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Статті в журналах з теми "Prenatal transmission"
Guibert, Floriane, Sophie Lumineau, Kurt Kotrschal, Erich Möstl, Marie-Annick Richard-Yris, and Cécilia Houdelier. "Trans-generational effects of prenatal stress in quail." Proceedings of the Royal Society B: Biological Sciences 280, no. 1753 (February 22, 2013): 20122368. http://dx.doi.org/10.1098/rspb.2012.2368.
Повний текст джерелаRodrigues, Celeste Souza, Mark Drew Crosland Guimarães, and Cibele Comini César. "Missed opportunities for congenital syphilis and HIV perinatal transmission prevention." Revista de Saúde Pública 42, no. 5 (October 2008): 851–58. http://dx.doi.org/10.1590/s0034-89102008000500010.
Повний текст джерелаPeters, Vicki, Kai-Lih Liu, Kenneth Dominguez, Toni Frederick, Sharon Melville, Ho-Wen Hsu, Idith Ortiz, Tamara Rakusan, Balwant Gill, and Pauline Thomas. "Missed Opportunities for Perinatal HIV Prevention Among HIV-Exposed Infants Born 1996–2000, Pediatric Spectrum of HIV Disease Cohort." Pediatrics 111, Supplement_1 (May 1, 2003): 1186–91. http://dx.doi.org/10.1542/peds.111.s1.1186.
Повний текст джерелаBoussemart, T., P. Babe, G. Sibille, C. Neyret, and C. Berchel. "Prenatal Transmission of Dengue: Two New Cases." Journal of Perinatology 21, no. 4 (June 2001): 255–57. http://dx.doi.org/10.1038/sj.jp.7200530.
Повний текст джерелаAndany, Nisha, Michelle Letchumanan, Lise Bondy, Kellie Murphy, and Mona R. Loutfy. "Amniocentesis in the HIV-Infected Pregnant Woman: Is There Still Cause for Concern in the Era of Combination Antiretroviral Therapy?" Canadian Journal of Infectious Diseases and Medical Microbiology 24, no. 3 (2013): e91-e95. http://dx.doi.org/10.1155/2013/185192.
Повний текст джерелаReitter, A., A. U. Stücker, H. Buxmann, E. Herrmann, A. E. Haberl, R. Schlößer, and F. Louwen. "Prenatal Ultrasound Screening for Fetal Anomalies and Outcomes in High-Risk Pregnancies due to Maternal HIV Infection: A Retrospective Study." Infectious Diseases in Obstetrics and Gynecology 2013 (2013): 1–10. http://dx.doi.org/10.1155/2013/208482.
Повний текст джерелаManjarrez, Gabriel, Ignacia Cisneros, Rocio Herrera, Felipe Vazquez, Alejandro Robles, and Jorge Hernandez. "Prenatal Impairment of Brain Serotonergic Transmission in Infants." Journal of Pediatrics 147, no. 5 (November 2005): 592–96. http://dx.doi.org/10.1016/j.jpeds.2005.06.025.
Повний текст джерелаAmutah-Onukagha, Ndidiamaka, Tonia J. Rhone, Mandy J. Hill, Alecia McGregor, and Rebecca Cohen. "Prevalence of Prenatal HIV Screening in Massachusetts: Examining Patterns in Prenatal HIV Screening Using the Massachusetts Pregnancy Risk Assessment Monitoring System (PRAMS), 2007-2016." Journal of the International Association of Providers of AIDS Care (JIAPAC) 21 (January 2022): 232595822110697. http://dx.doi.org/10.1177/23259582211069767.
Повний текст джерелаEuler, Gary L., Karen G. Wooten, Andrew L. Baughman, and Walter W. Williams. "Hepatitis B Surface Antigen Prevalence Among Pregnant Women in Urban Areas: Implications for Testing, Reporting, and Preventing Perinatal Transmission." Pediatrics 111, Supplement_1 (May 1, 2003): 1192–97. http://dx.doi.org/10.1542/peds.111.s1.1192.
Повний текст джерелаSetyoboedi, Bagus, Avianita Kusumawardhani, Agung Widodo, and Muhammad Akbar. "Tenofovir Disoproxil Fumarate Prenatal as A Complementary Treatment to Prevent Vertical Transmission of Hepatitis B Virus: A Systematic Review." International Journal of Medical Reviews and Case Reports 5, Reports in Dental Medicine and (2021): 1. http://dx.doi.org/10.5455/ijmrcr.tenofovir-disoproxil-fumarate-prenatal.
Повний текст джерелаДисертації з теми "Prenatal transmission"
LATTARD, CHANTAL. "Transmission maternofoetale de la toxoplasmose : estimation du risque sur une cohorte de 248 femmes suivies en 1988 et 1989 en vue de preciser les indications du diagnostic ante-natal." Lyon 1, 1993. http://www.theses.fr/1993LYO1M185.
Повний текст джерелаRichter, André. "Essays on the Intergenerational Transmission of Disadvantage : The Role of Prenatal Health and Fertility." Doctoral thesis, Stockholms universitet, Nationalekonomiska institutionen, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-135563.
Повний текст джерелаAt the time of the doctoral defense, the following papers were unpublished and had a status as follows: Paper 1: Manuscript. Paper 2: Manuscript. Paper 3: Manuscript.
Steiner, Kevin Lee. "Prenatal priming to malaria antigens increases susceptibility to HIV infection." Case Western Reserve University School of Graduate Studies / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=case1321827400.
Повний текст джерелаPONCELIN, DE RAUCOURT YVES. "Transmission materno-foetale de la toxoplasmose : estimation du risque sur une cohorte de 550 femmes suivies entre 1988 et 1992 en vue de preciser les indications du diagnostic ante-natal." Lyon 1, 1994. http://www.theses.fr/1994LYO1M182.
Повний текст джерелаLowe, Germaine. "Prenatal maternal infection and synaptic transmission in the hippocampus: from single cells to neural networks." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=104545.
Повний текст джерелаL'hippocampe est une des structures clés du cerveau dans les processus de mémorisation. Les altérations du fonctionnement de cette structure chez les patients schizophrènes pourraient expliquer en partie les troubles cognitifs associés à la maladie. Même si les causes exactes de la schizophrénie font encore l'objet de nombreux débats, plusieurs études ont pu mettre en évidence une augmentation de l'incidence de cette maladie chez les enfants des mères exposées à une infection bactérienne ou virale durant la grossesse. Afin de mieux comprendre les changements intervenant au sein des réseaux hippocampiques lors de la schizophrénie, nous avons utilisé un modèle d'infection prénatale chez le rongeur. Ce modèle animal, largement validé, a permis de caractériser les changements anatomiques survenant dans l'hippocampe. Cependant, soulignons qu'aucune étude n'a pour le moment étudié les altérations possibles de l'activité électrophysiologique, tant au niveau de la synapse que du réseau, occasionnées dans l'hippocampe par une infection prénatale. La caractérisation de ces altérations nous permettra par conséquent de mieux saisir d'une part les changements d'activité observés dans l'hippocampe suite à une infection prénatale et de cerner d'autre part leurs rôles possibles dans les troubles comportementaux communément décrits dans la schizophrénie.La première partie de notre étude porte ainsi sur les altérations de la transmission synaptique dans l'hippocampe de jeunes rats provenant de mères exposées à une infection bactérienne (en utilisant le lipopolysacharide ou LPS comme modèle d'infection) vers la fin de la grossesse, soit le quinzième et seizième jours de gestation chez le rat. Dans ce modèle d'infection prénatale, nous avons observé une diminution de la transmission excitatrice vers l'aire CA1 de l'hippocampe (altérations présynaptique) accompagnée d'une augmentation compensatoire au niveau postsynaptique. Ces changements semblent spécifiques à l'aire CA1 puisqu'ils ne sont pas présents dans l'aire CA3 ou le gyrus dentelé. Finalement, nous avons également mis en évidence une augmentation de l'excitabilité des cellules pyramidales de l'aire CA1.Ces résultats couplés aux nombreuses études mettant en évidence une perturbation de la transmission excitatrice et inhibitrice dans l'hippocampe suite à une infection prénatale, suggèrent que l'activité de réseau de l'hippocampe pourrait être altérée. Afin de tester cette hypothèse, nous avons étudié l'effet d'une infection prénatale au poly I:C sur l'activité oscillatoire de l'hippocampe et ce en utilisant une nouvelle approche expérimentale récemment développée au laboratoire, l'hippocampe intact isolé in vitro. Bien qu'aucune altération des rythmes thêta et gamma nà été observée, nous avons mis en évidence une plus grande sensibilité du rythme thêta aux récepteurs NMDA (N-methyl D-aspartate) exclusivement dans le subiculum. Dans cette region de l'hippocampe nous avons de plus observé une augmentation du couplage des oscillations gamma et theta. En conclusion, l'infection prénatale induit des changements neurodéveloppementaux qui affectent profondément la transmission synaptique et l'activité de réseau de façon précoce dans l'hippocampe. Ces changements, spécifiques au subiculum et à l'aire CA1, pourraient fortement compromettre le transfert d'information entre l'hippocampe et différentes structures corticales et sous-corticales et pourraient de ce fait contribuer aux déficits cognitifs observés dans la schizophrénie.
Bianda, Nkembi Lydie. "The Role of Prenatal Care and Systematic HIV Testing in Preventing Perinatal Transmission in Tanzania, 2011-2012." ScholarWorks, 2017. https://scholarworks.waldenu.edu/dissertations/3486.
Повний текст джерелаLeonard, Lynne. "Testing women as mothers : the policy and practice of prenatal HIV testing." Thesis, McGill University, 2003. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=84280.
Повний текст джерелаWorking with pregnant women in Ontario, the province with the highest level of HIV infection among Canadian women, this thesis articulates and interprets their experiences of prenatal HIV counselling and testing and details their perspectives on best practices. The pregnant women's evidence-based recommendations for the re-design of prenatal HIV testing programmes are provided. These unique data have important utility for federal and provincial policy makers as HIV counselling and testing policies and programmes that encompass and are grounded in pregnant womens' experiences and perspectives are likely to be maximally acceptable and thereby increase the number of pregnant women who can be apprised of prophylactic treatment to take care of their own health needs as well as those of their unborn children.
In order for pregnant women to increase control over their own health and that of their unborn children, there is clear value in all pregnant women being afforded the opportunity to know their HIV status. However, the voices of the women in this study suggest that the autonomy rights of pregnant women may well be at risk in a programme in which the current emphasis is on potential HIV infection of the foetus rather than on potential or actual infection of the pregnant woman.
Lindberg, Ann. "Epidemiology and eradication of bovine viral diarrhoea virus infections : studies on transmission and prenatal diagnosis of persistent infection /." Uppsala : Dept. of Ruminant Medicine and Veterinary Epidemiology, Swedish Univ. of Agricultural Sciences ([Institutionen för idisslarmedicin och epidemiologi], Sveriges lantbruksuniv.), 2002. http://epsilon.slu.se/v132.pdf.
Повний текст джерелаLanthony, Mathilde. "Impact de traits de personnalité des truies gestantes sur le comportement des porcelets : exemple de la réaction à l’humain et de l’agressivité en contexte de stress social. Étude des interactions avec le renforcement positif de la relation humain-animal des porcelets." Electronic Thesis or Diss., Rennes, Agrocampus Ouest, 2022. http://www.theses.fr/2022NSARB359.
Повний текст джерелаMixing with conspecifics and interactions with humans are two of the major stress sources for pregnant sows. They may respond to it in different ways. This variability of response, if it is time stable, draws the concept of animal personality. Stress response and personality could have an impact on the offspring behaviour, as well as early positive experiences with human. This study thus first focused on the investigation of the individual response variability and stability to a social stressor, and its impact on the stress response. Social status can modulate the stressor impact ; we thus previously investigated the characteristics of the hierarchy inside our groups. Since the aggressiveness seemed to be a valuable indicator of the social stress response variability, we investigated its impact on piglets’ behaviour. Finally, we studied the effects of sows’ reaction to humans and their interactions with early positive human handling on piglets’ behaviour.We demonstrated that the hierarchy inside groups of sows is linear, steep and stable, which limited the impact of the social stress. The most aggressive sows were also the most dominant and least injured, but no effects of sows aggressiveness were described on their stress levels, neither on their piglets’ behaviour. Piglets born from fearful sows showed behavioural responses in various tests that could be directly or indirectly attributed to the fear of human. Positive experiences with humans improved human-piglets relation but didn’t erase the maternal effects. Piglets born from fearful sows had lower performances in a spatial memory task that could be attributed to a general anxious state. This study raises the importance of the breeding of sows in stable groups to lower the impact of a possible social stress thanks to the hierarchy. It also points out the importance of a good human-animal relation in sow breedings, not only for their own welfare but also for their piglets’
Mimouni, Nour El Houda. "Elevated prenatal anti-Müllerian hormone reprograms the fetus and induces polycystic ovary syndrome (PCOS) in adulthood." Thesis, Lille, 2019. http://www.theses.fr/2019LILUS051.
Повний текст джерелаPolycystic ovary syndrome (PCOS) is the main cause of female infertility worldwide with high comorbidity and economic burden. It is mainly characterized by hyperandrogenism, oligo/anovulation and polycystic appearing ovaries. Moreover, most women with PCOS exhibit higher levels of circulating luteinizing hormone (LH), suggestive of heightened gonadotropin-releasing hormone (GnRH) release. Additionally, PCOS patients also exhibit 2-3x higher levels of Anti-Müllerian Hormone (AMH) as compared to healthy controls.While the exact origin of PCOS is unknown, familiar clustering and twin studies of PCOS patients and their relatives suggest a strong heritable component in PCOS. However, the candidate genes identified account for only <10% of the estimated 70% heritability of PCOS, implying that it may originate during intrauterine development and that environmental factors, such as hormonal imbalances during fetal life, could be involved in the onset of PCOS.In this study, we first measured AMH levels in a cohort of pregnant women with PCOS and control women which revealed that AMH is significantly more elevated in the former group versus the latter, we then modelized our clinical findings by exposing pregnant mice to high concentration of AMH during a specific temporal window and showed that this fetal exposure leads to a cascade of alterations impacting the maternal brain, the ovaries, and the placenta, which consequently reprogram the fetal brain and induce the acquisition of the major PCOS cardinal neuroendocrine reproductive features, namely hyperandrogenism, elevation in LH pulse frequency and oligo-anovulation, and a persistent rise in the GnRH neuronal firing activity in adulthood. Moreover, our results show that the long-term consequences of a short exposure to elevated AMH levels during gestation expand beyond the first generation exposed and that PCOS-like manifestations seem to be transmitted across subsequent generations of females.Intrestingly, using a pharmacological approach, we demonstrate that tempering GnRH signaling pathway rescues the neuroendocrine phenotype of PCOS-like animals, restoring their normal hormonal levels, estrus cyclicity and ovarian morphology.Lastly, we sought to understand how early exposure to AMH excess would affect the neuroendocrine and reproductive features of the male offspring. Here, we demonstrate that prenatal AMH treatment profoundly impacts the Hypothalamic-Pituitary-Gonadal (HPG) axis function in males, which fail to engage the testosterone surge at birth observed in control newborns, leading to a feminization of sexually dimorphic circuitries of their brains, an increase in LH, a drastic decrease in testosterone levels, severe alterations in the testicular steroidogenesis and morphology as well as a higher risk of developing cryptorchidism in adulthood. Thus, it could be of clinical interest to relate findings from this study to the reproductive phenotype of sons of PCOS women, who are exposed during gestation but not systematically investigated in adulthood.Collectively, our results challenge the concept of PCOS originating in utero and appear to consolidate the role of AMH as a trigger of the pathogenesis, suggesting that an altered hormonal milieu during early life associated with PCOS may not only affect the female fetus but also the male fetus exposed and that these alterations could be transmitted across multiple generations.These findings point to PAMH mouse model as an excellent preclinical tool to investigate both neuroendocrine disturbances of PCOS and how developmental programming effects are transmitted, while offering a therapeutic avenue for the treatment of the disease
Книги з теми "Prenatal transmission"
Renaud, Michelle. Profiles of activities to reduce perinatal transmission of HIV: Assessing the response. Washington, D.C: U.S. Conference of Mayors, 1997.
Знайти повний текст джерелаElizabeth, Kresse, and United States Conference of Mayors., eds. Profiles of activities to reduce perinatal transmission of HIV: Assessing the response. Washington, D.C: United States Conference of Mayors, 1997.
Знайти повний текст джерелаA, Stoto Michael, Almario Donna A, McCormick Marie C, Institute of Medicine (U.S.). Committee on Perinatal Transmission of HIV., and National Research Council (U.S.). Board on Children, Youth, and Families., eds. Reducing the odds: Preventing perinatal transmission of HIV in the United States. Washington, D.C: National Academy Press, 1999.
Знайти повний текст джерела(Editor), Michael A. Stoto, and Donna A. Almario (Editor), eds. Reducing the Odds: Preventing Perinatal Transmission of HIV in the United States. National Academy Press, 1999.
Знайти повний текст джерелаBurdmann, Emmanuel A. Syphilis. Edited by Vivekanand Jha. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0192.
Повний текст джерелаShort, William R., and Jason J. Schafer. Antiretroviral Therapy in Pregnant Women. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0026.
Повний текст джерелаPerinatal Hivaids. W.B. Saunders Company, 2010.
Знайти повний текст джерелаMathiesen, Amber, and Kali Roy. Assisted Reproductive Technology and Reproductive Options for the At-Risk Couple. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190681098.003.0008.
Повний текст джерелаSturt, Amy S., and Jennifer S. Read. Human Immunodeficiency Virus Type 1. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0008.
Повний текст джерелаGaitanis, John, Phillip L. Pearl, and Howard Goodkin. The EEG in Degenerative Disorders of the Central Nervous System. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0013.
Повний текст джерелаЧастини книг з теми "Prenatal transmission"
Moog, Nora K., Pathik D. Wadhwa, and Claudia Buss. "Intergenerational Transmission of Parental Early Life Stress." In Prenatal Stress and Child Development, 113–30. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-60159-1_6.
Повний текст джерелаCoutelle, Charles, Simon N. Waddington, and Michael Themis. "Monitoring for Potential Adverse Effects of Prenatal Gene Therapy: Mouse Models for Developmental Aberrations and Inadvertent Germ Line Transmission." In Prenatal Gene Therapy, 329–40. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-873-3_15.
Повний текст джерелаParris, Kerry M., and Shamanthi M. Jayasooriya. "Prenatal Risk Assessment for Preterm Birth in Low-Resource Settings: Infection." In Evidence Based Global Health Manual for Preterm Birth Risk Assessment, 31–39. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-04462-5_5.
Повний текст джерелаBatista, Sharon M., and Jocelyn Soffer. "HIV through the Life Cycle." In Handbook of AIDS Psychiatry. Oxford University Press, 2010. http://dx.doi.org/10.1093/oso/9780195372571.003.0007.
Повний текст джерелаProniewski, Andrzej. "Godność dziecka nieredukowalną wartością ontyczno-wychowawczą." In Dziecko w historii - między godnością a zniewoleniem. Tom 1. Dziecko jako fundament praw człowieka, 117–36. Wydawnictwo Uniwersytetu w Białymstoku, 2021. http://dx.doi.org/10.15290/dhmgz.01.2021.08.
Повний текст джерелаWillen, Elizabeth J., and Anai Cuadra. "Pediatric Human Immunodeficiency Virus." In Cognitive and Behavioral Abnormalities of Pediatric Diseases. Oxford University Press, 2010. http://dx.doi.org/10.1093/oso/9780195342680.003.0035.
Повний текст джерелаТези доповідей конференцій з теми "Prenatal transmission"
Kurniati, Nurul. "Analysis of Factors and Management of Hepatitis B Virus Screening in Mothers and Infants: A Scoping Review." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.03.67.
Повний текст джерелаCastro, Ana Flávia Silva, Natália Barros Salgado Vieira, and Sarah Joanny da Silva Pereira. "Correlation between Zika virus and microcephaly as a consequence of congenital infection." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.629.
Повний текст джерелаGiannelli, B. F. "MOLECULAR GENETICS OF HAEMOPHILIA." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643981.
Повний текст джерела