Добірка наукової літератури з теми "Associated gene"

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Статті в журналах з теми "Associated gene"

1

Gough, N. R. "Gene-Associated AMPK." Science Signaling 3, no. 138 (September 7, 2010): ec272-ec272. http://dx.doi.org/10.1126/scisignal.3138ec272.

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2

Leka, Sofia, Sofia Kitsiou-Tzeli, Ariadni Kalpini-Mavrou, and Emmanuel Kanavakis. "Short stature and dysmorphology associated with defects in the SHOX gene." HORMONES 5, no. 2 (April 15, 2006): 107–18. http://dx.doi.org/10.14310/horm.2002.11174.

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3

Divers, Jasmin, Nicholette D. Palmer, Lingyi Lu, Carl D. Langefeld, Michael V. Rocco, Pamela J. Hicks, Mariana Murea, Lijun Ma, Donald W. Bowden, and Barry I. Freedman. "Gene–gene interactions in APOL1-associated nephropathy." Nephrology Dialysis Transplantation 29, no. 3 (October 24, 2013): 587–94. http://dx.doi.org/10.1093/ndt/gft423.

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Amundsen, S. S., M. K. Viken, L. M. Sollid, and B. A. Lie. "Coeliac disease-associated polymorphisms influence thymic gene expression." Genes & Immunity 15, no. 6 (May 29, 2014): 355–60. http://dx.doi.org/10.1038/gene.2014.26.

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Zhao, N. N., S. Lin, Z. Q. Wang, and T. J. Zhang. "VLDLR gene polymorphism associated with abdominal fat in Gaoyou domestic duck breed." Czech Journal of Animal Science 60, No. 4 (July 15, 2016): 178–84. http://dx.doi.org/10.17221/8132-cjas.

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Sapir-Koren, Rony, Gregory Livshits, Tania Landsman, Kobyliansky, and Eugene. "Bone Mineral Density is Associated with Estrogen Receptor Gene Polymorphism in Men." Anthropologischer Anzeiger 59, no. 4 (January 18, 2002): 343–53. http://dx.doi.org/10.1127/anthranz/59/2002/343.

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7

Freedman, Barry I., and Karl Skorecki. "Gene–Gene and Gene–Environment Interactions in Apolipoprotein L1 Gene-Associated Nephropathy." Clinical Journal of the American Society of Nephrology 9, no. 11 (June 5, 2014): 2006–13. http://dx.doi.org/10.2215/cjn.01330214.

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Yacubian, J., T. Sommer, K. Schroeder, J. Glascher, R. Kalisch, B. Leuenberger, D. F. Braus, and C. Buchel. "Gene gene interaction associated with neural reward sensitivity." Proceedings of the National Academy of Sciences 104, no. 19 (May 2, 2007): 8125–30. http://dx.doi.org/10.1073/pnas.0702029104.

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9

Bhatnager, Richa, Ranjeet Kaur, and Amita Suneja Dang Twinkle Dahiya. "Computational prediction of damage associated non synonymous SNPs of CYP17A1 and CYP19A1 gene." International Journal of Trend in Scientific Research and Development Volume-1, Issue-6 (October 31, 2017): 635–46. http://dx.doi.org/10.31142/ijtsrd3581.

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Thuong, N. T. T., T. R. Hawn, T. T. H. Chau, N. D. Bang, N. T. B. Yen, G. E. Thwaites, Y. Y. Teo, et al. "Epiregulin (EREG) variation is associated with susceptibility to tuberculosis." Genes & Immunity 13, no. 3 (December 15, 2011): 275–81. http://dx.doi.org/10.1038/gene.2011.83.

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Дисертації з теми "Associated gene"

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Hofstra, Robert Martinus Wouter. "The RET gene and its associated diseases." [S.l. : [Groningen] : s.n.] ; [University Library Groningen] [Host], 1995. http://irs.ub.rug.nl/ppn/142201383.

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Jones, Richard Julian. "Novel gene therapy for EBV-associated malignancies." Thesis, University of Birmingham, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.274412.

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James, Pamela. "Characterization of Novel Lymphoid-Associated Genes Identified by Gene-Trapping: a Dissertation." eScholarship@UMMS, 2006. http://escholarship.umassmed.edu/gsbs_diss/231.

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Анотація:
The discovery of novel genes involved in hematopoietic development and lymphoid function is necessary for the understanding of these systems. To this end, we utilized transmembrane protein-specific gene trapping in embryonic stem (ES) cells, a method of forward genetics, to identify a novel, complex locus from which several splice variants arise. The trapped locus identified in the KST30 ES cell clone encodes several genes including outer membrane protein 25 (OMP25) and activin receptor interacting protein (ARIP2) and two novel genes, AK74 and AK88. AK74 is highly conserved between human and mouse with 85% identity at the amino acid level. The human homolog was cloned from CD34+ cord blood hematopoietic stem cell progenitors (HSCPs) implying that it may have a role in the hematopoietic system. We generated mice from the gene trapped ES cells, called KST30 mice, to analyze the expression pattern of transcripts from the trapped locus in the hematopoietic system. Utilizing the gene trap LacZ reporter and RT-PCR, we found that AK88 and AK74 are expressed in hematopoietic stem cells and thymocytes and that AK88 and ARIP2 are dramatically up-regulated in activated Band T lymphocytes. In addition, we found restricted expression of the gene trap in most non-lymphoid tissues. Interestingly, the expression pattern of the gene trap coincides with the expression of activin signaling components in many cell types including thymocytes, activated B cells, hematopoietic stem cells and the ductal cells of the pancreas. AK74, AK88 and ARIP2 share two exons that encode a 44 amino acid region. ARIP2 negatively regulates activin signaling through endocytosis of Activin type II receptors. The N-terminal PDZ domain associates with ActRII and mediates endocytosis via association with RalBP1. The region of ARIP2 that associates with RalBP1 encompasses the 44 amino acid region also found in AK74 and AK88, suggesting that these proteins may also associate with RalBP1, perhaps sequestering it from ARIP2. This possibility combined with the similarities between gene trap expression and expression of the components of activin signaling indicates a role of the trapped genes in activin signaling. AK74 and AK88 have a signal sequence and transmembrane domain that are predicted to direct them to mitochondria. To confirm this prediction, we examined the subcellular localization of AK74 and found that it localizes to a punctate, perinuclear structure identified as mitochondria using a mitochondria specific dye. AK74 was not seen in the cytoplasm, nucleus or at the plasma membrane of cells. To determine the function of these novel genes, AK74 was retrovirally over-expressed in a double positive thymoma cell line and examined the global expression profile using Affymetrix gene chip. AK74 changed the expression levels of 36 genes greater than 3-fold compared to vector alone. Of these genes, several are involved in cytoskeletal rearrangement, apoptosis or are regulated by calcium signaling. Using yeast two-hybrid, several candidate binding partners for AK74 were identified, one of which is the receptor for activated protein kinase C (RACK1). RACK1 was also identified as a potential binding partner for AK88. RACK1 is a WD40 domain-containing scaffolding protein that has been implicated in many pathways but most prominently in the protein kinase C signaling pathway. Association with RACK1 by either AK74 or AK88 suggests that they may be involved in RACK1 function. Both RACK1 and PKC are involved with Ca2+ signaling through different mechanisms. This, combined with global gene expression changes in AK74 over-expressing cells suggests a role for AK74, AK88 or ARIP2 in Ca2+ signaling. When we examined the expression of the trapped genes in mice homozygous for the gene-trapped allele (KST30tr/tr) we found that insertion of the gene trap caused a severe decrease in AK88 and ARIP2 but not AK74 transcripts. Analysis of KST30tr/tr mice showed no abnormalities in conventional lymphoid populations and precursors, however, intraepithelial lymphocyte (IEL) populations were altered by the loss of AK88 and/or ARIP2. There was an approximate 2-fold decrease CD8αα+ T cells in the small intestine while CD8αβ+ T cells were largely unaltered. Using gene trap technology, we have identified two novel, mitochondria-localized proteins. The cumulative findings described in this thesis, including the homology between AK74, AK88 and ARIP2, their expression pattern and the phenotype of KST30tr/tr mice, suggest possible roles of AK74 and AK88 in diverse pathways.
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Read, Tara. "Elucidating a novel gene associated with myoclonus dystonia." Thesis, University of Ottawa (Canada), 2009. http://hdl.handle.net/10393/28248.

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Myoclonus Dystonia (MD) is an autosomal dominant disease with high but incomplete penetrance and is characterized by both involuntary myoclonic jerks and dystonic posturing. Our group has found that mutations within the epsilon sarcoglycan (SGCE) gene on chromosome 7q21 are associated with MD in 30-40% of affected individuals in 31 families studied, supporting the basis for genetic heterogeneity. Novel mutations have been found in SGCE by screening these families for point mutations and large deletions and duplications through the use of sequencing, high performance liquid chromatography (HPLC) and multi˙ligation probe amplification (MLPA) analysis. A 10cM genome wide linkage analysis of a large Canadian family provided significant LOD scores for microsatellite markers within the 18p11 region, now designated as the DYT 15 locus. Further haplotype analysis has narrowed a non-recombinant region associated with the disease phenotype to a 3.18 Mb region in this locus. Since the current understanding of Myoclonus Dystonia is poor, it is difficult to predict genes that could be responsible for MD. Sarcoglycans are essential constituents of the dystrophin-glycoprotein complex and are involved in linking the extracellular lamanin matrix to the actin filaments within the cytoplasm; therefore focus is given to the examination of potentially related structural genes that are expressed in the brain. By analyzing such candidate genes in a panel of affected individuals, we believe that a novel gene will be elucidated and provide insight into the mechanism of Myoclonus Dystonia.
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Gordon, Linda Anne. "Investigation of potential problems associated with gene therapy." Thesis, University of Leicester, 1998. http://hdl.handle.net/2381/30742.

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Safety requirements for gene therapy currently focus on preventing acute patient toxicity caused by the introduction of exogenous DNA. This project aimed to investigate the possibility that more subtle changes in cell regulation may occur as a result of transfection or transgene expression. Mouse B16.F1 melanoma and CMT93 colorectal cell lines transfected with a plasmid (pNASS) or retroviral (pBabeNeo) construct were used as a model. Expression of the transgene was driven by a 2540bp (plasmid) or 769bp (retroviral) fragment of the mouse tyrosinase promoter. Results have shown that transfection per se can cause small changes in the proliferation rate of B16.F1 and CMT93 cells, but transgene expression did not produce this effect. However, expression of an IL-2 transgene in B16.F1 cells did decrease the rate of substrate attachment via an intracellular mechanics. These data suggest that cell regulation can be disrupted by transfection and transgene expression. Despite these changes, DDRT-PCR analysis of approximately 25% of the mRNA population indicated few changes in gene expression between parental and transfected cells. Loss of transgene expression has proved an obstacle to success in clinical trials and was observed in both cell lines. In B16.F1 cells containing pNASS/IL-2, levels of IL-2 protein were reduced by more than 90% after 15 weeks in culture and this was mirrored by a decrease in IL-2 mRNA levels. The transgene was intact in high passage cells and restriction digest analysis did not indicate changes in methylation status. The continued presence of the construct was confirmed by treatment with 10-6 M melanocyte stimulating hormone, which induced IL-2 trnasgene mRNA and protein expression in cells that had lost expression from pNASS/IL-2. Induction was not observed in cells with the shorter promoter fragment in pBabeNeo/IL-2, indicating that the mechanism of downregulation can be overridden if the promoter contains suitable response elements.
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6

Behrens, Renee F. "Changes in endometrial gene expression associated with Infertility." Thesis, Imperial College London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.511836.

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Sawhney, Namita. "Studies of the growth arrest associated gene - prohibitin." Thesis, King's College London (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.261713.

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8

Glod, Frank. "PCR generated gene probes for cloning fungal polykeptide synthase genes associated with squalestatin biosynthesis." Thesis, University of Bristol, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268525.

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9

John, Christopher Robert. "Gene expression associated with the evolution of C₄ photosynthesis." Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.709401.

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10

Childs, Andrew James. "Tex19 : a germ cell-specific gene associated with pluripotency." Thesis, University of Edinburgh, 2007. http://hdl.handle.net/1842/29064.

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Germ cells are the only population of cells in the adult organism shown to retain pluripotency – the ability to differentiate into all the different germ layers of the embryo. This property is shared with embryonic stem cells and tumour-derived embryonal carcinoma cells, and the molecular mechanisms underpinning pluripotency are likely to be similar in all three systems. The core circuitry of transcription factors required to establish and maintain pluripotency is relatively well characterised. However, many of these important transcription factors also regulate a large number of genes involved in processes other than transcription, some of which may also play an important role in maintaining stem cell plasticity. Understanding the entire molecular basis of pluripotency will improve the use of ES cells as a model system for differentiation and development, enhance the therapeutic potential of stem cells and provide insights into the mechanisms of tumourigenesis. Testis Expressed Gene 19 has been identified in screens for genes expressed in spermatogonia, embryonic stem cells, and for targets of translational regulation by the germ cell-specific protein Dazl. The work presented here characterises the expression pattern of Tex19 in the gonads and stem cells, establishing a correlation between the expression of Tex19 and pluripotent potentiality. A human homologue of Tex19 is found to be expressed in stem cells and cancer, and the evolution of the gene family in mammals is investigated. Finally, the function of Tex19 and its relationship with Dazl is also investigated.
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Книги з теми "Associated gene"

1

Jones, Richard Julian. Novel gene therapy for EBV-associated malignancies. Birmingham: University of Birmingham, 2003.

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2

Berns, Kenneth I., and Catherine Giraud, eds. Adeno-Associated Virus (AAV) Vectors in Gene Therapy. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-80207-2.

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3

Millar, Anna L. Frequency estimation of endometrial cancer associated with microsatellite instability and mismatch repair gene defects. Ottawa: National Library of Canada, 1999.

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4

Musk, P. Molecular events associated with the activation of a temperature sensitive p160 v-abl gene product. Manchester: UMIST, 1993.

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5

Small, Cherrie-Lee. The analysis of gene transcripts associated with conidiation in the insect pathogenic fungus, Metarhizium anisopliae. St. Catharines, Ont: Brock University, Dept. of Biological Sciences, 2004.

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6

Murray, Paul G. Epstein-Barr virus gene expression in the pathogenesis of Hodgkin's Disease and other EBV-associated diseases. Wolverhampton: University of Wolverhampton, 1996.

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7

Lai, Joe Heng. A polymorphic locus in the promoter region of the IGFBP3 gene is associated with mammographic breast density. Ottawa: National Library of Canada, 2003.

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8

Kellough, Gene Ross. The ancestry of Gene Ross: Including the Ross line and associated lines of Bennett, Grimes, Llewellyn, Knighton, Turnbull and Kyle. Seneca, S.C. (600 Old Salem Rd., Seneca 29678): G.R. Kellough, 1986.

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9

United, States Dept of Energy Assistant Secretary for Environment Safety and Health. Human radiation experiments associated with the U.S. Department of Energy and its predecessors. Washington, DC: U.S. Dept. of Energy, 1995.

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10

Flavin, Nora. Cloning and characterisation of the bovine activin receptor type II gene (ActRII): Its localisation to chromosome 2 (BTA2) by somatic cell genetic analysis and the genotyping of an associated microsatelltie UCD2. Dublin: University College Dublin, 1996.

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Частини книг з теми "Associated gene"

1

Miller, Daniel G. "AAV-Mediated Gene Targeting." In Adeno-Associated Virus, 301–15. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-370-7_13.

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2

Dillon, Lawrence S. "Nucleic Acid-Associated Protein Genes." In The Gene, 243–319. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4899-2007-2_5.

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3

Podoltsev, N., E. Podoltseva, and E. Morozova. "Multiple Myeloma Associated with Amyloidosis." In Gene Technology, 455–60. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-61122-3_35.

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4

Sands, Mark S. "AAV-Mediated Liver-Directed Gene Therapy." In Adeno-Associated Virus, 141–57. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-370-7_6.

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5

Hallek, M., C. M. Wendtner, R. Kotin, D. Michl, and E. L. Winnacker. "Recombinant Adeno-Associated Virus (r AAV) Vectors." In Gene Therapy, 73–91. Basel: Birkhäuser Basel, 1999. http://dx.doi.org/10.1007/978-3-0348-7011-5_6.

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6

Vogel, S. N., and M. J. Fultz. "Lps Gene-Associated Functions." In Genetics of Immunological Diseases, 165–70. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-50059-6_24.

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Martino, Ashley T., Roland W. Herzog, Ignacio Anegon, and Oumeya Adjali. "Measuring Immune Responses to Recombinant AAV Gene Transfer." In Adeno-Associated Virus, 259–72. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-370-7_11.

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Danos, Olivier. "Adeno-Associated Viral Vectors: Principles and in vivo Use." In Gene Therapy, 103–10. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-72160-1_11.

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Samulski, Richard Jude. "Adeno-associated Viral Vectors." In Viruses in Human Gene Therapy, 53–76. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0555-2_3.

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10

Smith, Richard H., Linda Yang, and Robert M. Kotin. "Chromatography-Based Purification of Adeno-Associated Virus." In Gene Therapy Protocols, 37–54. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-248-3_4.

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Тези доповідей конференцій з теми "Associated gene"

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Liu, Zhenqiu. "Detecting Disease Associated Genes and Gene-Gene Interactions with Penalized AUC Maximization." In 2008 Seventh International Conference on Machine Learning and Applications. IEEE, 2008. http://dx.doi.org/10.1109/icmla.2008.145.

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2

Pabst, S., TF Wienker, MM Noethen, and C. Grohe. "CD4 Gene Polymorphisms Are Associated with Sarcoidosis." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5411.

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Nersisyan, Lilit, Anna Hakobyan, and Arsen Arakelyan. "Telomere-associated gene network in lung adenocarcinoma." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.oa3493.

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Xu, Wenjing, Juan Li, Jianqiang Li, Yue Li, Weiliang Qiu, Ji-Jiang Yang, Chunjie Guo, and Minhua Lu. "Gene Network Modules Associated to DPSCs Differentiation." In 2017 IEEE 41st Annual Computer Software and Applications Conference (COMPSAC). IEEE, 2017. http://dx.doi.org/10.1109/compsac.2017.136.

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Wu, Mengmeng, Wanwen Zeng, Wenqiang Liu, Yijia Zhang, Ting Chen, and Rui Jiang. "Integrating embeddings of multiple gene networks to prioritize complex disease-associated genes." In 2017 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2017. http://dx.doi.org/10.1109/bibm.2017.8217651.

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Deng, Chao, Cui-Xiang Lin, and Hong-Dong Li. "Improving the Prediction of Disease-associated Genes by Integrating Annotated Gene Sets." In 2021 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2021. http://dx.doi.org/10.1109/bibm52615.2021.9669702.

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Tieppo, Eduardo Macedo de Souza, Miriam Eva Koch, and Alzira Alves de Siqueira Carvalho. "A deletion in CFL-2 gene associated with Severe Nemaline Myopathy with peculiar features." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.362.

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Context: Nemaline myopathy (NM) is the most common congenital myopathy characterized by muscle weakness and presence of nemaline bodies (rods) in muscle biopsy. Phenotype ranges from neonatal death to normal lifespan. 13 genes have been reported. We describe a new variant in cofilin 2 gene (CFL2;OMIM*601443). Case report: A 5-year-old boy born severely hypotonic and unable to breathe, in need of mechanic ventilation. Healthy non-consanguineous parents. Physical examination: severe hypotonia with only extraocular motricity preserved and multiple contractures. Dysmorphic features were observed as brachycephaly, hypertelorism, pseudohypertrophy, macroglossia, premature pubic hair. Deep reflexes were absent. CK: 1010U/l. DHEA-S elevated. Muscular biopsy: Rods, cores and dystrophic pattern. Exome: homozygous deletion in exons 1 to 4 of CFL2 and partial deletion of the next gene Sorting nexin-6 (SNX6) in Chr14:34.563.122-34.714.639. Conclusion: 9 cases were described previously: Age onset was before 31 months. 4 presented respiratory distress at birth, 1 presented macroglossia, 2 contractures, 2 spinal deformities and 3 delayed motor milestones. Our patient presents an extent deletion in homozygosis not described before. A second deletion was found in SNX6, which is involved in protein trafficking and is expressed in different cells, as endocrine and cardiac. The early puberty and dimorphisms could be due to SNX6, though there is no previous disease caused by this gene. Among differential diagnosis of macroglossia, congenital myopathy caused by CFL2 should be considered.
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K. Biwott, Felix, Chengsi Luo, Xiaoqin Lv, Han Zhang, and Nini Rao. "Identification of Gene Modules and Key Genes Associated with Atrial Fibrillation Based on Gene Co-expression Network Analysis." In ICBBE '21: 2021 8th International Conference on Biomedical and Bioinformatics Engineering. New York, NY, USA: ACM, 2021. http://dx.doi.org/10.1145/3502871.3502879.

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9

Stacey, Minviluz. "Utility of CRISPR/Cas in accelerating gene discovery in soybean." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/rzne1660.

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The use of CRISPR/Cas9 has been successfully applied in various plant species to induce targeted genome editing, including soybean. Soybean is recalcitrant to transformation and thus, plants with stable CRISPR gene edits are costly and take a long time to produce. Moreover, soybean is allotetraploid and editing paralogous genes are often necessary to obtain observable phenotype(s). For each gene target, we designed two gRNAs to increase editing efficiency and allow rapid genotyping by PCR. We also tested the CRISPR reagents in transient hairy root transformation to determine if the Cas9 and gRNAs would perform properly in transgenic soybean plants. Our results showed that we can indeed obtain highly efficient, cost-effective CRISPR/Cas editing in soybean to generate novel genotypes for gene discovery and downstream field propagation and breeding efforts. Examples of CRISPR-edited genes and their associated seed traits will be discussed.
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10

Zhou, Xiangdong, Keith C. C. Chan, and Danhong Zhu. "A multi-stage approach to detect gene-gene interactions associated with multiple correlated phenotypes." In 2017 IEEE Conference on Computational Intelligence in Bioinformatics and Computational Biology (CIBCB). IEEE, 2017. http://dx.doi.org/10.1109/cibcb.2017.8058563.

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Звіти організацій з теми "Associated gene"

1

Lamont, Susan J., Eyal Koren, Avigdor Cahaner, E. Dan Heller, and Jacob Pitcovski. Gene Expression Associated With Virus Resistance in Chickens. Ames (Iowa): Iowa State University, January 2006. http://dx.doi.org/10.31274/ans_air-180814-825.

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2

Tricoli, James V. CLAR1, A Novel Gene Associated with Prostate Tumor Progression. Fort Belvoir, VA: Defense Technical Information Center, March 2001. http://dx.doi.org/10.21236/ada395761.

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3

Sandford, Erin, Ceren Ciraci, Behnam Abasht, Jack C. M. Dekkers, and Susan J. Lamont. SNPs in Region of NF-Kappa-B Gene Associated with Expression of Immune-Related Genes. Ames (Iowa): Iowa State University, January 2010. http://dx.doi.org/10.31274/ans_air-180814-95.

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4

Sanders, Michael M. Regulation of the Multidrug Resistance-Associated Protein Gene by Estrogen. Fort Belvoir, VA: Defense Technical Information Center, March 2001. http://dx.doi.org/10.21236/ada410077.

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5

Chinnaiyan, Arul M. Discovery of Novel Gene Elements Associated with Prostate Cancer Progression. Fort Belvoir, VA: Defense Technical Information Center, December 2014. http://dx.doi.org/10.21236/ada612909.

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6

Collier, Jackie, L. A Novel, Photosynthesis-Associated Thioredoxin-Like Gene: Final Technical Report. Office of Scientific and Technical Information (OSTI), September 2005. http://dx.doi.org/10.2172/850272.

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7

Chinnaiyan, Arul M. Discovery of Novel Gene Elements Associated with Prostate Cancer Progression. Fort Belvoir, VA: Defense Technical Information Center, October 2012. http://dx.doi.org/10.21236/ada568345.

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8

Sanders, Michel. Regulation of the Multidrug Resistance-Associated Protein Gene by Estrogen. Fort Belvoir, VA: Defense Technical Information Center, October 1999. http://dx.doi.org/10.21236/ada381694.

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9

ZHENG, Jiansheng, and Tang ZHU. Polymorphism of fucosyltransferase 3 (FUT3) gene is associated with inflammatory bowel disease (IBD) — a systematic review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0001.

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Review question / Objective: Polymorphism of fucosyltransferase 3 (FUT3) gene is associated with inflammatory bowel disease (IBD) — a systematic review and Meta-analysis. Condition being studied: This study collected relevant literatures and analyzed the relationship between the polymorphism of the FUT3 genes at rs3745635, rs3894326, and rs28362459 to the IBD with Meta-analysis, in order to further explore the possible mechanism of the polymorphism of the FUT3 gene and IBD.
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10

Hasenstein, Jason, and Susan J. Lamont. Chicken Gallinacin Gene Cluster Associated with Salmonella Colonization in Two Advanced Intercross Lines. Ames (Iowa): Iowa State University, January 2007. http://dx.doi.org/10.31274/ans_air-180814-152.

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