Статті в журналах: "Transient function"

1

Song, Justin C. W. "Transient wave function twist." Nature Physics 16, no. 1 (November 4, 2019): 6–7. http://dx.doi.org/10.1038/s41567-019-0708-0.

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2

Grubin, H. L. "Transient Wigner Function Studies of DMS Barrier Devices." Advances in Science and Technology 52 (October 2006): 36–41. http://dx.doi.org/10.4028/www.scientific.net/ast.52.36.

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Incorporating diluted magnetic semiconductor (DMS) layers within barrier devices offers new device design potential. To study these devices we have generalized an existing time dependent transient algorithm that couples the Wigner transport equation to Poisson’s equation and an external circuit. For electron transport we have studied alterations in the dc and time dependent behavior of resonant tunneling diodes with DMS barriers and wells, have transformed a single barrier structure into a double barrier structure and examined the increased functionality of the devices. We present new results, including some preliminary calculations incorporating holes, discuss transients and the potential role that DMS layers will play in controlling the transient operation of superlattice structures.

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3

Fouad, A. A., and V. Vittal. "The transient energy function method." International Journal of Electrical Power & Energy Systems 10, no. 4 (October 1988): 233–46. http://dx.doi.org/10.1016/0142-0615(88)90011-7.

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4

Козир, Олег Васильович, and Юліан Михайлович Туз. "Study of thermocouple transient function models." Information systems, mechanics and control, no. 20 (June 30, 2019): 13–22. http://dx.doi.org/10.20535/2219-3804202019194307.

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5

ZHAO, J. "Executive function during transient ischemic attacks." Neural Regeneration Research 2, no. 3 (March 2007): 142–45. http://dx.doi.org/10.1016/s1673-5374(07)60032-2.

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6

Christensen, Niels B. "Sensitivity functions of transient electromagnetic methods." GEOPHYSICS 79, no. 4 (July 1, 2014): E167—E182. http://dx.doi.org/10.1190/geo2013-0364.1.

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Any geophysical measurement is a filter through which the distribution of a certain physical parameter in the subsurface is seen, and the sensitivity function is a characteristic of the method that reveals the nature of this filter. Insight into the structural pattern of the sensitivity function pertaining to a certain transmitter-receiver configuration provides the geophysicist with an image that allows an immediate qualitative understanding of the characteristics of the method. The assets and shortcomings of different measuring configurations can be discussed and understood, and the sensitivity function permits qualified predictions about resolution characteristics of new configurations and measuring strategies. I evaluated a rapid and accurate method for calculating 3D sensitivity functions of a homogeneous half-space model for a wide variety of transient electromagnetic configurations using the central loop and an airborne offset loop configuration as examples. Computations of 3D sensitivity functions were performed as convolutions in the time domain between the electric fields from the transmitter and the receiver, had it been used as a transmitter. The 2D and 1D sensitivity functions are found through numerical integration of the 3D functions. Beside offering insight into the resolution capability of the measuring configuration, the sensitivity functions lend themselves to rapid calculations of approximate responses and derivatives in various modeling and inversion strategies.

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7

Garimella, S. S., and K. Srinivasan. "Transient Response of Repetitive Control Systems." Journal of Dynamic Systems, Measurement, and Control 118, no. 4 (December 1, 1996): 795–97. http://dx.doi.org/10.1115/1.2802360.

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Upper bounds on transient response magnitudes for a SISO continuous-time repetitive control system are derived. Limiting the size of these transients is shown to be related to limiting the ∞-norm of a transfer function product of filters used in the repetitive controller. The decay rate of the transients is related to the peak of a function of frequency called the regeneration spectrum, which has previously been shown in the literature to be a measure of the relative stability of the system. Bounds derived here, although conservative, can be useful in the design of the repetitive controller, as illustrated by means of an example.

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8

Da-Zhong Fang, T. S. Chung, Yao Zhang, and Wennan Song. "Transient stability limit conditions analysis using a corrected transient energy function approach." IEEE Transactions on Power Systems 15, no. 2 (May 2000): 804–10. http://dx.doi.org/10.1109/59.867177.

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9

Robbins, Matthew S., David M. Breidbart, and Hilary Y. Robbins. "Transient global amnesia complicating pulmonary function testing." Respiratory Medicine CME 3, no. 4 (2010): 230–31. http://dx.doi.org/10.1016/j.rmedc.2009.12.002.

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10

McDonough, Paul G., and Carlos E. Sueldo. "Transient Liver Function Tests Abnormalities in OHSS." Fertility and Sterility 50, no. 6 (December 1988): 995–96. http://dx.doi.org/10.1016/s0015-0282(16)60392-8.

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11

Korkmaz, Serdar, Emrah Pekkan, and Yücel Güney. "Transient Analysis withMODFLOWfor Developing Water-Diversion Function." Journal of Hydrologic Engineering 21, no. 6 (June 2016): 05016009. http://dx.doi.org/10.1061/(asce)he.1943-5584.0001349.

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12

Earley, Scott, and Joseph E. Brayden. "Transient receptor potential channels and vascular function." Clinical Science 119, no. 1 (April 7, 2010): 19–36. http://dx.doi.org/10.1042/cs20090641.

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TRP (transient receptor potential) channels play important roles in the regulation of normal and pathological cellular function. In the vasculature, TRP channels are present both in ECs (endothelial cells) and vascular SMCs (smooth muscle cells) and contribute to vasomotor control mechanisms in most vascular beds. Vascular TRP channels are activated by various stimuli, such as mechanical perturbation, receptor activation and dietary molecules. Some of the specific roles of these channels in normal and impaired vascular function have emerged in recent years and include participation in vascular signalling processes, such as neurotransmission, hormonal signalling, NO production, myogenic tone and autoregulation of blood flow, thermoregulation, responses to oxidative stress and cellular proliferative activity. Current research is aimed at understanding the interactions of TRP channels with other vascular proteins and signalling mechanisms. These studies should reveal new targets for pharmacological therapy of vascular diseases, such as hypertension, ischaemia and vasospasm, and vascular proliferative states.

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13

Le, Hung T., and Edward J. Wegman. "Generalized function estimation of underwater transient signals." Journal of the Acoustical Society of America 89, no. 1 (January 1991): 274–79. http://dx.doi.org/10.1121/1.400670.

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14

Avelino, A., A. Charrua, B. Frias, C. Cruz, M. Boudes, D. de Ridder, and F. Cruz. "Transient receptor potential channels in bladder function." Acta Physiologica 207, no. 1 (November 1, 2012): 110–22. http://dx.doi.org/10.1111/apha.12021.

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15

Pérez, Claudia G., Julio A. Copello, Yanxia Li, Kimberly L. Karko, Leticia Gómez, Josefina Ramos-Franco, Michael Fill, Ariel L. Escobar, and Rafael Mejía-Alvarez. "Ryanodine receptor function in newborn rat heart." American Journal of Physiology-Heart and Circulatory Physiology 288, no. 5 (May 2005): H2527—H2540. http://dx.doi.org/10.1152/ajpheart.00188.2004.

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The role of ryanodine receptor (RyR) in cardiac excitation-contraction (E-C) coupling in newborns (NB) is not completely understood. To determine whether RyR functional properties change during development, we evaluated cellular distribution and functionality of sarcoplasmic reticulum (SR) in NB rats. Sarcomeric arrangement of immunostained SR Ca2+-ATPase (SERCA2a) and the presence of sizeable caffeine-induced Ca2+ transients demonstrated that functional SR exists in NB. E-C coupling properties were then defined in NB and compared with those in adult rats (AD). Ca2+ transients in NB reflected predominantly sarcolemmal Ca2+ entry, whereas the RyR-mediated component was ∼13%. Finally, the RyR density and functional properties at the single-channel level in NB were compared with those in AD. Ligand binding assays revealed that in NB, RyR density can be up to 36% of that found in AD, suggesting that some RyRs do not contribute to the Ca2+ transient. To test the hypothesis that RyR functional properties change during development, we incorporated single RyRs into lipid bilayers. Our results show that permeation and gating kinetics of NB RyRs are identical to those of AD. Also, endogenous ligands had similar effects on NB and AD RyRs: sigmoidal Ca2+ dependence, stronger Mg2+-induced inhibition at low cytoplasmic Ca2+ concentrations, comparable ATP-activating potency, and caffeine sensitivity. These observations indicate that NB rat heart contains fully functional RyRs and that the smaller contribution of RyR-mediated Ca2+ release to the intracellular Ca2+ transient in NB is not due to different single RyR channel properties or to the absence of functional intracellular Ca2+ stores.

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16

Dhole, G. M., and M. K. Khedkar. "Antigen energy function: a new energy function for transient stability assessment." Electric Power Systems Research 74, no. 2 (May 2005): 315–22. http://dx.doi.org/10.1016/j.epsr.2004.12.001.

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17

Mayer, Otto, Jitka Seidlerová, Jan Bruthans, Jan Opatrný, Milan Hromádka, Josef Jirák, and Jan Filipovský. "The prognostic impact of renal function decline during hospitalization for myocardial infarction." Journal of Comparative Effectiveness Research 10, no. 3 (February 2021): 219–28. http://dx.doi.org/10.2217/cer-2020-0085.

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Aim: We analyzed the mortality risk of myocardial infarction (MI) patients according to renal function, observed during hospitalization. Materials & methods: Patients hospitalized for MI between 2006 and 2018 were followed (n = 5659). We divided the sample into four groups by estimated glomerular filtration (eGFR) [ml/min]: normal functions (lowest eGFR during hospitalization >60); transiently moderate insufficiency (lowest eGFR >30 and ≤60, highest >60); permanently moderate insufficiency (highest eGFR >30 and ≤60); severe insufficiency (highest and lowest eGFR ≤30). Results: Permanently moderate renal insufficiency indicates increased 5-years all-cause mortality (hazard risk ratio: 2.27 [95% CIs: 1.87–2.75], p < 0.0001), but a similar risk was found in patients with the only transient decline of renal functions (hazard risk ratio: 2.08 [95% CIs: 1.70–2.55], p < 0.0001). Both moderate insufficiency subgroups (transient/permanent) did not statistically differ regarding mortality risk. Conclusion: Even just fluctuation of eGFR toward moderate insufficiency during hospitalization represents an important prognostic indicator in MI patients.

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18

Chydzinski, Andrzej. "On the Transient Queue with the Dropping Function." Entropy 22, no. 8 (July 28, 2020): 825. http://dx.doi.org/10.3390/e22080825.

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We deal with a queueing system, in which arriving packets are being dropped with the probability depending on the queue size. Such a scheme is used in several active queue management schemes proposed for Internet routers. In this paper, we derive and analyze a selected transient characteristic of the model, i.e., the probability that in a given time interval the queue size is kept under a predefined level. As the main purpose of the discussed queueing scheme is to maintain the queue size low, this is a natural characteristic to study. In addition to that, the average time to reach a given level is derived. Theoretical results for both characteristics are accompanied by numerical examples. Among other things, they demonstrate that the transient behavior of the queue may vary significantly with the shape of the dropping function, even if the steady-state performance remains unaltered.

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19

Fraser, W. D., M. D. Gardner, J. O'Donnell, and F. J. Dryburgh. "TRANSIENT IMPAIRMENT OF RENAL FUNCTION AFTER GENERALISED SEIZURES." Lancet 329, no. 8547 (June 1987): 1428. http://dx.doi.org/10.1016/s0140-6736(87)90612-x.

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20

Twamley, Elizabeth W., Barton W. Palmer, Dilip V. Jeste, Michael J. Taylor, and Robert K. Heaton. "Transient and executive function working memory in schizophrenia." Schizophrenia Research 87, no. 1-3 (October 2006): 185–90. http://dx.doi.org/10.1016/j.schres.2006.04.013.

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21

Payne, S. T., H. S. R. Hosker, M. B. Allen, H. Bradbury, and R. L. Page. "TRANSIENT IMPAIRMENT OF RENAL FUNCTION AFTER STREPTOKINASE THERAPY." Lancet 334, no. 8676 (December 1989): 1398. http://dx.doi.org/10.1016/s0140-6736(89)92010-2.

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22

Evans, Denis J., Debra J. Searles, and Stephen R. Williams. "The covariant dissipation function for transient nonequilibrium states." Journal of Chemical Physics 133, no. 5 (August 7, 2010): 054507. http://dx.doi.org/10.1063/1.3463439.

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23

Carbajo, S. "Transient work function gating: A new photoemission regime." Journal of Applied Physics 128, no. 2 (July 14, 2020): 023102. http://dx.doi.org/10.1063/5.0011737.

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24

Pai, M. A., M. Laufenberg, and P. W. Sauer. "Some clarifications in the transient energy function method." International Journal of Electrical Power & Energy Systems 18, no. 1 (January 1996): 65–72. http://dx.doi.org/10.1016/0142-0615(95)00062-3.

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25

Pai, M. "Some clarifications in the transient energy function method." Fuel and Energy Abstracts 37, no. 3 (May 1996): 195. http://dx.doi.org/10.1016/0140-6701(96)88720-0.

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26

Guenoun, Thierry, Olivier Montagne, Monique Laplace, and Bertrand Crozatier. "Propofol-induced Modifications of Cardiomyocyte Calcium Transient and Sarcoplasmic Reticulum Function in Rats." Anesthesiology 92, no. 2 (February 1, 2000): 542. http://dx.doi.org/10.1097/00000542-200002000-00038.

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Background Propofol is considered to be an anesthetic agent with few or no negative inotropic effects. This study evaluated a possible direct depressant effect of propofol on sarcoplasmic reticulum Ca2+ accumulation and cardiomyocytes. Methods The effects of propofol on intracellular Ca2+ transients were evaluated in isolated rat cardiomyocytes using a microfluorometric technique with Indo-1. Sarcoplasmic reticulum function was also assessed by measuring the oxalate-stimulated Ca2+ uptake from homogenates of rat ventricles. Results The Ca2+ uptake capacity of the sarcoplasmic reticulum was decreased by propofol (10(-4) M). Large concentrations of propofol decreased the rate of decrease of the intracellular Ca2+ transient, which resulted in an increase of diastolic Ca2+ when the diastolic interval was decreased. The increased diastolic Ca2+ also resulted in a decrease in Ca2+ transient. This effect appeared for lower doses (10(-5) M) after a short diastolic pause rather than after a long (2- to 3-min) rest (appearing at 10(-4) M). Conclusions For doses more than 10(-5) M, propofol induces a Ca2+ uptake capacity impairment of the sarcoplasmic reticulum. This is probably responsible for a slowing of the decrease of the Ca2+ transient, which in turn increases the diastolic Ca2+ for high heart rate. These diastolic modifications may participate in the slight negative inotropic effect of the drug.

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27

Duza, Tasmia, and Ingrid H. Sarelius. "Localized transient increases in endothelial cell Ca2+ in arterioles in situ: implications for coordination of vascular function." American Journal of Physiology-Heart and Circulatory Physiology 286, no. 6 (June 2004): H2322—H2331. http://dx.doi.org/10.1152/ajpheart.00006.2004.

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Intracellular Ca2+ transients were identified in endothelial cells (ECs) in intact blood-perfused arterioles. ECs in cremaster muscle arterioles (diameter ∼45 μm) in anesthetized mice were loaded with the Ca2+ indicator fluo 4-AM by intraluminal perfusion, after which blood flow was reestablished. Confocal microscopy was used to visualize Ca2+ as a function of fluo-4 intensity in real time. Separate sets of experiments were performed under the following conditions: control, ischemia, during inhibition of P2x or P1 purinoreceptors, and with the application of exogenous adenosine. In controls, spontaneous EC Ca2+ transients displayed a wide range of activity frequency (1–32 events/min) and about one-third of these transient events were synchronized between adjacent ECs. The increase in Ca2+ remained localized and did not spread to encompass the entire cell body. Ca2+ transient activity decreased significantly with ischemia (from 9.9 ± 0.6 to 3.1 ± 0.3 events/min, n = 135) but was unaffected by P2x or P1 receptor inhibition. Exogenous adenosine significantly increased the frequency of Ca2+ transients (to 12.8 ± 0.9 events/min) and increased synchronization so that 50% of all Ca2+ events were synchronized between ECs. This response to adenosine was not due to an increase in shear stress. These data indicate that localized Ca2+ transients are sensitive to flow conditions and, separately, to metabolically active pathways (exogenous adenosine), although the basal activity occurs independently of P2x or P1 receptors. These transients may represent a mechanism by which individual EC responses are integrated to result in coordinated arteriolar responses in situ.

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28

Al Majid, A., and R. Dufour. "Damping in High Transient Motion." Journal of Vibration and Acoustics 125, no. 2 (April 1, 2003): 223–27. http://dx.doi.org/10.1115/1.1547702.

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An original method for evaluating the dissipative effect in SDOF systems due to the transient phenomenon was presented in a previous article. This method based on the use of an additional dimension, and the general relativity concept was validated experimentally. However, the function of the forcing frequency required to establish the metric of the space was identified using an experimental transfer function. In the present paper the main objective is to solve the geodesic equations in order to avoid the experimental identification of the function contained in the metric. The variational problem of the metric of Riemannian space gives three geodesic equations for the SDOF system studied. Solving these equations gives in particular the transient forced response which, when compared with experimental results, permits validating the proposed method and therefore proving that the transient motion bends the space-time.

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29

Holloway, D. S., and M. R. Davis. "Green Function Solutions for the Transient Motion of Water Sections." Journal of Ship Research 46, no. 02 (June 1, 2002): 99–120. http://dx.doi.org/10.5957/jsr.2002.46.2.99.

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A time-domain boundary element method based on a Green function solution is derived for two-dimensional motions in the presence of a free liquid surface. Particular attention is given to the numerical evaluation of the required Green functions with regard to accuracy and speed of solution by choice of a computational algorithm appropriate to the domain of particular computations. The method is validated with reference to analytic solutions for submerged and floating cylinders in steady, transient and periodic motion. The intended application of the method is in the computation of wave response of slender ships at high Froude number by a fixed frame strip theory where the Green function obviates the necessity to panel the free surface with elements.

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30

Mouneimne, Ghassan, Lilian Soon, Vera DesMarais, Mazen Sidani, Xiaoyan Song, Shu-Chin Yip, Mousumi Ghosh, Robert Eddy, Jonathan M. Backer, and John Condeelis. "Phospholipase C and cofilin are required for carcinoma cell directionality in response to EGF stimulation." Journal of Cell Biology 166, no. 5 (August 30, 2004): 697–708. http://dx.doi.org/10.1083/jcb.200405156.

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The epidermal growth factor (EGF)–induced increase in free barbed ends, resulting in actin polymerization at the leading edge of the lamellipodium in carcinoma cells, occurs as two transients: an early one at 1 min and a late one at 3 min. Our results reveal that phospholipase (PLC) is required for triggering the early barbed end transient. Phosphoinositide-3 kinase selectively regulates the late barbed end transient. Inhibition of PLC inhibits cofilin activity in cells during the early transient, delays the initiation of protrusions, and inhibits the ability of cells to sense a gradient of EGF. Suppression of cofilin, using either small interfering RNA silencing or function-blocking antibodies, selectively inhibits the early transient. Therefore, our results demonstrate that the early PLC and cofilin-dependent barbed end transient is required for the initiation of protrusions and is involved in setting the direction of cell movement in response to EGF.

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31

Billa Elghali, Elmotaz, Marayati Marsadek, and Agileswari K. Ramasamy. "Risk of Transient Stability Using Rotor Trajectory Index as Severity Function." Indonesian Journal of Electrical Engineering and Computer Science 6, no. 3 (June 1, 2017): 591. http://dx.doi.org/10.11591/ijeecs.v6.i3.pp591-601.

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This paper presents a new approach to determine the risk of transient stability. It describes the implementation of rotor trajectory index (RTI) to assess the severity of power systems when it is subjected to a three-phase fault. The (RTI) is proposed as an index used to represent severity of transient instability. Risk of transient stability for three-phase fault is calculated using a well-known risk formula. Risk of transient stability provides a quantitative measure to evaluate the potential loss of synchronism of a generator that takes into account the probability and consequences. RTI index is calculated based on the machines rotor angles obtained at each step of a time domain simulation. RTI is proposed as an index to show the severity of the three-phase fault towards transient stability since it allows a fast and accurate measurement of the degree of stability of the system facing a fault. The proposed technique is implemented on the IEEE 39-bus system.

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32

Madiedo, Jose´ M., Jose´ M. Franco, Concepcio´n Valencia, and Crı´spulo Gallegos. "Modeling of the Non-Linear Rheological Behavior of a Lubricating Grease at Low-Shear Rates." Journal of Tribology 122, no. 3 (December 29, 1999): 590–96. http://dx.doi.org/10.1115/1.555406.

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This paper deals with modeling the non-linear rheological behavior of lubricating greases at very low shear rates. With this aim, dynamic linear viscoelastic, non-linear stress relaxation, transient and steady-state shear flow, and transient first normal stress difference measurements have been carried out on a diurea-derivative lubricating grease. A factorable non-linear viscoelasticity model, the Wagner integral model, derived from the K-BKZ constitutive equation, was used in order to predict the non-linear rheological response of the above-mentioned lubricating grease under shear. The time-dependent part of the model was described by its linear relaxation spectrum, whilst two different damping functions (Wagner and Soskey-Winter’s damping functions) were analysed as the strain-dependent factor. The continuous linear relaxation spectrum was estimated, using regularization techniques, from the dynamic linear viscoelasticity functions. The damping function was calculated from non-linear stress relaxation tests. The constitutive model, with Soskey-Winter’s damping function, predicted the steady-state flow curve, the transient shear stress and the transient first normal stress differences of the lubricating grease studied fairly well. [S0742-4787(00)01603-9]

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33

Shuti, Fu, Hu Jiaxiao, Ni Yixin, Chen Jianlin, Xu Youfang, and Zhao Zunlian. "A Practical Transient Stability Program Using Modified Transient Energy Function with HVDC and Load Models." IFAC Proceedings Volumes 26, no. 2 (July 1993): 727–30. http://dx.doi.org/10.1016/s1474-6670(17)48565-x.

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34

Latorre, Ramon, Cristián Zaelzer, and Sebastian Brauchi. "Structure–functional intimacies of transient receptor potential channels." Quarterly Reviews of Biophysics 42, no. 3 (August 2009): 201–46. http://dx.doi.org/10.1017/s0033583509990072.

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AbstractAlthough a unifying characteristic common to all transient receptor potential (TRP) channel functions remains elusive, they could be described as tetramers formed by subunits with six transmembrane domains and containing cation-selective pores, which in several cases show high calcium permeability. TRP channels constitute a large superfamily of ion channels, and can be grouped into seven subfamilies based on their amino acid sequence homology: the canonical or classic TRPs, the vanilloid receptor TRPs, the melastatin or long TRPs, ankyrin (whose only member is the transmembrane protein 1 [TRPA1]), TRPN after the nonmechanoreceptor potential C (nonpC), and the more distant cousins, the polycystins and mucolipins. Because of their role as cellular sensors, polymodal activation and gating properties, many TRP channels are activated by a variety of different stimuli and function as signal integrators. Thus, how TRP channels function and how function relates to given structural determinants contained in the channel-forming protein has attracted the attention of biophysicists as well as molecular and cell biologists. The main purpose of this review is to summarize our present knowledge on the structure of channels of the TRP ion channel family. In the absence of crystal structure information for a complete TRP channel, we will describe important protein domains present in TRP channels, structure–function mutagenesis studies, the few crystal structures available for some TRP channel modules, and the recent determination of some TRP channel structures using electron microscopy.

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35

Earley, Scott, and Joseph E. Brayden. "Transient Receptor Potential Channels in the Vasculature." Physiological Reviews 95, no. 2 (April 2015): 645–90. http://dx.doi.org/10.1152/physrev.00026.2014.

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The mammalian genome encodes 28 distinct members of the transient receptor potential (TRP) superfamily of cation channels, which exhibit varying degrees of selectivity for different ionic species. Multiple TRP channels are present in all cells and are involved in diverse aspects of cellular function, including sensory perception and signal transduction. Notably, TRP channels are involved in regulating vascular function and pathophysiology, the focus of this review. TRP channels in vascular smooth muscle cells participate in regulating contractility and proliferation, whereas endothelial TRP channel activity is an important contributor to endothelium-dependent vasodilation, vascular wall permeability, and angiogenesis. TRP channels are also present in perivascular sensory neurons and astrocytic endfeet proximal to cerebral arterioles, where they participate in the regulation of vascular tone. Almost all of these functions are mediated by changes in global intracellular Ca2+ levels or subcellular Ca2+ signaling events. In addition to directly mediating Ca2+ entry, TRP channels influence intracellular Ca2+ dynamics through membrane depolarization associated with the influx of cations or through receptor- or store-operated mechanisms. Dysregulation of TRP channels is associated with vascular-related pathologies, including hypertension, neointimal injury, ischemia-reperfusion injury, pulmonary edema, and neurogenic inflammation. In this review, we briefly consider general aspects of TRP channel biology and provide an in-depth discussion of the functions of TRP channels in vascular smooth muscle cells, endothelial cells, and perivascular cells under normal and pathophysiological conditions.

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36

Lee, Karen W., Sarah E. Webb, and Andrew L. Miller. "Requirement for a localized, IP3R-generated Ca2+ transient during the furrow positioning process in zebrafish zygotes." Zygote 14, no. 2 (May 2006): 143–55. http://dx.doi.org/10.1017/s0967199406003637.

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SummaryWe report that the first localized Ca2+ transient visualized in the blastodisc cortex of post-mitotic zebrafish zygotes has unique features. We confirm that this initial ‘furrow positioning’ Ca2+ transient precedes the physical appearance of the first cleavage furrow at the blastodisc surface and that it has unique dynamics, which distinguish it from the subsequent furrow propagation transients that develop from it. This initial transient displays a distinct rising phase that peaks prior to the initiation of the two linear, subsurface, self-propagating Ca2+ waves that constitute the subsequent furrow propagation transient. Through the carefully timed introduction of the Ca2+ buffer, dibromo-BAPTA, we also demonstrate the absolute requirement of this initial rising phase Ca2+ transient in positioning the furrow at the blastodisc surface: no rising phase transient, no cleavage furrow. Likewise, the introduction of the inositol 1,4,5-trisphosphate receptor (IP3R) antagonist, 2-aminoethoxydiphenyl borate, eliminates both the rising phase transient and the appearance of the furrow at the cell surface. On the other hand, antagonists of the ryanodine receptor and NAADP-sensitive channels, or simply bathing the zygote in Ca2+-free medium, have no effect on the generation of the rising phase positioning transient or the appearance of the furrow at the surface. This suggests that like the subsequent propagation and deepening/zipping Ca2+ transients, the rising phase furrow positioning transient is also generated specifically by Ca2+ released via IP3Rs. We propose, however, that despite being generated by a similar Ca2+ release mechanism, the unique features of this initial transient suggest that it might be a distinct signal with a specific function associated with positioning the cleavage furrow at the blastodisc surface.

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Strutz, Jasmin, Kathrin Baumann, Elisa Weiss, and Ursula Hiden. "Transient Hyperglycemia and Hypoxia Induce Memory Effects in AngiomiR Expression Profiles of Feto-Placental Endothelial Cells." International Journal of Molecular Sciences 22, no. 24 (December 13, 2021): 13378. http://dx.doi.org/10.3390/ijms222413378.

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Gestational diabetes (GDM) and preeclampsia (PE) are associated with fetal hyperglycemia, fetal hypoxia, or both. These adverse conditions may compromise fetal and placental endothelial cells. In fact, GDM and PE affect feto-placental endothelial function and also program endothelial function and cardiovascular disease risk of the offspring in the long-term. MicroRNAs are short, non-coding RNAs that regulate protein translation and fine tune biological processes. A group of microRNAs termed angiomiRs is particularly involved in the regulation of endothelial function. We hypothesized that transient hyperglycemia and hypoxia may alter angiomiR expression in feto-placental endothelial cells (fpEC). Thus, we isolated primary fpEC after normal, uncomplicated pregnancy, and induced hyperglycemia (25 mM) and hypoxia (6.5%) for 72 h, followed by reversal to normal conditions for another 72 h. Current vs. transient effects on angiomiR profiles were analyzed by RT-qPCR and subjected to miRNA pathway analyses using DIANA miRPath, MIENTURNET and miRPathDB. Both current and transient hypoxia affected angiomiR profile stronger than current and transient hyperglycemia. Both stimuli altered more angiomiRs transiently, i.e., followed by 72 h culture at control conditions. Pathway analysis revealed that hypoxia significantly altered the pathway ‘Proteoglycans in cancer’. Transient hypoxia specifically affected miRNAs related to ‘adherens junction’. Our data reveal that hyperglycemia and hypoxia induce memory effects on angiomiR expression in fpEC. Such memory effects may contribute to long-term adaption and maladaption to hyperglycemia and hypoxia.

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Nilius, Bernd. "Transient Receptor Potential (TRP) Channels in the Brain: the Good and the Ugly." European Review 20, no. 3 (May 2, 2012): 343–55. http://dx.doi.org/10.1017/s1062798711000597.

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The ‘transient receptor potential’ (TRP) multigene family encodes sixspan membrane proteins that function as ion channels in mostly tetrameric structures. Members of this family are conserved from yeast, worm, fly to invertebrate, vertebrate and man. These channels have been stigmatized to function only as cell sensors occupied by sensory function. It turns out that TRP channels fulfil a plethora of cellular functions, including non-sensory functions in our brain. This short paper will highlight the advent of novel ion channels in the brain serving different functions and being significantly involved in the genesis of multiple diseases. We will certainly witness a plethora of the novel roles of this protein family in physiological and pathophysiological functions in our central nervous system.

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Menjívar, M., F. Vilchis, M. Cárdenas, C. Cruz, G. Pérez-Palacios, and J. Pedraza-Chaverrí. "Transient alteration of the reproductive function in nephrotic rats." Life Sciences 61, no. 12 (August 1997): 1159–67. http://dx.doi.org/10.1016/s0024-3205(97)00657-7.

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Shibata, Y. "Evaluation of Anal Sphincter Function after Transient Loop Colostomy." Nippon Daicho Komonbyo Gakkai Zasshi 51, no. 1 (1998): 52–55. http://dx.doi.org/10.3862/jcoloproctology.51.52.

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Younis, Johnny S., Dan Zeevi, Ron Rabinowitz, Neri Laufer, and Joseph G. Schenker. "Transient liver function tests abnormalities in ovarian hyperstimulation syndrome." Fertility and Sterility 50, no. 1 (July 1988): 176–78. http://dx.doi.org/10.1016/s0015-0282(16)60029-8.

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Volkov, Alexander N. "Structure and Function of Transient Encounters of Redox Proteins." Accounts of Chemical Research 48, no. 12 (November 25, 2015): 3036–43. http://dx.doi.org/10.1021/acs.accounts.5b00343.

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ATKINSON, STEVEN A., and JAMES E. FISHER. "LOFT FUEL ROD TRANSIENT DNB PROBABILITY DENSITY FUNCTION STUDIES." Chemical Engineering Communications 37, no. 1-6 (August 1985): 233–47. http://dx.doi.org/10.1080/00986448508911283.

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Lo, K. L., and T. T. Ma. "UPFC damping control strategy based on transient energy function." Electric Power Systems Research 56, no. 3 (December 2000): 195–203. http://dx.doi.org/10.1016/s0378-7796(00)00098-5.

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Pattanaik, P. R., S. Pati, and S. K. Sanyal. "Transient Monitoring Function based Fault Classifier for Relaying Applications." International Journal of Electrical and Computer Engineering (IJECE) 8, no. 6 (December 1, 2018): 4089. http://dx.doi.org/10.11591/ijece.v8i6.pp4089-4095.

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<p>This paper proposes Transient monitoring function (TMF) based fault classification approach for transmission line protection. The classifier provides accurate results under various system conditions involving fault resistance, inception angle, location and load angle. The transient component during fault is measured by TMF and appropriate logics applied for fault classification. Simulation studies using MATLAB®/SIMULINK™ are carried out for a 400 kV, 50 Hz power system with variable system conditions. Results show that the proposed classifier has high classification accuracy. The method developed has been compared with a fault classification technique based on Discrete Wavelet Transform (DWT). The proposed technique can be implemented for real time protection schemes employing distance relaying.</p>

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El-Kady, M. A., C. K. Tang, V. F. Carvalho, A. A. Fouad, and V. Vittal. "Dynamic security assessment utilizing the transient energy function method." IEEE Power Engineering Review PER-6, no. 8 (August 1986): 55–56. http://dx.doi.org/10.1109/mper.1986.5527809.

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AISAKA, Kohzo, Saburo ANDO, Kentaro KOKUBO, Kohsuke YOSHIDA, and Hiroyuki MORI. "Effects of Transient or Occulted Hyperprolactinemia on Luteal Function." Folia Endocrinologica Japonica 62, no. 2 (1986): 117–25. http://dx.doi.org/10.1507/endocrine1927.62.2_117.

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Karlsson, Anders, Henrik Otterheim, and Rodney Stewart. "Transient wave propagation in composite media: Green’s function approach." Journal of the Optical Society of America A 10, no. 5 (May 1, 1993): 886. http://dx.doi.org/10.1364/josaa.10.000886.

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Cameron, E. L., J. C. Tai, and M. Carrasco. "Effects of transient covert attention on the psychometric function." Journal of Vision 1, no. 3 (March 14, 2010): 78. http://dx.doi.org/10.1167/1.3.78.

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Jing Shi, Yuejin Tang, Yajun Xia, Li Ren, Jingdong Li, and Fengshun Jiao. "Energy Function Based SMES Controller for Transient Stability Enhancement." IEEE Transactions on Applied Superconductivity 22, no. 3 (June 2012): 5701304. http://dx.doi.org/10.1109/tasc.2011.2177431.

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