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Добірка наукової літератури з теми "VO2 slow component"

Автор: Grafiati

Опубліковано: 11 березня 2023

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Статті в журналах з теми "VO2 slow component"

1

Fontana, Federico Y., Giorgia Spigolon, and Silvia Pogliaghi. "VO2 Slow Component." Medicine & Science in Sports & Exercise 48 (May 2016): 200. http://dx.doi.org/10.1249/01.mss.0000485602.73906.88.

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2

Wasserman, K. "Coupling of external to cellular respiration during exercise: the wisdom of the body revisited." American Journal of Physiology-Endocrinology and Metabolism 266, no. 4 (1994): E519—E539. http://dx.doi.org/10.1152/ajpendo.1994.266.4.e519.

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The changes in cellular respiration needed to increase energy output during exercise are intimately and predictably linked to external respiration through the circulation. This review addresses the mechanisms by which lactate accumulation might influence O2 uptake (VO2) and CO2 output (VCO2) kinetics. Respiratory homeostasis (a steady state with respect to VO2 and VCO2) is achieved by 3-4 min for work rates not associated with an increase in arterial lactate. When blood lactate increases significantly above rest for constant work rate exercise, VO2 characteristically increases past 3 min (slow

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3

Colosio, Alessandro L., Kevin Caen, Jan G. Bourgois, Jan Boone, and Silvia Pogliaghi. "Bioenergetics of the VO2 slow component between exercise intensity domains." Pflügers Archiv - European Journal of Physiology 472, no. 10 (2020): 1447–56. http://dx.doi.org/10.1007/s00424-020-02437-7.

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Abstract During heavy and severe constant-load exercise, VO2 displays a slow component (VO2sc) typically interpreted as a loss of efficiency of locomotion. In the ongoing debate on the underpinnings of the VO2sc, recent studies suggested that VO2sc could be attributed to a prolonged shift in energetic sources rather than loss of efficiency. We tested the hypothesis that the total cost of cycling, accounting for aerobic and anaerobic energy sources, is affected by time during metabolic transitions in different intensity domains. Eight active men performed 3 constant load trials of 3, 6, and 9 m

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4

Womack, C. J., S. E. Davis, J. L. Blumer, E. Barrett, A. L. Weltman, and G. A. Gaesser. "Slow component of O2 uptake during heavy exercise: adaptation to endurance training." Journal of Applied Physiology 79, no. 3 (1995): 838–45. http://dx.doi.org/10.1152/jappl.1995.79.3.838.

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Seven untrained male subjects [age 25.6 +/- 1.5 (SE) yr, peak O2 uptake (VO2) 3.20 +/- 0.19 l/min] trained on a cycle ergometer 4 days/wk for 6 wk, with the absolute training workload held constant for the duration of training. Before and at the end of each week of training, the subjects performed 20 min of constant-power exercise at a power designed to elicit a pronounced slow component of VO2 (end-exercise VO2-VO2 at minute 3 of exercise) in the pretraining session. An additional 20-min exercise bout was performed after training at this same absolute power output during which epinephrine (Ep

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5

Heck, Kristen L., Jeffrey A. Potteiger, Karen L. Nau, and Jan M. Schroeder. "Sodium Bicarbonate Ingestion Does Not Attenuate the VO2 Slow Component during Constant-Load Exercise." International Journal of Sport Nutrition 8, no. 1 (1998): 60–69. http://dx.doi.org/10.1123/ijsn.8.1.60.

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We examined the effects of sodium bicarbonate ingestion on the VO2 slow component during constant-load exercise. Twelve physically active males performed two 30-min cycling trials at an intensity above the lactate threshold. Subjects ingested either sodium bicarbonate (BIC) or placebo (PLC) in a randomized. counterbalanced order. Arterialized capillary blood samples were analyzed for pH, bicarbonate concentration ([HCO3−), and lactate concentration ([La]). Expired gas samples were analyzed for oxygen consumption (VO2). The VO2 slow component was defined as the change in VO2 from Minutes 3 and

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6

Billat, V. L. "VO2 slow component and performance in endurance sports." British Journal of Sports Medicine 34, no. 2 (2000): 83–85. http://dx.doi.org/10.1136/bjsm.34.2.83.

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7

Lucia, A. "The slow component of VO2 in professional cyclists." British Journal of Sports Medicine 34, no. 5 (2000): 367–74. http://dx.doi.org/10.1136/bjsm.34.5.367.

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8

Jones, A. "VO2 slow component and performance in endurance sports." British Journal of Sports Medicine 34, no. 6 (2000): 473. http://dx.doi.org/10.1136/bjsm.34.6.473.

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9

Poole, D. C., W. Schaffartzik, D. R. Knight, et al. "Contribution of excising legs to the slow component of oxygen uptake kinetics in humans." Journal of Applied Physiology 71, no. 4 (1991): 1245–60. http://dx.doi.org/10.1152/jappl.1991.71.4.1245.

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Анотація:

Rates of performing work that engender a sustained lactic acidosis evidence a slow component of pulmonary O2 uptake (VO2) kinetics. This slow component delays or obviates the attainment of a stable VO2 and elevates VO2 above that predicted from considerations of work rate. The mechanistic basis for this slow component is obscure. Competing hypotheses depend on its origin within either the exercising limbs or the rest of the body. To resolve this question, six healthy males performed light nonfatiguing [approximately 50% maximal O2 uptake (VO2max)] and severe fatiguing cycle ergometry, and simu

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10

Jones, Andrew M., and Mark Burnley. "Oxygen Uptake Kinetics: An Underappreciated Determinant of Exercise Performance." International Journal of Sports Physiology and Performance 4, no. 4 (2009): 524–32. http://dx.doi.org/10.1123/ijspp.4.4.524.

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The rate at which VO2 adjusts to the new energy demand following the onset of exercise strongly influences the magnitude of the “O2 defcit” incurred and thus the extent to which muscle and systemic homeostasis is perturbed. Moreover, during continuous high-intensity exercise, there is a progressive loss of muscle contractile efficiency, which is reflected in a “slow component” increase in VO2. The factors that dictate the characteristics of these fast and slow phases of the dynamic response of VO2 following a step change in energy turnover remain obscure. However, it is clear that these featur

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