Добірка наукової літератури з теми "Blindness"

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Статті в журналах з теми "Blindness":

1

Work, Henry H. "On Blindness: Magee on Blindness." Journal of the American Academy of Child & Adolescent Psychiatry 36, no. 4 (April 1997): 572. http://dx.doi.org/10.1097/00004583-199704000-00024.

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2

Kanai, Ryota, Chia-huei Tseng, and Vincent Walsh. "Perceptual blindness and attentional blindness." Neuroscience Research 65 (January 2009): S16. http://dx.doi.org/10.1016/j.neures.2009.09.1572.

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3

Jensen, Melinda S., Richard Yao, Whitney N. Street, and Daniel J. Simons. "Change blindness and inattentional blindness." Wiley Interdisciplinary Reviews: Cognitive Science 2, no. 5 (March 1, 2011): 529–46. http://dx.doi.org/10.1002/wcs.130.

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4

Woiwode, Larry. "Blindness." Antioch Review 44, no. 3 (1986): 276. http://dx.doi.org/10.2307/4611611.

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5

Smalec, Theresa. "Blindness." Theatre Journal 73, no. 4 (2021): 551–53. http://dx.doi.org/10.1353/tj.2021.0108.

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6

Intson, Camille. "Blindness." International Journal of Performance Arts and Digital Media 18, no. 1 (January 2, 2022): 195–96. http://dx.doi.org/10.1080/14794713.2022.2028339.

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7

Saramago, José, and Giovanni Pontiero. "Blindness." Grand Street, no. 64 (1998): 69. http://dx.doi.org/10.2307/25008298.

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8

El-Khayat, Abdul R. "Blindness." InnovAiT: Education and inspiration for general practice 5, no. 3 (January 20, 2012): 147–52. http://dx.doi.org/10.1093/innovait/inr224.

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9

Hoffmann, Stanley, and Michael Mandelbaum. "Blindness." Foreign Affairs 78, no. 6 (1999): 167. http://dx.doi.org/10.2307/20049603.

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10

Elledge, R. "Blindness." BMJ 341, oct20 3 (October 20, 2010): c5680. http://dx.doi.org/10.1136/bmj.c5680.

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Дисертації з теми "Blindness":

1

MacDonald, James. "Load induced blindness." Thesis, University College London (University of London), 2008. http://discovery.ucl.ac.uk/1444318/.

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This thesis has established the effects of perceptual load and working memory load on the conscious awareness of an expected task-unrelated stimulus. Participants performed a visual search task, in which perceptual load was manipulated, while attempting to detect the presence of a meaningless task-unrelated figure, referred to as the critical stimulus (CS). The results showed a consistent reduction in CS detection rate and detection sensitivity (with no accompanying change in response criterion), when the search task was of high perceptual load, compared to a low perceptual load condition. Alternative accounts of the results in terms of memory failure rather than the absence of conscious awareness, the differential search task reaction times in the low and high conditions of perceptual load, goal-neglect, and strategy were ruled out. The effects of perceptual load were generalised to a CS presented directly at fixation, while demonstrating that detection performance was superior for fixated stimuli than for stimuli in peripheral vision, despite size-scaling to account for cortical magnification. Furthermore, the experiments established a dissociation between the effect of perceptual load and the effect of working memory load on conscious awareness, and a second dissociation between the effect of working memory load on awareness and its effect on distractor interference: whereas detection sensitivity and distractor interference were both reduced alike under high perceptual load, working memory load led to increased distractor interference but had no effect on detection sensitivity. Overall, the results generalised perceptual load theory (e.g., Lavie, 1995) to measures of conscious perception, and established a contrast between the effect of working memory load on awareness and on distractibility.
2

Bavelier, Daphne. "Phonological repetition blindness." Thesis, Massachusetts Institute of Technology, 1992. http://hdl.handle.net/1721.1/13206.

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3

Taylor, E. D. "Blindness, education and society." Thesis, Loughborough University, 2000. https://dspace.lboro.ac.uk/2134/7264.

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This thesis looks at social scientific and disability related research on visual impairment and education. It starts from a historical perspective, and outlines the radical change of emphasis in research and thinking brought about by the Disabled People's Movement. After showing how this movement has developed, it looks at various aspects of visual impairment, concentrating on rehabilitation, personality research and the symbolism of the eye. The next section looks at the development of education for visually impaired people. It starts from a historical perspective, and relates this to mainstream Sociological research on classroom interaction and school culture. All of these sections highlight the importance of attitudes and social factors, whilst not denying the undoubted impact of visual impairment in itself. 23 visually impaired school pupils were interviewed, each individually, in a wide range of schools, and from a variety of social and educational backgrounds although an age range of 14 to 18 years seemed most suitable, for various reasons, the ages of those interviewed range from 13 to 19 years. For more detail see Chapter 7. The results highlighted a lack of understanding amongst educational establishment and society at large, especially with regard to partial sight. They showed that generally people can live normal lives, but that it is difficult for them to become fully part of groups which include sighted people, mainly due to problems in sighted people's attitudes and the effects of not seeing who and what is around. Varying levels of confidence were found in both mainstream and special schools, and these often related to the level of encouragement given by staff for the visually impaired people to mix with sighted people. 10 sighted colleagues of the visually impaired respondents at three of the schools were also interviewed, again individually. They were aged 15 and 16. Again, see Chapter 7 for more details. They highlighted issues including looking different, not "knowing how to act" around a visually impaired person, and in some schools, a lack of information about visual impairment. It was these issues, along with the availability of more mainstream information for visually impaired people, (especially "top shelf" material) that concerned respondents the most.
4

Godfrey, Andrew R. "Nietzsche and 'aspect-blindness'." Thesis, Birkbeck (University of London), 2014. http://bbktheses.da.ulcc.ac.uk/67/.

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In this thesis, I draw on Wittgensteinian philosophical thought to critically explore Nietzsche’s diagnosis of (and proposed therapy for) the alleged malaise in modernity. In chapter 1, I argue that Wittgenstein’s concepts of aspect-seeing and certainty provide the resources to resolve several apparently contradictory claims Nietzsche makes about the predicament of his fellow moderns. I show that the condition Wittgenstein calls ‘aspect-blindness’ offers a unified account of the predicament Nietzsche describes. In chapter 2, I explore Nietzsche’s claim that some overarching framework (what Wittgenstein calls a ‘form of life’) has ‘died’. I argue that the pathological attitude of Wittgenstein’s interlocutor towards frameworks provides the most satisfactory model for understanding the nature of the supposed ‘death’ of frameworks in modernity. In chapter 3, I argue that Nietzsche’s proposed remedy for the malaise of modernity is a form of therapy akin to Wittgenstein’s attempt to free his readers from a picture which holds them captive. I show that our understanding of Nietzsche’s therapeutic method will be improved if we bear in mind certain distinctions and ambiguities highlighted by Wittgensteinian discussion of pictures. In chapter 4, I address the concern that Nietzsche appears to characterise modernity in two contradictory ways as both excessively and insufficiently emotional and excessively and insufficiently sceptical. I resolve the appearance of contradiction by showing that (on Nietzsche’s view) the moderns are inherently prone to extreme shifts between poles and that many of the apparent emotions and ideals of the moderns are ‘fake’.
5

Russell, Charlotte Lisa. "Implicit and explicit measures of visual processing within 'inattentional blindness' and 'change blindness' paradigms." Thesis, University College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.270882.

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6

Bates, Kathleen. "A social history of blindness." Thesis, Boston Spa, U.K. : British Library Document Supply Centre, 1998. http://ethos.bl.uk/OrderDetails.do?did=1&uin=uk.bl.ethos.263622.

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7

Arnold, Sarah Ellen. "Blindness and Vision in "Middlemarch"." W&M ScholarWorks, 1989. https://scholarworks.wm.edu/etd/1539625528.

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8

Magnusson, Anna, and Madeleine Thyrsson. "Banner blindness : hur webbanvändare undviker reklambanners." Thesis, University West, Department of Economics and IT, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:hv:diva-1622.

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Reklam på internet är ständigt ökande och många internetanvändare är negativt inställda till den. Därför har de utvecklat olika medvetna och omedvetna strategier för att undvika den när de ska göra något på internet. För skapare av reklam på Internet kan det vara bra att känna till hur reklamen undviks för att kunna bli bättre på att skapa effektiv reklam. Denna undersökning har baserats på intervjuer med vana internetanvändare med olika bakgrund för att få reda på hur de hanterar reklambanners samt en förståelse för hur de tänker. Intervjuerna föregicks av en videoinspelning där försöksdeltagarna fick specifika uppgifter att utföra för att eftersträva ett naturligt beteende.

Resultatet av undersökningen visade att banner blindness är ett fenomen som används av många försöksdeltagare, både på ett medvetet och omedvetet plan. Det visade sig att bara några få gör något aktivt fö ratt slippa eller undvika reklambanners. Anledningarna till varför de undvek reklamen visade sig i många fall bero på tre anledningar som tas upp i Cho och Cheons (2004) teoretiska modell : reklamen stör deras uppgift; många och röriga banners samt tidigare negativa erfarenheter.

9

Keser, Vafa. "Molecular genetic studies of childhood blindness." Thesis, McGill University, 2013. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=117218.

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Childhood blindness due to hereditary retinal disease represents a huge clinical problem. Morbidity, blindness but also a major lack of understanding the molecular genetic events that lead to blindness in very young children hamper clinical management of this important and common problem. One way to get entry into the disease mechanism of childhood retinal blindness is to study Mendelian disease entities that are caused by relatively straightforward links between mutations in genes that lead to aberrant retinal protein function and the resulting disease phenotype. These discoveries in the Mendelian model would lead to understanding of the genetics, proteomics and most importantly retinal disease mechanisms and cycles that underlie the more complex diseases causing blindness. We have assembled a very exciting collection of 21 consanguineous pedigrees with a devastating autosomal recessive Mendelian disease, nonsyndromic congenital retinal nonattachment (NCRNA) and 16 families with autosomal recessive retinitis pigmentosa (arRP). Our main guiding hypothesis is that a significant portion of NCRNA will be caused by mutations in novel retinal genes as currently only one NCRNA gene is known (ATOH7). These novel genes may give us insight into the disease pathways of retinal detachment itself, a huge and clinically unmet medical need. Adult retinal detachments are common and the disease pathways are unknown.In a preliminary study of our cohort, we performed SNP genotyping where we aimed to identify homozygous (hmz) regions. Homozygous regions contain the causal gene(s) in consanguineous families. We found that in many of our patients known retinal detachment or RP gene(s) resided in one of the top 5 largest homozygous segments documented by the SNP array. Direct sequencing of these genes identified the causal mutations in known retinal genes in 11 out of our 21 retinal detachment families and 2 out of our 16 RP families. Thus, 35% of the patients were successfully genotyped in our study with six novel and seven previously reported mutations in known genes. Interestingly, 65% of our cohort of arRP and NCRNA patients remains genetically undetermined, despite our intensive search. After our prescreening for known mutations in NCRNA and RP genes using a combination of conventional genotyping methods, the genetic defects in 10 NCRNA and 14 RP patients remained unexplained suggesting new gene(s) in those patients. Although the clinical diagnosis of all children was NCRNA, in eight families, the molecular diagnosis determined that the disease process was in fact, familial exudative vitreoretinopathy (FEVR), a rare inherited vitreoretinal dystrophy characterized by the disruption of retinal vascular development (FEVR; MIM 133780), caused by pathogenic variation occurred in LRP5, TSPAN12 and NDP genes. However, the phenotype remains the very severe congenital retinal detachment, previously not known to be associated with FEVR mutations. Therefore, we have expanded the phenotypic spectrum of FEVR, a severe retinal detachment phenotype that clinically overlaps with NCRNA. Also, we identified in our Pakistani cohort, the previously identified large deletion (6523 bp del) in ATOH7 found in the Kurdish founder population of Northern Iranian with a high incidence of NCRNA (Ghiasvand et. al, 2011) [5]. We therefore establish for the first time genetic overlap between the Iranian and Pakistani populations.
La cécité infantile causée par des maladies héréditaires de la rétine représente un énorme problème clinique. La morbidité, la cécité mais aussi le manque important de la compréhension des événements moléculaires génétiques conduisant à la cécité chez les très jeunes enfants diminuent la capacité de gérer cliniquement ce problème important et commun. Un moyen de comprendre le mécanisme de la maladie de la cécité infantile est d'étudier les maladies mendéliennes causées par des liens relativement simples entre les mutations génétiques, qui rendent aberrant le fonctionnement des protéines rétiniennes, et le phénotype de la maladie qui en résulte. Ces découvertes dans le modèle mendélien conduiraient à la compréhension des mécanismes des maladies rétiniennes ainsi que d'autres maladies plus complexes provoquant la cécité. Nous avons rassemblé une collection très intéressante de 21 pedigrees consanguins dont les membres possèdent une maladie autosomique récessif aux effets dévastatrices: nonsyndromic congenital retinal nonattachement (NCRNA) ainsi que 16 familles avec rétinite pigmentaire autosomique récessive (arRP). Notre principale hypothèse de base est que une partie importante de NCRNA sera causée par des mutations de nouveaux gènes rétiniennes puisque actuellement un seul gène NCRNA est connu (ATOH7). Ces nouveaux gènes peuvent nous donner des informations sur la mécanisme de la maladie de la détachement rétinienne. Décollement de la rétine chez les adultes sont fréquents et les voies de la maladie sont inconnues. Dans une étude préliminaire de notre cohorte, nous avons effectué de génotypage de SNPs où nous avons cherché à identifier des régions homozygotes (hmz). Les régions homozygotes contiennent le gène responsable dans des familles consanguines. Nous avons constaté que, parmi beaucoup de nos patients, les gènes de la détachement rétinienne ou de RP résidaient dans l'un des 5 plus grands secteurs homozygotes documentés par l'ensemble SNP. Le séquençage direct de ces gènes nous a permis d'identifier les mutations responsables dans les gènes rétiniens connus pour 11 sur nos 21 familles de décollement de la rétine et 2 sur nos 16 familles de RP. Conséquemment, six nouvelles mutations et sept mutations rapportées dans des gènes connus se trouvent dans 35% des patients. Fait intéressant, 65% de notre cohorte de patients arRP et NCRNA demeurent génétiquement indéterminée, en dépit de notre recherche intensive. Après notre présélection pour les mutations connues dans les gènes NCRNA et RP en utilisant une combinaison de méthodes de génotypage conventionnelles, les défauts génétiques dans 10 patients NCRNA et 14 patients RP sont restées inexpliqués suggérant de nouveaux gène(s) chez ces patients. Bien que le diagnostic clinique de tous les enfants était NCRNA, dans huit familles, le diagnostic moléculaire a déterminé que le processus de la maladie était en fait familial exudative vitreoretinopathy FEVR, une rare dystrophie vitréo-rétinienne héréditaire caractérisée par la perturbation du développement vasculaire rétinienne (FEVR; MIM 133780), causé par des variations pathologiques survenues dans les gènes LRP5, TSPAN12 et NDP. Cependant, le phénotype reste d'être des détachements de la rétine congénitale sévères qui n'étaient pas connue pour être associée à des mutations FEVR. Par conséquent, nous avons élargi le spectre phénotypique de la FEVR, un phénotype de détachement rétinien sévère contenant des caractéristiques cliniques similaires à NCRNA. En outre, Nous avons trouvé, dans notre cohorte pakistanaise, la suppression de grande taille (6523 pb del) dans ATOH7 préalablement identifiée parmi la population fondatrice kurde du nord de l'Iran chez qui on retrouve une incidence élevée de NCRNA (Ghiasvand et. al., 2011). Nous avons donc établi, pour la première fois, le chevauchement génétique entre les populations iraniennes et pakistanaises.
10

Ward, Marchella. "Towards a grammar of theatrical blindness." Thesis, University of Oxford, 2018. http://ora.ox.ac.uk/objects/uuid:8376616c-d537-4e2e-93ad-6f06665d252d.

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Since the fifth century, the theatre has been a place for seeing. In spite of this, blind figures repeatedly appear on the stage, from Oedipus, Polymestor, Tiresias and the Cyclops to Shakespeare's Gloucester, Beckett's Hamm, Friel's Molly Sweeney and Kane's Ian. These blind characters have an important role to play in articulating the task of the spectator, both in their aural and imaginative construction of the fictional world in pre-naturalistic theatre, and also in their ability to see through the dramatic illusion in later drama. These scenes of blindness and blinding also have consequences for reception studies, since the relationship between them is not straightforwardly a textual reception history. Instead, these blind characters and the scenes in which they appear are read as what Deleuze and Guattari term an 'assemblage': a heterogenous multiplicity that is produced at the moment of reading / watching with reference to other scenes of blindness and blinding. This thesis sketches out a grammar for such an assemblage, and each chapter focuses on a rule in this grammar. When read as part of an assemblage of blindness, blind characters always have a special relationship with death (Chapter 2), showcase their own performance (Chapter 3), undermine the fictional setting that has been established onstage (Chapter 4), have access to a kind of superhuman knowledge (Chapter 5) and alter the position of their spectators (Chapter 6). Each chapter is structured around a particular moment when the theatre's interest in blind characters resurges, as a response to changes in the social, cultural or scientific understanding of vision and visual impairment. In each chapter, the grammar that is outlined in Chapter 1 with reference to ancient plays returns to the fore, but is refracted through the historical period back on to the grammar of the assemblage.

Книги з теми "Blindness":

1

Landau, Elaine. Blindness. New York: Twenty-First Century Books, 1994.

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2

Marcovitz, Hal. Blindness. Detroit: Lucent Books, 2009.

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3

Saramago, José. Blindness. Boston: Houghton Mifflin Harcourt, 2011.

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4

Saramago, José. Blindness. London, UK: Vintage Classics, 2017.

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5

Saramago, José. Blindness. Boston: Houghton Mifflin Harcourt, 2011.

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6

Henry, Green. Blindness. [Normal, IL]: Dalkey Archive Press, 2001.

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7

Saramago, José. Blindness. New York: Harcourt Brace & Company, 1998.

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8

Saramago, José. Blindness. Thorndike, Me: Thorndike Press, 1999.

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9

Saramago, José. Blindness. San Diego: Harcourt Brace & Co., 1999.

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10

Ferraro, Estella. Wilful Blindness. Wiesbaden: Springer Fachmedien Wiesbaden, 2021. http://dx.doi.org/10.1007/978-3-658-32654-8.

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Частини книг з теми "Blindness":

1

Friedman, Lawrence M., Curt D. Furberg, and David L. DeMets. "Blindness." In Fundamentals of Clinical Trials, 119–32. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-1586-3_7.

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2

Firth, Ashley. "Blindness." In Practical Web Inclusion and Accessibility, 17–53. Berkeley, CA: Apress, 2019. http://dx.doi.org/10.1007/978-1-4842-5452-3_2.

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3

Friedman, Lawrence M., Curt D. Furberg, and David L. DeMets. "Blindness." In Fundamentals of Clinical Trials, 82–93. New York, NY: Springer New York, 1998. http://dx.doi.org/10.1007/978-1-4757-2915-3_6.

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4

Galloway, N. R. "Blindness." In Common Eye Diseases and their Management, 267–71. London: Springer London, 1985. http://dx.doi.org/10.1007/978-1-4471-3521-0_24.

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5

Galloway, Nicholas Robert, and Winfried Mawutor Kwaku Amoaku. "Blindness." In Common Eye Diseases and their Management, 183–85. London: Springer London, 1999. http://dx.doi.org/10.1007/978-1-4471-3625-5_25.

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6

Welch, Therese R. "Blindness." In Encyclopedia of Autism Spectrum Disorders, 1–5. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4614-6435-8_18-3.

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7

Welch, Therese R. "Blindness." In Encyclopedia of Autism Spectrum Disorders, 1–5. New York, NY: Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4614-6435-8_18-4.

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8

Grote-Garcia, Stephanie A. "Blindness." In Encyclopedia of Child Behavior and Development, 263–64. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-79061-9_373.

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9

Galloway, Nicholas R., Winfried M. K. Amoaku, Peter H. Galloway, and Andrew C. Browning. "Blindness." In Common Eye Diseases and their Management, 255–58. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32869-0_25.

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10

Galloway, N. R., and S. A. Vernon. "Blindness." In Ophthalmology, 108–10. London: Springer London, 1988. http://dx.doi.org/10.1007/978-1-4471-1624-0_5.

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Тези доповідей конференцій з теми "Blindness":

1

Dalton, Nicholas S., Emily Collins, and Paul Marshall. "Display Blindness?" In CHI '15: CHI Conference on Human Factors in Computing Systems. New York, NY, USA: ACM, 2015. http://dx.doi.org/10.1145/2702123.2702150.

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2

Kang, Ruogu, Aimee Kane, and Sara Kiesler. "Teammate inaccuracy blindness." In CSCW'14: Computer Supported Cooperative Work. New York, NY, USA: ACM, 2014. http://dx.doi.org/10.1145/2531602.2531681.

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3

Lichev, Lachezar. "BLINDNESS, ANALYSIS AND DIAGNOSTIC." In SGEM2011 11th International Multidisciplinary Scientific GeoConference and EXPO. Stef92 Technology, 2011. http://dx.doi.org/10.5593/sgem2011/s10.102.

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4

Wood, Chip, Rob Gray, Jennifer Young, John Summers, Kari Torkkola, and Noel Massey. "Inattentional Blindness While Driving." In Driving Assessment Conference. Iowa City, Iowa: University of Iowa, 2005. http://dx.doi.org/10.17077/drivingassessment.1119.

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5

Jurja, Sanda. "EYES WIDE OPEN ON BLINDNESS." In 2nd International Multidisciplinary Scientific Conference on Social Sciences and Arts SGEM2015. Stef92 Technology, 2015. http://dx.doi.org/10.5593/sgemsocial2015/b11/s2.068.

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6

Allain, M. "Promoting blindness in structured illumination microscopy." In Mathematics in Imaging. Washington, D.C.: OSA, 2017. http://dx.doi.org/10.1364/math.2017.mm3c.1.

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7

Berger, Nils. "Overcoming Operational Blindness in Software Architecture." In WCX SAE World Congress Experience. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 2020. http://dx.doi.org/10.4271/2020-01-1336.

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8

Krogmeier, Claudia, Justin Heffron, Justin Legare, Michael Nelson, Ziyi Liu, and Christos Mousas. "Blindness Visualizer: A Simulated Navigation Experience." In 2020 IEEE Conference on Virtual Reality and 3D User Interfaces Abstracts and Workshops (VRW). IEEE, 2020. http://dx.doi.org/10.1109/vrw50115.2020.00107.

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9

Bygari, Ramya, Rachita Naik, and Uday Kumar P. "Blindness (Diabetic Retinopathy) Severity Scale Detection." In 2021 8th Swiss Conference on Data Science (SDS). IEEE, 2021. http://dx.doi.org/10.1109/sds51136.2021.00009.

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10

Lees, Monica N., JonDavid Sparks, John D. Lee, and Matthew Rizzo. "Change Blindness, Attention, and Driving Performance." In Driving Assessment Conference. Iowa City, Iowa: University of Iowa, 2007. http://dx.doi.org/10.17077/drivingassessment.1211.

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Звіти організацій з теми "Blindness":

1

Henderson, John M., and Andrew Hollingworth. Global Transsaccadic Change Blindness During Scene Perception. Fort Belvoir, VA: Defense Technical Information Center, September 2003. http://dx.doi.org/10.21236/ada422684.

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2

Gelernter, Judith. Spot Rare Occurrences More Frequently by Lessening Inattentional Blindness. Fort Belvoir, VA: Defense Technical Information Center, April 2013. http://dx.doi.org/10.21236/ada601393.

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3

Amberson, Joshua. Staring Into the Sun: Essays on Vision and Blindness. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.6341.

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4

Russas, Michael Sr E. Correcting Blindness In the Nerve Center: How To Improve Situational Awareness. Fort Belvoir, VA: Defense Technical Information Center, December 2015. http://dx.doi.org/10.21236/ad1009217.

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5

Cutler, Kelly. Making Race Matter: Interrupting Racial Color-blindness in Education Through the Implementation of Anti-racist Curriculum. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.7368.

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6

Pirohov, Vladyslav M., Anna M. Horlo, and Iryna S. Mintii. Software development of the algorithm of adaptating of the website design for people with color-blindness. [б. в.], December 2018. http://dx.doi.org/10.31812/123456789/2888.

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The purpose of the study is the program implementation of the basic algorithms of the website design adaptation for people with color perception. This article examines the implementation of the following algorithms: the conversion algorithm from the rgb color model to the lms model (there is a special algorithm precisely in the color model lms for modeling various types of color perception violations), simulation of different color blindness types in the lms-model (this is the transformation of normal values in the color model lms to values with different types of color perception violations), convert data from lms color model to rgb model, conversion from color rgb to hsl model, color filtering in hsl-model and conversion from hsl model to rgb color model.
7

Li, Bin, Fei Wen, Hongru Chen, and Ri-Li Ge. A meta analysis of the prevalence rate of diabetic retinopathy. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0112.

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Review question / Objective: P: diabetic retinopathy; I:—; C:—; O: prevalecne rate; S:cross-sectional study. Objective:To analyze the epidemiological characteristics of diabetic retinopathy and to provide scientific basis for its prevention and control. Condition being studied: Diabetic retinopathy is one of the common microvascular complications in patients with diabetes mellitus, which ultimately seriously affects the vision of patients. It is the leading cause of blindness among young and middle-aged workers worldwide. It is one of the main causes of binocular blindness in elderly patients in western countries.Because of the high incidence, wide range, complex pathogenesis, serious consequences and poor treatment effect of DM and its DR,many countries have actively carried out epidemiological research on the population of DM patients in order to understand the incidence, distribution and related risk factors of DR, and to provide scientific basis for the formulation of targeted public prevention and control measures.
8

Corrado, Giovanni. Tactical Enthusiasm and Operational Blindness: Civilian Casualties during the Allied Air Campaign in Italy in 1940-1945. Fort Belvoir, VA: Defense Technical Information Center, April 2015. http://dx.doi.org/10.21236/ad1001269.

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9

Swobodzinski, Martin, Amy Parker, Elizabeth Schaller, and Denise Snow. Pedestrian Wayfinding Under Consideration of Visual Impairment, Blindness, and Deafblindness: A Mixed-Method Investigation Into Individual Experiences and Supporting Elements. Transportation Research and Education Center (TREC), 2022. http://dx.doi.org/10.15760/trec.282.

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10

Harvey, Paul, and Habiba Mohamed. The Politics of Donor and Government Approaches to Social Protection and Humanitarian Policies for Assistance During Crises. Institute of Development Studies (IDS), March 2022. http://dx.doi.org/10.19088/basic.2022.010.

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This paper examines social protection policy processes in fragile and conflict-affected settings (FCAS). It explores what the policies of donor governments, aid agencies, and crisis-affected governments reveal about the politics of assistance during crises, and how aid agencies are navigating tensions between humanitarian and development approaches to social assistance. It finds that social protection policies are prone to conflict blindness. Commitments to state-building often ignore dilemmas inherent in supporting states that are parties to ongoing conflicts and the political rather than technical challenges involved. Government social protection policies in FCAS often make little mention of the fact that war or conflict are taking place.

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