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Статті в журналах з теми "ASM dysfunction"
Khan, Shah Dupesh, and N. Pandiyan. "Ejaculatory Dysfunction—A Mini Review." Advances in Sexual Medicine 05, no. 02 (2015): 39–42. http://dx.doi.org/10.4236/asm.2015.52005.
Повний текст джерелаEl-Tahlawi, Samar, Noha Ezzat Mohammad, Asmaa Younes Elsary, Noha Mohamed Yousef, and Talal Abdelreheem. "Female Sexual Dysfunction in Elfayoum Governorate." Advances in Sexual Medicine 08, no. 01 (2018): 1–13. http://dx.doi.org/10.4236/asm.2018.81001.
Повний текст джерелаJesudason, E. C., N. P. Smith, M. G. Connell, D. G. Spiller, M. R. H. White, D. G. Fernig, and P. D. Losty. "Peristalsis of airway smooth muscle is developmentally regulated and uncoupled from hypoplastic lung growth." American Journal of Physiology-Lung Cellular and Molecular Physiology 291, no. 4 (October 2006): L559—L565. http://dx.doi.org/10.1152/ajplung.00498.2005.
Повний текст джерелаAlidu, Huseini, W. K. B. A. Owiredu, Nafiu Amidu, Peter Paul Mwinsanga Dapare, Ahmed Tijani Bawah, Christian Kofi Gyasi-Sarpong, and Christian Obirikorang. "The Metabolic Syndrome and Sexual Dysfunction in a State of Inflammation." Advances in Sexual Medicine 07, no. 02 (2017): 82–96. http://dx.doi.org/10.4236/asm.2017.72006.
Повний текст джерелаDumbraveanu, Ion, Iurie Arian, Andrei Munteanu, Daniela Catereniuc, and Adrian Tanase. "Erectile Dysfunction in Male with Glans Penis Apocrine Hydrocystoma: Organic or Psychogenic?" Advances in Sexual Medicine 07, no. 03 (2017): 131–38. http://dx.doi.org/10.4236/asm.2017.73010.
Повний текст джерелаEyada, Moustafa M. K., Alaa-Aldin S. Abd-Elhamid, Riham A. F. Elboghdady, Ahmed M. Gadallah, and Mohamed Azab. "Assessment of Female Sexual Dysfunction in Patients with Premenopausal Female Pattern Hair Loss." Advances in Sexual Medicine 10, no. 03 (2020): 86–103. http://dx.doi.org/10.4236/asm.2020.103006.
Повний текст джерелаKeeler, Allison M., Donghai Liu, Marina Zieger, Lang Xiong, Jeffrey Salemi, Karl Bellvé, Barry J. Byrne, David D. Fuller, Ronghua ZhuGe, and Mai K. ElMallah. "Airway smooth muscle dysfunction in Pompe (Gaa−/−) mice." American Journal of Physiology-Lung Cellular and Molecular Physiology 312, no. 6 (June 1, 2017): L873—L881. http://dx.doi.org/10.1152/ajplung.00568.2016.
Повний текст джерелаRodríguez-Sureda, Víctor, Francesca Crovetto, Stefania Triunfo, Olga Sánchez, Fátima Crispi, Elisa Llurba, Eduard Gratacós, Francesc Figueras, and Carmen Domínguez. "Increased secretory sphingomyelinase activity in the first trimester of pregnancy in women later developing preeclampsia: a nested case-control study." Biological Chemistry 397, no. 3 (March 1, 2016): 269–79. http://dx.doi.org/10.1515/hsz-2015-0266.
Повний текст джерелаNkumu, Matthieu Marc Loposso, Christian Kane Kapinga, Mafuta Tsita Alpha, Dieu Donné Moningo Molamba, Nkodila Aliosha, Augustin Punga Maole Monga-Lembe, Jean Paul Esika Mokumo, Diangienda Nkutima Pablo, Mujinga Lukusa Elisabeth, and Dirk De Ridder. "A Cross-Sectional Study According to Risk Factors Associated with Erectile Dysfunction in Men." Advances in Sexual Medicine 10, no. 03 (2020): 104–18. http://dx.doi.org/10.4236/asm.2020.103007.
Повний текст джерелаBao, Jun-Xiang, Min Xia, Justin L. Poklis, Wei-Qing Han, Christopher Brimson, and Pin-Lan Li. "Triggering role of acid sphingomyelinase in endothelial lysosome-membrane fusion and dysfunction in coronary arteries." American Journal of Physiology-Heart and Circulatory Physiology 298, no. 3 (March 2010): H992—H1002. http://dx.doi.org/10.1152/ajpheart.00958.2009.
Повний текст джерелаДисертації з теми "ASM dysfunction"
Burnett, Hollie. "Executive dysfunction in high functioning autism." Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/25825.
Повний текст джерелаDavid, Dylan Naitraj. "Inflammation-Induced HSPC Dysfunction: Towards a Better Understanding of the Role of MAVS, ASC, and Caspase-1 in HSPC Dysfunction and Bone Marrow Failure." University of Cincinnati / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1626356668978688.
Повний текст джерелаSarn, Nicholas Brian. "MICROGLIA PATHOLOGY: AN INHERENT FEATURE OF CONSTITUTIONAL PTEN DYSFUNCTION." Case Western Reserve University School of Graduate Studies / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1619526347351812.
Повний текст джерелаPitzer, Ashley. "Contribution of ASC-Inflammasome to Vascular Endothelial Dysfunction: Role of RNA Receptor RIG-I." VCU Scholars Compass, 2017. http://scholarscompass.vcu.edu/etd/5050.
Повний текст джерелаHsin-Chin, Chen, and 陳信誌. "Clinical Assessment of Hand Writing , Shoulder/Elbow Motor Control Function for Upper Motor Neuron Dysfunction Patients and Development of a Rehabilitation Robotic Arm for Upper Extremity Motor Learning." Thesis, 2002. http://ndltd.ncl.edu.tw/handle/69320938591967511117.
Повний текст джерела長庚大學
機械工程研究所
90
Patients with writer’s cramp or upper motorneuron disease (ex. Parkinson’s disease, stroke, and other brain injuries) often have difficulty in processing upper arm movement because of involuntary muscle contraction or neuron regression. This often results in trembling of the involved extremity. Most patients , still experience palm/arm movement obstacles even after treatment/surgery. Therefore, clinical evaluation and rehabilitation training for writing with a pen and arm movement are important parts of the postoperative management.. In this research , clinical testing and quantitative evaluation of the writing function of the patients was done. A series of robotic manipulators for arm movement assessment and rehabilitation was designed and its applications were tested quantitatively and diagrammatically. In the first part of this research , an evaluation system was developed which aimed to quantitatively measure the patients’ arm function using four parameters : the contact pressure between paper and pen, the time delay before start of writing, the hesitation time spent between words, and the total written words in a given time. An evaluation standard for quantitative writing was set-up, clinical testing and subsequent statistical analysis of the results was done. A total of 26 patients and 13 control subjects were studied. The results were analyzed using the SPSS statistical software and showed that t test values of the four estimation parameters were all less than 0.05. This means that there was a significant difference between the evaluation parameters of the patients and normal control subjects. These results can provide a reference for clinicians when diagnosing patients. The second part of the research was aimed to decrease the time and energy spent by therapists who usually have to repeatedly say or demonstrate the correct motions for arm rehabilitation. A mechanical device was designed to manipulate the arm movement and provide the patient with a continuous passive rehabilitation. The machine used was a SCARA type robotic manipulator with four degrees of freedom. This was connected to a computer with a software based on Windows 2000 and Visual Basic 6.0. Also designed for this project to adjust the rehabilitation parameters (i.e. velocity, circle/ellipse movement locus size, etc.). This set up enables the doctor or therapist to choose the proper rehabilitation parameters and patterns for patients who will undergo a continuous upper arm continuous passive rehabilitation. The third part of the research was aimed to consolidate the mechanical 3-D Digitizer already existing in the market and the interface software we developed into a manipulator for the evaluation of upper arm movement function in order to improve the defects noted during initial rehabilitation. This would also provide diagrammatic (arm movement locus radial error, distribution diagram) and quantitative (radial error of normal value, radial error of absolute value, average drawing circle rate, etc.) evaluation indicators and clinical test standards. This consolidated robotic manipulator was further tested on 11 stroke paralysis patients, 5 normal elders, and 6 normal youths. The results showed that there was a significant difference between the three test groups. From the quantitative evaluation indicators, it is shown that the radial error of absolute value was 2 to 3 times more among the stroke patients compared to that of the normal elders, and 3 to 4 times more compared to that of the normal youths. From the diagrammatic evaluation indicators, we found that in the arm movement locus radial error distribution diagram, stroke paralysis patient had the widest area of radial error of absolute value compared to the rest. The error value was about 4 to 9 times more than the normal elder group and 9 to 16 times more than the normal youth group. This objectively result shows that the stroke patients had more serious functional defects. These results can provide a reference for clinicians in the diagnosis of functional defects of patients. The hand writing function evaluation system for using a pen and the manipulator for the assessment of arm function will be able to provide assistance to the clinician as new tools to evaluate patients’ palm/arm motor function. The robotic manipulator can be used for the postoperative rehabilitation the training of the patient. The standards and quantitative evaluation indicators developed in this research can be used as new tool and methods of the treatment for upper motorneuron or palm/arm movement dysfunction patients.
Rahn, Ingmar. "Neurohumorale Aktivierung in einem kardiovaskulären Risikokollektiv - Einfluss von diastolischer oder systolischer Dysfunktion." Doctoral thesis, 2011. http://hdl.handle.net/11858/00-1735-0000-0006-B173-D.
Повний текст джерелаLaranjo, Mariana Barradas Serrano. "The role of GPRASP2 on mGluR5 signalling." Master's thesis, 2018. http://hdl.handle.net/10316/86125.
Повний текст джерелаA perturbação do espectro do autismo (PEA) inclui um grupo heterogéneo de condições de neurodesenvolvimento, caracterizadas por défices na linguagem, alterações na interação social e na comunicação e por comportamentos repetitivos e estereotipados. Estudo epidemiológicos estimam que mais de 1% da população mundial tem uma PEA e que, 70% dos indivíduos com PEA também apresentam défice intelectual (DI).Há duas teorias para explicar a patofisiologia das PEA: uma destas teorias considera que é a perda da homeostasia neuronal, devido à disfunção sináptica e ao desequilíbrio entre a excitação e inibição que desencadeia as comorbidades características das PEAs. A outra teoria admite que são as alterações de circuitos neuronais específicos (conetividade neuronal) que desencadeiam as PEAs. Contudo, o exato mecanismo que desencadeia as PEAs não está totalmente definido. Estudo epidemiológicos sugerem uma forte componente genética nesta desordem. Para além disto, estudos genéticos têm identificado proteínas envolvidas na função sináptica com implciações nas PAE.Os recetores acoplados às proteínas G (GPCRs) desempenham várias funções na regulação da sinalização sináptica. Assim, as proteínas que interagem ou regulam estes recetores, permitindo a sua modulação, poderão emergir como possíveis genes associados a PAE. Um exemplo de uma proteína que poderá interagir e/ou modular os GPCRs é a GPRASP2, tendo já sido associada a desordens neuropsiquiátricas, incluindo PAE. A GPRASP2 regula a sinalização das GPCR, incluindo dos recetores metabotrópicos de glutamato (mGLuRs), que estão igualmente associados a PEA e DI.De forma a perceber o papel da GPRASP2 no sistema nervoso, um modelo de murganhos knockout (KO) para o gene Gprasp2 foi desenvolvido. Este modelo apresenta diferentes fenótipos associados aos PEA, incluindo alterações no comportamento social. A GPRASP2 é expressa em diferentes regiões do cérebro, como no hipocampo, tálamo e hipotálamo. Para além disto, alterações na expressão dos níveis de GPRASP2 afetam a morfologias as espiculas dendríticas, a complexidade neuronal e o tráfico dos mGluRs, em culturas do hipocampo. Contudo, a função sináptica desta proteína e o seu papel na modulação da sinalização dos GPCR continua por concluir.De forma a entender a função da GPRASP2 nos recetores mGluR5s, recorreu-se a culturas organotáticas do hipocampo incubadas na presença e ausência de DHPG de forma a estudar as vias de sinalização ERK, PI3K e mTOR utilizando a técnica de western blot. Foram também averiguadas as alterações em proteínas sinápticas associadas à deleção do gene Gprasp2. Paralelamente, animais Gprasp2-/y e Gprasp2+/y foram sujeitos a tarefas de comportamento dependentes do hipocampo. De forma a estudar possíveis alterações na comunicação neonatal destes animais, as vocalizações foram detetadas em animais nas primeiras duas semanas de vida. Estas tarefas tiveram como objetivos analisar a função da GPRASP2 na sinapse e os efeitos da deleção deste gene no hipocampo.
Autism spectrum disorder (ASD) comprises a group of heterogeneous neurodevelopmental conditions, characterised by deficits in language, impairments in social interaction and communication and restricted interests and repetitive or stereotyped behaviours. Epidemiological studies estimate that more than 1% of the world’s human population has an ASD and that more than 70% of those with autism also have intellectual disability. There are two main theories to explain the pathophysiology of ASD: one involves failure in maintaining neural homoeostasis (synaptic dysfunction and E/I imbalance) and another involves alterations in specific neural circuits (cerebral connectivity). However, the exact mechanism that triggers an ASD is not yet fully understood.Twin and epidemiological studies strongly suggest that genetic factors play an important role in this disease. Moreover, genetic studies have now identified several proteins involved in synaptic function as strongly implicated in ASD.G-protein coupled receptors (GPCRs) have a role in the regulation of synaptic signalling. As such, GPCRs partners that modulate these receptors might be candidate genes for ASD. One example is the family of G-protein coupled receptor associated sorting proteins (GPRASP). GPRASP2, in particular, has been implicated in neuropsychiatric disorders, including ASD. GPRASP2 regulates GPCRs signalling, including metabotropic glutamate receptor (mGluR), themselves associated with ASD and ID pathophysiology. To understand the role of GPRASP2 in the nervous system, a new knockout (KO) mouse model was generated, and several autistic phenotypes, including abnormal social behaviour, were identified in this animal model. GPRASP2 is expressed in several brain regions, such as the hippocampus, thalamus, and hypothalamus. Additionally, changes in the expression levels of GPRASP2 can affect dendritic spine morphology, neuronal complexity and mGluR5 trafficking in rat hippocampal neuronal cultures. However, the synaptic function of GPRASP2 and its role in modulating GPCR signalling remains largely unknown.To elucidate the role of GPRASP2 upon mGluR5, hippocampal organotypic slices were incubated in the presence/absence of DHPG and downstream signalling was analysed using western blot techniques to study ERK, PI3K and mTOR pathways. We also assessed alterations in synaptic protein when GPRASP2 is ablated in vivo. Additionally, we performed hippocampal-dependent tasks and measured ultrasonic vocalisations in GPRASP2 knockout mice to complement the characterisation of this line as a model for ASD. These tasks were performed to understand the role of GPRASP2 in the synapse function and to dissect the downstream effects of its deletion.
Книги з теми "ASM dysfunction"
Manfredi, Claudia, ed. Models and Analysis of Vocal Emissions for Biomedical Applications. Florence: Firenze University Press, 2013. http://dx.doi.org/10.36253/978-88-6655-470-7.
Повний текст джерелаManfredi, Claudia, ed. Models and Analysis of Vocal Emissions for Biomedical Applications. Florence: Firenze University Press, 2009. http://dx.doi.org/10.36253/978-88-6453-096-3.
Повний текст джерелаMcCarthy, Barry, and Lana M. Wald. Sexual Dysfunction and Couple Dysfunction. Edited by Erika Lawrence and Kieran T. Sullivan. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199783267.013.006.
Повний текст джерелаde Bie, Robertus M. A., and Susanne E. M. Ten Holter. “My Arm Is Not Working”. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190607555.003.0011.
Повний текст джерелаFeldman, Jamie, and Karin Larsen. Sexual Dysfunction. Edited by C. Steven Richards and Michael W. O'Hara. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.032.
Повний текст джерелаPorst, Hartmut. Erectile dysfunction. Edited by David John Ralph. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0103.
Повний текст джерелаSaccà, Antonino, and Andrea Salonia. Erectile dysfunction. Edited by David John Ralph. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0102.
Повний текст джерелаChong, Ji Y., and Michael P. Lerario. Progressive Gait Dysfunction. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0034.
Повний текст джерелаFoorsov, Victor, Omar Dyara, Robert Bolash, and Bruce Vrooman. Sacroiliac Joint Dysfunction. Edited by Mehul J. Desai. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199350940.003.0019.
Повний текст джерелаAmin, Sandeep. Cervical Facet Dysfunction. Edited by Mehul J. Desai. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199350940.003.0005.
Повний текст джерелаЧастини книг з теми "ASM dysfunction"
Zaragoza, Briana, and T. Gonzalez. "Discrimination as dysfunction." In Libraries as Dysfunctional Organizations and Workplaces, 109–31. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003159155-5.
Повний текст джерелаHoover, Jasmine. "Dysfunction by (dis)organization." In Libraries as Dysfunctional Organizations and Workplaces, 283–91. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003159155-14.
Повний текст джерелаDoran-Myers, Miranda, and Crystal Schimpf. "Workplace dysfunction and intellectual freedom in public libraries." In Libraries as Dysfunctional Organizations and Workplaces, 132–48. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003159155-6.
Повний текст джерелаAcadia, Spencer, and Kyndal Vogt. "An introduction to dysfunction in the library workplace." In Libraries as Dysfunctional Organizations and Workplaces, 1–50. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003159155-1.
Повний текст джерелаGamache, Éthel, and Spencer Acadia. "You are seen." In Libraries as Dysfunctional Organizations and Workplaces, 193–210. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003159155-9.
Повний текст джерелаLopez, Erica. "Improving dysfunctional recruitment and retention in academic libraries by honoring the whole person." In Libraries as Dysfunctional Organizations and Workplaces, 72–94. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003159155-3.
Повний текст джерелаFoster Kaufman, Amanda, Amy F. Fyn, Millicent Weber, and Christina Heady. "The dysfunctional library and academic librarian turnover." In Libraries as Dysfunctional Organizations and Workplaces, 51–71. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003159155-2.
Повний текст джерелаGeary, Carol Anne, and Spencer Acadia. "A descriptive study of workplace bullying in U.S. libraries during the COVID-19 pandemic." In Libraries as Dysfunctional Organizations and Workplaces, 211–39. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003159155-10.
Повний текст джерелаSasyk, Zorian M. "Work alienation in academic libraries." In Libraries as Dysfunctional Organizations and Workplaces, 240–53. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003159155-11.
Повний текст джерелаVong, Silvia. "Bamboo ceiling reframed." In Libraries as Dysfunctional Organizations and Workplaces, 254–68. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003159155-12.
Повний текст джерелаТези доповідей конференцій з теми "ASM dysfunction"
Hald, Eric S., Zaw Win, Marianne R. Scheitel, and Patrick W. Alford. "High-Throughput Microtissue Contractility Assay for In Vitro Analysis of Vascular Mechanics." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14604.
Повний текст джерелаBell, E. David, and Samir N. Ghadiali. "Finite Element Analysis of Clinical Pressure-Flow Waveforms in a Collapsed Respiratory Airway." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-175329.
Повний текст джерелаOtis, James C., Sharon M. Kenneally, Matthew L. Hansen, and Jonathan T. Deland. "A Cadaveric Foot Model for Posterior Tibial Tendon Dysfunction." In ASME 1998 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1998. http://dx.doi.org/10.1115/imece1998-0109.
Повний текст джерелаStewart, Kelley C., Rahul Kumar, John J. Charonko, Pavlos P. Vlachos, and William C. Little. "A Hydrodynamic Efficiency Parameter as a Novel Left Ventricular Diastolic Dysfunction Diagnostic Metric." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192954.
Повний текст джерелаKilu, Rufai, Mohammed-Aminu Sanda, Lilian Ama Afun, and Anna Alacovska. "The dysfunctional systems of creative entrepreneurship in Ghana." In 13th International Conference on Applied Human Factors and Ergonomics (AHFE 2022). AHFE International, 2022. http://dx.doi.org/10.54941/ahfe1002157.
Повний текст джерелаHayashi, Shigeto, Hiromichi Nakadate, Yuelin Zhang, Kojiro Mekata, Haruo Yamashita, Shinichi Nakayama, Eiji Kohmura, Yasuhiro Matsui, Hong Ji, and Shigeru Aomura. "Reproduction Analysis of Injury Condition Using Finite Element Modeling of the Head in Cases With Traumatic Higher Brain Dysfunction Caused by Traffic Accidents." In ASME 2018 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/imece2018-86945.
Повний текст джерелаSheer, Francis J., and Samir N. Ghadiali. "Parametric Analysis of Eustachian Tube Function Using Fully Coupled Fluid-Structure Interaction Models." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19425.
Повний текст джерелаZakaria, Hasballah, and Johan Agathon. "Endothelial Cell Dysfunction Assessment by Arm Positioning Reactive Hyperemia Induced." In 2019 2nd International Conference on Bioinformatics, Biotechnology and Biomedical Engineering (BioMIC) - Bioinformatics and Biomedical Engineering. IEEE, 2019. http://dx.doi.org/10.1109/biomic48413.2019.9034832.
Повний текст джерелаZaylor, William, Betty Sindelar, and John R. Cotton. "Finite Element Analysis Demonstrates Splinting in the Porcine Mandibular Condyle Causes Changes in Bone Volume Fraction and Stiffness Anisotropy." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80311.
Повний текст джерелаScott, Devon, Robin Shandas, and Wei Tan. "Effect of Vessel Stiffening and High Pulsatility Flow on Contractile Function and Proliferation of Small Arterial Cells." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19597.
Повний текст джерелаЗвіти організацій з теми "ASM dysfunction"
Neodo, Anna, Fiona Augsburger, Jan Waskowski, Joerg C. Schefold, and Thibaud Spinetti. Monocytic HLA-DR expression and clinical outcomes in adult ICU patients with sepsis – a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0119.
Повний текст джерелаPlatt, Michael L. Neural Basis of Empathy and Its Dysfunction in Autism Spectrum Disorders (ASD). Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada612863.
Повний текст джерелаPlatt, Michael L., and Steve W. Chang. Neural Basis of Empathy and Its Dysfunction in Autism Spectrum Disorders (ASD). Fort Belvoir, VA: Defense Technical Information Center, August 2012. http://dx.doi.org/10.21236/ada575868.
Повний текст джерелаPlatt, Michael L., Steve W. Chang, and Jean-Francois Gariepy. Neural Basis of Empathy and Its Dysfunction in Autism Spectrum Disorders (ASD). Fort Belvoir, VA: Defense Technical Information Center, August 2013. http://dx.doi.org/10.21236/ada591282.
Повний текст джерелаShi, Xiaohua, Yu Bai, and Aiguo Wang. Shaoyao Gancao Decoction for limb dysfunction after fractures around the knee: A protocol for systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, January 2022. http://dx.doi.org/10.37766/inplasy2022.1.0028.
Повний текст джерелаCHEN, TIAN, and SONG GU. Network meta-analysis on effect of different exercise interventions on improving social dysfunction in children and adolescents with autism spectrum disorder(ASD). INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0085.
Повний текст джерелаDing, Huaze, Yiling Dong, Kaiyue Zhang, Jiayu Bai, and Chenpan Xu. Comparison of dexmedetomidine versus propofol in mechanically ventilated patients with sepsis: A meta-analysis of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0103.
Повний текст джерелаTan, Junjie, Yanhui Chen, Jianwen Huang, and Weiguo Xu. Endovenous Ablation for the treatment of Small Saphenous Varicose Veins: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0134.
Повний текст джерелаZerbib, Olivier, Yaniv Hadi, Daniel Kovarsky, Gal Sahaf Levin, Tamar Gottesman, Mor Darkhovsky, and Shaul Lev. Multiple Recurrent Pneumothoraces and Thoracic Drain Insertion in a Mechanically Ventilated Patient Suffering from Methadone Induced Cardiomyopathy. Science Repository, January 2023. http://dx.doi.org/10.31487/j.jcmcr.2022.01.02.
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