Добірка наукової літератури з теми "MiR-204"
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Статті в журналах з теми "MiR-204"
Huang, Zhengbiao, and Tianling Deng. "miR-204-3p Regulates Glioma Cell Biological Behaviors via Targeting Protein Kinase B (AKT1)." Journal of Biomaterials and Tissue Engineering 12, no. 12 (December 1, 2022): 2395–400. http://dx.doi.org/10.1166/jbt.2022.3188.
Повний текст джерелаSun, Xueqin, Shan Su, Guoxiang Zhang, Hong Zhang, and Xiaohui Yu. "MiR-204 suppresses cell proliferation and promotes apoptosis in ovarian granulosa cells via targeting TPT1 in polycystic ovary syndrome." Biochemistry and Cell Biology 97, no. 5 (October 2019): 554–62. http://dx.doi.org/10.1139/bcb-2019-0019.
Повний текст джерелаWang, Zhiguo, Zehui Fang, Runzhang Lu, Hongli Zhao, Tiejun Gong, Dong Liu, Luojia Hong, Jun Ma, and Mei Zhang. "MicroRNA-204 Potentiates the Sensitivity of Acute Myeloid Leukemia Cells to Arsenic Trioxide." Oncology Research Featuring Preclinical and Clinical Cancer Therapeutics 27, no. 9 (September 23, 2019): 1035–42. http://dx.doi.org/10.3727/096504019x15528367532612.
Повний текст джерелаDu, Youyou, Guanghui Liu, Luosha Zhao, and Rui Yao. "Protective Effect of miR-204 on Doxorubicin-Induced Cardiomyocyte Injury via HMGB1." Oxidative Medicine and Cellular Longevity 2020 (November 19, 2020): 1–16. http://dx.doi.org/10.1155/2020/8819771.
Повний текст джерелаSong, Rui, Yufeng Zhai, Lihua Ao, David A. Fullerton, and Xianzhong Meng. "MicroRNA-204 Deficiency in Human Aortic Valves Elevates Valvular Osteogenic Activity." International Journal of Molecular Sciences 21, no. 1 (December 20, 2019): 76. http://dx.doi.org/10.3390/ijms21010076.
Повний текст джерелаSu, Qunxue, Hao Shen, Bei Gu, and Ning Zhu. "miR-204-5p Hampers Breast Cancer Malignancy and Affects the Cell Cycle by Targeting PRR11." Computational and Mathematical Methods in Medicine 2022 (January 27, 2022): 1–10. http://dx.doi.org/10.1155/2022/4010947.
Повний текст джерелаLi, Liang-Qing, Dun Pan, Qun Chen, Sheng-Wei Zhang, Di-Ya Xie, Xue-Lan Zheng, and Hui Chen. "Sensitization of Gastric Cancer Cells to 5-FU by MicroRNA-204 Through Targeting the TGFBR2-Mediated Epithelial to Mesenchymal Transition." Cellular Physiology and Biochemistry 47, no. 4 (2018): 1533–45. http://dx.doi.org/10.1159/000490871.
Повний текст джерелаEstephan, Leonard E., Michael V. Genuardi, Chad M. Kosanovich, Michael G. Risbano, Yingze Zhang, Nancy Petro, Annie Watson, et al. "Distinct plasma gradients of microRNA-204 in the pulmonary circulation of patients suffering from WHO Groups I and II pulmonary hypertension." Pulmonary Circulation 9, no. 2 (March 28, 2019): 204589401984064. http://dx.doi.org/10.1177/2045894019840646.
Повний текст джерелаQu, Liangliang, Zhongqiu Li, and Pinduan Liu. "mir-204-5p Acts as a Tumor Suppressor by Targeting DNM2 in Osteosarcoma Cells." Journal of Healthcare Engineering 2022 (February 9, 2022): 1–7. http://dx.doi.org/10.1155/2022/8944588.
Повний текст джерелаCheng, Yuan, Dandan Wang, Feng Wang, Jing Liu, Baorui Huang, Maria Angeles Baker, Jianyong Yin, et al. "Endogenous miR-204 Protects the Kidney against Chronic Injury in Hypertension and Diabetes." Journal of the American Society of Nephrology 31, no. 7 (June 2, 2020): 1539–54. http://dx.doi.org/10.1681/asn.2019101100.
Повний текст джерелаДисертації з теми "MiR-204"
Bhat, Rajeshwari Subray. "Study of the role of miR-204 in photoreceptor development." Thesis, Open University, 2016. http://oro.open.ac.uk/48207/.
Повний текст джерелаMigliore, Chiara Maria. "RNA-sequencing based identification of microRNA-204 targets." Doctoral thesis, Università degli studi di Trieste, 2011. http://hdl.handle.net/10077/4595.
Повний текст джерелаWith the completion of the sequencing and annotation of hundreds of genomes, and the accumulation of data on the mammalian transcriptome, greater emphasis has been placed on elucidating the function of non-coding DNA and RNA sequences. It is well known that the non-coding portion of the genome can transcribe functional RNAs. Several categories of non-coding RNAs (ncRNAs) have been defined, such as transport RNAs (tRNAs) ribosomal RNAs (rRNAs), small nuclear RNAs (snRNAs) and small nucleolar RNAs (snoRNAs). A larger group of ncRNAs comprises the so-called microRNAs (miRNAs) and long non-coding RNAs serving key regulatory roles. It has been shown that miRNAs directly target a large number of genes, thus affecting significantly major pathways. In my project, I focused on miR-204, a microRNA that is highly conserved from zebrafish to human and located in the sixth intron of the human TRPM3 gene. I sought to identify mir-204 targets by using the Medaka fish (Oryzias latipes), where mir-204 is expressed at very low levels in the nervous system, as a model for perturbation of the mir-204 network. Transient transgenic Medaka fish were produced to knock down and over-express mir-204. Next-generation sequencing was used to sequence the Medaka transcriptome, dissect the putative targets of miR-204, and thus gain further insight about its function. Potential target genes of mir-204 were selected by choosing genes, which presented lower expression in the wild-type (wt) fish than in the knock down, a lower expression in the over-expression than in the wt and, finally, a higher expression in the knock down than in the over-expression. At the same time, I collected a list of putative miR-204 mouse and human targets using the prediction softwares miRanda, PicTar and TargetScan, obtained the Medaka orthologues and verified that the selected genes in Medaka had a statistically significant enrichment in miR-204 targets as compared to the complete set of genes obtained from the RNA-Sequencing approach. The combined RNA-Sequencing and bioinformatics analysis revealed 147 predicted targets of mir-204, which showed a significant enrichment for the axon guidance pathway. In order to confirm this data, real time quantitative PCR has been performed on total RNA from wt and morphant fish. Results showed a higher expression in the knock down fish for 15 out of 25 putative targets (Neo1, Trim71, Ddx3y, Prkar1a, MyoX, Sema3B, Sema3F, Ptprg, Slit2, Epha4, Epha7, Amot, Lpp, Odz4, Jarid2). I further validated these genes by both Q-PCR and luciferase assays. To this aim, I cloned five putative target sequences into the 3’UTR of a luciferase reporter vector (pGL3-TK-luc Promega) to use them in luciferase assays: co-transfection with miR-204 reduced the luciferase activity of Sema3F, belonging to the class of receptors involved upstream of the axon guidance pathway. These results indicate that mir-204 directly targets key genes involved in the axon guidance pathway such as Sema3F in the nervous system. Further validation of the disruption of axon guidance in the transgenic fish has been undertaken in vivo by our collaborators: the experiment demonstrated a clear role of this microRNA in axon path finding during retinal development.
XXII Ciclo
1981
Книги з теми "MiR-204"
Music, Alfred. Cantata No. 204 -- Ich Bin in Mir Vergnugt: Soprano Solo. Alfred Publishing Company, Incorporated, 1985.
Знайти повний текст джерелаBach, Johann Sebastian. Cantatas Nos. 204, Ich bin in mir vergnught (G) and 205 Zerreisset, Zersprenget: Miniature Score, Kalmus Edition. Alfred Publishing Company, 1985.
Знайти повний текст джерелаЧастини книг з теми "MiR-204"
"Abkürzungen." In Das Geschlecht in mir, edited by Gerhard Schreiber, XXIII—XXIV. Berlin, Boston: De Gruyter, 2019. http://dx.doi.org/10.1515/9783110614626-204.
Повний текст джерелаТези доповідей конференцій з теми "MiR-204"
Ryan, Jacqueline M., Amanda Tivnan, Isabella Bray, Joanna Fay, Andrew M. Davidoff, Lorraine Tracey, and Raymond Stallings. "Abstract 130: MiR-204 acts as a tumor suppressor in neuroblastoma through down-regulation of the neurotrophic receptor TrkB." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-130.
Повний текст джерелаKoga, T., K. Migita, M. Umeda, F. Nonaka, S.-Y. Kawashiri, N. Iwamoto, K. Ichinose, et al. "FRI0620 MIR-204-3P inhibits the production of TLR4-related cytokines in familial mediterranean fever by targeting the PIK3 signaling pathway." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.2813.
Повний текст джерела