Готові списки джерел за темами / ABA1

Добірка наукової літератури з теми "ABA1"

Автор: Grafiati
Опубліковано: 10 грудня 2022
Оновлено: 28 січня 2023

Оформте джерело за APA, MLA, Chicago, Harvard та іншими стилями

Оберіть тип джерела:

Зміст

Ознайомтеся зі списками актуальних статей, книг, дисертацій, тез та інших наукових джерел на тему "ABA1".

Біля кожної праці в переліку літератури доступна кнопка «Додати до бібліографії». Скористайтеся нею – і ми автоматично оформимо бібліографічне посилання на обрану працю в потрібному вам стилі цитування: APA, MLA, «Гарвард», «Чикаго», «Ванкувер» тощо.

Також ви можете завантажити повний текст наукової публікації у форматі «.pdf» та прочитати онлайн анотацію до роботи, якщо відповідні параметри наявні в метаданих.

Статті в журналах з теми "ABA1"

1

Audran, Corinne, Sylviane Liotenberg, Martine Gonneau, Helen North, Anne Frey, Karine Tap-Waksman, Nicole Vartanian, and Annie Marion-Poll. "Localisation and expression of zeaxanthin epoxidase mRNA in Arabidopsis in response to drought stress and during seed development." Functional Plant Biology 28, no. 12 (2001): 1161. http://dx.doi.org/10.1071/pp00134.

Повний текст джерела
Анотація:
Abscisic acid (ABA) is involved in seed development and plant adaptation to environmental stresses. ABA is synthesized from cleaved xanthophylls and zeaxanthin epoxidase (ZEP) is the enzyme responsible for the conversion of zeaxanthin to violaxanthin. In this study, we have characterized the ABA1 gene (AtZEP) of Arabidopsis thaliana L. and show that this complements the aba1 mutant, defective in zeaxanthin epoxidation. The molecular basis for two aba1 mutant alleles has been determined and the reduction in their AtZEP transcript levels correlates with the molecular defect identified. As AtZEP mRNA abundance was not affected in two other ABA-deficient mutants (aba2 and aba3) and in two ABA-insensitive mutants (abi1 and abi2), no feedback regulation of ABA biosynthesis seems to occur at the level of ZEP transcription. Steady state transcript levels increased in roots during rapid water stress as well as progressive drought stress, providing evidence that zeaxanthin epoxidation contributed to the regulation of ABA biosynthesis in roots and consequently to the plant adaptive response to hydric stress. In seeds in situ hybridization analysis detectedAtZEP mRNA in the embryo cells from the globular stage to desiccation phase. In contrast, expression of AtZEP in maternal tissues was specific to the maturation phase. These results are discussed in relation to the role of ABA both in response to drought stress and in seed development.
Стилі APA, Harvard, Vancouver, ISO та ін.
2

Cramer, Grant R. "Response of abscisic acid mutants of Arabidopsis to salinity." Functional Plant Biology 29, no. 5 (2002): 561. http://dx.doi.org/10.1071/pp01132.

Повний текст джерела
Анотація:
Increases in abscisic acid (ABA) concentrations in plant tissues correlate with growth inhibition in salt-stressed plants. Therefore, it was hypothesized that Arabidopsis ABA mutants different in, or insensitive to, ABA would respond differently than wild type (wt) to salinity stress. Seeds (wt, abi1-1, abi2-1, abi3-1, and aba1-3) were germinated and grown hydroponically in three separate experiments with different environmental conditions: relative humidity at 80 or 100%, day/night temperatures at 21/18 or 23/20˚C, and light intensity at 125, 200 or 350 μmol photons m–2 s-1. Plants were exposed to salinity (either 0, 40 and 80 mM NaCl or 1, 5, and 9 dS m–1 with a Na/Ca ratio of 10 depending on the experiment) for one to several weeks before harvesting. The effect of salinity on root elongation rates of young seedlings was measured as well. Two-way ANOVA of root elongation rates of young seedlings and the growth of 3-week old plants in hydroponic solutions indicated that salinity inhibited growth, increased ABA and Na concentrations, and reduced K concentrations in all genotypes tested. However, there were no significant interactions with salinity and genotype for root elongation rates, total dry weight, shoot ABA and K concentrations. Shoot Na concentrations were significantly higher in wt plants relative to other genotypes subjected to high salinity stress. aba1-3 had significantly lower ABA concentrations than other genotypes, but the interaction of aba1-3 with salinity was the same as other genotypes. The lack of difference in interaction between genotype and salinity indicates that all genotypes responded in the same manner and amount to salinity for the particular parameter measured. Therefore, it appears that there are no significant differences in growth in response to salinity between the ABA mutants (ABA-deficient and ABA-insensitive) and wt. However, in contrast to the other genotypes, some of the ABA-deficient plants, aba1-3, died when exposed to high salinity and high light intensity. ABA appears to provide a protective role in conditions of high salinity and high light intensity.
Стилі APA, Harvard, Vancouver, ISO та ін.
3

Assmann, Sarah M., Jo Ann Snyder, and Yuh‐Ru Julie Lee. "ABA‐deficient (aba1)andABA‐insensitive (abi1‐1, abi2‐1) mutants ofArabidopsishave a wild‐type stomatal response to humidity." Plant, Cell & Environment 23, no. 4 (April 2000): 387–95. http://dx.doi.org/10.1046/j.1365-3040.2000.00551.x.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
4

Gao, F., J. F. Whitaker, Y. Liu, C. Uher, C. E. Platt, and M. V. Klein. "Terahertz transmission of aBa1−xKxBiO3film probed by coherent time-domain spectroscopy." Physical Review B 52, no. 5 (August 1, 1995): 3607–13. http://dx.doi.org/10.1103/physrevb.52.3607.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
5

Kaliff, Maria, Jens Staal, Mattias Myrenås, and Christina Dixelius. "ABA Is Required for Leptosphaeria maculans Resistance via ABI1- and ABI4-Dependent Signaling." Molecular Plant-Microbe Interactions® 20, no. 4 (April 2007): 335–45. http://dx.doi.org/10.1094/mpmi-20-4-0335.

Повний текст джерела
Анотація:
Abscisic acid (ABA) is a defense hormone with influence on callose-dependent and -independent resistance against Leptosphaeria maculans acting in the RLM1Col pathway. ABA-deficient and -insensitive mutants in Ler-0 background (aba1-3 and abi1-1) displayed susceptibility to L. maculans, along with a significantly decreased level of callose depositions, whereas abi2-1 and abi3-1 remained resistant, together with the abi5-1 mutant of Ws-0 background. Suppressor mutants of abi1-1 confirmed that the L. maculans-susceptible response was due to the dominant negative nature of the abi1-1 mutant. Highly induced camalexin levels made ABA mutants in Col-0 background (aba2-1, aba3-1, and abi4-1) appear resistant, but displayed enhanced susceptibility as double mutants with pad3-1, impaired in camalexin biosynthesis. β-Aminobutyric acid (BABA) pretreatment of Ler-0 contributed to an elevated level of endogenous ABA after L. maculans inoculation. Comparisons between (RLM1Col)pad3 and rlm1Lerpad3 showed that ABA and BABA enhancement of callose deposition requires induction from RLM1Col·ABI1, but not ABI2, was found to be involved in a feedback mechanism that modulates RLM1Col expression. Genetic analysis showed further that this feedback occurs upstream of ABI4 and that components downstream of ABI4 modulate ABI1 activity. ABA and BABA treatments of the L. maculans-susceptible callose synthase mutant pmr4 showed that ABA also induces a callose-independent resistance. Similar treatments enhanced callose depositions and induced resistance to L. maculans in oilseed rape, and BABA-induced resistance was found to be independent of salicylic acid.
Стилі APA, Harvard, Vancouver, ISO та ін.
6

Strizhov, Nicolai, Edit Ábrahám, László Ökrész, Stefan Blickling, Aviah Zilberstein, Jeff Schell, Csaba Koncz, and László Szabados. "Differential expression of two P5CS genes controlling proline accumulation during salt‐stress requires ABA and is regulated by ABA1 , ABI1 and AXR2 in Arabidopsis." Plant Journal 12, no. 3 (September 1997): 557–69. http://dx.doi.org/10.1046/j.1365-313x.1997.00537.x.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
7

Strizhov, Nicolai, Edit Ábrahám, László Ökrész, Stefan Blickling, Aviah Zilberstein, Jeff Schell, Csaba Koncz, and László Szabados. "Differential expression of two P5CS genes controlling proline accumulation during salt‐stress requires ABA and is regulated by ABA1, ABI1 and AXR2 in Arabidopsis." Plant Journal 12, no. 3 (September 1997): 557–69. http://dx.doi.org/10.1111/j.0960-7412.1997.00557.x.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
8

BARRERO, JOSÉ MARÍA, PEDRO L. RODRÍGUEZ, VÍCTOR QUESADA, DAVID ALABADÍ, MIGUEL A. BLÁZQUEZ, JEAN-PIERRE BOUTIN, ANNIE MARION-POLL, MARÍA ROSA PONCE, and JOSÉ LUIS MICOL. "The ABA1 gene and carotenoid biosynthesis are required for late skotomorphogenic growth in Arabidopsis thaliana." Plant, Cell & Environment 31, no. 2 (November 7, 2007): 227–34. http://dx.doi.org/10.1111/j.1365-3040.2007.01759.x.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
9

Chen, Huihuang, Linyi Lai, Lanxin Li, Liping Liu, Bello Hassan Jakada, Youmei Huang, Qing He, Mengnan Chai, Xiaoping Niu, and Yuan Qin. "AcoMYB4, an Ananas comosus L. MYB Transcription Factor, Functions in Osmotic Stress through Negative Regulation of ABA Signaling." International Journal of Molecular Sciences 21, no. 16 (August 10, 2020): 5727. http://dx.doi.org/10.3390/ijms21165727.

Повний текст джерела
Анотація:
Drought and salt stress are the main environmental cues affecting the survival, development, distribution, and yield of crops worldwide. MYB transcription factors play a crucial role in plants’ biological processes, but the function of pineapple MYB genes is still obscure. In this study, one of the pineapple MYB transcription factors, AcoMYB4, was isolated and characterized. The results showed that AcoMYB4 is localized in the cell nucleus, and its expression is induced by low temperature, drought, salt stress, and hormonal stimulation, especially by abscisic acid (ABA). Overexpression of AcoMYB4 in rice and Arabidopsis enhanced plant sensitivity to osmotic stress; it led to an increase in the number stomata on leaf surfaces and lower germination rate under salt and drought stress. Furthermore, in AcoMYB4 OE lines, the membrane oxidation index, free proline, and soluble sugar contents were decreased. In contrast, electrolyte leakage and malondialdehyde (MDA) content increased significantly due to membrane injury, indicating higher sensitivity to drought and salinity stresses. Besides the above, both the expression level and activities of several antioxidant enzymes were decreased, indicating lower antioxidant activity in AcoMYB4 transgenic plants. Moreover, under osmotic stress, overexpression of AcoMYB4 inhibited ABA biosynthesis through a decrease in the transcription of genes responsible for ABA synthesis (ABA1 and ABA2) and ABA signal transduction factor ABI5. These results suggest that AcoMYB4 negatively regulates osmotic stress by attenuating cellular ABA biosynthesis and signal transduction pathways.
Стилі APA, Harvard, Vancouver, ISO та ін.
10

Arenas-Huertero, Francisco, Analilia Arroyo, Li Zhou, Jen Sheen, and Patricia León. "Analysis of Arabidopsis glucose insensitive mutants, gin5 and gin6, reveals a central role of the plant hormone ABA in the regulation of plant vegetative development by sugar." Genes & Development 14, no. 16 (August 15, 2000): 2085–96. http://dx.doi.org/10.1101/gad.14.16.2085.

Повний текст джерела
Анотація:
Sugars have signaling roles in a wide variety of developmental processes in plants. To elucidate the regulatory components that constitute the glucose signaling network governing plant growth and development, we have isolated and characterized two Arabidopsisglucose insensitive mutants, gin5 and gin6, based on a glucose-induced developmental arrest during early seedling morphogenesis. The T-DNA-tagged gin6 mutant abrogates the glucose-induced expression of a putative transcription factor, ABI4, previously shown to be involved in seed-specific abscisic acid (ABA) responses. Thus, ABI4 might be a regulator involved in both glucose- and seed-specific ABA signaling. The characterization of thegin5 mutant, on the other hand, reveals that glucose-specific accumulation of ABA is essential for hexokinase-mediated glucose responses. Consistent with this result, we show that three ABA-deficient mutants (aba1-1, aba2-1, andaba3-2) are also glucose insensitive. Exogenous ABA can restore normal glucose responses in gin5 and aba mutants but not in gin6 plants. Surprisingly, only abi4 andabi5-1 but not other ABA-insensitive signaling mutants (abi1-1, abi2-1, and abi3-1) exhibit glucose insensitivity, indicating the involvement of a distinct ABA signaling pathway in glucose responses. These results provide the first direct evidence to support a novel and central role of ABA in plant glucose responses mediated through glucose regulation of both ABA levels by GIN5 and ABA signaling by GIN6/ABI4.
Стилі APA, Harvard, Vancouver, ISO та ін.
Більше джерел

Дисертації з теми "ABA1"

1

Ghassemian, Majid. "Genetic and molecular interactors of ABA insensitive1 gene (abi1) of Arabidopsis thaliana." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0025/NQ49826.pdf.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
2

Van, Thielen Nocha. "Genetic interactions between the ABA insensitive mutant, abi1-1, and the ABA supersensitive mutant, era1-4, in Arabidopsis thaliana." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0001/MQ45891.pdf.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
3

Juan, Emanuel Enrique. "ABAX S.R.L." Universidad Nacional de Cuyo. Facultad de Ingeniería, 2018. http://bdigital.uncu.edu.ar/10952.

Повний текст джерела
Анотація:
El presente informe, describe las principales actividades desarrolladas por el alumno durante la realización de la Práctica Profesional Supervisada (PPS). Estas se realizaron en la consultora de ingeniería estructural ABAX SRL, la cual desarrolla sus actividades en base al diseño de obras civiles varias. Los trabajos efectuados, fueron mayoritariamente de gabinete, valiéndose para ello de apoyo computacional, donde se utilizaron herramientas de software como AutoCAD ®, MathCAD ®, RAM Advanse ®, RAM Elements ®, Revit Structure ®, ETABS ®, Response2000 ® y consistieron principalmente sobre tres proyectos, con intervenciones expeditas sobre problemas específicos a resolver en otros proyectos. El informe se articula siguiendo un orden cronológico, comenzando por los objetivos de las prácticas, los trabajos realizados con sus dificultades y soluciones adoptadas y una evaluación posterior donde se destacan las conclusiones y aprendizajes de estas prácticas.
Fil: Juan, Emanuel Enrique. Universidad Nacional de Cuyo. Facultad de Ingeniería.
Стилі APA, Harvard, Vancouver, ISO та ін.
4

Dasgupta, Yashodhara. "NORMAL ABL1 IS A TUMOR SUPPRESSOR AND THERAPEUTIC TARGET IN BCR-ABL1-POSITIVE LEUKEMIAS." Diss., Temple University Libraries, 2014. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/239008.

Повний текст джерела
Анотація:
Biology
Ph.D.
BCR-ABL1 results from t(9;22)(q34;q11) reciprocal translocation resulting in BCR-ABL1 kinase expression, initiating chronic myeloid leukemia in chronic phase (CML-CP). At the initial stages of CML-CP both oncogenic BCR-ABL1 kinase and normal ABL1 kinase are expressed, however, loss of ABL1 kinase expression in CML-CP can result from an interstitial deletion in the normal chromosome 9 [del(9q34)] which may be combined with the transcriptional silencing of the alternative ABL1 promoter within the translocation eventually leading to disease progression and drug resistance. We found that BCR-ABL1 Abl1-/- cells generated a CML-blast phase (BP)-like disease phenotype in NOD-SCID mice compared to the BCR-ABL1 Abl1+/+ cells. To determine the mechanisms responsible for blastic transformation of BCR-ABL1 Abl1-/- cells, we examined the role of ABL1 in proliferation, differentiation, apoptosis, genomic instability, and stemness. The presence of ABL1 inhibited proliferation in BCR-ABL1 cells as BCR-ABL1 Abl1-/- cells had higher clonogenic activity and proliferative rate compared to their wild-type counterparts. ABL1 is essential for myeloid differentiation since BCR-ABL1 Abl1-/- cells showed an immature blast phenotype when stained with Wright-Giemsa and myeloid differentiation markers Gr-1 and CD11b. ABL1 promoted apoptosis in response to genotoxic stress as revealed by reduced clonogenicity and expression of p53, phosphoserine-15 p53 and activated caspase 3 in BCR-ABL1 Abl1+/+ compared to knock-out cells. Although the absence of ABL1 did not enhance ROS and oxidative DNA damage, it appears that an impaired DNA damage response may be responsible for higher chromosome numbers and an accumulation of high numbers of chromosomal aberrations in BCR-ABL1 Abl1-/- cells. We detected an expansion of Lin-c-Kit+Sca-1+ leukemia stem cells (LSCs) in BCR-ABL1 Abl1-/- cells compared to BCR-ABL1 Abl1+/+ or non-transformed counterparts; among the LSCs, there was a higher percentage of CD34-Flt3- long-term and CD34+Flt3- short-term stem cells. These results showed that ABL1 is involved in regulating the LSC compartment in BCR-ABL1 cells. DNA microarray analysis revealed changes in mRNA levels of several genes involved in proliferation, myeloid differentiation, apoptosis, DNA damage response and `stemness' in BCR-ABL1 Abl1-/- cells in comparison to BCR-ABL1 Abl1+/+ cells. Together, these results demonstrate a critical role of ABL1 as a tumor suppressor in BCR-ABL1-induced leukemia, prolonging survival in mice by suppressing proliferation and expansion of LSC, inducing myeloid differentiation, apoptosis and DNA damage response in BCR-ABL1 cells. Loss of ABL1 was also found to contribute to Imatinib resistance in BCR-ABL1 cells. Moreover, we hypothesized that enhancement of the tumor-suppressor function of ABL1 may have a significant impact on CML treatment. A small molecule activator of ABL1 kinase, 5-(1,3-diaryl-1H-pyrazol-4-yl)hydantoin (DPH), have been reported to interact with the myristoyl-binding site of ABL1 and destabilize the bent conformation of the á1 helix, thereby preventing the auto-inhibitory conformation. Western blot analysis revealed partially restored activation of ABL1 kinase when Imatinib-treated cells were incubated with DPH. DPH along with Imatinib was found to inhibit viability of BCR-ABL1 Abl1+/+ cells but not BCR-ABL1 Abl1-/- cells demonstrating its ABL1-specific mode of action. DPH when used in combination with tyrosine kinase inhibitors such as Imatinib and Ponatinib inhibited growth of CML CD34+ cells, Philadelphia chromosome-positive B-Acute Lymphoblastic Leukemia (Ph+B-ALL) cells and relapsed Ph+B-ALL cells harboring T315I mutation without affecting normal counterparts. A similar inhibitory effect was observed when TEL-ABL1-expressing cell lines and NUP214-ABL1-expressing murine bone marrow cells were treated with DPH and Imatinib, as well as Acute Myeloid Leukemia (AML) cells expressing FLT3-ITD mutation when treated with DPH in combination with AC220 which is the FLT3-ITD inhibitor. In summary, ABL1 is a potential tumor-suppressor in BCR-ABL1-induced leukemia and stimulation of its function may play a significant role in the development of novel therapeutic strategies for CML and other Fusion Tyrosine Kinase (FTK)-mediated hematologic malignancies.
Temple University--Theses
Стилі APA, Harvard, Vancouver, ISO та ін.
5

Kang, Martin Hubert. "Post-transcriptional regulation of ABCA1." Thesis, University of British Columbia, 2012. http://hdl.handle.net/2429/43655.

Повний текст джерела
Анотація:
Epidemiological studies consistently demonstrate an inverse relationship between HDL levels and cardiovascular disease (CVD), independent of LDL and triglyceride levels. Due to the crucial role ABCA1 plays in HDL biogenesis, increasing ABCA1 expression is considered an attractive strategy to increase plasma HDL levels. In this thesis we attempt to identify novel post-transcriptional and post-translational mechanisms that regulate ABCA1 expression and/or function. Prior to translation, ABCA1 protein expression is regulated by non-coding RNA molecules known as microRNAs which bind and inhibit translation of mature mRNA transcripts in the cytoplasm. In this study we used bioinformatic prediction programs to identify potential microRNA regulators of ABCA1. Using reporter constructs, protein expression analysis by immunoblotting, and cholesterol efflux assays, we validated microRNA-145 as a novel repressor of ABCA1 translation. The inhibition of endogenous microRNA-145 in HepG2 cells increases both ABCA1 protein levels and cholesterol efflux activity. The inhibition of this microRNA in the liver is a potential strategy to increase HDL levels. Following translation, numerous post-translational modifications and protein-protein interactions are required for the ABCA1 protein to function properly. In this study we identified palmitoylation as a novel post-translational modifier of ABCA1. The majority of ABCA1-mediated cholesterol efflux and HDL biogenesis occurs at the cell surface. We show that palmitoylation is a crucial lipid addition for proper ABCA1 plasma membrane localization. We also identify a number of enzymes that mediate the incorporation of radio-labeled palmitate onto ABCA1, and demonstrate that the overexpression of the palmitoyl transferase enzyme DHHC8 increases ABCA1 palmitoylation and cholesterol efflux activity. The increase of ABCA1 palmitoylation in the liver is a novel strategy to increase HDL levels. In this thesis, we have contributed to the understanding of ABCA1 biology by the identification of two novel regulators of ABCA1 expression and/or function.
Стилі APA, Harvard, Vancouver, ISO та ін.
6

Williams, Jeremy Hugh. "Monitoring the condition of semi-natural vegetation : the application of remote sensing and geographical information systems (GIS)." Thesis, Bangor University, 1992. https://research.bangor.ac.uk/portal/en/theses/monitoring-the-condition-of-seminatural-vegetation--the-application-of-remote-sensing-and-geographical-information-systems-gis(c90702cb-be4f-454f-aba1-1c74484c0093).html.

Повний текст джерела
Анотація:
The principal objective of this thesis was to investigate the use of remote sensing and Geographical Information System (GIS) technology in the survey and monitoring of semi-natural, vegetation. The effects of acidic deposition and airborne pollutants on vegetation were of particular interest during the 1980s and early 1990s. A first experiment studied the effect of simulated acid rain on the reflectance of birch seedlings. Plants exposed to acidic treatments lost the characteristic reflectance curve shape of healthy green vegetation. Spectroradiometer data were used to discriminate between plants in different rainfall treatments. A second experiment studied the effects of combinations of pollutant gases (03, S02+NO2, and 03+SO2+NO2) and acidic mists on the reflectance of white clover. Plants in the two treatments containing ozone showed marked changes in reflectance, and were statistically separable from the control. Simple and 4-waveband vegetation indices showed positive linear relationships with shoot dry weight. Plants in the treatments containing ozone showed marked decreases in shoot dry weight and vegetation index. Airborne Thematic Mapper (ATMý data were used to study the relationships between remotely-sensed radiance and water and soil chemistry on a large flood-plain mire in south Wales. Strong relationships between radiance and chemistry were found, suggesting associations between nutrient concentrations and the health and vigour of the mire vegetation. A study on the Glyderau mountains in Snowdonia investigated the potential for mapping upland vegetation using Landsat Thematic Mapper (TM) data. It addressed the problems involved in classifying highly variable ground cover on valley floors, steep slopes and high plateaux, and the problems involved in reconciling the need for a generalised vegetation map with the fine detail present on the ground and in TM data. Pre- and post-classification digital spatial filters were used to produce TM classmaps which agreed closely with the ground survey data. GIS was used to extract management information.
Стилі APA, Harvard, Vancouver, ISO та ін.
7

Dawalibi, Paul. "L'amour comme aban-don de soi." Paris 4, 2008. http://www.theses.paris-sorbonne.fr/these_dawalibi/paris4/2008/these_dawalibi/html/index-frames.html.

Повний текст джерела
Анотація:
Par l’épreuve de la perte de soi, de la perte de l’intimité de soi, la mienneté se laisse-être-prise selon les exigences d’un aban-don originaire à ce qui fait commencer en elle la dé-mesure, la non-limite et le surcroît de ce qui a déjà commencé en elle depuis sa naissance. Ainsi, toute ressemblance tient en elle sa dif-férence, toute appropriation s’expose à une altération, tout pouvoir se brise, tout orgueil s’aliène et tout narcissisme s’étouffe sous le charme de sa propre beauté. Et puisque la limite d’un « chez soi » est par définition non-familière, le paradoxe des frontières, ainsi produit, altérera en tout cas le pouvoir d’être soi et même la signification d’un « chez soi » ontologique. Dès lors, se perdre c’est laisser l’aban-don de soi faire advenir l’arrivage de l’originaire en soi, c’est être originairement selon l’arrivage de l’événement de sa naissance, du recommencement du don originaire de soi. Seul l’arrivage de ce qui fait naître l’ipséité originairement, possibilise le se-laisser-être-aban-donné de soi. Seul l’arrivage de l’Amour rend possible la donation originaire de l’ipséité comme aban-don de soi
By the event of the loss of one, the loss of the intimacy of one, the self leash-being-taken according to the requirements of a native abnegation or renunciation in what made begin in her the immoderation, the nonlimit and the increasing of what has already begun in her since her birth. So, any resemblance holds in her its difference, any appropriation exposes it self to a change, any power breaks, any pride alienates and any narcissism suffocate or suppresses under the charm of its own beauty. And because the limit of one "at home" is by definition non-familiar, the paradox of the borders, so produced, will distort (alter) at least the power to be one and even the meaning of an "at home" ontological. From then on, get lost it is to let the self-abnegation make the native come in itself, it is to be originally according to the arrival of the event of its birth, of the resumption of the donation native of one. Only the arrival of what creates the self originally makes possible to let-be-abandoned by one. Only the arrival of Love makes possible the native donation as the self-abnegation
Стилі APA, Harvard, Vancouver, ISO та ін.
8

Chambenoit, Olivier. "ABCA1 et l'homéostasie du cholestérol cellulaire." Aix-Marseille 2, 2003. http://www.theses.fr/2003AIX22023.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
9

Dawalibi, Paul Marion Jean-Luc Greisch Jean. "L'amour comme aban-don de soi." Paris : Université Paris Sorbonne - Paris IV, 2008. http://www.theses.paris4.sorbonne.fr/these_dawalibi/paris4/2008/these_dawalibi/html/index-frames.html.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
10

Bélair, Caroline. "Ab1-42 and Ab1-40 induce tau phosphorylation in human neurons." Thesis, McGill University, 1997. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=27279.

Повний текст джерела
Анотація:
Alzheimer's disease is characterized by (1) senile plaques formed of aggregated amyloid peptides of 40 to 42 amino acids (A$ beta sb{1-40}$ and A$ beta sb{1-42}$) which are derived from the metabolism of the amyloid precursor protein, (2) by neurofibrillary tangles involving a dysfunction of the cytoskeleton due to the hyperphosphorylation of the tau protein, and (3) by amyloid-laden cerebral vessels. The goal of this project is to determine if there is a link between the presence of aggregated amyloid peptides and tau hyperphosphorylation.
We incubated the human fetal primary neuron cultures with A$ beta sb{1-40}$ and A$ beta sb{1-42}$ (100nM). The western blots show that serine 202 epitope of tau protein is phosphorylated in a cyclic manner. With the tau phosphorylation assay, we showed that A$ beta sb{1-40}$ and A$ beta sb{1-42}$ activate one or more protein kinase(s) able to phosphorylate tau protein and more specifically the serine 202 epitope.
We conclude that pathological concentrations of aggregated peptides A$ beta sb{1-40}$ and A$ beta sb{1-42}$ activate protein kinases which induce the phosphorylation of tau protein and serine 202 epitope.
Стилі APA, Harvard, Vancouver, ISO та ін.
Більше джерел

Книги з теми "ABA1"

1

Nazarbaev, Nursultan. Abaĭ turaly sȯz =: Slovo ob Abae = Word on Abay. Almaty: "Rauan", 1995.

Знайти повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
2

Salahudin, Asep. Pangersa Abah Anom: Wali fenomenal abad 21 & ajarannya. Jagakarsa, Jakarta: Noura Books, 2013.

Знайти повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
3

Zhamsuev, B. B., and Balʹzhit Dambaeva. Abaĭ Gėsėr bogdo khan =: Abai geser boġda qaġan. Aginskoe: Izd-vo Buri︠a︡tskogo nauchnogo t︠s︡entra SO RAN, 2007.

Знайти повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
4

Atmowiloto, Arswendo. Abal-abal. Jakarta: Pustaka Utama Grafiti, 1994.

Знайти повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
5

(Publisher), Kosa Kata Kita. Negeri abal-abal. Jakarta: Penerbit Kosa Kata Kita, 2013.

Знайти повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
6

Belorgey, Jean-Michel. Abad. Saint-Pourçain-sur-Sioule: Bleu autour, 2005.

Знайти повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
7

Maĭtanov, B. Abaĭ. Almaty: Aruna, 2004.

Знайти повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
8

Massardi, Adhie Moelyadi. Pilpres abal-abal republik amburadul. Pasar Minggu, Jakarta: Republika Penerbit, 2011.

Знайти повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
9

Theobald, Patrick. Profikurs ABAP: Konkrete, praxisorientierte Lo sungen ; Tipps, Tricks und jede Menge Erfahrung. 2nd ed. Wiesbaden: Vieweg, 2007.

Знайти повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
10

Riekert, Rainer. ABAP-Programmierung: Fortgeschrittene Programmiertechniken fu r ABAP. Mu nchen: Addison-Wesley, 2001.

Знайти повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
Більше джерел

Частини книг з теми "ABA1"

1

Biswas-Fiss, Esther E., Stephanie Affet, Malissa Ha, Takaya Satoh, Joe B. Blumer, Stephen M. Lanier, Ana Kasirer-Friede, et al. "ABC1 (for ABCA1) and ABC2 (for ABCA2)." In Encyclopedia of Signaling Molecules, 21. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100025.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
2

Kirchner, Mark. "Abay." In Kindlers Literatur Lexikon (KLL), 1. Stuttgart: J.B. Metzler, 2020. http://dx.doi.org/10.1007/978-3-476-05728-0_1219-1.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
3

Markandeya, Sushil, and Kaushik Roy. "ABAP OOP." In SAP ABAP, 575–645. Berkeley, CA: Apress, 2014. http://dx.doi.org/10.1007/978-1-4302-4804-0_11.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
4

Morris, Christine M., and Suzanne M. Benjes. "BCR-ABL1." In Encyclopedia of Cancer, 1–14. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_571-3.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
5

Gellert, Ulrich, and Ana Daniela Cristea. "ABAP Dictionary." In Web Dynpro ABAP for Practitioners, 35–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-38247-5_4.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
6

Gellert, Ulrich, and Ana Daniela Cristea. "ABAP Dictionary." In Web Dynpro ABAP for Practitioners, 33–53. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-11385-7_4.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
7

Gellert, Ulrich, and Ana Daniela Cristea. "ABAP Dictionary." In Xpert.press, 35–55. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-11387-1_4.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
8

Morris, Christine M., and Suzanne M. Benjes. "BCR-ABL1." In Encyclopedia of Cancer, 460–72. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_571.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
9

Morris, Christine M., and Suzanne M. Benjes. "BCR-ABL1." In Encyclopedia of Cancer, 368–73. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_571.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
10

Markandeya, Sushil, and Kaushik Roy. "ERP and SAP Overview." In SAP ABAP, 1–20. Berkeley, CA: Apress, 2014. http://dx.doi.org/10.1007/978-1-4302-4804-0_1.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.

Тези доповідей конференцій з теми "ABA1"

1

Sundyreva, M. A., A. N. Rebrov, A. E. Mishko, and E. O. Lutsky. "The effect of sucrose concentration in the culture medium on the formation of abscisic acid and the activity of the photosynthetic apparatus of grape plants in vitro." In 2nd International Scientific Conference "Plants and Microbes: the Future of Biotechnology". PLAMIC2020 Organizing committee, 2020. http://dx.doi.org/10.28983/plamic2020.240.

Повний текст джерела
Анотація:
An increase in sucrose in the medium increased the content of pigments, gene expression of the photosynthetic apparatus, growth processes, and H2O2, but decreased the quantum yield of photosynthesis. With a change in the sucrose content in the medium, the expression of ABA1 increased most intensively. 30 g/l of sucrose in the medium inhibited the expression of genes involved in the formation of ABA.
Стилі APA, Harvard, Vancouver, ISO та ін.
2

Bhatt, Smriti, and Ravi Sandhu. "ABAC-CC." In SACMAT '20: The 25th ACM Symposium on Access Control Models and Technologies. New York, NY, USA: ACM, 2020. http://dx.doi.org/10.1145/3381991.3395618.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
3

Abirami, G., and Revathi Venkataraman. "Performance Analysis of ABAC and ABAC with Trust (ABAC-T) in Fine Grained Access Control Model." In 2019 11th International Conference on Advanced Computing (ICoAC). IEEE, 2019. http://dx.doi.org/10.1109/icoac48765.2019.246870.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
4

Morisset, Charles, Tim A. C. Willemse, and Nicola Zannone. "Efficient Extended ABAC Evaluation." In SACMAT '18: The 23rd ACM Symposium on Access Control Models and Technologies. New York, NY, USA: ACM, 2018. http://dx.doi.org/10.1145/3205977.3205980.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
5

Kamarova, Nagbdu S. "Foreign words included in the Abay dictionary in the Kazakh language: features of use." In Lexicography of the digital age. TSU Press, 2021. http://dx.doi.org/10.17223/978-5-907442-19-1-2021-93.

Повний текст джерела
Анотація:
The article says that Abay is not only a skilled user of the rich value of the national language, but also a poet who creates new words, new meanings. The article also deals with the purpose of creating a dictionary of Abay. Writing this article, we were looking for answers to the questions of how many words the great poet used in writing his works and in what sense he used them. The article analyzes foreign words included in the Abay dictionary in the Kazakh language and their features of use.
Стилі APA, Harvard, Vancouver, ISO та ін.
6

Savion, N., A. Gamliel, and N. Farzame. "THROMBIN INTERACTION WITH CULTURED AORTIC AND CAPILLARY ENDOTHELIAL CELLS: BINDING, INTERNALIZATION, DEGRADATION AND RELEASE OF PROTEASE NEXINS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644734.

Повний текст джерела
Анотація:
Thrombin (Th) binds specifically to confluent cultures of bovine aortic (ABAE) and brain capillary (BBC) endothelial cells. Saturation of 125I-Th binding is observed after 1 h exposure to the ligand and at an extracellular concentration of 0.5 and 1.0 µg/ml for ABAE and BBC cells, respectively. Under optimal conditions both ABAE and BBC cultures bind about 2 to 5 ng/106 cells, which represents about 20% of Th binding.to bovine corneal endothelial (BCE) cells. The cell associated 125I-Th in ABAE and BBC cells is internalized and degraded as described in BCE cells. The nature of the cell associated radioactivity is analyzed on SDS-polyacrylamide -gel electrophoresis and in ABAE and BBC cells about 30% of the I-Th appears in a complex with protease nexin I (PN I) while in BCE cells about 70% of the binding is mediated by PN I. ABAE cells possess 3 types of complexes, one which appears only on the cell surface with a molecular weight of 78 kDa, and two other complexes which appear only in the conditioned medium (CM) with molecular weights of 84 and 85 kDa. BBC and BCE cells demonstrate only one type of complex with a molecular weight of 77 kDa which appears both on the cell surface and in the CM. Preincubation of BCE cultures in the presence of Th is known to up-regulate the amount of PN I on the cell surface and in the CM, but this Th induced up-regulation effect is not observed in ABAE or BBC cells.The results described above indicate a difference between ABAE and BBC cells although both cell types growunder similar conditins and demonstrate similar morphological appearance. However, in both vascular endothelial cell types the total amount of PN I and its metabolism is relatively small compared to corneal endothelial cells. It, therefore, may indicate the lower capacity of vascular endothelial cells to control serine proteases activity at or near their cell surfaces as compared to corneal endothelial cells. This research was supported by a grant from the NationalCouncil for Research and Development, Israel and G.S.F. Munchen, Germany
Стилі APA, Harvard, Vancouver, ISO та ін.
7

He, Ping-Ping, Xin-Ping OuYang, Kai Yin, Wu-Jun Chen, Qian Lu, Zhong-Cheng Mo, Guo-Jun Zhao, Yun-Cheng Lv, and Chao-Ke Tang. "PDE Inhibitor Rolipram Increases Cholesterol Efflux and ABCA1 Expression: Rolipram Increases ABCA1 Expression." In 2012 International Conference on Biomedical Engineering and Biotechnology (iCBEB). IEEE, 2012. http://dx.doi.org/10.1109/icbeb.2012.292.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
8

Batra, Gunjan, Vijayalakshmi Atluri, Jaideep Vaidya, and Shamik Sural. "Incremental Maintenance of ABAC Policies." In CODASPY '21: Eleventh ACM Conference on Data and Application Security and Privacy. New York, NY, USA: ACM, 2021. http://dx.doi.org/10.1145/3422337.3447825.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
9

Chang, G. S. "Pu-Rich MOX Agglomerate-by-Agglomerate Model for Fuel Pellet Burnup Analysis." In 12th International Conference on Nuclear Engineering. ASMEDC, 2004. http://dx.doi.org/10.1115/icone12-49141.

Повний текст джерела
Анотація:
In support of potential licensing of the mixed oxide (MOX) fuel made from weapons-grade (WG) plutonium and depleted uranium for use in United States reactors, an experiment containing WG-MOX fuel is being irradiated in the Advanced Test Reactor (ATR) at the Idaho National Engineering and Environmental Laboratory (INEEL). The WG-MOX comprises five percent PuO2 and 95% depleted UO2. Based on the Post Irradiation Examination (PIE) observation, the volume fraction (VF) of MOX agglomerates in the fuel pellet is about 16.67%, and PuO2 concentration of 30.0 = (5 / 16.67 × 100) wt% in the agglomerate. A pressurized water reactor (PWR) unit WG-MOX lattice with Agglomerate-by-Agglomerate Fuel (AbAF) modeling has been developed. The effect of the irregular agglomerate distribution can be addressed through the use of the Monte Carlo AbAF model. The AbAF-calculated cumulative ratio of AGglomerate burnup to U-MAtrix burnup (AG/MA) is 9.17 at the beginning of life (BOL), and decreases to 2.88 at 50 GWd/t. The MCNP-AbAF-calculated results can be used to adjust the parameters in the MOX fuel fission gas release modeling.
Стилі APA, Harvard, Vancouver, ISO та ін.
10

Caminada, Martin, and Claudia Schulz. "On the Equivalence between Assumption-Based Argumentation and Logic Programming (Extended Abstract)." In Twenty-Seventh International Joint Conference on Artificial Intelligence {IJCAI-18}. California: International Joint Conferences on Artificial Intelligence Organization, 2018. http://dx.doi.org/10.24963/ijcai.2018/788.

Повний текст джерела
Анотація:
In this work, we explain how Assumption-Based Argumentation (ABA) is subsumed by Logic Programming (LP). The translation from ABA to LP (with a few restrictions on the ABA framework) results in a normal logic program whose semantics coincide with the semantics of the underlying ABA framework. Although the precise technicalities are beyond the current extended abstract (these can be found in the associated full paper) we provide a number of examples to illustrate the general idea.
Стилі APA, Harvard, Vancouver, ISO та ін.

Звіти організацій з теми "ABA1"

1

Olszewski, Neil, and David Weiss. DELLA, ABA and drought tolerance in tomato. United States Department of Agriculture, January 2015. http://dx.doi.org/10.32747/2015.7600021.bard.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
2

Hu, Vincent C., David Ferraiolo, Rick Kuhn, Adam Schnitzer, Kenneth Sandlin, Robert Miller, and Karen Scarfone. Guide to Attribute Based Access Control (ABAC) Definition and Considerations. National Institute of Standards and Technology, January 2014. http://dx.doi.org/10.6028/nist.sp.800-162.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
3

Virues-Ortega, J., T. Carrasco-Jiménez, C. Dempsey, M. X. Froján-Parga, O. Garcia-Leal, E. Freixa i Baque, and A. Zaragoza-Scherman. Glosario español-inglés de términos conductuales de ABA España. ABA España, February 2019. http://dx.doi.org/10.26741/glos.2019.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
4

Ferraiolo, David F., Ramaswamy Chandramouli, Vincent C. Hu, and D. Richard R. Kuhn. A Comparison of Attribute Based Access Control (ABAC) Standards for Data Service Applications. National Institute of Standards and Technology, October 2016. http://dx.doi.org/10.6028/nist.sp.800-178.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
5

Mosquna, Assaf, and Sean Cutler. Systematic analyses of the roles of Solanum Lycopersicum ABA receptors in environmental stress and development. United States Department of Agriculture, January 2016. http://dx.doi.org/10.32747/2016.7604266.bard.

Повний текст джерела
Анотація:
Drought and other abiotic stresses have major negative effects on agricultural productivity. The plant hormone abscisic acid (ABA) regulates many responses to environmental stresses and can be used to improve crop performance under stress. ABA levels rise in response to diverse abiotic stresses to coordinate physiological and metabolic responses that help plants survive stressful environments. In all land plants, ABA receptors are responsible for initiating a signaling cascade that leads to stomata closure, growth arrest and large-scale changes in transcript levels required for stress tolerance. We wanted to test the meaning of root derived ABA signaling in drying soil on water balance. To this end we generated transgenic tomato lines in which ABA signaling is initiated by a synthetic agonist- mandipropamid. Initial study using a Series of grafting experiments indicate that that root ABA signaling has no effect on the immediate regulation of stomata aperture. Once concluded, these experiments will enable us to systematically dissect the physiological role of root-shoot interaction in maintaining the water balance in plants and provide new tools for targeted improvement of abiotic stress tolerance in crop plants.
Стилі APA, Harvard, Vancouver, ISO та ін.
6

Liu, Xuan, and John Cogan. Stimulation of p53-Dependent Transciption by the Growth Suppressor, c-Ab1. Fort Belvoir, VA: Defense Technical Information Center, July 2000. http://dx.doi.org/10.21236/ada383345.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
7

Kotula, Leszek, and Xiaoling Xiong. Mechanisms of Abnormal Growth Regulation in Prostatic Adenocarcinoma Using Abi1/Hssh3bp1 Conditional Knockout Mouse Model. Fort Belvoir, VA: Defense Technical Information Center, June 2009. http://dx.doi.org/10.21236/ada508242.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
8

Yalovsky, Shaul, and Julian Schroeder. The function of protein farnesylation in early events of ABA signal transduction in stomatal guard cells of Arabidopsis. United States Department of Agriculture, January 2002. http://dx.doi.org/10.32747/2002.7695873.bard.

Повний текст джерела
Анотація:
Loss of function mutations in the farnesyltransferase β subunit gene ERA1 (enhanced response to abscisic acid), cause abscisic acid hypersensitivity in seedlings and in guard cells. This results in slowed water loss of plants in response to drought. Farnesyltransferase (PFT) catalyses the attachment of the 15-carbon isoprenoid farnesyl to conserved cysteine residues located in a conserved C-terminal domain designated CaaX box. PFT is a heterodimeric protein comprised of an a and b sununits. The a subunit is shared between PFT and geranylgeranyltransferase-I (PGGTI) which catalyses the attachemt of the 20-carbon isoprenoid geranylgeranyl to CaaX box proteins in which the last amino acid is almost always leucine and in addition have a polybasic domain proximal to the CaaL box. Preliminary data presented in the proposal showed that increased cytoplasmic Ca2+ concentration in stomal guard cells in response to non-inductive ABA treatements. The goals set in the proposal were to characterize better how PFT (ERA1) affects ABA induced Ca2+ concentrations in guard cells and to identify putative CaaX box proteins which function as negative regulators of ABA signaling and which function is compromised in era1 mutant plants. To achieve these goals we proposed to use camelion Ca2+ sensor protein, high throughput genomic to identify the guard cell transcriptome and test prenylation of candidate proteins. We also proposed to focus our efforts of RAC small GTPases which are prenylated proteins which function in signaling. Our results show that farnesyltransferaseprenylates protein/s that act between the points of ABA perception and the activation of plasma membrane calcium influx channels. A RAC protein designated AtRAC8/AtRop10 also acts in negative regulation of ABA signaling. However, we discovered that this protein is palmitoylated and not prenylated although it contains a C-terminal CXXX motif. We further discovered a unique C-terminal sequence motif required for membrane targeting of palmitoylatedRACs and showed that their function is prenylation independent. A GC/MS based method for expression in plants, purification and analysis of prenyl group was developed. This method would allow highly reliable identification of prenylated protein. Mutants in the shared α subunit of PFT and PGGT-I was identified and characterized and was shown to be ABA hypersensitive but less than era1. This suggested that PFT and PGGT-I have opposing functions in ABA signaling. Our results enhanced the understanding of the role of protein prenylation in ABA signaling and drought resistance in plants with the implications of developing drought resistant plants. The results of our studies were published 4 papers which acknowledge support from BARD.
Стилі APA, Harvard, Vancouver, ISO та ін.
9

Virues-Ortega, Javier, ed. Preventive Measures against COVID-19 for Therapist of Applied Behavior Analysis (ABA) Services. ABA España, June 2020. http://dx.doi.org/10.26741/covid19.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
10

Caerols Mateo, Raquel. La Gestión de la Comunicación en los Museos De Madrid: Auditoría de Relaciones Públicas, de Mónica Viñarás Abad. Revista Internacional de Relaciones Públicas, December 2012. http://dx.doi.org/10.5783/rirp-4-2012-14-303-306.

Повний текст джерела
Стилі APA, Harvard, Vancouver, ISO та ін.
Також вас можуть зацікавити розширені добірки на тему "ABA1" для конкретних типів джерел:
Статті в журналах Дисертації Книги
Ми пропонуємо знижки на всі преміум-плани для авторів, чиї праці увійшли до тематичних добірок літератури. Зв'яжіться з нами, щоб отримати унікальний промокод!

До бібліографії