Дисертації з теми "Migration Adhesion"
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Burthem, John. "Hairy cell adhesion and migration." Thesis, University of Liverpool, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.240394.
Повний текст джерелаSundar, Rajan Vinoth Edal Joseph. "Adhesion and transendothelial migration of cancer cells." Thesis, Université Grenoble Alpes (ComUE), 2016. http://www.theses.fr/2016GREAV065/document.
Повний текст джерелаCancer metastasis is associated with 90% cancer-associated deaths, when cancer cells escape from the primary tumor and form metastatic colonies in secondary sites. Extravasation is an important step in cancer metastasis, where cancer cells carried in blood, adhere and transmigrate through the endothelium. Therefore identifying the key molecules involved during the adhesion process could enable to develop new anticancer cancer drugs able to inhibit the adhesion of cancer cells to the endothelium. We have previously shown that InterCellular Adhesion Molecule-1 (ICAM-1) expressed by endothelial cells is involved in the interactions of bladder cancer cells (BCs) with the endothelium. However the ICAM-1 ligands have never been investigated. In this study, we combined adhesion assays and Atomic Force Microscopy (AFM) to identify the ligands involved and to quantify the forces relevant in such interactions. We report the expression of MUC1 and CD43 on BCs and demonstrate that these ligands interact with ICAM-1 to mediate cancer cell-endothelial cell adhesion in the case of the more invasive BCs. AFM experiments were performed to quantify the force ranges involved by MUC1 and CD43 during their interaction with ICAM-1. AFM measurements combined with a Gaussian Mixture Model showed distinct force ranges for the interaction of ICAM-1 with MUC1 and ICAM-1 with CD43. Furthermore, a detailed analysis of the rupture events suggests that CD43 is strongly connected to the cytoskeleton and that its interaction with ICAM-1 mainly corresponds to force ramps followed by sudden jumps. On the contrary, MUC1 seems to be weakly connected to the cytoskeleton as its interactions with ICAM-1 are mainly associated with the formation of tethers. The forces involved during the transmigration of cancer cells through the endothelium was investigated using Traction Force Microscopy (TFM). Preliminary results showed that tractions exerted by cancer cells during transmigration can be studied and quantified using TFM
Baghdadchi, Negin. "CYTOKINE CONTROL OF GLIOMA ADHESION AND MIGRATION." CSUSB ScholarWorks, 2014. https://scholarworks.lib.csusb.edu/etd/93.
Повний текст джерелаChen, Ning. "Role of cell adhesion molecules in melanoma transendothelial migration." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ58734.pdf.
Повний текст джерелаGolé, Laurent. "Migration of Dictyostelium Amoeba : role of Adhesion and Quorum sensing." Phd thesis, Université Claude Bernard - Lyon I, 2011. http://tel.archives-ouvertes.fr/tel-00846586.
Повний текст джерелаBoespflug, Nicholas. "ATF3 regulates neutrophil migration in mice." University of Cincinnati / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1382372804.
Повний текст джерелаLi, Yi-Yang Cheung H. Tak. "Basement membrane and its components on lymphocyte adhesion, migration, and proliferation." Normal, Ill. Illinois State University, 1992. http://wwwlib.umi.com/cr/ilstu/fullcit?p9234466.
Повний текст джерелаTitle from title page screen, viewed January 27, 2006. Dissertation Committee: H. Tak Cheung (chair), Anthony Otsuka, Alan Katz, Brian Wilkinson, David Weber. Includes bibliographical references (leaves 108-120) and abstract. Also available in print.
Li, ShuShun. "Thrombospondin 1, an autocrine regulator in T cell adhesion and migration." Doctoral thesis, Umeå : Klinisk mikrobiologi, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-599.
Повний текст джерелаCichon, Monika Agnieszka. "Axl regulates adhesion, migration and stem-like properties of cancer cells." Thesis, Queen Mary, University of London, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610930.
Повний текст джерелаVielkind, Susina. "Role of the GTPase Rho in T cell adhesion and migration." Thesis, University College London (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406052.
Повний текст джерелаRabut, Anne. "Regulation of Drosophila E-cadherin mediated adhesion during border cell migration." Université Louis Pasteur (Strasbourg) (1971-2008), 2004. https://publication-theses.unistra.fr/public/theses_doctorat/2004/RABUT_Anne_2004.pdf.
Повний текст джерелаClassic cadherins are major mediators of homophilic cell-cell adhesion during animal development. In many developmental processes cadherin-dependent adhesive interactions between cells are likely to be regulated. A lot has been done to understand how adhesion is regulated in tissue culture experiments, but so far little is known about the relevance of the studied regulatory mechanisms in vivo. We used Drosophila as a model to study these putative regulatory mechanisms during development. Several mutant variants of Drosophila Epithelial cadherin (DE-cadherin) were generated. Their ability to substitute for endogenous DE-cadherin activity was analyzed in multiple cadherin-dependent processes during Drosophila development and oogenesis, in particular during border cell migration, a process that probably requires dynamic adhesion. Using different DE-cadherin variants, I showed that DE-cadherin/p120ctn interaction, DE-cadherin juxtamembrane domain as well as the conserved tyrosines in DE-cadherin cytoplasmic domain are surprisingly all dispensable for DE-cadherin function. DE-cadherin-Db/a-catenin (a-catenin fused after DE-cadherin juxtamembrane domain) partially substitutes for endogenous DE-cadherin in border cells, showing that regulation of the link between DE-cadherin and a-catenin is not strictly required for the migration. DE-cadherin-DCyt/a-catenin (a-catenin fused after DE-cadherin transmembrane domain) is able to mediate adhesion in the follicular epithelium but does not substitute for endogenous DE-cadherin in border cells, suggesting that some regulatory signals important for the migration may be located in DE-cadherin cytoplasmic domain. However, DE-cadherin-DCyt/a-catenin also have some clear subcellular localization defects and it is so far not completely clear if the observed migration defects are really due to lack of adhesion regulation. If this is the case, the precise regulatory mechanisms will still need to be identified
Lamberti, Giuseppina. "A BIOMIMETIC MICROFLUIDIC DEVICE FOR MODELING THE LEUKOCYTE ADHESION/MIGRATION CASCADE." Diss., Temple University Libraries, 2014. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/265841.
Повний текст джерелаPh.D.
There is a clear need for testing targeted drug carrier systems in a more realistic microenvironment where both biochemical interactions and shear forces are present. This is critical both for understanding of the molecular mechanisms involved in this process and during the drug discovery process. Current in vitro models of the leukocyte adhesion cascade cannot be used for real-time studies of the entire leukocyte adhesion cascade including rolling, adhesion and migration in a single assay. In this study, we have developed and validated a novel bioinspired microfluidic device (bMFD) and used it to test the hypothesis that blocking of specific steps in the adhesion/migration cascade significantly affects other steps of the cascade. The bMFD consists of an endothelialized microvascular network in communication with a tissue compartment via a 3 µm porous barrier. Human neutrophils in bMFD preferentially adhered to activated human endothelial cells near bifurcations with rolling and adhesion patterns in close agreement with in vivo observations. Treating endothelial cells with monoclonal antibodies to E-selectin or ICAM-1 or treating neutrophils with wortmannin reduced rolling, adhesion, and migration of neutrophils to 60%, 20% and 18% of their respective control values. Antibody blocking of specific steps in the adhesion/migration cascade (e.g. mAb to E-selectin) significantly downregulated other steps of the cascade (e.g. migration). This novel in vitro assay provides a realistic human cell based model for basic science studies, identification of new treatment targets, selection of pathways to target validation, and rapid screening of candidate agents.
Temple University--Theses
Lee, Eun-ju Yousaf Muhammad. "Development of dynamic substrates for studies of cell adhesion and migration." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2009. http://dc.lib.unc.edu/u?/etd,2284.
Повний текст джерелаTitle from electronic title page (viewed Jun. 26, 2009). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Department of Chemistry." Discipline: Chemistry; Department/School: Chemistry.
Bliss, Katherine Theresa. "Elucidating the Role of Lasp-2 in Cell Adhesion and Migration." Diss., The University of Arizona, 2012. http://hdl.handle.net/10150/255198.
Повний текст джерелаNewe, Abigail Lucy. "Unearthing the molecular mechanisms that govern L-selectin-dependent adhesion and migration." Thesis, King's College London (University of London), 2015. http://kclpure.kcl.ac.uk/portal/en/theses/unearthing-the-molecular-mechanisms-that-govern-lselectindependent-adhesion-and-migration(8f22370e-34b7-447f-98c2-dd1713036a84).html.
Повний текст джерелаMangiante, Lee Elena Taylor Joan M. "The role of focal adhesion kinase in vascular smooth muscle cell migration." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2008. http://dc.lib.unc.edu/u?/etd,2137.
Повний текст джерелаTitle from electronic title page (viewed Feb. 17, 2009). "... in partial fulfillment of the requirements for the degree of Master of Science in the Department of Pathology and Laboratory Medicine." Discipline: Pathology and Laboratory Medicine; Department/School: Medicine.
Deem, Tracy L. "VCAM-1 Signaling in Endothelial Cells for Lymphocyte Migration." University of Cincinnati / OhioLINK, 2004. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1096565014.
Повний текст джерелаTse, Kathy Wan-Kei. "The role of Pyk2 and FAK in B cell migration, adhesion, and spreading." Thesis, University of British Columbia, 2010. http://hdl.handle.net/2429/25041.
Повний текст джерелаKang, Inkyung. "CHANGES IN THE BIOMECHANICAL PROPERTIES OF ENDOTHELIAL CELLS DURING NEUTROPHIL ADHESION AND MIGRATION." Case Western Reserve University School of Graduate Studies / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=case1149881623.
Повний текст джерелаFERRARIS, G. M. SARRA. "NON-INTEGRIN CELL ADHESION TRIGGERS LIGAND-INDEPENDENT INTEGRIN SIGNALING." Doctoral thesis, Università degli Studi di Milano, 2011. http://hdl.handle.net/2434/157459.
Повний текст джерелаChon, John H. "Mediation of vascular smooth muscle cell adhesion and migration by cell surface heparan sulfate glycosaminoglycans." Diss., Georgia Institute of Technology, 1999. http://hdl.handle.net/1853/11315.
Повний текст джерелаImtiaz, Sarah [Verfasser], Philippe [Akademischer Betreuer] Philippe, and Leif [Gutachter] Dehmelt. "Distinct focal adhesion protein modules control the adhesion site segregation and cell migration behavior / Sarah Imtiaz ; Gutachter: Leif Dehmelt ; Betreuer: Philippe Philippe." Dortmund : Universitätsbibliothek Dortmund, 2017. http://d-nb.info/1175625469/34.
Повний текст джерелаHu, Shiqiong. "Mechanics and Dynamics of Cell Adhesion : Experimental Study of the Osteoclasts." Lyon, Ecole normale supérieure, 2010. http://www.theses.fr/2010ENSL0594.
Повний текст джерелаLes ostéoclastes sont des cellules multinucléées, responsables de la résorption osseuse. Quand ils sont déposés sur un substrat, des structures ponctuelles riches en actine, les podosomes, apparaissent et s'assemblent en clusters, anneaux ou ceinture. Nous avons étudié expérimentalement leur fonction et leur dynamique, depuis une population entière jusqu'à l'échelle d'un unique podosome. Sur une population de cellules, des mesures cinétiques montrent que la surface de la cellule A varie comme A ~ K2, où K est le nombre de noyaux ; ce résultat indique une forme aplatie. Par ailleurs, la mesure de quantités qui prennent en compte l'organisation spatiale de l'actine dans la cellule montre que l'organisation des podosomes ne dépend que du temps écoulé après différentiation, et non de K. Dans un seul ostéoclaste, l'observation d'un fort couplage entre l'étalement d'une cellule et la formation des podosomes nous a conduit a suggérer que les podosomes jouent un rôle important dans la mobilité des ostéoclastes. L'analyse de la migration d'ostéoclastes, ainsi que des forces appliquées sur le substrat, montre que la dynamique interne de l'actine dans la cellule est non seulement corrélée avec la migration cellulaire, mais la gouverne. Enfin, afin de comprendre la dynamique interne d'un podosome, nous avons amélioré le modèle de Biben et al. (2005), en prenant en compte d'une part, la polymérisation de l'actine, et d'autre part, la diffusion et la cinétique d'attachement de la gelsoline, une protéine responsable de la coupe des filaments d'actine. Nous montrons que les podosomes sont principalement gouvernés par la dynamique de l'actine, indépendamment de la concentration en gelsoline
Jennifer, Leigh Quizi. "SLK-mediated Phosphorylation of Paxillin Is Required for Focal Adhesion Turnover and Cell Migration." Thèse, Université d'Ottawa / University of Ottawa, 2011. http://hdl.handle.net/10393/20483.
Повний текст джерелаLemma, S. (Siria). "Migration and adhesion associated molecules in lymphoma biology and their potential roles as biomarkers." Doctoral thesis, Oulun yliopisto, 2017. http://urn.fi/urn:isbn:9789526216041.
Повний текст джерелаTiivistelmä Lymfoomat ovat heterogeeninen ryhmä imukudossyöpiä, joista diffuusi suurisoluinen B-solulymfooma (DLBCL) on yleisin alatyyppi. Se on aggressiivinen maligniteetti, jonka insidenssi on noussut viime vuosina. DLBCL potilaiden ennuste on parantunut merkittävästi, mutta yhä osa potilaista menehtyy tautiinsa. DLBCL:n keskushermostorelapsin kliininen merkitys on tänä päivänä aiempaa suurempi. Sekundaarisen keskushermostolymfooman (sCNSL) ja primaarin aivolymfooman (PCNSL) ennusteet ovat nykyhoidoilla huonoja, joten keskushermostorelapsin ennaltaehkäiseminen on tärkeää. Perifeeriset T-solulymfoomat (PTCLs) ovat ryhmä harvinaisia neoplasioita, joka sisältää useita eri alatyyppejä, joiden morfologiset ja immunofenotyyppiset ominaisuudet ovat monimuotoisia ja osin päällekkäisiä. Eri entiteettien indentifiointi on parantunut, mutta PTCL:ien biologinen tietämys on yhä DLBCL:aa heikompaa. PTCL:ien optimaalinen hoito ei ole selvillä ja tätä tautiryhmää on pitkään hoidettu samoilla hoidoilla kuin DLBCL:aa, mutta huonommilla hoitotuloksilla. Tutkimuksen tavoitteena oli löytää huonon ennusteen markkereita, joilla myös pystyttäisiin ennustamaan DLBCL:n keskushermostorelapsia. Aineisto koostui DLBCL, sCNSL, PCNSL ja PTCL näytteistä. Immunohistokemiallisilla värjäyksillä tutkittiin epiteliaalis-mesenkymaalisen transition (EMT) transkriptiotekijöitä (TF), kemokiinireseptoreita sekä adheesioon-, migraatioon ja inflammaatioon assosioituja molekyylejä. Immunoelektronimikroskopialla varmennettiin molekyylien lokalisaatio soluissa. Geeniekspressioprofiloinnilla (GEP) verrattiin kahdentoista hyvän ja huonon ennusteen ryhmään kuuluvan PTCL näytteen välisiä geeniekspressioeroja sekä kahden DLBCL potilaan näytteitä, joista toiselle kehittyi keskushermostorelapsi. EMT TF:ien ekspressiota nähtiin DLBCL ja PTCL näytteissä, joissa niillä myös todettiin olevan ennusteellista merkitystä. PTCL:ssa TF:t pystyivät erottelemaan tautiryhmän, jolla oli oma spesifinen geeniekspressioprofiilinsa. CXCR4, CXCR5, ITGA10, PTEN ja CD44 ekspressio oli erilaista systeemisissä DLBCL tapauksissa verrattuna sCNSL tapauksiin. Edellä mainituilla molekyyleillä näyttää olevan oma roolinsa keskushermostotaudin kehittymisessä ja jos nämä tulokset pystytään vahvistamaan tulevissa tutkimuksissa, johtavat ne toivottavasti kohti keskushermostorelapsiriskin tarkempaa tunnistamista
Ferro, Valerie Anne. "The role of endothelial cells in promoting adhesion, spreading and migration of B16F10 cells." Thesis, University of St Andrews, 1989. http://hdl.handle.net/10023/14067.
Повний текст джерелаTimpson, Paul. "A study examining the role of Rho family GTPases in the intracellular targeting of Src kinase during cell polarisation and migration." Thesis, University of Glasgow, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.248188.
Повний текст джерелаEl-Hariry, Iman Ahmed. "The relationship between FGF/FGFR and E-cadherin/catenin systems in pancreatic adenocarcinoma." Thesis, Imperial College London, 1999. http://hdl.handle.net/10044/1/7552.
Повний текст джерелаPorts, Michael O. "LATERALLY ASSOCIATED PROTEINS MODULATE A6 INTEGRIN CLEAVAGE, A PERMISSIVE PROCESS UTILIZED DURING CANCER METASTASIS." Diss., The University of Arizona, 2009. http://hdl.handle.net/10150/194361.
Повний текст джерелаGarcia, Emilien. "Rôle de la tyrosine kinase SYK dans la régulation du processus métastatique du mélanome." Thesis, Université Côte d'Azur (ComUE), 2016. http://www.theses.fr/2016AZUR4154/document.
Повний текст джерелаThe progression of tumors to metastatic disease involves the loss of metastatic suppressor functions, as it is the case in melanoma. Thus, aberrant cell migration is a key feature of melanoma progression, and is required for metastasis. SYK (Spleen tyrosine kinase) is a cytoplasmic tyrosine kinase that has been implicated in tumor suppression of breast cancer and melanoma. In skin cells, SYK is found expressed in melanocytes but SYK is frequently downregulated in melanoma by epigenetic silencing. We showed previously that its loss has been associated with senescence escape. Whether it also regulates tumor cell migration and subsequent metastasis remains poorly understood. In this work we used gain- and loss-of-function approaches to analyze SYK’s effects on metastatic abilities of human and murine melanoma cells. Respectively, the reexpression or knockdown of SYK results in decreased or increased migration, invasion and metastasis of melanoma cells. Notably, SYK knockdown cells displayed a mesenchymal-like phenotype with upregulation of mesenchymal markers. Our study unveils a novel role for SYK in suppressing integrin-mediated adhesion, both a points of traction and a signaling platform during cell migration, and outlines the importance of SYK inactivation in acquisition of a metastatic phenotype. To clarify the role of SYK in melanoma formation and progression, we have generated a conditional Syk KO mouse model in melanoma based on melanocyte-specific Pten loss and BrafV600E activating mutation. Preliminary results suggest that Syk loss does not accelerate Pten/Braf-driven melanoma formation but leads to deep invasion of Braf/Pten tumor cells into the dermis
Pischedda, F. "THE IGLON FAMILY MEMBER NEGR1 PROMOTES NEURONAL ARBORIZATION AND MIGRATION VIA FGFR2." Doctoral thesis, Università degli Studi di Milano, 2016. http://hdl.handle.net/2434/363912.
Повний текст джерелаXiong, Siyuan. "The role of Activin B and TGFb1 in regulating endometrial cancer cell adhesion and migration." Thesis, University of British Columbia, 2016. http://hdl.handle.net/2429/58581.
Повний текст джерелаMedicine, Faculty of
Obstetrics and Gynaecology, Department of
Graduate
Dawson, Cassandra. "Investigating the role of protein tyrosine phosphatase alpha in focal adhesion dynamics and cell migration." Thesis, University of British Columbia, 2017. http://hdl.handle.net/2429/62137.
Повний текст джерелаMedicine, Faculty of
Experimental Medicine, Division of
Medicine, Department of
Graduate
Smith, Liisa Sundberg Taylor Joan M. "A role for focal adhesion kinase in vascular smooth muscle cell proliferation, migration and differentiation." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2007. http://dc.lib.unc.edu/u?/etd,1258.
Повний текст джерелаTitle from electronic title page (viewed Mar. 26, 2008). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Department of Pathology and Laboratory Medicine." Discipline: Pathology and Laboratory Medicine; Department/School: Medicine.
Bola, Becky Melanie. "The role of Kinesin-1 and Rab Proteins in cell migration and focal adhesion dynamics." Thesis, University of Manchester, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.491863.
Повний текст джерелаRioja, Ana Ysabel. "Cell Adhesion and Migration on NDGA Cross-Linked Fibrillar Collagen Matrices for Tendon Tissue Engineering." Scholar Commons, 2012. http://scholarcommons.usf.edu/etd/4214.
Повний текст джерелаInfante, Elvira. "Role of Rap and Rho GTPases in T-acute lymphoblastic leukaemia cell adhesion and migration." Thesis, King's College London (University of London), 2013. https://kclpure.kcl.ac.uk/portal/en/theses/role-of-rap-and-rho-gtpases-in-tacute-lymphoblastic-leukaemia-cell-adhesion-and-migration(e1c1aff6-96a7-4dc6-9ee5-355bb41b01ee).html.
Повний текст джерелаTanizaki, Hideaki. "Rho-mDia1 pathway is required for adhesion, migration, and T cell stimulation in dendritic cells." Kyoto University, 2011. http://hdl.handle.net/2433/142071.
Повний текст джерелаAbedi, Syeda Husna Bano. "Mechanisms of migration of vascular smooth muscle and endothelial cells : role of the focal adhesion kinase pathway." Thesis, King's College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.286793.
Повний текст джерелаKerjouan, Adèle. "Décodage des fonctions spatio-temporelles de la signalisation Src impliqué dans la migration et l'invasion par une approche optogénétique." Thesis, Université Grenoble Alpes (ComUE), 2018. http://www.theses.fr/2018GREAV040.
Повний текст джерелаCells sense and integrate a multitude of instructional signals from their microenvironment through a diverse set of transmembrane receptors. This information is then collected at intracellular signaling nodes to later disperse down signaling cascades to drive cell fate. How one signaling node can interpret multiple stimuli and spatio-temporally transmit the appropriate information remains poorly understood. The proto-oncogene c-Src is a pleiotropic tyrosine kinase, is one such node essential for driving many cellular processes, such as migration, invasion, degradation, and cell division. We developed a synthetic approach to explore the relationship between SRC structure and the multiplicity of cellular processes it regulates. Our approach resulted in the decoupling of the different modules composing SRC protein to understand how each of them impacts its activity in space and time. Our approach to control multiple state of SRC conformation was the design of an OptoSrc both capable of forming oligomers and to be recruited at the plasma membrane. To do so, we modified SRC structure to be potentially active in the dark and fused it with light sensitive CRY2. Light stimulation induces CRY2 hetero-dimerization with a CIBN anchored at the plasma membrane and its homo-oligomerisation triggering relocalization of OptoSrc at the plasma and/or its oligomerization. This system generated two different type of mobility of OptoSrc inside focal adhesion inducing two different adhesion fates, the formation of invadosome in one case and the formation of lamellipodia on the other
Hamadi, Abdelkader. "Rôle de FAK (Focal Adhesion Kinase) dans le turnover des points d’adhérence durant la migration cellulaire." Université Louis Pasteur (Strasbourg) (1971-2008), 2008. https://publication-theses.unistra.fr/public/theses_doctorat/2008/HAMADI_Abdelkader_2008.pdf.
Повний текст джерелаNg, Lui, and 吳磊. "Actopaxin: a novel regulator of cell migration and invasion in human hepatocellular carcinoma." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B47752610.
Повний текст джерелаpublished_or_final_version
Surgery
Doctoral
Doctor of Philosophy
Comber, Kate. "Investigation into the molecular mechanisms governing Drosophila embryonic hemocyte migration in vivo." Thesis, University of Bath, 2014. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.606669.
Повний текст джерелаSroka, Thomas Charles. "Synthetic Peptide Ligand Mimetics and Tumor Cell Motility." Diss., Tucson, Arizona : University of Arizona, 2005. http://etd.library.arizona.edu/etd/GetFileServlet?file=file:///data1/pdf/etd/azu%5Fetd%5F1325%5F1%5Fm.pdf&type=application/pdf.
Повний текст джерелаBirner, Ulrieke. "Cell adhesion molecule mechanisms for neutrophil and monocyte migration to joints of rats with adjuvant arthritis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape8/PQDD_0017/MQ49315.pdf.
Повний текст джерелаHur, Sung-Sik. "Roles of 3D traction forces in migration and focal adhesion dynamics of bovine aortic endothelial cells." Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2007. http://wwwlib.umi.com/cr/ucsd/fullcit?p3259061.
Повний текст джерелаTitle from first page of PDF file (viewed June 11, 2007). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references (p. 77-79).
Strugnell, Scott Stuart. "The role of tyrosine phosphorylated caveolin-1 in regulating focal adhesion dynamics and cancer cell migration." Thesis, University of British Columbia, 2009. http://hdl.handle.net/2429/15218.
Повний текст джерелаEssex, Rachel R. "Determining the Effects of CD151 and β1 on Tumor Cell Adhesion and Migration". UKnowledge, 2015. http://uknowledge.uky.edu/cme_etds/56.
Повний текст джерелаHamadi, Abdelkader Rondé Philippe. "Rôle de FAK (Focal Adhesion Kinase) dans le turnover des points d'adhérence durant la migration cellulaire." Strasbourg : Université Louis Pasteur, 2008. http://eprints-scd-ulp.u-strasbg.fr:8080/1011/01/HAMADI_Abdelkader3_2008.pdf.
Повний текст джерелаHoover, Ashtyn. "The Role of Small GTPase RhoG in Focal Adhesion Dynamics and Contractility." University of Toledo / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1556712457014336.
Повний текст джерела