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Статті в журналах з теми "BAFF"
Krejsek, Jan, Martina Koláčková, Irena Lindrová, Radovan Slezák, and Ctirad Andrýs. "Increase of Intracellular BAFF in B Cells of Sjögren’s Patients Is Not Affected by Decrease of BAFFR." Acta Medica (Hradec Kralove, Czech Republic) 58, no. 1 (2015): 25–31. http://dx.doi.org/10.14712/18059694.2015.88.
Повний текст джерелаSaidoune, F., N. Charles, J. Chezel, B. Escoubet, T. Papo, A. Nicoletti, and K. Sacre. "AB0140 BAFF NEUTRALIZATION HAS JANUS-FACED EFFECT ON ATHEROSCLEROSIS ASSOCIATED WITH SYSTEMIC LUPUS ERYTHEMATOSUS." Annals of the Rheumatic Diseases 79, Suppl 1 (June 2020): 1370.3–1370. http://dx.doi.org/10.1136/annrheumdis-2020-eular.988.
Повний текст джерелаAlexaki, Vassilia-Ismini, Vassiliki Pelekanou, George Notas, Maria Venihaki, Marilena Kampa, Valérie Dessirier, Sanaa Sabour-Alaoui, Efstathios N. Stathopoulos, Andreas Tsapis, and Elias Castanas. "B-Cell Maturation Antigen (BCMA) Activation Exerts Specific Proinflammatory Effects in Normal Human Keratinocytes and Is Preferentially Expressed in Inflammatory Skin Pathologies." Endocrinology 153, no. 2 (February 1, 2012): 739–49. http://dx.doi.org/10.1210/en.2011-1504.
Повний текст джерелаXu, Haifeng C., Jun Huang, Vishal Khairnar, Vikas Duhan, Aleksandra A. Pandyra, Melanie Grusdat, Prashant Shinde, et al. "Deficiency of the B Cell-Activating Factor Receptor Results in Limited CD169+Macrophage Function during Viral Infection." Journal of Virology 89, no. 9 (February 11, 2015): 4748–59. http://dx.doi.org/10.1128/jvi.02976-14.
Повний текст джерелаPrieto-Peña, D., S. Remuzgo Martinez, F. Genre, V. Pulito-Cueto, B. Atienza-Mateo, B. Sevilla, J. Llorca, et al. "POS0113 BAFF-APRIL-BAFFR PATHWAY ON THE PATHOGENESIS OF IMMUNOGLOBULIN-A VASCULITIS." Annals of the Rheumatic Diseases 80, Suppl 1 (May 19, 2021): 267.2–268. http://dx.doi.org/10.1136/annrheumdis-2021-eular.707.
Повний текст джерелаBossen, Claudia, Teresa G. Cachero, Aubry Tardivel, Karine Ingold, Laure Willen, Max Dobles, Martin L. Scott, et al. "TACI, unlike BAFF-R, is solely activated by oligomeric BAFF and APRIL to support survival of activated B cells and plasmablasts." Blood 111, no. 3 (February 1, 2008): 1004–12. http://dx.doi.org/10.1182/blood-2007-09-110874.
Повний текст джерелаSecreto, Frank J., Michelle K. Manske, Tammy Price-Troska, Steven C. Ziesmer, Stephen M. Ansell, James R. Cerhan, and Anne J. Novak. "A Lymphoma-Associated Mutation in BAFF-R Drives Constitutive PI3K Signaling and Increased Expression of Pro-Survival Genes." Blood 118, no. 21 (November 18, 2011): 2642. http://dx.doi.org/10.1182/blood.v118.21.2642.2642.
Повний текст джерелаHildebrand, Joanne M., Zhenghua Luo, Michelle Manske, Steven Ziesmer, Tammy Price-troska, Wai Lin, Bruce Hostager, et al. "A BAFF-R Mutation Associated with Non-Hodgkin Lymphoma Exhibits Altered TRAF Binding and Reveals New Insights Into Proximal BAFF-R Signaling." Blood 116, no. 21 (November 19, 2010): 468. http://dx.doi.org/10.1182/blood.v116.21.468.468.
Повний текст джерелаTada, S., T. Yasui, T. Okuno, Y. Nakatsuji, H. Mochizuki, S. Sakoda, and H. Kikutani. "BAFF controls neural cell survival through BAFF receptor." Journal of the Neurological Sciences 333 (October 2013): e689. http://dx.doi.org/10.1016/j.jns.2013.07.2380.
Повний текст джерелаTada, Satoru, Teruhito Yasui, Yuji Nakatsuji, Tatsusada Okuno, Toru Koda, Hideki Mochizuki, Saburo Sakoda, and Hitoshi Kikutani. "BAFF Controls Neural Cell Survival through BAFF Receptor." PLoS ONE 8, no. 7 (July 29, 2013): e70924. http://dx.doi.org/10.1371/journal.pone.0070924.
Повний текст джерелаДисертації з теми "BAFF"
Tun, Paul Fei-Tun. "BAFF, B cells and tumour immunity." Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.534210.
Повний текст джерелаSutherland, Andrew Peter Robert St Vincents Clinical School UNSW. "BAFF regulation of peripheral T cell responses." Awarded by:University of New South Wales. St Vincents Clinical School, 2005. http://handle.unsw.edu.au/1959.4/22788.
Повний текст джерелаBitoun, Samuel. "Mise au point de modèles animaux pour étudier la physiopathologie de la polyarthrite rhumatoïde et le rôle du méthotrexate dans la tolérisation." Thesis, Université Paris-Saclay (ComUE), 2018. http://www.theses.fr/2018SACLS174.
Повний текст джерелаTitle : Development of new animal models to study the pathophysiology of RA and the role of methotrexate-induced tolerance.Keywords : Rheumatoid arthritis, shared epitope, ACPA, immunogenicity, methotrexate, TNF inhibitorsAbstract: Rheumatoid arthritis (RA) is an autoimmune disease (AID) where antibodies directed against citrullinated peptides (ACPA) are highly specific for the diagnosis. One of the key treatments of RA is methotrexate. It has an action on both the disease and reinforces the effect of second line TNF inhibitors (TNFi). MTX might act via prevention of anti-drug antibodies (ADAb) directed against TNFi that are implicated in loss of efficacy of TNFi. We have developed a macaque model to recapitulate the human disease by immunization with citrullinated peptides in the context of a genetic factor favoring RA: the shared epitope on the HLA. Immunization of macaques with citrullinated peptides and intra-articular boost cause an anti-citrulline T and B cell response and a chronic monoarthritis.The role of MTX-induced tolerance against TNFI has been studied in autoimmune BAFF transgenic (tg) mice using MTX just before treatment with TNFi we were able to prevent ADAb formation in BAFFtg mice and not wild type mice or macaques. We identified that BAFFtg mice expressed elevated CD73 leading to more adenosine and regulatory B cells as actors in MTX-induced tolerance. This MTX-BAFF interaction was further confirmed in humans in the ABIRISK cohort where MTX was more efficient to prevent ADAb formation in RA patients with elevated BAFF levels.Setting up two new animal models allows better understanding of RA pathophysiology and better use of biologics that extend to other domains of medicine
Sellam, Jérémie. "Etude de nouveaux acteurs impliqués dans la physiopathologie du syndrome de Sjögren primaire : Id3, BAFF, BAFF-R et les microparticules circulantes." Paris 11, 2009. http://www.theses.fr/2009PA11T012.
Повний текст джерелаGroom, Joanna Ruth School of Medicine UNSW. "Loss of immune regulatory checkpoints in BAFF transgenic mice." Awarded by:University of New South Wales. School of Medicine, 2006. http://handle.unsw.edu.au/1959.4/27281.
Повний текст джерелаMurphy, Deirdre. "Investigating the BAFF/APRIL cytokine system in atherosclerosis pathology." Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.709281.
Повний текст джерелаLahiri, Ayan. "The complexity of the BAFF forms and their functional implications." Thesis, Brest, 2014. http://www.theses.fr/2014BRES0006/document.
Повний текст джерелаElevated expression of ‘B cell activating factor’ (BAFF), a potent B cell survival factor contributes to the expansion of low-affinity self-reactive B cells during the establishment of tolerance. However, mechanisms leading to BAFF over-expression in autoimmune diseases are not understood. We reported the discovery of a new variant for BAFF, 4BAFF in humans (in which exon 4 is excised) or 5BAFF in mice (in which exon 5 is excised), which acts as a transcription factor of the full-length form of BAFF, and which is preferentially found in cells isolated from patients with autoimmune diseases. When transfected in human B cells, D4BAFF upregulates a large number of genes associated with immune response and especially innate immunity and regulation of apoptotis. Furthermore D4BAFF acts, in association with p50 from the NF- B pathway, as a transcription factor for its own parent gene. Another important finding is that 4BAFF is an important component of the efficacy of regulatory B cell activity. Our work introduces an entirely novel concept in biology suggesting that a human cytokine gene can be transcriptionally regulated by the activity of one of its own splice variants.We have also tried to understand the complexity of the various forms of BAFF. We observed that epithelial cells expressed BAFF-receptor (BR3) and produce BAFF suggesting autocrine properties. Blocking BR3 results in nuclear translocation of PKC promoting epithelial cell apoptosis.Furthermore, only some forms of BAFF are required for epithelial cell survival. Finally, we studied the consequences of the expression of TLR9 on the B cell surface and demonstrated that TLR9 acts as a co-receptor of the B cell receptor to influence B cell fate independently of CpG binding. We show that CpG activation of B cells, acting synergistically with BCR signals, was inhibited by anti-TLR9 stimulation. Induction of CD25 expression and proliferation of B cells were thus down-regulated by engagement of cell surface TLR9. Overall, our results indicate that TLR9 expressed on B cell plasma membrane might be a negative regulator of endosomal TLR9, and could provide a novel control by which activation of autoreactive B cells is restrained. All these findings contribute to a better understanding on immunopathology of autoimmune diseases with potential applications in therapy
Fountain, Kathryn. "The role of the BAFF receptor in B cell survival." Thesis, University College London (University of London), 2017. http://discovery.ucl.ac.uk/10040551/.
Повний текст джерелаSaidoune, Fanny. "Rôle de BAFF dans l'athérome accéléré associé au lupus systémique." Thesis, Université de Paris (2019-....), 2020. http://www.theses.fr/2020UNIP7011.
Повний текст джерелаAccelerated atherosclerosis is now recognized as the main cause of death in SLE patients. Of note, the increased prevalence of premature atherosclerosis in SLE is not fully explained by traditional cardiovascular risk factors. Although we and others showed that lupus immunopathogenesis may promote accelerated atherosclerosis, the mechanisms by which SLE pathogenesis might contribute to accelerate atherosclerosis remains elusive.B cells, by their ability to produce pathogenic autoantibodies, play a causative role in SLE. Consistently, there are strong evidence to connect B-cell activating factor (BAFF), a cytokine required for maturation and survival of B2 B cells, to lupus. Belimumab (GSK), a monoclonal antibody specifically directed against BAFF, is the only approved biotherapy for SLE treatment. BAFF is involved in atherosclerosis and atherosclerosis-prone mice in which hematopoietic cells did not express the receptor for the pro-B2 BAFF cytokine displayed a significant reduction in atherosclerotic lesion development. My thesis project consists of studying the contribution of BAFF in accelerated atherosclerosis associated with SLE.We first showed that ApoE-/-D227K mice were an appropriate model for the study of lupus associated atherosclerosis, when compared to the ApoE-/- pristane treated mice.We then demonstrated that, in the ApoE-/-D227K model, the neutralization of BAFF by using an antibody specifically targeting this cytokine, efficiently treated lupus by inducing the depletion of mature B cells in the secondary lymphoid organs. On the other hand, the atheroprotective effect of the BAFF neutralization, despite the depletion of B cells in the aortic tertiary lymphoid organs, was apparent in mice with low cholesterol levels, only. For high cholesterol levels, the neutralization of BAFF tended to aggravate atherosclerotic lesions by promoting the foam cell transformation of macrophages that expressed TACI.We then asked whether the observations made in our mouse model were relevant to human atherosclerosis associated with SLE and observed a positive correlation between BAFF serum levels, peripheral expansion of B lymphocytes and progression of subclinical atherosclerosis in lupus patients at apparent low risk for cardiovascular events. The effect of Belimumab treatment on atherosclerosis was also analyzed ex vivo and in vitro in a small number of patients and we showed that BAFF neutralization in lupus could enhance or dampen the development of atherosclerosis according to the body mass index (BMI).In conclusion, our work shows 1- that B cells and BAFF play a pathogenic role in atherosclerosis associated with lupus and 2-that BAFF can be atheroprotective or atheroaggravative depending on the mechanisms involved. In absence of classical cardiovascular risk factors (such as dysplipidemia or overweight), BAFF neutralizing therapy, by disrupting the BAFF/BAFFR interaction on B cells, depletes mature B cells in tertiary lymphoid organs and is atheroprotective. In the presence of classical cardiovascular risk factors, BAFF neutralizing therapy, by disrupting the BAFF/TACI interaction on macrophages, promotes the foam cell transformation of macrophages and aggravates atherosclerosis
Chhabra, Manu. "Targeting the BAFF/April signalling axis for prevention of humoral rejection." Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708468.
Повний текст джерелаКниги з теми "BAFF"
Sateren, Shelley Swanson. Banff. New York: Crestwood House, 1989.
Знайти повний текст джерелаBarf. Lāhaur: Sang-i Mīl Pablīkeshanz, 2010.
Знайти повний текст джерелаJamīlah, Kadīvar, ed. Barf. Tihrān: Umīd-i Īrāniyān, 2004.
Знайти повний текст джерелаBaffo, Giorgio. giorgio baffo poesie Baffo osceno. Roma: Stampa alternativa, 2001.
Знайти повний текст джерелаAkbar, Akbar Ḥusain. Barf barf cingārī: Shiʻrī majmūʻah. Kolkata: Raʾīsah Akbar va Munavvar Ḥusain, 2003.
Знайти повний текст джерелаHashmi, Saira. Siah baif. Lahore: India-e-Naqush, 1986.
Знайти повний текст джерелаThakkar, Anil. Garm barf. Delhi: Modern Books, 1990.
Знайти повний текст джерелаAhmad, Razia F. Kālī barf. Lāhaur: Maqbūl Ikāḍamī, 1992.
Знайти повний текст джерелаKālī barf. Lāhaur: Maqbūl Ikaiḍamī, 1992.
Знайти повний текст джерелаKrzyżanowski, Jerzy Ryszard. Banff: Powieść. Paryż: Instytut Literacki, 1988.
Знайти повний текст джерелаЧастини книг з теми "BAFF"
Bittenbinder, Elise, Jenny Baron, and Silvia Schriefers. "Eine gewagte Kooperation: BAMF und BAfF." In Grenzbereiche der Supervision - Verwaltung in Bewegung, 65–77. Göttingen: Vandenhoeck & Ruprecht, 2015. http://dx.doi.org/10.13109/9783666403699.65.
Повний текст джерелаMyles, Arpita, Jean L. Scholz, and Michael P. Cancro. "BAFF/BLyS Family." In Encyclopedia of Signaling Molecules, 523–31. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101556.
Повний текст джерелаWarnatz, Klaus. "Baff-Receptor Deficiency." In Encyclopedia of Medical Immunology, 1–2. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-9209-2_29-1.
Повний текст джерелаSmulski, Cristian R., Patricia Odermatt, and Hermann Eibel. "BAFF Receptor Deficiency." In Humoral Primary Immunodeficiencies, 131–47. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91785-6_11.
Повний текст джерелаMyles, Arpita, Jean L. Scholz, and Michael P. Cancro. "BAFF/BLyS Family." In Encyclopedia of Signaling Molecules, 1–10. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101556-1.
Повний текст джерелаWarnatz, Klaus. "Baff-Receptor Deficiency." In Encyclopedia of Medical Immunology, 47–49. New York, NY: Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4614-8678-7_29.
Повний текст джерелаDavidson, Anne. "BAFF and APRIL and Their Receptors." In Encyclopedia of Medical Immunology, 181–87. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-0-387-84828-0_563.
Повний текст джерелаMackay, Fabienne, William A. Figgett, Pali Verma, and Xavier Mariette. "The BAFF/APRIL System in Autoimmunity." In BLyS Ligands and Receptors, 125–60. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-013-7_7.
Повний текст джерелаHildebrand, Joanne M., Ping Xie, and Gail A. Bishop. "Signal Transduction by Receptors for BAFF and APRIL." In BLyS Ligands and Receptors, 93–114. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-013-7_5.
Повний текст джерелаRyan, Maureen C., and Iqbal S. Grewal. "Targeting of BAFF and APRIL for Autoimmunity and Oncology." In Advances in Experimental Medicine and Biology, 52–63. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-89520-8_4.
Повний текст джерелаТези доповідей конференцій з теми "BAFF"
Al-Fatlawi, Noor Alhuda Ghanem, Arshad N. G. Al-Dujaili, and Talib Hussein Nooruldeen Kammona. "Assessment B-cell-activating factor (BAFF) in thrombocytopenia patients." In INTERNATIONAL CONFERENCE OF NUMERICAL ANALYSIS AND APPLIED MATHEMATICS ICNAAM 2019. AIP Publishing, 2020. http://dx.doi.org/10.1063/5.0027533.
Повний текст джерелаSamuel, Du, Jacobs Holly M, Arkatkar Tanvi, Scharping Nicole, Rawlings David J, and Jackson Shaun W. "AI-16 Integration of BCR, TLR, BAFF receptor and TACI signals promotes autoantibody production by transitional B cells in BAFF transgenic mice." In LUPUS 21ST CENTURY 2018 CONFERENCE, Abstracts of the Fourth Biannual Scientific Meeting of the North and South American and Caribbean Lupus Community, Armonk, New York, USA, September 13 – 15, 2018. Lupus Foundation of America, 2018. http://dx.doi.org/10.1136/lupus-2018-lsm.16.
Повний текст джерелаJha, Saket, Jagdeep Singh, Ranjana Minz, Aadhar Dhooria, Gsrsnk Naidu, Rajiv Kumar, Manish Rathi, Sanjay Jain, Shashi Anand, and Aman Sharma. "FRI0280 BAFF AND APRIL GENE EXPRESSION IN PATIENTS WITH ANCA ASSOCIATED VASCULITIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.2217.
Повний текст джерелаHsu, H., M. Zhang, K. Miner, G. Kevin, J. Whoriskey, R. Jacobsen, C. Chen, and K. Ishida. "FRI0306 Development of baff and icosl bispecific inhibitor amg 570 for sle treatment." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.5767.
Повний текст джерелаFiggett, W., A. Baldolli, F. Vincent, and F. Mackay. "36 Identification of disease-associated gut microbiota in lupus-prone baff-tg mice." In LUPUS 2017 & ACA 2017, (12th International Congress on SLE &, 7th Asian Congress on Autoimmunity). Lupus Foundation of America, 2017. http://dx.doi.org/10.1136/lupus-2017-000215.36.
Повний текст джерелаPolverino, F., S. Baraldo, E. Bazzan, G. Turato, S. Agostini, E. Balestro, M. Damin, et al. "B Cell Activating Factor (BAFF) Is Expressed by Lymphoid Follicles of COPD Patients." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2951.
Повний текст джерелаCampos, J., T. Slocombe, S. Nayar, V. Iannizzotto, DH Gardner, CD Buckley, A. Haynes, RB Henderson, and F. Barone. "P061 Targeting B-cell activating factor (BAFF) impairs ectopic lymphoneogenesis in murine models of sjÖgren’s syndrome." In 38th European Workshop for Rheumatology Research, 22–24 February 2018, Geneva, Switzerland. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-ewrr2018.80.
Повний текст джерелаAlfano, Gianvincenzo, Sergio Greco, Francesco Parisi, and Irina Trubitsyna. "Defining the Semantics of Abstract Argumentation Frameworks through Logic Programs and Partial Stable Models (Extended Abstract)." In Thirtieth International Joint Conference on Artificial Intelligence {IJCAI-21}. California: International Joint Conferences on Artificial Intelligence Organization, 2021. http://dx.doi.org/10.24963/ijcai.2021/641.
Повний текст джерелаFlessa, Christina Maria, Adrianos Nezos, Evangelia Zampeli, Haralampos M. Moutsopoulos, and Clio Mavragani. "AB0181 ASSOCIATION BETWEEN SINGLE NUCLEOTIDE POLYMORPHISMS (SNPS) OF THE BAFF GENE AND FATIGUE IN PRIMARY SJöGREN’S SYNDROME." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.7024.
Повний текст джерелаParodis, I., F. Söder, F. Faustini, Z. Kasza, F. Wermeling, and I. Gunnarsson. "PS7:134 Rituximab-mediated late-onset neutropenia in systemic lupus erythematosus – distinct roles of baff and april." In 11th European Lupus Meeting, Düsseldorf, Germany, 21–24 March 2018, Abstract presentations. Lupus Foundation of America, 2018. http://dx.doi.org/10.1136/lupus-2018-abstract.177.
Повний текст джерелаЗвіти організацій з теми "BAFF"
Dutra, Jose. OERDifferentiation Presentation April Banff Canana. NPT Educacional, March 2015. http://dx.doi.org/10.17699/ind.2015.2.
Повний текст джерелаCouture, R., and S. G. Evans. Five-Mile Creek debris flow, Banff National Park, near Banff, Alberta, August 4, 1999. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2000. http://dx.doi.org/10.4095/211547.
Повний текст джерелаYeo, W. B. When Banff was somewhere else down the track. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1985. http://dx.doi.org/10.4095/298182.
Повний текст джерелаJordan, Tyler. Development of an ultra-fast BaF2-based detector. Office of Scientific and Technical Information (OSTI), November 2020. http://dx.doi.org/10.2172/1726114.
Повний текст джерелаCarter, J. L. Lower carboniferous brachiopods from the Banff formation of western Alberta. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1987. http://dx.doi.org/10.4095/122462.
Повний текст джерелаLourenço-Leitzke, Milene Rocha, and Marlene Marchiori. Comunicação de Risco: um descompasso entre discurso e prática no caso Shell/Basf / Risk Communication: Mismatch between speech and practice in Shell/Basf case. Revista Internacional de Relaciones Públicas, December 2016. http://dx.doi.org/10.5783/rirp-12-2016-07-107-124.
Повний текст джерелаMedioli, B. E., and M. N. Demuth. Reconnaissance sub-bottom profiling studies of Peyto Lake, Banff National Park. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2009. http://dx.doi.org/10.4095/247448.
Повний текст джерелаMedioli, B. E., and M. N. Demuth. Sub-bottom profiling results from Peyto Lake, Banff National Park, Alberta. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2009. http://dx.doi.org/10.4095/247419.
Повний текст джерелаGardner C., L. Ahrens, J. W. Glenn, J. A. Kozak, and J. F. Ryan. Booster Fault Study for the BAF Penetration Site. Office of Scientific and Technical Information (OSTI), January 2000. http://dx.doi.org/10.2172/1132453.
Повний текст джерелаJordan, Tyler Alexander. Development of an ultra-fast BaF₂-based detector. Office of Scientific and Technical Information (OSTI), April 2020. http://dx.doi.org/10.2172/1615640.
Повний текст джерела