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-180252.

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Catecholamines are stress hormones that are produced and released by a rare tumor called pheochromocytoma. This tumor can cause hypertension which if undiagnosed and untreated leads to death. Since good therapy is available, it is important to find the tumor in time. The most common way to diagnose the tumor is measurement of the biochemical markers; catecholamines and their metabolites, methylated catecholamines. After observation that almost all normetanephrine results for women were higher than the upper reference limit and therefore pathological, the accuracy of the present reference intervals was questioned. Therefore new reference intervals for both urinary catecholamines and methylated catecholamines were developed by analysis of 46 samples using HPLC. Creatinine was analysed in acidified urine in order to see if the results became the same as when analysed in non-acidified urine. Urinary catecholamines and methylated catecholamines were analysed using HPLC. Comparison between measurement of creatinine in acidified urine and non-acidified urine with an enzymatic method was performed using Architect ci 8200, Abbott. As suspected, there was a difference between the present and new intervals. Therefore the new intervals will be used for future diagnosis. There was no difference between the two treatments of creatinine samples wherefore it can be measured in both.In conclusion reference intervals determind in this study will be used and it was shown that creatinine can be measured in acidified urine.

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Achola, Kohath Jenge. "Measurements of catecholamines during anaesthesia." Thesis, University of Leicester, 1988. http://hdl.handle.net/2381/33571.

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Factors affecting catecholamine levels in vitro were studied using the modified high pressure liquid chromatography method with electrochemical detection. Differential centrifugation showed that platelet-rich plasma contained significantly higher catecholamine levels than platelet-poor plasma. Serum samples had significantly higher catecholamine levels than plasma. Plasma or serum samples clotted with glass beads had significantly higher catecholamine levels than those without. Therefore, consistent results can only be obtained when catecholamine samples are spun at the same speed, either plasma or serum can be used, but not both in a single study. Post-dated blood for transfusion was used to study stability of catecholamines, and showed that catecholamines are stable. Hence, the collection of blood samples for catecholamine measurements was modified. Blood samples were collected in Vacutainer tubes containing lithium heparin without antioxidants and not pre-cooled, samples were spun at the convenient time. This was welcomed by the clinicians who did not have to interrupt clinical assessment to care for blood samples as with the former method. The three clinical studies showed no significant differences in catecholamine levels in patients undergoing laryngoscopy with and without tracheal intubation, whether or not the patients were beta blocked or had received topical tracheal analgesia. The mean catecholamine levels were within the normal range. No relationships between baseline catecholamine levels and the baseline blood pressures or heart rate nor between the changes in catecholamine levels from the baseline and the corresponding changes in blood pressures or heart rate. The Injury Severity Score in minor injured patients had no relationship with plasma catecholamine levels, and no significant rise in noradrenaline levels when the ISS<30 and adrenaline levels when the ISS<17. The studies suggest that catecholamine levels are of no value in assessing the severity of minor injuries, or changes in blood pressures or heart rate during anaesthesia.

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Henegar, Jeffrey R. "The role of catecholamines in angiotensin II - related myocardial damage." free to MU campus, to others for purchase, 1996. http://wwwlib.umi.com/cr/mo/fullcit?p9821330.

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Khorchid, Amani. "The effects of catecholamines on oligodendrocytes /." Thesis, McGill University, 2002. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=82901.

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The aim of the studies underlying this thesis was to characterize the effect of catecholamines on developing rat brain oligodendrocytes by characterizing the alpha1-adrenoceptor (alpha1-AR) expressed in oligodendrocytes, determining the signaling pathway downstream from the second messenger molecules, and demonstrating a cytotoxic response to catecholamines.
Norepinephrine (NE) caused a time- and concentration-dependent increase in total inositol phosphate formation (IPt). Using various selective antagonists, we identified that, similar to progenitors, the alpha 1A-AR subtype in mature oligodendrocytes is mediating NE-induced IP t formation.
In examining the signaling transduction pathway, we found that in oligodendrocyte progenitors NE only in the presence of propranolol, a beta-AR antagonist, increased mitogen-activated protein kinase (MAPK) activity.
We further revealed that catecholamines exposure results in cytotoxicity to oligodendrocyte cultures, which is dependent on the dose of dopamine or norepinephrine used, and on the developmental stage of the cultures, with oligodendrocyte progenitors being more vulnerable. Catecholamines caused an increase in oxidative stress as elucidated from reduced intracellular glutathione levels, and increased accumulation in reactive oxygen species and in stress-induced protein, heme oxygenase-1.
In conclusion, our findings showed that developing oligodendrocytes express all three alpha1-AR subtypes but that only the alpha1A -AR was involved in NE-mediated IPt, formation. We also demonstrated that NE activated MAPK and c-fos expression in oligodendrocyte progenitors, suggesting trophic response. Lastly, we demonstrate that catecholamine could induce cytotoxicity in oligodendrocyte cultures, which is developmentally regulated, mediated by oxidative stress and have characteristics of apoptosis in progenitor cells. (Abstract shortened by UMI.)

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AZOUI, RACHIDA. "Catecholamines intra-erythrocytaires : metabolisme et transport." Paris 6, 1995. http://www.theses.fr/1995PA066511.

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Ce travail a ete realise afin d'evaluer le role joue par les globules rouges dans la clairance metabolique des catecholamines. Nous avons confirme, in vitro, qu'il existait une accumulation de catecholamines au sein des erythrocytes humains. Cette accumulation varie selon la structure de l'amine (dopamine>adrenaline>noradrenaline). La cinetique d'accumulation des catecholamines decroit progressivement quand leurs concentrations plasmatiques augmentent ; elle est fonction de la structure de l'amine. Cette accumulation est reversible, dependante de la temperature et de la p0#2. La cinetique de ce phenomene a ete exploree chez le rat anesthesie au cours de perfusion i. V. De catecholamines. A faibles doses, (30, 300 pmole), l'accumulation des catecholamines par les erythrocytes est alors activee. Au dela de ces concentrations le phenomene devient saturable. Nous avons cherche a identifier un metabolisme intra-erythrocytaire des catecholamines en particulier de la dopamine. La catechol-o-methyl transferase a ete inhibee par l'injection i. P. De 1,2-dimethyl-3-hydroxypyridin-4-one (cp20), chez le rat conscient. Ce traitement induit une augmentation significative de la da plasmatique et erythrocytaire. Nos resultats montrent que cette incubation, in vitro, conduit a une augmentation de la da glycuroconjuguee dans les erythrocytaires. La structure responsable de cette accumulation a ete identifiee comme etant le transporteur de choline, car la choline inhibe l'influx de la dopamine et stimule son efflux. Nous avons montre que ce mecanisme de transport semblait etre module par l'insuline aussi bien in vivo qu'in vitro

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Baik, Sonya A. "Catecholamines and basal metabolism in the myocardium." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ34085.pdf.

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Siraki, Arno Garakhanian. "Antioxidant and pro-oxidant nature of catecholamines." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/MQ54170.pdf.

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Tippett, P. A. T. "Metabolism of catecholamines in some clinical diseases." Thesis, University of Southampton, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.383298.

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Avkiran, Metin. "Catecholamines and arrhythmias in the anaesthetized rat." Thesis, University of Bath, 1987. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.374610.

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Holtbäck, Ulla. "The role of catecholamines in regulation of renal tubular sodium transport /." Stockholm, 1998. http://diss.kib.ki.se/1998/91-628-2854-1.

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Книги з теми "Catecholamines"

H, Bönisch, Trendelenburg U. 1922-, and Weiner Norman 1928-, eds. Catecholamines. Berlin: Springer-Verlag, 1988.

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G, Bouloux P. M., ed. Catecholamines. London: Baillière Tindall, 1993.

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Trendelenburg, U., and N. Weiner, eds. Catecholamines II. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73551-6.

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Trendelenburg, U., and N. Weiner, eds. Catecholamines I. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-46625-0.

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A, Arnold M., Trendelenburg U, and Weiner Norman, eds. Catecholamines II. Berlin: Springer-Verlag, 1989.

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K, Ganguly Pallab, ed. Catecholamines and heart disease. Boca Raton, Fla: CRC Press, 1991.

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Nira, Ben-Jonathan, Bahr Janice M, and Weiner Richard I, eds. Catecholamines as hormone regulators. New York: Raven Press, 1985.

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Calif.) International Catecholamine Symposium (10th 2012 Pacific Grove. Catecholamine research in the 21st century: Abstracts and graphical abstracts, 10th International Catecholamine Symposium, 2012. Amsterdam: Elsevier/AP, Academic Press is an imprint of Elsevier, 2013.

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Siraki, Arno Garakhanian. Antioxidant and pro-oxidant nature of catecholamines. Ottawa: National Library of Canada, 2000.

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P, Buckley Joseph, Ferrario Carlos M, and Lokhandwala Mustafa F, eds. Brain peptides and catecholamines in cardiovascular regulation. New York: Raven Press, 1987.

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Частини книг з теми "Catecholamines"

Stanley, Takara. "Catecholamines and Catecholamine Metabolites." In Endocrine Conditions in Pediatrics, 201–3. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-52215-5_34.

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Hoyer, Daniel, Eric P. Zorrilla, Pietro Cottone, Sarah Parylak, Micaela Morelli, Nicola Simola, Nicola Simola, et al. "Catecholamines." In Encyclopedia of Psychopharmacology, 276. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_452.

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Sanzone, Marla. "Catecholamines." In Encyclopedia of Clinical Neuropsychology, 496–502. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_1760.

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Trachte, George J. "Catecholamines." In Encyclopedia of Behavioral Medicine, 400–401. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_237.

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Trachte, George J. "Catecholamines." In Encyclopedia of Behavioral Medicine, 359–60. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_237.

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Sanzone, Marla, and Efrain Gonzalez. "Catecholamines." In Encyclopedia of Clinical Neuropsychology, 1–8. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_1760-2.

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Cosentino, Marco, and Franca Marino. "Catecholamines." In Encyclopedia of Pathology, 1–2. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-28845-1_5125-1.

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Sanzone, Marla, and Efrain Antonio Gonzalez. "Catecholamines." In Encyclopedia of Clinical Neuropsychology, 697–703. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_1760.

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Landsberg, Lewis. "Catecholamines." In Contemporary Endocrinology, 1–14. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-77048-2_1.

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Trachte, George J. "Catecholamines." In Encyclopedia of Behavioral Medicine, 1–2. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4614-6439-6_237-2.

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Тези доповідей конференцій з теми "Catecholamines"

Costa, Susana, M. Sameiro Gonçalves, and Maria José Fernandes. "Release of catecholamines from pyrenylmethyl urethane conjugates by light." In The 13th International Electronic Conference on Synthetic Organic Chemistry. Basel, Switzerland: MDPI, 2009. http://dx.doi.org/10.3390/ecsoc-13-00163.

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Gonzalez, Reniel Suarez, Yousef M. Aljohani, Kennth Kellar, and Ghazaul Dezfuli. "Metformin Potentiates Glutamate-Stimulated Catecholamines Release in Rat Brain." In ASPET 2024 Annual Meeting Abstract. American Society for Pharmacology and Experimental Therapeutics, 2024. http://dx.doi.org/10.1124/jpet.528.991880.

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Morrison, J. EJ, A. B. Latif, C. Mason, P. Bramley, and T. R. Criag. "THE PROFILE OF IN VIVO PLATELET ACTIVATION IN NOCTURNAL ASTHMA." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643495.

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The precise pathophysiology of nocturnal asthma still remains to be elucidated. Activated platelets have the ability to release potent broncho- and vaso-constrictors and therefore, have been implicated in asthma. However, there is no information on the status of in vivo platelet activation in patients with nocturnal asthmaIn a randomised controlled study five healthy volunteers and five asthmatics were investigated during a period of 24h after acclimatisation for one day. Both peak flow rate (PFR) and blood samples were obtained at 4 hourly intervals. Plasma levels of platelet factor 4 (PF4) and beta- thromboglobulin(BTG) were measured by radioimmunoassay and adrenaline (A), noradrenaline (NA) and dopamine (DOP) by radioenzymatic analysis.PFR (1/min) for the 24h period was significantly lower in asthmatics (401±15SEM, P<0.001) than in controls (598+4SEM) with an apparent circadian rhythm peak (442±73, P<0.05 Wilcoxon's test) at 4.00pm only in asthmatics. Although there was no significant differences in either PF4 and BTG (ng/ml) or A, NA and DOP (nmol/1) between asthmatics and controls an apparent circadian rhythm in all of these parameters was demonstrated in both groups. Peak values (mean+SEM) for PF4 (8.9±1.5) and BTG (44.4±3.8) occurred at 8.00am whereas the highest values for the catecholamines (A: 0.36±0.08, NA: 1.75±0.23, DOP: 0.78±0.16) were observed at 4.00pm indicating a lag of 8h between the peaks for catecholamines and the platelet specific proteinsThese initial data demonstrate a clear difference in PFR between asthmatics and controls which is apparently not associated with changes in either PF4 or BTG but which may concur with circadian changes in plasma levels of catecholamines at least in asthmatic patients. Thus, in vivo platelet activation is probably not a contributing factor in nocturnal asthma. Finally, the phase lag between peak plasma levels of platelet proteins and catecholamines requires further investigation

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Yaglova, N. V. "DEVELOPMENT AND FUNCTION OF THE ADRENAL MEDULLA IN EXPOSED TO ENDOCRINE DISRUPTOR DDT ORGANISMS." In NOVEL TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2022. http://dx.doi.org/10.47501/978-5-6044060-2-1.119-123.

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Development and function of the adrenal medulla in exposed to endocrine disruptor DDT organisms was studied. The investigation revealed disrupted by DDT morphogenesis and se-cretion of adrenal medulla resulted in decreased production of catecholamines.

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Chang, Ha Kyun, Hyun Jung Kim, Sung Jong Lee, and Kyun Heo. "PR002/#853 Catecholamines promote ovarian cancer progression through secretion of CXC-chemokines." In IGCS 2023 Annual Meeting Abstracts. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/ijgc-2023-igcs.45.

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Yeung, Edward S., Wei Tong, and Sheri Lillard. "Cell Imaging by Laser-Induced Native Fluorescence Microscopy." In Laser Applications to Chemical and Environmental Analysis. Washington, D.C.: Optica Publishing Group, 1998. http://dx.doi.org/10.1364/lacea.1998.lma.2.

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The high degree of heterogeneity of the nervous and endocrine systems makes it extremely important for real-time monitoring of dynamic chemical changes at the single-cell level to gain a better understanding of the interaction of cells with their environment. Secretion mediated by exocytosis is one of the fundamental phenomena whose mechanism mimics the release of neurotransmitters at synaptic sites. Although the regulation of the secretory pathway has been studied extensively, its molecular mechanism is still not clear. It is important to develop methods that can follow real-time secretory processes with both high temporal and high spatial resolution. The native fluorescence of some proteins and neurotransmitters excited by a deep-UV laser has been shown to be a powerful probe for single-cell analysis. The advantages of direct native fluorescence detection include: (1) no chemical derivatization with fluorescent dyes is needed so no contamination or additional background will be introduced; (2) uncertainties about the efficiencies of the derivatization reaction are eliminated to ensure fast and quantitative response, without influences from slow reaction kinetics or incomplete equilibrium; and (3) the biological integrity of the cells will not be unnecessarily disturbed by having additional reagents or from exposure to artificial environments. We report the coupling of laser-induced native fluorescence detection with capillary electrophoresis (CE) to quantitatively monitor the secretion of insulin, serotonin and catecholamines from single cells. The uptake of serotonin by single living astrocytes was also recorded by native fluorescence imaging microscopy. The catecholamine (mainly epinephrine and norepinephrine) secreting adrenal chromaffin cells have been used as “model nerve terminals” to elucidate the molecular mechanism of neurotransmitter secretion at the nerve terminal. The in vitro dynamics of catecholamine release from bovine adrenal chromaffin cells was monitored with both high spatial and high temporal resolution.

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Silva, Lurdes I. B., Ana C. Freitas, Teresa A. P. Rocha-Santos, M. E. Pereira, and Armando C. Duarte. "Optical fiber biosensor based on enzymatic coating matrix for catecholamines assessment in human urine." In (EWOFS'10) Fourth European Workshop on Optical Fibre Sensors, edited by José Luís Santos, Brian Culshaw, José Miguel López-Higuera, and William N. MacPherson. SPIE, 2010. http://dx.doi.org/10.1117/12.865820.

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Seeley, Eric J., Sophia S. Kim, Michael A. Matthay, and Paul J. Wolters. "Endogenous Catecholamines Impair Innate Immune Responses And Worsen Survival During Septic Peritonitis In Mice." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a3852.

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Kawada, T., M. Inagaki, C. Zheng, M. Li, K. Sunagawa, and M. Sugimachi. "Insignificant Effects of Plasma Catecholamines on Dynamic Heart Rate Regulation by the Cardiac Sympathetic Nerve." In 2005 IEEE Engineering in Medicine and Biology 27th Annual Conference. IEEE, 2005. http://dx.doi.org/10.1109/iembs.2005.1616566.

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Baba, A., T. Mannen, R. Ishigami, Y. Ohdaira, K. Shinbo, K. Kato, F. Kaneko, N. Fukuda, and H. Ushijima. "Detection of Catecholamines with Poly(3-aminobenzylamine) Thin Films using Electrochemical-Surface Plasmon Resonance Spectroscopy." In 2008 International Conference on Solid State Devices and Materials. The Japan Society of Applied Physics, 2008. http://dx.doi.org/10.7567/ssdm.2008.i-6-2.

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Звіти організацій з теми "Catecholamines"

Thomas, Steven A. Catecholamines in Post-Traumatic Stress Disorder. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada585061.

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Abrass, Itamar B., and Christine K. Abrass. Influence of Stress-Induced Catecholamines on Macrophage Phagocytosis. Fort Belvoir, VA: Defense Technical Information Center, April 1989. http://dx.doi.org/10.21236/ada206608.

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Jankowski, Jeffrey A., Timothy J. Schroeder, Ronald W. Holz, and R. M. Wightman. Quantal Secretion of Catecholamines Measured from Individual Bovine Adrenal Medullary Cells Permeabilized with Digitonin. Fort Belvoir, VA: Defense Technical Information Center, May 1992. http://dx.doi.org/10.21236/ada251716.

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Klipp, Robert. Catecholamine Interactions with the Cardiac Ryanodine Receptor. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.1438.

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Wurtman, Richard. Use of Tyrosine or Foods to Amplify Catecholamine Release. Fort Belvoir, VA: Defense Technical Information Center, February 1988. http://dx.doi.org/10.21236/ada195686.

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Schwieger, Alexandra, Kaelee Shrewsbury, and Paul Shaver. Dexmedetomidine vs Fentanyl in Attenuating the Sympathetic Surge During Endotracheal Intubation: A Scoping Review. University of Tennessee Health Science Center, July 2021. http://dx.doi.org/10.21007/con.dnp.2021.0007.

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Purpose/Background Direct laryngoscopy and endotracheal intubation after induction of anesthesia can cause a reflex sympathetic surge of catecholamines caused by airway stimulation. This may cause hypertension, tachycardia, and arrhythmias. This reflex can be detrimental in patients with poor cardiac reserve and can be poorly tolerated and lead to adverse events such as myocardial ischemia. Fentanyl, a potent opioid, with a rapid onset and short duration of action is given during induction to block the sympathetic response. With a rise in the opioid crisis and finding ways to change the practice in medicine to use less opioids, dexmedetomidine, an alpha 2 adrenergic agonist, can decrease the release of norepinephrine, has analgesic properties, and can lower the heart rate. Methods In this scoping review, studies published between 2009 and 2021 that compared fentanyl and dexmedetomidine during general anesthesia induction and endotracheal intubation of surgical patients over the age of 18 were included. Full text, peer-reviewed studies in English were included with no limit on country of study. The outcomes included post-operative reviews of decrease in pain medication usage and hemodynamic stability. Studies that were included focused on hemodynamic variables such as systolic blood pressure, diastolic blood pressure, mean arterial pressure, heart rate, and use of opioids post-surgery. Result Of 2,114 results from our search, 10 articles were selected based on multiple eligibility criteria of age greater than 18, patients undergoing endotracheal intubation after induction of general anesthesia, and required either a dose of dexmedetomidine or fentanyl to be given prior to intubation. Dexmedetomidine was shown to effectively attenuate the sympathetic surge during intubation over fentanyl. Dexmedetomidine showed a greater reduction in heart rate, systolic blood pressure, diastolic blood pressure, mean arterial pressure than fentanyl, causing better hemodynamic stability in patients undergoing elective surgery.Implications for Nursing Practice Findings during this scoping review indicate that dexmedetomidine is a safe and effective alternative to fentanyl during induction of general anesthesia and endotracheal intubation in attenuating the hemodynamic response. It is also a safe choice for opioid-free anesthesia.

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Chen, Guangyao, and Andrew G. Ewing. Multiple Classes of Catecholamine Vesicles Observed during Exocytosis from the Planorbis Cell Body. Fort Belvoir, VA: Defense Technical Information Center, September 1995. http://dx.doi.org/10.21236/ada300360.

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Ghambaryan, Anna. Heart Rate Variability, Catecholamine and Hemodynamic Responses During Rest and Stress in Coronary Artery Disease Patients: The PIMI Study. Fort Belvoir, VA: Defense Technical Information Center, January 2007. http://dx.doi.org/10.21236/ad1013978.

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Palazzolo, D. L., and K. S. Kumar. Effects of S-2-(3-Methylaminopropylamino)ethyl Phosphorothioic Acid (WR- 3689), Alone or Combined with Caffeine, on Catecholamine Content of Mouse Hypothalamus. Fort Belvoir, VA: Defense Technical Information Center, January 1993. http://dx.doi.org/10.21236/ada268505.

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Czerwaty, Katarzyna, Karolina Dżaman, Krystyna Maria Sobczyk, and Katarzyna Irmina Sikrorska. The Overlap Syndrome of Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0077.

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Review question / Objective: To provide the essential findings in the field of overlap syndrome of chronic obstructive pulmonary disease and obstructive sleep apnea, including prevalence, possible predictors, association with clinical outcomes, and severity compared to both chronic obstructive pulmonary disease and obstructive sleep apnea patients. Condition being studied: OSA is characterized by complete cessation (apnea) or significant decrease (hy-popnea) in airflow during sleep and recurrent episodes of upper airway collapse cause it during sleep leading to nocturnal oxyhemoglobin desaturations and arousals from rest. The recurrent arousals which occur in OSA lead to neurocognitive consequences, daytime sleepiness, and reduced quality of life. Because of apneas and hypopneas, patients are experiencing hypoxemia and hypercapnia, which result in increasing levels of catecholamine, oxidative stress, and low-grade inflammation that lead to the appearance of cardio-metabolic consequences of OSA. COPD is a chronic inflammatory lung disease defined by persistent, usually pro-gressive AFL (airflow limitation). Changes in lung mechanics lead to the main clini-cal manifestations of dyspnea, cough, and chronic expectoration. Furthermore, patients with COPD often suffer from anxiety and depression also, the risk of OSA and insomnia is higher than those hospitalized for other reasons. Although COPD is twice as rare as asthma but is the cause of death eight times more often.

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