Добірка наукової літератури з теми "Virus de poisson"
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Статті в журналах з теми "Virus de poisson"
Widyaningsih, Yekti, and Rugun Ivana. "WEIBULL-POISSON DISTRIBUTION AND THEIR APPLICATION TO SYSTEMATIC PARALLEL RISK." BAREKENG: Jurnal Ilmu Matematika dan Terapan 18, no. 1 (March 13, 2024): 0029–42. http://dx.doi.org/10.30598/barekengvol18iss1pp0053-0064.
Повний текст джерелаNourinejhad Zarghani, Shaheen, Mehran Monavari, Amin Nourinejhad Zarghani, Sahar Nouri, Jens Ehlers, Joachim Hamacher, Martina Bandte, and Carmen Büttner. "Quantifying Plant Viruses: Evolution from Bioassay to Infectivity Dilution Curves along the Model of Tobamoviruses." Viruses 16, no. 3 (March 12, 2024): 440. http://dx.doi.org/10.3390/v16030440.
Повний текст джерелаPakes, Anthony G., and A. C. Trajstman. "Some properties of continuous-state branching processes, with applications to Bartoszyński’s virus model." Advances in Applied Probability 17, no. 1 (March 1985): 23–41. http://dx.doi.org/10.2307/1427050.
Повний текст джерелаPakes, Anthony G., and A. C. Trajstman. "Some properties of continuous-state branching processes, with applications to Bartoszyński’s virus model." Advances in Applied Probability 17, no. 01 (March 1985): 23–41. http://dx.doi.org/10.1017/s0001867800014634.
Повний текст джерелаManeking, Faranika Deysi G., Deiby Tineke Salaki, and Djoni Hatidja. "Model Regresi Poisson Tergeneralisasi untuk Anak Gizi Buruk di Sulawesi Utara." JURNAL ILMIAH SAINS 20, no. 2 (October 31, 2020): 141. http://dx.doi.org/10.35799/jis.20.2.2020.29133.
Повний текст джерелаAkbarzadeh Baghban, Alireza, Asma Pourhoseingholi, Farid Zayeri, Ali Akbar Jafari, and Seyed Moayed Alavian. "Application of Zero-Inflated Poisson Mixed Models in Prognostic Factors of Hepatitis C." BioMed Research International 2013 (2013): 1–5. http://dx.doi.org/10.1155/2013/403151.
Повний текст джерелаSeghier, Fatma Zohra, Halim Zeghdoudi, and Vinoth Raman. "A Novel Discrete Distribution: Properties and Application Using Nipah Virus Infection Data Set." European Journal of Statistics 3 (January 9, 2022): 3. http://dx.doi.org/10.28924/ada/stat.3.3.
Повний текст джерелаSeghier, Fatma Zohra, Halim Zeghdoudi, and Abbes Benchaabane. "A Size-Biased Poisson-Gamma Lindley Distribution with Application." European Journal of Statistics 1, no. 1 (October 22, 2021): 132–47. http://dx.doi.org/10.28924/ada/stat.1.132.
Повний текст джерелаNourinejhad Zarghani, Shaheen, Mehran Monavari, Jens Ehlers, Joachim Hamacher, Carmen Büttner, and Martina Bandte. "Comparison of Models for Quantification of Tomato Brown Rugose Fruit Virus Based on a Bioassay Using a Local Lesion Host." Plants 11, no. 24 (December 9, 2022): 3443. http://dx.doi.org/10.3390/plants11243443.
Повний текст джерелаZuhrat, Lily, Dodi Devianto, and Izzati Rahmi HG. "Pemodelan Jumlah Kasus DBD Yang Meninggal Di Kota Padang Dengan Menggunakan Regresi Poisson." Jurnal Matematika UNAND 4, no. 4 (July 26, 2019): 57. http://dx.doi.org/10.25077/jmu.4.4.57-64.2015.
Повний текст джерелаДисертації з теми "Virus de poisson"
Lacasa, Michel. "Le virus herpétique du poisson-chat : structure et clonage du génome, transcription in vivo." Paris 6, 1986. http://www.theses.fr/1986PA066233.
Повний текст джерелаLacasa, Michel. "Le Virus herpétique du Poisson-chat structure et clonage du génome, transcription in vivo." Grenoble 2 : ANRT, 1986. http://catalogue.bnf.fr/ark:/12148/cb375988602.
Повний текст джерелаGordon, John Carroll. "A new computationally facile approximation of electrostatic potential suitable for macromolecules." Thesis, Virginia Tech, 2007. http://hdl.handle.net/10919/31216.
Повний текст джерелаMaster of Science
Nzonza, Angella. "Utilisation d'un rhabdovirus de poisson pour la mise en place d'une nouvelle plateforme vaccinale : exemple du virus West Nile (VWN)." Paris 7, 2013. http://www.theses.fr/2013PA077208.
Повний текст джерелаWest Nile Virus (WNV) is an arbovirus that can cause disease in mammals including humans and horses. There is no specific treatment or vaccines for WNV in humans. Our study aims at developing a WNV vectored vaccine which consists in using a fish Novirhabdovirus vector: the Viral Hemorrhagic Septicemia virus (VHSV). VHSV replicates at temperatures lower than 20°C and is naturally inactivated at higher temperatures. A reverse genetics system has recently been developed for VHSV allowing the addition of genes in the viral genome and the generation of the respective recombinant viruses rVHSVs. We have generated rVHSV vectors bearing on the one hand the complete WNV envelope gene (EwNv) (rVHSV-EwNv and rVHSV-EwNΔtm) or deleted of his transmembrane domain and on the other hand, fragments encoding E subdomains (either domain III alone or fused to domain II) (rVHSV-DJIIwNv and rVHSV-DIIDIIIwNv, respectively) in the VHSV genome. With the objective to enhance the targeting of the EwNv protein or EwNv-derived domains to the surface of VHSV virions, Novirhabdovirus G-derived signal peptide and transmembrane domain (SPG and TMG) were fused to EwNv at its amino and carboxy termini, respectively. We demonstrated that both the EWNV and the DIIIwnv could be expressed at the viral surface of rVHSV upon addition of SPG. Constructs expressing EwNv fused to SPG protected mice against WNV lethal challenge and specifically rVHSV-SPGEwNv induced a neutralizing antibody response that correlated with protection. Surprisingly, rVHSV expressing EwNv-derived domain III or II and III were unable to protect mice against WNV challenge, although these domains were highly expressed at the viral surface
Passoni, Gabriella. "Unraveling viral encephalitis in vivo : dynamic imaging of neuro-invasion and neuro inflammation processes in the zebrafish." Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066644/document.
Повний текст джерелаThe zebrafish (Danio rerio) is an important model organism, particularly for studies of development and more recently host pathogen interactions. As opposed to other vertebrate model organisms, its optical clarity and ease of genetic manipulations allow to visualize highly dynamic cellular processes in vivo at the whole organism scale. These assets make the zebrafish a perfect model for the study of viral infections in vivo, such as those caused by neurotropic viruses. The aim of this project has been to gain insights in some of the interactions that determine encephalitis, by characterizing the neurotropic Sindbis virus (SINV). This Thesis project has consisted therefore in: 1) the development of a SINV infection model in zebrafish larvae, 2) the characterization of SINV neuroinvasion upon its inoculation in the bloodstream, thanks to the use of high resolution microscopy, 3) the study of SINV mechanism of entry in the CNS, 4) the characterization of the innate immune response, both at the whole organism and organ specific level. Thanks to the use of a SINV recombinant strain, engineered to express the green fluorescent protein “GFP” in infected cells upon viral replication, we have been able to follow the onset and the progression of the infection. We have suggested infection of peripheral neurons and subsequent axonal transport to the CNS as SINV entry mechanism. At the cellular level, we have identified neutrophils as the main IFN producing cells
Chaumont, Lise. "Functional study of key fish interferon-stimulated genes using an in vitro knock-out approach in fish cell lines : from comparative immunology to interest for vaccine production." Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASL038.
Повний текст джерелаIn jawed vertebrates, innate antiviral defenses are primarily based on type I interferons (IFNs). These master cytokines are secreted following virus recognition and induce the expression of hundreds of IFN-stimulated genes (ISGs). ISGs encode proteins with diverse functions, including enhancers of the type I IFN pathway and antiviral effectors, which all work towards establishing an antiviral state refractory to viral infection. Overall, the type I IFN system is well-conserved between mammals and fish but the ISGs repertoire is more diverse in fish, largely due to their complex evolutionary history and physiological specificities. Consequently, most mammalian ISGs have one or more orthologs in fish. However, it is still unclear whether fish ISGs are true functional homologs and their mechanisms of action remain to be explored in detail.In this context, my thesis aimed to functionally characterize two key fish ISGs, namely dsRNA-dependent protein kinase (pkr) and virus inhibitory protein endoplasmic reticulum-associated, interferon-inducible (viperin), by using an in vitro knock-out approach. In mammals, both proteins are primarily regarded as antiviral effectors: PKR is involved in host translation inhibition and apoptosis, while Viperin operates by generating antiviral ribonucleotides and modulating metabolic pathways exploited during viral life cycles. However, the extent to which these functions are conserved in fish remains largely unknown. The objectives of my thesis were articulated along three axes: (1) to develop and validate pkr-/- and viperin-/- fish cell lines using the CRISPR/Cas9 technology; (2) to functionally characterize these cell lines, in order to identify the mechanisms of action of fish PKR and Viperin and their role in regulating the type I IFN response through feedback loops; (3) to assess their permissivity to viral infections and their ability to produce viral particles at higher yields than their wild-type counterparts.Using complementary overexpression and knockout approaches, I first studied the molecular mechanisms of action of PKR in Chinook salmon (Oncorhynchus tshawytscha) CHSE-EC cells. Our findings show that salmonid PKR has conserved molecular functions, including apoptosis activation and inhibition of host protein synthesis. However, endogenous PKR did not play a major antiviral role during viral hemorrhagic septicemia virus (VHSV) infection. In fact, our results suggest that VHSV has evolved strategies to subvert PKR antiviral action, by limiting early induction of pkr expression, evading PKR-mediated translational arrest and taking advantage of PKR-mediated apoptosis at a late infection stage to favor viral spread.In parallel, we conducted a comparative RNA-seq analysis of the viperin-/- and wild-type fathead minnow (Pimephales promelas) EPC-EC cell lines with or without stimulation with recombinant type I IFN to have a global overview of the regulatory role of fish Viperin. Our data show that cyprinid Viperin is not involved in the regulation of the canonical type I IFN but negatively regulates specific inflammatory pathways. Our analysis further indicates that it plays a regulatory role in other metabolic processes, even in non-induced conditions, including extracellular matrix organization, cell adhesion and one carbon metabolism.During the development process of initial pkr-/- cell lines, two CHSE-EC cell lines were found to be persistently infected with infectious pancreatic necrosis virus (IPNV), presumably due to inadvertent contamination. I set out to characterize these persistently IPNV-infected cell lines over the course of 40 passages. A striking feature in both cell lines was the periodic oscillatory pattern of extracellular titers and intracellular viral RNA levels over passages. We further showed that the type I IFN response was not triggered during persistent infection, suggesting that persistent IPNV is able to evade the host innate immune response
Passoni, Gabriella. "Unraveling viral encephalitis in vivo : dynamic imaging of neuro-invasion and neuro inflammation processes in the zebrafish." Electronic Thesis or Diss., Paris 6, 2015. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2015PA066644.pdf.
Повний текст джерелаThe zebrafish (Danio rerio) is an important model organism, particularly for studies of development and more recently host pathogen interactions. As opposed to other vertebrate model organisms, its optical clarity and ease of genetic manipulations allow to visualize highly dynamic cellular processes in vivo at the whole organism scale. These assets make the zebrafish a perfect model for the study of viral infections in vivo, such as those caused by neurotropic viruses. The aim of this project has been to gain insights in some of the interactions that determine encephalitis, by characterizing the neurotropic Sindbis virus (SINV). This Thesis project has consisted therefore in: 1) the development of a SINV infection model in zebrafish larvae, 2) the characterization of SINV neuroinvasion upon its inoculation in the bloodstream, thanks to the use of high resolution microscopy, 3) the study of SINV mechanism of entry in the CNS, 4) the characterization of the innate immune response, both at the whole organism and organ specific level. Thanks to the use of a SINV recombinant strain, engineered to express the green fluorescent protein “GFP” in infected cells upon viral replication, we have been able to follow the onset and the progression of the infection. We have suggested infection of peripheral neurons and subsequent axonal transport to the CNS as SINV entry mechanism. At the cellular level, we have identified neutrophils as the main IFN producing cells
Quesnel-Vallières, Mathieu. "Développement de lignées de poissons zébrés transgéniques pour l'étude du rôle de la protéine F dans la pathogenèse de l'hépatite C." Thèse, 2010. http://hdl.handle.net/1866/4144.
Повний текст джерелаHepatitis C virus (HCV) is a major cause of liver steatosis, fibrosis and hepatocellular carcinoma. HCV F protein is expressed from an alternative reading frame within the Core sequence. F protein was discovered after many of the pathogenic determinants of HCV had been associated with the effects of Core. Hence, we propose that a part of the functions attributed to Core result from the activity of the F protein. We produced and selected 19, 21 and 36 transgenic zebrafish (Danio rerio) to give rise to 3 independent lines expressing different versions of the F protein. Of these founders, 9, 11 and 11 were raised to maturity and will be bred to generate stable transgenic lines. Characterizing the phenotype of these transgenic fish will help determine the precise role of the F protein in the pathogenesis of hepatitis C.
Pagliuzza, Amélie. "Étude de l’implication de la protéine F du virus de l’hépatite C dans le développement de pathologie hépatique chez deux lignées de poissons zébrés transgéniques." Thèse, 2012. http://hdl.handle.net/1866/9190.
Повний текст джерелаHepatitis C virus (HCV) core protein is thought to be responsible for the major pathogenic effects of HCV, including the development of fibrosis, steatosis, cirrhosis, and hepatocellular carcinoma. An alternate translational open reading frame exists in the core gene that allows the synthesis of another protein called ARFP (alternate reading frame protein) or F protein (frameshift), the role of which remains poorly understood. Since we cannot exclude the presence of F protein in most studies of core biological functions, it is possible that the roles attributed to core reflect the activity of ARFP. To determine the biological functions of F protein in hepatocytes and their influence on HCV-associated pathogenesis, we generated transgenic lines of zebrafish (Danio rerio) in which the liver fatty acid binding protein (L-FABP) promoter was used to direct liver-specific expression of two forms of ARFP (AF11opti and AUG26opti). The phenotype of F2 transgenic zebrafish was analyzed for morphological, histological and microscopic signs of liver-associated pathology. Our results demonstrated the implication of the HCV F protein in the development of hepatic steatosis in transgenic zebrafish liver but not fibrosis or oncogenesis. Identification of the cellular and molecular mechanisms underlying F protein-induced lipid accumulation will lead to a better understanding of the role of ARFP in HCV-associated pathology, which could lead to the development of novel antiviral strategies.
Книги з теми "Virus de poisson"
Copyright Paperback Collection (Library of Congress), ed. Poison. New York: Scholastic, 1997.
Знайти повний текст джерелаHoh, Diane. Poison. Bt Bound, 2003.
Знайти повний текст джерелаHoh, Diane. Poison (Med Center No. 6). Scholastic, 1997.
Знайти повний текст джерелаCyber Insecurity. Kent, WA, USA: Self-published, 2018.
Знайти повний текст джерелаAdams, Joseph 1756-1818. Observations on Morbid Poisons, Phagedaena, and Cancer: Containing a Comparative View of the Theories of Dr. Swediaur, John Hunter, Messrs. Foot, Moore and Bell, on the Laws of the Venereal Virus. and Also Some Preliminary Remarks on the Language And... Creative Media Partners, LLC, 2021.
Знайти повний текст джерелаЧастини книг з теми "Virus de poisson"
Mirchev, Miroslav, and Ljupco Kocarev. "Non-poisson Processes of Email Virus Propagation." In ICT Innovations 2009, 187–96. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-10781-8_20.
Повний текст джерелаAshcraft, Karen Lee. "Metaphor Matters: Poison or Pandemic?" In Wronged and Dangerous, 191–98. Policy Press, 2022. http://dx.doi.org/10.1332/policypress/9781529221398.003.0019.
Повний текст джерела"Diana Primrose (fl. 1630)." In Early Modern Women Poets (1520-1700), edited by Jane Stevenson Peter Davidson, Meg Bateman, Kate Chedgzoy, and Julie Saunders, 227. Oxford University PressOxford, 2001. http://dx.doi.org/10.1093/oso/9780198184263.003.0083.
Повний текст джерелаFalaki, Faezeh. "Citrus Virus and Viroid Diseases." In Advances in Citrus Production and Research [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.108578.
Повний текст джерелаKantagba, Yves M. K., Seydou Golo Barro, Serge L. W. Nikiema, and Pascal Staccini. "In Silico Screening of Phytocompounds from West African Traditional Medicine and Molecular Docking Targeting Dengue Virus Protein NS2B/NS3." In Studies in Health Technology and Informatics. IOS Press, 2024. http://dx.doi.org/10.3233/shti240492.
Повний текст джерелаТези доповідей конференцій з теми "Virus de poisson"
Nugraha, Jaka, and Welly Nur Armawati. "Modelling on human immunodeficiency virus case using Poisson bivariate regression." In 2ND INTERNATIONAL CONFERENCE ON CHEMISTRY, CHEMICAL PROCESS AND ENGINEERING (IC3PE). Author(s), 2018. http://dx.doi.org/10.1063/1.5065060.
Повний текст джерелаOkamura, Hiroyuki, Kazuya Tateishi, and Tadashi Dohi. "Statistical Inference of Computer Virus Propagation Using Non-Homogeneous Poisson Processes." In The 18th IEEE International Symposium on Software Reliability (ISSRE '07). IEEE, 2007. http://dx.doi.org/10.1109/issre.2007.28.
Повний текст джерелаRodionov, Nikolay I., and Shalabh C. Maroo. "Charge Distribution and Surface Properties of the Tobacco Mosaic Virus 4-nm Central-Pore." In ASME 2012 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/imece2012-87098.
Повний текст джерелаRakityanskaya, Irina Anisimovna, Tatiana Sergeevna Ryabova, Usmonali Adgaralievich Tajibaev, and Anastasia Andreevna Kalashnikova. "NEW APPROACHES IN THE TREATMENT OF CHRONIC VIRAL EPSTEIN-BARR INFECTION." In Themed collection of papers from Foreign intemational scientific conference «Joint innovation - joint development». Medical sciences . Part 2. Ьу НNRI «National development» in cooperation with PS of UA. June 2023. Crossref, 2023. http://dx.doi.org/10.37539/230629.2023.23.77.016.
Повний текст джерелаSchussnig, Richard, Douglas R. Q. Pacheco, Manfred Kaltenbacher, and Thomas-Peter Fries. "Efficient and Higher-Order Accurate Split-Step Methods for Generalised Newtonian Fluid Flow." In VI ECCOMAS Young Investigators Conference. València: Editorial Universitat Politècnica de València, 2021. http://dx.doi.org/10.4995/yic2021.2021.12217.
Повний текст джерела