Добірка наукової літератури з теми "Methionine supplementation"
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Статті в журналах з теми "Methionine supplementation"
Kalbande, V. H., K. Ravikanth, S. Maini, and D. S. Rekhe. "Methionine Supplementation Options in Poultry." International Journal of Poultry Science 8, no. 6 (May 15, 2009): 588–91. http://dx.doi.org/10.3923/ijps.2009.588.591.
Повний текст джерелаTripodi, Farida, Andrea Castoldi, Raffaele Nicastro, Veronica Reghellin, Linda Lombardi, Cristina Airoldi, Ermelinda Falletta, et al. "Methionine supplementation stimulates mitochondrial respiration." Biochimica et Biophysica Acta (BBA) - Molecular Cell Research 1865, no. 12 (December 2018): 1901–13. http://dx.doi.org/10.1016/j.bbamcr.2018.09.007.
Повний текст джерелаRatriyanto, Adi. "Pola Produksi Telur Puyuh yang Diberi Ransum Disuplementasi Betain dan Metionin." Caraka Tani: Journal of Sustainable Agriculture 33, no. 1 (March 29, 2018): 1. http://dx.doi.org/10.20961/carakatani.v33i1.19354.
Повний текст джерелаHu, Yili, Xiaocui Chai, Jun Men, Shen Rao, Xin Cong, Shuiyuan Cheng, and Zhixian Qiao. "Does Methionine Status Influence the Outcome of Selenomethinione Supplementation? A Comparative Study of Metabolic and Selenium Levels in HepG2 Cells." Nutrients 14, no. 18 (September 8, 2022): 3705. http://dx.doi.org/10.3390/nu14183705.
Повний текст джерелаMeng, Bin, Weina Gao, Jingyu Wei, Lingling Pu, Zhenchuang Tang, and Changjiang Guo. "Quercetin Increases Hepatic Homocysteine Remethylation and Transsulfuration in Rats Fed a Methionine-Enriched Diet." BioMed Research International 2015 (2015): 1–8. http://dx.doi.org/10.1155/2015/815210.
Повний текст джерелаDel Vesco, Ana Paula, Eliane Gasparino, Daiane de Oliveira Grieser, Vittor Zancanela, Maria Amélia Menck Soares, and Adhemar Rodrigues de Oliveira Neto. "Effects of methionine supplementation on the expression of oxidative stress-related genes in acute heat stress-exposed broilers." British Journal of Nutrition 113, no. 4 (January 23, 2015): 549–59. http://dx.doi.org/10.1017/s0007114514003535.
Повний текст джерелаBrouwerv, Ingeborg A., Marijke van Dusseldorp, Marinus Duran, Chris M. G. Thomas, Joseph G. A. J. Hautvast, Tom K. A. B. Eskes, and Régine P. M. Steegers-Theunissen. "Low-dose folic acid supplementation does not influence plasma methionine concentrations in young non-pregnant women." British Journal of Nutrition 82, no. 2 (August 1999): 85–89. http://dx.doi.org/10.1017/s0007114599001221.
Повний текст джерелаWidiyastuti, Titin, and Tri Rahardjo Sutardi. "Amino Acid and Mineral Supplementation in Fermentation Process of Concentrate Protein of Jatropha Seed Cake (Jatropha curcas L.)." ANIMAL PRODUCTION 18, no. 3 (October 10, 2016): 141. http://dx.doi.org/10.20884/1.anprod.2016.18.3.574.
Повний текст джерелаHuisman, J., E. J. van Weerden, P. van Leeuwen, and F. Koch. "Effect of methionine compounds on rumen activity of cows." Netherlands Journal of Agricultural Science 36, no. 2 (May 1, 1988): 111–17. http://dx.doi.org/10.18174/njas.v36i2.16684.
Повний текст джерелаLambert, B. D., C. A. Löest, and Evan C. Titgemeyer. "Effect of methionine supplementation on methionine metabolism in growing cattle." Kansas Agricultural Experiment Station Research Reports, no. 1 (January 1, 2002): 17–19. http://dx.doi.org/10.4148/2378-5977.1696.
Повний текст джерелаДисертації з теми "Methionine supplementation"
Plank, Johanna E. "Methionine and Methionine Analog Supplementation: Comparison of Bioavailability In Dairy Cows and Differential Utilization by Rumen Microbes in Batch Culture." The Ohio State University, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=osu1306946206.
Повний текст джерелаLatham, Christine M. "Effects of Dietary Amino Acid Supplementation on Measures of Whole-Body and Muscle Protein Metabolism in Aged Horses." UKnowledge, 2016. http://uknowledge.uky.edu/animalsci_etds/63.
Повний текст джерелаGarner, Justine. "The effects of 5,10-Methylenetetrahydrofolate Reductase deficiency and Methionine supplementation on the DNA Methylation patterns of early male germ cells." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=110667.
Повний текст джерелаL'acquisition inexacte de patrons de méthylation d'ADN des cellules germinales pendant la période fœtale chez la souris mâle est associée à des désordres de méiose et d'infertilité. L'enzyme MTHFR joue un rôle clé dans le processus de méthylation d'ADN puisqu'elle est impliquée dans une cascade produisant les groupements méthyles nécessaires à la réaction. L'objectif des travaux présentés dans ce mémoire était d'évaluer les profiles de méthylation d'ADN dans les spermatogonies provenant de souris hétérozygotes pour une suppression ciblée de Mthfr (Mthfr+/-), en comparaison avec celles découlant des compagnons de type sauvage de la même portée (Mthfr +/+). Nous avons déterminé que dans les spermatogonies néonatales de souris Mthfr+/+ et Mthfr+/- les patrons de méthylation sont demeurés similaires au niveau des gènes à empreinte ainsi qu'à divers sites intergéniques retrouvés à travers le chromosome 9. Subséquemment nous avons établi un système de culture de cellules souches spermatogoniales (SSC) à partir de souris Mthfr+/+ et Mthfr+/- afin d'examiner la stabilité des profiles de méthylation en culture et de déterminer si un supplément de méthionine peut restaurer un dérèglement de méthylation d'ADN causé par une haploinsuffisance de MTHFR. La période de culture (nombre de passages faible vs élevé) n'a nullement altérée les patrons de méthylation d'ADN, ce qui suggère que cette modification épigénétique est globalement très stable dans les SSCs en culture. Le traitement de ces même SSCs avec un milieu 20 fois plus élevé en méthionine occasionne par contre une augmentation des niveaux globaux de méthylation d'ADN à travers le chromosome 9, démontrant que cette modification post-traductionnelle peut être perturbée en culture. De plus, la culture des SSCs Mthfr+/- dans diverses concentrations de methionine démontre une augmentation significative de la variance des niveaux de méthylation d'ADN pour une multitude de loci à travers le chromosome 9 comparativement aux SSCs Mthfr+/+ soumis aux mêmes traitements. Ensemble, nos résultats suggèrent que les patrons de méthylation d'ADN des SSCs sont normalement stables en culture mais peuvent cependant être perturbés par les niveaux de méthionine et MTHFR.
Fowler, Colleen Marie. "Evaluation of 2-Hydroxy-4-(methylthio) Butanoic Acid Isopropyl Ester and Methionine Supplementation on Efficiency of Microbial Protein Synthesis and Rumen Bacterial Populations." The Ohio State University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=osu1248875016.
Повний текст джерелаGomes, Tarsila Daysy Ursula Hermogenes. "Efeito da concentração de metionina na dieta durante o período pré e pósnatal sobre o estresse oxidativo, a instabilidade genômica e expressão de RNAm de Mat1a, Bhmt e Cbs em camundongos." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/60/60134/tde-10022014-143246/.
Повний текст джерелаMethionine is the main methyl donor for the DNA methylation, a process responsible for gene expression modifications. Since this essential amino acid is required for normal growth and development, variations of this compound in the diet may lead to alterations on DNA stability. Thus, this study aimed to evaluate the effect of deficient and supplemented methionine diets on oxidative stress and genomic instability in mice and their dams treated during pregnancy and lactation, and the expression of Mat1a, Bhmt and Cbs mRNA of transmethylation, remetthylation, and transulfuration pathways, respectively, in dams livers. The dams were divided into three methionine diets groups (control, 0,3% DL-methionine; supplemented, 2,0%; and deficient, 0%) until the end of lactation (10 weeks). For each dams groups, the offspring were subdivided and were also treated with the same diets during 18 weeks after weaning. The parameters evaluated were food intake, body weight, relative liver and kidney weights, and survival of the offspring. Also, it was carried out theevaluation of lipid peroxidation (thiobarbituric acid reactive substances, TBARS), quantification of glutathione (GSH) and catalase activity, and genomic instability (comet assay) in liver and kidneys; andMat1a, Bhmt, and CbsmRNA analysis only in dams liver. The deficient diet resulted in lower food intake and body weights in both phases and reduced the survival of the offspring that were treated with this diet. The supplemented diet reduced the TBARS concentration in both tissues of dams and the deficient diet did not differ. In the offspring, the supplementation reduced liver TBARS, whereas the deficiency raised. The supplementation increased the liver GSH concentration of dams, as well as the deficiency in kidneys. In the offspring, the responses were different; the supplementation reduced the liver GSH, as well as the deficiency, also observed in kidneys. There were no differences of catalase parameter of dams, but there was a reduction in both tissues of the offspring. Both supplementation and deficiency reduced the liver DNA damage of dams, however the supplementation increased and the deficiency reduced the DNA damage of kidneys. In the offspring, both diets increased the DNA damage in both tissues. The methionine supplementation did not differ the mRNA expression and the deficiency only reduced the Cbs, mRNA expression. It was concluded that the methionine supplementation or deficiency did not resulted in oxidative stress in dams, but the supplementation reduced the genomic instability of liver and raised the kidney one. The deficiency resulted in lower genomic instability in both tissues in dams. In the offspring, both methionine supplementation and deficiency presented variation of oxidative stress in both tissues and resulted more genomic instability.
Pestana, Tiago Roberto Gomes. "Methionine supplementation improves the efficacy of breast cancer immunotherapy." Master's thesis, 2018. http://hdl.handle.net/10362/53243.
Повний текст джерелаLeonardi, Claudia. "Effect of methionine supplementation on productive performance of dairy cattle." 2001. http://catalog.hathitrust.org/api/volumes/oclc/48191871.html.
Повний текст джерелаDavidson, Shannon. "Supplementation of rumen-protected forms of methionine, betaine, and choline to early lactation Holstein cows." 2006. http://www.lib.ncsu.edu/theses/available/etd-08162006-141227/unrestricted/etd.pdf.
Повний текст джерела趙文綺. "Effects of Methionine Supplementation on the Glutathione Metabolism of Rats during Long-term High Selenium Consumption." Thesis, 1997. http://ndltd.ncl.edu.tw/handle/49712835627908512556.
Повний текст джерела輔仁大學
食品營養學系
85
The purpose of this study was to investigate the effect of dietary methionine supplementation on glutathione metabolism of rats during long term Se consumption. Male weanling Sprague-Dawley rats were randomly assigned to recive diets containing 3 levels of dietary Se (adequate: 0.1 mg Se/kg diet, moderately high: 3 mg Se/kg diet, high: 5 mg Se/kg diet, as sodium selenite) and 3 levels of dietary methionine supplement (0, 0.3, or 0.6 g Met/100 g diet). Results showed that hepatic and erythrocyte glutathione (GSH) and GSH/GSSG ratio decreased significantly whereas glutathione disulfide (GSSG) increased significantly due to long term excessive Se ingestion. Methionine supplementation significantly elevates hepatic GSH level and GSH/GSSG ratio, and lowered hepatic GSSG level as well as erythrocyte GSH level and GSH/GSSG ratio. In terms of activities of hepatic GSH related enzymes, γ-Glutamylcysteine synthetase activity increased with elevated dietary methionine supplementation. The hepatic glutathione reductase activity increased by long term high Se ingestion without supplementing methionine. Dietary methionine supplement decreased glutathione reductase activity. Long term high Se consumption ignificantly elevated glutathione-S-transferase activity, which was reduced by dietary methionine supplement. Although experimental diets of this study offered 23.2 mg/kg diet vitamin B2, consumption of 5 mg Se/kg diet which no added methionine for 12 weeks still elevated erythrocyte glutathione reductase activity coefficient, probably to the stage of marginal vitamin B2 deficiency. In summary, long term consumption of high Se diet caused growth retardation and hair loss, altered hepatic GSH metabolism and probably raised vitamin B2 requirement of rats. Dietary methionine supplementation may assist in relieving Se toxicity probably increasing GSH supply.
Chareontesprasit, Numchai. "The utilization of roasted full-fat soybean and methionine supplementation in diets for juvenile freshwater prawns Macrobrachium rosenbergii." 1992. http://hdl.handle.net/1993/18000.
Повний текст джерелаЧастини книг з теми "Methionine supplementation"
Leclerc, J. "Effect of methionine supplementation of low protein diets in the rat: A review." In Amino Acids, 1108–13. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-011-2262-7_139.
Повний текст джерелаHuang, Henry H., and E. Hawrylewicz. "Effect of Dietary Protein and Methionine Supplementation on Mammary Tumorigenesis." In Dietary Proteins, 123–50. AOCS Publishing, 1992. http://dx.doi.org/10.1201/9781439831878.ch9.
Повний текст джерела