Дисертації з теми "Mutant Phenotype"
Оформте джерело за APA, MLA, Chicago, Harvard та іншими стилями
Ознайомтеся з топ-50 дисертацій для дослідження на тему "Mutant Phenotype".
Біля кожної праці в переліку літератури доступна кнопка «Додати до бібліографії». Скористайтеся нею – і ми автоматично оформимо бібліографічне посилання на обрану працю в потрібному вам стилі цитування: APA, MLA, «Гарвард», «Чикаго», «Ванкувер» тощо.
Також ви можете завантажити повний текст наукової публікації у форматі «.pdf» та прочитати онлайн анотацію до роботи, якщо відповідні параметри наявні в метаданих.
Переглядайте дисертації для різних дисциплін та оформлюйте правильно вашу бібліографію.
Sofia, Francesca. "Emx1 null mutant mouse phenotype : potential implications for human epilepsy." Thesis, Open University, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.251365.
Повний текст джерелаRejeski, Kai Dannebom [Verfasser], and Michael [Akademischer Betreuer] Lübbert. "Elucidation of a hypersplicing phenotype in SRSF2 mutant myeloid malignancy." Freiburg : Universität, 2021. http://d-nb.info/1236500849/34.
Повний текст джерелаMorrison, Gillian M. "Analysis of the pulmonary inflammatory phenotype of the CF mutant mouse." Thesis, University of Edinburgh, 1999. http://hdl.handle.net/1842/22507.
Повний текст джерелаBenn, Caroline Louise. "Targeting mutant huntingtin to the nucleus accelerates phenotype onset in transgenic mice." Thesis, King's College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.401268.
Повний текст джерелаDonzelli, A. "BEHAVIOURAL PHENOTYPE AND ELECTROENCEPHALOGRAPHIC PROFILE OF ADOLESCENT AND ADULT SNAP-25+/- MUTANT MICE." Doctoral thesis, Università degli Studi di Milano, 2013. http://hdl.handle.net/2434/214980.
Повний текст джерелаJolivet, Katell. "Cloning and characterisation of LDW1, an Arabidopsis thaliana mutant displaying a lesion-mimic, dwarf phenotype." Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.405621.
Повний текст джерелаSkowrońska, Anna Maria. "ATM mutant cellular phenotype in B-cell chronic lymphocytic leukaemia : clinical consequences and therapeutic implications." Thesis, University of Birmingham, 2013. http://etheses.bham.ac.uk//id/eprint/4720/.
Повний текст джерелаBukowski-Wills, Jimi-Carlo. "Molecular links between genotype and phenotype in the albino-Swiss PKC-gamma mutant (AS/AGU) rat." Thesis, University of Glasgow, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.443440.
Повний текст джерелаPlong, Alexander. "Developmental and leaf senescence phenotype analysis in Arabidopsis thaliana| The atx4 (AT4G27910) mutant displays delayed leaf senescence and the kdm5b_1 (AT5G46910) mutant has larger seeds." Thesis, California State University, Long Beach, 2015. http://pqdtopen.proquest.com/#viewpdf?dispub=1603973.
Повний текст джерелаLeaf senescence is the final stage of leaf development that results in the highly ordered degeneration of cellular organelles and reallocation of nutrients to younger developing tissue. These cellular changes are accompanied by global changes in gene expression. Epigenetic modifications, such as DNA methylation, changes in histone variants, and covalent modification of histones have been shown to influence the expression of genes. Previous ChIP-seq and RNA-seq analyses revealed a correlation between the trimethylation of histone H3 lysine 4 (H3K4me3), a mark associated with active transcription, and the expression of a subset of senescence associated genes (SAGs) during leaf senescence in Arabidopsis thaliana. In this study, T-DNA insertion mutants of five histone methyltransferases (HMTases or ATX/ATXR) and five histone demethylases (HDMases or KDM5b) were examined to determine whether the expression of targeted SAGs was affected. In addition, total chlorophyll and protein levels, as well as plant development were investigated to determine whether these mutants function in regulating plant development and catabolism during senescence. The results show that while the expression of target SAGs were not affected by the mutants, other processes were affected. atx4 was observed to have higher levels of chlorophyll and protein, which indicates ATX4 may function in positively regulating catabolism during leaf senescence. Additionally, kdm5b_1 was observed to have larger seeds, which indicates KDM5b_1 may function to restrict seed size in Arabidopsis.
Li, Xiaona. "Mass spectrometry-based metabolomics study on KRAS-mutant colorectal cancer and rheumatoid arthritis." HKBU Institutional Repository, 2018. https://repository.hkbu.edu.hk/etd_oa/540.
Повний текст джерелаBégot, Laurent. "Caractérisation du mutant dal1-2 d'Arabidopsis thaliana, affecté dans le développement précoce du chloroplaste." Université Joseph Fourier (Grenoble ; 1971-2015), 1999. http://www.theses.fr/1999GRE10054.
Повний текст джерелаКоваль, Вікторія Миколаївна. "Генетичний поліморфізм шиншил". Магістерська робота, ЗНУ, 2020. https://dspace.znu.edu.ua/jspui/handle/12345/1394.
Повний текст джерелаUA : Робота викладена на 82 сторінках друкованого тексту, містить 14 таблиць та 12 рисунків. Перелік посилань включає 52 джерела. Об’єктом дослідження була приватна колекція шиншил. Мета роботи – оцінити генетичний потенціал приватної колекції шиншил за рівнем поліморфізму забарвлення. Методи дослідження – аналіз наукової літератури, гібридологічний аналіз та генетичне прогнозування. Складений та систематизований визначник алелів забарвлення шиншили. Проведено генетичний опис приватної колекції шиншил та складено генетич-ний портрет 15 тварин. Визначено особливо генетично цінні форми –гомозиготний бежевий, сапфір та гомобежевий фіолет. Підтверджено генетичну чистоту форм колекції та визначено перспективні напрямки проведення гібри-дизації з метою розкриття та реалізації генетичного потенціалу. Проаналізовано роботу генетичного калькулятора для розрахунку можливих забарвлень шин-шил, підтверджено його зручність як інструменту для зручного та швидкого моделювання можливих варіантів забарвлення за умови знання генотипів бать-ків. Отримані результати можуть бути використані як посібник для ефектив-ної роботи з штучними популяціями шиншил.
EN : The work is presented at 82 pages of printed text, containing 14 tables and 12 figures. The list of references includes 52 sources. The object of the study was a private collection of chinchillas. The purpose of this work was to evaluate the genetic potential of a private chinchilla collection by the level of color polymorphism. Research methods – scientific literature analysis, hybridological analysis and genetic prediction. The determinant of the chinchilla color alleles was compiled and organized. A genetic description of a private collection of chinchillas was conducted and a genetic portrait of 15 animals was completed. Particularly genetically valuable forms have been identified – homozygous beige, sapphire, and homobeige violet. The genetic purity of collection forms has been confirmed and promised directions for hybridization to identify and realize the genetic potential. The work of the genetic calculator for calculating the possible color of chinchillas is analyzed, and its convenience as a tool for the convenient and fast modeling of possible color variants, provided the knowledge of the parents' genotypes is confirmed. The obtained results can be used as a guide for effective work with artificial chinchilla populations.
Bennett, William R. "Characterisation of two developmentally important genes mutated by transgene insertion in the laboratory mouse." Thesis, University of Bath, 1999. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.301968.
Повний текст джерелаCharles, Tysheena Perkins. "Unraveling the phenotype of colicin cytoplasmic import (cim) mutants." [College Station, Tex. : Texas A&M University, 2008. http://hdl.handle.net/1969.1/ETD-TAMU-2321.
Повний текст джерелаAlaynick, William Arthur. "Phenotypic characterization of estrogen-related receptor gamma mutant mice." Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2006. http://wwwlib.umi.com/cr/ucsd/fullcit?p3235013.
Повний текст джерелаTitle from first page of PDF file (viewed December 6, 2006). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.
RANERI, MATTEO. "PLEIOTROPIC PHENOTYPE OF PSEUDOMONAS AERUGINOSA MUTANTS DEFECTIVE IN GLUCOSE UPTAKE." Doctoral thesis, Università degli Studi di Milano, 2019. http://hdl.handle.net/2434/618277.
Повний текст джерелаPseudomonas aeruginosa is an opportunistic pathogen causing a wide range of infections in humans. Pathologies leading to hyperglycaemia have been associated with augmented risk of developing serious P. aeruginosa infections due to the ability of the bacterium to utilize glucose as carbon source for the growth. Therefore, preventing the import of glucose might be a good strategy to develop anti-pseudomonas drugs. To address this hypothesis, a collection of single and multiple glucose uptake defective mutants was generated in P. aeruginosa PAO1 and tested for virulence-related phenotypes in in vitro assays and in vivo, in two different infection models. In particular, we engineered mutants in genes encoding inner membrane (IM) proteins involved in the internalization of glucose and its oxidized derivatives gluconate and 2-ketogluconate, i.e. the Glt, GntP and KguT transporters. A triple mutant lacking these transporters was demonstrated to be completely unable to grow on glucose as sole carbon source (Glucose Uptake Null mutant, GUN). The transcriptomic analysis revealed a strong divergence in the GUN transcriptional profile relative to the parental strain, with more than 500 differentially expressed genes, controlling both metabolic functions and virulence traits. Consistent with the transcriptomic data, the GUN mutant showed a pleiotropic phenotype in the in vitro assays, with a reduction in biofilm formation and an increased secretion of proteases, pyocyanin and pyoverdine. Furthermore, the production of quorum sensing signal molecules was altered in this mutant. Interestingly, the gene expression profile and most phenotypic traits differ between GUN and the parental strain irrespective of the presence of glucose, suggesting a possible additional role for the deleted transporter(s). Finally, the in vivo assays demonstrated that while the virulence of all mutants in the Caenorhabditis elegans infection model was essentially comparable to that of the wild type strain, all mutants lacking kguT (i.e. the 2-ketogluconate transporter gene) and especially the double ΔgntP ΔkguT and GUN mutants, were attenuated in the Galleria mellonella model. This suggests that targeting glucose metabolism with specific drugs may alter P. aeruginosa pathogenicity depending on the ability of this pathogen to adapt to the specific nutritional context encountered at the site of infection. Therefore, a deeper knowledge of host-pathogen metabolic transactions is pivotal to develop effective antibacterial strategies based on hampering bacterial metabolic functions.
Zhao, Yuming. "Phenotypic analysis of osteoclast lineage in c-fos mutant mice." Thesis, King's College London (University of London), 2003. https://kclpure.kcl.ac.uk/portal/en/theses/phenotypic-analysis-of-osteoclast-lineage-in-cfos-mutant-mice(fafcec7f-6480-4f8c-87b6-3cca60a475fb).html.
Повний текст джерелаWebster, Margaret Ann. "Floral morphogenesis in Primula : inheritance of mutant phenotypes, heteromorphy, and linkage analysis." Thesis, University of Leeds, 2005. http://etheses.whiterose.ac.uk/11275/.
Повний текст джерелаRoth, Ronelle. "Phenotypic characterization of maize bundle sheath defective mutants." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.339349.
Повний текст джерелаFrostell-Birge, Malin, and Mia Skocic. "Phenotypic Characterization of hns mutants of Aggregatibacter actinomycetemcomitans." Thesis, Umeå universitet, Tandläkarutbildning, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-97867.
Повний текст джерелаGautam, Deepshila, Imdadul Haq, and Aruna Kilaru. "Phenotypic Characterization of FAAH Mutants in Physcomitrella Patens." Digital Commons @ East Tennessee State University, 2020. https://dc.etsu.edu/etsu-works/7733.
Повний текст джерелаGuatam, Deepshila, Imdadul Haq, and Aruna Kilaru. "Phenotypic Characterization of FAAH Mutants in Physcomitrella Patens." Digital Commons @ East Tennessee State University, 2020. https://dc.etsu.edu/etsu-works/7734.
Повний текст джерелаWong, Kung-yen Corinne, and 黃共欣. "Analysis of abnormal phenotypes of Hoxb3 mouse mutants generated by gene targeting." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B29158904.
Повний текст джерелаDean, Deyrick Osmond. "Isolation and phenotypic characterisation of deletion mutants of dnaK." Thesis, University of East Anglia, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.292657.
Повний текст джерелаKennedy, Daniel Edward II. "Phenotypic Characterization and Gene Expression Analyses of a Penicillium marneffei Septin Mutant." Youngstown State University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=ysu1340653587.
Повний текст джерелаRobins, Tiina. "Functional and structural studies on CYP21 mutants in congenital adrenal hyperplasia /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-529-1/.
Повний текст джерелаWeaver, Jun Eon. "Characterizing phenotypes of Pichia pastoris mutants that show enhanced secretion of recombinant proteins." Scholarly Commons, 2014. https://scholarlycommons.pacific.edu/uop_etds/188.
Повний текст джерелаKryzskaya, Darya. "Genetic interactions between suppressors of the slow defecation of phenotype of clk-1 mutants." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=106585.
Повний текст джерелаPlusieurs des gènes impliqués dans l'utilisation des nutriments et de l'énergie ainsi que pour leur stockage sont conservés de l'humain à C. elegans. Un des gènes impliqué dans la respiration mitochondriale du nématode est clk-1. Ce gène encode pour une enzyme requise à la synthèse d'ubiquinone. Un des phénotypes des mutants clk-1 est une altération du cycle de défécation. Chez ces mutants, le cycle de défécation est ralenti et celui-ci ne s'accélère pas à des températures plus élevées, comme observé chez le nématode sauvage. Ces phénotypes sont indépendants l'un de l'autre. Plusieurs résultats expérimentaux suggèrent que le ralentissement du cycle de défécation chez les mutants clk-1 est du à des altérations du métabolisme lipoprotéines/cholestérol car ce phénotype peut être supprimé par la réduction du cholestérol provenant de la diète, par l'administration de composés affectant le métabolisme des lipoprotéines et par mutation des gènes impliqués dans le métabolisme des lipoprotéines. Ces mutations peuvent êtres divisées en 2 classes : celles qui suppriment le ralentissement du cycle de défécation à 20oC aussi bien qu'à 25oC (classe I) et celles qui suppriment ce phénotypes seulement à 20oC (classe II). La mutation dsc-4 chez le nématode (impliqué dans l'assemblage des lipoprotéines) ainsi que la mutation dsc-3 (impliqué dans le métabolisme des molécules de type bile) agissent comme suppresseurs de classe I. Chacune de ces deux mutations restaure la capacité des mutants clk-1 à réagir à des changements de température. Dans cette étude, nous avons identifié un nouveau suppresseur de classe I, pgp-2 et classifié sod-4 comme étant un suppresseur de classe II. pgp-2 encode une protéine essentielle à la biosynthèse des organelles intestinales de type lysosome. Les mutations pgp-2 et sod-4 combinées ne suffisent pas à restaurer la capacité de clk-1 à répondre aux changements de température. La combinaison de différentes mutations de classe I ainsi que des mutations de classe I et la mutation de classe II, sod-4, nous a démontré que les interactions génétiques entre les différents mutants sont difficiles à prévoir.
Barman, Soumi. "Construction and Senescence Phenotype Analysis of Double Mutants Encoding H3K4me3 Methyltransferases in Arabidopsis thaliana." Thesis, California State University, Long Beach, 2017. http://pqdtopen.proquest.com/#viewpdf?dispub=10257592.
Повний текст джерелаLeaf senescence, which is the final process of leaf development, involves a complex regulation of thousands of genes to recover and recycle valuable nutrients and mobilize them to growing part of the plant for high yield of fruits and grains. A greater understanding of the complex senescence gene regulation could be helpful for higher crop yield. This study is focused on three genes (ATX1, ATX3 and ATX4) that code for H3K4me3 methyl transferases to investigate their effect on flowering transition time, and their importance during senescence by assaying total chlorophyll and protein levels, and quantifying the mRNA expression of senescence marker gene WRKY75. An additive early flowering phenotype was observed for double mutants. However, no senescence alteration was found for double mutants. An increased level of total chlorophyll was shown by single mutant atx4. Significant differences for total protein were observed in leaf 6 vs. leaf 7 for double mutants atx1atx3 and atx1atx4, suggesting a faster protein degradation rate or smaller variability (reduced confidence interval) in leaf 7 data. Due to the gene redundancy of the ten-member ATX family, knocking out two genes may not adequately affect the function of H3K4 methyltransferase activity. Therefore, phenotypic analyses of triple and quadruple mutants of senescence-expressed H3K4me3 methyltransferase coding genes may show stronger senescence phenotypes. Of importance, these data show that significantly early flowering does not dictate early leaf senescence.
Lam, Sonya Hoan Linh. "Neu tyrosine autophosphorylation site mutants exhibit similar and distinct mammary tumour phenotypes." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=112529.
Повний текст джерелаSanderson, Micheline. "Genetic and phenotypic characterisation of an Arabidopsis thaliana jasmonic acid signalling mutant, jam2." Master's thesis, University of Cape Town, 2003. http://hdl.handle.net/11427/8590.
Повний текст джерелаJasmonic acid (JA) is a plant hormone with diverse functions, ranging from development to stress responses. Moreover, a role for JA in mediating defence against pathogen attack has been established, seemingly specific against necrotrophic pathogens such as Botrytis cinerea. Despite these known roles of JA, it is not known exactly how JA activated downstream responses, such as induced gene expression. To further our understanding of JA signalling, this work aimed to identify new components involved in JA signal transduction. A novel screening method based on lack of anthocyanin accumulation after exogenous application of the methyl ester of JA, methyl jasmonate (MeJA), was employed. A recessive, monogenic mutant, jasmonic acid modified2 (jam2), was isolated from T-DNA activation tagged lines and characterized genetically and phenotypically. jam2 was found not to be T-DNA tagged as the T-DNA segregates independently of the mutation. jam2 is unlikely to be an anthocyanin biosynthetic mutant but shows delayed anthocyanin accumulation after exogenous MeJA treatment. Resistance to MeJA root growth inhibition is a phenotype shared by all JA insensitive mutants. Contrary to this, jam2, like Col-0, exhibits stunted root growth on MeJA. The expression of the antifungal peptide, PDF1.2 can be induced by exogenous MeJA treatment. To assess how PDF1.2 expression was affected in jam2, plants were treated with external liquid and vaporous MeJA. Interestingly, the PDF1.2 expression pattern after MeJA application (liquid or gaseous) was biphasic for Col-0, jam2 and jar1. However, compared to Col-0 and jar1, jam2 appeared to be affected in the first induction peak upon liquid MeJA treatment, whilst in the second after gaseous treatment. PDF1.2 expression can also be seen as a marker for JA-mediated defence responses. Upon infection with B. cinerea, jam2 and jar1 showed intermediate resistance and faster PDF1.2 expression, compared to Col-0 and coil-1. These findings suggest that jam2 is possibly involved in temporal regulation of anthocyanin accumulation and PDF1.2 expression after MeJA application and B. cinerea infection. Therefore, jam2 may define a novel component within the JA signalling pathway and further genetic and phenotypic characterisation could confirm this.
Collins, Emma Alice Maria. "Characterisation of in vivo and in vitro phenotypes of Pseudomonas aeruginosa elastase mutants." Thesis, Queen Mary, University of London, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.522328.
Повний текст джерелаKumar, Sudhir. "Molecular genetic and phenotypic analysis of ENU-induced mutant mouse models for biomedical research." Diss., lmu, 2011. http://nbn-resolving.de/urn:nbn:de:bvb:19-134175.
Повний текст джерелаMackenzie, Francesca Emily. "Positional cloning and phenotypic characterisation of the novel hearing-impaired mouse mutant Cloth-ears." Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.442824.
Повний текст джерелаCanas, Simoes Mariana. "Molecular genetic and phenotypic analysis of a new C. elegans MAB mutant, mab-29." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.670160.
Повний текст джерелаHoslin, Angela. "Genetic and phenotypic characterisation of a novel Efl1 mouse mutant of Shwachman Diamond syndrome." Thesis, University of Oxford, 2016. https://ora.ox.ac.uk/objects/uuid:78fdeb8d-ed5c-4bc7-aca2-e71c50df49a0.
Повний текст джерелаTona, Risa. "The Phenotypic Landscape of a Tbc1d24 Mutant Mouse Includes Convulsive Seizures Resembling Human Early Infantile Epileptic Encephalopathy." Kyoto University, 2019. http://hdl.handle.net/2433/242396.
Повний текст джерелаLeyser, Henrietta Miriam Ottoline. "An analysis of fasciated mutants of Arabidopsis thaliana and the role of cytokinin in this phenotype." Thesis, University of Cambridge, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.357803.
Повний текст джерелаGazzard, James. "Genetic and phenotypic analysis of five alleles of the mutant mouse shaker-with-syndactylism (sy)." Thesis, University of Nottingham, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.342067.
Повний текст джерелаWang, An-Li [Verfasser]. "Behavioral phenotypes of a transgenic rat overexpressing the full-length non-mutant human DISC1 gene / An-Li Wang." Düsseldorf : Universitäts- und Landesbibliothek der Heinrich-Heine-Universität Düsseldorf, 2018. http://d-nb.info/1161182756/34.
Повний текст джерелаRodrigues, Tania 1979. "Identification and analysis of new mutations that suppress the slow defecation phenotype of clk-1(qm30) mutants." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=98781.
Повний текст джерелаUnderwood, M. I. "The relationship between ADAMTS13 genotype and phenotype in congenital thrombotic thrombocytopenic purpura and characterisation of ADAMTS13 mutants." Thesis, University College London (University of London), 2015. http://discovery.ucl.ac.uk/1462706/.
Повний текст джерелаNakouzi, Ghunwa Akram. "Genetic and Phenotypic Response of Neural Tube Defect Mouse Mutants to Folic Acid." Case Western Reserve University School of Graduate Studies / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=case1246538783.
Повний текст джерелаDilling, Bradley Paul. "Mapping and phenotypic characterization of temperature sensitive vaccinia virus mutants cts6 and cts9." [Gainesville, Fla.] : University of Florida, 2004. http://purl.fcla.edu/fcla/etd/UFE0004844.
Повний текст джерелаTypescript. Title from title page of source document. Document formatted into pages; contains 51 pages. Includes Vita. Includes bibliographical references.
Kesavan, Jayati. "Genetic analysis and phenotypic characterization of two Shewanella putrefaciens iron reduction-deficient mutants." Thesis, Georgia Institute of Technology, 1998. http://hdl.handle.net/1853/25231.
Повний текст джерелаWahab, Adbul. "Regulation of antibiotic production in Streptomyces coelicolor : phenotypic and transcriptomic analysis of AbsA mutants." Thesis, University of Surrey, 2007. http://epubs.surrey.ac.uk/843422/.
Повний текст джерелаHawkins, Thomas. "The control of central nervous system myelination and the phenotypic characterisation of a novel zebrafish mutant, akineto(u45)." Thesis, University College London (University of London), 2004. http://discovery.ucl.ac.uk/1446716/.
Повний текст джерелаKumar, Sudhir [Verfasser], and Bernhard [Akademischer Betreuer] Aigner. "Molecular genetic and phenotypic analysis of ENU-induced mutant mouse models for biomedical research / Sudhir Kumar. Betreuer: Bernhard Aigner." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2011. http://d-nb.info/1015203213/34.
Повний текст джерелаMcPhee, Scott William John. "Phenotypic characterisation of the tremor mutant and AAV mediated aspartoacylase gene transfer in the rat model of Canavan disease." Thesis, University of Auckland, 2004. http://wwwlib.umi.com/dissertations/fullcit/3136372.
Повний текст джерелаSubscription resource available via Digital Dissertations only.
Mangoli, Maryam. "Molecular and phenotypic characterisation of zebrafish mutants displaying defects in axonal development in the central nervius system." Thesis, University College London (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.408677.
Повний текст джерела