Книги: "Skin carcinoma"

1

National Cancer Institute (U.S.), ed. Skin cancers: Basal cell and squamous cell carcinomas. [Bethesda, Md.?]: National Cancer Institute, 1990.

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2

Agnew, Karen L. Skin cancer. Oxford: Health Press, 2005.

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3

Shuck, Carolyn. Saving face: My victory over skin cancer. Forest Dale, Vt: Paul S. Eriksson, 2000.

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4

Society, American Cancer. Basal and squamous cell skin cancer: What you need to know-- now. Atlanta, Ga: American Cancer Society/Health Promotions, 2012.

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5

S, Weber Randal, Miller Michael J. 1955-, and Goepfert Helmuth, eds. Basal and squamous cell skin cancers of the head and neck. Baltimore: Williams & Wilkins, 1996.

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6

National Cancer Institute (U.S.). Nonmelanoma skin cancers: Basal and squamous cell carcinomas. Bethesda, Md: U.S. Department of Health and Human Services, Public Health Service, National Institutes of Health, National Cancer Institute, 1988.

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7

Waes, Carter Van, and Adam B. Glick. Signaling pathways in squamous cancer. New York: Springer, 2011.

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8

Japanese Society for Ultrastructural Cutaneous Biology. Symposium. Merkel cells, Merkel cell carcinoma, and neurobiology of the skin: Proceedings of the 1st Symposium of the Japanese Society for Ultrastructural Cutaneous Biology held in Tokyo, Japan, 24-25 November 1999. Edited by Ono Tomomichi and Suzuki Hiroyuki. Amsterdam: Elsevier, 2000.

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9

National Cancer Institute (U.S.), ed. Nonmelanoma skin cancers: Basal and squamous cell carcinomas. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, National Cancer Institute, 1988.

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10

Matin, Rubeta, Jane McGregor, and Catherine Harwood. Skin cancer. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0259.

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Skin cancer is very common in the UK, and its incidence is rising rapidly. There are two broad classes of primary skin cancer: non-melanoma and melanoma. Non-melanoma skin cancer is the commonest form (100 000 cases diagnosed annually in the UK), accounting for nine out of ten skin cancers and includes basal cell carcinoma (BCC) and squamous cell carcinoma (SCC). Cutaneous melanoma is less common (10 000 cases diagnosed in the UK annually) but confers a significantly worse prognosis and accounts for 75% of skin cancer related deaths. There are also a number of other, rarer, non-melanoma skin cancers (e.g. appendageal carcinomas, Merkel cell carcinoma, sarcomas, vascular malignancies, and cutaneous lymphomas); however, these account for less than 1% of all skin cancers in the UK and so will not be specifically discussed in this chapter. Cutaneous metastases can occur secondary to any internal cancer or, indeed, to skin cancer (e.g. melanoma). In most cases, cutaneous metastasis occurs after the diagnosis of a primary cancer and usually in late stages of the disease but, in some cases, it may be the first presentation, in which case it should prompt a thorough investigation for the primary malignancy.

11

Alam, Murad, Jeremy S. Bordeaux, and Siegrid S. Yu. Merkel Cell Carcinoma. Springer, 2013.

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12

Alam, Murad, Jeremy S. Bordeaux, and Siegrid S. Yu. Merkel Cell Carcinoma. Springer London, Limited, 2013.

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13

Alam, Murad, Jeremy S. Bordeaux, and Siegrid S. Yu. Merkel Cell Carcinoma. Springer, 2016.

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14

Alam, Murad, Jeremy S. Bordeaux, and Siegrid S. Yu. Merkel Cell Carcinoma. Springer, 2013.

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15

Carton, James. Skin pathology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199591633.003.0015.

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Eczemas 286Psoriasis 287Lichen planus 288Erythema multiforme 289Granuloma annulare 290Pemphigus vulgaris 291Bullous pemphigoid 292Dermatitis herpetiformis 293Erythema nodosum 294Pyoderma gangrenosum 295Skin infections 296Benign cutaneous lumps 298Basal cell carcinoma 300Squamous cell carcinoma 301Malignant melanoma ...

16

Sondak. Merkel Cell Carcinoma. World Scientific Publishing Co Pte Ltd, 2010.

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17

Carton, James. Skin pathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0016.

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This chapter discusses skin pathology, including eczema/dermatitis, psoriasis, lichen planus, erythema multiforme, granuloma annulare, pemphigus vulgaris, bullous pemphigoid, dermatitis herpetiformis, erythema nodosum, pyoderma gangrenosum, acne vulgaris, rosacea, skin infections, benign epidermal tumours, benign melanocytic tumours, benign cutaneous soft tissue tumours, benign skin adnexal tumours, basal cell carcinoma, squamous cell carcinoma, malignant melanoma, mycosis fungoides, mycosis fungoides, and dermatofibrosarcoma protuberans.

18

Corner, C., and Peter Hoskin. Skin cancer. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199696567.003.0018.

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Chapter 13 discusses skin tumours and that they differ in their radiotherapy planning from most other sites in that the volume definition is based principally upon clinical examination and the majority will be treated by single applied beams using low-energy X-rays or electrons with clinical verification. Three major histological groups are squamous cell carcinoma, basal cell carcinoma and malignant melanoma with a fourth comprising the rarer entities of adnexal tumours and Merkel cell tumours.

19

Wassberg, Cecilia. Ultraviolet Radiation & Squamous Cell Carcinoma in Human Skin. Uppsala Universitet, 2001.

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20

Green, Adèle C., Catherine M. Olsen, and David J. Hunter. Skin Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0015.

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Skin cancer is one of the few types of cancer for which exposure to the major carcinogen, solar ultraviolet (UV) radiation, is strongly implicated on the basis of descriptive epidemiologic data alone. There are three major forms of skin cancer considered in this chapter—melanoma, basal cell carcinoma (BCC), and squamous cell carcinoma (SCC)—and each appears to have different causal relations to the pattern and total amount of sun exposure. High-intensity UV exposure and long-term UV exposure appear to be involved differentially in the various skin cancers and their subtypes. Underlying molecular mechanisms are becoming better understood, though many aspects like the cells of origin and the exact roles of intermediate lesions like actinic keratoses and nevi remain unclear. Because exposure of skin to UV radiation is modifiable, skin cancers are substantially preventable.

21

Ajithkumar, Thankamma, Ann Barrett, Helen Hatcher, and Natalie Cook. Skin cancer. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235636.003.0011.

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Basal cell carcinoma (BCC) is a slow growing, locally invasive (hence called rodent ulcer) malignant epidermal skin tumour. The exact incidence is difficult to obtain although there is a worldwide trend in increasing incidence. Approximately 1 million new cases are diagnosed per year in the USA....

22

Warwick, David, Roderick Dunn, Erman Melikyan, and Jane Vadher. Skin conditions. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199227235.003.0008.

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Surgical incisions 228Nail 229Benign skin tumours 230Malignant skin tumours 232Squamous cell carcinoma 234Malignant melanoma 236Other malignant skin tumours 240Dupuytren's disease 242The hand is second only to the face in awareness of appearance by both patients and those with whom they interact. Surgeons should always consider this when designing incisions....

23

Carton, James. Gynaecological pathology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199591633.003.0011.

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Vulval skin diseases 186Benign vulval tumours 187Vulval carcinoma 188Vaginal infections 190Vaginal tumours 191Cervical carcinoma 192Cervical screening 194Endometriosis 195Endometrial carcinoma 196Uterine leiomyomas 198Functional ovarian cysts 200Polycystic ovarian syndrome 202Benign ovarian tumours 204Ovarian carcinomas ...

24

Schmults, Chrysalyne D. High-Risk Cutaneous Squamous Cell Carcinoma: A Practical Guide for Patient Management. Springer, 2016.

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25

Schmults, Chrysalyne D. High-Risk Cutaneous Squamous Cell Carcinoma: A Practical Guide for Patient Management. Springer, 2018.

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26

Schmults, Chrysalyne D. High-Risk Cutaneous Squamous Cell Carcinoma: A Practical Guide for Patient Management. Springer London, Limited, 2016.

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27

Basal and squamous cell skin cancers of the head and neck. Media, Pa: Williams & Wilkins, 1995.

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28

Ono, T., H. Suzuki, and Tomomichi Ono. Merkel Cells, Merkel Cell Carcinoma and Neurobiology of the Skin. Elsevier Science Pub Co, 2000.

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29

Carton, James. Gynaecological pathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0012.

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This chapter covers gynaecological pathology and includes vulval skin diseases, benign vulval tumours, vulval carcinoma, vaginal infections, vaginal tumours, cervical carcinoma, cervical screening, endometriosis, endometrial carcinoma, uterine leiomyomas (fibroids), uterine leiomyosarcoma, functional ovarian cysts, benign non-epithelial ovarian tumours, benign epithelial ovarian tumours, borderline epithelial ovarian tumours, ovarian carcinomas, pelvic inflammatory disease, ectopic pregnancy, polycystic ovarian syndrome, hydatidiform mole, and pre-eclampsia.

30

Sierakowski, Adam, and Roderick Dunn. Skin conditions. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757689.003.0008.

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This chapter provides an overview of skin conditions affecting the hand, including nail pathology, benign and malignant skin tumours, and Dupuytren’s disease (DD). Although distortion of the nail occurs most commonly after trauma, nail changes may indicate other systemic causes (e.g. psoriasis), and may occasionally be due to underlying malignancy. Hands are exposed to sunlight and other occupational hazards (chemicals, radiation), and are vulnerable to skin cancer, most commonly squamous cell carcinoma. DD is often familial, commoner in men, and can affect the feet (plantar fibromatosis) and penis (Peyronie’s disease). Discreet areas of DD are now treatable by collagenase injection. Surgery is still indicated to restore function, either by fasciectomy (excision of DD) or dermofasciectomy (fasciectomy plus full thickness skin graft) where skin is involved or there is a secondary skin defect following fasciectomy. Patients should be counselled realistically about the post-operative recovery to full function, and that DD is not curable by surgery.

31

Selected abstracts on diagnosis and therapy of basal cell carcinoma of the skin. [Bethesda, M.D.?]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, International Cancer Research Data Bank, National Cancer Institute, 1987.

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32

Reichrath, Jörg. Molecular Mechanisms of Basal Cell and Squamous Cell Carcinomas. Springer, 2006.

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33

Reichrath, Jörg. Molecular Mechanisms of Basal Cell and Squamous Cell Carcinomas. Springer, 2014.

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34

Glick, Adam B., and Carter Van Waes. Signaling Pathways in Squamous Cancer. Springer, 2014.

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35

Glick, Adam B., and Carter Van Waes. Signaling Pathways in Squamous Cancer. Springer, 2011.

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36

Gilchrest, Barbara A., Karen L. Agnew, and Christopher B., M.D. Bunker. Skin Cancer Fast Facts: All You Need to Keep up to Speed (Fast Facts). Not Avail, 2005.

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37

Prosacco, Dora. Aldara Treatment: Ultimate Guide to Using Aldara in Treating Skin Cancer[Superficial Basal Cell Carcinoma] Actinic Keratosis and Genital Warts. Independently Published, 2022.

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38

Adelstein, David J. Squamous Cell Head and Neck Cancer: Recent Clinical Progress and Prospects for the Future. Humana Press, 2005.

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39

Adelstein, David J. Squamous Cell Head and Neck Cancer: Recent Clinical Progress and Prospects for the Future (Current Clinical Oncology). Humana Press, 2005.

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40

Adelstein, David J. Squamous Cell Head and Neck Cancer: Recent Clinical Progress and Prospects for the Future. Humana Press, 2007.

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41

News, PM Medical Health. 21st Century Complete Medical Guide to Skin Cancer (Basal and Squamous Cell Carcinoma) - Authoritative Government Documents and Clinical References for ... on Diagnosis and Treatment Options. Progressive Management, 2002.

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42

Frisch, Morten. Penile Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0055.

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Penile cancers are rare primary malignancies located on the glans, foreskin, or shaft of the penis, excluding the urethra. The vast majority of penile cancers are epithelial tumors representing histological subtypes of squamous cell carcinoma (SCC). Most penile SCCs are believed to develop through pre-invasive lesions known as penile intraepithelial neoplasia and penile carcinoma in situ. They account for 0.1%–0.3% of all incident cancers (excluding non-melanoma skin cancers) in the United States and other developed countries and up to 1% of all cancers in some countries in sub-Saharan Africa. Penile cancers are rare in men younger than 40 years, and are typically diagnosed among men above age 60. The two most important risk factors are pathological phimosis and infection with high-risk types of human papillomaviruses (HPV), both of which are preventable conditions.

43

Winkler, Nicole S. Intracystic/Intraductal Mass. Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0046.

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This chapter, appearing in the section on nipple, skin, and lymph nodes, reviews the key clinical features, associated imaging findings, imaging protocols and pitfalls, differential diagnoses, and management recommendations for intracystic and intraductal masses. The differential diagnoses of intraductal and intracystic masses are similar, with papilloma the most common cause for both. Therefore, we review descriptive terms for both together, with similar implications and management recommendations. Intracystic masses have a higher association with malignancy than intraductal masses, due in part to overlap of imaging appearances of intracystic mass and complex cystic and solid mass. Topics discussed include intraductal and intracystic masses, evaluation of ducts, papilloma, papillary carcinoma and nipple discharge.

44

Winkler, Nicole S. Nipple Abnormalities. Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0045.

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New nipple retraction and new nipple inversion can be secondary to malignancy, post-surgical change, inflammation, or infection. Paget disease of the nipple is characterized by an inflammatory response of the nipple epidermis to malignant cells extending from ductal carcinoma in the lactiferous sinus. A mass arising within the nipple is rare and usually a variant of a papilloma arising in the nipple (nipple adenoma). This chapter, appearing in the section on nipple, skin, and lymph nodes, reviews the key clinical features, associated imaging findings, imaging protocols and pitfalls, differential diagnoses, and management recommendations for patients presenting with nipple retraction. Topics discussed include imaging features of nipple retraction, both benign and malignant causes of nipple retraction, Paget disease of the nipple, and masses occurring in the nipple.

45

Molecular Mechanisms of Basal Cell and Squamous Cell Carcinomas (Medical Intelligence Unit). Springer, 2007.

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46

Green, Adèle C., and David C. Whiteman. Ultraviolet Radiation. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0014.

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Ultraviolet (UV) radiation is the principal cause of over 95% of keratinocyte cancers (basal cell carcinomas and squamous cell carcinomas of the skin), the most common cancers in white populations worldwide. UV radiation also causes an estimated 60%–90% of cutaneous melanoma, the cancer affecting the skin’s pigment-producing cells. In addition, UV radiation is the major cause of many eye diseases, including ocular cancers and cataract, the commonest cause of blindness, and is responsible for the underlying changes in skin aging, on which billions of dollars are spent annually in efforts to repair the damage. The sun is the principal source of human exposure to UV radiation. However, artificial sources are encountered in a wide range of industrial and medical settings, and increasingly from commercial tanning facilities. By the late twentieth century, nearly epidemic increases in skin cancer incidence had occurred in white populations, especially in Australia and New Zealand.

47

Lane, William I., and Linda Comac. The Skin Cancer Answer: The Natural Treatment for Basal and Sqamous Cell Carcinomas and Keratoses. Avery, 1998.

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48

Lee, Olivia T., Jennifer N. Wu, Frederick J. Meyers, and Christopher P. Evans. Genitourinary aspects of palliative care. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0084.

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Genitourinary tract diseases in the palliative care setting most commonly involve urinary tract obstruction, intractable bleeding, fistulae, and bladder-associated pain. Sources of obstruction in the lower urinary tract include benign prostatic hyperplasia, invasive prostate or bladder cancer, urethral stricture, or bladder neck contracture. Upper tract obstruction includes intraluminal or extraluminal blockage of the renal collecting system and ureters, such as transitional cell carcinoma, fibroepithelial polyps, stricture, stones, pelvic or retroperitoneal malignancy, fibrosis, or prior radiation. Untreated, obstructive uropathy leads to elevated bladder, ureter, and kidney pressures, bladder dysfunction, urolithiasis, renal failure, pyelonephritis, or urosepsis. Intractable haematuria can cause problematic anaemia, frequent transfusions, clot retention, haemorrhagic shock, and death. In addition, urinary tract fistulae such as vesicovaginal and vesicoenteric fistulae are common in patients who have had prior pelvic surgery or radiation especially in the setting of immunocompromise, poor nutrition, and infection. Untreated, these symptoms lead to rash, skin breakdown, ulcers, chronic infection, and sepsis. Lastly, pelvic and bladder pain, depending on aetiology can be treated with oral medications, intravesical therapies, or surgical therapies such as palliative resection or urinary diversion. Selection of tests and treatment modalities in the palliative care setting should be based on using the least invasive means to achieve the most relief in suffering. Some genitourinary conditions are potentially fatal, and in the acute or subacute setting, require re-evaluation of the end-of-life goals and wishes of the patient and family.

49

Kuypers, Dirk R. J., and Maarten Naesens. Immunosuppression. Edited by Jeremy R. Chapman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0281_update_001.

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Combination immunosuppressive therapy produces excellent short-term results after kidney transplantation. Long-term graft survival has improved, but less dramatically. Death with a functioning graft remains the primary cause of graft loss. Dosing of current immunosuppressive therapy balances between careful clinical interpretation of time-driven immunological risk assessments and drug-related toxicity on the one hand, and the use of simple surrogate drug exposure indicators like blood/plasma concentrations on the other. The combined use of calcineurin-inhibitors (CNIs) with mycophenolic acids and corticosteroids has been fine-tuned over the last decade, based on empirically derived observations as well as on the results of large multicentre randomized clinical studies. Corticosteroid withdrawal and avoidance are feasible, at least in patients with a low immunological risk, but CNI-free protocols have had few long-term successes. Some minimization strategies have increased risk of developing acute rejection or (donor-specific) anti-HLA antibodies, with deleterious effects on the graft. Mammalian target of rapamycin inhibitors (mTORi) have shown limited benefit in early CNI replacement regimens and their long-term use as primary drug is hampered by intolerance. In the setting of particular malignant disease occurring after transplantation, such as squamous cell carcinoma of the skin and Kaposi’s sarcoma, mTORi seem promising. Induction agents (anti-interleukin 2 receptor monoclonal antibodies, antithymocyte globulins) effectively diminish the risk of early immunological graft loss in recipients with moderate to high immunological risk but at the price of more infectious or malignant complications. While personalized transplantation medicine is only in its early stages of development, attempts are made to quantitatively measure the clinical degree of immunosuppression, to tailor immunosuppressive therapy more specifically to the patient’s individual profile, and to monitor graft status by use of invasive (e.g. surveillance renal biopsies) and non-invasive biomarkers. These scientific endeavours are a necessity to further optimize the current immunosuppressive therapy which will remain for some time to come.

50

Webber/Sekely. In Vitro Models for Cancer Research: Carcinomas of the Mammary Gland, Uterus, and Skin (Crc Series on Vitro Models for Cancer Research). CRC, 1986.

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Книги з теми "Skin carcinoma"

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