Добірка наукової літератури з теми "MRP4 (Multidrug Resistance Associated Protein 4)"
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Статті в журналах з теми "MRP4 (Multidrug Resistance Associated Protein 4)"
Steinbach, Daniel, Susann Wittig, Gunnar Cario, Susanne Viehmann, Angelika Mueller, Bernd Gruhn, Ralf Haefer, Felix Zintl, and Axel Sauerbrey. "The multidrug resistance-associated protein 3 (MRP3) is associated with a poor outcome in childhood ALL and may account for the worse prognosis in male patients and T-cell immunophenotype." Blood 102, no. 13 (December 15, 2003): 4493–98. http://dx.doi.org/10.1182/blood-2002-11-3461.
Повний текст джерелаAnsari, Marc, Geraldine Sauty, Albert Moghrabi, and Maja Krajinovic. "Polymorphisms in Multidrug Resistance-Associated Protein Genes Are Associated with Worse Outcome in Childhood Acute Lymphoblastic Leukemia." Blood 110, no. 11 (November 16, 2007): 1443. http://dx.doi.org/10.1182/blood.v110.11.1443.1443.
Повний текст джерелаDonepudi, Ajay C., Gregory J. Smith, Oladimeji Aladelokun, Yoojin Lee, Steven J. Toro, Marisa Pfohl, Angela L. Slitt, et al. "Lack of Multidrug Resistance-associated Protein 4 Prolongs Partial Hepatectomy-induced Hepatic Steatosis." Toxicological Sciences 175, no. 2 (April 24, 2020): 301–11. http://dx.doi.org/10.1093/toxsci/kfaa032.
Повний текст джерелаHorvah, L. G., L. Ho, J. G. Kench, J. A. Allen, G. L. Scheffer, P. D. Stricker, J. J. Grygiel, R. L. Sutherland, and S. M. Henshall. "Elevated multidrug resistance-associated protein 4 (MRP4) expression in localized prostate cancer—A potential androgen regulated protein." Journal of Clinical Oncology 24, no. 18_suppl (June 20, 2006): 20022. http://dx.doi.org/10.1200/jco.2006.24.18_suppl.20022.
Повний текст джерелаZhang, Jing, Ka-Yun Ng, and Paul C. Ho. "Interaction of Oxazaphosphorines with Multidrug Resistance-Associated Protein 4 (MRP4)." AAPS Journal 12, no. 3 (April 20, 2010): 300–308. http://dx.doi.org/10.1208/s12248-010-9189-x.
Повний текст джерелаHardwick, Rhiannon N., Marina Snellings, Brian C. Ferslew, Yang Lu, and Kim L. R. Browuer. "Tyrosine and aurora kinase inhibitors diminish transport function of multidrug resistance-associated protein (MRP) 4 and breast cancer resistance protein (BCRP)." ADMET and DMPK 4, no. 4 (December 26, 2016): 302. http://dx.doi.org/10.5599/admet.4.4.322.
Повний текст джерелаHayashi, Hisamitsu, Sotaro Naoi, Takayuki Nakagawa, Toru Nishikawa, Hiroyuki Fukuda, Shinobu Imajoh-Ohmi, Ayano Kondo, et al. "Sorting Nexin 27 Interacts with Multidrug Resistance-associated Protein 4 (MRP4) and Mediates Internalization of MRP4." Journal of Biological Chemistry 287, no. 18 (March 12, 2012): 15054–65. http://dx.doi.org/10.1074/jbc.m111.337931.
Повний текст джерелаAnsari, Marc, Géraldine Sauty, Malgorzata Labuda, Vincent Gagné, Caroline Laverdière, Albert Moghrabi, Daniel Sinnett, and Maja Krajinovic. "Polymorphisms in multidrug resistance-associated protein gene 4 is associated with outcome in childhood acute lymphoblastic leukemia." Blood 114, no. 7 (August 13, 2009): 1383–86. http://dx.doi.org/10.1182/blood-2008-11-191098.
Повний текст джерелаMay, M., C. Alejandro, N. Gomez, F. Diez, S. Copsel, J. Iturbe, N. Mohr, N. Fernandez, C. Shayo, and C. Davio. "P-020 Targeting multidrug resistance – associated protein 4 (MRP4/ABCC4) in pancreatic cancer." Annals of Oncology 27 (June 2016): ii6. http://dx.doi.org/10.1093/annonc/mdw199.20.
Повний текст джерелаSassi, Y., S. El Haou, Y. Fromes, N. Mougenot, G. Vandecasteel, P. Lechat, S. Hatem, A. M. Lompre, and J. S. Hulot. "J016 Inhibition of the multidrug resistance-associated protein 4, MRP4 promotes cardiac hypertrophy." Archives of Cardiovascular Diseases 102 (March 2009): S108. http://dx.doi.org/10.1016/s1875-2136(09)72391-5.
Повний текст джерелаДисертації з теми "MRP4 (Multidrug Resistance Associated Protein 4)"
Carillion, Aude. "Physiopathologie de la dysfonction bêta-adrénergique et rôle de la protéine MRP4 au cours du vieillissement, du diabète et du syndrome métabolique." Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066485/document.
Повний текст джерелаThe studies presented in this report looked for a better understanding of the altered response to stimulation of the β-adrenergic receptors in several physiopathological contexts. The first study confirms the alteration of the β-adrenergic response at the cardiomyocyte level in the senescent cardiomyopathy. The role of MRP4 (multidrug resistance associated protein 4) in the reduced inotropic response to isoproterenol is emphasized. The second study evaluates the response to β-adrenoceptors stimulation in the metabolic syndrome and shows mild dysfunction in this context even in obesity associate with diabetes. These functional results are explained by a reduced expression of β1- and β2-adrenergic receptors but no overexpression of β3-adrenoceptor as observed in type 1 diabetes. The third study analyzes the role of atorvastatin on the β-adrenergic response in the diabetic cardiomyopathy and the mechanisms involved by study of the cardiac transcriptome. The inhibition of nitrite oxide production improves the response to β-adrenoceptors stimulation in diabetic heart. The fourth study explained part of the β-adrenergic dysfunction in the diabetic cardiomyopathy by the overexpression of MRP4. The inhibition of this protein restored the response to isoproterenol during diabetic cardiomyopathy. All together the present results carry on with description of the mechanisms involved in the β-adrenergic dysfunction in aging and diabetes and underline the role of MRP4
Lemaire, Laurine. "Étude des propriétés physico-chimiques de la membrane plasmique comme facteurs modulant l'interaction de molécules et des structures protéiques exogènes." Electronic Thesis or Diss., Compiègne, 2022. http://www.theses.fr/2022COMP2713.
Повний текст джерелаThe plasma membrane was often described as a structure delimiting and protecting the cell from its external environment. However, his role is much more complex and multifunctional. The membrane is an exchange platform at the cellular external and internal environments. Many cellular functions are closely related to it, such as migration, transport of molecules, some pathways of metabolic signaling, or the contact with micro-organisms. This thesis focuses on the study of some cellular processes occurring at the membrane interface using a system that can mimic the lipid bilayer properties. This membrane models that allow a precise control of the in vitro conditions, represent a good alternative to the often inconclusive studies on whole cells. Liposomes allow focusing on a particular function or constituent. In this thesis, the use of the biomimetic model was declined for the study of several processes. The mechanisms of adhesion of flagellated bacteria to lipid bilayers were studied as a function of the physical properties of the lipid bilayers. This information is of paramount importance in the context of antibiotic resistance, giving more information for the potential development of alternative therapies. The liposome model was also used for forming proteoliposomes to study of a transmembrane protein, MRP4 (multidrug resistance associated protein). The study of this protein is an issue in multi-drug treatments. Indeed, this protein is widely involved in drug interactions. Finally, the liposome model was used to characterize the interaction with lipid bilayers of molecules with high therapeutic potential: polyphenols. All of this work was done in collaboration with the team of the Prof Patrick Trouillas (INSERM U1248 team, Limoges University Hospital) working on the development of biomimetic cell models in silico
Tan, Kah Poh. "Nuclear Factor (Erythroid 2-like) Factor 2 (Nrf2) as Cellular Protector in Bile Acid and Retinoid Toxicities." Thesis, 2008. http://hdl.handle.net/1807/17287.
Повний текст джерелаТези доповідей конференцій з теми "MRP4 (Multidrug Resistance Associated Protein 4)"
Kochel, Tyler, Xinrong Ma, Namita Kundu, Jocelyn Reader, Olga Goloubeva, and Amy M. Fulton. "Abstract 4132: Multiple drug resistance-associated protein 4 (MRP4): Role in triple negative breast cancer." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-4132.
Повний текст джерелаLeslie, Elaine M., Janet R. Zhou, and Gurnit Kaur. "Arsenic Hepatic Sinusoidal Export is Stimulated by Methylselenocysteine and Mediated by Multidrug Resistance Protein 4 (MRP4/ABCC4)." In ASPET 2023 Annual Meeting Abstracts. American Society for Pharmacology and Experimental Therapeutics, 2023. http://dx.doi.org/10.1124/jpet.122.537850.
Повний текст джерелаKochel, Tyler J., Namita Kundu, Xinrong Ma, Jocelyn Reader, and Amy Fulton. "Abstract 2257: Multiple drug resistance-associated protein 4 (MRP4) may contribute to breast cancer metastasis by exporting the COX-2 product PGE2." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-2257.
Повний текст джерелаKochel, Tyler J., Jocelyn Reader, Namita Kundu, Yanchun Li, Xinrong Ma, Dawn Holt, and Amy Fulton. "Abstract 5119: Multiple drug resistance-associated protein 4 (MRP4) may contribute to breast cancer progression by exporting the COX-2 product PGE2." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5119.
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