Готові списки джерел за темами / Neurological soft sign

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Добірка наукової літератури з теми "Neurological soft sign"

Автор: Grafiati
Опубліковано: 11 березня 2023

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Статті в журналах з теми "Neurological soft sign"

1

Dazzan, Paola, Tuhina Lloyd, Kevin D. Morgan, et al. "Neurological abnormalities and cognitive ability in first-episode psychosis." British Journal of Psychiatry 193, no. 3 (2008): 197–202. http://dx.doi.org/10.1192/bjp.bp.107.045450.

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BackgroundIt remains unclear if the excess of neurological soft signs, or of certain types of neurological soft signs, is common to all psychoses, and whether this excess is simply an epiphenomenon of the lower general cognitive ability present in psychosis.AimsTo investigate whether an excess of neurological soft signs is independent of diagnosis (schizophrenia v. affective psychosis) and cognitive ability (IQ).MethodEvaluation of types of neurological soft signs in a prospective cohort of all individuals presenting with psychoses over 2 years (n=310), and in a control group from the general
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Thioritz, Wempy, Erlyn Limoa, J. C. Hutomo, Saidah Syamsuddin, and Sonny T. Lisal. "Differentiation in Neurological Soft Sign Scores on Schizophrenic Patients with Antipsychotic Treatment." Open Access Macedonian Journal of Medical Sciences 9, T3 (2021): 249–53. http://dx.doi.org/10.3889/oamjms.2021.6356.

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Background: Schizophrenia is a chronic mental illness that affects cognitive aspect of a patient which need long term care with antipsychotics. Long term use of antipsychotic itself causes neurobiological change in the brain which results in alteration of cognitive function. The latest research had demonstrated that NSS (Neurological Soft Sign) reflect a rather wide range of cognitive impairments in schizophrenia which was not accounted for by age, education or severity of global cognitive deficits. Therefore, we examined the effects and impact of antipsychotic Haloperidol and Risperidone trea
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3

Phillips, P. H. "Intermittent exotropia increasing with near fixation: a "soft" sign of neurological disease." British Journal of Ophthalmology 89, no. 12 (2005): 1120–22. http://dx.doi.org/10.1136/bjo.2004.063123.

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Phillips, P. H., K. J. Fray, and M. C. Brodsky. "Intermittent Exotropia Increasing with Near Fixation: A “Soft” Sign of Neurological Disease." Journal of American Association for Pediatric Ophthalmology and Strabismus 10, no. 2 (2006): 188. http://dx.doi.org/10.1016/j.jaapos.2006.03.006.

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Picard, Hernàn, Anne Le Seac'h, Isabelle Amado, Raphael Gaillard, Marie-Odile Krebs, and Cécile Beauvillain. "Impaired saccadic adaptation in schizophrenic patients with high neurological soft sign scores." Psychiatry Research 199, no. 1 (2012): 12–18. http://dx.doi.org/10.1016/j.psychres.2012.04.039.

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GARDNER, DAVID, PETER B. LUCAS, and REX W. COWDRY. "Soft Sign Neurological Abnormalities in Borderline Personality Disorder and Normal Control Subjects." Journal of Nervous and Mental Disease 175, no. 3 (1987): 177–80. http://dx.doi.org/10.1097/00005053-198703000-00009.

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King, D. J., A. Wilson, S. J. Cooper, and J. L. Waddington. "The Clinical Correlates of Neurological Soft Signs in Chronic Schizophrenia." British Journal of Psychiatry 158, no. 6 (1991): 770–75. http://dx.doi.org/10.1192/bjp.158.6.770.

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Among 16 chronic schizophrenic in-patients, all had at least one neurological soft sign (NSS), and 6 (40%) had definite neurodysfunction. NSS and TD scores were highly intercorrelated, and NSS were significantly correlated with neuroleptic drug exposure. NSS correlated positively with both positive and negative symptoms and cognitive impairment but not with cerebral ventricular size on CT. Patients with neurodysfunction had more positive and negative psychopathology, cognitive impairment and TD than those without. Cerebral ventricular sizes and family histories of schizophrenia were similar in
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Aronowitz, Bonnie R., Concetta Decaria, Andrea Allen, et al. "The Neuropsychiatry of Autism and Asperger's Disorder: Review of the Literature and Case Reports." CNS Spectrums 2, no. 5 (1997): 43–60. http://dx.doi.org/10.1017/s1092852900004892.

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AbstractThe literature on neuropsychiatric, neuropsychological, and neuroimaging findings in autism and Asperger's disorder is briefly reviewed. Neurological soft sign, neuropsychological, and positron-emission tomography findings in one patient with Asperger's disorder and one patient with autism are highlighted, discussed as examples of heterogeneity, and integrated with the relevant literature.
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Toro Espinoza, P., M. E. Ceballos, D. Valenzuela, M. F. Inostroza, and J. Schröder. "Subtests of the heidelberg neurological soft sign scale that discriminate HIV patients with and without hand." Journal of the Neurological Sciences 357 (October 2015): e5. http://dx.doi.org/10.1016/j.jns.2015.08.096.

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Bajraktari, Mustafa, Majlinda Naco, Gentian Huti, Blerim Arapi, and Rudin Domi. "Fat Embolism Syndrome Without Bone Fracture: Is It Possible?" Open Access Macedonian Journal of Medical Sciences 10, no. C (2022): 331–35. http://dx.doi.org/10.3889/oamjms.2022.11169.

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BACKGROUND: Fat embolism syndrome is a life challenge syndrome. Early diagnosing and treatment can significantly improve the patient’s prognosis and likelihood of success. This syndrome occurs mainly after long bones fractures or orthopedic surgery up to 95% of diagnosed cases, but in unusual situation can be faced as well. These rare situations include diabetes mellitus, video-assisted thoracoscopies, fatty liver, and fat injection in plastic and cosmetic procedures. The likelihood of this syndrome can be increased if multiplex long bones fractures occur in the same patient simultaneously. Th
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Дисертації з теми "Neurological soft sign"

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Chen, Eric Y. H. "Soft neurological signs in schizophrenia." Thesis, University of Edinburgh, 2004. http://hdl.handle.net/1842/24331.

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This dissertation describes a series of studies addressing the prevalence, correlates and longitudinal changes in soft neurological signs (SNS) in schizophrenia. SNS are found to be increased in schizophrenia. The increase appeared to have both a genetic and a non-genetic component. It has been proposed that SNS could be considered as one of the biological markers expressing a mediating risk for schizophrenia. In order to clarify the role of SNS in this perspective it is important to understand factors that affect the expression of SNS in a given population. Previous studies have identified po
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Oskamp, Andrea [Verfasser]. "Neurological Soft Signs in Stadien der Anorexia nervosa / Andrea Oskamp." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2013. http://d-nb.info/103563824X/34.

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Goldhahn, Klaus [Verfasser]. "Neurological Soft Signs bei Patientinnen mit Anorexia nervosa / Klaus Goldhahn." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2009. http://d-nb.info/1023956985/34.

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Dazzan, Paola. "Neurological soft signs in first episode psychoses : their clinical and neuroanatomical correlates." Thesis, King's College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.430827.

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Galindo, Guarin Liliana. "Neurological soft signs, temperament and schizotypy in patients with schizophrenia and unaffected relatives: an FMRI study." Doctoral thesis, Universitat Autònoma de Barcelona, 2017. http://hdl.handle.net/10803/403815.

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La esquizofrenia es un trastorno psiquiátrico grave, un síndrome complejo y heterogéneo originado por la alteración del desarrollo del cerebro por factor genéticos o ambientales. Las bases genéticas pueden estar presentes en individuos sin enfermedad como en hermanos de pacientes y pueden ser identificadas a través de marcadores biológicos. Los signos neurológicos menores son discretas alteraciones sensitivo-motoras asociadas con un desarrollo cerebral alterado que se han propuesto como un endofenotipo de la esquizofrenia. Un perfil específico de temperamento y carácter así como la presencia d
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Negash, Alemayehu. "Bipolar disorder in rural Ethiopia : community-based studies in Butajira for screening, epidemiology, follow-up, and the burden of care." Doctoral thesis, Umeå universitet, Psykiatri, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-21743.

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Background: The challenges of research in economically stunted countries’ settings remains a profound concern and is linked to socioeconomic development of these countries. More research is needed regarding psychiatric morbidity in rural areas of the developing and poverty stricken countries. The present studies were undertaken within the framework of a broader ongoing community-based project on the course and outcome of major psychiatric disorders in the rural Butajira district located in Ethiopia. This thesis treats the course and outcome of bipolar I disorder in the district. Objectives: Th
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Rothman, David J. "An Investigation of Neurological soft signs as a discriminating factor between Veterans with Post-traumatic Stress Disorder, mild Traumatic Brain Injury, and co-occurring Post-traumatic Stress Disorder and mild Traumatic Brain Injury." VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/5915.

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While multiple Operation Enduring Freedom/Operation Iraqi Freedom/Operation New Dawn veterans suffer from mild Traumatic Brain Injury (mTBI), Post-traumatic Stress Disorder (PTSD), and co-morbid mTBI and PTSD, there remains difficulty disentangling the specific symptoms associated with each disorder using self-report and neurocognitive assessments. We propose that neurological soft signs (NSS), which are tasks associated with general neurologic compromise, may prove useful in this regard. Based on our review of the literature we hypothesized that individuals with PTSD would present with a grea
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Gay, Olivier. "Marqueurs neurodéveloppementaux en psychiatrie : intérêt dans les troubles schizophréniques." Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCB016/document.

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Le terme de neurodéveloppement dans son acception la plus large renvoie à l'ensemble des processus permettant le développement du système nerveux depuis les étapes les plus précoces de sa formation in utero jusqu'aux étapes plus tardives de maturation à l'adolescence aboutissant au système nerveux adulte. Les travaux de ces quarante dernières années ont conduit à proposer un modèle neurodéveloppemental des troubles psychiatriques, notamment schizophréniques, sur la base d'arguments génétiques, épidémiologiques et d'imagerie. Ce modèle propose que l'apparition de la maladie soit liée à une/des
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Aguirre, Pacheco Cándida Isabel. "Los signos neurológicos menores en la esquizofrenia: correlatos con las características clínicas, la función cognitiva y los cambios cerebrales estructurales." Doctoral thesis, Universitat Autònoma de Barcelona, 2018. http://hdl.handle.net/10803/665695.

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Los Signos Neurológicos Menores (SNM), entendidos como alteraciones neurológicas que no tienen una localización específica a nivel cerebral, presentan una mayor prevalencia en los pacientes con esquizofrenia que en la población sana. También se ha descrito una mayor frecuencia de los mismos en los familiares de los pacientes con esquizofrenia (aunque en menor medida que en los pacientes). Algunos aspectos de la asociación de los SNM con el cuadro clínico de la esquizofrenia permanecen sin esclarecer. Otros temas de interés en la literatura se dirigen hacia la relación entre los SNM y los défic
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Martinez, Gilles. "Continuum autisme-schizophrénie : apport de l’étude de la cognition sociale et de marqueurs phénotypiques développementaux." Thesis, Sorbonne Paris Cité, 2017. http://www.theses.fr/2017USPCB065/document.

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Autisme et schizophrénie sont deux troubles psychiatriques neuro-développementaux. L’étude des formes précoces de schizophrénie, fréquemment associées aux troubles du spectre de l’autisme (TSA), a suggéré un possible continuum développemental entre ces troubles. Des arguments cliniques et épidémiologiques, et issus des études en génétique moléculaire ou en imagerie cérébrale, sont progressivement venus étayer cette hypothèse. Dans ce contexte, l’étude de la cognition sociale a fait l’objet d’un intérêt particulier, des altérations étant rapportées dans les deux troubles avec toutefois des résu
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Книги з теми "Neurological soft sign"

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E, Tupper David, ed. Soft neurological signs. Grune & Stratton, 1987.

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Soft neurological signs in clumsy children: Indicators of timing and force control dysfunction. 1990.

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Частини книг з теми "Neurological soft sign"

1

Ojagbemi, Akin. "Neurological Soft Signs." In Encyclopedia of Personality and Individual Differences. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-24612-3_782.

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Ojagbemi, Akin. "Neurological Soft Signs." In Encyclopedia of Personality and Individual Differences. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-28099-8_782-1.

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Shaffer, David, Cornelius S. Stokman, Patricia A. O’Connor, et al. "Early Soft Neurological Signs and Later Psychopathology." In Life-Span Research on the Prediction of Psychopathology. Routledge, 2021. http://dx.doi.org/10.4324/9781003165187-3.

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Neeper, Ronald, and Robert S. Greenwood. "On the Psychiatric Importance of Neurological Soft Signs." In Advances in Clinical Child Psychology. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-9826-4_6.

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"Neurological Soft Signs." In Encyclopedia of Clinical Neuropsychology. Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_4874.

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Schröder, Johannes, and Christina J. Herold. "Neurological Soft Signs in Schizophrenia Spectrum Disorders." In Movement Disorders in Psychiatry, edited by Antonio L. Teixeira, Erin Furr Stimming, and William G. Ondo. Oxford University PressNew York, 2022. http://dx.doi.org/10.1093/med/9780197574317.003.0010.

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Abstract Neurological soft signs (NSS) are minor motor and sensory changes that are frequently found in schizophrenia spectrum disorders, including subjects with an increased liability to them, but can also be observed in bipolar disorder and dementia. In schizophrenia, high NSS-scores, which typically mark acute psychotic states, decrease in the course of illness with remission of acute symptoms. This process continues in patients with a favorable course but does not lead to a complete “normalization” of NSS, which remain in the range typical for subjects with an increased liability. As a transdiagnostic phenomenon, NSS are associated with similar symptoms and neurocognitive deficits across the respective disorders, such as negative symptoms, apathy or executive and declarative memory dysfunction. Neuroimaging studies in schizophrenia identified frontal cortices, including pre- and postcentral gyrus, inferior and middle frontal gyrus, and premotor area, alongside cerebellum, caudate, and thalamus as important sites for NSS. NSS can be used for early recognition and clinical diagnosis of schizophrenia and have the potential to facilitate our understanding of the cerebral mechanisms involved in its pathophysiology.
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Mehta, Gautam, and Bilal Iqbal. "Central Nervous System." In Clinical Medicine for the MRCP PACES. Oxford University Press, 2010. http://dx.doi.org/10.1093/oso/9780199542550.003.0011.

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As with all neurological patients, you will be more likely to pick up the diagnosis if you take a step back and look at the whole patient. Take some time to assess their facial expressions, speech, tremor, and posture. A common instruction at this station, with the patient seated on a chair is ‘Look at this patient, and examine as appropriate’. Candidates are often baffled, when given this instruction. Often the patients with Parkinson’s disease are given specific instructions to interlock the fingers of both hands, or place hands flat on their lap to mask the tremor. Picking up an expressionless face and low volume monotonous speech from the outset will provide useful clues to the diagnosis. If you are not sure at this stage, proceed to examining the gait. Once you are certain, that this is Parkinson’s disease, you may proceed to demonstrate the other features. 1. Patients with Parkinson’s disease have characteristic expressionless facies (hypomimia), often described as ‘mask-like’. This is a manifestation of bradykinesia. There is a reduced blink rate. The glabellar tap (Myerson’s sign) is an unreliable sign and is not recommended in the examination. This involves tapping the patient’s forehead repeatedly. Normal subjects will stop blinking, but in Parkinson’s disease, the patient will continue to blink. The patient may be drooling saliva (resulting from dysphagia and sialorrhoea-due to autonomic dysfunction) 2. Patients may have soft speech (hypophonia). This is also a manifestation of bradykinesia, and characteristically, the speech is low-volume, monotonous and tremulous (appears slurred). 3. Blepharoclonus is tremor of the eyelids. This will only be demonstrated if the eyes are gently closed, as opposed to tightly closing the eyes. 4. The classic tremor is present at rest and asymmetrical (more marked on one side). It is classically described as being 4–6Hz and is the initial symptom in 60% of cases, although 20% of patients never have a tremor. The tremor may appear as a ‘pill-rolling’ motion of the hand or a simple oscillation of the hand or arm. It is easier to spot a tremor if you ask the patient to rest their arms in their lap in the semi-prone position.
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Schröder, Johannes, and Christina J. Herold. "Neurological Soft Signs – A Transdiagnostic Phenomenon in Neuropsychiatric Conditions." In Reference Module in Neuroscience and Biobehavioral Psychology. Elsevier, 2020. http://dx.doi.org/10.1016/b978-0-12-809324-5.24016-9.

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Sechi, Elia, and Dean M. Wingerchuk. "Rapidly Progressive Numbness and Weakness After Soft-Tissue Abscess." In Mayo Clinic Cases in Neuroimmunology, edited by Andrew McKeon, B. Mark Keegan, and W. Oliver Tobin. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780197583425.003.0002.

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A previously healthy 45-year-old man had development of neck pain and swelling, followed 1 week later by fevers, chills, and night sweats. Cervical computed tomography showed a left-sided cervical soft-tissue abscess. The patient was treated with oral cephalexin for 10 days, without benefit. Fine-needle aspiration biopsy of the mass showed granulomatous inflammation and a heterogeneous lymphocyte population without evidence of malignancy. Meropenem and gentamicin were started. Ten days later, he had development of acute urinary retention, numbness and weakness in the lower extremities, and numbness in the upper extremities. At symptom nadir 2 days later, he required the aid of a walker to ambulate. Lhermitte sign and erectile dysfunction were also present. The patient was admitted to the hospital. Spinal cord magnetic resonance imaging showed a longitudinally extensive, nonenhancing, T2-hyperintense lesion predominantly affecting the ventral and lateral parenchyma of the cervical and thoracic spinal cord. Cerebrospinal fluid examination showed a white blood cell count of 581 cells/µL with 42% neutrophils, 35% lymphocytes, and 22% monocytes, increased protein concentration (109 mg/dL), and normal glucose concentration. A diagnosis of postinfectious idiopathic transverse myelitis was made. The patient was treated with intravenous immunoglobulin, intravenous methylprednisolone, and broad-spectrum antibiotics, with improvement of both the abscess and his neurologic symptoms. After discharge, he was able to walk unassisted. At follow-up evaluation 6 months after the initial evaluation, neurologic examination showed only mild weakness of the left iliopsoas muscle and brisk reflexes in the lower extremities. Acute transverse myelopathies are a heterogeneous group of spinal cord disorders characterized by acute or subacute signs and symptoms of spinal cord dysfunction, typically a combination of sensory, motor, and autonomic manifestations. Underlying causes include vascular, infectious, neoplastic, postirradiation, traumatic, and inherited/metabolic, and inflammatory processes.
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Тези доповідей конференцій з теми "Neurological soft sign"

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Silva, Bruno Custódio, Fernanda Silva dos Santos, Victória Porcher Simioni, Ana Luíza Kolling Konopka, Paulo Ricardo Gazzola Zen, and Rafael Fabiano Machado Rosa. "Type 1 neurofibromatosis and its relation to the occurrence of cerebral vascular accident." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.077.

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Context: Neurofibromatosis type 1 (NF1) is characterized by “café-au-lait” (CAL) spots, ephelids, cutaneous neurofibromas and iris Lisch nodules. Case report: A 63 year-old female patient came to evaluation due to NF1. She had a history of CAL spots, nodules on the skin, cognitive deficit and seizures. Chest X-ray showed several small nodules scattered in soft tissues of the thoracic wall. Magnetic resonance imaging of the skull demonstrated a possible increase in intracranial pressure. Computed tomography scan of the skull showed a hypodense area in the left hemisphere, consistent with a rece
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Schülke, Rasmus, Kyra Liepach, AnnaL Brömstrup, et al. "Neurological soft signs are increased in major depressive disorder irrespective of antidepressant treatment." In Abstracts of the 3rd Symposium of the Arbeitsgemeinschaft für Neuropsychopharmakologie und Pharmakopsychiatrie (AGNP) and Deutsche Gesellschaft für Biologische Psychiatrie (DGBP). Georg Thieme Verlag, 2022. http://dx.doi.org/10.1055/s-0042-1757665.

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Kaneko, Miki, Keiji Iramina, Takashi Ohya, et al. "A measurement of soft neurological signs by pronosupination using wireless acceleration and angular velocity sensors." In 2011 Biomedical Engineering International Conference (BMEiCON) - Conference postponed to 2012. IEEE, 2012. http://dx.doi.org/10.1109/bmeicon.2012.6172050.

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Звіти організацій з теми "Neurological soft sign"

1

Pitman, Roger K. Can Post mTBI Neurological Soft Signs Predict Postconcussive and PTSD Symptoms: A Pilot Study. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada602492.

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Pitman, Roger K. Can Post mTBI Neurological Soft Signs Predict Postconcussive and PTSD Symptoms: A Pilot Study. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada602679.

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