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Статті в журналах з теми "Pro-inflammatory cytokine TNF -α"
Dumaine, Jennifer E., and Noah T. Ashley. "Acute sleep fragmentation does not alter pro-inflammatory cytokine gene expression in brain or peripheral tissues of leptin-deficient mice." PeerJ 6 (February 19, 2018): e4423. http://dx.doi.org/10.7717/peerj.4423.
Повний текст джерелаChen, Wei, Prabhu Balan, and David G. Popovich. "The Effects of New Zealand Grown Ginseng Fractions on Cytokine Production from Human Monocytic THP-1 Cells." Molecules 26, no. 4 (February 22, 2021): 1158. http://dx.doi.org/10.3390/molecules26041158.
Повний текст джерелаParamalingam, Sivalingam Suppiah, Julian Thumboo, Sheila Vasoo, Szu Tien Thio, Connie Tse, and Kok-Yong Fong. "In vivo Pro- and Anti-inflammatory Cytokines in Normal and Patients with Rheumatoid Arthritis." Annals of the Academy of Medicine, Singapore 36, no. 2 (February 15, 2007): 96–99. http://dx.doi.org/10.47102/annals-acadmedsg.v36n2p96.
Повний текст джерелаPakdemirli, Ahu, and Gizem Calibasi Kocal. "TNF-alpha Induces Pro-Inflammatory Factors in Colorectal Cancer Microenvironment." Medical Science and Discovery 7, no. 4 (April 30, 2020): 466–69. http://dx.doi.org/10.36472/msd.v7i4.368.
Повний текст джерелаMusa, Fauzia, Nathan Shaviya, Fidelis Mambo, Collins Abonyo, Erick Barasa, Philemon Wafula, George Sowayi, Mustafa Barasa, and Tom Were. "Cytokine profiles in highly active antiretroviral treatment non-adherent, adherent and naive HIV-1 infected patients in Western Kenya." African Health Sciences 21, no. 4 (December 14, 2021): 1584–92. http://dx.doi.org/10.4314/ahs.v21i4.12.
Повний текст джерелаSutcigil, Levent, Cagatay Oktenli, Ugur Musabak, Ali Bozkurt, Adnan Cansever, Ozcan Uzun, S. Yavuz Sanisoglu, et al. "Pro- and Anti-Inflammatory Cytokine Balance in Major Depression: Effect of Sertraline Therapy." Clinical and Developmental Immunology 2007 (2007): 1–6. http://dx.doi.org/10.1155/2007/76396.
Повний текст джерелаMcGeehan, Gerard M., та Joanne Uhl. "TNF-α in Human Diseases". Current Pharmaceutical Design 2, № 6 (грудень 1996): 662–67. http://dx.doi.org/10.2174/1381612802666221004191458.
Повний текст джерелаНlushchenko, T. A. "Investigation of the Cytokine Spectrum of Patients' Oral Fluid with Generalized Periodontitis and Metabolic Syndrome." Ukraïnsʹkij žurnal medicini, bìologìï ta sportu 7, no. 1 (March 22, 2022): 208–12. http://dx.doi.org/10.26693/jmbs07.01.208.
Повний текст джерелаMourouzis, Konstantinos, Evangelos Oikonomou, Gerasimos Siasos, Sotiris Tsalamadris, Georgia Vogiatzi, Alexios Antonopoulos, Petros Fountoulakis, Athina Goliopoulou, Spyridon Papaioannou, and Dimitris Tousoulis. "Pro-inflammatory Cytokines in Acute Coronary Syndromes." Current Pharmaceutical Design 26, no. 36 (October 23, 2020): 4624–47. http://dx.doi.org/10.2174/1381612826666200413082353.
Повний текст джерелаStumpf, Christian, Sebastian Petzi, Katrin Seybold, Gerald Wasmeier, Martin Arnold, Dorette Raaz, Atilla Yilmaz, Werner G. Daniel, and Christoph D. Garlichs. "Atorvastatin enhances interleukin-10 levels and improves cardiac function in rats after acute myocardial infarction." Clinical Science 116, no. 1 (November 28, 2008): 45–52. http://dx.doi.org/10.1042/cs20080042.
Повний текст джерелаДисертації з теми "Pro-inflammatory cytokine TNF -α"
Michlewska, Sylwia. "Macrophage phagocytosis of apoptotic neutrophils is critically regulated by the opposing actions of pro-inflammatory and anti-inflammatory agents : key role for TNF-α". Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/5594.
Повний текст джерелаMejías, Luque Raquel. "Regulatory effects of pro-inflammatory cytokines on genes associated with gastric carcinogenesis." Doctoral thesis, Universitat Pompeu Fabra, 2009. http://hdl.handle.net/10803/7156.
Повний текст джерелаLa gastritis crònica produïda per la infecció per Helicobacter pylori és un dels principals determinants en la patogènesi del càncer gàstric, i comporta nivells elevats de citoquines proinflamatòries com TNF-α, IL-1β o IL-6, que poden regular l'expressió de gens implicats en la transformació neoplàsica gàstrica. Vam analitzar l'efecte de citoquines proinflamatòries en l'expressió de glicosiltransferases i antígens Lewis en cèl·lules de càncer gàstric va ser analitzat i vam observar que el tractament d'aquestes cèl·lules amb IL-1β augmentava l'expressió d'antígens Lewis de tipus 2. A més, vam observar que tumors gàstrics amb inflamació crònica presentaven nivells més elevats d'estructures glicosídiques de tipus 2, suggerint que determinades glicosiltransferases podrien estar regulades per la inflamació.
Els adenocarcinomes gàstrics de tipus intestinal s'originen a partir d'una sèrie successiva de lesions precanceroses que porten a una transdiferenciació intestinal de la mucosa gàstrica. En aquest procés s'activa l'expressió de diferents gens intestinals a les cèl·lules gàstriques, com és el cas dels gens de les mucines intestinals MUC2 i MUC4 i del factor de transcripció intestinal CDX2. Nosaltres vam estudiar l'efecte de citoquines inflamatòries i de les seves vies de senyalització associades en l'expressió d'aquests gens va ser estudiat. El primer que vam constatar va ser que IL-6 regulava l'expressió de MUC4 a través de la via de gp130/STAT3 a línies cel·lulars de càncer gàstric, mentre que l'expressió de MUC2 era induïda per TNF-α a través de la via de senyalització de NF-κB. A més, vam observar que l'expressió de CDX2 no era regulada per citoquines inflamatòries. Finalment, vam trobar que les diferències en l'expressió de les mucines intestinals MUC2 i MUC4 a tumors gàstrics podien ser explicades per diferències en el tipus d'inflamació predominant.
De tots aquests resultats podem concloure que la inflamació i les vies de senyalització associades modulen l'expressió de gens associats a la carcinogènesi gàstrica.
In the gastric carcinogenetic process the specific expression pattern of glycosyltransferases and Lewis antigens displayed by the normal gastric mucosa is lost. We detected that changes in the expression of Lewis antigens induced by the transfection of FUT1 cDNA in HT-29/M3 cells determined a less invasive and metastatic phenotype.
Chronic gastritis caused by H. pylori infection is a major determinant in the pathogenesis of gastric cancer, and present increased levels of the pro-inflammatory cytokines TNF-α, IL-1β and IL-6, which can regulate the expression of genes involved in the gastric neoplastic transformation. We analysed the effect of pro-inflammatory cytokines on the expression of glycosyltransferases and Lewis antigens in gastric cancer cells. IL-1β treatment increased the expression of type 2 Lewis antigens, and, in addition, we observed that gastric tumours with chronic inflammation displayed increased levels of type 2 glycan structures, suggesting that specific glycosyltransferases may be regulated by inflammation.
Intestinal-type gastric adenocarcinomas develop from successive pre-cancerous lesions that lead to an intestinal transdifferentiation of the gastric mucosa. In this process many intestinal genes are activated in gastric cells, such as the intestinal mucin MUC2 and MUC4 genes and the transcription factor CDX2. We studied the effect of pro-inflammatory cytokines and their associated signalling pathways on the expression of these genes. IL-6 regulated the expression of MUC4 through the gp130/STAT3 signalling pathway in gastric cancer cell lines, while MUC2 expression was induced by TNF-α through the NF-κB signalling pathway. No effects on CDX2 expression were observed after cytokine treatment in gastric tumour cells. Finally, we found that the differences observed in the expression of the intestinal mucins MUC2 and MUC4 in gastric tumours could be explained by differences in the predominant type of inflammation present in gastric adenocarcinomas.
In conclusion, our results suggest that inflammation and its associated signalling pathways modulate the expression of genes associated with gastric carcinogenesis.
Grandjean-Laquerriere, Alexia. "Mécanismes de régulation de la production de cytokines pro-inflammatoires (TNF-α,IL-6,IL-)8 et IL-18 et anti-inflammatoire (IL-10) : modèle du kératinocyte humain irradié par des UVB/Alexia Grandjean". Reims, 2001. http://www.theses.fr/2001REIMP202.
Повний текст джерелаBetina-Bencharif, Soumeya. "Isolement et caractérisation de saponosides extraits de deux plantes médicinales : Cyclamen africanum, Zygophyllum cornutum et évalution de leur activité anti-inflammatoire." Thesis, Dijon, 2014. http://www.theses.fr/2014DIJOPE03/document.
Повний текст джерелаThe appearance of several diseases, such as cancer, diabetes, high blood pressure and spread of infections mutagenic virus type can be linked to the quality and lifestyle that we lead today. Indeed, several studies on the factors triggering these so-called "morbid" long-or short-term illnesses are related to stress and quality of food consumed, whether of plant and animal origin. These diseases are becoming a common occurrence, they affect different races and all classes of society. According ethnobotanical research, naturally occurring substances, allowed civilizations to survive deadly diseases. For example, we thus find references to periods of malarial fever in China and one of the symptoms of this disease in the "Huangdi Neijiang" The Canon of Medicine dating from around the first century BC, more than 2000 years, which relates to the use of herbal medicines to relieve fevers (Desgrouas et al., 2014).Around 186 BC appears, in some parts of China, the use in herbal tea, Qing hao su, later known as artemisinin in the West and extracted from a medicinal plant used as antipyretic called "Qing hao" Artemisia annua or annual wormwood. Artemisinin blocks an enzyme which enables the parasite to pump calcium and prevents it from developing. As of today the Artemisinin-based combination therapy in French Combination therapy of artemisinin and ACT acronym, is a therapy and tertiary prevention in cases of uncomplicated malaria.From this perspective our study adds to a long series of studies on medicinal plants and natural substances extracted. It aims to reveal new biomolecules, highlighting their biological activities through techniques of biotechnology on the one hand. Moreover, these investigations will develop natural resources that are characterized by endemic.To do this, our choice is focused on two endemic medicinal plants in Algeria Cyclamen africanum Boiss. & Reuter and Zygophyllum cornutum Coss. After an ethnobotanical research on traditional medicine in Northern Africa, which showed the effectiveness of these plants in minimal inflammatory problems among Aboriginal, we undertook biochemical pharmacological investigations.The latter allowed us to isolate, five compounds from the methanol extract of the roots of the species Cyclamen africanum Boiss. Reuter & two new oleanane triterpene saponins type, Afrocyclamin A and B (1, 2) and three triterpenoid saponins known lysikokianoside of (3), deglucocyclamin I (4) and its derivative dicrotalique acid (5) September and known from the methanol extract saponins from the whole plant of Zygophyllum cornutum Coss. these saponins are ursane type, type triterpenes are reported in this species for the first time and can be considered a chemotherapy marker Taxonomic (chemotype) of Zygophyllum kind. The structures were elucidated on the basis of the analysis of NMR spectra of the experience-1D and 2D-NMR (COSY, TOCSY, NOESY, HSQC and HMBC) and mass spectrometry method negative ion FAB source. The biological activities of saponosidiques FR.1 and Fr.2 fractions were tested on lines of male and female rats of the Winstar rats to evaluate the anti-inflammatory activity. The saponosidique fraction FR.1 Cyclamen africanum the 5 mg dose, showed a significant effect on inflammation caused by carrageenan, reducing edema and immune response, which resulted in the concentration of protein the inflammatory response (PRI) through their action on the pro-inflammatory mediators (COX-2, PGE2, TNF -α, iNOS). The fraction of Fr.2 saponosidique Zygophyllum dose 20 mg did not show a significant effect on inflammation in general
Österberg, Johanna. "Inflammatory Reactions in Peritonitis and Malignant Obstructive Jaundice : Clinical and Experimental Studies with Special Emphasis on the Cellular Immune Response." Doctoral thesis, Uppsala University, Department of Surgical Sciences, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-4767.
Повний текст джерелаPatients with peritonitis or malignant obstructive jaundice (HPB+) have an increased morbidity and mortality due to sepsis. An altered cell-mediated immunity in the intestinal mucosa might promote gut barrier failure, increased endotoxin and cytokine release and bacterial translocation (BT) in these conditions. A clinically relevant rat model of polymicrobial peritonitis induced sepsis by cecal ligation and puncture (CLP) was used. Septic animals demonstrated a superficial injury in the small intestinal mucosa, and a significant reduction in T lymphocytes in the villi, as well as increased number of macrophages in the villi and in the MLNs as compared to sham. CLP caused increased concentration of TNF-α and IL-6 in ascitic fluid. CLP + the immunomodulator Linomide decreased the TNF-α level, reduced mucosal damage and attenuated the changes in T lymphocytes and macrophages observed following CLP. CLP + selective cyclooxygenase (COX)-2 inhibitor (SC-236) or nonselective COX inhibitor (indometacin) decreased the amount of macrophages in the mucosa and the MLNs compared to untreated CLP. CLP + indometacin decreased T lymphocytes in the villi and MLNs. SC-236 + CLP reduced mucosal injury and cytokine release as compared to indometacin. An increased rate of apoptosis in both the mucosa and MLNs was seen following CLP; COX inhibitors enhanced this phenomenon in the MLNs.
BT occurred infrequently in patients with acute peritonitis and in HPB+ there was no evidence of BT. Peritonitis and HPB+ causes significant inflammatory cellular reactions as increased endotoxin and cytokine plasma levels and an altered immune cell distribution in MLNs, in HPB+ a high rate of apoptosis in MLNs was observed.
An altered pattern of immunocompetent cells within the mucosa and in MLNs was found in experimental and clinical peritonitis as in HPB+. Lymphocyte depletion may be a result of increased apoptosis, which could reduce the ability of septic or jaundice patients to eradicate infection.
Bollenbach, Maud. "Nouvelles cibles pour l'étude et le développement d'outils pharmacologiques originaux pour le traitement des douleurs neuropathiques." Thesis, Strasbourg, 2017. http://www.theses.fr/2017STRAF022.
Повний текст джерелаNeuropathic pains correspond to a central sensory nervous system hypersensitivity. It is a chronic and disabling disease, which touch around 6% of the French population. Nowadays, there is no specific and efficient treatment. In my PhD project, we used two different strategies in order to develop innovative pharmacological tools to treat those pains: a phenotypic approach around two TNFα overproduction inhibitor (a 2-aminopyrimidine derivative and a pyridin-2-yl guanidine derivative) and a molecular approach around MY 5445 (a phthalazine PDE5 inhibitor). Our work was based on the design, synthesis and structure-activity relationship study around various hits and we obtained compounds i.p. and orally effective on a murin neuropathic pain model.In parallel to this pharmacological work, we developed different catalytic systems (Pd, Cu) under micellar conditions to form C-N bonds at almost room temperature
Fotheringham, Julie A. "Regulation of the pro-inflammatory cytokine TNF-[alpha] by adenosine receptors in monocytes and macrophages." 2003. http://hdl.handle.net/1993/19851.
Повний текст джерелаКниги з теми "Pro-inflammatory cytokine TNF -α"
K. Gautam, Rupesh, Lokesh Deb, and Kamal Dua, eds. Natural Products for the Management of Arthritic Disorders. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/97898150507761220101.
Повний текст джерелаЧастини книг з теми "Pro-inflammatory cytokine TNF -α"
Vogler, D., G. Schmittat, and S. Ohrndorf. "Rheumatism and wIRA Therapy." In Water-filtered Infrared A (wIRA) Irradiation, 225–32. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-92880-3_19.
Повний текст джерелаBashir, Haamid, Mohammad Hayat Bhat, and Sabhiya Majid. "Molecular Pathogenesis of Inflammatory Cytokines in Insulin Resistance Diabetes Mellitus." In Insights on Antimicrobial Peptides [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.100971.
Повний текст джерелаFrançozo de Melo, Adriana, Giulia Oliveira Timo, and Mauricio Homem-de-Mello. "Vitamin C and Sepsis." In Antioxidants - Benefits, Sources, Mechanisms of Action. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.95623.
Повний текст джерелаZolty, Ronald. "The Role of Neurohormonal Systems, Inflammatory Mediators and Oxydative Stress in Cardiomyopathy." In Cardiomyopathy - Disease of the Heart Muscle. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.97345.
Повний текст джерелаKumar, Vikas, Md Sakhawat Hossain, Janice A. Ragaza, and Marina Rubio Benito. "The Potential Impacts of Soy Protein on Fish Gut Health." In Soybean for Human Consumption and Animal Feed. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.92695.
Повний текст джерелаArora, Disha, Sanjay Sharma, and Sumeet Gupta. "Natural Products Targeting Various Mediators in Rheumatoid Arthritis." In Natural Products for the Management of Arthritic Disorders, 135–63. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/9789815050776122010009.
Повний текст джерелаSahu, Bijayani, Amy R. Mackos, Angela M. Floden, Loren E. Wold, and Colin K. Combs. "Particulate Matter Exposure Exacerbates Amyloid-β Plaque Deposition and Gliosis in APP/PS1 Mice." In Advances in Alzheimer’s Disease. IOS Press, 2021. http://dx.doi.org/10.3233/aiad210013.
Повний текст джерелаHu, Li, Shaoping Zhu, Xiaoping Peng, Kanglan Li, Wanjuan Peng, Yu Zhong, Chenyao Kang, Xingxing Cao, Zhou Liu, and Bin Zhao. "High Salt Elicits Brain Inflammation and Cognitive Dysfunction, Accompanied by Alternations in the Gut Microbiota and Decreased SCFA Production." In Advances in Alzheimer’s Disease. IOS Press, 2022. http://dx.doi.org/10.3233/aiad220029.
Повний текст джерелаMalhotra, Hitesh, Anjoo Kamboj, and Peeyush Kaushik. "Phytochemicals: A New Approach for Targeting Biomarkers in the Management of Arthritis." In Natural Products for the Management of Arthritic Disorders, 218–42. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/9789815050776122010013.
Повний текст джерелаAli, Anwar, Quratul Ain, Ayesha Saeed, Waseem Khalid, Munir Ahmed, and Ahmed Bostani. "Bio-Molecular Characteristics of Whey Proteins with Relation to Inflammation." In Whey Proteins - Uses and Biological Roles [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99220.
Повний текст джерелаТези доповідей конференцій з теми "Pro-inflammatory cytokine TNF -α"
Danilova, Galina, та Anna Klinnikova. "INFLUENCE OF GLUCOCORTICOID HORMONES ON THE INFLAMMATORY EFFECTS OF THE PRO-INFLAMMATORY CYTOKINE TNF-α". У XVIII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2022. http://dx.doi.org/10.29003/m2739.sudak.ns2022-18/120.
Повний текст джерелаWolfe, Valerie M., Seonghun Park, Marjana Tomic, Peter A. Torzilli, and C. T. Christopher Chen. "Load Down-Regulates TNF-Alpha Induced Cartilage Degradation in Part Through NF-KB and P38 Pathways." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176541.
Повний текст джерелаRajan, Neena, Nathaniel Stetson, Passquale Razzano, Mitchell Levine, Daniel Grande, and Nadeen Chahine. "Response of Mesenchymal Stem Cells and Intervertebral Disc Cells to Inflammatory Challenge." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53937.
Повний текст джерелаMaidhof, Robert, Neena Rajan, and Nadeen O. Chahine. "Effect of Inflammation on the Osmotic Response of Nucleus Pulposus Cells." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80358.
Повний текст джерелаRajan, Neena, Nate Stetson, Robert Maidhof, Mitchell Levine, and Nadeen Chahine. "In Vivo Administration of an Inflammatory Stimulant can Trigger Loss of Biomechanical and Biochemical Properties of the Intervertebral Disc." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80495.
Повний текст джерелаDanilova, G. А., A. A. Klinnikova та N. P. Aleksandrova. "The role of prostaglandins in the realization of respiratory effects of TNF-α in case of hypoxia". У VIII Vserossijskaja konferencija s mezhdunarodnym uchastiem «Mediko-fiziologicheskie problemy jekologii cheloveka». Publishing center of Ulyanovsk State University, 2021. http://dx.doi.org/10.34014/mpphe.2021-86-88.
Повний текст джерелаKashyap, Meghana, Kristen T. Carter, Brent C. Sauer та Christopher T. Chen. "NF-κB Mediates Cartilage Degradation Induced by Trauma Injury and Interleukin-1". У ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14513.
Повний текст джерелаAl-Haidose, Amal. "Effect of Omega-3 Polyunsaturated Fatty Acids on Inflammatory Biomarkers in Chronic Obstructive Pulmonary Disease." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0144.
Повний текст джерелаNa Takuathung, M., A. Wongnoppavich, A. Panthong, P. Khonsung, N. Chiranthanut, N. Soonthornchareonnon та S. Sireeratawong. "Anti-psoriatic Effects of Wannachawee Recipe on TNF-α- and IFN-γ-Induced Inflammatory Cytokine production in HaCaT Human Keratinocytes". У GA 2017 – Book of Abstracts. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1608165.
Повний текст джерела"Pro-inflammatory cytokines “Tumor necrosis factor alpha (TNF-α) and Interleukin beta (IL-1 β)” levels in albino rats after co-administration of diclofenac sodium and dexamethasone". У 2nd Hawler Pharmaceutical Sciences Conference. Hawler Medical University, 2020. http://dx.doi.org/10.15218/hpsc.02.13.
Повний текст джерелаЗвіти організацій з теми "Pro-inflammatory cytokine TNF -α"
Cao, Xianling, Xuanyou Zhou, Naixin Xu, Songchang Chang, and Chenming Xu. Association of IL-4 and IL-10 Polymorphisms with Preterm Birth Susceptibility: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0044.
Повний текст джерелаSchwartz, Bertha, Vaclav Vetvicka, Ofer Danai, and Yitzhak Hadar. Increasing the value of mushrooms as functional foods: induction of alpha and beta glucan content via novel cultivation methods. United States Department of Agriculture, January 2015. http://dx.doi.org/10.32747/2015.7600033.bard.
Повний текст джерела