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Статті в журналах з теми "Selectively bred mice"
Smolen, Andrew, Toni N. Smolen, and Jennifer L. van de Kamp. "Sensitivity of inbred and selectively bred mice to ethanol." Alcohol 4, no. 1 (January 1987): 57–62. http://dx.doi.org/10.1016/0741-8329(87)90061-9.
Повний текст джерелаEwalds-Kvist, S. Béatrice M., Ritva-Kajsa Selander, and N. Kenneth Sandnabba. "Sex-Related Coping Responses in Mice Selectively Bred for Aggression." Perceptual and Motor Skills 84, no. 3 (June 1997): 911–14. http://dx.doi.org/10.2466/pms.1997.84.3.911.
Повний текст джерелаKvist, Béatrice. "Learning in Mice Selectively Bred for High and Low Aggressiveness." Psychological Reports 64, no. 1 (February 1989): 127–30. http://dx.doi.org/10.2466/pr0.1989.64.1.127.
Повний текст джерелаBuckman, Jennifer F., and Charles K. Meshul. "Immunocytochemical analysis of glutamate and GABA in selectively bred mice." Brain Research 760, no. 1-2 (June 1997): 193–203. http://dx.doi.org/10.1016/s0006-8993(97)00281-3.
Повний текст джерелаNehrenberg, Derrick L., Ramona M. Rodriguiz, Michel Cyr, Xiaodong Zhang, Jean M. Lauder, Jean-Louis Gariépy, and William C. Wetsel. "An anxiety-like phenotype in mice selectively bred for aggression." Behavioural Brain Research 201, no. 1 (July 2009): 179–91. http://dx.doi.org/10.1016/j.bbr.2009.02.010.
Повний текст джерелаMeek, T. H., B. P. Lonquich, R. M. Hannon, and T. Garland. "Endurance capacity of mice selectively bred for high voluntary wheel running." Journal of Experimental Biology 212, no. 18 (August 28, 2009): 2908–17. http://dx.doi.org/10.1242/jeb.028886.
Повний текст джерелаCrabbe, John C., Lauren C. Kruse, Alexandre M. Colville, Andy J. Cameron, Stephanie E. Spence, Jason P. Schlumbohm, Lawrence C. Huang, and Pamela Metten. "Ethanol Sensitivity in High Drinking in the Dark Selectively Bred Mice." Alcoholism: Clinical and Experimental Research 36, no. 7 (February 8, 2012): 1162–70. http://dx.doi.org/10.1111/j.1530-0277.2012.01735.x.
Повний текст джерелаMathes, Wendy Foulds, Derrick L. Nehrenberg, Ryan Gordon, Kunjie Hua, Theodore Garland, and Daniel Pomp. "Dopaminergic dysregulation in mice selectively bred for excessive exercise or obesity." Behavioural Brain Research 210, no. 2 (July 2010): 155–63. http://dx.doi.org/10.1016/j.bbr.2010.02.016.
Повний текст джерелаKosobud, Ann E., Stephen J. Cross, and John C. Crabbe. "Neural sensitivity to pentylenetetrazol convulsions in inbred and selectively bred mice." Brain Research 592, no. 1-2 (October 1992): 122–28. http://dx.doi.org/10.1016/0006-8993(92)91666-3.
Повний текст джерелаSteward, Michael W., Carolynne Stanley, and Maria D. Furlong. "Antibody affinity maturation in selectively bred high and low-affinity mice." European Journal of Immunology 16, no. 1 (1986): 59–63. http://dx.doi.org/10.1002/eji.1830160112.
Повний текст джерелаДисертації з теми "Selectively bred mice"
Knapman, Alana. "Cognitive Deficits and their Underlying Structural and Functional Alterations in Mice Selectively Bred for High Stress Reactivity." Diss., lmu, 2010. http://nbn-resolving.de/urn:nbn:de:bvb:19-125411.
Повний текст джерелаHeinzmann, Jan-Michael [Verfasser], and Rainer [Akademischer Betreuer] Landgraf. "Central and peripheral aspects of hypothalamic-pituitary-adrenal (HPA) axis dysfunction : insights from mice selectively bred for extremes in stress reactivity / Jan-Michael Heinzmann. Betreuer: Rainer Landgraf." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2012. http://d-nb.info/1028191367/34.
Повний текст джерелаGreen, Alexis Suzanne. "Intravenous Self-Administration of Alcohol in Selectively Bred High- and Low- Alcohol Preferring Mice." Thesis, 2011. http://hdl.handle.net/1805/2612.
Повний текст джерелаGenetic vulnerability to alcoholism is theorized to be caused by multiple interacting genetic loci, each with a small to modest effect combining under certain environmental influences to contribute to vulnerability to ethanol dependence. Animal models such as selectively bred rodent lines can be used to address this hypothesis of genetic vulnerability. High-drinking lines are implicitly assumed by many to be evidence of high ethanol reinforcement without consideration for variables such as differential pre- and post ingestive effects, low response to alcohol or novelty-seeking. Therefore, it is an open question as to whether animal studies support the idea that genetic differences in free-choice drinking are correlated with genetic differences in other assessments of ethanol-reinforced behavior, including those utilizing operant and classical conditioning. Thus, the present study utilizes selectively bred High- and Low- Alcohol Preferring mice tested for operant intravenous alcohol administration to address the hypothesis that High Alcohol Preferring mice would show evidence of greater alcohol reinforcement than their selectively bred opposite, Low Alcohol Preferring mice. Evidence for greater reinforcement was supported by High Alcohol Preferring mice voluntarily pressing a lever to administer an intravenous dose of alcohol in a two lever choice paradigm, administering higher doses of intravenous alcohol, and tracking the location of the active alcohol lever during a lever reversal procedure in comparison to Low Alcohol Preferring mice. This study supports the High- and Low- Alcohol Preferring mice as a useful genetic model of alcohol-related vulnerability even when utilizing a route of administration that bypasses the digestive system.
Carron, Claire R. "A Novel Risky Decision-Making Task in High and Low Alcohol Preferring Mice." Thesis, 2018. http://hdl.handle.net/1805/17957.
Повний текст джерелаDeficits in impulse control and decision-making have been implicated in the development and maintenance of alcohol use disorders (AUDs). Individuals with AUD often make disadvantageous choices under conditions of probabilistic risk. The Iowa Gambling Task (IGT) is often used to measure risky decision-making, in which impaired individuals tend to favor large, infrequent rewards even when punished for these choices, rather than smaller, safer, and more advantageous rewards. It remains poorly understood if these deficits are behaviors under genetic control and if ethanol intoxication may alter decision-making. High and Low Alcohol Preferring (HAP3 and LAP3, respectively) mice were trained on a novel gambling task to investigate these possible influences. In Experiment 1, HAP3s and LAP3s responded for a 0.1% saccharin solution, choosing between a risky and a safe option. Importantly, choosing the risky option was meant to be ultimately disadvantageous. In Experiment 2, these same HAP3 mice responded for saccharin or saccharin plus 10% ethanol. Contrary to hypothesis, LAP3s preferred the risky option more than HAP3s. Alcohol increased preference for the risky lever, but only in male mice. HAP3 preference for the safe lever may be explained by higher motivation to obtain sweet rewards, or higher overall avidity for responding. Ethanol-induced changes in male risk behavior may be explained by higher androgen levels, but further investigation is required. Similarly, continued research is necessary to optimize a risky decision-making task for both lines, and thus investigate possible genetic differences in risk acceptance that correlate with differences in alcohol intake.
Knapman, Alana [Verfasser]. "Cognitive deficits and their underlying structural and functional alterations in mice selectively bred for high stress reactivity / Alana Knapman." 2010. http://d-nb.info/100997419X/34.
Повний текст джерела(5931026), Claire Carron. "A Novel Risky Decision-Making Task in High and Low Alcohol Preferring Mice." Thesis, 2019.
Знайти повний текст джерелаDeficits in impulse control and decision-making have been implicated in the development and maintenance of alcohol use disorders (AUDs). Individuals with AUD often make disadvantageous choices under conditions of probabilistic risk. The Iowa Gambling Task (IGT) is often used to measure risky decision-making, in which impaired individuals tend to favor large, infrequent rewards even when punished for these choices, rather than smaller, safer, and more advantageous rewards. It remains poorly understood if these deficits are behaviors under genetic control and if ethanol intoxication may alter decision-making. High and Low Alcohol Preferring (HAP3 and LAP3, respectively) mice were trained on a novel gambling task to investigate these possible influences. In Experiment 1, HAP3s and LAP3s responded for a 0.1% saccharin solution, choosing between a risky and a safe option. Importantly, choosing the risky option was meant to be ultimately disadvantageous. In Experiment 2, these same HAP3 mice responded for saccharin or saccharin plus 10% ethanol. Contrary to hypothesis, LAP3s preferred the risky option more than HAP3s. Alcohol increased preference for the risky lever, but only in male mice. HAP3 preference for the safe lever may be explained by higher motivation to obtain sweet rewards, or higher overall avidity for responding. Ethanol-induced changes in male risk behavior may be explained by higher androgen levels, but further investigation is required. Similarly, continued research is necessary to optimize a risky decision-making task for both lines, and thus investigate possible genetic differences in risk acceptance that correlate with differences in alcohol intake.
Частини книг з теми "Selectively bred mice"
Nagao, Mototsugu, Jonathan Lou S. Esguerra, Anna Wendt, Akira Asai, Hitoshi Sugihara, Shinichi Oikawa, and Lena Eliasson. "Selectively Bred Diabetes Models: GK Rats, NSY Mice, and ON Mice." In Methods in Molecular Biology, 25–54. New York, NY: Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0385-7_3.
Повний текст джерелаMcCarty, Richard. "Stress and Schizophrenia." In Stress and Mental Disorders: Insights from Animal Models, 225–66. Oxford University Press, 2020. http://dx.doi.org/10.1093/med-psych/9780190697266.003.0008.
Повний текст джерелаЗвіти організацій з теми "Selectively bred mice"
Coyner, Jennifer L. Differential Expression of Phosphorylated Mitogen-Activated Protein Kinase (pMAPK) in the Lateral Amygdala of Mice Selectively Bred for High and Low Fear. Fort Belvoir, VA: Defense Technical Information Center, June 2013. http://dx.doi.org/10.21236/ad1012913.
Повний текст джерела