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Статті в журналах з теми "SoxB"
Friedrich, Cornelius G., Armin Quentmeier, Frank Bardischewsky, Dagmar Rother, Regine Kraft, Susanne Kostka, and Heino Prinz. "Novel Genes Coding for Lithotrophic Sulfur Oxidation of Paracoccus pantotrophus GB17." Journal of Bacteriology 182, no. 17 (September 1, 2000): 4677–87. http://dx.doi.org/10.1128/jb.182.17.4677-4687.2000.
Повний текст джерелаRother, Dagmar, Grazyna Orawski, Frank Bardischewsky, and Cornelius G. Friedrich. "SoxRS-mediated regulation of chemotrophic sulfur oxidation in Paracoccus pantotrophus." Microbiology 151, no. 5 (May 1, 2005): 1707–16. http://dx.doi.org/10.1099/mic.0.27724-0.
Повний текст джерелаRother, Dagmar, Hans-Jürgen Henrich, Armin Quentmeier, Frank Bardischewsky, and Cornelius G. Friedrich. "Novel Genes of the sox Gene Cluster, Mutagenesis of the Flavoprotein SoxF, and Evidence for a General Sulfur-Oxidizing System in Paracoccus pantotrophusGB17." Journal of Bacteriology 183, no. 15 (August 1, 2001): 4499–508. http://dx.doi.org/10.1128/jb.183.15.4499-4508.2001.
Повний текст джерелаTurner, Monte E., Daniel Ely, Jeremy Prokop, and Amy Milsted. "Sry, more than testis determination?" American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 301, no. 3 (September 2011): R561—R571. http://dx.doi.org/10.1152/ajpregu.00645.2010.
Повний текст джерелаHaseeb, Abdul, and Véronique Lefebvre. "The SOXE transcription factors—SOX8, SOX9 and SOX10—share a bi-partite transactivation mechanism." Nucleic Acids Research 47, no. 13 (June 13, 2019): 6917–31. http://dx.doi.org/10.1093/nar/gkz523.
Повний текст джерелаZhu, Chuankun, Lei Zhang, Huaiyu Ding, and Zhengjun Pan. "Transcriptome-wide identification and characterization of the Sox gene family and microsatellites for Corbicula fluminea." PeerJ 7 (October 22, 2019): e7770. http://dx.doi.org/10.7717/peerj.7770.
Повний текст джерелаOgawa, Takuro, Toshinari Furusawa, Ryohei Nomura, Daisuke Seo, Naomi Hosoya-Matsuda, Hidehiro Sakurai, and Kazuhito Inoue. "SoxAX Binding Protein, a Novel Component of the Thiosulfate-Oxidizing Multienzyme System in the Green Sulfur Bacterium Chlorobium tepidum." Journal of Bacteriology 190, no. 18 (July 18, 2008): 6097–110. http://dx.doi.org/10.1128/jb.00634-08.
Повний текст джерелаAwad, Nemat M., A. A. Abd El-Kader, M. Attia, and A. K. Alva. "Effects of Nitrogen Fertilization and Soil Inoculation of Sulfur-Oxidizing or Nitrogen-Fixing Bacteria on Onion Plant Growth and Yield." International Journal of Agronomy 2011 (2011): 1–6. http://dx.doi.org/10.1155/2011/316856.
Повний текст джерелаWang, Yubin, Xiangzhong Luo, Chunjuan Qu, Tao Xu, Guiwei Zou, and Hongwei Liang. "The Important Role of Sex-Related Sox Family Genes in the Sex Reversal of the Chinese Soft-Shelled Turtle (Pelodiscus sinensis)." Biology 11, no. 1 (January 6, 2022): 83. http://dx.doi.org/10.3390/biology11010083.
Повний текст джерелаHeadd, Brendan, and Annette Summers Engel. "Evidence for Niche Partitioning Revealed by the Distribution of Sulfur Oxidation Genes Collected from Areas of a Terrestrial Sulfidic Spring with Differing Geochemical Conditions." Applied and Environmental Microbiology 79, no. 4 (December 7, 2012): 1171–82. http://dx.doi.org/10.1128/aem.02812-12.
Повний текст джерелаДисертації з теми "SoxB"
Richardson, Nainoa. "Sox8 compense la perte de Sox9 pendant le développement testiculaire physiopathologique chez la souris présentant une perte de fonction du gène R-spondin1." Electronic Thesis or Diss., Université Côte d'Azur (ComUE), 2019. http://www.theses.fr/2019AZUR6005.
Повний текст джерелаIn humans and mice, testicular development in XY gonads involves SRY/SOX9 signaling to promote Sertoli cell differentiation and their formation as testis chords. For ovarian development in XX gonads, RSPO1/WNT/beta-catenin signaling is the main pathway for granulosa cell differentiation and their subsequent assembly into follicles. Indeed, XY Sox9 mutant mice develop ovaries, and XX Rspo1 mutant mice develop ovo-testes, a gonad containing a testicular and an ovarian part. In XX Rspo1 mutant mice, ovo-testicular development involves precocious differentiation of some granulosa cells and their and reprogramming as Sertoli cells. Thus, these single mutant studies demonstrated that SOX9 and RSPO1 are required for testicular and ovarian development respectively, and that SRY is dispensable for testicular development in XX Rspo1 mice. Interestingly, gonad development in XY and XX Rspo1 Sox9 double knockout (DKO) mice has challenged the requirement of SOX9 for testicular development. In XX Rspo1 single mutants, it was assumed that Sertoli cell differentiation was SOX9-dependent, but co-inactivation of Sox9 in DKO mice does not impair the ovo-testicular phenotype. For XY Sox9 single mutant mice developing ovaries, co-inactivation of Rspo1 in XY DKO mice rescues the sex reversal, though the testes are hypo-plastic. Thus, in XY and XX Rspo1 Sox9 DKO mice, SOX9 and/or SRY are dispensable for testicular differentiation, indicating that an alternate testis factor exists. For my research project, we hypothesized that a SOX9-related transcription factor, SOX8, acts redundantly for testicular development in XY and XX Rspo1 Sox9 DKO mice. Thus, to first establish redundancy among the SOX factors, we first analyzed their expression in Rspo1 mutant mice lacking Sox8 or Sox9, and then generated and analyzed gonad development in XY and XX Rspo1 Sox8 DKO mice. Then to test our hypothesis, we studied Rspo1 Sox8 Sox9 triple knockout (TKO) mice. We predicted that a loss of both Sox genes in TKO mice would prevent granulosa cell reprogramming as Sertoli cells and subsequent testicular development. To characterize gonad development and their effects in DKO and TKO mice, we performed analyses in embryonic day 17.5 (E17.5) mice, when granulosa-to-Sertoli cell reprogramming begins in XX Rspo1 single mutants; in juvenile post-natal day 10 (P10) mice, when gonad fate is set; and in young adult P40 mice. We examined a variety of parameters including gonad morphology and secondary sex characteristics, as well as gonad organization and cell population by histological and immunostaining analyses. We report that SOX8 and SOX9 are expressed independently in XY and XX Rspo1 Sox9 DKO and Rspo1 Sox8 DKO gonads in embryonic and juvenile mice. Next, XY and XX Rspo1 Sox8 DKO mice developed testes and ovo-testes, indicating that loss of one SOX factor does not impair testicular differentiation, as in XY and XX Rspo1 Sox9 DKO mice. In XY and XX Rspo1 Sox8 Sox9 TKO mice, granulosa-to-Sertoli cell reprogramming was impaired at E17.5 and post-natal gonads lacked testicular development. Thus, SOX8 can compensate for the loss of SOX9 in Rspo1 Sox9 DKO mice. In addition, gonads in XY and XX TKO mice developed as atrophied ovaries, indicating that ovarian fate is partially maintained.In total, we investigated the etiology of pathophysiological testicular development in RSPO1 loss-of-function mice. Remarkably, though SOX8 is dispensable for male sex determination in mice, it can promote testicular differentiation in the absence of SRY and SOX9 because of functional redundancy with SOX9. Thus, in human cases of sex reversal where testicular development cannot be explained by misexpression of SRY or SOX9, SOX8 could be a causative factor
Sandberg, Magnus. "Sox proteins and neurogenesis." Stockholm, 2010. http://diss.kib.ki.se/2010/978-91-7409-873-0/.
Повний текст джерелаRammah-Bouazza, Cyrine. "Implication de SOX9 et de MiniSOX9 dans la tumorigenèse colorectale." Thesis, Montpellier 1, 2012. http://www.theses.fr/2012MON1T020.
Повний текст джерелаSOX9 is an HMG transcription factor known to regulate transcription by binding of its HMG domain to DNA. We previously demonstrated that SOX9 has anti-oncogenic-properties but SOX9 is overexpressed in colon tumors when compared to adjacent healthy tissu. This overexpression appears paradoxical, unless its anti-oncogenic activity cannot be exert. In this study, we report the discovery of MiniSOX9, a new SOX9 splice variant, which is highly expressed in colon tumors. MiniSOX9 acts as a SOX9 dominant negative isoform. Our hypothesis was that MiniSOX9 antagonizes the SOX9 anti-oncogenic activity in tumors.Using tumors cells lines inducible for SOX9 and MiniSOX9 overexpression, we showed that SOX9 reduces cell proliferation, migration and invasion. Surprisingly, MiniSOX9 has no effect on cell proliferation, suggesting that SOX9 effects could be du to his transcriptionnal activity. However, SOX9 and MiniSOX9 decrease cell clonal ability and tumorosphere formation. In this case, it is likely that SOX9 and MiniSOX9 modulate the activity of proteins partners
Farhat, Andalib. "Implication de la voie Prostaglandine D synthase/PGD2/SOX9 dans l'ovaire normal et pathologique et régulation par la signalisation estrogénique." Thesis, Montpellier 2, 2010. http://www.theses.fr/2010MON20206.
Повний текст джерелаThe prostaglandin D2 (PGD2) pathway is involved in numerous biological processes and while it has been identified as a partner of the embryonic sex determining male cascade, the roles it plays in ovarian function remain largely unknown. PGD2 is secreted by two prostaglandin D synthases (Pgds); the male-specific lipocalin (L)-Pgds and the hematopoietic (H)-Pgds. Here, we report the localization of H-Pgds mRNA in the granulosa cells from the primary to pre-ovulatory follicles. We used adult female mice treated with HQL-79, a specific inhibitor of H-Pgds enzymatic activity, to provide evidence of an interaction between H-Pgds-produced PGD2 signaling and FSH signaling. This leads to the activation of steroidogenic Scc and StAR gene expression through increased FshR and LhR receptor expression leading to progesterone secretion. We also identify a role whereby H-Pgds-produced PGD2 is involved in the regulation of follicular growth through inhibition of granulosa cell proliferation in the growing follicles. Indeed, we report an altered H-Pgds expression in human ovarian tumors alongside a partial or complete absence of H-Pgds protein in granulosa cell tumors, suggesting a potential association between decreased levels of H-Pgds expression and a tumoral phenotype. Together, these results show PGD2 signaling to be essential for FSH action within granulosa cells, thus identifying an important and unappreciated role for PGD2 signaling in controlling the balance of proliferation, differentiation and steroidogenic activity of these cells
Agustí, Benito Cristina. "Mecanisme d'activació de fibronectina i LEF1 per Snail1 durant la transició epili-mesènquima." Doctoral thesis, Universitat Pompeu Fabra, 2007. http://hdl.handle.net/10803/7107.
Повний текст джерелаEpithelial to mesenchymal transitions takes place during embryo development and in the late stages of tumorigenesis allowing metastasis formation. These transitions require E-Cadherin downregulation and can be reproduced in cell culture by ectopic expression of Snail1, an E-Cadherin gene repressor. During Snail-induced transition a rapid upregulation of mesenchymal genes such as Fibronectin and LEF1 has been characterized. Forced expression of E-Cadherin strongly down-regulates Fibronectin and LEF1 RNA levels, indicating that an E-Cadherin sensitive cofactor is involved in the activation of these genes. Accordingly, transcription of Fibronectin and LEF1 was dependent on -Catenin and NFB. E-Cadherin over-expression downregulated the transcriptional activity of both factors and decreased their interaction to Fibronectin promoter. Similarly to -Catenin, NFB was detected associated to E-Cadherin and other cell adhesion components. Association of NFB to E-Cadherin required the integrity of this complex; conditions that disrupts adherens junctions, such as Snail over-expression, decreased E-Cadherin-NFB interaction and up-regulates NFB and -Catenin transcriptional activity. Therefore, -Catenin and NFB transcriptional activities are required for expression of the studied mesenchymal genes and these activities are inactivated by immobilizing -Catenin and NFB to functional E-Cadherin structures.
Boopathi, Ramachandran. "Structure de haute résolution du complexe nucleosome-H1 et son interaction avec le facteur de transcription Sox6." Thesis, Université Grenoble Alpes (ComUE), 2016. http://www.theses.fr/2016GREAV020/document.
Повний текст джерелаUnderstanding the structural organization of chromatin is a fundamental issue in the field of gene regulation. X-ray crystallography and other biophysical techniques have enabled understanding of the nucleosome structure nearly at atomic precision. Despite numerous studies, the structural information beyond the nucleosome core particle (NCP) remains elusive. Over the last few decades several attempts have been made to reveal how the linker histone H1 interacts with the nucleosome particles and condenses them into a chromatin fiber. These studies have led to different models describing the position of linker histone H1 on chromatin. Recent advancements in linker histone H1 studies suggest that globular domain of histone H1 (GH1) interacts with the nucleosomal dyad and its C-terminal domain interacts with the linker DNA forming a stem like structure. However, the precise conformation of linker histone H1 and position of other domains still remains unknown.In this study, we resolved the three-dimensional structure of H1-containing nucleosomes by using cryo-electron microscopy (cryo-EM) and X-ray crystallography. We have used the chaperone NAP-1 to deposit linker histone H1 onto nucleosomes reconstituted from recombinant core histones and 197 base-pair of 601 strong nucleosome positioning DNA sequence. Our cryo-EM results showed that association of H1 gives a more compact appearance of the nucleosome as it restricts the mobility of the two linker DNAs keeping them in close proximity and thereby stabilizing contacts between the histone core and nucleotides preceding NCP exit. Our X-ray crystallography results at 7 Ä resolution reveal that the globular domain of histone H1 (GH1) is positioned onto the nucleosome pseudodyad and recognizes the nucleosome core and both linker arms by contacting the DNA backbone in the minor groove. The N- and C-terminal domains of H1 are oriented away from the nucleosome core towards different DNA linkers. We further validated the orientation of GH1 by cross-linking experiments followed after cysteine substitutions mutagenesis, hydroxyl radical footprinting and by molecular docking. Our results reveal the effect of H1 on nucleosome dynamics and also provide a detailed view of the nucleosome stem conformation upon H1 incorporation.We also studyed the nucleosome accessibility of transcription factor Sox6 and the impact of linker histone H1 incorporation to Sox6 binding on nucleosome by using UV laser biphotonic footprinting. Our results reveal that Sox6 HMG domain binds specifically to its consensus binding located deep inside of the nucleosomal DNA, but not at the nucleosomal dyad. Our in vitro footprinting results reveal that the “locking” of DNA linkers by incorporation of histone H1 on nucleosome does not show any impact on Sox6 HMG domain binding, evidencing an alternative to the Widom model based on thermal fluctuation “opening” of the nucleosome at the linkers.. The finding that Sox6 is able to overcome nucleosome (chromatosome) barrier in presence or absence of H1, strongly suggest that the HMG domain - based Sox family proteins it can act as a pioneer factor in transcription regulation, in particular in initiation of cell differentiation
Dupasquier, Sébastien. "SOX9, un lien moléculaire entre voie Wnt/APC et PKCalpha dans l'épithélium intestinal sain et tumoral." Montpellier 1, 2008. http://www.theses.fr/2008MON1T041.
Повний текст джерелаNeirijnck, Yasmine. "Contrôle transcriptionnel du développement rénal par la famille de gènes Sox." Thesis, Nice, 2013. http://www.theses.fr/2013NICE4109.
Повний текст джерелаCongenital abnormalities of the kidney and the urinary tract (CAKUT) belong to the mostcommon birth defects in human and are caused by defects in the program governing organ development. The Sox gene family encodes 20 transcription factors that ensure multiple and essential functions during mouse and human organogenesis. We have previously shown that the homologous genes Sox8 and Sox9 are required for the branching process of the ureter and their loss results in renal agenesis. In this thesis project, we aimed to identify and characterize the role of the Sox-C genes (Sox4/11/12), in vivo using mouse models. Expression analysis revealed that Sox4, Sox11 and Sox12 are coexpressed in the self-renewing nephron precursors cells that are destined to undergo mesenchyme-to-eptihelial transition (MET) to form vesicles that elongate to give rise to the functional nephrons. Phenotypical analysis revealed a functional redundancy between Sox4 and Sox11 in MET and nephron maturation processes: double mutants display renal hypodysplasia, due to a dramatic reduction in the number and size of nephrons. The nephron precursor pool is intact in these mutants but unable to commit to nephrogenesis, probably because of a cell identity change. In addition, in the absence of Sox11, ectopic ureteric buds form, leading to duplex kidneys, a phenotype found in a proportion of CAKUT patients. Importantly, mutation analysis of a cohort suffering from CAKUT syndrome identified a series of SOX11 variants, thus suggesting an involvement of SOX11 mutations in this human disease
Hess, Samuel Joseph. "Sox2 target network in regulating adult Schwann cell plasticity : new insights into peripheral nerve regeneration and pathology." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/25778.
Повний текст джерелаRoberts, Neil Alistair. "The role of SOX9 during human pancreas development." Thesis, University of Manchester, 2011. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-sox9-during-human-pancreas-development(dab5d8da-4c02-4592-b05e-471984461dcc).html.
Повний текст джерелаКниги з теми "SoxB"
SOB. Alexander, AR: Sibling Rivalry Press, 2011.
Знайти повний текст джерелаSeixas, Noémia. Beethoven sob. Lisboa: Ulmeiro, 1989.
Знайти повний текст джерелаDavis, Carol Anne. Sob story. London: Snowbooks, 2006.
Знайти повний текст джерелаSilva, Alencar e. Sob vésper: Poesia. Manaus: Edições Puxirum, 1986.
Знайти повний текст джерелаChicago White Sox. Edina, Minn: ABDO Pub. Co., 2011.
Знайти повний текст джерелаKawai, Mitsuko. Sob dois horizontes. São Paulo: Editora do Escritor, 1988.
Знайти повний текст джерелаItalia, Bob. Boston Red Sox. Edina, MN: Abdo & Daughters, 1997.
Знайти повний текст джерелаChicago White Sox. Minneapolis, Minnesota: Sportszone, an imprint of ABDO Publishing, 2015.
Знайти повний текст джерелаGilbert, Sara. Chicago White Sox. Mankato, MN: Creative Paperbacks, 2014.
Знайти повний текст джерелаAbramson, Phyllis Leslie. Sob sister journalism. New York: Greenwood Press, 1990.
Знайти повний текст джерелаЧастини книг з теми "SoxB"
Lefebvre, Véronique, Benoit de Crombrugghe, and Richard R. Behringer. "The transcription factors L-Sox5 and Sox6 are essential for cartilage formation." In The Many Faces of Osteoarthritis, 91–100. Basel: Birkhäuser Basel, 2002. http://dx.doi.org/10.1007/978-3-0348-8133-3_10.
Повний текст джерелаNongmeikapam, Kishorjit, Wahengbam Kanan Kumar, and Aheibam Dinamani Singh. "Prn-Sorb-Slam." In Autonomous Driving and Advanced Driver-Assistance Systems (ADAS), 167–92. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.1201/9781003048381-8.
Повний текст джерелаChiang, Pen-Chi, and Xiang Gao. "SOx Control." In Air Pollution Control and Design, 7–47. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-13-7488-3_2.
Повний текст джерелаHall, Beverley. "Sorb-German Bilingual Education." In Bilingual Education, 151–56. Dordrecht: Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-4531-2_15.
Повний текст джерелаShimozaki, Koji. "Sox2 (SRY-Box 2)." In Encyclopedia of Signaling Molecules, 5093–100. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101970.
Повний текст джерелаShimozaki, Koji. "Sox2 (SRY-Box 2)." In Encyclopedia of Signaling Molecules, 1–8. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101970-1.
Повний текст джерелаJozsa, Frank P. "Boston Red Sox." In SpringerBriefs in Economics, 9–15. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-25996-3_2.
Повний текст джерелаJozsa, Frank P. "Chicago White Sox." In SpringerBriefs in Economics, 41–48. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-25996-3_6.
Повний текст джерелаRay, Sujay, Arundhati Banerjee, and Angshuman Bagchi. "Molecular Level Insight into the Interactions of SoxC and SoxD from Epsilonproteobacteria Sulfurimonas denitrificans: A Biomolecular Computational Approach." In Advances in Intelligent Systems and Computing, 401–10. New Delhi: Springer India, 2015. http://dx.doi.org/10.1007/978-81-322-2517-1_39.
Повний текст джерелаSchrum, Kelly. "Introduction." In Some Wore Bobby Sox, 1–10. New York: Palgrave Macmillan US, 2004. http://dx.doi.org/10.1007/978-1-349-73134-3_1.
Повний текст джерелаТези доповідей конференцій з теми "SoxB"
Jiang, Shih-Sheng, Wen-Tsen Fang, I.-Shou Chang, and Chih-Ting Huang. "Abstract 1292: Mutually exclusive expression of SOX2 and SOX9 in lung adenocarcinoma cells and its implications." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1292.
Повний текст джерелаHanlin, Heath. "Sorb." In ACM SIGGRAPH 99 Electronic art and animation catalog. New York, New York, USA: ACM Press, 1999. http://dx.doi.org/10.1145/312379.313083.
Повний текст джерелаCapasso, Giulio, Jani Achrén, Mirko Colapietro, Sergio D'Orsi, Sergio Campana, Riccardo Claudi, Pietro Schipani, et al. "SOXS control electronics design." In Software and Cyberinfrastructure for Astronomy V, edited by Juan C. Guzman and Jorge Ibsen. SPIE, 2018. http://dx.doi.org/10.1117/12.2312780.
Повний текст джерелаYoung, David, Marco Landoni, Stephen Smartt, Sergio Campana, Riccardo U. Claudi, Pietro Schipani, Matteo Aliverti, et al. "The SOXS data-reduction pipeline." In Software and Cyberinfrastructure for Astronomy VI, edited by Juan C. Guzman and Jorge Ibsen. SPIE, 2020. http://dx.doi.org/10.1117/12.2561015.
Повний текст джерелаCosentino, Rosario, Matteo Aliverti, Salvatore Scuderi, Sergio Campana, Riccardo U. Claudi, Pietro Schipani, Andrea Baruffolo, et al. "The VIS detector system of SOXS." In Ground-based and Airborne Instrumentation for Astronomy VII, edited by Hideki Takami, Christopher J. Evans, and Luc Simard. SPIE, 2018. http://dx.doi.org/10.1117/12.2312539.
Повний текст джерелаClaudi, Riccardo, Federico Biondi, Nancy Elias-Rosa, Matteo Genoni, Matteo Munari, Kalyan Radhakrishnan, Davide Ricci, et al. "Operational modes and efficiency of SOXS." In Ground-based and Airborne Instrumentation for Astronomy VIII, edited by Christopher J. Evans, Julia J. Bryant, and Kentaro Motohara. SPIE, 2020. http://dx.doi.org/10.1117/12.2562321.
Повний текст джерелаCortes, Carlos, and Hideharu Amano. "Switching region analysis for SOTB technology." In 2017 International Caribbean Conference on Devices, Circuits and Systems (ICCDCS). IEEE, 2017. http://dx.doi.org/10.1109/iccdcs.2017.7959717.
Повний текст джерелаGupta, Sumit. "SOX Compliant Agile Processes." In Agile 2008 Conference. IEEE, 2008. http://dx.doi.org/10.1109/agile.2008.48.
Повний текст джерелаPetersen, Rebecca. "Loss of Sox4 Impacts Early Ocular Development." In Virtual 12th Light Sheet Fluorescence Microscopy Conference 2020. Royal Microscopical Society, 2020. http://dx.doi.org/10.22443/rms.lsfm2020.18.
Повний текст джерелаBrucalassi, Anna, Oz Diner, Hanindyo Kuncarayakti, Adam Rubin, José Antonio Araiza-Durán, Andrea Bianco, Mirko Colapietro, et al. "Architecture of the SOXS instrument control software." In Software and Cyberinfrastructure for Astronomy V, edited by Juan C. Guzman and Jorge Ibsen. SPIE, 2018. http://dx.doi.org/10.1117/12.2310092.
Повний текст джерелаЗвіти організацій з теми "SoxB"
Gupta, R. P., S. K. Gangwal, and G. P. Khare. Fluidized-bed testing of Z-SORB III sorbent. Office of Scientific and Technical Information (OSTI), August 1994. http://dx.doi.org/10.2172/78556.
Повний текст джерелаYuan, Xin. SOX9 Is a Progressive Factor in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2013. http://dx.doi.org/10.21236/ada593774.
Повний текст джерелаPrensner, John. The Role of Sox4 In Prostate Cancer Metastases. Fort Belvoir, VA: Defense Technical Information Center, September 2011. http://dx.doi.org/10.21236/ada560517.
Повний текст джерелаCampbell, W. M., J. J. O`Donnell, S. Katta, T. Grindley, G. Delzer, and G. Khare. Desulfurization of hot fuel with Z-Sorb III sorbent. Office of Scientific and Technical Information (OSTI), December 1993. http://dx.doi.org/10.2172/10192619.
Повний текст джерелаLee, G. K., and R. J. Philp. Low NOx/SOx burner trials: CFB Gagetown. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1988. http://dx.doi.org/10.4095/304408.
Повний текст джерелаLee, G. K. The Rockwell low NOx /SOx burner development. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1986. http://dx.doi.org/10.4095/302630.
Повний текст джерелаOnaitis, Mark. The Role of Sox2 in Lung Cancer Initiation and Progression. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada598798.
Повний текст джерелаOnaitis, Mark. The Role of Sox2 in Lung Cancer Initiation and Progression. Fort Belvoir, VA: Defense Technical Information Center, October 2012. http://dx.doi.org/10.21236/ada599530.
Повний текст джерелаOnaitis, Mark. The Role of Sox2 in Lung Cancer Initiation and Progression. Fort Belvoir, VA: Defense Technical Information Center, October 2011. http://dx.doi.org/10.21236/ada603904.
Повний текст джерелаLee, G. K., and R. J. Philp. Gagetown low NOx/SOx burner project test program. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1987. http://dx.doi.org/10.4095/304366.
Повний текст джерела