Добірка наукової літератури з теми "Rat placenta"
Оформте джерело за APA, MLA, Chicago, Harvard та іншими стилями
Ознайомтеся зі списками актуальних статей, книг, дисертацій, тез та інших наукових джерел на тему "Rat placenta".
Біля кожної праці в переліку літератури доступна кнопка «Додати до бібліографії». Скористайтеся нею – і ми автоматично оформимо бібліографічне посилання на обрану працю в потрібному вам стилі цитування: APA, MLA, «Гарвард», «Чикаго», «Ванкувер» тощо.
Також ви можете завантажити повний текст наукової публікації у форматі «.pdf» та прочитати онлайн анотацію до роботи, якщо відповідні параметри наявні в метаданих.
Статті в журналах з теми "Rat placenta"
Trifunović, Svetlana, Branka Šošić Jurjević, Nataša Ristić, Nataša Nestorović, Branko Filipović, Ivana Stevanović, Vesna Begović-Kuprešanin, and Milica Manojlović-Stojanoski. "Maternal Dexamethasone Exposure Induces Sex-Specific Changes in Histomorphology and Redox Homeostasis of Rat Placenta." International Journal of Molecular Sciences 24, no. 1 (December 29, 2022): 540. http://dx.doi.org/10.3390/ijms24010540.
Повний текст джерелаAchur, Rajeshwara N., Sean T. Agbor-Enoh, and D. Channe Gowda. "Rat Spongiotrophoblast-specific Protein Is Predominantly a Unique Low Sulfated Chondroitin Sulfate Proteoglycan." Journal of Biological Chemistry 281, no. 43 (September 5, 2006): 32327–34. http://dx.doi.org/10.1074/jbc.m605841200.
Повний текст джерелаClifton, Vicki L., Phillip C. Owens, Phillip J. Robinson, and Roger Smith. "Identification and characterization of a corticotrophin-releasing hormone receptor in human placenta." European Journal of Endocrinology 133, no. 5 (November 1995): 591–97. http://dx.doi.org/10.1530/eje.0.1330591.
Повний текст джерелаCharest, Phanie L., Vanessa Vrolyk, Pauline Herst, Maryse Lessard, Deborah M. Sloboda, Mathieu Dalvai, Julius Haruna, Janice L. Bailey, and Marie-Odile Benoit-Biancamano. "Histomorphologic Analysis of the Late-term Rat Fetus and Placenta." Toxicologic Pathology 46, no. 2 (February 2018): 158–68. http://dx.doi.org/10.1177/0192623318755135.
Повний текст джерелаBorke, James L., Ariel Caride, Anil K. Verma, Lucky K. Kelley, Carl H. Smith, John T. Penniston, and Rajiv Kumar. "Calcium pump epitopes in placental trophoblast basal plasma membranes." American Journal of Physiology-Cell Physiology 257, no. 2 (August 1, 1989): C341—C346. http://dx.doi.org/10.1152/ajpcell.1989.257.2.c341.
Повний текст джерелаLacroix, MC, H. Jammes, and G. Kann. "Occurrence of a growth hormone-releasing hormone-like messenger ribonucleic acid and immunoreactive peptide in the sheep placenta." Reproduction, Fertility and Development 8, no. 3 (1996): 449. http://dx.doi.org/10.1071/rd9960449.
Повний текст джерелаShearman, Lauren P., Alison M. McReynolds, Feng C. Zhou, and Jerrold S. Meyer. "Relationship between [125I]RTI-55-labeled cocaine binding sites and the serotonin transporter in rat placenta." American Journal of Physiology-Cell Physiology 275, no. 6 (December 1, 1998): C1621—C1629. http://dx.doi.org/10.1152/ajpcell.1998.275.6.c1621.
Повний текст джерелаWhite, Elizabeth A., Joffre B. Baker, Michael McGrogan, and Paul A. Kitos. "Protease Nexin 1 Is Expressed in the Human Placenta." Thrombosis and Haemostasis 69, no. 02 (1993): 119–23. http://dx.doi.org/10.1055/s-0038-1651566.
Повний текст джерелаVaswani, Kanchan, Hsiu-Wen Chan, Hassendrini N. Peiris, Marloes Dekker Nitert, Ryan J. Wood Bradley, James A. Armitage, Gregory E. Rice, and Murray D. Mitchell. "Gestation Related Gene Expression of the Endocannabinoid Pathway in Rat Placenta." Mediators of Inflammation 2015 (2015): 1–9. http://dx.doi.org/10.1155/2015/850471.
Повний текст джерелаSobočan, Nikola, Marta Himelreich-Perić, Ana Katušić-Bojanac, Jure Krasić, Nino Sinčić, Željka Majić, Gordana Jurić-Lekić та ін. "Extended Prophylactic Effect of N-tert-Butyl-α-phenylnitron against Oxidative/Nitrosative Damage Caused by the DNA-Hypomethylating Drug 5-Azacytidine in the Rat Placenta". International Journal of Molecular Sciences 23, № 2 (6 січня 2022): 603. http://dx.doi.org/10.3390/ijms23020603.
Повний текст джерелаДисертації з теми "Rat placenta"
Robinson, N. R. "Calcium transfer across the rat placenta." Thesis, University of Manchester, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.234186.
Повний текст джерелаFreitas, Murilo Rodrigues Barbosa de. "O efeito do selênio em ratas Wistar prenhas infectadas pela cepa Y de Trypanosoma cruzi." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/60/60135/tde-31102014-102620/.
Повний текст джерелаThe selenium (Se) is an essential micronutrient in the diet ofmammals and has an important role in the immune function. A range of 25 selenoproteinshas Sein its structure and most of them in the form of amino acid selenocysteine, being this element involved in the in maintenance of the antioxidant defense. Diet with Se is beneficial in the treatment of diseases correlated with high levels of oxidative stress, like Chagas\' disease, a neglected illness caused by Trypanosoma cruzi. The objective of this study was to evaluate the effects of selenium in the immune response of pregnant Wistar rats infected withtheY strain of T. cruzi. Se treatment triggered enhanced fetal weight and length and placental diameter and weight. It was observed decreased parasitemia. No significant alterations in NO concentrations and amastigote nests in heart were observed. The histological evaluation of placenta displayed an enhanced number of amastigote nests in infected and Se treated animals. The reduction of pro-inflammatory cytokines and T cell populations triggered a Th-2 immune response, which is the hallmark of the gestation period. This fact probably led to the raise in parasite nests in placenta of infected and Se treated animals. So it is possible that the Se supplementation during pregnancy could impair the local placental immune response. Further studies are needed to assess the interaction between selenium and the acute Chagas\' disease during pregnancy.
Taylor, Louise. "Comparative analyses of ABC transporters and metabolising enzymes in human and rat placental models." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/comparative-analyses-of-abc-transporters-and-metabolising-enzymes-in-human-and-rat-placental-models(3daff296-0364-4e4b-89f8-337dac6dbf10).html.
Повний текст джерелаCarter, Wayne Grant. "Site specificity and purification of an insulin receptor associated serine kinase from human placenta and rat liver." Thesis, University of Southampton, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295913.
Повний текст джерелаCisse, Ouma. "Conséquences transgénérationnelles d'une programmation fœtale par dénutrition maternelle et d'un régime hyperlipidique chez le rat : focus sur le placenta." Phd thesis, Université du Droit et de la Santé - Lille II, 2013. http://tel.archives-ouvertes.fr/tel-01064268.
Повний текст джерелаCisse, Ouma. "Conséquences transgénérationnelles d’une programmation fœtale par dénutrition maternelle et d’un régime hyperlipidique chez le rat : focus sur le placenta." Thesis, Lille 2, 2013. http://www.theses.fr/2013LIL2S005/document.
Повний текст джерелаThe concept of DOHaD (Developmental Origins of Health and Disease) which derives from the theory of Barker, replace the origin of metabolic diseases in adults during fetal development and / or perinatal period. Many epidemiological data indicate that maternal dysnutrition (undernutrition, overnutrition) affects fetal growth showing abnormal birth weight (intrauterine growth retardation : IUGR / large birth weight macrosomia) and predispose individuals to development of metabolic diseases. To better understand the mechanisms invovle in transmission of the metabolic vulnerability from one generation to another, we have developed a model combining transgenerational rat fetal programming in the F0 by maternal undernutrition (model FR30) and dysnutrition in F1 with an hyperlipidic diet.Our results show that dietary restriction of 70% throughout pregnancy (FR30 model) contributes to emphasize development of metabolic syndrome traits into female F1 progeny. F1 females from undernourished mothers have an adult glucose intolerance and hyperleptinemia. These metabolic disturbances will be increase by a high fat diet (HF) but will not lead to obesity in female F1 regardless of the mother (F0) diet/statut. This can be explain by the low palatability of the diet, and the lack of carbohydrates largely involve in the development of obesity. These F1 phenoypes conduce to different F2 phenotypes at birth depending of fetal growth trajectory.Newborns from F1 mothers C or FR with HF diet during pregestation and gestation (C HF-HF and FR HF-HF) present IUGR. In contrast, infants born from F1 undernourished mothers with HF diet during pregestation following by standard diet during pregnancy (FR HF-S) show macrosomia. Like metabolic and hormonal disturbances into F1 mothers can not explain themselves the occurrence of these phenotypes, we studied the organ at the interface between maternal and fetal compartments in charge of the crosstalk between mother and fetus : the placenta. Morphological and molecular analysis indicate that placenta is not only sensitive to metabolic changes in the mother, but also able to adapt to the fetus needs. We observe strong correlation between gene expression (decrease or increase) and phenotype (IUGR/macrosomia) with gender specificity.Our work therefore suggests that IUGR or macrosomia phenotypes are not only depending on maternal hormonal and metabolic abnormalities but also in the sex-specific placental gene response to these exposure
Hering, Lydia. "Die Bedeutung des Renin-Angiotensin-Systems im Tiermodell für Präeklampsie." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2012. http://dx.doi.org/10.18452/16550.
Повний текст джерелаDysregulation of the renin-angiotensin-system is important in preeclampsia, a pregnancy specific disorder, characterized by high blood pressure and albuminuria. Aim of this study is to characterize the effects of circulation and uteroplacental renin-angiotensin-system during pregnancy in a rat model. Female rats transgenic for the human angiotensinogen gene crossed with males transgenic for the human renin gene develop preeclampsia, whereas those of the opposite cross do not. We used this model to study the role of angiotensin II in trophoblast invasion, which is shallow in human preeclampsia but deeper in this model. We investigated the following groups: preeclampsia rats, opposite-cross rats, angiotensin II–infused rats and control rats. Angiotensin II infusion increased only circulating angiotensin II levels, opposite cross influenced only uteroplacental angiotensin II and preeclampsia rats showed increased circulating and uteroplacental angiotensin II. Blood pressure and albuminuria occurred in the models with high circulating angiotensin II but not in other models. Control rats showed physiological heart hypertrophy during pregnancy whereas pathological heart hypertrophy occurred in preeclampsia rats. High uteroplacental angiotensin II influenced deep trophoblast invasion in distant spiral arteries whilst the effect of circulating angiotensin II was more diffuse. We then studied human trophoblast cell line and villous explants derived from first-trimester pregnancy. Local angiotensin II dose-dependently increased migration, invasion and motility. The data suggest that angiotensin II stimulates trophoblast invasion in vivo in the rat and in vitro in human cells, a hitherto fore unrecognized function.
Chartrel, Nicolas. "Identification de quelques facteurs impliqués dans l'induction de l'hypotrophie foetale chez la rate rendue expérimentalement diabétique." Rouen, 1989. http://www.theses.fr/1989ROUES016.
Повний текст джерелаNash, Peppi. "Experimental and Clinical Studies of Oxidative Stress in Pre-Eclampsia." Doctoral thesis, Uppsala University, Department of Medical Cell Biology, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7717.
Повний текст джерелаImpaired placentation and oxidative stress are proposed to play major roles in the pathogenesis of pre-eclampsia (PE). It has recently been pointed out that PE might be more than one disease and may have several different pathogeneses. This thesis describes a new animal model for PE and examines the role of oxidative stress in early respective late onset PE.
The effects of Suramin injections on day 10 and 11 of pregnancy were investigated in normal and diabetic rats of two strains (U and H), with or without additional vitamin E treatment. Suramin caused placental dysfunction in both rat strains: foetal growth restriction, increased resorption rate, reduced placental blood flow, and decreased maternal blood volume in the placenta. In the U strain Suramin also caused maternal hypertension and reduced renal blood flow. Oxidative stress in the Suramin treated rats was indicated by increased levels of isoprostane 8-iso-PGF2α in the placenta. Antioxidative treatment with vitamin E partly protected against the effects of Suramin. Streptozotocin-induced diabetes seemed to cause similar placental effects as Suramin, and in the diabetic rats the additional effects of Suramin were only moderate. In conclusion, Suramin-injected pregnant rats constitute a valid animal model for placental dysfunction (U and H rats) and PE (U rats).
Oxidative stress was estimated in women with early onset (≤ 32 weeks) or late onset (≥ 35 weeks) PE, in normotensive pregnant women of respective gestational length, and in healthy non-pregnant women. The ratio of PAI-1/PAI-2 was measured in serum, and the amount of isoprostane 8-iso-PGF2α was measured in placenta, serum, and urine. The ratio of PAI-1/PAI-2 and placental isoprostane levels were higher in women with early onset PE compared with all other groups. Serum levels of isoprostane were similar between groups. Urinary levels of isoprostane were similar in all pregnant women, but lower in non-pregnant women. These data indicate that pregnancy increases general oxidative stress, and that early onset, but not late onset PE, causes increased oxidative stress also in placental tissue. The pathogeneses of early and late onset PE are, therefore, not likely to be identical.
Lippi, Luciana Lucinio. "Estudo da toxicidade da Ipomoea carnea em ratas durante o período perinatal. Avaliação dos possíveis efeitos lesivos no tecido placentário." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/10/10133/tde-17122009-173640/.
Повний текст джерелаIpomoea carnea is a toxic plant widely distributed in Brazil and other tropical countries. During periods of drought, animals graze on this plant which grows even in the presence of adverse climatic conditions. After prolonged periods of plant intake, the animals exhibit a variety of clinical signs as depression, general weakness, body weight loss, staggering gait, muscle tremors, ataxia, posterior paresis, and paralysis. Two kinds of toxic principles were isolated from the plant, the nortropane alkaloids calystegines B1, B2, B3 and C1 and mainly the indolizidine alkaloid swainsonine. The latter alkaloid is a potent inhibitor of two distinct intracellular enzymes, the lysosomal α-mannosidase which results in lysosomal accumulation of incompletely processed oligosaccharides moieties inside vacuoles, which progresses to cellular function loss and, ultimately, to cell death and the Golgi mannosidase II enzyme causes alteration of the N-linked glycoprotein process, modifying the glycoprotein synthesis, processing and carrier. Histologically, cellular vacuolization of Purkinje cells, thyroid follicles, exocrine pancreas, liver and kidney cells have been observed. Recently, many studies in our laboratory have shown that Ipomoea carnea have teratogenic effects in rats, goats and rabbits. However, it is not known yet if the alterations observed in the fetuses are due to alterations in the placenta or if they can be directly related to the transplacental transfer of the active principle. The present study was performed to evaluate the effects of Ipomoea carnea in the placental tissue and in the litter of female rats treated during. Pregnant rats of the experimental groups were treated orally by gavage, once a day from GD6 to GD19, with 1,0; 3,0; 7,0 g/kg of Ipomoea carnea AF. The control and peer-feeding group received tap water by gavage. Total body weight gain, water and food consumption were measured each three days during the experimental period. At the end of pregnancy period some animals were, for cesarean section, mainly for evaluation of placental tissue and the other animals followed the pregnancy until the birth to the term analysis of offspring. From the animals that came from the cesarean section, were collected the fetuses and their placental, those being collected for anatomopathological, histochemistry (lectins and TUNEL) and morphometric analysis, as the fetuses measured about their sizes and weight and assessed to external malformation and bone and visceral analysis and performed the reproductive performance of those females. The pregnants that followed up to the birth of their offspring, which were assessed regarding their physical and reflexology development daily and at days 4, 8, 15 e 22 of lactation, an offspring of each mother was euthanized so representatives fragments of the liver, kidney, SNC and pancreas could be collected for a histopathological assessment, this procedure was also performed at those mothers submitted to the cesarean section at the 20th pregnancy day, as well as at the breastfeeding at the 22nd lactation day. The obtained results clearly show the teratogenic potential produced by the Ipomoea carnea, because both the fetuses as the pups had some congenital anomalies, decrease in birth weight and the delayed negative geotaxis. At both the mother and offsprings liver and kidneys was observed a vacuolar degeneration, showing the maternal and fetal toxicity promoted by the plant exposed during the pregnancy period. An important data noted here refers to the placental tissue assessment, which showed some histopathological alterations, as the labirinth zone thickening and the reduction of the junctional zone thickness, however the vacuolar degeneration was not observed in this organ, although when performed the lectin-histochemistry technique, it was possible to observe the accumulation of some sugars in some cells located at several regions of the placenta, this way showing that this tissue has also suffered some injury promoted by the plant action, not being possible anymore to consider this organs just as a plac of passage for these toxic active principle.
Книги з теми "Rat placenta"
Husain, Shahid Masud. Tr ansport of calcium and magnesium across the placenta of the diabetic rat. Manchester: University of Manchester, 1996.
Знайти повний текст джерелаHudson, Lionel. The Rats of Rangoon: The inside story of the 'fiasco' that took placeat the end of the war in Burma. London: Arrow Bks., 1989.
Знайти повний текст джерелаRizzuto, Gabrielle A., and Anna I. Bakardjiev. Listeria monocytogenes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0020.
Повний текст джерелаKoo, Paul. The regulation of placental glutathione S-transferase (GST-P or -7-7) expression in rat liver in relation to chemical hepatocarcinogenesis. 1992.
Знайти повний текст джерелаЧастини книг з теми "Rat placenta"
Ogle, T. F., T. M. Nosek, and T. M. Mills. "Action of Progesterone and RU38486 on Protein Synthesis in Rat Placenta." In Advances in Experimental Medicine and Biology, 689–92. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5395-9_43.
Повний текст джерелаGambling, L., W. Bruce-Johnson, R. G. Lea, M. J. Bingham, and H. J. McArdle. "Ceruloplasmin Oxidase Expression in the Rowett Hooded rat and Human Placenta During Pregnancy." In Trace Elements in Man and Animals 10, 383–84. New York, NY: Springer US, 2002. http://dx.doi.org/10.1007/0-306-47466-2_119.
Повний текст джерелаMiatello, R. M., M. C. Lama, E. S. González, and H. L. Nolly. "Kallikrein-Like Activity in Nonpregnant and Pregnant Rat Uterus, Fetal Membranes, Placenta and Amniotic Fluid." In Recent Progress on Kinins, 106–13. Basel: Birkhäuser Basel, 1992. http://dx.doi.org/10.1007/978-3-0348-7321-5_14.
Повний текст джерелаWang, Shoou-Lih, Mohan K. Raizada, and Kathleen T. Shiverick. "Alteration of Insulin Receptor Binding and Protein Kinase Activity in Rat Liver and Placenta by ß-Naphthoflavone." In Insulin, Insulin-like Growth Factors, and Their Receptors in the Central Nervous System, 93–106. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5380-5_7.
Повний текст джерелаMasuda, Ryuichi, Michihiro C. Yoshida, and Motomichi Sasaki. "Chromosomal Mapping of the Placental Glutathione S-Transferase Gene and Its Expression in Livers of LEC Rats." In The LEC Rat, 341–48. Tokyo: Springer Japan, 1991. http://dx.doi.org/10.1007/978-4-431-68153-3_37.
Повний текст джерелаMeyer, R. K. "Enzymes in the Placentoma of the Rat." In Ciba Foundation Symposium - Steroid Hormones and Enzymes (Book II of Colloquia on Endocrinology, Vol. 1), 266–70. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470718742.ch7.
Повний текст джерелаDuckworth, Mary Lynn, May C. Robertson, Ingo C. Schroedter, Claude Szpirer, and Henry G. Friesen. "Molecular Genetics and Biology of the Rat Placental Prolactin Family." In Trophoblast Cells, 169–90. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4612-2718-2_12.
Повний текст джерелаWierman, Margaret E., Wei Sun, Chun Wang, David F. Gordon, and William W. Wood. "Control of Rat Gonadotropin Releasing Hormone Promoter Activity in Placental Cells." In Modes of Action of GnRH and GnRH Analogs, 106–15. New York, NY: Springer New York, 1992. http://dx.doi.org/10.1007/978-1-4612-2916-2_7.
Повний текст джерелаSato, Sanai, Susan Old, Deborah Carper, and Peter F. Kador. "Purification and Characterization of Recombinant Human Placental and Rat Lens Aldose Reductases Expressed in Escherichia Coli." In Advances in Experimental Medicine and Biology, 259–68. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1965-2_32.
Повний текст джерелаHazelhoff Roelfzema, W., A. M. Roelofsen, J. H. J. Copius Peereboom-Stegemann, and R. F. M. Herber. "ZINC AND CADMIUM CONCENTRATIONS IN LIVER AND PLACENTAS OF RATS: ARE THEY INTERRELATED?" In Proceedings of the 4. International Workshop, Neuherberg, F. R. G., April 1986, edited by Peter Brätter, 301–10. Berlin, Boston: De Gruyter, 1987. http://dx.doi.org/10.1515/9783111692449-025.
Повний текст джерелаТези доповідей конференцій з теми "Rat placenta"
Novell, Anthony, Vanda Mendes, Arthuis Chloe, Ayache Bouakaz, and Franck Perrotin. "Notice of Removal: Evaluation of utero-placental perfusion in intrauterine growth restriction rat model using CEUS." In 2017 IEEE International Ultrasonics Symposium (IUS). IEEE, 2017. http://dx.doi.org/10.1109/ultsym.2017.8092965.
Повний текст джерелаLukovnikova, L. V., L. A. Lelbiks, and E. E. Lesiovskaya. "EFFECT OF NICKEL AND ITS INSOLUBLE INORGANIC COMPOUNDS ON REPRODUCTIVE FUNCTION OF WHITE RATS." In The 16th «OCCUPATION and HEALTH» Russian National Congress with International Participation (OHRNC-2021). FSBSI “IRIOH”, 2021. http://dx.doi.org/10.31089/978-5-6042929-2-1-2021-1-326-329.
Повний текст джерелаGodal, H. C., F. Brosstad, and B. Holm. "CONGENITAL HYPOFIBRINOGENEMIA, DEEP VEIN THROMBOSIS AND RECURRENT PLACENTAL ABRUPTION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644279.
Повний текст джерелаYadav, Sushma. "Unusual clinical presentation of chriocarcinoma in young patients – Neulological meatastasis." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685389.
Повний текст джерелаPhilips, M., A. G. Juul, S. Thorsen, J. Selmer, and L. Thim. "PURIFICATION AND CHARACTERIZATION OF REACTIVE AND NON-REACTIVE PLASMINOGEN ACTIVATOR INHIBITOR-1 (PAI-1) FROM HUMAN PLACENTA." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642806.
Повний текст джерелаRayer, Debby Jacqueline Jochebed, and dan Jacklin Stella Salome Manoppo. "Production of Superior Pigs as Breeding Stocks by Improving Maternal Uterus and Placental Environment during Pregnancy." In Unima International Conference on Science and Technology 2022. Switzerland: Trans Tech Publications Ltd, 2023. http://dx.doi.org/10.4028/p-x30och.
Повний текст джерелаBajaj, Kanika. "Poster Abstract." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685361.
Повний текст джерелаShipkova, L. N., N. V. Shipkov, S. V. Kalmanovich, and A. K. Cherednichenko. "TOXOPLASMOSIS AMONG THE POPULATION OF THE KRASNODAR TERRITORY." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. All-Russian Scientific Research Institute for Fundamental and Applied Parasitology of Animals and Plant – a branch of the Federal State Budget Scientific Institution “Federal Scientific Centre VIEV”, 2023. http://dx.doi.org/10.31016/978-5-6048555-6-0.2023.24.544-548.
Повний текст джерелаCherkashova, Elvira, Veronika Burunova, Daria Namestnikova, Ilya Gubskiy, Tatiana Bukharova, Diasna Salikhova, Georgy Leonov, et al. "THE EFFECTIVENESS AND DISTRIBUTION OF INTRAVENOUS TRANSPLANTATION OF MESENCHYMAL STEM CELLS DERIVED FROM HUMAN PLACENTA AND NEURAL PROGENITOR CELLS DERIVED FROM INDUCED PLURIPOTENT STEM CELLS IN RATS WITH FOCAL CEREBRAL ISCHEMIA." In XVII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2021. http://dx.doi.org/10.29003/m2398.sudak.ns2021-17/417-418.
Повний текст джерела"Gestational choriocarcinoma after term pregnancy: A case report." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685341.
Повний текст джерелаЗвіти організацій з теми "Rat placenta"
Sophia Johnson, Sophia Johnson. Human placentophagy: What is the microbial composition of raw and processed placental tissue? Experiment, May 2018. http://dx.doi.org/10.18258/11227.
Повний текст джерелаBaszler, Timothy, Igor Savitsky, Christopher Davies, Lauren Staska, and Varda Shkap. Identification of bovine Neospora caninum cytotoxic T-lymphocyte epitopes for development of peptide-based vaccine. United States Department of Agriculture, March 2006. http://dx.doi.org/10.32747/2006.7695592.bard.
Повний текст джерела