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Готові списки джерел за темами / Rye Genetics / Книги

Щоб переглянути інші типи публікацій з цієї теми, перейдіть за посиланням: Rye Genetics.

Книги з теми "Rye Genetics"

Автор: Grafiati

Опубліковано: 4 червня 2021

Оновлено: 30 січня 2023

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1

Molski, Bogusław. An analysis of the protein content and its nutritional value in the grains of rye cultivars from collection and the determination of the amino acid composition of selected cultivars. Warszawa: Botanical Garden of the Polish Academy of Sciences, 1987.

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2

Kazman, M. Ebrahim. Eine neue Methode zur Substitution von D-Chromosomen in das A- und B-Genom des hexaploiden Triticale. Göttingen: Cuvillier, 1992.

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3

A, Salmenkova E., and Omelʹchenko V. T, eds. Populi͡at͡sionnai͡a genetika lososevykh ryb. Moskva: Nauka, 1997.

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4

A, Strunnikov V., and Kirpichnikov Valentin Sergeevich, eds. Genetika i selekt͡s︡ii͡a︡ ryb. 2nd ed. Leningrad: Izd-vo "Nauka," Leningradskoe otd-nie, 1987.

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5

Genetic diversity in landraces of rye (Secale cereale L.) and turnip (Brassica rapa L. ssp. rapa) from the Nordic area. Alnarp: Swedish University of Agricultural Sciences, 2000.

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6

Sergeevich, Kirpichnikov Valentin, Institut biologii mori͡a (Akademii͡a nauk SSSR) та Soviet Union Ikhtiologicheskai͡a komissii͡a, ред. Genetika v akvakulʹture: Trudy 3-go Vsesoi͡uznogo soveshchanii͡a po genetike, selekt͡sii i gibridizat͡sii ryb, Tartu, 1986 g. Leningrad: "Nauka," Leningradskoe otd-nie, 1989.

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7

Van Heyningen, Veronica. E diteur scientifique, ed. Advances in genetics. Amsterdam: Elsevier, 2008.

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8

Vsesoi͡uznoe soveshchanie po genetike, selekt͡sii i gibridizat͡sii ryb (3rd 1986 Tartu, Estonia). Geneticheskie issledovanii͡a morskikh gidrobiontov: Materialy III Vsesoi͡uznogo soveshchanii͡a po genetike, selekt͡sii i gibridizat͡sii ryb, senti͡abrʹ 1986 g., Tartu. Moskva: Vses. nauchno-issl. in-t morskogo rybnogo khozi͡aĭstva i okeanografii, 1987.

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9

Makoedov, A. N. Kariologii͡a︡, biokhimicheskai͡a︡ genetika i populi͡a︡t͡s︡ionnai͡a︡ fenetika lososevidnykh ryb Sibiri i Dalʹnego Vostoka: Sravnitelʹnyĭ aspekt. Moskva: UMK "Psikhologii͡a︡", 1999.

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10

T, Leondes Cornelius, ed. Control and dynamic systems. San Diego, Calif: Academic Press, 1998.

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11

Schlegel, Rolf H. J. Rye: Genetics, Breeding, and Cultivation. Taylor & Francis Group, 2016.

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12

Rye: Genetics, Breeding, and Cultivation. Taylor & Francis Group, 2013.

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13

Schlegel, Rolf H. J. Rye: Genetics, Breeding, and Cultivation. Taylor & Francis Group, 2013.

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14

Schlegel, Rolf H. J. Rye: Genetics, Breeding, and Cultivation. Taylor & Francis Group, 2013.

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15

P, Rowland Lewis, ed. Molecular genetics in diseases of brain, nerve, and muscle. New York: Oxford University Press, 1989.

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16

Genetika v akvakulture: Trudy 3-go Vsesoiuznogo soveshchaniia po genetike, selektsii i gibridizatsii ryb, Tartu, 1986 g. "Nauka," Leningradskoe otd-nie, 1989.

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17

S, Zonova A., та Krupkin V. Z, ред. Razvedenie i selekt͡s︡ii͡a︡ ryb v uslovii͡a︡kh industrialʹnykh teplovodnykh khozi͡a︡ĭstv. Leningrad: Promrybvod, 1986.

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18

Lawrence, Davis, ed. Genetic algorithms and simulated annealing. London: Pitman, 1987.

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19

Kariologiia, biokhimicheskaia genetika i populiatsionnaia fenetika lososevidnykh ryb Sibiri i Dalnego Vostoka: Sravnitelnyi aspekt. Psikhologiia, 1999.

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20

Donaghy, Michael, ed. Brain's Diseases of the Nervous System. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.001.0001.

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The definitive guide to clinical neurology, the twelfth edition of Brain's Diseases of the Nervous System provides detailed coverage of the full range of major neurological conditions, and includes updated sections on genetics, development neurology, and re-written introductory chapters.

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21

Molecular Basis of Cardiovascular Disease: A Companion to Braunwald's Heart Disease. Saunders, 2003.

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22

Kretzschmar, William A. Addressing “Emergence” in a HEL Classroom. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190611040.003.0011.

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“Emergence” is the key term from the study of complex systems, a new science currently useful in physics, genetics, evolutionary biology, and economics, but also a perfect fit for the humanities. The science of complexity describes how massive numbers of random interactions can give rise to order, regularities that “emerge” from the interactions without specific causes. This chapter will present an argument for designing History of English Language (HEL) courses that bear “emergence” in mind, offering as an option the story of the language centered on the continual emergence and re-emergence of lexical, phonological, and grammatical forms of English out of the interaction of its speakers and the contingencies of their history.

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23

Yazdani, Shahram, Audrey Kamzan, Deepa Kulkarni, Carlos Lerner, and Charles A. Newcomer, eds. General Pediatrics Board Review. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190848712.001.0001.

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General Pediatrics Board Review is a comprehensive guide for recent residency graduates and re-certifiers preparing for the American Board of Pediatrics (ABP) board exam. The text consists of over 1000 multiple-choice questions, organized into 25 chapters covering pediatrics topics such as fetal and neonatal care, adolescent and young adult medicine, genetics, child maltreatment, pediatric infectious diseases, gastrointestinal disorders, and more. Chapters include questions, answers with detailed explanations, and references to primary or landmark articles to help better navigate a standardized exam. Questions are written in a case-based format that emulates the ABP board exam, and are supplemented by figures, tables, and boxes. A Quick Facts section helps to emphasize important and frequently tested information.

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24

Abed, Riadh, and Paul St John-Smith, eds. Evolutionary Psychiatry. Cambridge University Press, 2022. http://dx.doi.org/10.1017/9781009030564.

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Evolutionary psychiatry attempts to explain and examine the development and prevalence of psychiatric disorders through the lens of evolutionary and adaptationist theories. In this edited volume, leading international evolutionary scholars present a variety of Darwinian perspectives that will encourage readers to consider 'why' as well as 'how' mental disorders arise. Using insights from comparative animal evolution, ethology, anthropology, culture, philosophy and other humanities, evolutionary thinking helps us to re-evaluate psychiatric epidemiology, genetics, biochemistry and psychology. It seeks explanations for persistent heritable traits shaped by selection and other evolutionary processes, and reviews traits and disorders using phylogenetic history and insights from the neurosciences as well as the effects of the modern environment. By bridging the gap between social and biological approaches to psychiatry, and encouraging bringing the evolutionary perspective into mainstream psychiatry, this book will help to inspire new avenues of research into the causation and treatment of mental disorders.

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25

Nishime, Leilani. Seeing Multiracial. University of Illinois Press, 2017. http://dx.doi.org/10.5406/illinois/9780252038075.003.0007.

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This chapter uses the artwork of Kip Fulbeck as a lever to pry open some of the thornier matters surrounding the twinned issues of recognition and state-sponsored discipline. Fulbeck's most famous work, The Hapa Project, is included in the traveling anthropological exhibit “Race: Are We So Different?” The chapter puts Fulbeck's artwork in dialogue with the history of race-based scientific photography and argues that the exhibit's representation of multiracial Asian Americans can provide a counternarrative to the re-racialization of genetic science. Thus, Fulbeck's work demonstrates how audiences might view multiracial visual culture less as an antidote to racial hierarchies and more as a tool that can break open the smooth surface of naturalized and transcendent notions of racial difference.

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26

Liberzon, Israel, and Kerry Ressler, eds. Neurobiology of PTSD: From Brain to Mind. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190215422.001.0001.

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Post-traumatic stress disorder (PTSD) is a maladaptive and debilitating psychiatric disorder characterized by an extreme sense of fear at the time of trauma occurrence, with characteristic re-experiencing, avoidance, and hyperarousal symptoms in the months and years following the trauma. PTSD can occur in up to 25% of subjects who have experienced severe psychological trauma, such as combat veterans, refugees, and assault victims. Why are some people resilient, whereas others develop debilitating PTSD? Notably, PTSD is among the most likely of psychiatric disorders to be understood from the perspective of environmental influences interacting with genetic vulnerability, since diagnosis requires a specific, highly traumatizing, fear-evoking experience. In addition, a large amount of evidence now supports a model in which PTSD can be viewed, in part, as a disorder of fear dysregulation. This is particularly exciting because the neural circuitry underlying fear behavior in mammals is among the most well-understood behavioral circuits in neuroscience. Further, the study of fear behavior and its underlying circuitry has led to rapid progress in understanding learning and memory processes. By combining molecular-genetic approaches with a mechanistic understanding of fear circuitry, great progress is possible in the understanding, diagnosis, and treatment of PTSD. This book examines the basic neural mechanisms that mediate complex responses and adaptations to psychological trauma; it describes what is currently known about how these biological processes are impaired in individuals with PTSD, and how environmental exposure to trauma interacts with the brain to create the syndrome of PTSD.

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27

Hausen, Harald zur. Infections Causing Human Cancer. Wiley & Sons, Limited, John, 2006.

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28

Hausen, Harald zur. Infections Causing Human Cancer: Softcover Edition. Wiley & Sons, Limited, John, 2010.

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29

Hausen, Harald zur. Infections Causing Human Cancer. Wiley & Sons, Incorporated, John, 2007.

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30

Mammen, Andrew L., and Jessica R. Nance. Evaluation of hyperCKaemia. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0007.

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Serum creatine kinase (CK) levels may be elevated in patients with muscle weakness or pain. In asymptomatic patients with CK elevations, the focus should be on identifying reversible causes, followed by investigation for inherited muscle diseases. In asymptomatic patients with an incidental finding of elevated CK, clinicians should look for reversible causes, then re-test the CK after 10 days of rest in the absence of potential triggers. If the CK remains markedly elevated and/or electromyography proves myopathic, a muscle biopsy should be considered. Women of childbearing age with elevation of serum CK should be evaluated for dystrophin mutation. Genetic causes of hyperCKaemia can be pursued with targeted gene sequencing, or whole exome or next generation sequencing. Patients with inherited skeletal muscle diseases may also have associated cardiac disease, so a cardiology evaluation should be considered in all patients with unexplained CK elevations.

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31

Hausen, Harald zur. Infections Causing Human Cancer. Wiley-VCH, 2006.

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32

Hausen, Harald zur. Infections Causing Human Cancer. Wiley & Sons, Incorporated, John, 2007.

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33

Somsen, Han. From Improvement Towards Enhancement. Edited by Roger Brownsword, Eloise Scotford, and Karen Yeung. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780199680832.013.42.

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This chapter discusses a host of what mostly are still isolated ad hoc technology-driven initiatives, usually in support of human (rights) imperatives, which effectively endeavour to engineer and re-engineer living and non-living environments in ways that have no natural, legal, or historical precedent. The umbrella term I propose to capture such initiatives is ‘environmental enhancement’. Potential examples that fit this definition include genetic modification of disease-transmitting mosquitoes to protect human health, solar radiation-management initiatives and other forms of climate engineering to sustain human life on earth, the creation of new life forms to secure food supplies and absorb population growth, and de-extinction efforts that help restore the integrity of ecosystems. The question this paper asks, in the words of Brownsword, is whether conventional environmental law ‘connects’ with environmental enhancement, focusing on EU environmental law, and whether states may be duty-bound to enhance environments in pursuit of human rights imperatives.

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34

Schindler, Thomas E. A Hidden Legacy. Oxford University Press, 2021. http://dx.doi.org/10.1093/oso/9780197531679.001.0001.

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This biography of Esther Zimmer Lederberg highlights the importance of her research work, which revealed the unique features of bacterial sex, essential for our understanding of molecular biology and evolution. A Hidden Legacy relates how, she and her husband Joshua Lederberg established the new field of bacterial genetics together, in the decade leading up to the discovery of the DNA double helix. Their impressive series of achievements include: the discovery of λ‎ bacteriophage and of the first plasmid, known as the F-factor; the demonstration that viruses carry bacterial genes between bacteria; and the elucidation of fundamental properties of bacterial sex. This successful collaboration earned Joshua the 1958 Nobel Prize, which he shared with two of Esther’s mentors, George Beadle and Edward Tatum. Esther Lederberg’s contributions, however, were overlooked by the Nobel committee, an example of institutional discrimination known as the Matilda Effect. Esther Lederberg should also have been recognized for inventing replica plating, an elegant technique that she originated by re-purposing her compact makeup pad as a kind of ink stamp for conveniently transferring bacterial colonies from one petri dish to another. Instead, the credit for the invention is given to her famous husband, or, at best, to Dr. and Mrs. Lederberg. Within a few years of winning the Nobel Prize, Joshua Lederberg divorced his wife, leaving Esther without a laboratory, cut off from research funding, and facing uncertain employment. In response, she created a new social circle made up of artists and musicians, including a new soulmate. She devoted herself to a close-knit musical ensemble, the Mid-Peninsula Recorder Orchestra, an avocation that flourished for over forty years, until the final days of her life.

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35

Inherited Neuromuscular Diseases Translation From Pathomechanisms To Therapies. Springer, 2009.

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36

Morgan Wortham, Simon. Fleeced: Derrida and ‘the Deciding Discourse of Castration’. Edinburgh University Press, 2018. http://dx.doi.org/10.3366/edinburgh/9781474429603.003.0005.

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This chapter explores the relationship of deconstruction to psychoanalysis, and reads the Genet column of Glas in terms of the deconstructibility of ‘the deciding discourse of castration’, as Derrida puts it. The fleece that Genet imagines Harcamone wearing in The Miracle of the Rose takes centre stage, as much as Genet’s flowers. The fleece is both garb and pelt, at once a talismanic scalp, a part that has been brutally cut away, and a covering used to shield or shelter what is vulnerable or exposed. It is both something stolen, and a protective barrier against loss. To get ‘fleeced’ already carries a double and ambiguous set of possible meanings, then, and Derrida puts it to work in the interests of a double-sexed deconstruction of castratability. If the erection cannot ‘fall’ without re-elevating the entire edifice or column of that phallogocentrism of which castration would paradoxically form an uncastratable part, Derrida’s insertion of a deconstructive ‘hole in erection’ exposes to a powerfully deciphering reading this tale of castration’s uncastratability. The chapter reads into the Hegel column of Glas precisely this deconstructibility of a ‘deciding discourse of castration’, notably in terms of the Hegelian interpretation of Antigone’s politics.

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37

Barsoum, Rashad S. Schistosomiasis. Edited by Neil Sheerin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0181_update_001.

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AbstractSchistosomes are blood flukes that parasitize humans, apes, cattle, and other animals. In these definitive hosts they are bisexual, and lay eggs which are shed to fresh water where they complete an asexual cycle in different snails, ending in the release of cercariae which infect the definitive hosts to complete the life cycle.Seven of over 100 species of schistosomes are human pathogens, causing disease in different organs depending on the parasite species. Racial and genetic factors are involved in susceptibility, severity, and sequelae of infection.Morbidity is induced by the host’s immune response to schistosomal antigens. The latter include tegument, microsomal, gut, and oval antigens. The former are important in the process of invasion and establishment of infection, oval antigens in formation of granulomata which lead to fibrosis in different sites, and the gut antigens constitute the main circulating antigens in established infection, leading to immune-complex disease, particularly in the kidneys. The host immunological response includes innate and adaptive mechanisms, the former being the front line responsible for removing 90% of the infecting cercarial load. Adaptive immunity includes a Th1 phase, dominated by activation of an acute inflammatory response, followed by a prolonged Th2 phase which is responsible for immunity to re-infection as well as progression of tissue injury. Switching from Th1 to Th2 phases is controlled by functional and morphological change in the antigen-presenting cells, which is achieved by molecules of host as well as parasitic origin.Many cells participate in parasite killing, but also in the induction of tissue injury. The most potent of these is the eosinophil, which by binding antibodies to the parasite, particularly immunoglobulin E, facilitates parasite elimination. However, this process is complex, including agonist as well as antagonist pathways, which provide escape mechanisms for the parasite to survive, thereby achieving a delicate balance that permits schistosomes to live for decades in the infected host.

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38

Lamas, Santiago. Nitric Oxide, Cell Signaling, and Gene Expression. Taylor & Francis Group, 2005.

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39

(Editor), Santiago Lamas, and Enrique Cadenas (Editor), eds. Nitric Oxide, Cell Signaling, and Gene Expression (Oxidative Stress and Disease). CRC, 2005.

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40

Lamas, Santiago. Nitric Oxide, Cell Signaling, and Gene Expression. Taylor & Francis Group, 2005.

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41

Lamas, Santiago, and Enrique Candenas. Nitric Oxide, Cell Signaling, and Gene Expression. Taylor & Francis Group, 2010.

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42

Alexander, D. J., N. Phin, and M. Zuckerman. Influenza. Edited by I. H. Brown. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0037.

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Influenza is a highly infectious, acute illness which has affected humans and animals since ancient times. Influenza viruses form the Orthomyxoviridae family and are grouped into types A, B, and C on the basis of the antigenic nature of the internal nucleocapsid or the matrix protein. Infl uenza A viruses infect a large variety of animal species, including humans, pigs, horses, sea mammals, and birds, occasionally producing devastating pandemics in humans, such as in 1918 when it has been estimated that between 50–100 million deaths occurred worldwide.There are two important viral surface glycoproteins, the haemagglutinin (HA) and neuraminidase (NA). The HA binds to sialic acid receptors on the membrane of host cells and is the primary antigen against which a host’s antibody response is targeted. The NA cleaves the sialic acid bond attaching new viral particles to the cell membrane of host cells allowing their release. The NA is also the target of the neuraminidase inhibitor class of antiviral agents that include oseltamivir and zanamivir and newer agents such as peramivir. Both these glycoproteins are important antigens for inducing protective immunity in the host and therefore show the greatest variation.Influenza A viruses are classified into 16 antigenically distinct HA (H1–16) and 9 NA subtypes (N1–9). Although viruses of relatively few subtype combinations have been isolated from mammalian species, all subtypes, in most combinations, have been isolated from birds. Each virus possesses one HA and one NA subtype.Last century, the sudden emergence of antigenically different strains in humans, termed antigenic shift, occurred on three occasions, 1918 (H1N1), 1957 (H2N2) and 1968 (H3N2), resulting in pandemics. The frequent epidemics that occur between the pandemics are as a result of gradual antigenic change in the prevalent virus, termed antigenic drift. Epidemics throughout the world occur in the human population due to infection with influenza A viruses, such as H1N1 and H3N2 subtypes, or with influenza B virus. Phylogenetic studies have led to the suggestion that aquatic birds that show no signs of disease could be the source of many influenza A viruses in other species. The 1918 H1N1 pandemic strain is thought to have arisen as a result of spontaneous mutations within an avian H1N1 virus. However, most pandemic strains, such as the 1957 H2N2, 1968 H3N2 and 2009 pandemic H1N1, are considered to have emerged by genetic re-assortment of the segmented RNA genome of the virus, with the avian and human influenza A viruses infecting the same host.Influenza viruses do not pass readily between humans and birds but transmission between humans and other animals has been demonstrated. This has led to the suggestion that the proposed reassortment of human and avian influenza viruses takes place in an intermediate animal with subsequent infection of the human population. Pigs have been considered the leading contender for the role of intermediary because they may serve as hosts for productive infections of both avian and human viruses, and there is good evidence that they have been involved in interspecies transmission of influenza viruses; particularly the spread of H1N1 viruses to humans. Apart from public health measures related to the rapid identification of cases and isolation. The main control measures for influenza virus infections in human populations involves immunization and antiviral prophylaxis or treatment.

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