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Статті в журналах з теми "Virulence Operon"
Kitten, Todd, Cindy L. Munro, Suzanne M. Michalek, and Francis L. Macrina. "Genetic Characterization of a Streptococcus mutans LraI Family Operon and Role in Virulence." Infection and Immunity 68, no. 8 (August 1, 2000): 4441–51. http://dx.doi.org/10.1128/iai.68.8.4441-4451.2000.
Повний текст джерелаSalvail, Hubert, Jeongjoon Choi, and Eduardo A. Groisman. "Differential synthesis of novel small protein times Salmonella virulence program." PLOS Genetics 18, no. 3 (March 4, 2022): e1010074. http://dx.doi.org/10.1371/journal.pgen.1010074.
Повний текст джерелаDay, William A., and Anthony T. Maurelli. "Shigella flexneri LuxS Quorum-Sensing System Modulates virB Expression but Is Not Essential for Virulence." Infection and Immunity 69, no. 1 (January 1, 2001): 15–23. http://dx.doi.org/10.1128/iai.69.1.15-23.2001.
Повний текст джерелаMiranda-CasoLuengo, Raúl, Aleksandra A. Miranda-CasoLuengo, Enda P. O’Connell, Ruth J. Fahey, Clara A. Boland, Jose A. Vázquez-Boland, and Wim G. Meijer. "The vapA co-expressed virulence plasmid gene vcgB (orf10) of the intracellular actinomycete Rhodococcus equi." Microbiology 157, no. 8 (August 1, 2011): 2357–68. http://dx.doi.org/10.1099/mic.0.049759-0.
Повний текст джерелаTobe, Toru, Tetsuya Hayashi, Chang-Gyun Han, Gary K. Schoolnik, Eiichi Ohtsubo, and Chihiro Sasakawa. "Complete DNA Sequence and Structural Analysis of the Enteropathogenic Escherichia coli Adherence Factor Plasmid." Infection and Immunity 67, no. 10 (October 1, 1999): 5455–62. http://dx.doi.org/10.1128/iai.67.10.5455-5462.1999.
Повний текст джерелаMole, Beth, Sohrab Habibi, Jeffery L. Dangl, and Sarah R. Grant. "Gluconate Metabolism Is Required for Virulence of the Soft-Rot Pathogen Pectobacterium carotovorum." Molecular Plant-Microbe Interactions® 23, no. 10 (October 2010): 1335–44. http://dx.doi.org/10.1094/mpmi-03-10-0067.
Повний текст джерелаVu-Khac, Hung, and Kurt W. Miller. "Regulation of Mannose Phosphotransferase System Permease and Virulence Gene Expression in Listeria monocytogenes by the EIItMan Transporter." Applied and Environmental Microbiology 75, no. 21 (September 4, 2009): 6671–78. http://dx.doi.org/10.1128/aem.01104-09.
Повний текст джерелаOnasanya, Amos, R. O. Onasanya, Abiodun A. Ojo, and B. O. Adewale. "Genetic Analysis and Molecular Identification of Virulence in Xanthomonas oryzae pv. oryzae Isolates." ISRN Molecular Biology 2013 (September 8, 2013): 1–8. http://dx.doi.org/10.1155/2013/160157.
Повний текст джерелаWright, Anita C., Jan L. Powell, James B. Kaper, and J. Glenn Morris. "Identification of a Group 1-Like Capsular Polysaccharide Operon for Vibrio vulnificus." Infection and Immunity 69, no. 11 (November 1, 2001): 6893–901. http://dx.doi.org/10.1128/iai.69.11.6893-6901.2001.
Повний текст джерелаO’Neill, Erinn M., Tatiana S. Mucyn, Jon B. Patteson, Omri M. Finkel, Eui-Hwan Chung, Joshua A. Baccile, Elisabetta Massolo, Frank C. Schroeder, Jeffery L. Dangl, and Bo Li. "Phevamine A, a small molecule that suppresses plant immune responses." Proceedings of the National Academy of Sciences 115, no. 41 (September 20, 2018): E9514—E9522. http://dx.doi.org/10.1073/pnas.1803779115.
Повний текст джерелаДисертації з теми "Virulence Operon"
Zorgani, Mohamed Amine. "Caractérisation des ARN régulateurs chez Streptococcus agalactiae." Thesis, Tours, 2016. http://www.theses.fr/2016TOUR3309.
Повний текст джерелаThe opportunistic pathogen group B Streptococcus (GBS) is the leading cause of neonatal infections. The aim of this work is the characterization of a 680 nt-long regulatory RNA, CetR (cell-envelope-targeting RNA). It modulates antimicrobial peptides (AMPs) resistance and virulence through posttranscriptional regulation of dltD mRNA which encodes a D-alanyl-lipoteichoic acid biosynthesis protein. Deletion of cetR leads to cell morphology changes, reduced biofilm formation and AMPs resistance. A 27 nt-long CetR-dltD interacting region is predicted in silico. Compensatory base pair exchanges in GBS demonstrate that CetR interacts directly with dltD mRNA and that disruption of this RNA pairing is sufficient to observe the CetR-associated phenotypes. By quantifying both mRNA and protein, we demonstrate that CetR enhances dltD translation and disruption of the CetR/dltD mRNA interaction results in a dramatic decrease in DltD protein. Moreover, using an infection murine model and quantifying bacterial survival in macrophages, we observe that both CetR and DltD are crucial for GBS virulence. Finally, we highlight CetR pleiotropic role in the expression of several moonlighting proteins and potential virulence factors. This regulatory RNA may play an important role in the ability of GBS to settle in its biotope and express its virulence factors
Franza, Thierry, and ANDRE KLIER. "Caracterisation d'un nouvel operon intervenant dans le transport et la biosynthese de la chrysobactine, siderophore implique dans la virulence d'une enterobacterie phytopathogene erwinia chrysanthemi." Paris 7, 1991. http://www.theses.fr/1991PA077233.
Повний текст джерелаToukoki, Chadia. "MTSR is a Dual Regulator that Controls Virulence Genes and Metabolic Functions in Addition to Metal Homeostasis in Group A Streptococcus." Digital Archive @ GSU, 2009. http://digitalarchive.gsu.edu/biology_diss/66.
Повний текст джерелаBernardi, Adilson César Abreu [UNESP]. "Estudo de amostras de Staphylococcus coagulase-negativa quanto a formação de biofilme." Universidade Estadual Paulista (UNESP), 2005. http://hdl.handle.net/11449/103991.
Повний текст джерелаUniversidade Estadual Paulista (UNESP)
Os Staphylococcus coagulase-negativa, particularmente, os Staphylococcus epidermidis são a causa mais freqüente de infecções relacionadas ao cateter por sua habilidade em aderir a uma superfície e entre si (aderência intercelular) formando biofilme em multicamadas sobre superfícies de polímeros. O objetivo do presente estudo foi avaliar cepas hospitalares de Staphylococcus coagulasenegativa isoladas de cateteres intravenosos, quanto à resistência a oxacilina, produção de slime, aderência ao poliestireno, habilidade de formar biofilme sobre superfícies abióticas (cateter esterilizado) e a presença de genes icaAD. Na presente pesquisa, a presença de icaA e icaD foi determinada pelo método PCR, em uma coleção de 27 amostras Staphylococcus coagulase-negativa (10 Staphylococcus epidermidis, 4 S. haemolyticus, 2 S. hominis, 2 S. lugdunensis, 1 S. saprophyticus, 1 S. schleiferi, 2 S. xylosus e 4 S. warneri). Os genes icaAD foram detectados em dez cepas S. epidermidis...
Coagulase-negative Staphylococcus, particularly, Staphylococcus epidermidis are frequent cause of infections associated with catheters and is attributed to the attachment ability on a surface and each other (intercellular adhesion) forming a multilayered biofilm on polymeric surfaces. The objective of the present study was to evaluate coagulase-negative Staphylococcus strains isolated from intravenous catheters by oxacillin resistance, slime production (qualitative method) and spectrophotometric assay (quantitative method), ability to form biofilm on abiotic surfaces (steriled catheter) and the presence of icaAD genes. In the present study icaA and icaD were determined by PCR method, in a collection of 27 coagulasenegative Staphylococcus (10 Staphylococcus epidermidis, 4 S. haemolyticus, 2 S. hominis, 2 S. lugdunensis, 1 S. saprophyticus, 1 S. schleiferi, 2 S. xylosus and 4 S. warneri). The icaA genes were detected in nine S. epidermidis and icaD in ten. The slime-producing ability was determined by culture on Congo red agar plates in which slime-producing strains formed black colonies in 10 S. epidermidis, 4 S.haemolyticus, 4 S. warneri, 2 S. xylosus and 1 S. chromogenes, while nonslimeformingones develop red colonies. The quantitative assay of coagulase-negative Staphylococcus was observed in 19 strains, including: 10 S. epidermidis, 3 S.haemolyticus, 3 S. warneri, 2 S. xylosus, 1 S. chromogenes. The ability of coagulasenegative Staphylococcus to form biofilm embedded in an amorphous substance wasobserved by scanning electronic microscope on abiotic surface in 10 S. epidermidis,3 S. haemolyticus, 2 S. hominis, 2 S. lugdunensis, 1 S. saprophyticus, 1 S. schleiferi,2 S. xylosus and 3 S. warneri. The oxacillin resistance was observed in 9 strains S.epidermidis, 3 S. haemolyticus, 3 S. warneri, 1 S. xylosus and 1 S. chromogenes. All strains of staphylococci were susceptible... (Complete abstract, click eletronic address below)
Silva, Filho Renato Geraldo da. "Produção de biofilme em amostras clínicas de S. epidermidis: influência de concentrações subinibitórias de antissépticos (etanol e clorexidina) e associação com potenciais marcadores de virulência." reponame:Repositório Institucional da FIOCRUZ, 2014. https://www.arca.fiocruz.br/handle/icict/10997.
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Fundação Oswaldo Cruz. Instituto Nacional de Controle de Qualidade em Saúde
S. epidermidis é o principal agente de infecções associadas a dispositivos médicos implantados, sendo sua habilidade para formar biofilme em superfícies inertes o fator determinante para a persistência desse micro-organismo. Neste estudo avaliamos 52 isolados clínicos desta espécie quanto à susceptibilidade a antimicrobianos, produção de biofilme/natureza química, presença de genes relacionados à virulência (atlE, capB,aap, embp, bhp, IS256 e IS257), e o efeito de concentrações subinibitórias (sub-CIMs) de etanol e clorexidina na produção de biofilme. Além disso, algumas das amostras biofilme-positivas foram estudadas quanto ao efeito de sub-CIMs destes antissépticos na expressão de icaA, icaR, sigB e sarA. Mais de 60% das amostras apresentaram resistência para ≥ 10 drogas e as amostras produtoras de biofilme mostraram, no geral, maior percentual de resistência a antimicrobianos. No teste em placa de microtitulação (MTP), 23 amostras foram produtoras de biofilme, sendo 14 de natureza polissacarídica, 8 proteica e 3 indeterminada. No teste em Ágar Vermelho Congo, somente amostras produtoras de biofilme polissacarídico apresentaram reação positiva. Genes do operon ica foram detectados em 23 isolados, sendo 17 destes classificados como produtores e 6 como não produtores de biofilme no MTP. A frequência dos outros genes relacionados à produção de biofilme foi: embp (69%), aap (29%) e bhp (12%), não sendo detectada correlação entre estes e a produção de biofilme do tipo PIA-independente. Os genes aap (29%) e IS256 (23%) mostraram correlações significativas com: produção de biofilme, presença de ica, perfil biofilme+/ica+, e produção de nível forte de biofilme. O gene IS256 foi ainda correlacionado significativamente com resistência a alguns antimicrobianos. Sub-CIMs de etanol (2 e/ou 4%) determinaram aumento na produção de biofilme em 15 das 17 amostras PIA-dependentes e nas 8 PIA-independentes, mas não induziram produção de biofilme em amostras originalmente não produtoras. Ao contrário do etanol, sub-CIMs de clorexidina não somente não induziram produção, como determinaram redução da produção de biofilme nas amostras biofilme-positivas. Nas amostras PIA-dependentes, o etanol (1%) acarretou aumento da expressão relativa de icaA e redução da expressão de icaR, além de aumento da expressão dos reguladores globais (sarA e sigB), enquanto a amostra PIA-independente mostrou redução na expressão destes reguladores globais. Ao contrário do etanol, a clorexidina (0,5 μg/mL) determinou aumento da expressão de icaR e redução de icaAnas amostras PIA-dependentes, além de redução na expressão de sarA e sigB na amostra PIA-independente. Os resultados indicaram que a produção de biofilme mostrou-se associada com alguns dos potenciais marcadores de virulência, sendo também evidenciada associação de alguns desses marcadores com resistência a certos antimicrobianos. As amostras PIA-dependentes foram prevalentes, destacando-se, porém, o encontro de número expressivo de amostras PIA-independentes. Os genes aap,embp e bhp não se mostraram correlacionados com a produção de biofilme proteico, indicando existência de outros mecanismos envolvidos na formação desse tipo de biofilme. Nas amostras PIA-dependentes, etanol e clorexidina mostraram efeitos opostos na expressão de icaA e icaR, corroborando dados fenotípicos previamente obtidos, e enfatizando a necessidade de ampliação do estudo da clorexidina, tendo em vista o potencial de aplicação prática deste achado.
S. epidermidis is the main agent of infections associated with implanted medical devices, being its ability to form biofilms on inert surfaces the determinant factor for the persistence of this microorganism. Fifth two clinical isolates of this species were evaluated for susceptibility to antimicrobials, biofilm production/chemical nature, presence of genes related to virulence (atlE, capB, aap, embp, bhp, IS256 andIS257), and the effect of subinibitory concentrations (sub-MICs) of ethanol and chlorhexidine in biofilm production. Moreover, some of biofilm-positive samples were studied for the effect of sub-MICs of these antiseptics in the expression of icaA, icaR, sigB and sarA. Over 60% of the samples showed resistance to ≥ 10 drugs and biofilm producers showed, in general, a higher percentage of antimicrobial resistance. In microtiter plate test (MTP), 23 strains were biofilm producers, being 4 of polysaccharide nature, 8 proteinaceous and 3 undetermined. In Congo Red Agar test, only biofilm polysaccharide producer strains showed a positive reaction. ica operon genes were detected in 23 isolates, being 17 of these classified as producers and 6 as non-biofilm producers in MTP. The frequency of other production-related biofilm genes was: embp (69%), aap (29%) and bhp (12%), no being detect a correlation between them and the production of PIA-independent biofilm. The aap (29%) and IS256 (23%) genes showed significant correlations with: biofilm production, presence of ica biofilm, biofilm+/ica+ profile, and strong level of production of biofilm. The IS256 gene was also significantly correlated with resistance to some antibiotics. Sub-MIC of ethanol (2 and / or 4%) led to an increase in biofilm production in 15 of 17 samples PIA-dependent and in the 8 PIA-independent, but did not induce biofilm production in not originally producing samples. Unlike ethanol, sub-MICs of chlorhexidine not only did not induce production as determined reduction of biofilm production in biofilm-positive samples. In PIA-dependent strains, ethanol (1%) caused an increase in the relative expression of icaAand reduced expression of icaR, in addition to increased expression of global regulators (sarA and sigB), while the PIA-independent strain showed reduction in the expression of these global regulators. Unlike ethanol, chlorhexidine (0.5 mg/mL) determined increased expression of icaR and reduction of icaA in PIA-dependent strains, besides a reduction in the expression of sarA and sigB in the PIA-independent strain. The results indicated that biofilm production was associated with some of potential virulence markers, and also evidenced some combination of these markers and resistance to certain antibiotics. The PIA-dependent strains were prevalent, highlighting, however, the encounter of significant number of PIA-independent strains. The aap, embp and bhp genes were not correlated with the production of proteinaceous biofilm, indicating the existence of other mechanisms involved in the formation of such biofilms. In PIA-dependent strains, ethanol and chlorhexidine showed opposite effects on the expression of icaA and icaR, corroborating phenotypic data previously obtained, and emphasizing the need to expand the study of chlorhexidine, in view of the potential of practical application of this finding.
Kamar, Rita. "Mécanismes de résistance aux peptides antimicrobiens chez Bacillus thuringiensis : rôle de dltX dans la D-alanylation des acides téichoïques." Electronic Thesis or Diss., Paris, AgroParisTech, 2014. http://www.theses.fr/2014AGPT0029.
Повний текст джерелаThe Bacillus cereus pathogenic spectrum ranges from strains used as probiotics to human-lethal strains (causing gastrointestinal disorders or local and severe systemic infections). However, prediction of the pathogenic potential of a strain remains difficult. In this work, we studied different phenotypes of colonization/adaptation to the host (adhesion, cytotoxicity, motility, biofilm formation, resistance to antimicrobial peptides and virulence), analyzing the correlation between these phenotypes and human disease in a collection of strains representative of the pathological diversity of B. cereus in humans. Statistical analysis revealed correlations between several phenotypes, and principal component analysis grouped the strains into two distinct subpopulations. We found that strains differed in pathogenic potential and that virulent strains could be differentiated from non-pathogenic strains. We believe that these findings will facilitate the identification of a phenotype or a combination of phenotypes of potential use in the development of effective prevention strategies and/or diagnostic tools for distinguishing between pathogenic and non-pathogenic B. cereus strains. Our result suggests that B. cereus is not an exclusively opportunistic pathogen and could instead be considered a real pathogen per se. However, virulence is a multifactorial phenomenon involving numerous factors from both the host and the invading pathogen. As cationic antimicrobial peptides (CAMPs) are the primary defense mechanism against invading organisms, virulence of pathogens like B. cereus requires bacterial resistance to such compounds. Consequently, many pathogens have developed resistance strategies involving the reduction of the cell envelope negative charge, thereby influencing the binding and interaction of these CAMPs. The incorporation of Dalanine esters into teichoic acids (TAs) represents one of the most common bacterial resistance mechanisms that depend on such charge modifications. That D-alanylation process is accomplished by the gene products of an operon containing five genes, dltXABCD, that is highly conserved among nearly all gram-positive bacteria. The small first ORF, dltX, encodes a protein of unknown function. The aim of the other part of this work was then to establish whether this protein is involved in the D-alanylation process in Bacillus thuringiensis. We therefore constructed an in frame deletion mutant of dltX, without affecting the expression of the other genes of the operon. The growth characteristics of the dltX mutant and those of the wild type strain were similar under standard in vitro conditions. However disruption of dltX drastically impaired the resistance of B. thuringiensis to CAMPs and significantly attenuated its virulence in two insect species: Galleria mellonella and Drosophila melanogaster. Moreover, HPLC studies showed the dltX mutant was devoid of D-alanine, and electrophoretic mobility measurements indicated that the cells carried a higher negative surface charge. Scanning electron microscopy experiments showed morphological alterations of these mutant bacteria, suggesting that depletion of D-alanine from TAs affects cell wall structure. Our findings suggest that DltX is essential for the incorporation of D-alanyl esters into teichoic acids. Moreover, we found that DltX does not affect the expression of the operon. We therefore conclude that dltX is translated into a functional protein that plays a direct biosynthetic, transport or addresser role. Altogether, our results clearly indicate that DltX plays a direct role in the resistance to CAMPs, thus contributing to the survival and virulence of B. thuringiensis in insects. The exact function of that protein remains to be elucidated. This work is the first report examining the involvement of dltX in the Dalanylation of TAs
Azuama, Onyedikachi Cecil. "Recherche de nouveaux actifs d'origine végétale contre le pathogène opportuniste de l'homme Pseudomonas aeruginosa Battling Pseudomonas aeruginosa virulence with natural plant bioactive compounds Membrane-interactive compounds from Pistacia lentiscus L. thwart Pseudomonas aeruginosa virulence Tackling Pseudomonas aeruginosa virulence by mulinane-like diterpenoids from Azorella atacamensis Pseudomonas aeruginosa virulence attenuation by extracts of Parastrephia terestiuscula, Baccharis grisebachii, Haplopappus rigidus medicinal plants of the Asteraceae family from the Atacama Desert area The absence of SigX results in impaired carbon metabolism and membrane fluidity in Pseudomonas aeruginosa Activation of the Cell Wall stress response in Pseudomonas aeruginosa infected by a Pf4 Phage Variant The temperature-regulation of Pseudomonas aeruginosa cmaX-cfrX-cmp-X operon reveals an intriguing molecular network involving the Sigma factors AlgU and SigX." Thesis, Normandie, 2020. http://www.theses.fr/2020NORMR077.
Повний текст джерелаAntimicrobial resistance has become a great challenge in therapeutic medicine so much so that the World health organization forecasts the possibility of a post-antibiotic era where minor injuries may lead to mortality. Pseudomonas aeruginosa is among the list of organisms that are highly resistant to conventional antibiotics, partly due to its broad genome, which facilitates the elaboration of virulence determinants and rapid adaptation to various environments, in addition to its inherent resistance mechanisms. In view of this, alternative measures of controlling microbial virulence activities using novel approaches that do not disturb its growth and viability, also known as anti-virulence strategy, are gaining wider attention. Since plants are repositories of several metabolites with chemical defense system against environmental pathogens, through ethnobotanical led studies, the effect of Pistacia lentiscus fruit extracts originating from Algeria and forty plant extracts originating from North-Chile were biologically and chemically evaluated with the aim of deciphering their anti-virulence effects against P. aeruginosa. Furthermore, this study tried to gain more insight into the bioactive compounds and possible mechanism of action. From the results obtained, selected plant extracts attenuated P. aeruginosa mainly pyocyanin activity and /or elastase and rhamnolipids virulence production which appears to be associated with the inhibition of quorum sensing activities and the alteration in membrane activities. The anti-virulence effect of the selected extracts (P. lentiscus, Azorella atacamensis, Baccharis grisebachii, Haplopappus rigidus and Parastrephia terestiucula) were also validated in biological models of infections where they mediated the toxicity of P. aeruginosa towards A549 human monolayer cells and/or Caenorhabditis elegans nematode. Interestingly, growth of the pathogen was not affected. Further chemical profiling of P. Lentiscus, and A atacamensis extracts revealed the presence of gingkolic acid and azorellane/mulinane diterpenoids as the putative bioactive compounds. Future studies intend to explore these extracts and their derived compounds on the potentiation of antibiotic activity in a panel of clinical strains. In general, this study sets the pace for the possible use of these plant extracts as adjuvants in treatment of P. aeruginosa infections
Tortuel, Damien. "Vers la compréhension du rôle de SigX dans la réponse au stress de l'enveloppe chez Pseudomonas aeruginosa Pf4 phage infection induces SOS and cell envelope stress responses in Pseudomonas aeruginosa Pf4 phage infection reduced virulence-associated phenotypes in Pseudomonas aeruginosa The temperature-regulation of Pseudomonas aeruginosa cmaX-cfrX-cmpX operon reveals an intriguing molecular network involving the sigma factors AlgU and SigX." Thesis, Normandie, 2020. http://www.theses.fr/2020NORMR002.
Повний текст джерелаPseudomonas aeruginosa is a very resistant opportunistic pathogen, for which it is critical to find new therapies. This bacterium easily adapts to its environment, through its large genome and proportion of regulators allowing a very fine regulation of its genes. The cell wall is the first barrier in contact with environment, and therefore represents a very important place ofexchange. The cell wall thus represents an interesting potential therapeutic target. SigX is an extracytoplasmic function sigma factor, responding to cell wall stresses detected by the bacterium, but the precise stimulus remains to discover. This sigma factor could be part of a new atypical signal transduction system that could couple SigX with a mechanosensitive channel. This work has led to the discovery of three new sigX activating conditions, which are Pf4 phage infection, loss of the CmpX mechanosensitive channel, and cold shock. These conditions seem to cause strong perturbations and an increase in membrane stiffness that could be the activating stimulus of SigX. This work has led to a better understanding of the activating condition of SigX, and to the clarification of the cellular and regulatory functions of the SigXCfrX-CmpX system members, highlighting the involvement of a mechanosensitive channel in the physiology of Pseudomonas aeruginosa
Lacroix, Jean-Marie Bohin Jean-Pierre. "Les glucanes périplasmiques osmorégulés (OPG) chez les entérobactéries de la régulation osmotique à la virulence /." Villeneuve d'Ascq : Université des sciences et technologies de Lille, 2007. https://iris.univ-lille1.fr/dspace/handle/1908/1000.
Повний текст джерелаN° d'ordre (Lille 1) : 510. Titre provenant de la page de titre du document numérisé. Bibliogr. p. 41-56.
Zhang, Honghao. "Modulation of virulence of Streptococcus pneumoniae by an operon in conjugative transposon Tn5252." 2006. http://digital.library.okstate.edu/etd/umi-okstate-2057.pdf.
Повний текст джерелаЧастини книг з теми "Virulence Operon"
Cai, Hongyan, Jiaying Yu, Qiu Li, Youyu Zhang, and Lixing Huang. "Research Progress on Virulence Factors of Vibrio alginolyticus: A Key Pathogenic Bacteria of Sepsis." In Sepsis - New Perspectives [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.108206.
Повний текст джерела"Infection." In Standards for the Management of Open Fractures, edited by Simon Eccles, Bob Handley, Umraz Khan, Iain McFadyen, Jagdeep Nanchahal, and Selvadurai Nayagam, 125–34. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198849360.003.0013.
Повний текст джерелаPruitt, Nicholas T. "Introduction." In Open Hearts, Closed Doors, 1–22. NYU Press, 2021. http://dx.doi.org/10.18574/nyu/9781479803545.003.0001.
Повний текст джерелаChinnici, Joseph P. "Diversities, Silence, and Open Conflict." In American Catholicism Transformed, 53–83. Oxford University Press, 2021. http://dx.doi.org/10.1093/oso/9780197573006.003.0003.
Повний текст джерелаCorredor, Mauricio, Juan David Patiño-Salazar, Diana Carolina Castaño, and Amalia Muñoz-Gómez. "The Pangenome of Pseudomonas aeruginosa." In Pseudomonas aeruginosa - New Perspectives and Applications [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.108187.
Повний текст джерелаЗвіти організацій з теми "Virulence Operon"
Elbaum, Michael, and Peter J. Christie. Type IV Secretion System of Agrobacterium tumefaciens: Components and Structures. United States Department of Agriculture, March 2013. http://dx.doi.org/10.32747/2013.7699848.bard.
Повний текст джерелаKapulnik, Yoram, and Donald A. Phillips. Isoflavonoid Regulation of Root Bacteria. United States Department of Agriculture, January 1996. http://dx.doi.org/10.32747/1996.7570561.bard.
Повний текст джерелаLindow, Steven E., Shulamit Manulis, Dan Zutra, and Dan Gaash. Evaluation of Strategies and Implementation of Biological Control of Fire Blight. United States Department of Agriculture, July 1993. http://dx.doi.org/10.32747/1993.7568106.bard.
Повний текст джерелаMcElwain, Terry F., Eugene Pipano, Guy H. Palmer, Varda Shkap, Stephn A. Hines, and Wendy C. Brown. Protection of Cattle against Babesiosis: Immunization against Babesia bovis with an Optimized RAP-1/Apical Complex Construct. United States Department of Agriculture, September 1999. http://dx.doi.org/10.32747/1999.7573063.bard.
Повний текст джерела