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Книги з теми "Murine Mycobacterium tuberculosis infection"

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Автор: Grafiati

Опубліковано: 10 грудня 2022

Оновлено: 28 січня 2023

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1

O, Thoen Charles, and Steele James H, eds. Mycobacterium bovis infection in animals and humans. Ames, Iowa: Iowa State University Press, 1995.

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2

O, Thoen Charles, Steele James H, and Gilsdorf Michael J, eds. Mycobacterium bovis infection in animals and humans. 2nd ed. Ames, Iowa: Blackwell Pub., 2006.

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3

Venketaraman, Vishwanath, ed. Understanding the Host Immune Response Against Mycobacterium tuberculosis Infection. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-97367-8.

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4

Hasnain, Seyed Ehtesham, Nasreen Z. Ehtesham, and Sonam Grover, eds. Mycobacterium Tuberculosis: Molecular Infection Biology, Pathogenesis, Diagnostics and New Interventions. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-32-9413-4.

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5

Hingley-Wilson, Suzanne Maria. The detection and identification of macrophage genes induced by intracellular infection with mycobacterium tuberculosis. Birmingham: University of Birmingham, 1998.

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6

Alli, Oyebode Armstrong Terry. Development of a RAP-PCR strategy to study mycobacterium tuberculosis gene expression during intracellular infection of macrophages. Birmingham: University of Birmingham, 1998.

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7

Steele, James H., Charles O. Thoen, and Michael J. Gilsdorf. Mycobacterium Bovis Infection in Animals and Humans. Wiley & Sons, Incorporated, John, 2008.

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8

Understanding the Host Immune Response Against Mycobacterium tuberculosis Infection. Springer, 2018.

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9

Understanding the Host Immune Response Against Mycobacterium tuberculosis Infection. Springer, 2018.

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10

Steele, James H., Charles O. Thoen, and Michael J. Gilsdorf. Mycobacterium Bovis Infection in Animals and Humans. Wiley & Sons, Incorporated, John, 2010.

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11

Aston, Stephen, Geraint Davies, and Nick Beeching. Mycobacterial infection other than tuberculosis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0311.

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Анотація:

Mycobacteria are aerobic bacilli with a lipid-rich cell wall and are widespread both in the environment and in animals. Many species within the genus cause disease in humans, most notably those of the Mycobacterium tuberculosis complex, which cause tuberculosis, and Mycobacterium leprae, the causative agent of leprosy. Several other species, termed non-tuberculous mycobacteria, can cause chronic cutaneous, pulmonary, and disseminated infections. This chapter will briefly review infection with non-tuberculous mycobacteria and Mycobacterium leprae.

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12

Mycobacterium tuberculosis Pathogenesis, Infection Prevention and Treatment. MDPI, 2020. http://dx.doi.org/10.3390/books978-3-03936-659-0.

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13

Mycobacterium Tuberculosis: Molecular Infection Biology, Pathogenesis, Diagnostics and New Interventions. Springer Singapore Pte. Limited, 2020.

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14

Cuervo, Lilian María Mederos. Infection for Mycobacterium tuberculosis and Nontuberculous Mycobacteria in the HIV. INTECH Open Access Publisher, 2011.

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15

Hasnain, Seyed Ehtesham, Nasreen Z. Ehtesham, and Sonam Grover. Mycobacterium Tuberculosis: Molecular Infection Biology, Pathogenesis, Diagnostics and New Interventions. Springer, 2019.

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16

Haldar, Pranabashis. Tuberculosis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0130.

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Анотація:

Tuberculosis (TB) is an infectious disease caused by the bacterial organism Mycobacterium tuberculosis. In this context, reference to the word ‘disease’ is important, as TB implies Mycobacterium tuberculosis infection associated with symptoms. Approximately 10% of Mycobacterium tuberculosis infection is manifest as disease. In the large majority of cases, Mycobacterium tuberculosis infection is latent and defined by evidence of a measurable and significant cell-mediated immune response to mycobacterial antigens, in the absence of clinical or radiological evidence of disease. TB may be clinically classified further according to the site of disease. Miliary TB refers to systemic disease that may affect multiple organs.

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17

Joint Commission on Accreditation of Healthcare Organizations., ed. Tuberculosis: Arresting everyone's enemy. Oakbrook Terrace, IL: Joint Commission on Accreditation of Healthcare Organizations, 1996.

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18

Garnett, Ben. Behavioural aspects of bovine tuberculosis (Mycobacterium bovis) transmission and infection in badgers (Meles meles). 2003.

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19

A, Jensen Paul, Centers for Disease Control and Prevention (U.S.), and National Center for HIV, STD, and TB Prevention (U.S.), eds. Guidelines for preventing the transmission of Mycobacterium tuberculosis in health-care settings, 2005. Atlanta, GA: U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, 2005.

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20

David, Schlossberg, ed. Tuberculosis and nontuberculous mycobacterial infections. 5th ed. New York: McGraw-Hill, Medical Pub. Division, 2005.

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21

Hansoti, Bhakti. Pulmonary Tuberculosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0028.

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Анотація:

Mycobacterium tuberculosis (TB) is most commonly known for its manifestations in the lungs; symptoms include fever and chest pain (retrosternal pain and/or dull intracapsular pain). In the reactivation stage of TB, typical symptoms may include cough, weight loss, fatigue, fever, night sweats, chest pain, dyspnea, and/or hemoptysis. Symptoms may remain undiagnosed for several years. Poverty, HIV, and drug resistance are major contributors to the resurging global TB epidemic. Two kinds of tests are used to detect TB: the tuberculin skin test or a TB blood test. These tests only tell you if a person has been infected with the bacteria. The do not differentiate between latent TB infection and active TB. This distinction clinically suspected when the clinical picture of active TB matches with initial investigations (such as acid-fast bacilli stains, chest x-ray, or CT) and is definitively confirmed by the growth of M. tuberculosis in a clinical specimen.

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22

Heyns, Chris. Tuberculosis and parasitic infestations involving the urogenital system. Edited by Rob Pickard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0006.

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Urogenital tuberculosis is caused by Mycobacterium tuberculosis, which evokes a granulomatous tissue reaction leading to caseous necrosis, fibrosis, and eventual calcification. It most commonly presents as cystitis with sterile pyuria but can show many other symptoms and signs requiring a high index of suspicion to make the diagnosis. Schistosomiasis (Bilharzia) affecting the urinary tract is caused by the flatworm Schistosoma haematobium. Humans are infested by contact with fresh water harbouring the intermediate snail host. Echinococcosis (hydatid disease), is caused by the tapeworm Echinococcus granulosis or multilocularis. Human infection results from close contact with the parasite host (usually dogs and sheep). Filariasis, caused by the roundworm Wuchereria bancrofti, is transmitted by mosquito bite

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23

B, Heifets Leonid, and Cynamon Michael H, eds. Drug susceptibility in the chemotherapy of mycobacterial infections. Boca Raton, Fla: CRC Press, 1991.

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24

Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Neurological infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0015.

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This chapter reviews the clinical features and management of meningitis (community-acquired bacterial meningitis and chronic meningitis), Mycobacterium tuberculosis, encephalitis and myelitis, Lyme disease, brain abscess and parameningeal infection, neurological infections in the immunocompromised, fungal infection, parasitic infection, and bacterial neurotoxins.

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25

Eastwood, John, Cathy Corbishley, and John Grange. Mycobacterial infections. Edited by Vivekanand Jha. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0196.

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Анотація:

The genus Mycobacterium contains over a hundred species including the M. tuberculosis complex and M. leprae, the causative agents of, respectively, tuberculosis and leprosy. The many other species are environmental saprophytes, present particularly in free and piped water sources, and some species are causes of opportunist disease in humans, especially in those who are immune compromised.The genitourinary tract is a common site of both primary and post-primary tuberculosis. In most cases of renal tuberculosis there are gross lesions consisting of caseating granulomas from which tubercle bacilli enter the urinary tract, often with the development of secondary lesions in the ureters, bladder, epididymis, and testis. Tuberculous interstitial nephritis is a less common condition with an insidious course and may result in renal failure. The urine is often negative for tubercle bacilli, emphasizing the need for biopsy in those with renal insufficiency.The risk of developing pulmonary or disseminated tuberculosis after infection is greatly enhanced by any form of immune compromise including renal failure and post-renal transplant immunosuppression.

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