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Статті в журналах з теми "Periodontitis Longitudinal studies"
Leite, Fábio R. M., Gustavo G. Nascimento, Stina Baake, Lisa D. Pedersen, Flemming Scheutz, and Rodrigo López. "Impact of Smoking Cessation on Periodontitis: A Systematic Review and Meta-analysis of Prospective Longitudinal Observational and Interventional Studies." Nicotine & Tobacco Research 21, no. 12 (July 13, 2018): 1600–1608. http://dx.doi.org/10.1093/ntr/nty147.
Повний текст джерелаJu, Xiangqun, Jane Harford, Liana Luzzi, Gloria Mejia, and Lisa M. Jamieson. "A Longitudinal Study of Chronic Periodontitis in Two Cohorts of Community-Dwelling Elderly Australians." International Journal of Environmental Research and Public Health 19, no. 18 (September 19, 2022): 11824. http://dx.doi.org/10.3390/ijerph191811824.
Повний текст джерелаKhammissa, R. A. G., R. Ballyram, Y. Jadwat, J. Fourie, J. Lemmer, and L. Feller. "Vitamin D Deficiency as It Relates to Oral Immunity and Chronic Periodontitis." International Journal of Dentistry 2018 (October 1, 2018): 1–9. http://dx.doi.org/10.1155/2018/7315797.
Повний текст джерелаYan, Gabriel Lee Keng, Mei Na Tan, Mun Loke Wong, Chong Meng Tay, and Patrick Finbarr Allen. "Functional Dentition, Chronic Periodontal Disease and Frailty in Older Adults—A Narrative Review." International Journal of Environmental Research and Public Health 20, no. 1 (December 28, 2022): 502. http://dx.doi.org/10.3390/ijerph20010502.
Повний текст джерелаde Vries, Charlotte, Guillermo Ruacho, Elin Kindstedt, Barbara Aleksandra Potempa, Jan Potempa, Björn Klinge, Pernilla Lundberg, Elisabet Svenungsson, and Karin Lundberg. "Antibodies to Porphyromonas gingivalis Are Increased in Patients with Severe Periodontitis, and Associate with Presence of Specific Autoantibodies and Myocardial Infarction." Journal of Clinical Medicine 11, no. 4 (February 15, 2022): 1008. http://dx.doi.org/10.3390/jcm11041008.
Повний текст джерелаMootha, Archana, Sankari Malaiappan, N. D. Jayakumar, Sheeja S. Varghese, and Julie Toby Thomas. "The Effect of Periodontitis on Expression of Interleukin-21: A Systematic Review." International Journal of Inflammation 2016 (2016): 1–8. http://dx.doi.org/10.1155/2016/3507503.
Повний текст джерелаBortolini, Bruna Martinazzo, Pedro Henrique de Carli Rodrigues, Lidiane Ura Afonso Brandão, Danielle Shima Luize, Gladson Ricardo Flor Bertolini, Carlos Augusto Nassar, and Patricia Oehlmeyer Nassar. "BONE TISSUE BEHAVIOR OF RATS WITH EXPERIMENTAL PERIODONTITIS SUBJECTED TO PHYSICAL EXERCISE." Revista Brasileira de Medicina do Esporte 25, no. 2 (April 2019): 133–36. http://dx.doi.org/10.1590/1517-869220192502170693.
Повний текст джерелаQi, Xiang, Zheng Zhu, and Bei Wu. "Reciprocal Association Between Cognitive Function and Oral Health: A Systematic Review and Meta-Analysis." Innovation in Aging 4, Supplement_1 (December 1, 2020): 158. http://dx.doi.org/10.1093/geroni/igaa057.514.
Повний текст джерелаSantana, Danilo Dias, Gilberto Kac, Pedro Paulo Teixeira dos Santos, Thainá Castro da Silva, Camila Benaim, Paula Guedes Cocate, Maria Beatriz Trindade de Castro, Berit Lilienthal Heitmann, and Amanda Rodrigues Amorim Adegboye. "Association between Pre-Pregnancy BMI and Inflammatory Profile Trajectories during Pregnancy and Postpartum in Brazilian Women with Periodontitis: The IMPROVE Trial." International Journal of Environmental Research and Public Health 19, no. 5 (February 25, 2022): 2705. http://dx.doi.org/10.3390/ijerph19052705.
Повний текст джерелаKellesarian, Sergio Varela, Hans Malmstrom, Tariq Abduljabbar, Fahim Vohra, Tammy Varela Kellesarian, Fawad Javed, and Georgios E. Romanos. "“Low Testosterone Levels in Body Fluids Are Associated With Chronic Periodontitis”." American Journal of Men's Health 11, no. 2 (September 21, 2016): 443–53. http://dx.doi.org/10.1177/1557988316667692.
Повний текст джерелаДисертації з теми "Periodontitis Longitudinal studies"
Mário, Ticiane de Góes. "CICATRIZAÇÃO DE FUMANTES E NUNCA FUMANTES APÓS UM ANO DE TERAPIA PERIODONTAL NÃO CIRÚRGICA." Universidade Federal de Santa Maria, 2013. http://repositorio.ufsm.br/handle/1/6154.
Повний текст джерелаPeriodontal diseases are the result of complex interrelationships between bacterial biofilm and factors related to the host response. These relationships can be modified by several factors, among them the smoke. Many studies support the evidence that the smoke has a detrimental effect on the health of the periodontium. However, the effect of this habit in the results of periodontal treatment is still controversial and longitudinal studies of smokers and never smokers undergoing periodontal therapy are needed. Initially, 14 never smokers and 11 smokers completed the non-surgical periodontal treatment. After examination of three months, three participants were lost: one from the first group and two from the second. Thus, this study aimed to evaluate the response of 13 never smokers and nine smokers with chronic periodontitis, 12 months after this therapeutic approach. This study aimed to evaluate the response to non-surgical periodontal therapy in smokers and never smokers with chronic periodontitis, after a period of 12 months. Patients in both groups who received non-surgical periodontal treatment, were analyzed with respect to changes in bleeding on probing (BoP, BoP1 and BoP2), probing depth (PD), clinical attachment level (CAL), visible plaque index (VPI) and gingival bleeding index (GBI). To evaluate these parameters, trained and calibrated examiners performed periodontal clinical examinations prior to periodontal therapy and three, six and 12 months after treatment completion. The changes in scores for bleeding on probing were considered primary outcome and changes in other clinical parameters were secondary outcomes. The response to non-surgical periodontal therapy was similar between smokers and never smokers in changes related to VPI, mean and different strata of PD, BoP and BoP2. Similarities were also observed in BoP, BoP 1 and BoP 2 in different strata of PD, except for BoP 1 on PD 1-3mm in ever smokers had greater reductions. Differences between groups were also observed in the presence of GBI and BoP1. Changes in CAL were higher in the group of never smokers, the statistic difference being borderline.
As doenças periodontais são resultantes de inter-relações complexas entre biofilme bacteriano e fatores relacionados à resposta do hospedeiro. Essas relações podem ser modificadas por vários fatores, dentre eles, o fumo. Muitos estudos suportam a evidência de que o fumo prejudica a saúde do periodonto. Porém, o efeito desse hábito nos resultados do tratamento periodontal ainda é controverso e estudos de acompanhamento longitudinal de fumantes e nunca fumantes submetidos à terapia periodontal são necessários. Inicialmente, 14 nunca fumantes e 11 fumantes completaram o tratamento periodontal não cirúrgico. Após o exame de três meses, foram perdidos três participantes: um do primeiro grupo e dois do segundo. Assim, esse trabalho objetivou avaliar a resposta de 13 nunca fumantes e nove fumantes com periodontite crônica, 12 meses decorridos do término dessa abordagem terapêutica. Pacientes de ambos os grupos que receberam tratamento periodontal não cirúrgico, foram analisados com relação a alterações no sangramento à sondagem (SS, SS1 e SS2), na profundidade de sondagem (PS), no nível de inserção clínica (NIC) e nos índices de placa visível (IPV) e de sangramento gengival (ISG). Para a avaliação desses parâmetros, examinadores treinados e calibrados realizaram exames clínicos periodontais previamente à terapia periodontal e três, seis e 12 meses após o tratamento concluído. As mudanças nos escores de sangramento à sondagem foram consideradas desfecho primário e as alterações nos demais parâmetros clínicos avaliados, os desfechos secundários. A resposta à terapia periodontal não cirúrgica foi semelhante entre fumantes e nunca fumantes em relação às alterações de IPV, médias e diferentes estratos de PS, SS e SS2. Semelhanças também foram observadas em SS, SS1 e SS2 nos diferentes estratos de PS, com exceção de SS1 em PS de 1-3mm em que nunca fumantes tiveram maiores reduções. Diferenças entre os grupos também foram observadas no ISG e na presença de SS1. As alterações no NIC foram maiores no grupo de nunca fumantes, sendo a diferença estatística borderline.
Haas, Alex Nogueira. "Epidemiologia da progressão de perda de inserção periodontal : estudo de Porto Alegre." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2008. http://hdl.handle.net/10183/14770.
Повний текст джерелаThe aim of this thesis was to estimate the occurrence of progression of periodontal attachment loss (PAL) and determine its risk factors in an urban sample from South Brazil. The “Porto Alegre Study” started in 2001 and was an epidemiological survey with a representative sample of the Porto Alegre metropolitan area obtained by a multistage probability sampling. At baseline, the sample comprised 1465 dentate and 121 edentulous individuals (mean age: 37.9 years, standard deviation:13.3). After 5 years, 697 (47.6%) dentate (294 males and 403 females) and 48 edentulous subjects were re-evaluated. Demographics, behavioral and socioeconomic data were collected using a structured questionnaire. PAL was obtained in six sites of all teeth present by calibrated periodontists. Two analyses were performed, one aiming at estimating the occurrence of PAL progression, and another aiming at determining risk factors for PAL progression. Overall, 56.0% and 36.2% of the subjects presented PAL progression ≥3mm in 2+ and 4+ teeth, respectively. In general, extent of progression was localized with 3.76 teeth presenting PAL progression ≥3mm. Overall mean PAL progression was 0.3mm/year. When subjects were categorized according to the mean annual PAL progression, 67.0% of subjects showed a moderate progression rate (>0.1mm/year to ≤0.5mm/year), whereas 16.1% were deemed as having no/ slight (≤0.1mm/year) and 16.9% as rapid progression (>0.5mm/year) rates. PAL progression increased significantly with age and was statistically higher among males. The multivariable model showed that: 30-49 and 50+ years-old individuals had approximately two times higher risk than <30 year-olds; low education increased the risk of PAL progression by 53%; among never-smokers, males were 33% more likely to experience PAL progression than females, while the risk increased by 8% for smoker males and by 21% for smoker females per 10 packyears of lifetime smoking exposure. It can be concluded that a large proportion of this urban Brazilian sample was affected by PAL progression underscoring the need for health promotion initiatives aiming at preventing progression of destructive periodontal disease. Age, gender, education and smoking were found to be independent risk factors for PAL progression.
Lavin, Emilie, and Richard Truong. "Progression av obehandlad apikal parodontit vid rotfyllda tänder - En röntgenologisk, retrospektiv kohortstudie." Thesis, Malmö högskola, Odontologiska fakulteten (OD), 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:mau:diva-19990.
Повний текст джерелаThere is a high frequency of endodontically treated teeth with apical periodontitis in Sweden, but a lack of a well-established practice of how to deal with these. This study aims to investigate radiographic progression of untreated apical periodontitis in previously root-filled teeth left without intervention and to identify factors that affect the progression in order to reach a better foundation for making treatment decisions. Radiographs of root-filled teeth with apical periodontitis (n = 120) were examined with regards to the change of the apical periodontitis over at least three years during 2002-2011.Progression of the apical periodontitis was observed in 22.5 % of the studied teeth in spite of probable clinical decisions that treatment was not needed for the apical periodontitis or treatment could be postponed. None of the studied variables: the size of the apical periodontitis, the length of the root-filling, the sealing quality of the root-filling, the type of the coronal restoration or the sealing quality of the coronal restoration were found to be correlated to the progression of the apical periodontitis. The apical periodontitis was unchanged in 50 % of the teeth and observed more often in the maxilla (p=0.02).
Частини книг з теми "Periodontitis Longitudinal studies"
Ferrari Cestari, José Augusto, Gisele Maria Campos Fabri, Jorge Kalil, Ricardo Nitrini, Wilson Jacob-Filho, José Tadeu Tesseroli De Siqueira, and Silvia Regina D. T. Siqueira. "Oral Infections and Cytokine Levels in Patients with Alzheimer’s Disease and Mild Cognitive Impairment Compared with Controls." In Advances in Alzheimer’s Disease. IOS Press, 2022. http://dx.doi.org/10.3233/aiad220039.
Повний текст джерела