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Книги з теми "Oral cancers"

Щоб переглянути інші типи публікацій з цієї теми, перейдіть за посиланням: Oral cancers.

Автор: Grafiati

Опубліковано: 10 грудня 2022

Оновлено: 29 січня 2023

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1

Bahadur, Sudhir, and Subramania Iyer, eds. Management of Oral Cancers. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-6499-4.

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2

Ogbureke, Kalu U. E. Oral cancer. Rijeka, Croatia: InTech Europe, 2012.

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3

W, Johnson Newell, and Batsakis John G, eds. Oral cancer. London: Martin Dunitz, 2003.

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4

Kirita, Tadaaki, and Ken Omura, eds. Oral Cancer. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-54938-3.

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5

1926-, Silverman Sol, and American Cancer Society, eds. Oral cancer. 3rd ed. Atlantic: American Cancer Society, 1990.

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6

Oral cancer metastasis. New York: Springer, 2010.

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7

Myers, Jeffrey, ed. Oral Cancer Metastasis. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-0775-2.

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8

M. Fribley, Andrew, ed. Targeting Oral Cancer. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-27647-2.

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9

Panta, Prashanth, ed. Oral Cancer Detection. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-61255-3.

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10

Appleton, Julia. Working with oral cancer. Bicester: Winslow, 1995.

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11

Al Moustafa, Ala-Eddin, ed. Development of Oral Cancer. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-48054-1.

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12

Pathak, K. Alok, and Richard W. Nason, eds. Controversies in Oral Cancer. New Delhi: Springer India, 2012. http://dx.doi.org/10.1007/978-81-322-2574-4.

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13

Warnakulasuriya, Saman, and John S. Greenspan, eds. Textbook of Oral Cancer. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-32316-5.

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14

Oral contraceptives & breast cancer. Washington, D.C: National Academy Press, 1991.

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15

Oral cancer: Causes, symptoms, signs, diagnosis, treatments, stages of oral cancer. United States: [CreateSpace Independent Publishing Platform], 2012.

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16

William, Carl, and Sako Kumao, eds. Cancer and the oral cavity. Chicago: Quintessence Pub. Co., 1986.

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17

Zaidi, Shabih H. A monograph on oral cancer. Karachi, Pakistan: Royal Book Co., 1995.

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18

Kademani, Deepak, ed. Improving Outcomes in Oral Cancer. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-30094-4.

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19

Oral cancers: Research report. [Bethesda, Md.?]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1991.

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20

Iyer, Subramania, and Sudhir Bahadur. Management of Oral Cancers. Springer Singapore Pte. Limited, 2021.

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21

Iyer, Subramania, and Sudhir Bahadur. Management of Oral Cancers. Springer Singapore Pte. Limited, 2020.

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22

Sundaresan, Sivapatham, Zahra Jassim Mohammed Al Timimi, Nihat Akbulut, and Neeti Kapre Gupta. Prevention of Oral Cancers. DI Press, 2022.

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23

Singh, Surya P., and Arja M. Kullaa. Diagnosis of Oral Cancers: An Optical Perspective. SPIE PRESS, 2016. http://dx.doi.org/10.1117/3.2235415.

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24

Hashibe, Mia, Erich M. Sturgis, Jacques Ferlay, and Deborah M. Winn. Oral Cavity, Oropharynx, Lip, and Salivary Glands. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0029.

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Анотація:

Cancers of the oral cavity, oropharynx, lip, and salivary glands are malignancies of the head and neck. Some of these cancer sites share risk factors, although each has distinctive anatomic, epidemiologic, and clinical features. Oral cavity cancers arise on the inner lip and buccal mucosa, anterior two-thirds of the tongue, gum, hard palate, and floor of mouth. These cancers are strongly associated with the use of smoked and smokeless tobacco products, heavy alcohol consumption, and chewing of betel quid or pan, but only minimally associated with prior infection with human papillomavirus (HPV). In contrast, oropharyngeal cancers affect the posterior one-third (base) of the tongue, tonsils, soft palate, and other oropharyngeal tissues and are strongly associated with HPV-16 infection as well as with the use of tobacco, alcohol, and betel quid. In principle, tumors of the oral cavity, oropharynx, and lip are among the most preventable forms of cancer.

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25

National Cancer Institute (U.S.), ed. What you need to know about oral cancers. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1989.

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26

Kayla, Bobby. Guidebook on Oral Cancers: The Most Effective Guide on Natural Solution to Cure Oral Cancers, Symptoms, Treatment, Health Tips and More. Independently Published, 2022.

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27

Rider, Jennifer R., Paul Brennan, and Pagona Lagiou. Oral and Pharyngeal Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0007.

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Анотація:

This chapter covers cancer of the oral cavity and the oropharynx, which includes the base of the tongue, soft palate, tonsils, and back and side walls of the throat. Many important risk factors for oral and oropharyngeal cancer have been identified, and in 2007 the World Health Organization determined there was sufficient evidence to include human papilloma virus (HPV) type 16 as a cause of these cancers. Tobacco and alcohol remain important modifiable risk factors, but the increasing incidence of HPV-associated tumors is now evident. While these tumors are more amenable to treatment than HPV-negative tumors, they are still a source of considerable morbidity and mortality. Moreover, the lack of a precursor lesion and limited data on efficacy of the HPV vaccine in preventing oral HPV infection are barriers to primary and secondary prevention efforts. Dietary patterns high in fruits and vegetables and low in meats may confer some protection.

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28

Cancers of the oral cavity and pharynx: A statistics review monograph 1973-1987. Atlanta, GA: National Center for Prevention Services, National Center for Chronic Disease Prevention and Health Promotion, Centers for Disease Control, Public Health Service, U.S. Dept. of Health and Human Services, 1992.

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29

V, Kleinman D., National Institutes of Health (U.S.), and Centers for Disease Control (U.S.), eds. Cancers of the oral cavity and pharynx: A statistics review monograph, 1973-1987. Atlanta, GA: U.S. DHHS, PHS, CDC, 1991.

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30

V, Kleinman D., National Institutes of Health (U.S.), and Centers for Disease Control (U.S.), eds. Cancers of the oral cavity and pharynx: A statistics review monograph, 1973-1987. Atlanta, GA: U.S. DHHS, PHS, CDC, 1991.

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31

V, Kleinman D., United States. Public Health Service., National Institutes of Health (U.S.), and Centers for Disease Control (U.S.), eds. Cancers of the oral cavity and pharynx: A statistics review monograph, 1973-1987. Atlanta, GA: U.S. DHHS, PHS, CDC, 1991.

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32

Tworoger, Shelley S., Amy L. Shafrir, and Susan E. Hankinson. Ovarian Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0046.

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Worldwide, ovarian cancer is the seventh most commonly diagnosed cancer and the eighth most common cause of death from cancer. In 2012, 239,000 women were diagnosed with ovarian cancer and 152,000 women died of the disease worldwide. In the United States in 2015, an estimated 21,290 women were newly diagnosed with ovarian cancer and 14,180 died from the disease. Both incidence and mortality have decreased over time in the United States, with a 1.6% and 2.1% annual decrease, respectively, from 2003 to 2012. Ovarian cancers can arise from epithelial, germ, or stromal cells, although about 90% are epithelial in origin. Risk factors best confirmed to increase risk of ovarian cancer include age and a family history of ovarian cancer, while parity, oral contraceptive use, and tubal ligation decrease risk. Several etiologic pathways, including hormonal and inflammatory pathways, have garnered substantial support from both epidemiologic and laboratory studies, although many questions remain.

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33

L, Sutton Amy, ed. Dental care and oral health sourcebook: Basic consumer health information about dental care, including oral hygiene, dental visits, pain management, cavities, crowns, bridges, dental implants, and fillings, and other oral health concerns, such as gum disease, bad breath, dry mouth, genetic and developmemtal abnormalities, oral cancers, orthodontics, and temporomandibular disorders ; along with updates on current research in oral health, a glossary, a directory of dental and oral health organizations, and resources for people with dental and oral health disorders. 2nd ed. Detroit, MI: Omnigraphics, 2003.

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34

1926-, Silverman Sol, ed. Oral cancer. 2nd ed. New York: American Cancer Society, 1985.

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35

Batsakis, John G., Newell W. Johnson, and Jatin P. Shah. Oral Cancer. Taylor & Francis Group, 2002.

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36

Shah, Jatin P., Newell W. Johnson, and John G. Batsakis, eds. Oral Cancer. CRC Press, 2002. http://dx.doi.org/10.1201/9780367804435.

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37

Shah, Jatin, Newell Johnson, and John Batsakis, eds. Oral Cancer. CRC Press, 2002. http://dx.doi.org/10.1201/b14725.

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38

1926-, Silverman Sol, and American Cancer Society, eds. Oral cancer. 4th ed. Hamilton, ON: B.C. Decker, 1998.

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39

E, Dawson C., ScottishForum for Public Health Medicine., Scottish Needs Assessment Programme, Special Needs Action Programme, and Health Promotion Network, eds. Oral cancer. Glasgow: Scottish Forum for Public Health Medicine, 1996.

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40

1926-, Silverman Sol, and American Cancer Society, eds. Oral cancer. 5th ed. Hamilton, ON: B.C. Decker, 2003.

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41

Ogbureke, Kalu U. E., ed. Oral Cancer. InTech, 2012. http://dx.doi.org/10.5772/1260.

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42

Gapstur, Susan M., and Philip John Brooks. Alcohol and Cancer Risk. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0012.

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In 2010, alcoholic beverage consumption caused an estimated 3.3 million deaths worldwide, and contributed to injuries, violence, liver cirrhosis, social disruption and at least seven different types of cancer. The International Agency for Research on Cancer (IARC) classifies exposure to both ethanol in alcoholic beverages and acetaldehyde, the primary metabolite of ethanol, as carcinogenic to humans (Group 1) based on “sufficient” evidence that alcoholic beverage consumption is causally related to cancers of the oral cavity, pharynx, larynx, esophagus, liver, colorectum and female breast. The biologic mechanisms by which alcohol and its primary metabolite acetaldehyde affect cancer risk appear to vary across anatomic sites. Broadly, these mechanisms involve DNA and protein damage from acetaldehyde and oxidative stress, nutritional malabsorption and metabolic effects, and for breast cancer, increased estrogen levels. The World Health Organization has increased global surveillance of alcohol consumption and encourages national efforts to apply evidence-based policies to reduce consumption.

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43

Crous-Bou, Marta, Immaculata de Vivo, and Pagona Lagiou. Endometrial Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0018.

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Lifestyle factors contribute appreciably to endometrial cancer risk, with obesity accounting for over one-third of incident cases in high-income societies. Unlike cervical cancer, which is a model of viral carcinogenesis, endometrial cancer is considered a model of hormonal carcinogenesis, as use of unopposed estrogens postmenopausally and obesity are the best-established risk factors. Endometrial cancer is also the only known malignancy for which cigarette smoking has been shown to confer protection. Risk reduction conferred by current smoking, past oral contraceptive use, childbearing, and physical activity is believed to be mediated by hormones. This may also apply to the increase in risk associated with obesity, which increases peripheral production of estrogens, and with diabetes mellitus. Hence, it should be possible to prevent a substantial fraction of endometrial cancers through lifestyle modification. Pathological classification of endometrial cancer is currently evolving and studies are revealing different molecular subtypes within the same histological groups.

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44

Yousefshahi, Fardin, Giuliano Michelagnoli, and Juan Francisco Asenjo. Ketamine Use and Opioid-Tolerant Cancer Patients. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190271787.003.0031.

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Pain occurs in up to 70% of cancer patients and it can be challenging to manage. The standard for analgesic therapy is the World Health Organization ladder; however, up to 25% of patients don’t reach a level of comfort using this approach. Ketamine has been recognized as an excellent adjuvant for cancer pain treatment, especially when other analgesics have failed. Some randomized clinical trials have confirmed ketamine’s efficacy in refractory cancer pain, but most had small sample sizes and low power. Some publications have confirmed the beneficial effect of oral, intranasal, subcutaneous, or intravenous ketamine in treatment of refractory chronic cancer pain, while others are less conclusive. While ketamine is rapidly gaining ground as an adjuvant in treating pain in patients with cancers refractory to conventional therapy and/or patients with opioid tolerance, care should be taken to identify patients with ketamine contraindications in order to offer the greatest benefit with the lowest risk of side effects.

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45

Ye, Weimin, Olof Nyrén, and Hans-Olov Adami. Stomach Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0010.

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The epidemiology of stomach cancer is characterized by its wide variation of incidence by geography and population. Despite a declining secular trend of incidence in most Western populations, it is still one of the most common cancers worldwide. Helicobacter pylori infection is the strongest and most important risk factor known today. Although eradication of H. pylori might be an efficient primary prevention strategy, a deeper understanding of effect-modifying factors, including bacterial genetics, human genetics, and environmental risk factors, may enable us to focus even more precisely on the relevant high-risk groups. Dietary factors, particularly the low intake of antioxidant-rich fruits and vegetables and high intake of salt, are likely to be important. The roles of microbes other than H. pylori in the oral cavity and stomach have recently gained increasing attention. A subtype of stomach cancer, cardia cancer, appears to have a different epidemiology and risk factor profile.

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46

Castle, David J., Peter F. Buckley, and Fiona P. Gaughran. Other physical health problems in people with schizophrenia. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198811688.003.0004.

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While cardiovascular risk remains the most important factor in early death among people with schizophrenia, a host of other physical health maladies are also found in excess in this group of individuals. These include pulmonary problems, poor bone health with associated risk of fractures, sexual health problems, infectious diseases, and poor oral health. Certain cancers are seen in excess in people with schizophrenia, but what is perhaps more of a shameful indictment of our health systems is that if they develop cancer, they are less likely to be effectively treated than people without a mental illness. Intriguingly, there is some evidence of higher pain tolerance among people with schizophrenia, as well as remarkably low rates of degenerative musculoskeletal conditions.

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47

Nicholas, Stafford, and Waldron John, eds. Managementof oral cancer. Oxford: Oxford University Press, 1989.

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48

Myers, Jeffrey. Oral Cancer Metastasis. Springer, 2014.

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49

Santoro, A., Walter Giaretti, and Dziegiel Piotr. Oral Cancer 3. DI Press, 2022.

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50

Santoro, A., Walter Giaretti, Dziegiel Piotr, and Takashi Muramatsu. Oral Cancer 3. DI Press, 2022.

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