Добірка наукової літератури з теми "Pisa syndrome"
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Статті в журналах з теми "Pisa syndrome"
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Uemura, Takeshi, Yuichi Kasai, Kentaro Araki, and Atsumasa Uchida. "Pisa Syndrome." Journal of Spinal Disorders & Techniques 21, no. 6 (August 2008): 455–57. http://dx.doi.org/10.1097/bsd.0b013e3181570889.
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Michel, Sáenz Farret, Arias Carrión Oscar, Thalia Estefania Sánchez Correa, Pellene Luis Alejandro, and Federico Micheli. "Pisa Syndrome." Clinical Neuropharmacology 38, no. 4 (2015): 135–40. http://dx.doi.org/10.1097/wnf.0000000000000092.
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Stubner, Susanne, Frank Padberg, Renate Grohmann, Harald Hampel, Matthias Hollweg, Hanns Hippius, Hans-Jurgen Moller, and Eckart Ruther. "Pisa Syndrome (Pleurothotonus)." Journal of Clinical Psychiatry 61, no. 8 (August 15, 2000): 569–74. http://dx.doi.org/10.4088/jcp.v61n0805.
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Remington, Gary J. "The Pisa Syndrome." Journal of Clinical Psychopharmacology 8, no. 3 (June 1988): 228. http://dx.doi.org/10.1097/00004714-198806000-00025.
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Amore, Mario. "THE PISA SYNDROME." Clinical Neuropharmacology 15 (1992): 265B. http://dx.doi.org/10.1097/00002826-199202001-00511.
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Harada, Kenichi, Nobuyuki Sasaki, Hiroshi Ikeda, Norihito Nakano, Hiroki Ozawa, and Toshikazu Saito. "Risperidone-Induced Pisa Syndrome." Journal of Clinical Psychiatry 63, no. 2 (February 15, 2002): 166. http://dx.doi.org/10.4088/jcp.v63n0213b.
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Mascia, Marcello Mario, and Giovanni Defazio. "Cocaine-induced Pisa syndrome." Neurological Sciences 42, no. 9 (June 19, 2021): 3927–28. http://dx.doi.org/10.1007/s10072-021-05404-x.
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Teng, Po-Ren, and Te-Jen Lai. "Paliperidone-Related Pisa Syndrome." Journal of Clinical Psychopharmacology 33, no. 1 (February 2013): 129–31. http://dx.doi.org/10.1097/01.jcp.0000426181.89572.18.
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Smith, Connor, Naomi Gwynn, and Jean-Pierre Lindenmayer. "Pisa Syndrome and Aripiprazole." Journal of Clinical Psychopharmacology 40, no. 1 (2020): 99–101. http://dx.doi.org/10.1097/jcp.0000000000001162.
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Lee, Yen-Feng. "Antipsychotic-Induced Pisa Syndrome." Clinical Neuropharmacology 41, no. 2 (2018): 60–63. http://dx.doi.org/10.1097/wnf.0000000000000274.
Повний текст джерелаДисертації з теми "Pisa syndrome"
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DE, ICCO ROBERTO. "TRANSCRANIAL DIRECT CURRENT STIMULATION (t-DCS) AS ADD-ON TO NEUROREHABILITATION OF PISA SYNDROME IN PARKINSON’S DISEASE: A RANDOMIZED CONTROLLED TRIAL." Doctoral thesis, Università degli studi di Pavia, 2020. http://hdl.handle.net/11571/1371675.
Повний текст джерелаАнотація:
Background: Pisa Syndrome (PS) is a lateral trunk flexion frequently associated to Parkinson’s disease (PD). The management of PS is still a challenge for the physicians, because it poorly responds to the anti-parkinsonian drugs, and the improvement achieved with neurorehabilitation or botulinum toxin injections tends to fade in 6 months or less. Transcranial direct current stimulation (t-DCS) is a non-invasive neuromodulation technique, which showed promising results in movement disorders. The aim of our study is to evaluate the role of bi-hemispheric t-DCS as add-on to neurorehabilitation in PS.
Methods: Twenty-eight patients with PD and PS (21 male, age 72.9±5.1 years, PD duration 9.3±7.4 years, PS duration 3.0±1.9 years) received a 4-week intensive neurorehabilitation treatment and were randomized to receive t-DCS (t-DCS group, n=13), 5 daily sessions (20 minutes - 2 mA) with cathode over the primary motor cortex (M1) contralateral to PS, and anode over the M1 cortex ipsilateral to PS, or sham group (sham group, n=15). At baseline (T0), end of rehabilitation (T1) and 6 months later, patients were evaluated with trunk kinematic analysis in static and dynamic conditions, UPDRS-III, FIM, and VAS for lumbar pain rating. At T0, the evaluations were completed by an EMG study of trunk muscles.
Results: The study groups were comparable for clinical/demographic features and EMG phenotypes. When compared to sham group, t-DCS group achieved better results in several variables: overall posture (p=0.014), lateral inclination (p=0.013) of trunk during upright standing position, total range of motion (ROM) of the trunk (p=0.012), ROM of bending ipsilateral to PS (p=0.037), and ROM of anterior trunk flexion (p=0.014). The improvement in the overall trunk posture in upright standing position was persistent in t-DCS group at 6 months (T2 vs. T0: p<0.05). UPDRS-III scores decreased after rehabilitation (p=0.001), without significant differences between t-DCS and sham groups (p=0.942). In contrast, FIM score and lumbar pain intensity improved the most in t-DCS group when compared to sham group (p=0.048, and p=0.017 respectively). The EMG pattern was not a predictor of the efficacy of the t-DCS treatment.
Conclusions: Our data supports the use of neuromodulation with t-DCS as add-on to neurorehabilitation for the treatment of patients affected by PS in PD. t-DCS is a non-invasive and repeatable approach that proved effective even in those patients with an EMG pattern not amenable to botulinum toxin injections.
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Esparza-Romero, Julian. "RISK FACTORS OF TYPE 2 DIABETES IN MEXICAN AND U.S. PIMA INDIANS: ROLE OF ENVIRONMENT." Diss., The University of Arizona, 2010. http://hdl.handle.net/10150/195732.
Повний текст джерелаАнотація:
Introduction. Pima Indians living in the United States (U.S.) have the highest prevalence of type 2 diabetes mellitus in the world. Their Mexican counterparts, living a traditional lifestyle in the mountain of Sonora, Mexico, have at least five times less diabetes than the U.S. Pima Indians. The effects of a traditional lifestyle in reducing type 2 diabetes risk factors and the association of factors to type 2 diabetes were evaluated in a sample of 1211 genetically related Pima Indians living different lifestyles (224 from Mexico and 887 from U.S.). Subsets of these populations were used to address specific questions. First, differences in insulin resistance between subjects with normal glucose tolerance (194 Mexican versus 449 U.S. Pima) were evaluated. Second, the effect of physical activity and obesity explaining differences in metabolic syndrome prevalence were evaluated in 224 and 447 Mexican and U.S. Pima Indians. Third, factors associated with type 2 diabetes were evaluated in each Pima Indian population (224 from Mexico and 887 from U.S.).Methods. Demographic, physical, biochemical, and lifestyle factors were measured in 1996 in a cross-sectional study of Pima Indians 20 years of age or older living in Maycoba, Sonora Mexico and contrasted to results from a sample of U.S. Pima Indians participating in an ongoing epidemiological study that used similar methods and selection criteria. Insulin resistance was estimated by both fasting insulin and homeostasis model assessment-insulin resistance (HOMA-IR). Metabolic syndrome was defined using the Third Report of the National Cholesterol Education Program's Adult Treatment Panel III (NCEP III) criteria. Body mass index (BMI) was calculated by dividing weigh in kilograms by the square of height in meters (Kg/m2). Physical activity was measured using a questionnaire developed for the U.S. Pima Indians and adapted to the Mexican Pima Indian population. Type 2 diabetes was defined according to the 1999 WHO criteria after an oral glucose tolerance test. Multiple linear regression analysis was used to answer the first question (related to differences in insulin resistance) and multiple logistic regressions analysis to answer the second (related with differences in metabolic syndrome) and third questions (related to factors associated with type 2 diabetes).Results. Insulin resistance was much lower in the Mexican Pima Indians than in genetically related U.S. counterparts, even after controlling for differences in obesity, age and sex. In addition, the unadjusted prevalence of metabolic syndrome was 24.1% and 56.6 % in the Mexican and U.S. Pima Indians, respectively. However, most of the difference in metabolic syndrome prevalence was explained by differences in obesity and physical activity. Furthermore, in Mexican Pima Indians, type 2 diabetes was independently associated with age, fasting insulin, and waist circumference. In the U.S. Pima Indians, type 2 diabetes was associated with with age, sex, fasting insulin, total cholesterol, blood pressure and physical activity.Conclusion. The findings underscore the importance of lifestyle in the prevention of type 2 diabetes risk factors, such as insulin resistance and metabolic syndrome, even in individuals with high propensity to develop diabetes.
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Murray, Helen B., Jennifer J. Thomas, Andreas Hinz, Simone Munsch, and Anja Hilbert. "Prevalence in Primary School Youth of Pica and Rumination Behavior: The Understudied Feeding." Universitätsklinikum Leipzig, 2018. https://ul.qucosa.de/id/qucosa%3A36121.
Повний текст джерелаАнотація:
Objective: Little epidemiological evidence exists on rumination disorder behavior (RB) and pica behavior (PB). We examined prevalence of RB and PB and presence of comorbid feeding/eating disorder symptoms among school-aged children. Methods: In elementary schools in Switzerland, 1,430 children (54.0% female) ages seven to 13 completed Eating Disorder Examination-Questionnaire for children (ChEDE-Q) and Eating Disturbances in Youth Questionnaire (EDY-Q). Results: EDY-Q data behavior frequency showed 9.7% reported RB only, 10.0% reported PB only, and 3.1% reported RB+PB (≥1 on 0-6 Likert scale). At a clinical cut-off score of ≥4 (at least “often true”), 1.7% had RB only, 3.8% had PB only, and 1.1% had RB+PB. Avoidant/restrictive food intake disorder symptoms were most common in those with RB+PB, and more common in those with RB or PB than those without. Degree of eating disorder symptoms (by ChEDE-Q) over the past 28 days were similar among those with RB, PB, or RB+PB, but less common in those without RB or PB. Discussion: RB and PB were commonly reported in our sample of school-aged children, even at a potential clinically significant cut-off. Our findings also suggest that degree of eating disorder symptom comorbidity is similar between those with RB and PB.
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Rocha, ?rika Dantas de Medeiros. "Interven??o dietoter?pica em portadores de lipodistrofia generalizada cong?nita do Rio Grande do Norte." Universidade Federal do Rio Grande do Norte, 2008. http://repositorio.ufrn.br:8080/jspui/handle/123456789/13223.
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Previous issue date: 2008-10-20Congenital generalized lipodystrophy is a rare genetic disease with autosomal recessive inheritance characterized by the generalized absence of subcutaneous adipose tissue and insulin resistance. The aim of our study was to determine the profile of patients with congenital generalized lipodystrophy (Berardinelli-Seip syndrome) through their clinical history, eating habits, and socioeconomic and cultural aspects; assess food consumption and nutritional status of the study group; propose and evaluate a diet therapy model associated to oral supplementation with zinc to help in the control and prevention of metabolic complications associated to the pathology. Initial assessment of food consumption indicated a voracious appetite in all the patients studied. The introduction of zinc reduced appetite, contributing to patient adherence to the food plan proposed. It was also observed that the proposed diet contributed mainly to glycidic control, specifically with respect to HbA1c. The nutritional status of the patients investigated was adequate in terms of body mass index (BMI), arm muscle circumference (AMC), arm muscle area AMA, but triceps skinfold (TSF) indicated serious malnutrition. Our study is unique in the literature and provides important information to the field of nutrition and to individuals with this pathology. Furthermore, it contemplates the interdisciplinary and multidisciplinary requirements of the Postgraduate Program in Health Sciences of the Federal University of Rio Grande do Norte (UFRN), Natal, Brazil
A lipodistrofia generalizada cong?nita ? uma rara doen?a gen?tica com heran?a autoss?mica recessiva caracterizada por aus?ncia generalizada de tecido adiposo subcut?neo e resist?ncia insul?nica. O nosso estudo objetivou conhecer o perfil de portadores de lipodistrofia generalizada cong?nita mediante hist?ria cl?nica, h?bitos alimentares, aspectos s?cio-econ?micos e culturais; avaliar o consumo alimentar e o estado nutricional do grupo estudado; propor e avaliar um modelo de interven??o dietoter?pica associada ? suplementa??o oral com o elemento zinco para auxiliar o controle e a preven??o de complica??es metab?licas associada ? patologia. A avalia??o inicial do consumo alimentar indicou apetite voraz comum em todos os portadores investigados. Com a introdu??o do elemento zinco, verificou-se redu??o do apetite contribuindo para a ades?o dos mesmos ao plano alimentar proposto. Observou-se, tamb?m, que a dieta proposta contribuiu, principalmente, ao controle do metabolismo glic?dico, especificamente, em rela??o ? hemoglobina glicada (HbA1c). O estado nutricional dos portadores investigados revelou-se adequado em rela??o ao ?ndice de massa corporal (IMC), circunfer?ncia do m?sculo do bra?o (CMB) e ?rea da massa do bra?o (AMB), e indicou desnutri??o grave em rela??o ? prega cut?nea do tr?ceps (PCT). O nosso estudo ? ?nico na literatura e traz contribui??es importantes ao campo da nutri??o e ? popula??o portadora desta patologia. Al?m disto, contemplou os requisitos de interdisciplinaridade e multidisciplinaridade exigidos pelo Programa de P?sgradua??o em Ci?ncias da Sa?de
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Polverino, Giovanni. "Personality and pace-of-life syndrome in fishes: New perspectives." Doctoral thesis, Humboldt-Universität zu Berlin, 2017. http://dx.doi.org/10.18452/18569.
Повний текст джерелаАнотація:
Individuelle Verhaltensunterschiede (sog. „Animal personality“) werden oft als konsistent über die Zeit und situationsunabhängig angenommen. Vielfach werden solche Persönlichkeitsunterschiede zwischen Tieren einer Art durch individuelle Unterschiede im Energiehaushalt sowie Lebenszyklusvariablen (sog. ‚state variables‘, dt. Zustandsgrößen) erklärt. Dies ist in der „pace-of-life“ Hypothese zusammengefasst. In neueren Arbeiten wurde jedoch die Konsistenz von Persönlichkeitsmerkmalen über den Lebensverlauf und deren strikte Abhängigkeit von Zustandsgrößen in Frage gestellt. Die vorliegende Dissertation soll neue Einblicke in die Mechanismen, die zur Entstehung von Persönlichkeitsunterschieden im Verlauf des Lebens von Fischen, deren Situationsabhängigkeit sowie ihre Verbindung zu individuellen Unterschieden in Zustandsgrößen liefern. In einer Abfolge von fünf unabhängigen Studien untersuchte ich die genannten Annahmen und fand, dass (1) Persönlichkeitsunterschiede sich im Laufe des Lebens von Tieren vergrößern; (2) Persönlichkeitsabschätzungen bei jungen Tieren oft stärker vom experimentellen Aufbau beeinflusst werden als bei Erwachsenen; (3) der Energiehaushalt und Lebenszyklusvariablen Persönlichkeitsunterschiede sowohl unter Laborbedingungen als auch im Freiland nicht hinreichend erklären können; (4) Beziehungen zwischen Persönlichkeitsunterschieden und Unterschieden im Energiehaushalt und in Lebenszyklusvariablen fanden sich nur bei Fischen einer Population mit langsamer Lebenszyklusstrategie nicht jedoch in einer Population mit schnellem Lebenszyklus. Die vorliegende Arbeit suggeriert daher, dass sich erst im Verlauf des Lebens eines Tieres Persönlichkeitsunterschiede unvermeidbar entwickeln. Dies stellt Persönlichkeitsmessungen bei juvenilen Tieren grundsätzlich in Frage. Weiterhin scheinen Persönlichkeitsunterschiede und Zustandsgrößen unter bestimmten Umweltbedingungen und evolutiven Szenarien voneinander unabhängig zu sein.Among-individual differences in behavior (i.e., animal personality) are assumed to be consistent over time and contexts. In theory, they are often explained by individual variations in energy costs of self-maintenance as well as life history among animals (i.e., state variables), commonly expressed as the pace-of-life syndrome hypothesis. Yet, recent theories have disputed the consistency of personality types over lifetime and their rigid state-dependency. This thesis aims to offer novel insights on the mechanisms behind the emergence and development of personality over lifetime of fishes, its context dependency, and its link to individual variation in state variables. In a sequence of five independent yet interconnected studies, I tested the assumptions above and observed that (1) personality differences increased during lifetime as a function of consistent declines in the behavioral plasticity with increasing age of animals; (2) personality estimates in young animals were weaker and thus more vulnerable to experimental biases compared to adults; (3) personality variation did not depend upon individual differences in energy costs of self-maintenance and life-history traits among individuals, under both laboratory and natural settings; and (4) the relationship between behavioral, metabolic, and life-history traits was manifested only in fish populations with slow rather than fast life-history strategies. This thesis suggests that personality variation in animals might be the inevitable outcome of development, raising questions about the reliability of personality estimates in juvenile individuals. Furthermore, individual variation in personality and “states” may act independently (i.e., phenotypes are uncorrelated) under environmental conditions and evolutionary contexts that mask or select against their trade-offs.
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Moffett, Carol D. "The Impact of Childhood Measures of Glycemia and Insulin Resistance Factors on Follow-Up Glycemic Measures." Diss., The University of Arizona, 2007. http://hdl.handle.net/10150/194096.
Повний текст джерелаАнотація:
The purpose of this research was to evaluate the impact of glycemic measures, and changes in identified risk factors (BMI, waist circumference, lipids, blood pressure) on follow-up glycemia, in Pima children at high risk for type two diabetes (type 2 DM).I computed incidence and cumulative incidence of type 2 DM in Pima children 5-19 years of age between 1983 and 2004. Cox proportional hazards rates for development of type 2 DM were calculated by glycemic measure (HbA1C, 20PG, FPG) controlling for confounding factors (age, sex, BMI, blood pressure, and cholesterol). Diabetes was defined by the presence of at least one of four criteria: 1) 20PG of >200 mg/dl, 2) FPG of >126 mg/dl, 3) HbA1C > 8.0%, or 4) hypoglycemic treatment. Linear regression models were computed to identify the impact of changes in risk factors on changes in HbA1C. Only exams performed in non-diabetic children during childhood were included in the regression models.Among 2658 non-diabetic children, 258 cases of diabetes occurred during mean 9.1 years of follow-up (1.5 - 21.7). The age-sex adjusted incident rate of diabetes was 19.0 cases per 1000 person-years, and cumulative incidence was 54% by age 40. Incidence rates increased with increasing baseline values of 20PG, and FPG, but not for HbA1C. For HbA1C the relationship was u-shaped with the lowest and highest quartiles having the highest DM rates. After adjustment for confounding risk factors using Cox proportional hazards analysis, the risk for diabetes increased 2-fold for every 10 mg/dl increase in FPG. Changes in waist circumference best predicted changes in HbA1C (R2 = 0.48, Ï <0.001). However, the ability of waist circumference to predict change is limited due to the powerful effect of regression to the mean, suggesting that these risk factors contribute very little to changes in HbA1C, at least in childhood.Childhood levels of glycemia predict development of type 2 DM later in life. While changes in waist circumference are associated with only moderate changes in HbA1C, this does not refute the significant contribution of adiposity in childhood to the development of type 2 DM.
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Ara?jo, Edilene Maria Queiroz. "Interven??o dietoter?pica na s?ndrome metab?lica e sua associa??o com o perfil gen?tico da intoler?ncia ? lactose." Universidade Estadual de Feira de Santana, 2016. http://localhost:8080/tede/handle/tede/528.
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Metabolic syndrome (MS) is a complex disorder with a strong genetic basis and multifactorial etiology. Insulin resistance (IR) causes MS and it can be triggered by intestinal inflammation like the use of lactose in patients intolerant of this carbohydrate. It was found that variants in the lactase gene are associated with lactase non persistence LNP and MS in a population sample of Salvador/Bahia; and whether these variants are modifying the response to diet-therapeutic intervention in patients with this syndrome; also compared the biochemical test of lactose tolerance (LTT) with genetic diagnosis; and tested the association of mutations in the lactase gene with cofactors SM (TGL, HDL-C, blood pressure, glucose levels, waist circumference), with anthropometric variables (Arm Circumference, Body Mass Index, Hip Circumference, hip-waist ratio,lean massand fat mass percentages) and other factors associated with MS: insulin, total cholesterol, LDL-C, VLDL-C, C-reactive protein, HOMA-IR, renal function (creatinine, urea, uric acid, microalbuminuria) and vitamin D. There were two studies: a case-control with 257 cases (MS) and 210 controls and other clinical trial study, which was conducted with three types of diet in patients with metabolic syndrome: diet 1 - No lactose; Diet 2 - Lactose and energy restriction; Diet 3 - Only energy restriction. In all groups were also evaluated for nine SNPs in the lactase (LCT) gene. The genotyping of SNPs was carried out by TaqMan assays. Data were analyzed using SPSS, 20.0, and the Hardy-Weinberg Equilibriumhaplotype frequencies were calculated using Arlequin, 2000 program. The results showed that all diets improve several MSaspects after two months of intervention, especially in the diet 1, that also decreased inflammation, insulin resistance and dyslipidemia (LDL-C). In addition,it was the diet that most took out patients of the MS: 2.72 times more likely to get out of MS than diet 3. LNP was high in both cases and controls. There was compatibility between clinical diagnosis for LNP by Lactose Tolerance Test and two of the studied SNPs, they were rs4988253 and rs182549, those that have proved functional studies. Thus, it is suggested the analysis of LCT gene polymorphisms before the nutritional therapeutics for patients with MS, as well as to take out the lactose in their diet.
A S?ndrome Metab?lica (SM) ? uma desordem complexa, de forte base gen?tica e de etiologia multifatorial. Dentre as suas causas, encontra-se a Resist?ncia ? Insulina (RI) que pode ser desencadeada pela inflama??o intestinal, pelo uso de lactose em pacientes intolerantes a este carboidrato. Verificou-se quais variantes no gene da lactase est?o associados ? IL e SM em amostra da popula??o de Salvador/Bahia; e tamb?m se estas variantes s?o modificadoras da resposta ? interven??o dietoter?pica em portadores desta s?ndrome; comparou-se tamb?m o teste bioqu?mico de toler?ncia ? lactose (TTL) com o diagn?stico gen?tico; e testou-se a associa??o das muta??es no gene da lactase com os cofatores da SM (TGL, HDL-c, press?o arterial, glicemia, circunfer?ncia da cintura), com vari?veis antropom?tricas (circunfer?ncia do bra?o, ?ndice de massa corporal, circunfer?ncia do quadril, raz?o cintura quadril, percentual de massa magra e massa gorda) e com outros fatores associados ? SM: insulina, colesterol total, LDL-C, VLDL, Prote?na C reativa, HOMA-IR, fun??o renal (creatinina, ur?ia, ?cido ?rico, microalbumin?ria) e vitamina D. Foram realizados dois estudos: um caso-controle com 257 casos (SM) e 210 controles e outro estudo de tipo ensaio cl?nico, que foi realizado com tr?s tipos de dieta com os pacientes com SM: dieta 1 ? sem lactose; dieta 2 ? sem lactose e com restri??o energ?tica; Dieta 3 ? apenas restri??o energ?tica. Em ambos os grupos tamb?m foram avaliados 9 SNPs no gene da lactase. A genotipagem dos SNPs foi realizada pela tecnologia de ensaios TaqMan. Os dados foram analisados pelo programa SPSS ver 20.0 e a adequa??o das frequ?ncias genot?picas ao Equilibrio de Hardy-Weinberg e c?lculo da frequ?ncia dos hapl?tipos formados pelos polimorfismos foram obtidos atrav?s do programa Arlequin ver 2000. Os resultados mostraram que todas as dietas melhoram o quadro da SM ap?s dois meses de interven??o, com destaque para a dieta 1, que tamb?m diminuiu a inflama??o, resist?ncia ? insulina e a dislipidemia (LDL-C). Al?m disso foi a dieta que mais tirou paciente da SM: apresentou 2,72 vezes mais chances de sair da SM que a dieta 3. A intoler?ncia ? lactose foi alta tanto em casos como em controles. Houve compatibilidade do TTL com os SNPsrs4988253 e rs182549, os ?nicos que possuem estudos funcionais. Assim, sugere-se an?lise de polimorfismos do gene da lactase antes da prescri??o nutricional para pacientes com SM, bem como, a retirada da lactose da dieta.
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Silva, Ana Flávia Silveira. "Avaliação da qualidade ambiental interior de um edifício climatizado artificialmente com ênfase na análise do conforto térmico." Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/18/18138/tde-28122010-131829/.
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Ocupantes de ambientes internos climatizados artificialmente estão expostos contínua e prolongadamente a condições ambientais muitas vezes desfavoráveis à execução de suas atividades e à manutenção da saúde. O objetivo desta pesquisa foi avaliar alguns parâmetros de qualidade do ar interior e de conforto térmico de um edifício climatizado artificialmente e relacioná-los à utilização de sistema de condicionamento de ar com distribuição pelo piso em ambientes que não são caracterizados como escritórios abertos. Considerando que o edifício estudado apresenta usos distintos de seus pavimentos, optou-se pela avaliação de dois deles, um pavimento cujo layout é de salas de aula e outro de escritório semi-aberto. Os parâmetros foram analisados em três momentos distintos. O primeiro se caracteriza por três ciclos semestrais de medições de temperatura, umidade relativa e velocidade do ar, concentração de aerossóis, dióxido de carbono e fungos. A segunda etapa consiste em uma campanha única de medições, com duração de quatro dias, permitindo a realização de perfis de temperatura e umidade relativa, avaliação das velocidades do ar em pontos de desconforto e cálculo dos índices de conforto térmico PMV (Predicted Mean Vote) e PPD (Predicted Percentage of Dissatisfied). Em um terceiro e último momento, aplicou-se o questionário de qualidade ambiental interior aos ocupantes de ambos os pavimentos. Resultados obtidos nos ciclos permitiram identificar concentrações de fungos e dióxido de carbono acima dos limites indicados. Os perfis de temperatura revelaram a predominância das mesmas abaixo do recomendado. O cálculo dos índices PMV e PPD apontaram para um cenário de maior conforto térmico nos ambientes estudados com temperatura operativa igual a 24 graus Celsius. Os resultados dos questionários corroboraram as medições de temperatura, indicando a prevalência das sensações térmicas relacionadas ao frio entre os ocupantes do edifício, em especial aqueles do sexo feminino. Ficou evidenciado que o conforto térmico nos ambientes pesquisados é um fator perturbador das atividades exercidas em ambos os pavimentos. Houve um grande número de relatos de ocupantes com sintomas típicos da Síndrome dos Edifícios Doentes (SED), sugerindo que medidas relativas à qualidade ambiental devem ser tomadas em prol da saúde, bem estar e produtividade dos ocupantes do edifício. Concluiu-se que a operação e manutenção do sistema de condicionamento de ar com insuflamento pelo piso em ambientes distintos de escritórios abertos são mais complexas e, por isso, dificultam o alcance de uma qualidade ambiental interior satisfatória.Occupants of artificially conditioned indoor spaces are continuously and lengthily exposed to environmental conditions mostly adverse to their activities performance and health maintenance. The purpose of this research was to evaluate some indoor air quality and thermal comfort parameters of an artificially conditioned building, and relate them to the employment of underfloor air conditioning system in spaces that differ from open-plan office spaces. Considering the studied building presents different activities on each of its floors it was necessary to choose two of them, one characterized by classrooms layout and the other by a semi open-plan office layout. The on-site data collection took place in three different steps. Step one was distinguished by three six-month cycles of air temperature, relative humidity and air velocity measurements, and fungi, particulate matter and carbon dioxide concentrations. The second step consisted of a four-day single campaign of measurements, when air temperature and relative humidity profiles were carried out, air velocity was quantified in complaining areas, and the Predicted Mean Vote (PMV) and Predicted Percentage of Dissatisfied (PPD) thermal comfort indexes were determined. During the third and last step, indoor environmental quality questionnaire surveys were distributed to the occupants of both studied floors. Data analysis and assessment originated from the cycles identified exceeding fungi and carbon dioxide concentrations. Temperature profiles indicated their prevalence below the recommended range. PMV and PPD indexes determination pointed to a scenery of best thermal environmental conditions for the researched spaces, with an operative temperature of 24 Celsius degrees. The results of the questionnaire surveys supported the air temperature measurements, showing supremacy of cold related thermal sensations among the occupants, especially those of female gender. It was evident that the thermal comfort of the studied environments is a disturbing factor for the activities practiced on both floors. There was a great number of occupants reporting Sick Building Syndrome (SBS) typical symptoms, what suggested actions related to indoor environmental quality should be taken in order to provide the desired health, welfare and productivity for the building occupants. It was conclusive that the operation and maintenance of underfloor air conditioned systems in indoor environments unlike open-plan offices are more complex and therefore make it harder to reach an acceptable indoor environmental quality.
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GEROIN, Christian. "Neurophysiological and biomechanical investigations of postural disorders in patients with Parkinson disease: implications for rehabilitation." Doctoral thesis, 2016. http://hdl.handle.net/11562/939178.
Повний текст джерелаАнотація:
Le deformità posturali rappresentano una delle complicazioni che colpiscono il paziente affetto da Malattia di Parkinson (MP). Tali deformità inducono un’importante disabilità nel paziente, limitandolo nelle attività di vita quotidiana. Tra le diverse deformità della postura si possono annoverare la sindrome di Pisa, la camptocormia, l’anterocollo e la scoliosi.In particolare, la sindrome di Pisa (SP) è una deformità clinicamente definita come una flessione laterale del tronco di almeno 10° che può essere ridotta completamente attraverso la mobilizzazione passiva o durante la posizione supina. Essa può essere concomitante anche con altre deformità come per esempio la scoliosi, la camptocormia e l’anterocollo. Recentemente, attraverso uno studio osservazionale multicentrico abbiamo stimato la prevalenza della SP, in Italia essere dell’ 8%.Le evidenze suggeriscono che le deformità posturali nella MP possono avere due differenti meccanismi patofisiologici: 1) un meccanismo centrale, confermato da studi sull’animale e clinici sull’essere umano, relativo a un difetto di funzionamento dei gangli della base il quale causa distonia e problemi d’integrazione sensorimotoria; e 2) meccanismi periferici attribuibili a cambiamenti del sistema muscoloscheletrico e miopatia.Il mio progetto di dottorato si pone l’obiettivo di valutare gli aspetti neurofisiologici e biomeccanici della SP nei pazienti con MP. Un miglioramento delle conoscenze riguardo questi aspetti ci permetterà di sviluppare e implementare strategie riabilitative per la gestione di queste complicanze che sono molto disabilitanti nel paziente affetto da MP.Postural deformities are a common and disabling complication of Parkinson’s disease (PD). These deformities include Pisa syndrome, camptocormia, antecollis and scoliosis.In particular, Pisa Syndrome (PS) has been clinically defined as a lateral trunk flexion of 10° which can be alleviated completely by passive mobilization or on lying supine and can be concomitant or not with other postural deformities such as scoliosis, camptocormia and antecollis. In our recent multicenter observational study involving 1631 PD patients, we estimated its prevalence in Italy of 8.8%. The evidence to date suggests that postural deformities have two different pathophysiologic mechanisms: (1) a central mechanism, sustained by both animal studies and clinical findings and related to a defect in basal ganglia functioning with dystonia along with impaired sensory-motor integration; and (2) a peripheral mechanism, referred to spinal tissues changes and paraspinal myopathy.My PhD research program intends to evaluate the neurophysiological and biomechanical aspects of PS in patients with PD. Improved understanding of the mechanisms underlying postural deformities in PD might ultimately lead us to more effective rehabilitation strategies for these disabling and drug-refractory complications.
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Abenhaim, Samantha. "Identification de la sumoylation de ZNF74 et de l'interaction de cette protéine à multidoigt de zinc avec UBC9 et PIAS1." Thèse, 2004. http://hdl.handle.net/1866/14787.
Повний текст джерелаКниги з теми "Pisa syndrome"
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D, Matthews Dawn, ed. Eating disorders sourcebook: Basic consumer health information about eating disorders, including information about anorexia nervosa, bulimia nervosa, binge eating, body dysmorphic disorder, pica, laxative abuse, and night eating syndrome, along with information about causes, adverse effects, and treatment and prevention issues, and featuring a section on concerns specific to children and adolescents, a glossary, and resources for further help and information. Detroit, MI: Omnigraphics, 2001.
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Kaplan, Tamara, and Tracey Milligan. Cerebrovascular Disease 3: Brainstem Syndromes (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190650261.003.0006.
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The video in this chapter explores cerebrovascular disease, and focuses on brainstem syndromes. It includes the hallmark ‘crossed signs’ feature of brainstem syndromes (ipsilateral cranial nerve deficits, contralateral long track signs), the characteristics of lateral medullary syndrome due to a PICA stroke, and palsies that may result from a ventral pontine stroke, and medical midbrain stroke.
Частини книг з теми "Pisa syndrome"
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Bhidayasiri, Roongroj, and Daniel Tarsy. "Parkinsonism with Pisa Syndrome." In Current Clinical Neurology, 18–19. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60327-426-5_9.
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Diederich, N. J. "Pisa Syndrome." In Encyclopedia of Movement Disorders, 462–64. Elsevier, 2010. http://dx.doi.org/10.1016/b978-0-12-374105-9.00509-8.
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"Botulinum Toxin Therapy for Trunk Dystonia/Camptocormia/Pisa Syndrome." In Botulinum Toxin Dosing Manual. New York, NY: Springer Publishing Company, 2021. http://dx.doi.org/10.1891/9780826147660.0007.
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"Transthoracic anatomy and pathology: Valves." In Echocardiography, edited by Paul Leeson, Christiana Monteiro, Daniel Augustine, Harald Becher, Paul Leeson, Christiana Monteiro, Daniel Augustine, and Harald Becher, 287–386. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198804161.003.0004.
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This chapter deals with the anatomy and pathology of valves in transthoracic echocardiography. It covers the mitral valve (stenosis, regurgitation, PISA, and prolapse); the aortic valve (stenosis and low-flow stenosis and regurgitation); the bicuspid, quadricuspid, and tricuspid valves; carcinoid heart syndrome; the pulmonary valve (stenosis, regurgitation, and prolapse); prosthetic valves (mechanical and bioprosthetic), along with stenosis, regurgitation, and regurgitation; and finally endocarditis.
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Thomas, Sherine. "Post-Infection Syndrome." In Tutorial Topics in Infection for the Combined Infection Training Programme. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780198801740.003.0051.
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Post-infection syndrome (PIS) or chronic fatigue syndrome (CFS) is a complex debilitating disorder. It is usually characterized by fatigue that is worsened by physical activity or mental exertion, and is experienced in the aftermath, or with ongoing concurrent infections. Other symptoms may also be present including myalgia, impaired concentration, impaired memory, insomnia, and post-exertion malaise that can last for more than twenty-four hours after exertion. PIS/ CFS is a complex disorder with symptoms related to cognitive, autonomous, and immune dysfunction. No single causal factor has been identified, but there is some evidence that indicates that immunological dysfunction and infections interacting with genetic and psychosocial factors probably contribute to the development of PIS/ CFS. There are no tests to diagnose PIS/ CFS. There are many conditions where the symptoms of PIS/ CFS can appear, and therefore diagnosing PIS/ CFS may rely on ruling out other conditions. There are published guidelines that are available in order to help with diagnosing these conditions. The most frequently used ones are from the CDC (the 1994 Fukuda criteria) and the 1991 Oxford criteria. The CDC case definition for CFS requires individuals to meet three criteria before receiving this diagnosis. These are: 1. Severe chronic fatigue which must have been present for six or more consecutive months, and not as a result of other medical conditions associated with fatigue. 2. Fatigue that interferes significantly with activities of daily life. 3. Four or more of the following symptoms are present: a. Post-exertion malaise that lasts for longer than twenty-four hours. b. Impairment of short-term memory. c. Myalgia. d. Unrefreshed sleep. e. Headache (of new type or severity). f. Arthralgia (without swelling or erythema around the joints). g. A frequent or recurring sore throat. h. Tender lymphadenopathy. However, the Oxford criteria differentiates CFS of unknown aetiology and CFS related to PIS, which is CFS that either follows an infection or is associated with an ongoing current infection. These guidelines suggest that in order to diagnose CFS, individuals must meet the following criteria: 1. The principle symptom experienced by patients should be fatigue that affects physical and mental functioning, and should have been present for at least six months.
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Doumbia, A., Y. Coulibaly, I. Amadou, M. Keita, O. Coulibali, B. Kamaté, M. K. Djiré, et al. "Surgical Complications of Pica Syndrome: A Case Series." In Research Developments in Medicine and Medical Science Vol. 1, 37–46. B P International (a part of SCIENCEDOMAIN International), 2023. http://dx.doi.org/10.9734/bpi/rdmms/v1/4619a.
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Fiorani, Helga. "How to Bring Together Two Generations so Distant in Age, yet so Close by Heart." In Handbook of Research on Didactic Strategies and Technologies for Education, 379–87. IGI Global, 2013. http://dx.doi.org/10.4018/978-1-4666-2122-0.ch032.
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The focus of this chapter is on the annual project “Adottiamo i nonni dell’ospizio,” translated “Let’s adopt grandparents in retirement home” (LAGR), winner of the Special Prize of Giornata delle Marche (2008). The main aim of the project is to promote the education of young people to active citizenship. It was developed by the teacher L. Del Papa, with the collaboration of 19 pupils of the 5th class, section C, including a girl suffering from Down’s syndrome and the elderly guests of the Opera Pia Ceci nursing home in Camerano (Italy). The project is part of an educational trend called “the thread that links the senior and youth generations,” sponsored by the USR (Ufficio Scolastico Regionale delle Marche).
Тези доповідей конференцій з теми "Pisa syndrome"
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Pinheiro, Renato Serquiz Elias, Emanuelly da Costa Nobre Soares, Maria Eduarda Bezerra Figueiredo, and Stella Mandu Cicco. "Pisa syndrome in Parkinson’s disease: case description." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.644.
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Context: Pisa Syndrome (PS) is a rare postural disorder, characterized by dystonia of the trunk muscles, lateral deviation as well as rotation of the axial axis. There is a strong association with Parkinson’s disease (PD) due to the possible imbalance between neurotransmitters. It happens either due to a decrease in dopaminergic stimuli, either because of an excess of cholinergic stimuli or drugs (an example of antidopaminergics). The diagnosis is clinical, showing at least a 10-degree trunk flexion with improvement of pharmacological and non- pharmacological measures. Case report: A 60-year-old man was diagnosed with PD five years ago due to tipical clinical complaints and physical examination. The treatment recquired an increase of Pramipexole as well as the use of Levodopa and Benserazide. After two years, he complained about neck pain, low back pain, hip pain and a slight trunk twisting. After six months, his pain was worse and he reported right hemidystonia. Thus, he was diagnosed with PS associated with PD. It was decided to optimize the therapy with Pregabalin, muscle relaxants and rehabilitation. However, it did not show any good result. In 2020, the application of botulinum toxin (BTX) evidenced excellent results, improving both the pain and the spasticity of the patient. Conclusions: Early recognition is necessary to introduce the right treatment as soon as possible, especially BTX and rehabilitation, ensuring functionality and avoiding negative outcomes.
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Panero, Elisa, Daniele Borzelli, Carlo Alberto Artusi, and Giuseppe Massazza. "Biomechanical assessment of botulinum toxin effects in Pisa syndrome disease." In 2022 IEEE International Symposium on Medical Measurements and Applications (MeMeA). IEEE, 2022. http://dx.doi.org/10.1109/memea54994.2022.9856455.
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Goules, Andreas, Chiara Baldini, Saviana Gandolfo, Aristea Papageorgiou, Dimitris Ziogas, Francesco Ferro, Valentina Donati, et al. "SAT0190 LYMPHOMA IN PRIMARY SJÖGREN’S SYNDROME: A RETROSPECTIVE CLINICAL STUDY WITH PATIENTS FROM THE UPA (UDINE, PISA, ATHENS) GROUP." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.5638.
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