Добірка наукової літератури з теми "Negative regulators"
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Статті в журналах з теми "Negative regulators"
Thomson, Christopher W., Boris P. L. Lee, and Li Zhang. "Double-Negative Regulatory T Cells: Non-conventional Regulators." Immunologic Research 35, no. 1-2 (2006): 163–78. http://dx.doi.org/10.1385/ir:35:1:163.
Повний текст джерелаMassagué, Joan, and Robert A. Weinberg. "Negative regulators of growth." Current Opinion in Genetics & Development 2, no. 1 (February 1992): 28–32. http://dx.doi.org/10.1016/s0959-437x(05)80317-x.
Повний текст джерелаPouwels, J., J. Nevo, T. Pellinen, J. Ylanne, and J. Ivaska. "Negative regulators of integrin activity." Journal of Cell Science 125, no. 14 (July 15, 2012): 3271–80. http://dx.doi.org/10.1242/jcs.093641.
Повний текст джерелаFrank, Steven A., and Paul Schmid-Hempel. "Evolution of negative immune regulators." PLOS Pathogens 15, no. 8 (August 1, 2019): e1007913. http://dx.doi.org/10.1371/journal.ppat.1007913.
Повний текст джерелаKile, Benjamin T., Nicos A. Nicola, and Warren S. Alexander. "Negative Regulators of Cytokine Signaling." International Journal of Hematology 73, no. 3 (April 2001): 292–98. http://dx.doi.org/10.1007/bf02981953.
Повний текст джерелаMonticelli, Silvia, and Federica Sallusto. "Negative regulators take center stage." Nature Immunology 13, no. 8 (July 19, 2012): 719–20. http://dx.doi.org/10.1038/ni.2377.
Повний текст джерелаJohnson, Terry C. "Negative regulators of cell proliferation." Pharmacology & Therapeutics 62, no. 1-2 (January 1994): 247–65. http://dx.doi.org/10.1016/0163-7258(94)90013-2.
Повний текст джерелаWang, John L., and Yen-Ming Hsu. "Negative regulators of cell growth." Trends in Biochemical Sciences 11, no. 1 (January 1986): 24–26. http://dx.doi.org/10.1016/0968-0004(86)90227-6.
Повний текст джерелаHARDT, S. "Negative regulators of cardiac hypertrophy." Cardiovascular Research 63, no. 3 (August 2004): 500–509. http://dx.doi.org/10.1016/j.cardiores.2004.03.015.
Повний текст джерелаEllis, Ronald E. "Negative regulators of programed cell death." Current Opinion in Genetics & Development 2, no. 4 (January 1992): 635–41. http://dx.doi.org/10.1016/s0959-437x(05)80184-4.
Повний текст джерелаДисертації з теми "Negative regulators"
Lively, Julie C. (Julie Christina) 1971. "Beta 3 integrins : negative regulators of angiogenesis." Thesis, Massachusetts Institute of Technology, 2002. http://hdl.handle.net/1721.1/8386.
Повний текст джерелаIncludes bibliographical references (leaves 199-219).
A method was developed to isolate and purify primary murine endothelial cells from lung tissue (MLEC). The cells generated by this method were characterized by immuno-fluorescence detection and FACS analysis and expressed specific antigens including PECAM-1, ICAM-1, ICAM-2, VCAM-1 and VE-cadherin. Using this method, cells from wild-type and beta 3-integrin-deficient animals were purified and used to determine the specificity of a novel potential anti-angiogenic drug. This study shows that tumstatin, a fragment of the alpha 3 chain of collagen IV, inhibits proliferation, inhibits total protein synthesis and specifically inhibits CAP-dependent protein synthesis in MLEC. These effects do not occur when beta 3-null MLEC are treated with tumstatin or any of its derivatives. Nor do they occur in mouse embryonic fibroblasts which do express beta 3 integrin. The inhibition by tumstatin also occurs in in vivo angiogenesis assayed using a Matrigel plug insert. Similarly to in vitro assays, tumstatin failed to inhibit angiogenesis in beta 3 integrin-deficient animals. These results suggest that avf33 integrin is necessary but not sufficient for the activity of tumstatin. Further studies are required to identify avf33 integrin-associated factors in endothelial cells which determine tumstatin's endothelial cell specificity. Matrigel plug assays were also used to demonstrate that the loss of beta-3 integrin enhanced VEGF-induced angiogenesis. Results also show that VEGF-induced angiogenesis was enhanced in aortic ring explants from beta 3-null animals. These data suggest a new role for beta 3 integrin as a negative regulator of angiogenesis, both as a receptor for an endogenous inhibitory molecule and as an inhibitor of VEGF-induced angiogenesis.
by Julie C. Lively.
Ph.D.
Kearns, Jeffrey D. "Distinct functions of negative regulators of NF-kappaB." Diss., [La Jolla] : University of California, San Diego, 2009. http://wwwlib.umi.com/cr/ucsd/fullcit?p3360060.
Повний текст джерелаTitle from first page of PDF file (viewed August 11, 2009). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references (p. 197-205).
Datar, Ila. "Positive and negative regulators of tumorigenesis and/or metastasis." University of Toledo Health Science Campus / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=mco1438962728.
Повний текст джерелаSubedee, Ashim. "Molecular Determinants and Transcriptional Regulators in Triple Negative Breast Cancer." Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:23845415.
Повний текст джерелаMedical Sciences
Carlsson, Emil Karl Viktor. "Biochemical, molecular and cellular studies on negative regulators of TLR-signalling." Thesis, Imperial College London, 2015. http://hdl.handle.net/10044/1/38528.
Повний текст джерелаSpencer, William John. "Negative regulators of chromosome replication in the dimorphic bacterium Caulobacter crescentus." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=103183.
Повний текст джерелаIn Caulobacter, chromosome replication is repressed, in part, by the binding of the response regulator CtrA to five binding sites (a-e) within the Caulobacter origin of replication (Cori ). Periodic phosphorylation of CtrA stimulates binding to the consensus sequence TTAA-N7-TTAA (N= any nucleotide) found in Cori and many cell-cycle regulated genes. This thesis presents an alternate mode of CtrA binding, namely, that phosphorylation does not stimulate binding to a specific class of CtrA-regulated promoters. This work shows that CtrA and CtrA-phosphate bind to two ctrA promoters with equal and weak affinity. As well, in vivo binding assays reveal that a non-proteolyzable CtrA allele (CtrADelta3) can occupy the ctrA promoters continuously without altering the temporal regulation of these promoters. The data suggest phosphorylation, while not increasing affinity for weak CtrA binding sites, provides allosteric signals that permit the recruitment of components required for transcription.
The proposed allosteric mechanism of CtrA-regulated transcription may also be important for CtrA-mediated repression of chromosome replication. Chromatin Immunoprecipitation assays (ChIP) allow for the sensitive detection of specific protein/DNA complexes in vivo. ChIP reveals that CtrA binds to Cori in swarmers but not in stalk cells when chromosome replication commences. The protein chaperone, ClpX, was recruited to Cori prior to the start of S-phase and correlates with the loss of CtrA binding to Cori. Expression of a non-proteolyzable CtrADelta3 allele showed increased affinity for Cori DNA. The increase in CtrADelta3 binding stimulated a corresponding increase in C1pX binding to Cori. This evidence suggests that C1pX recruitment to Cori is likely CtrA-dependant. The absence of CtrA binding in stalk cells suggests other mechanisms may be required to prevent re-replication in stalk cells.
An analysis of the Caulobacter genome identifies two DnaA-like genes. The first, cdl-1, is a homolog of the E. coli hda gene, a protein essential for regulated inactivation of DnaA (RIDA). The second, cdl-2, is a novel gene restricted to the alpha-proteobacteria group and whose function is unknown. Overexpression of either gene in Caulobacter produced filamentous cells that could not divide. DNA synthesis in these cells is also impaired and suggests the intracellular concentrations of these two proteins are important for coordinating proper cell cycle progression.
Hooker, Erika. "Negative regulators of the Src family kinases in renal epithelial cells." Thesis, McGill University, 2013. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=116932.
Повний текст джерелаLes kinases Src sont des tyrosine-kinases cytosoliques qui sont impliquées dans multiples processus dans les cellules épithéliales et autres. Originalement identifiée comme un oncogène viral, la kinase Src est maintenant caractérisée comme une régulatrice de la prolifération, la différenciation et la motilité cellulaire. Nous avons précédemment montré que les kinases Src sont capables de modifier l'expression génique dans les tubules des reins durant le domage rénal par ischémie et réperfusion. Cependant, les mécanismes de signalisation qui contrôle la réponse transcriptionelle des kinases Src ne sont pas bien compris. La présente thèse décrit deux nouveaux inhibiteurs endogènes de la famille de kinases Src dans les cellules rénale épithéliales.Les deux premiers manuscrits établissent que la protéine adaptatrice Dok-4 fonctionne comme un inhibiteur des kinases Src. Contrairement à la plus part de protéines adaptatrices, la famille Dok est caractérisée par des actions inhibitrices durant la signalisation par les tyrosines kinases. Malgré que Dok-4 soit le membre de la famille Dok exprimé de manière la plus ubiquitaire, sa fonction est encore mal connue. Le premier manuscrit que je présente (Manuscrit I) décrit le domaine PTB de Dok-4. On y a démontré que le domaine PTB contient une extension C-terminal consistant probablement en une hélice alpha et que celle-ci est essentielle pour les interactions canoniques du domaine PTB de Dok-4. De plus, nous avons identifié la phosphatase lipidique Ship1 comme un nouveau partenaire de ce domaine PTB redéfini. Cette interaction est augmentée quand les kinases Src sont actives et elle implique un motif NPXpY dans la région C-terminale de Ship1. Contrairement à l'interaction entre Dok-4 et Ship1, l'interaction décrite dans le deuxième manuscrit (Manuscrit II) entre Dok-4 et le facteur de transcription, Elk4, implique le domaine PTB, mais se fait dans une manière atypique. L'interaction entre Dok-4 et Elk4 induit la relocalisation d'Elk4 du noyau au cytoplasme et cause la dégradation de la protéine Elk4. Dans les cellules rénales, Dok-4 inhibe l'activation d'Elk4 par les kinases Src et réprime l'expression des gènes de réponse précoce ("immediate early genes"), comme egr-1 et fos, et quelques cibles transcriptionelles de ces gènes. En accord avec ces données, suppression de Dok-4 est associée avec une augmentation de prolifération. En utilisant un modèle in vivo d'ischémie-reperfusion rénale, où la surexpression de gène de réponse précoce a déjà été démontrée, nous avons détecté une forte activation des kinases Src suivie d'une augmentation retardée de l'expression d'Elk4 dans les lysates de reins. Ces données suggèrent que dans ce modèle Dok-4 pourrait être critique pour limiter les dommages aux reins causé par l'induction des gènes de réponse précoce par Elk4. En plus d'activer l'expression des gènes de réponse précoce, nous avons précédemment montré que les kinases Src sont impliquées dans l'induction transcriptionnelle du récepteur tyrosine-kinase, EphA2, durant l'ischémie-reperfusion rénale. Dans le manuscrit préliminaire que je présente, nous avons noté que dans un modèle de déplétion et réplétion d'ATP, les kinases Src sont activées et les protéines Stat, des effecteurs des kinases Jak, sont déphsophorylés et inactives. Comme corollaire de cette observation, la surexpression de trois membres de de la famille Jak inhibent l'activation du promoteur d'EphA2 par les Src kinases. En plus, l'inhibition des kinases Jak endogènes par traitement aux siRNA ou par un inhibiteur pharmacologique, Jak Inhibitor I, active le promoteur d'EphA2. Étonnement, l'inhibition de l'expression d'EphA2 par les kinases Jak se fait indépendamment des protéines Stat et les récepteurs à cytokines. Mises ensemble, les données de cette thèse démontrent deux nouveaux inhibiteurs de la famille Src dans les cellules rénales épithéliales, la protéine adaptatrice, Dok-4 et les kinases, Jak1 et Jak2.
Evans, Abigail Alexandra. "An analysis of selected negative regulators of growth in breast cancer." Thesis, Queen Mary, University of London, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.287979.
Повний текст джерелаGadbois, Ellen L. (Ellen Louise) 1968. "Functional antagonism of the RNA polymerase II holoenzyme by negative regulators." Thesis, Massachusetts Institute of Technology, 1997. http://hdl.handle.net/1721.1/43553.
Повний текст джерелаMiller, Allan. "Negative regulators of gene expression in yeast : a1/α2 and SIR". Thesis, University of Cambridge, 1987. https://www.repository.cam.ac.uk/handle/1810/270426.
Повний текст джерелаКниги з теми "Negative regulators"
McPhee, Jennifer. ID-1 and GDF-8 as negative regulators of skeletal muscle mass. Sudbury, Ont: Laurentian University, Behavioural Neuroscience Program, 1998.
Знайти повний текст джерелаBeth, McNeil, and Johnson Denise J, eds. Patron behavior in libraries: A Handbook of Positive Approaches to Negative Situations. Chicago: American Library Association, 1996.
Знайти повний текст джерелаRetnakaran, Ravi. Identification of RVR, a novel orphan nuclear receptor that acts as a negative regulator of transcription. Ottawa: National Library of Canada, 1994.
Знайти повний текст джерелаek, Slavoj Z. iz. Tarrying with the negative: Kant, Hegel, and the critique of ideology. Durham: Duke University Press, 1993.
Знайти повний текст джерелаLi, Georgia Xiaojie. Both positive and negative regulatory elements may be required for the expression of the human VAMP-1 gene. Ottawa: National Library of Canada, 1994.
Знайти повний текст джерелаPhilippines. Foreign Investments Act of 1991, R.A. 7042: As amended by R.A. 8179 : implementing rules and regulations : second regular foreign investment negative list. [Makati, Metro Manila, Philippines: Dept. of Trade and Industry, 1996.
Знайти повний текст джерелаGrafkina, Marina. Labor protection. ru: INFRA-M Academic Publishing LLC., 2021. http://dx.doi.org/10.12737/1173489.
Повний текст джерелаUnited States. Congress. House. Committee on Banking, Finance, and Urban Affairs. Subcommittee on Financial Institutions Supervision, Regulation, and Deposit Insurance. Agency actions to reduce the negative impact of regulations on credit availability: Hearing before the Subcommittee on Financial Institutions Supervision, Regulation, and Deposit Insurance of the Committee on Banking, Finance, and Urban Affairs, House of Representatives, One Hundred Third Congress, first session, June 29, 1993. Washington: U.S. G.P.O., 1993.
Знайти повний текст джерелаSuryawan, Agus. Positive and negative regulators of adipocyte differentiation in primary culture. 1995.
Знайти повний текст джерелаNegative regulators of hematopoiesis: Studies on their nature, action, and potential role in cancer therapy. New York, N.Y: New York Academy of Sciences, 1991.
Знайти повний текст джерелаЧастини книг з теми "Negative regulators"
Suchting, Steven, Catarina Freitas, Ferdinand le Noble, Rui Benedito, Christiane Bréant, Antonio Duarte, and Anne Eichmann. "Negative Regulators of Vessel Patterning." In Vascular Development, 77–86. Chichester, UK: John Wiley & Sons, Ltd, 2007. http://dx.doi.org/10.1002/9780470319413.ch7.
Повний текст джерелаCampion, Dennis R., and William Kelly Jones. "Regulation of Growth by Negative Growth Regulators." In Animal Growth Regulation, 123–40. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-8872-2_7.
Повний текст джерелаZimmermann, Wolfgang, and Robert Kammerer. "Negative Regulators in Cancer Immunology and Immunotherapy." In Experimental and Applied Immunotherapy, 229–49. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-980-2_11.
Повний текст джерелаHenriksen, Melissa A., and Aurel Betz. "Negative Regulators of STAT Function in Drosophila." In Signal Transducers and Activators of Transcription (STATs), 609–21. Dordrecht: Springer Netherlands, 2003. http://dx.doi.org/10.1007/978-94-017-3000-6_38.
Повний текст джерелаSmolinski, Kara N., and Stephen J. Meltzer. "Inactivation of Negative Growth Regulators During Neoplastic Transformation." In The Molecular Basis of Human Cancer, 81–111. Totowa, NJ: Humana Press, 2002. http://dx.doi.org/10.1007/978-1-59259-125-1_5.
Повний текст джерелаWang, Helen Y., and Rong-Fu Wang. "Innate Immune Signaling and Negative Regulators in Cancer." In Innate Immune Regulation and Cancer Immunotherapy, 61–88. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-9914-6_6.
Повний текст джерелаWright, E. G., and I. B. Pragnell. "The Stem Cell Compartment: Assays and Negative Regulators." In Current Topics in Microbiology and Immunology, 137–49. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76912-2_11.
Повний текст джерелаPeluso, J. J. "Steroids as Negative Regulators of Granulosa Cell Proliferation and Differentiation." In Cell Culture in Pharmaceutical Research, 215–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-662-03011-0_11.
Повний текст джерелаMurphy, Caroline, and Luke A. J. O’Neill. "Negative Regulators of NF-κB Activation and Type I Interferon Pathways." In Innate Immune Regulation and Cancer Immunotherapy, 267–87. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-9914-6_15.
Повний текст джерелаSaxena, Amit, and Nikolaos G. Frangogiannis. "Negative Regulators of Inflammation as Endogenous Protective Mechanisms in Postinfarction Remodeling." In Cardiac Remodeling, 313–30. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-5930-9_18.
Повний текст джерелаТези доповідей конференцій з теми "Negative regulators"
Kong, Moufu, Bin Wang, BingKe Zhang, Ke Huang, Jiaxin Guo, and Jiawei Xu. "Novel CMOS Positive and Negative Voltage Mutual Conversion Circuits and Regulators." In 2020 IEEE 15th International Conference on Solid-State & Integrated Circuit Technology (ICSICT). IEEE, 2020. http://dx.doi.org/10.1109/icsict49897.2020.9278190.
Повний текст джерелаHailemichael, Yared, Glenn Winn, and Michael Davies. "914 Regulating negative immune regulators to enhance immune checkpoint blockade antitumor potential." In SITC 37th Annual Meeting (SITC 2022) Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-sitc2022.0914.
Повний текст джерелаBurnett, Joseph Patrick, Garrett Johnson, Nathan Truchan, Michael Brooks, Max S. Wicha, and Duxin Sun. "Abstract 500: Discovering epigenetic regulators of cells states in triple negative breast cancers." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-500.
Повний текст джерелаJizhen Fu, Zhiliang Zhang, W. Eberle, Yan-Fei Liu, and P. C. Sen. "A high efficiency current source driver with negative gate voltage for buck voltage regulators." In 2009 IEEE Energy Conversion Congress and Exposition. ECCE 2009. IEEE, 2009. http://dx.doi.org/10.1109/ecce.2009.5316231.
Повний текст джерелаHancock, BA, Y.-H. Chen, JP Solzak, MN Ahmad, DC Wedge, D. Brinza, C. Scafe, et al. "Abstract P2-07-04: Molecular regulators of resistance and relapse in chemorefractory triple-negative breast cancers." In Abstracts: 2017 San Antonio Breast Cancer Symposium; December 5-9, 2017; San Antonio, Texas. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.sabcs17-p2-07-04.
Повний текст джерелаГинда, Елена, та Наталья Трескина. "Использование регуляторов роста растений для реализации продуктивного потенциала столового сорта винограда велика в зависимости от гидротермических условий периода вегетации". У VIIth International Scientific Conference “Genetics, Physiology and Plant Breeding”. Institute of Genetics, Physiology and Plant Protection, Republic of Moldova, 2021. http://dx.doi.org/10.53040/gppb7.2021.37.
Повний текст джерелаLee, E., K. Ito, HY Irie, and J. Zhu. "Abstract P5-08-02: Identify key regulators to modulate chemo-sensitivity of triple negative breast cancer by integrative analysis." In Abstracts: 2017 San Antonio Breast Cancer Symposium; December 5-9, 2017; San Antonio, Texas. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.sabcs17-p5-08-02.
Повний текст джерелаWorsham, MJ, KM Chen, I. Datta, JK Stephen, D. Chitale, and G. Divine. "Abstract P1-04-06: Network integration of epigenomic data: Leveraging the concept of master regulators in ER negative breast cancer." In Abstracts: 2016 San Antonio Breast Cancer Symposium; December 6-10, 2016; San Antonio, Texas. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.sabcs16-p1-04-06.
Повний текст джерелаLee-Verges, Eriong, Arnau Montraveta, Magda Pinyol, Pedro Jares, Cristina Arimany-Nardi, Marta Aymerich, Neus Villamor, et al. "Abstract 993: Upregulation of B-cell activation genes and negative regulators of apoptosis determines resistance to bendamustine in chronic lymphocytic leukemia cells." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-993.
Повний текст джерелаWorsham, Maria J., Kang Mei Chen, Indrani Datta, Josena K. Stephen, Dhananjay Chitale, and George Divine. "Abstract 4484: Differentially methylation between ER negative and ER positive breast cancer identifies master regulators to expose potential epigenetic drivers of aggressive disease." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-4484.
Повний текст джерелаЗвіти організацій з теми "Negative regulators"
Li, Luyuan. A Novel Negative Regulator of Angiogenesis. Fort Belvoir, VA: Defense Technical Information Center, August 1999. http://dx.doi.org/10.21236/ada390998.
Повний текст джерелаLi, Luyuan. A Novel Negative Regulator of Angiogenesis. Fort Belvoir, VA: Defense Technical Information Center, August 2000. http://dx.doi.org/10.21236/ada391074.
Повний текст джерелаOviedo, Daniel, Daniel Perez Jaramillo, and Mariajosé Nieto. Governance and Regulation of Ride-hailing Services in Emerging Markets: Challenges, Experiences and Implications. Inter-American Development Bank, August 2021. http://dx.doi.org/10.18235/0003579.
Повний текст джерелаGalili, Gad, Harry J. Klee, and Asaph Aharoni. Elucidating the impact of enhanced conversion of primary to secondary metabolism on phenylpropanoids secondary metabolites associated with flavor, aroma and health in tomato fruits. United States Department of Agriculture, January 2012. http://dx.doi.org/10.32747/2012.7597920.bard.
Повний текст джерелаAndoniou, Christopher E. The Target Sites on EGF Receptor for CBL Its Negative Regulator. Fort Belvoir, VA: Defense Technical Information Center, April 1999. http://dx.doi.org/10.21236/ada367438.
Повний текст джерелаAndoniou, Christopher E., and Hamid Band. The Target Sites on EGF Receptor for Cbl, It's Negative Regulator. Fort Belvoir, VA: Defense Technical Information Center, April 2000. http://dx.doi.org/10.21236/ada384088.
Повний текст джерелаBarash, Itamar, and Robert Rhoads. Translational Mechanisms Governing Milk Protein Levels and Composition. United States Department of Agriculture, 2006. http://dx.doi.org/10.32747/2006.7696526.bard.
Повний текст джерелаMarsden, Eric. Risk regulation, liability and insurance: literature review of their influence on safety management. Fondation pour une culture de sécurité industrielle, September 2014. http://dx.doi.org/10.57071/337rrl.
Повний текст джерелаHuang, Ran, and Stacy Lee. Perceived Deception or Perceived Relevance? The Role of Self-Regulatory Focus in Processing Negative Online Reviews. Ames (Iowa): Iowa State University. Library, January 2019. http://dx.doi.org/10.31274/itaa.8459.
Повний текст джерелаYalovsky, Shaul, and Julian Schroeder. The function of protein farnesylation in early events of ABA signal transduction in stomatal guard cells of Arabidopsis. United States Department of Agriculture, January 2002. http://dx.doi.org/10.32747/2002.7695873.bard.
Повний текст джерела