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Добірка наукової літератури з теми "Pulmonary alveoli Cytology"

Автор: Grafiati
Опубліковано: 10 грудня 2022
Оновлено: 29 січня 2023

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Статті в журналах з теми "Pulmonary alveoli Cytology"

1

Kavarnos, Ioannis, Dimitra Pardali, Georgia D. Brellou, Elias Papadopoulos, Maria Kritsepi-Konstantinou, and Katerina K. Adamama-Moraitou. "Bronchoscopy and Lung Fine-Needle Aspiration for Antemortem Evaluation of Pulmonary Involvement in Dogs with Naturally Occurring Canine Leishmaniosis." Pathogens 11, no. 3 (March 17, 2022): 365. http://dx.doi.org/10.3390/pathogens11030365.

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Анотація:
Clinical manifestations from the lower respiratory tract are rare in canine leishmaniosis (CanL), making bronchoscopy and lung fine-needle aspiration (FNA) seldomly justified. The aim of this prospective study was to investigate the involvement of Leishmania infantum in the lungs of dogs with naturally occurring CanL by bronchoscopy and examination of bronchoalveolar lavage fluid (BALF), bronchial mucosa biopsies, and FNA, using immunodiagnostics. Dogs with relevant concurrent diseases and azotemia were excluded. Cough was detected in 5/31 (16.1%) dogs. Lesions (hyperemia, edema, mucosal granularity, secretions) were identified upon bronchoscopy in 19/31 (61.3%) dogs. The cytology of BALF revealed histiocytic inflammation in 14/31 (45.2%) dogs; the parasite was identified in one dog (3.2%). The immunofluorescence antibody test in BALF was positive in 15/31 (48.4%) dogs. Histopathology of bronchial mucosa and/or adjacent alveoli revealed lesions (mononuclear cell infiltration, fibrosis, edema, thickening of the inter-alveolar septa) in 24/31 (77.4%) dogs, with no Leishmania amastigotes. Positive antigen staining was observed within the cytoplasm of mononuclear cells in immunocytochemistry and immunohistochemistry. Μononuclear cells showed antigenic positivity in bronchial mucosa (27/31; 87.1%), BALF (30/31; 96.8%), and lung FNA (27/31; 87.1%). In conclusion, lungs seem to be affected from CanL more commonly than previously believed, and bronchoscopy allows obtaining valuable samples for antemortem diagnosis.
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2

Laucirica, Rodolfo, and Mary L. Ostrowski. "Cytology of Nonneoplastic Occupational and Environmental Diseases of the Lung and Pleura." Archives of Pathology & Laboratory Medicine 131, no. 11 (November 1, 2007): 1700–1708. http://dx.doi.org/10.5858/2007-131-1700-conoae.

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Abstract Context.—Cytologic examination of the respiratory tract has been a useful diagnostic tool when evaluating neoplastic lesions of the respiratory tract. However, we have limited experience in the application of this technique in the management of nonneoplastic occupational and environmental diseases of the lung and pleura. This review focuses on the cytologic characteristics of a variety of occupational lung diseases, grouping them into 2 broad diagnostic categories: reactive cellular changes and noncellular elements. The former includes entities such as reactive mesothelial proliferation, goblet cell metaplasia, Creola bodies, and reserve cell hyperplasia, and the latter encompasses Curschmann spirals, Charcot-Leyden crystals, and asbestos bodies. Objective.—To illustrate the cytologic features of several nonneoplastic occupational and environmental diseases and correlate the cytology with various etiologic agents. Data Sources.—Case-derived material and literature review. Conclusions.—The role of cytology in the diagnosis of nonneoplastic occupational and environmental lung diseases is limited. This may be because more than one agent can elicit a similar host reaction and/or the offending agent can be associated with more than one pathologic process. However, in the appropriate clinical and radiographic setting, the cytology can render valuable diagnostic information. Examples include pulmonary alveolar proteinosis in patients with acute silicoproteinosis and asbestos bodies in bronchoalveolar lavage samples of patients with asbestos exposure.
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3

Yan, Lei, Prih Rohra, Lin Cheng, and Paolo Gattuso. "Pleural Effusion in Pulmonary and Extrapulmonary Blastomycosis." Acta Cytologica 64, no. 3 (July 2, 2019): 241–47. http://dx.doi.org/10.1159/000500973.

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Objective: Pleural effusion secondary to blastomycosis infection is an uncommon clinical manifestation of the disease. We undertook a retrospective study to assess the incidence and involvement of pleural effusion in patients with blastomycosis infection. Study Design: Institutional cytology and surgical pathology records were searched from December 1995 to October 2017 for cases of blastomycosis. The cytologic, surgical pathology, and clinical pertinent information was reviewed in detail. Results: A total of 77 cases of blastomycosis infection were recorded, with a male-to-female ratio of 1.7:1.0. Forty-eight cases of blastomycosis were pulmonary (62.3%), while 29 cases of blastomycosis were found in extrapulmonary sites (37.7%). The diagnosis of pulmonary blastomycosis was established by 24 lung biopsies/wedge resections, 22 bronchial alveolar lavages, and 2 lung fine needle aspirations. The 29 cases of extrapulmonary blastomycosis included 13 cases of bone (44.8%), 8 cases of skin (27.6%), 6 cases of soft tissue (20.7%), and 2 cases of brain infections (6.8%). Twenty-eight of 48 pulmonary cases were complicated by unilateral or bilateral pleural effusion (58.3%) detected by imaging studies. Four of the 28 pleural effusions were aspirated and examined by cytology. Two of the 4 pleural fluid cytologies showed involvement by blastomycosis (50%). In the extrapulmonary blastomycosis group, 9 of 29 patients showed unilateral or bilateral pleural effusions (31.0%), including 4 cases of bone, 4 cases of skin, and 1 case of brain involvement. Only 2 of the 9 pleural effusions were aspirated for cytology study. One of the 2 pleural fluid cytologies showed blastomycosis (50%). Conclusion: Pleural effusion detected by imaging is common in blastomycosis patients. Blastomycosis can involve pleural fluid in both pulmonary and extrapulmonary diseases. A broad infectious differential that includes blastomycosis should be considered to make a timely diagnosis and initiate antifungal therapy to prevent systemic infection and further dissemination of the disease.
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4

Burkhalter, Ann, Jan F. Silverman, Marbry B. Hopkins, and Kim R. Geisinger. "Bronchoalveolar Lavage Cytology in Pulmonary Alveolar Proteinosis." American Journal of Clinical Pathology 106, no. 4 (October 1, 1996): 504–10. http://dx.doi.org/10.1093/ajcp/106.4.504.

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5

Haley, P. J., B. A. Muggenburg, A. H. Rebar, G. M. Shopp, and D. E. Bice. "Bronchoalveolar Lavage Cytology in Cynomolgus Monkeys and Identification of Cytologic Alterations Following Sequential Saline Lavage." Veterinary Pathology 26, no. 3 (May 1989): 265–73. http://dx.doi.org/10.1177/030098588902600312.

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Total and differential cell counts were determined on cytolytic specimens obtained by fiberoptic bronchoscopy and bronchoalveolar lavage (BAL) of five normal cynomolgus monkeys. Total nucleated cell counts ranged from 100 to 430 cells/μl. Macrophages were approximately 91% of total nucleated cells, while lymphocytes were 3%, neutrophils 4%, and eosinophils 2% of the initial BAL from each monkey. Less than 1% of the cells were mast cells and ciliated or nonciliated epithelial cells. The effects of repeated saline BAL on pulmonary cell populations were evaluated. Saline lavage of individual lung lobes resulted in a marked rise in circulating blood neutrophils at 4 hr after BAL; there was a similar rise in neutrophils in lavage fluids 24 hr after the initial lavage. Differential and total cell counts of both blood and lavage fluid returned to normal if subsequent lavages were spaced at 48-hr intervals. Lymphocytes were not present in saline-lavaged lung lobes, and protein levels of lavage fluids did not rise significantly. BAL produced a transient, reversible, intra-alveolar influx of neutrophils which was preceded by mobilization of bone marrow-stored neutrophils. Neutrophilia in the lavage fluid and blood was not detectable if lavage and blood sampling procedures were done at 48-hr intervals (which did not alter Ia antigen expression among BAL cells). These observations indicate that BAL is a valid method for sampling and assessing pulmonary cellular and fluid constituents if the procedures are done at intervals of at least 48 hr.
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6

González, Eva Tejerina, Rosario Sánchez-Yuste, and José A. Jiménez-Heffernan. "Cytologic Features of Pulmonary Alveolar Adenoma." Acta Cytologica 52, no. 6 (2008): 739–40. http://dx.doi.org/10.1159/000325634.

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7

Kishore, Manjari, Manju Kaushal, Desh Deepak, and Manju Kumari. "Cytopathological examination of bronchoalveolar lavage fluid in diagnosis of pulmonary alveolar proteinosis." Journal of Laboratory Physicians 10, no. 01 (January 2018): 109–12. http://dx.doi.org/10.4103/jlp.jlp_109_17.

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Анотація:
AbstractPulmonary alveolar proteinosis (PAP) is a rare disease characterized by the deposition of extracellular lipoproteinaceous material within the air spaces. Although the diagnosis is mainly based on histopathological findings, sometimes, the diagnostic yield of transbronchial and even open lung biopsy can be unsatisfactory. The advantage with bronchoalveolar lavage (BAL) cytology is that apart from being safer for the patient, it can sample a much wider area and help in giving an early diagnosis and treatment to the patient. Herein, we present a case of PAP diagnosed on BAL fluid cytology in an elderly female.
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8

MERMOLJA, M., T. ROTT, and A. DEBELJAK. "Cytology of Bronchoalveolar Lavage In Some Rare Pulmonary Disorders: Pulmonary Alveolar Proteinosis and Amiodarone Pulmonary Toxicity." Cytopathology 5, no. 1 (February 1994): 9–16. http://dx.doi.org/10.1111/j.1365-2303.1994.tb00122.x.

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9

Prusty, B. Saroj Kumar, and Majed Abdul Basit Momin. "Dengue Fever with pulmonary alveolar hemorrhage - Common infection with uncommon association - rare case report." Bangladesh Journal of Medicine 30, no. 1 (January 22, 2019): 38–40. http://dx.doi.org/10.3329/bjmed.v30i1.39922.

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Анотація:
Dengue fever (DF) is major health hazard around the world in recent decades especially in India. Flu like illness to polyserositis among common presentation of DF. Dengue fever presenting as pulmonary hemorrhage is rare. Here we present a case of 16 -year young male patient presented with fever and shortness of breath, initially diagnosed as dengue fever by serological examination. CT chest findings mimiking as pneumonia ,however further fall in hemoglobin and progressive dysnoea, bronchoscopy and bronchial wash cytology done which reveal association of pulmonary hemorrhage. This case report alerted clinicians about rare presentation and high index of suspicion of pulmonary hemorrhage in DF with dysnoea for early diagnosis and to avoid its fatal outcome. Bangladesh J Medicine Jan 2019; 30(1) : 38-40
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10

Medenica, Milic, and Miras Medenica. "Diffuse alveolar hemorrhage in isolated pulmonary capillaritis: Case report." Srpski arhiv za celokupno lekarstvo 142, no. 3-4 (2014): 233–38. http://dx.doi.org/10.2298/sarh1404233m.

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Анотація:
Introduction. Pulmonary capillaritis is a small-diameter vessel vasculitis of the lung, which may occur in isolation as in isolated pauci-immune capillaritis, usually associated with the systemic vasculitis but it could be also related to collagen vascular diseases and in lung transplant rejection. Pulmonary capillaritis leads to diffuse alveolar hemorrhage. The clinical presentation includes symptoms like dyspnea, cough, pleuritic chest pain, fever and hemoptysis. Case Outline. A 48 year-old female patient, smoker, presented with progressive dyspnea. Serum tests for infectious diseases, collagen disorders and vasculitis were negative. Radiography and computed tomography of the chest showed diffuse alveolar infiltrates. Cytology of bronchoalveolar lavage showed presence of siderophages. A thoracoscopic lung biopsy was performed to clarify the diagnosis. The histopathological findings showed capillaritis and diffuse intraalveolar hemorrhage. Patient was treated with steroids, and good clinical and minimal radiographic response was obtained. Recently described pauci-immune pulmonary capillaritis has been characterized as p-ANCA (antineutrophil cytoplasmic antibodies) negative isolated pulmonary capillaritis. Conclusion. Isolated pauci-immune pulmonary capillaritis is a rare disease. First clinical manifestations of the isolated pulmonary capillaritis were the symptoms of progressive dyspnea, radiographic and functional signs of the interstitial fibrosis. At the same time, the signs of extrapulmonary diseases were not found. Presence of siderophages in bronchoalveolar lavage indicated alveolar hemorrhage. Histopathological tests of the sample of the lung pointed to pulmonary capillaritis and intraalveolar hemorrhage. Prolonged treatment with corticosteroids was necessary.
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Більше джерел

Книги з теми "Pulmonary alveoli Cytology"

1

J, Henry-Stanley Michelle, and Iber Conrad, eds. Bronchoalveolar lavage: Cytology and clinical applications. New York: Igaku-Shoin, 1991.

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2

M, Effros Richard, and Chang H. K. 1940-, eds. Fluid and solute transport in the airspaces of the lungs. New York: M. Dekker, 1994.

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3

Chang, H. K. Fluid and Solute Transport in the Airspace of the Lungs (Lung Biology in Health and Disease). Informa Healthcare, 1994.

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