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Статті в журналах з теми "Ventricular synchronization"

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Автор: Grafiati

Опубліковано: 7 липня 2024

Оновлено: 7 липня 2024

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1

Takemoto, Yoshio, Toshiyuki Osaka, Eriko Yokoyama, Masahide Harada, Michio Takikawa, Tomoyuki Suzuki, Susumu Takeuchi, Atsushi Ito, and Itsuo Kodama. "Right ventricular midseptal pacing preserves left ventricular mechanical synchronization." Heart Rhythm 2, no. 5 (May 2005): S165. http://dx.doi.org/10.1016/j.hrthm.2005.02.516.

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2

Arias, Miguel A., Rafael Peinado, and José L. Merino. "Better ventricular synchronization via an accessory pathway." EP Europace 8, no. 8 (August 1, 2006): 616–17. http://dx.doi.org/10.1093/europace/eul076.

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3

Agadi, Smitha G., Helen Lonergan-Thomas, Sharon Brennan, Pamela Cianci, Mark Slaughter, and Marc Silver. "IMPACT OF LEFT VENTRICULAR ASSIST DEVICE DESTINATION THERAPY ON VENTRICULAR RE-SYNCHRONIZATION." Chest 128, no. 4 (October 2005): 281S. http://dx.doi.org/10.1378/chest.128.4_meetingabstracts.281s-b.

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4

Wu, Shengjie, Parikshit S. Sharma, and Weijian Huang. "Novel left ventricular cardiac synchronization: left ventricular septal pacing or left bundle branch pacing?" EP Europace 22, Supplement_2 (December 2020): ii10—ii18. http://dx.doi.org/10.1093/europace/euaa297.

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Abstract It is well recognized that a high burden of right ventricular pacing results in deleterious clinical outcomes over the long term. His bundle pacing can achieve optimal ventricular synchronization; however, relatively high pacing thresholds, low R-wave amplitudes, and the long-term performance have been concerns. Recently, left ventricular (LV) septal endocardium pacing (LVSP) has demonstrated improved acute haemodynamics. Another novel technique of intraseptal left bundle branch pacing (LBBP) via transvenous approach has been adopted rapidly and has demonstrated its feasibility and effectiveness. This article reviews the clinical application and differences between LVSP and LBBP. Compared with LVSP, LBBP has strict criteria for left conduction system capture and lead location. In addition to LV septal capture it also stimulates the proximal left bundle branch, resulting in rapid and physiological LV activation. With a uniformity and standardization of the implant procedure and definitions, it may be possible to achieve widespread application of this form of physiological pacing.

5

Ly, Cheng, and Seth H. Weinberg. "Automaticity in ventricular myocyte cell pairs with ephaptic and gap junction coupling." Chaos: An Interdisciplinary Journal of Nonlinear Science 32, no. 3 (March 2022): 033123. http://dx.doi.org/10.1063/5.0085291.

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Spontaneous electrical activity, or automaticity, in the heart is required for normal physiological function. However, irregular automaticity, in particular, originating from the ventricles, can trigger life-threatening cardiac arrhythmias. Thus, understanding mechanisms of automaticity and synchronization is critical. Recent work has proposed that excitable cells coupled via a shared narrow extracellular cleft can mediate coupling, i.e., ephaptic coupling, that promotes automaticity in cell pairs. However, the dynamics of these coupled cells incorporating both ephaptic and gap junction coupling has not been explored. Here, we show that automaticity and synchronization robustly emerges via a Hopf bifurcation from either (i) increasing the fraction of inward rectifying potassium channels (carrying the [Formula: see text] current) at the junctional membrane or (ii) by decreasing the cleft volume. Furthermore, we explore how heterogeneity in the fraction of potassium channels between coupled cells can produce automaticity of both cells or neither cell, or more rarely in only one cell (i.e., automaticity without synchronization). Interestingly, gap junction coupling generally has minor effects, with only slight changes in regions of parameter space of automaticity. This work provides insight into potentially new mechanisms that promote spontaneous activity and, thus, triggers for arrhythmias in ventricular tissue.

6

Ahmed, Walid, Wael Samy, Osama Tayeh, Noha Behairy, and Alia Abd El Fattah. "Left ventricular scar impact on left ventricular synchronization parameters and outcomes of cardiac resynchronization therapy." International Journal of Cardiology 222 (November 2016): 665–70. http://dx.doi.org/10.1016/j.ijcard.2016.07.158.

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7

Biner, Simon, Asim Rafique, Arik Wolak, Kirsten Tolstrup, and Robert J. Siegel. "Visual Assessment of Left Ventricular Dyssynchrony Using Tissue Synchronization Imaging." Cardiology 114, no. 2 (2009): 90–99. http://dx.doi.org/10.1159/000217742.

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8

ZHANG, Ye, Zhi-An LI, Yi-Hua HE, Hai-Bo ZHANG, and Xu MENG. "Utility of echocardiographic tissue synchronization imaging to redirect left ventricular epicardial lead placement for cardiac resynchronization therapy." Chinese Medical Journal 126, no. 22 (November 20, 2013): 4222–26. http://dx.doi.org/10.3760/cma.j.issn.0366-6999.20130606.

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Background Cardiac resynchronization therapy (CRT) with biventricular pacing has demonstrated cardiac function improvement for treating congestive heart failure (HF). It has been documented that the placement of the left ventricular lead at the longest contraction delay segment has the optimal CRT benefit. This study described follow-up to surgical techniques for CRT as a viable alternative for patients with heart failure. Methods Between April 2007 and June 2012, a total of 14 consecutive heart failure patients with New York Heart Association (NYHA) Class III-IV underwent left ventricular epicardial lead placements via surgical approach. There were eight males and six females, aged 36 to 79 years ((59.6±9.2) years). The mean left ventricular ejection fraction (LVEF) was (33.6±7.4)%. All patients were treated with left ventricular systolic dyssynchrony and underwent left ventricular epicardial lead placements via a surgical approach. Tissue Doppler imaging (TDI) and intraoperative transesophageal echocardiography were used to assess changes in left heart function and dyssynchronic parameters. Also, echo was used to select the best site for left ventricular epicardial lead placement. Results Left ventricular epicardial leads were successfully implanted in the posterior or lateral epicardial wall without serious complications in all patients. All patients had reduction in NYHA score from III-IV preoperatively to II-III postoperatively. The left ventricular end-diastolic diameter (LVEDD) decreased from (67.9±12.7) mm to (61.2±7.1) mm (P<0.05), and LVEF increased from (33.6±7.4)% to (42.2±8.8)% (P<0.05). Left ventricular intraventricular dyssynchrony index decreased from (148.4±31.6) ms to (57.3±23.8) ms (P<0.05). Conclusions Minimally invasive surgical placement of the left ventricular epicardial lead is feasible, safe, and efficient. TDI can guide the epicardial lead placement to the ideal target location.

9

Kaufmann, Michael R., Matthew S. McKillop, Thomas A. Burkart, Mark Panna, Jamie B. Conti, and William M. Miles. "Iatrogenic Ventricular Fibrillation after Direct-Current Cardioversion of Preexcited Atrial Fibrillation Caused by Inadvertent T-Wave Synchronization." Texas Heart Institute Journal 45, no. 1 (February 1, 2018): 39–41. http://dx.doi.org/10.14503/thij-16-6162.

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Direct-current cardioversion is an important means of managing arrhythmias. During treatment, carefully synchronizing energy delivery to the QRS complex is necessary to avoid ventricular fibrillation caused by a shock during the vulnerable period of ventricular repolarization, that is, a shock on the T wave. The presence of an accessory pathway and ventricular preexcitation can lead to difficulty in distinguishing the QRS complex from the T wave because of bizarre, wide, irregular QRS complexes and prominent repolarization. We present the cases of 2 patients who had iatrogenic ventricular fibrillation from inappropriate T-wave synchronization during direct-current cardioversion of preexcited atrial fibrillation. Our experience shows that rapidly recognizing the iatrogenic cause of VF and immediate treatment with unsynchronized defibrillation can prevent adverse clinical outcomes.

10

Hirayama, Yasutaka, Tatsuya Saito, Masaru Tsukamoto, Nobuyuki Sato, Yuichiro Kawamura, and Naoyuki Hasebe. "Right Ventricular Outflow Tract Pacing Reveals a Different Left Ventricular Synchronization According to the Degree of the Atrio-Ventricular Conductivity." Journal of Arrhythmia 27, Supplement (2011): OP64_6. http://dx.doi.org/10.4020/jhrs.27.op64_6.

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11

Azazy, AhmedS, Mahmoud Soliman, Rehab Yaseen, Morad Mena, and Haitham Sakr. "Left ventricular dyssynchrony assessment using tissue synchronization imaging in acute myocardial infarction." Avicenna Journal of Medicine 9, no. 2 (2019): 48. http://dx.doi.org/10.4103/ajm.ajm_168_18.

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12

Carlisle, Euan J. F., J. D. Allen, A. Bailey, W. George Kernohan, J. Anderson, and A. A. Jennifer Adgey. "Fourier analysis of ventricular fibrillation and synchronization of DC countershocks in defibrillation." Journal of Electrocardiology 21, no. 4 (January 1988): 337–43. http://dx.doi.org/10.1016/0022-0736(88)90110-0.

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13

Hirayama, Yasutaka, Yuichiro Kawamura, Nobuyuki Sato, Tatsuya Saito, Hideichi Tanaka, Yasuaki Saijo, Kenjiro Kikuchi, Katsumi Ohori, and Naoyuki Hasebe. "Functional characteristics of left ventricular synchronization via right ventricular outflow-tract pacing detected by two-dimensional strain echocardiography." Journal of Arrhythmia 33, no. 1 (February 2017): 28–34. http://dx.doi.org/10.1016/j.joa.2016.04.009.

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14

Heyndrickx, G. R., P. J. Vantrimpont, M. F. Rousseau, and H. Pouleur. "Effects of asynchrony on myocardial relaxation at rest and during exercise in conscious dogs." American Journal of Physiology-Heart and Circulatory Physiology 254, no. 5 (May 1, 1988): H817—H822. http://dx.doi.org/10.1152/ajpheart.1988.254.5.h817.

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The effect of left ventricular asynchrony induced by right ventricular pacing on relaxation indexes was studied at rest and during exercise in seven conscious dogs instrumented for chronic measurements of left ventricular pressure, coronary blood flow, and arterial pressure and with right atrial and ventricular pacing electrodes. Increasing heart rate with atrial pacing resulted in an increase in both left ventricular maximum and minimum rates of pressure development, LV dP/dtmax and LV dP/dtmin, respectively, as well as in a decrease in the relaxation constant T. In contrast, increasing heart rate with ventricular pacing resulted in a decrease in LV dP/dtmax, a small increase in LV dP/dtmin, and a significant decrease in T. During exercise with heart rate kept constant with atrial pacing, both LV dP/dtmax and LV dP/dtmin increased and T decreased to the same extent as during exercise in sinus rhythm. In contrast, exercising during right ventricular pacing resulted in a significant increase in T, expressing a slowing of relaxation. It is concluded that increasing heart rate alone in the presence of asynchrony of LV contraction induced by abnormal electrical activation results in a depressed contractile response, while the relaxation phase is not significantly affected. However, during sympathetic stimulation, a condition where synchronization should be improved, the relaxation phase is considerably lengthened.

15

Паршина, С. С., С. Н. Самсонов, Т. Н. Афанасьева, П. Г. Петрова, А. А. Стрекаловская, В. Д. Петрова, А. И. Кодочигова, К. В. Комзин та Л. К. Токаева. "ОСОБЕННОСТИ ОТВЕТНОЙ РЕАКЦИИ СЕРДЕЧНО-СОСУДИСТОЙ СИСТЕМЫ НА ГЕОМАГНИТНУЮ ВОЗМУЩЕННОСТЬ НА РАЗЛИЧНЫХ ШИРОТАХ". Биофизика 65, № 6 (2020): 1161–70. http://dx.doi.org/10.31857/s0006302920060162.

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This paper describes the principles of multi-latitude monitoring “Heliomed-2” conducted for obtaining new data on the impact of space weather on human health. The paper presents results of synchronous monitoring oriented to the assessment of the response of the cardiovascular system and psycho-emotional state of healthy volunteers to changes in the geomagnetic disturbance at high (Tixie settlement and the city of Yakutsk) and middle (city of Saratov) latitudes. In all the volunteers, the effects of synchronization of ventricular myocardial repolarization processes (according to the data on the symmetry coefficient of the T wave of the electrocardiogram) and geomagnetic disturbance as well as synchronization of reactive anxiety and geomagnetic disturbance were observed. It has been established that regardless of latitude, cardiac sensitivity and psychological sensitivity to geomagnetic disturbances in volunteers from the same group were similar.

16

Deodhar, Ajita, Timm Dickfeld, Gordon W. Single, William C. Hamilton, Raymond H. Thornton, Constantinos T. Sofocleous, Majid Maybody, Mithat Gónen, Boris Rubinsky, and Stephen B. Solomon. "Irreversible Electroporation Near the Heart: Ventricular Arrhythmias Can Be Prevented With ECG Synchronization." American Journal of Roentgenology 196, no. 3 (March 2011): W330—W335. http://dx.doi.org/10.2214/ajr.10.4490.

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17

Saito, I., T. Chinzei, Y. Abe, S. Mochizuki, K. Takiura, T. Ono, T. Isoyama, et al. "A STUDY OF NATURAL HEARTBEAT SYNCHRONIZATION FOR THE UNDULATION PUMP VENTRICULAR ASSIST DEVICE." ASAIO Journal 49, no. 2 (March 2003): 166. http://dx.doi.org/10.1097/00002480-200303000-00104.

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18

Murphy, Ross T., Gardar Sigurdsson, Sumanth Mulamalla, Deborah Agler, Zoran B. Popovic, Randall C. Starling, Bruce L. Wilkoff, James D. Thomas, and Richard A. Grimm. "Tissue Synchronization Imaging and Optimal Left Ventricular Pacing Site in Cardiac Resynchronization Therapy." American Journal of Cardiology 97, no. 11 (June 2006): 1615–21. http://dx.doi.org/10.1016/j.amjcard.2005.12.054.

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19

Ahmed, Walid, Wael Samy, Osama Tayeh, Noha Behairy, and Alia Abd el Fattah. "Temporal changes of left ventricular synchronization parameters and outcomes of cardiac resynchronization therapy." Egyptian Journal of Critical Care Medicine 4, no. 2 (August 2016): 105–12. http://dx.doi.org/10.1016/j.ejccm.2016.04.001.

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20

Gurevitz, Osnat, Shemy Carasso, David Bar-Lev, Sharona Bachar, David M. Luria, Michael Eldar, Micha S. Feinberg, and Michael Glikson. "Cardiac re-synchronization may have a beneficial effect on right ventricular mechanical performance." Heart Rhythm 2, no. 5 (May 2005): S166. http://dx.doi.org/10.1016/j.hrthm.2005.02.519.

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21

Engels, Elien B., Bernard Thibault, Jan Mangual, Nima Badie, Luke C. McSpadden, Leonardo Calò, Philippe Ritter, et al. "Dynamic atrioventricular delay programming improves ventricular electrical synchronization as evaluated by 3D vectorcardiography." Journal of Electrocardiology 58 (January 2020): 1–6. http://dx.doi.org/10.1016/j.jelectrocard.2019.09.026.

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22

WANG, Chao-hong, Yue-heng WANG, Ning-ning NIU, Ying-xin XIE, and Lin WANG. "Evaluation of the asynchronization and function of the left ventricle in patients with chronic pulmonary hypertension by velocity vector imaging." Chinese Medical Journal 126, no. 23 (December 5, 2013): 4457–62. http://dx.doi.org/10.3760/cma.j.issn.0366-6999.20132060.

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Background Pulmonary hypertension (PH) is a set of pathophysiological syndromes characterized by increased pulmonary artery pressure and pulmonary vascular resistance, resulting in increased right ventricular afterload. The left and right ventricles interact through hemodynamics. What impact will PH have on synchronization and function of the left ventricle (LV)? The aim of this study was to evaluate the synchronization of the left ventricular wall motion and left ventricular function in patients with varying degrees of PH using velocity vector imaging (VVI) technology. Methods Sixty patients with chronic PH served as the experimental group, and 20 healthy volunteers served as the control group. According to the different degrees of pulmonary artery systolic pressure, the experimental group was divided into three groups: mild, moderate, and severe PH groups. The time to peak systolic longitudinal velocity (Tvl), the peak systolic longitudinal velocity (Vsl), the peak diastolic longitudinal velocity (Vel), the peak systolic longitudinal strain (Sl), and strain rate (SRl) in 18 segments were measured in each group. Results Tvl in the control group and each group with PH was reduced from basal to apical segment, and in control group Tvl in various segments of the same wall and in different walls showed no significant difference (P >0.05). With increase in pulmonary artery pressure, Tvl values measured showed an increasing trend in groups with PH. In groups with PH, Vsl and Vel of each wall were reduced sequentially from basal to apical segments, showing gradient change; Vsl and Vel values measured showed a decreasing trend with increase in pulmonary artery pressure, in which the differences of Vel values measured in the control group and the mild PH group were statistically significant (P <0.01), and the differences between other groups were statistically significant (P <0.01). In groups with PH, Sl and SRl in basal segment and the middle segment of each wall were decreased; the difference between groups was statistically significant (P <0.01). Conclusions Asynchronization of the LV and decreased left ventricular function were present in patients with chronic PH; VVI technology can accurately evaluate left ventricular function in patients with PH, and indicators such as Tvl, Vsl, and Vel are valuable.

23

Pak, Hui-Nam, Yen-Bin Liu, Hideki Hayashi, Yuji Okuyama, Peng-Sheng Chen, and Shien-Fong Lin. "Synchronization of ventricular fibrillation with real-time feedback pacing: implication to low-energy defibrillation." American Journal of Physiology-Heart and Circulatory Physiology 285, no. 6 (December 2003): H2704—H2711. http://dx.doi.org/10.1152/ajpheart.00366.2003.

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Wavefront synchronization is an important aspect preceding the termination of ventricular fibrillation (VF). We evaluated the defibrillation efficacy of a novel multisite pacing algorithm using optical recording-guided synchronized pacing (SyncP) in the excitable gaps. We compared the effects of SyncP with traditional overdrive pacing (ODP) at 90% of the VF cycle length (VFCL) and high-frequency pacing (HFP; 43–215 Hz) on spontaneous VF termination in isolated rabbit hearts. For SyncP, the pacing current was triggered by the activation of a reference site and was delivered when the optical potential of the pacing site was in an excitable gap. We measured VFCL and the spatial dispersion of VFCL (SDCL) from five points (3 points in the paced area and 2 points in the nonpaced area) and the distribution of phase singularities during the prepacing, pacing, and postpacing periods. The results showed that 1) the VF termination rate of SyncP (16.0%, n = 106) was higher than that of ODP (2.1%, n = 48, P < 0.01) or HFP (1.6%, n = 129, P < 0.0001); 2) energy consumption for SyncP (7.6 ± 9.3 mJ) was significantly lower than that of ODP (14.0 ± 14.8 mJ, P < 0.0001); and 3) SyncP, but not ODP or HFP, decreased SDCL in the paced area during the pacing ( P < 0.01) and postpacing ( P < 0.05) periods compared with the prepacing period. We conclude that SyncP is effective in inducing wavefront synchronization and is more effective at facilitating spontaneous VF termination than non-SyncP.

24

Lafitte, Stephane, Stephane Garrigue, Jean-Marie Perron, Pierre Bordachar, Sylvain Reuter, Pierre Jaïs, Michel Haïssaguerre, Jacques Clementy, and Raymond Roudaut. "Improvement of left ventricular wall synchronization with multisite ventricular pacing in heart failure: a prospective study using Doppler tissue imaging." European Journal of Heart Failure 6, no. 2 (March 2004): 203–12. http://dx.doi.org/10.1016/j.ejheart.2003.10.008.

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25

Ryabov, I. A., I. N. Mamchur, T. Yu Chichkova, S. E. Mamchur, I. N. Sizova, N. S. Bokhan, E. A. Khomenko, and O. M. Chistyukhin. "Positive impact of cardiac contractile modulation on myocardial contractility and left ventricular synchronization in a patient with a left ventricular noncompaction." Siberian Journal of Clinical and Experimental Medicine 35, no. 2 (July 10, 2020): 157–62. http://dx.doi.org/10.29001/2073-8552-2020-35-2-157-162.

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The article provides a clinical case of a 58-year-old man with the fi rst clinical manifestation of chronic heart failure in the presence of a signifi cant decrease in the left ventricular ejection fraction. Left ventricular non-compaction cardiomyopathy was diagnosed by echocardiography. After 12 months, a cardiac contractility modulation device was implanted to the patient in the presence of disease progression despite optimal medical therapy. We assessed the course of disease, quality of life, exercise tolerance, and myocardial contractility of the patient before and six months after surgery. The methods of assessment were collection of patient complaints, physical examination, electrocardiography (ECG), fi lling out the Minnesota Living with Heart Failure Questionnaire (MLHFQ), sixminute walk test, spiroergometry, and echocardiography.

26

Ahmed, MohammedA, MahmoudA Soliman, and RehabI Yaseen. "Left ventricular dyssynchrony in hypertensive patients with normal systolic function: tissue synchronization imaging study." Menoufia Medical Journal 27, no. 2 (2014): 407. http://dx.doi.org/10.4103/1110-2098.141716.

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27

Morris-Thurgood, J. "Pacing in heart failure: improved ventricular interaction in diastole rather than systolic re-synchronization." Europace 2, no. 4 (October 2000): 271–75. http://dx.doi.org/10.1053/eupc.2000.0133.

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28

Li, Xinghui, Lunchao Zhong, Lin Luo, Shidong Zhu, Kai Ni, Qian Zhou, Bibo Yang, and Xiaohao Wang. "Synchronization control of pulsatile ventricular assist devices by combination usage of different physiological signals." Computer Assisted Surgery 24, sup1 (February 14, 2019): 105–12. http://dx.doi.org/10.1080/24699322.2018.1560089.

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29

Manoach, Mordechai, Dalia Varon, and Mordechai Erez. "The role of catecholamines on intercellular coupling, myocardial cell synchronization and self ventricular defibrillation." Molecular and Cellular Biochemistry 147, no. 1-2 (1995): 181–85. http://dx.doi.org/10.1007/bf00944799.

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30

Gurevitz, O., S. Carasso, D. Bar-Lev, S. Bahar, D. Luria, M. Eldar, M. Feinberg, and M. Glikson. "271 Cardiac re-synchronization may have a beneficial effect on right ventricular mechanical performance." EP Europace 7, Supplement_1 (2005): 65. http://dx.doi.org/10.1016/eupace/7.supplement_1.65-a.

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31

Abdrahmanova, A. I., N. B. Amirov, and N. A. Cibulkin. "Application of Perfusion Single Photon Emission Computed Tomography of the Myocardium in Pain-Free Myocardial Ischemia." Russian Archives of Internal Medicine 10, no. 5 (October 9, 2020): 340–47. http://dx.doi.org/10.20514/2226-6704-2020-10-5-340-347.

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This literature review provides data on the use of single-photon emission computed tomography of myocardium in silent myocardial ischemia. The presence of silent myocardial ischemia increases the risk of cardiovascular complications several times and may be the first manifestation of coronary heart disease. Assessing the state of morphofunctional processes in the myocardium is the main goal of diagnostic imaging using singlephoton emission computed tomography of the myocardium. This allows to get three-dimensional image of left ventricle with information about distribution of perfusion volume across myocardium, makes it possible to more accurately differentiate such condition as silent myocardial ischemia. Conducting single-photon emission computed tomography in ECG synchronization mode allows you to visualize the kinetics of the myocardial walls in different phases of the cardiac cycle and thereby simultaneously assess the functional state of the left ventricular myocardium. Indicators of contractile function of the left ventricular myocardium in areas of transient hypoperfusion can be predictors of cardiac events after myocardial infarction and independent predictors of perioperative cardiac events in patients undergoing cardiac surgery. Performing single-photon emission computed tomography in ECG-synchronization mode allows visualizing kinetics of myocardial walls in different phases of cardiac cycle and thereby simultaneously assessing functional state of left ventricle myocardium. In combination with physical exercise and pharmacological tests, it helps to identify coronary stenosis among patients with silent myocardial ischemia. Perfusion single-photon emission computed tomography of myocardium is a necessary tool for stratification and assessment of prognosis of cardiac diseases in asymptomatic patients.

32

Garnier, Antoine, and Grégoire Girod. "Cardiac re-synchronization therapy in a patient with isolated ventricular non-compaction: a case report." European Heart Journal - Cardiovascular Imaging 10, no. 5 (April 30, 2009): 713–15. http://dx.doi.org/10.1093/ejechocard/jep040.

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33

Kolesnyk, M. Yu, and M. V. Sokolova. "Deformation of the left heart chambers in hypertensive postmenopausal women, depending on the presence of left ventricular hypertrophy and left atrium dilation." Ukrainian Journal of Cardiology 26, no. 3 (July 30, 2019): 17–26. http://dx.doi.org/10.31928/1608-635x-2019.3.1726.

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The aim – to assess the longitudinal deformation (strain) of the left heart chambers in postmenopausal women with essential hypertension (EH), depending on the presence of left ventricular hypertrophy (LVH) and left atrial (LA) dilation. Materials and methods. The study involved 126 postmenopausal women: 100 patients with EH I–II stages of the main group and 26 practically healthy women of the comparison group. Patients with EH were divided into two groups: 32 patients without structural changes of the myocardium and 68 women with LVH and/or LA dilation. In all patients we performed ambulatory blood pressure monitoring, standard transthoracic echocardiography and speckle-tracking echocardiography. The global longitudinal strain (GLS) of LV and deformation of the endocardial (endo), middle (mid) and epicardial (epi) layers of myocardium were analyzed. Analysis of LA deformation was performed using two (from the beginning of the R-wave and from the apex of the R-wave) variants of ECG-synchronization. The LA longitudinal strain (LS) was evaluated in reservoir and contraction phase in two positions with the calculation of the GLS LA. Results and discussion. We found changes in LV multilayer deformation as LS decreasing in the endocardial, middle and epicardial layers in hypertensive patients in the early stages of disease, even before the development of LVH. Damage of LA deformation preceded its dilation. Both types of ECG-synchronization showed a statistically significant decrease of LA strain in the reservoir phase in all hypertensive patients in comparison with healthy women. A decreasing LA GLS in women with EH and structurally normal heart compared to the healthy group was detected only by using ECG-synchronization with R-wave, which is considered more universal. Conclusion. A decrease of LA and LV LS in postmenopausal women is recorded even before the development of LVH and LA dilation. The LV LS became lower in all layers of myocardium – from endocardial to epicardial. Changes in the LA LS in postmenopausal women with EH begin with a damage of reservoir phase even with normal size of LA and a LV myocardial mass index.

34

Wyman, Bradley T., William C. Hunter, Frits W. Prinzen, Owen P. Faris, and Elliot R. McVeigh. "Effects of single- and biventricular pacing on temporal and spatial dynamics of ventricular contraction." American Journal of Physiology-Heart and Circulatory Physiology 282, no. 1 (January 1, 2002): H372—H379. http://dx.doi.org/10.1152/ajpheart.2002.282.1.h372.

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Resynchronization is frequently used for the treatment of heart failure, but the mechanism for improvement is not entirely clear. In the present study, the temporal synchrony and spatiotemporal distribution of left ventricular (LV) contraction was investigated in eight dogs during right atrial (RA), right ventricular apex (RVa), and biventricular (BiV) pacing using tagged magnetic resonance imaging. Mechanical activation (MA; the onset of circumferential shortening) was calculated from the images throughout the left ventricle for each pacing protocol. MA width (time for 20–90% of the left ventricle to contract) was significantly shorter during RA (43.6 ± 17.1 ms) than BiV and RVa pacing (67.4 ± 15.2 and 77.6 ± 16.4 ms, respectively). The activation delay vector (net delay in MA from one side of the left ventricle to the other) was significantly shorter during RA (18.9 ± 8.1 ms) and BiV (34.2 ± 18.3 ms) than during RVa (73.8 ± 16.3 ms) pacing. Rate of LV pressure increase was significantly lower during RVa than RA pacing (1,070 ± 370 vs. 1,560 ± 300 mmHg/s) with intermediate values for BiV pacing (1,310 ± 220 mmHg/s). BiV pacing has a greater impact on correcting the spatial distribution of LV contraction than on improving the temporal synchronization of contraction. Spatiotemporal distribution of contraction may be an important determinant of ventricular function.

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Masumiya, H. "Contribution of Nav1.1 to cellular synchronization and automaticity in spontaneous beating cultured neonatal rat ventricular cells." General Physiology and Biophysics 30, no. 1 (2011): 28–33. http://dx.doi.org/10.4149/gpb_2011_01_28.

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Gedikli, Omer, Merih Baykan, Kubra Kaynar, Gulsum Ozkan, Levent Korkmaz, Serkan Ozturk, Ismet Durmus, Sahin Kaplan, and Sukru Celik. "Assessment of Left Ventricular Systolic Synchronization in Patients with Chronic Kidney Disease and Narrow QRS Complexes." Echocardiography 26, no. 5 (May 2009): 528–33. http://dx.doi.org/10.1111/j.1540-8175.2008.00836.x.

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VANDEVEIRE, N., G. BLEEKER, J. DESUTTER, C. YPENBURG, E. HOLMAN, E. VANDERWALL, M. SCHALIJ, and J. BAX. "940 Tissue synchronization imaging accurately measures left ventricular dyssynchrony and predicts response to cardiac resynchronization therapy." European Journal of Echocardiography 7 (December 2006): S160. http://dx.doi.org/10.1016/s1525-2167(06)60595-7.

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PEIX, A. "Ventricular synchronization assessed by phase analysis in a radionuclide angiography in individuals with normal cardiac function." Journal of Nuclear Cardiology 6, no. 1 (February 1999): S106. http://dx.doi.org/10.1016/s1071-3581(99)90535-4.

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Alcaraz, Raúl, and José J. Rieta. "Nonlinear synchronization assessment between atrial and ventricular activations series from the surface ECG in atrial fibrillation." Biomedical Signal Processing and Control 8, no. 6 (November 2013): 1000–1007. http://dx.doi.org/10.1016/j.bspc.2013.01.009.

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40

Stöllberger, Claudia, and Josef Finsterer. "Left ventricular synchronization by biventricular pacing in Becker muscular dystrophy as assessed by tissue Doppler imaging." Heart & Lung 34, no. 5 (September 2005): 317–20. http://dx.doi.org/10.1016/j.hrtlng.2005.03.003.

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Saito, Itsuro, Yusuke Abe, Tsuneo Chinzei, Takashi Isoyama, Shuichi Mochizuki, Akimasa Kouno, Toshiya Ono, et al. "A STUDY OF HEARTBEAT SYNCHRONIZATION FOR THE UNDULATION PUMP VENTRICULAR ASSIST DEVICE USING THE INFLOW PRESSURE." ASAIO Journal 52, no. 2 (March 2006): 50A. http://dx.doi.org/10.1097/00002480-200603000-00217.

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42

Sun, Zhijun, Beibing Di, Huikuan Gao, Dihui Lan, and Hui Peng. "Assessment of ventricular mechanical synchronization after left bundle branch pacing using 2‐D speckle tracking echocardiography." Clinical Cardiology 43, no. 12 (October 21, 2020): 1562–72. http://dx.doi.org/10.1002/clc.23481.

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43

Stukalova, O. V., A. A. Beliaevskaia, and S. K. Ternovoy. "ASSESSMENT OF LEFT VENTRICULAR EJECTION FRACTION USING CINE-MRI WITHOUT BREATH-HOLDING AND SYNCHRONIZATION WITH ECG." Russian Electronic Journal of Radiology 14, no. 1 (2024): 89–97. http://dx.doi.org/10.21569/2222-7415-2024-14-1-89-97.

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44

Pascual, Domenic, Matthias Heinke, Reinhard Echle, and Johannes Hörth. "Electrode Model and Simulation of His- Bundle Pacing for Cardiac Resynchronization Therapy." Current Directions in Biomedical Engineering 6, no. 3 (September 1, 2020): 555–58. http://dx.doi.org/10.1515/cdbme-2020-3142.

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AbstractA disturbed synchronization of the ventricular contraction can cause a highly developed systolic heart failure in affected patients with reduction of the left ventricular ejection fraction, which can often be explained by a diseased left bundle branch block (LBBB). If medication remains unresponsive, the concerned patients will be treated with a cardiac resynchronization therapy (CRT) system. The aim of this study was to integrate His-bundle pacing into the Offenburg heart rhythm model in order to visualize the electrical pacing field generated by His-Bundle-Pacing. Modelling and electrical field simulation activities were performed with the software CST (Computer Simulation Technology) from Dessault Systèms. CRT with biventricular pacing is to be achieved by an apical right ventricular electrode and an additional left ventricular electrode, which is floated into the coronary vein sinus. The non-responder rate of the CRT therapy is about one third of the CRT patients. His- Bundle-Pacing represents a physiological alternative to conventional cardiac pacing and cardiac resynchronization. An electrode implanted in the His-bundle emits a stronger electrical pacing field than the electrical pacing field of conventional cardiac pacemakers. The pacing of the Hisbundle was performed by the Medtronic Select Secure 3830 electrode with pacing voltage amplitudes of 3 V, 2 V and 1,5 V in combination with a pacing pulse duration of 1 ms. Compared to conventional pacemaker pacing, His-bundle pacing is capable of bridging LBBB conduction disorders in the left ventricle. The His-bundle pacing electrical field is able to spread via the physiological pathway in the right and left ventricles for CRT with a narrow QRS-complex in the surface ECG.

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DOHI, KAORU, MATTHEW SUFFOLETTO, LEONARD GANZ, MARCO ZENATI, and JOHN GORCSAN. "Utility of Echocardiographic Tissue Synchronization Imaging to Redirect Left Ventricular Lead Placement for Improved Cardiac Resynchronization Therapy." Pacing and Clinical Electrophysiology 28, no. 5 (May 2005): 461–65. http://dx.doi.org/10.1111/j.1540-8159.2005.40056.x.

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46

Tang, Jiaojiao, Silin Chen, Lie Liu, Hongtao Liao, Xianzhang Zhan, Shulin Wu, Yuanhong Liang, et al. "Assessment of Permanent Selective His Bundle Pacing in Left Ventricular Synchronization Using 3-D Speckle Tracking Echocardiography." Ultrasound in Medicine & Biology 45, no. 2 (February 2019): 385–94. http://dx.doi.org/10.1016/j.ultrasmedbio.2018.10.006.

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Sumitomo, Naofumi F., Naoya Fukushima, and Masaru Miura. "Flecainide improves cardiac synchronization in an early infant with Wolff–Parkinson–White syndrome with left ventricular dyssynchrony." Journal of Cardiology Cases 22, no. 1 (July 2020): 1–4. http://dx.doi.org/10.1016/j.jccase.2020.03.004.

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Gurevitz, Osnat, Chava Granit, Shemy Carasso, David M. Luria, David Bar-Lev, Michael Eldar, and Michael Glikson. "Evolution of left and right ventricular capture thresholds over time in patients receiving cardiac re-synchronization therapy." Heart Rhythm 2, no. 5 (May 2005): S289. http://dx.doi.org/10.1016/j.hrthm.2005.02.910.

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49

Choquet, Caroline, Pierre Sicard, Juliette Vahdat, Thi Hong Minh Nguyen, Frank Kober, Isabelle Varlet, Monique Bernard, et al. "Nkx2-5 Loss of Function in the His-Purkinje System Hampers Its Maturation and Leads to Mechanical Dysfunction." Journal of Cardiovascular Development and Disease 10, no. 5 (April 27, 2023): 194. http://dx.doi.org/10.3390/jcdd10050194.

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The ventricular conduction or His-Purkinje system (VCS) mediates the rapid propagation and precise delivery of electrical activity essential for the synchronization of heartbeats. Mutations in the transcription factor Nkx2-5 have been implicated in a high prevalence of developing ventricular conduction defects or arrhythmias with age. Nkx2-5 heterozygous mutant mice reproduce human phenotypes associated with a hypoplastic His-Purkinje system resulting from defective patterning of the Purkinje fiber network during development. Here, we investigated the role of Nkx2-5 in the mature VCS and the consequences of its loss on cardiac function. Neonatal deletion of Nkx2-5 in the VCS using a Cx40-CreERT2 mouse line provoked apical hypoplasia and maturation defects of the Purkinje fiber network. Genetic tracing analysis demonstrated that neonatal Cx40-positive cells fail to maintain a conductive phenotype after Nkx2-5 deletion. Moreover, we observed a progressive loss of expression of fast-conduction markers in persistent Purkinje fibers. Consequently, Nkx2-5-deleted mice developed conduction defects with progressively reduced QRS amplitude and RSR’ complex associated with higher duration. Cardiac function recorded by MRI revealed a reduction in the ejection fraction in the absence of morphological changes. With age, these mice develop a ventricular diastolic dysfunction associated with dyssynchrony and wall-motion abnormalities without indication of fibrosis. These results highlight the requirement of postnatal expression of Nkx2-5 in the maturation and maintenance of a functional Purkinje fiber network to preserve contraction synchrony and cardiac function.

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Ayhan, Selçuk, Veli Gökhan Cin, and Sabri Abuş. "Comparing ICD shock ratios between Type 1, Type 2 and non diabetic patients." Medical Science and Discovery 10, no. 5 (May 8, 2023): 275–84. http://dx.doi.org/10.36472/msd.v10i5.938.

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Objective: The use of permanent pacemakers and implantable cardioverter defibrillators (ICDs) is increasing. With technological advances, the devices' implantation techniques and programmable features are improving, and indications are expanding. Several technical and clinical problems are encountered during implantation and follow-up of these devices. Methods: Our study retrospectively evaluated diabetic and non-diabetic patients who underwent ICD implantation in Mersin University Cardiology Department between January 2010 and December 2013 according to current indications. All clinical characteristics, baseline, 1-month and 6-month follow-up pacemaker data and baseline laboratory values were recorded. Results: A total of 106 ICD patients (57 diabetics and 49 nondiabetics) were enrolled at the 1st and 6th months of follow-up. 47.2% of the patients were male and the mean age was 56±7.3 years. ICDs were implanted for secondary prevention in 83.9% of patients. ICD was implanted for coronary artery disease +/- ventricular tachycardia (VT) or ventricular fibrillation (VF)+/- synchronization disorder. 75.4% of patients had CAD, and 89.6% had heart failure. The mean ejection fraction ratio (EF) was 31.3%. Early complications were observed in 10.3% of patients. 47.1% of patients received any treatment by the ICD, 34.9% had the appropriate shock, and 12.2% had inappropriate shock. The complication rate was 10.3%. While there was no difference in ventricular impedance and threshold values in diabetic patients compared to the control group, ventricular lead R amplitude values were found to be higher. The rate of atrial fibrillation was significantly different in the treated group. Treatment response was obtained in 71.4% of patients who underwent treatment change due to appropriate shock, incorrect shock and ATP. Conclusion: In conclusion, the incidence of appropriate shock and anti-tachycardic pacing was higher in diabetic patients than non-diabetic patients.