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Готові списки джерел за темами / Prevention liver cancer / Книги

Книги з теми "Prevention liver cancer"

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Автор: Grafiati

Опубліковано: 30 травня 2022

Оновлено: 31 травня 2022

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Ознайомтеся з топ-25 книг для дослідження на тему "Prevention liver cancer".

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1

Colvin, Heather M. Hepatitis and liver cancer: A national strategy for prevention and control of hepatitis B and C. Edited by Institute of Medicine (U.S.). Committee on the Prevention and Control of Viral Hepatitis Infections, Institute of Medicine (U.S.). Board on Population Health and Public Health Practice, and National Academies Press (U.S.). Washington, DC: National Academies Press, 2010.

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2

S, Luxenberg Jay, and Better Health Foundation, eds. You can't live forever, you can live 10 years longer with better health: A practical guide to reducing your risk of dying early from heart disease, cancer, stroke, osteoporosis & accidents. San Francisco, Calif: Better Health Foundation & UCSF/Mount Zion, 1993.

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3

McKinnell, Robert Gilmore. The understanding, prevention and control of human cancer: The historic work and lives of Elizabeth Cavert Miller and James A. Miller. Leiden: Brill, 2016.

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4

Okita, K., and M. Omata. Therapy for Viral Hepatitis and Prevention of Hepatocellular Carcinoma. Springer, 2012.

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5

Bamia, Christina, Sherri Stuver, and Lorelei Mucci. Cancer of the Liver and Biliary Tract. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0012.

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Primary liver cancer is one of the most commonly occurring cancers globally, and is the second most common cause of cancer death worldwide. There are two major histologic forms of primary liver cancer: hepatocellular carcinoma (HCC) and cholangiocarcinoma. It is a rapidly and almost uniformly fatal disease, yet there is already sufficient knowledge about its major risk factors, many of which are modifiable, to make primary prevention effective. Primary liver cancer is one of the first common human cancer that was found to have an infectious etiology, with hepatitis B virus (HBV) and hepatitis C virus (HCV) for HCC, and parasitic liver flukes for cholangiocarcinoma. Obesity is emerging as an important risk factor, particularly in Western countries, where primary liver cancer rates appear to be increasing over time. A number of additional risk factors and potential preventive factors are considered in this chapter.

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6

1925-, Blumberg Baruch S., ed. Hepatitis B and the prevention of primary cancer of the liver: Selected publications of Baruch S. Blumberg. Singapore: World Scientific, 2000.

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7

Blumberg, Baruch S. Hepatitis B and the Prevention of Primary Cancer of the Liver. WORLD SCIENTIFIC, 2000. http://dx.doi.org/10.1142/3550.

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8

S, Cappell Mitchell, ed. Screening, prevention, and treatment of esophageal, gastric, and hepatic malignancies. Philadelphia: Saunders, 2005.

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9

Keshav, Satish, and Alexandra Kent. Prevention of gastrointestinal disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0346.

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Disease prevention is usually directed where there is considerable morbidity or mortality, and etiological factors that can be controlled, treated, or reduced. The greatest morbidity and mortality from gastrointestinal disease is related to infectious diarrhoea and gastrointestinal cancer, both of which can be prevented. Smoking has been closely associated with oesophageal, gastric, and liver cancer and also has a significant effect in inflammatory bowel disease. In addition, alcohol consumption and viral hepatitis are preventable causes of liver disease, liver failure, and hepatic cancer. This chapter addresses the prevention of gastrointestinal disease, focusing on alcohol, smoking, peptic ulcer disease, colorectal cancer, oesophageal adenocarcinoma, hepatitis B, hepatitis C, non-alcoholic fatty liver disease, and gastrointestinal infection.

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10

M, Colvin Heather, Mitchell Abigail E, Institute of Medicine (U.S.). Committee on the Prevention and Control of Viral Hepatitis Infections., Institute of Medicine (U.S.). Board on Population Health and Public Health Practice., and National Academies Press (U.S.), eds. Hepatitis and liver cancer: A national strategy for prevention and control of hepatitis B and C. Washington, DC: National Academies Press, 2010.

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11

Colon Cancer Screening, Surveillance, Prevention and Treatment: Conventional and Novel Technologies, Part II, An Issue of Medical Clinics. Saunders, 2005.

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12

Muraki, Terumi. Yomei san-kagetsu no gan ga kieta ichi nichi isshoku kiseki no reshipi: Makki no kanzōgan o kokufuku shita kome · miso · shōyu · gohan. 2014.

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13

Therapy for viral hepatitis and prevention of hepatocellular carcinoma. Tokyo: Springer, 2004.

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14

(Editor), M. Omata, and K. Okita (Editor), eds. Therapy for Viral Hepatitis and Prevention of Hepatocellular Carcinoma. Springer, 2004.

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15

Bulterys, Marc, Julia Brotherton, and Ding-Shinn Chen. Prevention of Infection-Related Cancers. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0066.

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Анотація:

This chapter discusses primary prevention measures that disrupt transmission of oncogenic infections. It begins by discussing vaccination against hepatitis B virus (HBV) and human papillomavirus (HPV), two major causes of cancer for which safe and effective vaccines are currently available. It briefly discusses the importance of treatment and prophylaxis against human immunodeficiency virus type 1 (HIV-1), which potentiates the virulence of other viral infections as well as directly increasing the incidence of non-Hodgkin lymphoma. It does not discuss the treatment of HBV or hepatitis C virus (HCV) infection, since these are considered in Chapters 25 and 33. Also beyond the scope of this chapter are the randomized clinical trials currently underway to assess the efficacy and feasibility of eradication of Helicobacter pylori (Chapters 24, 31), vaccination against Epstein-Barr virus (EBV) (Chapters 24, 26, 39), or the prevention of schistosomiasis and liver flukes (Chapters 24, 33, and 52).

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16

Blumberg, Baruch S. Hepatitis B and the Prevention of Primary Cancer of the Liver: Selected Publications of Baruch S. Blumberg (World Scientific Series in 20th Century Biology). World Scientific Pub Co Inc, 2000.

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17

Grant, Warren, and Martin Scott-Brown. Prevention of cancer. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0350.

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In the UK, the four commonest cancers—lung cancer, breast cancer, colon cancer, and prostate cancer—result in around 62 000 deaths every year. Although deaths from cancer have fallen in the UK over the last 20 years, the UK still suffers from higher cancer death rates than many other countries in Western Europe. In 1999, the UK government produced a White Paper called Saving Lives: Our Healthier Nation that outlined a national target to reduce the death rate from cancer by at least 20% in people under 75 by 2010. The subsequent NHS Cancer Plan of 2000 designed a framework by which to achieve this target through effective prevention, screening, and treatment programmes as well as restructuring and developing new diagnostic and treatment facilities. But do we know enough about the biology of the development of cancer for government health policies alone to force dramatic changes in survival? The science behind the causes of cancer tells us that its origin lies in acquired or inherited genetic abnormalities. Inherited gene mutation syndromes and exposure to environmental mutagens cause cancer, largely through abnormalities in DNA repair mechanisms, leading to uncontrolled cell proliferation. Although screening those thought to be at highest risk, and regulating exposure to environmental carcinogens such as tobacco or ionizing radiation, have reduced, and will continue to reduce, cancer deaths, there are many other environmental factors that have been shown to increase the population risk of cancer. These will be outlined in this chapter. However, the available evidence is largely from retrospective and cross-sectional population-based studies and therefore limits the ability to apply this knowledge to the risk of the individual patient who may been seen in clinic. Although we may be able to put him or her into a high-, intermediate-, or low-risk category, the question ‘will I get cancer, doc?’ is one that we cannot answer with certainty. The NHS Cancer Plan of 2000, designed to reduce cancer deaths in this country and to bring UK treatment results in line with those other countries in Europe, focuses on preventing malignancy as part of its comprehensive cancer management strategy. It highlights that the rich are less likely to develop cancer, and will survive longer if they are diagnosed than those who live in poverty. This may reflect available treatment options, but is more likely to be related to the lifestyle of those with regular work, as they may be more health aware. The Cancer Plan, however, suggests that relieving poverty may be more labour intensive and less rewarding than encouraging positive risk-reducing behaviour in all members of the population. Eating well can reduce the risk of developing many cancers, particularly of the stomach and bowel. The Cancer Plan outlines the ‘Five-a-Day’ programme which was rolled out in 2002 and encouraged people to eat at least five portions of fruit and vegetables per day. Obese people are also at higher risk of cancers, in particular endometrial cancer. A good diet and regular exercise not only reduce obesity but are also independent risk-reducing factors. Alcohol misuse is thought to be a major risk factor in around 3% of all cancers, with the highest risk for cancers of the mouth and throat. As part of the Cancer Plan, the Department of Health promotes physical activity and general health programmes, as well as alcohol and smoking programmes, particularly in deprived areas. Focusing on these healthy lifestyle points can potentially reduce an individual lifetime risk of all cancers. However, our knowledge of the biology of four cancers in particular has led to the development of specific life-saving interventions. Outlined in this chapter are details regarding ongoing prevention strategies for carcinomas of the lung, the breast, the bowel, and the cervix.

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18

Oliver, Nora, and Elizabeth Chiao. Malignant Diseases in HIV. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0033.

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Malignancies were one of the earliest recognized manifestations that led to the eventual description of the AIDS epidemic. Kaposi’s sarcoma was one of the first entities described in association with AIDS. Subsequently, intermediate-grade and high-grade non-Hodgkin’s lymphoma, invasive cervical cancer, and primary central nervous system lymphoma were defined by the Centers for Disease Control and Prevention as “AIDS-defining conditions.” Since the advent of combination antiretroviral therapy, several other cancers that are not AIDS-defining have been found to have an increased incidence in patients with HIV. These include, but are not limited to, Hodgkin’s disease and anal, liver, lung, oropharyngeal, colorectal, and renal cancers. They are generally referred to as “non-AIDS-defining cancers.” The increasing longevity of persons living with HIV as well as concurrent modifiable risk factors such as tobacco use may also influence the epidemiology of these malignancies.

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19

(Editor), Janet E. Joy, Edward E. Penhoet (Editor), and Diana B. Petitti (Editor), eds. Saving Women's Lives: Strategies for Improving Breast Cancer Detection and Diagnosis. National Academies Press, 2005.

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20

1953-, Joy Janet E., Penhoet Edward E, Petitti Diana B, National Cancer Policy Board (U.S.). Committee on New Approaches to Early Detection and Diagnosis of Breast Cancer., National Research Council (U.S.). Policy and Global Affairs., and National Research Council (U.S.). Board on Science, Technology, and Economic Policy., eds. Saving women's lives: Strategies for improving breast cancer detection and diagnosis. Washington, DC: National Academies Press, 2005.

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21

Roger, Herdman, Norton Larry, and National Cancer Policy Board (U.S.). Committee on New Approaches to Early Detection and Diagnosis of Breast Cancer., eds. Saving women's lives: Strategies for improving breast cancer detection and diagnosis. Washington, DC: National Academies Press, 2005.

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22

Committee on New Approaches to Early Detection and Diagnosis of Breast Cancer. Saving Women's Lives: Strategies for Improving Breast Cancer Detection and Diagnosis -- A Breast Cancer Research Foundation and Institute of Medicine Symposium. National Academies Press, 2004.

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23

Magee, Elaine. Tell Me What to Eat to Help Prevent Breast Cancer: Nutrition You Can Live With (Tell Me What to Eat). Career Press, 2000.

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24

Ellinson, Michelle, and Tommy Rampling. Normal nutritional function. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0331.

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Normal nutritional function requires a healthy diet. Healthy eating incorporates a variety of nutrients that are essential for energy expenditure, prevention of disease, and maintenance of normal physiological function. An unhealthy diet can result in malnutrition, and this contributes to illness and death throughout the world. The core principle of healthy eating is obtaining an adequate balance, and the diseases resulting from overnourishment differ greatly from those resulting from undernourishment. In the third world, diets tend to rely heavily on staple crops, and can be very seasonal. Energy sources are predominantly cereals, whereas meat and fish are limited. Malnutrition tends to occur from a lack of essential nutrients, leading to conditions such as vitamin deficiencies, kwashiorkor, and iodine deficiency syndromes. In first-world countries, people have more freedom to choose what they eat. Thus, diets tend to be high in fat and dense in energy. Obesity, diabetes, coronary heart disease, cancer, and hypertension are major contributors to morbidity and mortality. A healthy diet should contain adequate proportions of carbohydrates, fats, proteins, vitamins, and trace elements. The intake of these constituents is sporadic, with meals constituting major boluses of potential energy. Energy expenditure, conversely, is continuous. The human body has, therefore, developed complex mechanisms directing nutrients into storage when in excess, and mobilizing these stores as they are needed, and it is essential that sufficient energy is always available to maintain the basal metabolic rate, which is the amount of energy expended while at rest in a neutrally temperate environment. This energy is sufficient only for the functioning of the vital organs, such as the heart, the lungs, the liver, the kidneys, and the CNS.

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25

Attain a Happy & Peaceful Life by Nikhil Anshuman: Live a life filled with happiness and inner peace. Nikhil Anshuman, 2019.

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