Добірка наукової літератури з теми "Structure du caillot"
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Статті в журналах з теми "Structure du caillot":
Chiore, Valeria. "Les structures matérielles de l’intentionnalité. Bachelard, Caillois, Corbin." Cahiers Gaston Bachelard 8, no. 1 (2006): 121–39. http://dx.doi.org/10.3406/cgbac.2006.1034.
Dionne, Jean-Claude. "Erratiques de dolomie au Cap Colombier, sur la haute Côte-Nord du Saint-Laurent estuarien." Note 55, no. 1 (October 2, 2002): 101–7. http://dx.doi.org/10.7202/005656ar.
Dionne, Jean-Claude. "Les erratiques lointains de l’embouchure du Saguenay, Québec." Géographie physique et Quaternaire 48, no. 2 (November 30, 2007): 179–94. http://dx.doi.org/10.7202/032995ar.
Portier, Natacha. "Stabilité polynômiale des corps différentiels." Journal of Symbolic Logic 64, no. 2 (June 1999): 803–16. http://dx.doi.org/10.2307/2586502.
Felczak, Mateusz. "Ludic guilt, paidian joy: Killing and ecocriticism in the theHunter series." Journal of Gaming & Virtual Worlds 12, no. 2 (June 1, 2020): 183–200. http://dx.doi.org/10.1386/jgvw_00013_1.
Чупова, А. Г. "Denatured Mythology and Artistic “Ambigu”: “Cailles en Sarcophage” by Salvatore Sciarrino." OPERA MUSICOLOGICA, no. 2023 (March 27, 2023): 32–65. http://dx.doi.org/10.26156/om.2023.15.1.003.
Song, Doo Heon, Hae Kyung Rhee, Ji-eun Kim, and Jong Hee Lee. "From Agasa Cristie to Group Image Play-Analysis of Horror Survival Game Panic Room : Escaping from the Den on Emotional Elements Development." International Journal of Electrical and Computer Engineering (IJECE) 8, no. 2 (April 1, 2018): 644. http://dx.doi.org/10.11591/ijece.v8i2.pp644-650.
Van Cutsem, Pierre. "Le fantastique de Maurice Sandoz : Le cas du "Labyrinthe"." Acta Universitatis Lodziensis. Folia Litteraria Romanica 18, no. 1 (October 30, 2023): 199–211. http://dx.doi.org/10.18778/1505-9065.18.15.
Murphy, Dean A., James C. Wilson, and David Moore. "Playing hard: Young men’s experiences of drinking in inner-city Melbourne." Journal of Sociology 53, no. 2 (June 25, 2016): 398–412. http://dx.doi.org/10.1177/1440783316654223.
GROSCLAUDE, F. "Le polymorphisme génétique des principales lactoprotéines bovines. Relations avec la quantité, la composition et les aptitudes fromagères du lait." INRAE Productions Animales 1, no. 1 (February 10, 1988): 5–17. http://dx.doi.org/10.20870/productions-animales.1988.1.1.4430.
Дисертації з теми "Structure du caillot":
Seyve, Landry. "Analyse de la structure du caillot en conditions physiologiques et pathologiques." Thesis, Université Grenoble Alpes (ComUE), 2019. http://www.theses.fr/2019GREAS027.
Physiologically, the blood function of the clot is to stop bleeding following a vascular breach. Initially, platelets stop blood flow, quickly supported by the formation of a fibrin fibers network that strengthens and gives properties to resist the blood pressure and fibrinolysis. Fibrinogen is the basic element of the fibrin network. During a vascular breach, the release of tissue factor triggers the coagulation cascade that results in the conversion of fibrinogen to fibrin monomers by the action of thrombin. These aggregate longitudinally to form protofibrils, then laterally to form a network of fibrin fibers.To date, many stages of the clot formation have been described in detail in the literature, however the mechanisms and driving forces of the lateral aggregation of protofibrils are still poorly understood.During this work, we studied different coagulation profiles: from hypo-coagulant to hyper-coagulant, through the normal profile and using a varied range of techniques: thrombin generation, plasmin generation, Fibrinography, Fibrinography in "fibrinolysis" mode, confocal microscopy, thromboelastometry and X-ray diffraction at small angles.We have highlighted the relationship between the amount of thrombin present during clot formation and the clot structure. Indeed, the more thrombin there is, the lower the protofibrils number per fiber and the greater the number of fibers. In addition, we correlated the initiation time of lateral fibers aggregation in Fibrinography with the initiation of plasmin generation. We have thus demonstrated the production of an abnormal fibrin clot structure in the presence of dabigatran, thanks to the combined use of confocal microscopy and Fibrinography.This multimodal analysis of the clot structure under different conditions provides additional information to the scientific community to better understand the mechanisms of fibrin clot formation
Garcia, gonzalez Xabel. "Influence de la nature du fibrinogène sur la structure et la mécanique du caillot de fibrine." Thesis, Université Grenoble Alpes (ComUE), 2016. http://www.theses.fr/2016GREAI076/document.
Fibrin clot formation is one of the major processes leading to blood clotting. It involves the polymerization of fibrin monomers into a network of fibrin fibres. This network controls the mechanical properties of the clot and serves as a skeleton for wound healing. Environmental factors (pH, concentration, …) have been proved to influence polymerization, however the role of fibrinogen composition on the structure of fibrin remains unexplored. This aspect might be important for the case of cardiovascular pathologies, which present abnormal fibrin structures.We have determined the relation between different sources of fibrinogen with the nano- and micro-metric structural and mechanical properties of fibrin clots. The composition in co-purified proteins of the fibrinogens has no significant importance, however the polydispersity profile controls the multiscale properties of fibrin. Indeed, x-ray scattering, multi-wavelength spectrophotometry and confocal microscopy measurements have proved that fibres from monodisperse fibrinogens are quasi-crystalline, straight and rigid. Fibres from polydisperse fibrinogens are less organised, curbed and less rigid. Finally, the mechanical properties of fibrin showed that the response of clots to deformation, as well as the scenarios of rupture are closely related to the structure, and consequently related to the profiles of polydispersity. This opens outstanding perspectives in many fields such the optimisation of fibrinogen’s use on dysfibrinogenemias or haemorrhages, tissue regeneration or the understanding between the abnormal structure of clots and cardiovascular diseases
Dassi, Carhel. "La fibrinographie : une méthode multi-longueurs d’ondes pour la détermination de la structure du caillot en plasma." Thesis, Université Grenoble Alpes (ComUE), 2016. http://www.theses.fr/2016GREAS028.
The physiological role of the clot is to avoid excessive bleeding in the presence of a vascular breach. Once this function is filled, the clot must be able to be easily destroyed, so that it is not transported in the venous system and does not hamper blood circulation. The formation of a fibrin clot and its lysis are key processes of hemostasis, implying simultaneously the polymerization of the fibrinogen monomers in a fibrin fibers network, and the destruction of this constituted network.Although this network controls the physical and mechanical properties of the clot, its structure at scales smaller than the micron is poorly characterized. The main problem in the physical characterization of clot in clinical settings is the current absence of a quantitative, sensitive and reproducible measurement method.We demonstrated in this work, thanks to our method using several wavelengths, that the analysis of the visible spectra of light transmitted through a clot allows to determine simultaneously, quantitatively and in quasi-physiological conditions, several essential parameters of structure of the fibrin clot, namely the number of protofibrils per fibrin fibers, the radius and the density of fibers, and various times of clotting and lysis of the clot. This method was validated by the results with CV inferior to 6 % under all test conditions and various plasmatic profiles: normal, hypo / hyper coagulant and hypo / hyper fibrinolytic. This demonstrates the robustness and reliability of the measurement method when measuring both clotting and clot lysis.This spectrophotometric method was implemented on a modified automaton dedicated to diagnosis of patients presenting hemostatic disorders. The clinical information and the interests expected from this new test concern at the same time the quality of the fibrin network, its accelerated lysis or its resistance to fibrinolysis, and the resultant of the coagulo-lytic balance
Sefiane, Thibaud. "Vers une meilleure compréhension des mécanismes moléculaires régissant l'action des thérapies non-substitutives dans l'hémophilie A et comparaison avec le facteur VIII." Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASQ004.
Hemophilia A is a bleeding disorder linked to a deficiency in plasma coagulation protein FVIII. For a long time, the standard treatment consisted in replacing the missing FVIII with exogenous FVIII, but the emergence of inhibitors against FVIII has made replacement therapies ineffective in 30% of patients. Recently, the bispecific anti-FIX/anti-FX antibody emicizumab, which mimics the cofactor action of FVIII, was approved. The aim of my thesis was to study how emicizumab compares with FVIII in terms of its molecular and functional properties.First, we analyzed the case of a patient undergoing prophylaxis with emicizumab. After 6 months of treatment, the patient developed hemarthrosis and anti-emicizumab antibodies were identified, leading to a decrease of emicizumab plasma concentration without directly affecting its activity. Further analysis revealed the first clinical case treated by emicizumab with antibodies causing accelerated clearance of emicizumab.In a second part, we investigated the impact of FVIII/FVIII-Fc vs emicizumab on clot formation, stabilization and structure. The previously reported in vitro studies were limited by the absence of cellular components, therefore leading to contradicting results.We developed a murine bleeding model which demonstrated that emicizumab alters the kinetics of clot formation, which could explain the bleeding observed in 5% of patients treated with emicizumab. Microscopic analysis of clot structure indicates that FVIII and FVIII-Fc have a similar impact on the clot structure, while emicizumab, with its unique mode of action, induces morphological differences.Finally, we investigated the potential use of murine bleeding models to determine equivalence between non-factor therapies (emicizumab and anti-TFPI) and FVIII. Indeed, the important question linked to the use of these therapies concerns the determination of possible equivalence with FVIII. As the data from in vitro activity tests did not answer this question, we proposed to assess this equivalence in vivo using a panel of murine bleeding models. The results showed that equivalence varied according to the severity of the model and led us to propose that absolute equivalence was unrealistic. Indeed, it would be more realistic to talk about an equivalence range in terms of FVIII activity
Montout, Chantal. "Le modèle expérimental caille de l'entérocolite ulcéronécrosante du nouveau-né : la structure comparée de l'histologie du tube digestif du nouveau-né humain et de l'oiseau explique-t-elle la validité du modèle ?" Paris 5, 1999. http://www.theses.fr/1999PA05P117.
Книги з теми "Structure du caillot":
Desplaces, Philippe Viguié. C'était hier-- le VIIème arrondissement: Saint-Thomas d'Aquin, Invalides, Ecole militaire, Gros-Caillou. [Charenton-le-Pont]: L.M.-Le Point, 1995.
Panoff, Michel. Les frères ennemis: Roger Caillois et Claude Lévi-Strauss. Paris: Payot, 1993.
Gournay, Brigitte. Vie et histoire du VIIe Arrondissement: Saint-Thomas d'Aquin, Invalides, Ecole militaire, Gros Caillou : histoire, anecdotes, curiosités, monuments, musées ... Paris: Hervas, 1986.
architectes, TNA. De voûtes et de briques: Piscine de la Butte-aux-Cailles : histoire d'un renouveau. Paris: Archibooks, 2014.
Toma, Mihai, and Oana Andrada Alexiu-Toma. Genetica si cancerul. Editura Universitara, 2021. http://dx.doi.org/10.5682/9786062812683.
Частини книг з теми "Structure du caillot":
Kolb, Sarah. "Impossible! Bergson after Duchamp after Caillois." In Speculative Art Histories. Edinburgh University Press, 2017. http://dx.doi.org/10.3366/edinburgh/9781474421041.003.0014.