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Добірка наукової літератури з теми "OCCLUSIONE PORTALE"

Автор: Grafiati

Опубліковано: 11 березня 2023

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Статті в журналах з теми "OCCLUSIONE PORTALE"

Bridwell, Rachel E., Sean Clerkin, Nathaniel R. Walker, Brit Long, and Sarah Goss. "Portal Venous Thrombosis in a Special Operations Paratrooper: A Case Report." Military Medicine 187, no. 1-2 (September 30, 2021): 256–58. http://dx.doi.org/10.1093/milmed/usab387.

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ABSTRACT Portal vein thrombosis is the thrombotic occlusion of the extrahepatic portal system, which can propagate towards the vena caval system. Although rare, it occurs primarily in those with cirrhosis, intra-abdominal infections, malignancy, or hypercoagulable disorders. This report describes the first reported case of a soldier within special operations without identifiable risk factors who was found to have a completely occlusive portal vein thrombosis after approximately 10 days of insidious abdominal pain. This case emphasizes the importance of considering this rare but dangerous pathology among this highly screened and capable special operations population.

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Iqbal, Nabeela, Syed Khalid Shah, and Shamima Haneef. "Polycythemia Vera Complicated by Portal Vein Thrombosis and Budd-Chiari Syndrome:." Journal of Bahria University Medical and Dental College 10, no. 02 (March 18, 2021): 163–65. http://dx.doi.org/10.51985/jbumdc2020019.

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Polycythemia vera is a medical condition characterized by raised hematocrit. Owing to increased viscosity, the blood flow in the vessels become sluggish leading to the clinical features of polycythemia such as headache, blurring of vision, red skin, dizziness, raised blood pressure, itching and more serious medical events like vaso occlusion, thrombosis and strokes. In this case report, polycythemia vera presenting unusually with heamatemesis, melena and abdominal distension. Physical examination of this case revealed massive ascites with dilated veins around the umbilicus. The diagnosis of polycythemia vera complicated by Budd Chiari Syndrome and Portal Vein Thrombosis was made. Patients with polycythemia vera are at risk of vaso occlusive sequelea like portal vein thrombosis and Budd chiari syndrome

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Strbe, Sanja, Slaven Gojkovic, Ivan Krezic, Helena Zizek, Hrvoje Vranes, Ivan Barisic, Dean Strinic, et al. "Over-Dose Lithium Toxicity as an Occlusive-like Syndrome in Rats and Gastric Pentadecapeptide BPC 157." Biomedicines 9, no. 11 (October 20, 2021): 1506. http://dx.doi.org/10.3390/biomedicines9111506.

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Due to endothelial impairment, high-dose lithium may produce an occlusive-like syndrome, comparable to permanent occlusion of major vessel-induced syndromes in rats; intracranial, portal, and caval hypertension, and aortal hypotension; multi-organ dysfunction syndrome; brain, heart, lung, liver, kidney, and gastrointestinal lesions; arterial and venous thrombosis; and tissue oxidative stress. Stable gastric pentadecapeptide BPC 157 may be a means of therapy via activating loops (bypassing vessel occlusion) and counteracting major occlusion syndromes. Recently, BPC 157 counteracted the lithium sulfate regimen in rats (500 mg/kg/day, ip, for 3 days, with assessment at 210 min after each administration of lithium) and its severe syndrome (muscular weakness and prostration, reduced muscle fibers, myocardial infarction, and edema of various brain areas). Subsequently, BPC 157 also counteracted the lithium-induced occlusive-like syndrome; rapidly counteracted brain swelling and intracranial (superior sagittal sinus) hypertension, portal hypertension, and aortal hypotension, which otherwise would persist; counteracted vessel failure; abrogated congestion of the inferior caval and superior mesenteric veins; reversed azygos vein failure; and mitigated thrombosis (superior mesenteric vein and artery), congestion of the stomach, and major hemorrhagic lesions. Both regimens of BPC 157 administration also counteracted the previously described muscular weakness and prostration (as shown in microscopic and ECG recordings), myocardial congestion and infarction, in addition to edema and lesions in various brain areas; marked dilatation and central venous congestion in the liver; large areas of congestion and hemorrhage in the lung; and degeneration of proximal and distal tubules with cytoplasmic vacuolization in the kidney, attenuating oxidative stress. Thus, BPC 157 therapy overwhelmed high-dose lithium intoxication in rats.

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Wang, L. Q., B. G. Persson, and S. Bengmark. "The Influence of Portal Deviation on the Effect of Repeat Dearterializations of a Transplantable Adenocarcinoma to the Rat Liver." HPB Surgery 8, no. 1 (January 1, 1994): 37–41. http://dx.doi.org/10.1155/1994/68212.

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As liver tumours receive some of their blood supply from the portal vein, we wanted to illustrate the influence of portal blood flow in combination with dearterialization in the treatment of liver tumours. Forty male, inbred Wistar/Furth rats with an adenocarcinoma transplanted to the liver were treated with various inflow occlusions repeated daily for 5 days. Deviation of the portal blood flow alone with an end-side porto-caval shunt did not alter the tumour growth (p = 0.089). Thirty min of repeat dearterializations was potentiated by portal deviation so that tumour growth was delayed (p = 0.004). However, repeat dearterializations for 60 min in portal deviated rats induced irreversible liver damage and all rats died in a few days. Repeated dearterializations for 60 minutes alone retarded the tumour growth as efficiently (p = 0.007). Simultaneous occlusion of the hepatic artery and the portal vein for 30 minutes with a side-side porto-caval shunted (total devascularization) did not affect tumour growth (p = 0.154). Liver aminotransferases (ASAT and ALAT) were substantially increased following dearterialization for 30 min in rats with either an end-side or a side-side porto-caval shunt. Dearterialization for 60 min in rats with end-side porto-caval shunts gave a further release of ASAT and ALAT.In conclusion, portal deviation did not augment the therapeutic benefit of repeat dearterializations for the treatment of this experimental liver tumour. Repeat dearterializations alone seemed to be a feasible and efficient therapy for liver tumours.

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Marn, C. S., and I. R. Francis. "CT of portal venous occlusion." American Journal of Roentgenology 159, no. 4 (October 1992): 717–26. http://dx.doi.org/10.2214/ajr.159.4.1326882.

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Shibamoto, Toshishige, Sen Cui, Zonghai Ruan, Wei Liu, Hiromichi Takano, and Yasutaka Kurata. "Hepatic venoconstriction is involved in anaphylactic hypotension in rats." American Journal of Physiology-Heart and Circulatory Physiology 289, no. 4 (October 2005): H1436—H1441. http://dx.doi.org/10.1152/ajpheart.00368.2005.

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We determined the roles of liver and splanchnic vascular bed in anaphylactic hypotension in anesthetized rats and the effects of anaphylaxis on hepatic vascular resistances and liver weight in isolated perfused rat livers. In anesthetized rats sensitized with ovalbumin (1 mg), an intravenous injection of 0.6 mg ovalbumin caused not only a decrease in systemic arterial pressure from 120 ± 9 to 43 ± 10 mmHg but also an increase in portal venous pressure that persisted for 20 min after the antigen injection (the portal hypertension phase). The elimination of the splanchnic vascular beds, by the occlusions of the celiac and mesenteric arteries, combined with total hepatectomy attenuated anaphylactic hypotension during the portal hypertension phase. For the isolated perfused rat liver experiment, the livers derived from sensitized rats were hemoperfused via the portal vein at a constant flow. Using the double-occlusion technique to estimate the hepatic sinusoidal pressure, presinusoidal ( Rpre) and postsinusoidal ( Rpost) resistances were calculated. An injection of antigen (0.015 mg) caused venoconstriction characterized by an almost selective increase in Rpre rather than Rpost and liver weight loss. Taken together, these results suggest that liver and splanchnic vascular beds are involved in anaphylactic hypotension presumably because of anaphylactic presinusoidal contraction-induced portal hypertension, which induced splanchnic congestion resulting in a decrease in circulating blood volume and thus systemic arterial hypotension.

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Terada, N., S. Koyama, J. Horiuchi, and T. Takeuchi. "Participation of adrenoceptors in liver blood flow regulation in anesthetized dogs." American Journal of Physiology-Heart and Circulatory Physiology 253, no. 5 (November 1, 1987): H1053—H1058. http://dx.doi.org/10.1152/ajpheart.1987.253.5.h1053.

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We evaluated involvement of adrenergic receptors in the responses of the hepatic vasculature to reduction either of portal venous flow or hepatic arterial inflow. Portal vein occlusion caused an increase in hepatic arterial blood flow (HAF) and decreases in hepatic arterial pressure (HAP) and hepatic arterial vascular resistance (HAR) in the intact group. After pretreatment with either yohimbine or prazosin, but not propranolol, occlusion of the portal vein produced a greater decrease in HAP as compared with that in the intact group. No significant changes in HAF, HAR, or hepatic tissue blood flow (HTF) occurred after the treatment. These results indicate that the compensatory response of the hepatic arterial vasculature to altered portal blood flow (PVF) is regulated independently of the intrahepatic adrenergic receptors. Hepatic arterial occlusion caused a significant decrease in portal venous pressure, PVF, and HTF. Portal venous vascular resistance (PVR) was reduced slightly, but not significantly. After pretreatment with either yohimbine or prazosin, but not propranolol, occlusion of the hepatic artery produced an opposite effect: to increase PVF and significantly decrease PVR. These results indicate that intrahepatic alpha-adrenoceptors participate in the regulation of portal vascular tone to maintain portal vein pressure at a steady level, when inflow from the hepatic artery is reduced.

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Kamikado, Chiaki, Toshishige Shibamoto, Minoru Hongo, and Shozo Koyama. "Effects of Hct and norepinephrine on segmental vascular resistance distribution in isolated perfused rat livers." American Journal of Physiology-Heart and Circulatory Physiology 286, no. 1 (January 2004): H121—H130. http://dx.doi.org/10.1152/ajpheart.01136.2002.

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We studied the effects of blood hematocrit (Hct), blood flow, or norepinephrine on segmental vascular resistances in isolated portally perfused rat livers. Total portal hepatic venous resistance ( Rt) was assigned to the portal ( Rpv), sinusoidal ( Rsinus), and hepatic venous ( Rhv) resistances using the portal occlusion (Ppo) and the hepatic venous occlusion (Phvo) pressures that were obtained during occlusion of the respective line. Four levels of Hct (30%, 20%, 10%, and 0%) were studied. Rpv comprises 44% of Rt, 37% of Rsinus, and 19% of Rhv in livers perfused at 30% Hct and portal venous pressure of 9.1 cmH2O. As Hct increased at a given blood flow, all three segmental vascular resistances of Rpv, Rsinus, and Rhv increased at flow >15 ml/min. As blood flow increased at a given Hct, only Rsinus increased without changes in Rpv or Rhv. Norepinephrine increased predominantly Rpv, and, to a smaller extent, Rsinus, but it did not affect Rhv. Finally, we estimated Ppo and Phvo from the double occlusion maneuver, which occluded simultaneously both the portal and hepatic venous lines. The regression line analysis revealed that Ppo and Phvo were identical with those measured by double occlusion. In conclusion, changes in blood Hct affect all three segmental vascular resistances, whereas changes in blood flow affect Rsinus, but not Rpv or Rhv. Norepinephrine increases mainly presinusoidal resistance. Ppo and Phvo can be obtained by the double occlusion method in isolated perfused rat livers.

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Bayraktar, Yusuf, Abdurrahman Kadayifci, Ferhun Balkanci, Burhan Kayhan, David H. Van Thiel, and Sevket Ruacan. "Splenic Artery Occlusion Masking Portal Hypertension." Journal of Clinical Gastroenterology 22, no. 4 (June 1996): 326–28. http://dx.doi.org/10.1097/00004836-199606000-00021.

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Cheng, Yu Fan, Chien Fu Hung, Yang Han Liu, Koon Kwan Ng, and Chung Chueng Tsai. "Hepatic actinomycosis with portal vein occlusion." Gastrointestinal Radiology 14, no. 1 (December 1989): 268–70. http://dx.doi.org/10.1007/bf01889213.

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Дисертації з теми "OCCLUSIONE PORTALE"

Bretagnol, Frédéric. "Les métastases hépatiques expérimentales d'origine colorectale : détection précoce et croissance tumorale après occlusion portale." Paris 6, 2010. http://www.theses.fr/2010PA066120.

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Un tiers des patients ayant un cancer colorectal développent des métastases hépatiques (MH) qui ne sont résécables que dans 30% des cas. L’occlusion portale induisant une hypertrophie hépatique permet cette résécabilité. Une récidive survient dans 30% des cas et suggère la présence de cellules "dormantes". La détection des micrométastases n’est pas possible par imagerie standard. Or, il a été montré que les MH étaient associées à des modifications de la perfusion hépatique. Par ailleurs, l’occlusion portale pourrait stimuler non seulement la régénération hépatique mais aussi la croissance tumorale. Le but était avec un modèle expérimental de MH chez le rat, d’évaluer la détection des micrométastases par scanner de perfusion hépatique et d’étudier la croissance tumorale des MH après occlusion portale. Méthodes. Evaluer dans un modèle de MH par injection intraportale de cellules DHDK12 chez le rat, la détection des micrométastases par scanner de perfusion et étudier la croissance tumorale des MH après occlusion portale. Résultats. Des modifications significatives de la perfusion hépatique ont été détectées par scanner de perfusion pour des MH <1,5 mm. Après ligature portale, le volume des micrométastases augmentait dans le foie ligaturé et celui des macrométastases augmentait dans le foie restant. Nous avons montré une diminution de la croissance des macrométastases au niveau du foie embolisé. Cette étude confirme l’intérêt de l’indice de perfusion comme facteur pronostique du risque de développer des MH en permettant de mieux sélectionner les patients "à risque". La stimulation de la croissance tumorale après occlusion portale justifie une sélection rigoureuse des patients

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IWASE, MASANORI. "The Influence of Portal Vein Occlusion on Liver Mitochondria in Rats after Releasing Biliary Obstruction." Nagoya University School of Medicine, 1986. http://hdl.handle.net/2237/17487.

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KRIEF, YANN. "Maladie veino-occlusive pulmonaire : a propos d'un cas, revue de la litterature portant sur 50 observations." Nantes, 1990. http://www.theses.fr/1990NANT145M.

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Petruželka, Jiří. "Výpočet viditelnosti v 3D bludišti." Master's thesis, Vysoké učení technické v Brně. Fakulta informačních technologií, 2014. http://www.nusl.cz/ntk/nusl-236125.

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Hallot, Véronique. "Invagination intestinale aigüe du nourrisson et de l'enfant : étude rétrospective portant sur 83 dossiers pendant une période de vingt ans à l'Hôpital d'Enfants de Saint-Denis de la Réunion." Caen, 1994. http://www.theses.fr/1994CAEN3042.

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Brohan, Kevin Patrick. "Search and attention for machine vision." Thesis, University of Manchester, 2012. https://www.research.manchester.ac.uk/portal/en/theses/search-and-attention-for-machine-vision(a4747c9b-ac13-46d1-8895-5f2d88523d80).html.

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This thesis addresses the generation of behaviourally useful, robust representations of the sensory world in the context of machine vision and behaviour. The goals of the work presented in this thesis are to investigate strategies for representing the visual world in a way which is behaviourally useful, to investigate the use of a neurally inspired early perceptual organisation system upon high-level processing in an object recognition system and to investigate the use of a perceptual organisation system on driving an object-based selection process. To address these problems, a biologically inspired framework for machine attention has been developed at a high level of neural abstraction, which has been heavily inspired by the psychological and physiological literature. The framework is described in this thesis, and three system implementations, which investigate the above issues, are described and analysed in detail. The primate brain has access to a coherent representation of the external world, which appears as objects at different spatial locations. It is through these representations that appropriate behavioural responses may be generated. For example, we do not become confused by cluttered scenes or by occluded objects. The representation of the visual scene is generated in a hierarchical computing structure in the primate brain: while shape and position information are able to drive attentional selection rapidly, high-level processes such as object recognition must be performed serially, passing through an attentional bottleneck. Through the process of attentional selection, the primate visual system identifies behaviourally relevant regions of the visual scene, which allows it to prioritise serial attentional shifts towards certain locations. In primates, the process of attentional selection is complex, operating upon surface representations which are robust to occlusion. Attention itself suppresses neural activity related to distractor objects, while sustaining activity relating to the target, allowing the target object to have a clear neural representation upon which the recognition process can operate. This thesis concludes that dynamic representations that are both early and robust against occlusion have the potential to be highly useful in machine vision and behaviour applications.

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Burrows, Fiona. "Investigation of haemodynamic changes and pathophysiology in a remote filament model of stroke." Thesis, University of Manchester, 2014. https://www.research.manchester.ac.uk/portal/en/theses/investigation-of-haemodynamic-changes-and-pathophysiology-in-a-remote-filament-model-of-stroke(e5f65ed8-d665-4787-a12b-775604c3600d).html.

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The initial hours following an ischaemic event in the brain represent a critically important window in which therapeutic interventions to reduce neuronal damage and improve patient outcome can be made. Nevertheless, the dynamics of cerebral blood flow and oxygenation, as well as the local physiological changes, in the first few hours after reperfusion following ischaemic stroke are not well understood. In the first study, a remote filament approach was used to obtain multispectral imaging data before, during and after middle cerebral artery occlusion to investigate early changes in haemodynamic concentration of oxy-/deoxy-haemoglobin and total blood volume, in anaesthetised mice. We use immunohistochemistry to establish the extent of cortical injury and correlate the severity of damage with the change of oxygen perfusion during and after the ischaemic event. Increased numbers of platelets and activated microglia, expression of interleukin-1α, evidence of BBB breakdown and neuronal stress are all seen within the stroked hemisphere of MCAo mice and correlate with the severity of oxy-haemoglobin concentration deficit at experimental but not with the change in oxy-haemoglobin concentration during the acute stroke. In the second study, we used the same remote filament and optical imaging approach to investigate the effects of acute systemic inflammation on haemodynamics pre, during, and after induced cerebral ischaemia. We found that an acute systemic inflammatory challenge exacerbates oxy-haemoglobin deficit after 3 h of reperfusion following an ischaemic event. We investigated known pathophysiological markers to elucidate potential mechanisms that may contribute to this exacerbated oxygenation deficit and found hyper-coagulated platelets within the large and microvessels of the ipsilateral cortex. Our findings demonstrate that despite initial restoration of HbO2 supply after 30 min MCAo there is a delayed compromise that coincides with inflammatory processes that could be a future target for improved stroke outcome after thrombolysis. We also show that acute systemic inflammation exacerbates this oxy-haemoglobin deficit after an ischaemic challenge and increases pathophysiology.

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Haumont, Dominique. "Calcul et représentation de l'information de visibilité pour l'exploration interactive de scènes tridimensionnelles." Doctoral thesis, Universite Libre de Bruxelles, 2006. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210880.

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La synthèse d'images, qui consiste à développer des algorithmes pour générer des images à l'aide d'un ordinateur, est devenue incontournable dans de nombreuses disciplines.

Les méthodes d'affichage interactives permettent à l'utilisateur d'explorer des environnements virtuels en réalisant l'affichage des images à une cadence suffisamment élevée pour donner une impression de continuité et d'immersion. Malgré les progrès réalisés par le matériel, de nouveaux besoins supplantent toujours les capacités de traitement, et des techniques d'accélération sont nécessaires pour parvenir à maintenir une cadence d'affichage suffisante. Ce travail s'inscrit précisemment dans ce cadre. Il est consacré à la problématique de l'élimination efficace des objets masqués, en vue d'accélérer l'affichage de scènes complexes. Nous nous sommes plus particulièrement intéressé aux méthodes de précalcul, qui effectuent les calculs coûteux de visibilité durant une phase de prétraitement et les réutilisent lors de la phase de navigation interactive. Les méthodes permettant un précalcul complet et exact sont encore hors de portée à l'heure actuelle, c'est pourquoi des techniques approchées leur sont préférée en pratique. Nous proposons trois méthodes de ce type.

La première, présentée dans le chapitre 4, est un algorithme permettant de déterminer de manière exacte si deux polygones convexes sont mutuellement visibles, lorsque des écrans sont placés entre eux. Nos contributions principales ont été de simplifier cette requête, tant du point de vue théorique que du point de vue de l'implémentation, ainsi que d'accélérer son temps moyen d'exécution à l'aide d'un ensemble de techniques d'optimisation. Il en résulte un algorithme considérablement plus simple à mettre en oeuvre que les algorithmes exacts existant dans la littérature. Nous montrons qu'il est également beaucoup plus efficace que ces derniers en termes de temps de calcul.

La seconde méthode, présentée dans le chapitre 5, est une approche originale pour encoder l'information de visibilité, qui consiste à stocker l'ombre que générerait chaque objet de la scène s'il était remplacé par une source lumineuse. Nous présentons une analyse des avantages et des inconvénients de cette nouvelle représentation.

Finalement, nous proposons dans le chapitre 6 une méthode de calcul de visibilité adaptée aux scènes d'intérieur. Dans ce type d'environnements, les graphes cellules-portails sont très répandus pour l'élimination des objets masqués, en raison de leur faible coût mémoire et de leur grande efficacité. Nous reformulons le problème de la génération de ces graphes en termes de segmentation d'images, et adaptons un algorithme classique, appelé «watershed», pour les obtenir de manière automatique. Nous montrons que la décomposition calculée de la sorte est proche de la décomposition classique, et qu'elle peut être utilisée pour l'élimination des objets masqués.

Doctorat en sciences appliquées
info:eu-repo/semantics/nonPublished

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RUSSOLILLO, NADIA. "ALCOLIZZAZIONE ASSOCIATA ALLA LEGATURA PORTALE: UNA TECNICA EFFICACE PER INCREMENTARE LA RIGENERAZIONE EPATICA." Doctoral thesis, 2016. http://hdl.handle.net/11562/939243.

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INTRODUZIONE E OBIETTIVO: L’occlusione della vena porta mediante legatura (PVL) o embolizzazione (PVE) è utilizzata nella pratica clinica per favorire la rigenerazione epatica nei pazienti con volume di fegato residuo (FLRV) all’intervento programmato inadeguato. Alcuni Autori hanno descritto un più elevato tasso di rigenerazione epatica nei pazienti sottoposti a PVE rispetto alla PVL, come conseguenza della formazione in questi ultimi di collaterali porto-portali tra il segmento 4 e i segmenti 5 e 8. L’obiettivo dello studio era di paragonare il tasso di rigenerazione epatica dopo legatura portale con (gruppo Alc+) e senza (gruppo Alc-) la simultanea iniezione intra-portale di alcool. PAZIENTI E METODI: 42 pazienti affetti da metastasi colorettali sottoposti tra 01/2004 e 06/2014 a PVL preoperatoria sono stati analizzati. A partire dal 09/2011 la legatura portale destra è stata preceduta dall’iniezione intraportale di alcool puro. IL FLRV è stato calcolato sulla base degli esami TC. Due radiologi hanno rivisto le immagini delle TC post-PVL al fine di identificare la presenza di cavernomi e ricanalizzazioni del flusso nel ramo portale occluso. La rigenerazione epatica è stata valutata in termini di Incremento Volumetrico (VI) calcolato con la seguente formula: [(FLRVpost PVL% – FLRVpre PVL%)/FLRVpre PVL%]. RISULTATI: I pazienti del gruppo Alc+ (n=23) e Alc- (n=19) erano simili in termini di età, sesso, diabete, FLRV pre-PVL e per la somministrazione di chemioterapia. Il tasso di ricanalizzazione portale (63.1% vs. 4.3%, p<0.001) e la presenza di cavernomi (36.8% vs. 8.7%, p=0.055) dopo PVL erano significativamente superiori nei pazienti del gruppo Alc-. Il FLRV post-PVL (43.3±14.3% vs. 34.6±6.4%, p=0.013) e l’incremento volumetrico VI (0.44±0.24 vs. 0.28±0.20, p=0.029) erano significativamente superiori nel gruppo Alc+. All’analisi univariata il sesso maschile (0.23±024 vs.0.40±0.19, p=0.005) e la PVL senza alcolizzazione (0.35±0.24 vs. 0.26±0.20, p=0.035) sono risultati negativamente correlati con il valore di VI. L’analisi multivariata ha mostrato che il sesso maschile (B=−0.149, p=0.035) e l’iniezione intraportale di alcool (B=0.143 p=0.041) correlavano significativamente con il valore di VI [F(1,40)=5.200, p=0.010]. CONCLUSIONI: L’iniezione di alcool prima della legatura portale incrementa significativamente la rigenerazione del FLRV, riducendo la ricanalizzazione del flusso nel ramo portale occluso
BACKGROUND AND AIM: Portal vein occlusion by ligation (PVL) or embolization (PVE) is routinely performed to increase inadequate future liver remnant volume (FLRV) . The higher liver regeneration rate observed after PVE than after PVL may be due to the formation in the latter of intrahepatic porto-portal collaterals between the portal branches of segment 4 and the branches of the adjacent right segments 5 and 8. The aim of the study was to compare liver regeneration rate following portal vein ligation (PVL) with (Alc+) and without (Alc-) simultaneous intraportal alcohol injection METHODS: Forty-two patients with colorectal liver metastases who underwent PVL between January 2004 and June 2014 were analyzed. Beginning in September 2011, absolute alcohol was injected prior to right PVL. Future liver remnant volume (FLRV) was assessed by CT-scan. CT-scans were reviewed to assess recanalization and/or cavernous transformation of the occluded portal vein. Liver regeneration was assessed as Volumetric Increase (VI) [(FLRVpost% – FLRVpre%)/FLRVpre%]. RESULTS: The Alc+ (n=23) and Alc- (n=19) groups were similar in age, sex, diabetes, pre-PVL FLRV and administration of chemotherapy. The rate of recanalization of the occluded portal vein was significantly higher (63.1% vs. 4.3%, p<0.001) and the rate of cavernous transformation higher (36.8% vs. 8.7%, p=0.055) in the Alc- than in the Alc+ group. Post-PVL FLRV (43.3±14.3% vs. 34.6±6.4%, p=0.013) and VI (0.44±0.24 vs. 0.28±0.20, p=0.029) were significantly higher in the Alc+ group. Univariate analysis showed that male sex (0.23±024 vs.0.40±0.19, p=0.005) and PVL without alcohol injection (0.35±0.24 vs. 0.26±0.20, p=0.035) were negatively correlated with VI. Multiple regression analysis showed that male sex (B=−0.149, p=0.035) and alcohol injection (B=0.143 p=0.041) significantly predicted VI [F(1,40)=5.200, p=0.010]. CONCLUSIONS: Alcohol injection prior to PVL significantly increased regeneration rate of the future liver remnant, reducing recanalization of the occluded portal vein.

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Ying-JrLin and 林盈志. "Development of a portable Near-infrared system for evaluation of peripheral arterial occlusive disease." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/51593291022016907057.

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碩士
國立成功大學
醫學工程研究所碩博士班
98
Peripheral artery occlusive disease (PAOD) has been a common symptom in many diseases and its risk increases sharply with age. Therefore detecting lower limb PAOD is important for patients to prevent disabling claudication, ischaemic rest pain and gangrene. The purpose of this research is to develop a portable and non-invasive microcirculation measurement system using the photo-electronic technique based on photoplethesmography (PPG) and arterial oxyhemoglobin saturation (SaO2) to evaluate the microcirculation state of PAOD patients. This device provides a help for diagnosis of peripheral artery abnormalities and a tool for health home-care. This device was mainly divided into two steps. First, the three channels PPG and one channel ECG was set up for measuring physiological parameters such as PTTf, PTTp, RT, and SaO2. Secondary, we evaluated the microcirculation by those parameters in different measuring point. The statistical results show that the significant differences between the bilateral sites (ΔPTTp andΔPTTf) are useful parameters for evaluating microcirculation in PAOD patients. Those results also indicated that the laser-based microcirculation measurement system in this study could be a tool for diagnosis or healthy home-care to determine the artery circulation state of PAOD patients.

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Книги з теми "OCCLUSIONE PORTALE"

Masrani, Abdulrahman, and Bulent Arslan. Deployment of Direct Intrahepatic Portocaval Shunt (DIPS) from a Femoral Access. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0078.

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The transjugular intrahepatic portosystemic shunt (TIPS) has been shown to be effective in management of esophageal varices bleeding in patients with liver cirrhosis when endoscopic manuvers fail to control it. Ascites refractory to optimal medical therapy is another indication for TIPS procedure. Occasionally, TIPS cannot be performed due to vascular anatomical difficulties such as occluded central venous access, small hepatic veins, or portal vein occlusion. Direct intrahepatic portocaval shunt (DIPS) can be considered as an alternative option in such circumstances. DIPS is typically performed utilizing jugular access with direct puncture from the inferior vena cava (IVC) to the right portal vein. However, the interventionalist may be challenged by jugular or brachiocephalic veins occlusion. This chapter discusses perfroming DIPS procedure utilizing femoral access in a patient with bilateral occluded brachiocephalic veins and thrombosed right portal vein.

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McCabe, Sam, Christopher Harnain, and Grigory Rozenblit. Use of an Elongated Radiopaque Gelatin Sponge Plug for Tract Occlusion After Hepatic Interventions. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0088.

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This chapter describes the use of an elongated radiopaque gelatin sponge plug for tract occlusion after percutaneous biliary, portal venous, or hepatic venous access or intervention. Often, embolization coils or a gelatin sponge “slurry” is sufficient for hemostasis following liver intervention. This technique offers an inexpensive, temporary, and readily available option for achieving hemostasis following liver interventions. A contrast-soaked Gelfoam plug is loaded into a delivery cylinder that is advanced into the access sheath and positioned and deployed under fluoroscopic guidance. This technique is an alternative option to the use of embolization coils or a gelatin sponge slurry when seeking hepatic parenchymal tract hemostasis following intervention.

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Ferral, Hector. Optimal Imaging Techniques of the Portal Vasculature During TIPS Creation: Use of the CO2 Portogram. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0074.

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Transjugular intrahepatic portosystemic shunt creation is one of the most complex interventional procedures. It requires skills in imaging, vessel catheterization, guidewire techniques, balloon angioplasty, endovascular stent deployment, and embolization techniques. The key step during this procedure is obtaining access into the portal vein. This chapter discusses how to perform a CO2 portogram to obtain access to the portal vein during the creation of a transjugular intrahepatic portosystemic shunt (TIPS). The CO2 portogram may be performed with an angiographic catheter wedged in the hepatic vein, with an occlusion balloon inflated within the hepatic vein, or with the transhepatic needle within the liver parenchyma—the so-called “intraparenchymal” injection. Those performing a CO2 portogram should have knowledge of CO2 and a user-friendly CO2 delivery system to avoid common CO2 complications.

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Частини книг з теми "OCCLUSIONE PORTALE"

Linecker, Michael, Henrik Petrowsky, and Pierre-Alain Clavien. "Two-Stage Liver Surgery with Portal Vein Occlusion." In Extreme Hepatic Surgery and Other Strategies, 217–30. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-13896-1_14.

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Lu, W. C., S. H. Lu, M. F. Chen, T. C. Fu, K. P. Lin, and C. L. Tsai. "Portable Near-Infrared Spectroscopy for Detecting Peripheral Arterial Occlusion." In Precision Medicine Powered by pHealth and Connected Health, 109–13. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-7419-6_19.

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Kobayashi, Kosuke, Nicolas Villard, Alban Denys, and Emmanuel Melloul. "Portal Vein Embolization with and without Hepatic Vein Occlusion." In Colorectal Liver Metastasis, 371–76. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-09323-4_40.

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Kubo, Shoji, Hiroaki Kinoshita, Kazuhiro Hirohashi, Nagahisa Fujio, Ryutaro Iwasa, and Hiroki Nakamura. "Combination of Hepatic Arterial Infusion Chemotherapy and Portal Vein Occlusion for Unresectable Hepatocellular Carcinoma." In Recent Advances in Management of Digestive Cancers, 678–80. Tokyo: Springer Japan, 1993. http://dx.doi.org/10.1007/978-4-431-68252-3_202.

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"Portal Vein Occlusion." In Diagnostic Imaging: Gastrointestinal, 700–705. Elsevier, 2015. http://dx.doi.org/10.1016/b978-0-323-37755-3.50214-2.

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"Portal Vein Occlusion." In Diagnostic Ultrasound: Abdomen and Pelvis, 264–67. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-323-37643-3.50048-7.

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"Portal Vein Occlusion." In Imaging in Gastroenterology, 330–33. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-323-55408-4.50165-8.

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Lee, Christine U., and James F. Glockner. "Case 2.16." In Mayo Clinic Body MRI Case Review, edited by Christine U. Lee and James F. Glockner, 92–93. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199915705.003.0047.

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83-year-old man being evaluated in the emergency department for abdominal pain Axial postgadolinium portal venous phase images (Figure 2.16.1) demonstrate nearly occlusive thrombus throughout the main portal vein. Reformatted oblique images (Figure 2.16.2) show thrombus extension into the right and left portal veins and the superior mesenteric vein....

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Fei, Yanxia, Yanhua Peng, Huiping Sun, Shuangfa Zou, and Jinfeng Yang. "Sinusoidal Obstruction Syndrome." In Portal Hypertension - Recent Advances. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96370.

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Sinusoidal obstructive syndrome (SOS) is a fibrous occlusive disease of hepatic sinusoids or hepatic venules. Small hepatic blood vessel damage, especially hepatic sinusoidal endothelial cell damage, is its main feature. Based on etiology, SOS is mainly classified into pyrrolidine alkaloids-related SOS, hematopoietic stem cell transplantation-related SOS, and SOS of unknown etiology. In recent years, the incidence of SOS has been increasing. However, due to the complexity of the etiology, the lack of specificity in clinical manifestations, the difficulty of early diagnosis, and the limited treatment options, it often leads to poor treatment effects and even death. This chapter aims to analyze and organize the pathogenesis, pathological characteristics, diagnosis, treatment, and prognosis of different types of SOS, to provide certain references for the prevention and treatment of the disease.

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Becker, Richard C., and Frederick A. Spencer. "Aspirin." In Fibrinolytic and Antithrombotic Therapy. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195155648.003.0012.

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Aspirin, considered the prototypic platelet antagonist, has been available for over a century and currently represents a mainstay both in the prevention and treatment of vascular events that include stroke, myocardial infarction, peripheral vascular occlusion, and sudden death. Aspirin irreversibly acetylates cyclooxygenase (COX), impairing prostaglandin metabolism and thromboxane A2 (TXA2) synthesis. As a result, platelet aggregation in response to collagen, adenosine diphosphate (ADP), and thrombin (in low concentrations) is attenuated (Roth and Majerus, 1975). Because aspirin more selectively inhibits COX-1 activity (found predominantly in platelets) than COX-2 activity (expressed in tissues following an inflammatory stimulus), its ability to prevent platelet aggregation is seen at relatively low doses, compared with the drug’s potential antiinflammatory effects, which require much higher doses (Patrono, 1994). Several alternative mechanisms of platelet inhibition by aspirin have been proposed, including: (1) inhibition of platelet activation by neutrophils and (2) enhanced nitric oxide production. In addition, aspirin may prevent the progression of atherosclerosis by protecting low-density lipoprotein (LDL) cholesterol from oxidation and scavenging hydroxyl radicals. Following oral ingestion, aspirin is promptly absorbed in the proximal gastrointestinal (GI) tract (stomach, duodenum), achieving peak serum levels within 15 to 20 minutes and platelet inhibition within 40 to 60 minutes. Enteric-coated preparations are less well absorbed, causing an observed delay in peak serum levels and platelet inhibition to 60 and 90 minutes, respectively. The antiplatelet effect occurs even before acetylsalicylic acid is detectable in peripheral blood, probably from platelet exposure in the portal circulation. The plasma concentration of aspirin decays rapidly with a circulating half-life of approximately 20 minutes. Despite the drug’s rapid clearance, platelet inhibition persists for the platelet’s life span (7 ± 2 days) due to aspirin’s irreversible inactivation of COX-1. Because 10% of circulating platelets are replaced every 24 hours, platelet activity (bleeding time, primary hemostasis) returns toward normal (≥50% activity) within 5 to 6 days of the last aspirin dose (O’Brien, 1968). A single dose of 100 mg of aspirin effectively reduces the production of TXA2 in many (but not all) individuals.

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Тези доповідей конференцій з теми "OCCLUSIONE PORTALE"

Cubeddu, Rinaldo, Eleonora Giambattistelli, Antonio Pifferi, Paola Taroni, and Alessandro Torricelli. "Portable 8-channel time-resolved optical imager for functional studies of biological tissues." In European Conference on Biomedical Optics. Washington, D.C.: Optica Publishing Group, 2001. http://dx.doi.org/10.1364/ecbo.2001.4431_260.

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We developed a portable dual-wavelength 8-channel system for time-resolved reflectance measurements based on pulsed laser diodes, a multianode photomultiplier and a time-correlated single-photon counting board. The performances of the system were tested on phantoms in terms of stability, reproducibility among channels, and accuracy in the determination of absolute values of the absorption and transport scattering coefficients. Preliminary in vivo measurements (cuff occlusion protocols) were performed on healthy volunteers to monitor spatial changes in tissue oxygenation and blood volume.

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D'Cruz, Roshan, Deborah Rabinowitz, Abigail E. Martin, and Stephen Dunn. "Early Percutaneous Intervention in Pediatric Chronic Extrahepatic Portal Vein Occlusion Is Feasible And Safe." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.903-a.

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Garrone, R., and R. Munden. "Development and utilization of a wireless and portable photoplethysmograph to study the post-occlusive reactive hyperemic response in a finger." In 2015 IEEE Signal Processing in Medicine and Biology Symposium (SPMB). IEEE, 2015. http://dx.doi.org/10.1109/spmb.2015.7405455.

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Schwartz, R., E. Kavanagh, K. Bauer, R. Rosenberg, J. Ballard, J. Latino, V. Strother, M. Mosesson, W. Haire, and M. DeLeo. "ANTITHROMBIN III CONCENTRATE (AT-III) FOR PROPHYLAXIS ANDTREATMENT OF CONGENITAL AND ACQUIREDAT-III DEFCIENCY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643678.

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An investigation has been undertakenin 13 patient studies to determine the efficacy of AT-III in prevention and treatment of thrombosis in patients with congenital (cong.) and acquired (acq.) AT-III deficiency. The mean in vivo incremental recovery of 17 infusions of AT-III in 7 patientswith cong. AT-III deficiency (2 kinetic studies, 5 prophylaxis) was 1.4%/U/kg (functional assay) administered. The mean in vivo recovery of 38 infusions in 4 non-bleeding patients(3 cong., 1 acq.) treated for thrombosis or pulmonary embolism (P.E.) with heparin was 1.33%/U/kg which was not significantly different. The half-life determined in an earlier study exceeded 2.5 days. All patients treated prophylactically or therapeutically received a loading dose to increase plasma AT-III to 120%, and then received maintenance doses, generally every 24 hours, to maintain plasma AT-III levels in the general range 80-120%. AT-III levels were monitored every 12 hours initially and doses and intervals modified accordingly. None of 5 patients with cong. deficiency treated prophylactically for high risk situations (surgery, delivery, catheterization) developed a thrombotic complication. Six patients (3 cong., 2 acq., 1 probably acq.) were treated for thrombosis/P.E., two of whom were pregnant. Heparin resistance was reversed in two, both pregnant. One patient with superior mesenteric-portal vein thrombosis (cong. deficiency) and another with superior mesenteric artery-aortic occlusion (acq. deficiency) survived without further progression of thrombosis. DIC resolved in one patient treated with AT-III and benefit was observed in a 2nd patient as well. AT-III was well tolerated, with but a single mild reaction in 176 infusions. We conclude AT-III may be beneficialas prophylaxis in patients with cong.AT-III deficiency during high thrombotic risk situations, as well as anadjunct to heparin in treatment for thrombotic complications in congenital and acquired AT-III deficiency.

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