Добірка наукової літератури з теми "Zymosan opsonisé"

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Статті в журналах з теми "Zymosan opsonisé"

1

van der Bruggen, T., P. T. Kok, J. A. Raaijmakers, J. W. Lammers, and L. Koenderman. "Cooperation between Fc gamma receptor II and complement receptor type 3 during activation of platelet-activating factor release by cytokine-primed human eosinophils." Journal of Immunology 153, no. 6 (1994): 2729–35. http://dx.doi.org/10.4049/jimmunol.153.6.2729.

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Abstract After priming with cytokines, such as granulocyte-macrophage colony-stimulating factor (GM-CSF), IL-3, or IL-5, eosinophils are stimulated potently by opsonized particles like serum-treated zymosan (STZ), resulting in activation of the respiratory burst and production of lipid mediators, such as platelet-activating factor (PAF) and leukotriene C4 (LTC4). In the present study, the role of the opsonin receptors Fc gamma RII and CR3 during both STZ-induced activation of the respiratory burst and PAF release by human eosinophils was investigated. Inhibition studies with blocking mAbs (alp
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2

Sahlin, Herman, and Håkan Nygren. "Cytotoxicity Testing of Wound-Dressing Materials." Alternatives to Laboratory Animals 29, no. 3 (2001): 269–75. http://dx.doi.org/10.1177/026119290102900319.

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A method was developed for testing the cytotoxicity of various bandage-like wound dressings and gel wound dressings. In this method, the ability of human polymor-phonuclear neutrophils (PMNs) to initiate a respiratory burst after exposure to the various wound dressings is used as a marker of cytotoxicity. Luminol-amplified chemiluminescence stimulated with opsonised zymosan or phorbol 12-myristate 13-acetate (PMA) is used to measure the degree of activation of the respiratory burst, i.e. the NADPH oxidase activity, after exposure to wound dressings. Opsonised zymosan (material from yeast cell
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3

Le Cabec, Véronique, Carine Cols, and Isabelle Maridonneau-Parini. "Nonopsonic Phagocytosis of Zymosan and Mycobacterium kansasii by CR3 (CD11b/CD18) Involves Distinct Molecular Determinants and Is or Is Not Coupled with NADPH Oxidase Activation." Infection and Immunity 68, no. 8 (2000): 4736–45. http://dx.doi.org/10.1128/iai.68.8.4736-4745.2000.

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ABSTRACT Complement receptor type 3 (CR3) was initially described as an opsonic receptor. Subsequently, CR3-mediated lectin-sugar recognition mechanisms have been shown to play a major role in the nonopsonic phagocytosis of several pathogens, among them Mycobacterium tuberculosis. Little is known about the binding and signal transduction mechanisms operating during nonopsonic ingestion through CR3 of different microorganisms. In the present study, we used CHO cells stably transfected with CR3 to show that CR3 was able to mediate internalization of zymosan and pathogenic mycobacteria (Mycobacte
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4

Klebanoff, S. J., M. A. Vadas, J. M. Harlan, et al. "Stimulation of neutrophils by tumor necrosis factor." Journal of Immunology 136, no. 11 (1986): 4220–25. http://dx.doi.org/10.4049/jimmunol.136.11.4220.

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Abstract Human recombinant tumor necrosis factor (TNF) was shown to be a weak direct stimulus of the neutrophil respiratory burst and degranulation. The stimulation, as measured by iodination, H2O2 production, and lysozyme release, was considerably increased by the presence of unopsonized zymosan in the reaction mixture, an effect which was associated with the increased ingestion of the zymosan. TNF does not act as an opsonin but, rather, reacts with the neutrophil to increase its phagocytic activity. TNF-dependent phagocytosis, as measured indirectly by iodination, is inhibited by monoclonal
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5

ROUIS, Mustapha, Fabienne NIGON, Thomas L. EGGERMAN, H. Bryan BREWER, and M. John CHAPMAN. "Apolipoprotein E expression by human-monocyte-derived macrophages. Modulation by opsonised zymosan and cholesterol." European Journal of Biochemistry 189, no. 2 (1990): 447–53. http://dx.doi.org/10.1111/j.1432-1033.1990.tb15509.x.

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6

Nair, P. K. Raveendran, Steven J. Melnick, Ziad A. Khatib, Reshma Ramachandran, Enrique A. Escalon та Cheppail Ramachandran. "Mechanism of Immune System Activation by (1,4)-α-D-glucan Isolated from Tinospora cordifolia in Macrophages." Blood 108, № 11 (2006): 3833. http://dx.doi.org/10.1182/blood.v108.11.3833.3833.

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Анотація:
Abstract We have characterized and reported the immunostimulating properties of a novel polysaccharide - (1,4)-α-D-glucan (RR1)- isolated from the medicinal plant Tinospora cordifolia [23]. This novel glucan is water soluble and having (1, 4)-α-D-glycosidic linkages in the main chain and (1, 6)-α-D-glycosidic linked side chains at an interval of 6, 7 glucose units. The signaling mechanism of the novel (1,4)-a-D-glucan (RR1) was investigated in macrophages to evaluate its immunostimulating properties. When RAW264.7 macrophages were incubated with RR1 at 4°C, the novel glucan inhibited the phago
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7

Melnick, D. A., W. M. Nauseef, S. D. Markowitz, J. P. Gardner, and H. L. Malech. "Biochemical analysis and subcellular localization of a neutrophil-specific antigen, PMN-7, involved in the respiratory burst." Journal of Immunology 134, no. 5 (1985): 3346–55. http://dx.doi.org/10.4049/jimmunol.134.5.3346.

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Анотація:
Abstract The adherence of serum-opsonized yeast to neutrophils results in phagocytosis of these particulate stimuli and activation of the respiratory burst. Both events are mediated or modulated in part by the surface receptors for IgG and complement. The link between the binding of complex particulate stimuli to the cell surface, and the triggering of these neutrophil functions, is not completely understood. We have previously described an anti-human neutrophil, murine monoclonal antibody PMN7C3, which specifically inhibits the respiratory burst of neutrophils stimulated with serum-opsonized
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8

Diniz, S. N., R. Nomizo, P. S. Cisalpino, et al. "PTX3 function as an opsonin for the dectin-1-dependent internalization of zymosan by macrophages." Journal of Leukocyte Biology 75, no. 4 (2004): 649–56. http://dx.doi.org/10.1189/jlb.0803371.

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9

Worku, Mulumebet, Max J. Paape, Andrea Di Carlo, Marcus E. Kehrli, and Warren W. Marquardt. "Complement component C3b and immunoglobulin Fc receptors on neutrophils from calves with leukocyte adhesion deficiency." American Journal of Veterinary Research 56, no. 4 (1995): 435–39. http://dx.doi.org/10.2460/ajvr.1995.56.04.435.

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SUMMARY Receptors for opsonins, such as complement component C3b (CR1) and immunoglobulins, Fc receptors, interact with adhesion glycoproteins in mediating immune functions. Defects in expression of the adhesion glycoproteins CD11/CD18 results in severely hampered in vitro and in vivo adherence- related functions of leukocytes. Little is known regarding the effect of leukocyte adhesion deficiency (lad) on ligand binding and receptor expression. We investigated the binding and expression of CR1 and Fc receptors by bovine neutrophils isolated from dairy calves suffering from lad, compared with c
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Bartoskova, A., P. Ondrackova, L. Leva, et al. "The effects of in vitro exposure to progesterone and estradiol-17β on the activity of canine neutrophils." Veterinární Medicína 59, No. 4 (2014): 202–9. http://dx.doi.org/10.17221/7481-vetmed.

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To date, only limited information about the influence of ovarian hormones on canine immune system cells has been available. The study investigated the in vitro influence of progesterone and estradiol-17β on the activity of canine neutrophils. Treatment of cells by both hormones led to a significant decrease in phagocytosis-induced oxidative burst as detected using luminometry after stimulation with opsonised zymosan. The increase in oxidative burst, not connected with phagocytosis, was recorded after stimulation with a soluble stimulator. Using flow cytometry, the tendency of both hor
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Дисертації з теми "Zymosan opsonisé"

1

Liu, Jie. "Activation of the phagocyte NADPH oxidase (NOX2) in human neutrophils : study of p47phox phosphorylation during phagocytosis." Electronic Thesis or Diss., Université Paris Cité, 2024. http://www.theses.fr/2024UNIP5213.

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Анотація:
Les polymorphonucléaires neutrophiles, sont des acteurs essentiels du système immunitaire inné, responsables de la phagocytose des agents pathogènes. Lors de la phagocytose, les neutrophiles produisent des quantités substantielles d'anion superoxyde, qui génèrent ensuite des espèces réactives de l'oxygène (ERO) telles que le peroxyde d'hydrogène, les radicaux hydroxyles et l'acide hypochloreux, indispensables à la destruction des microbes. L'enzyme responsable de la production de superoxyde est le complexe NADPH oxydase, composé de protéines membranaires (gp91phox/NOX2 et p22phox) et de protéi
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